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Livros sobre o tema "Inhibit"

Autor: Grafiati
Publicado: 4 de junho de 2021
Última modificação: 20 de fevereiro de 2023

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1

Coleman, Michael C. The major factors that inhibit better policing. Dublin: University College Dublin, 1989.

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2

Norgrove, Heather. Professional tribalism: Does it inhibit collaborative working? : differing stakeholder perceptions which inhibit collaborative working across organizational boundaries. [s.l.]: typescript, 1999.

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3

Quantifying competitive ability of perennial grasses to inhibit scotch broom. Portland, OR: U.S. Department of Agriculture, Pacific Northwest Research Station, 2011.

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4

Kibble, Karen-Jane. Growing pains: An analysis of the factors that inhibit growth in micro enterprises. Oxford: Oxford Brookes University, 2000.

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5

Abraham, Katharine G. Does employment protection inhibit labor market flexibility?: Lessons from Germany, France and Belgium. Cambridge, MA: National Bureau of Economic Research, 1993.

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6

Peter, Chalk, ed. Terrorism & development: Using social and economic development to inhibit a resurgence of terrorism. Santa Monica, CA: Rand, 2003.

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7

Hollyoak, Ken. Does the inconsistent application of visibility standards inhibit the development of brownfield sites. Oxford: Oxford Brookes University, 2000.

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8

Clements, Harold William. An analysis of stress absorbing membrane interlayers used to inhibit tensile fatigue reflective cracking. [s.l: The Author], 2001.

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9

Cowey, Lisa. University spinout companies: An investigation of the factors perceived to inhibit sucessful technology transfer. Oxford: Oxford Brookes University, 2004.

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10

Huisken, Ronald. Globalising the INF treaty: The best way to inhibit the proliferation of long-range missiles? Canberra: Strategic and Defence Studies Centre, The Australian National University, 2008.

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11

Evans, Dorothy. Women and management: A study of the factors that inhibit womens entry into management positions. London: NELP, 1985.

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12

Andu, Ademola. A consideration of the extent to which the criminal and civil law inhibit money laundering. Wolverhampton: University of Wolverhampton, 1995.

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13

Huisken, Ronald. Globalising the INF treaty: The best way to inhibit the proliferation of long-range missiles? Canberra: Strategic and Defence Studies Centre, The Australian National University, 2008.

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14

Westlake, Michael John. Addressing marketing and processing constraints that inhibit agrifood exports: A guide for policy analysts and planners. Rome: Food and Agirculture Organization of the United Nations, 2005.

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15

Newell, Graham. The factors which promote or inhibit the process of action research: (case studies in two schools). (UK): (s.n.), 1991.

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16

Tong, Hui. Does trade globalization induce or inhibit corporate transparency?: Unbundling the growth potential and product market competition channels. Cambridge, MA: National Bureau of Economic Research, 2011.

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17

Wong, Philip. The potential of two new inhibitors of mechanisms that regulate intracellular pH, cariporide and S3705, to inhibit tumour cell proliferation. Ottawa: National Library of Canada, 2001.

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18

Csonka, Jacqueline V. Electrical stimulation of tibialis anterior to inhibit the stretch reflex of soleus resulting from passive stretch and gait, and its effect on spasticity. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1992.

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19

Changing behavior in individuals, couples, and groups: Identifying, analyzing, and manipulating the elements involved in change in order to promote or inhibit alteration of behavior. Springfield, Ill: C. Thomas, 1996.

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20

Aono, Toshihiro, Hiromu Sugino e Wylie W. Vale, eds. Inhibin, Activin and Follistatin. New York, NY: Springer New York, 1997. http://dx.doi.org/10.1007/978-1-4612-1874-6.

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21

Commission, Virginia Marine Resources. Report of the Virginia Marine Resources Commission on an analysis of organic statutes and regulations which inhibit shellfish aquaculture operations to the Governor and the General Assembly of Virginia. Richmond: Commonwealth of Virginia, 1996.

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22

Matsukuru, Makoto. Phosphorothioate analogs of oligodeoxynucleotide inhibit viral replication of human immunodeficiency virus (HIV): Inhibition of de novo infection in uninfected cells and regulation of viral expression in chronically infected cells. [Bethesda, Md.?]: National Cancer Institute, Office of Cancer Communications, 1988.

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23

T, Addison John, ed. Do works councils inhibit investment? Bonn, Germany: IZA, 2005.

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24

Meyers, Coby V., e Marlene Darwin. Enduring Myths That Inhibit School Turnaround. Information Age Publishing, Incorporated, 2017.

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25

Meyers, Coby V., e Marlene Darwin. Enduring Myths That Inhibit School Turnaround. Information Age Publishing, Incorporated, 2017.

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26

Hofmann, Ron. Using ammonia to inhibit bromate formation during ozonation. 2000.

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27

Mattoo, Aaditya, Arvind Subramanian, Dominique van der Mensbrugghe e Jianwu He. Can Global De-Carbonization Inhibit Developing Country Industrialization ? The World Bank, 2009. http://dx.doi.org/10.1596/1813-9450-5121.

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28

Liu, Xiaoshun. Multiple Approaches to Inhibit Restenosis After Percutaneous Coronary Interventions. Leuven Univ Pr, 2004.

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29

Lu, Shennan. Retinoids can inhibit cell growth through AP-1-independent pathways. [s.n.], 2000.

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30

Mansfield, Ian David. The synthesis of novel agents which inhibit tumour-stimulated bone resorption. 1999.

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31

Normal development of five attentional processes: Focus, select, sustain, shift, and inhibit. Ottawa: National Library of Canada, 1996.

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32

Gajda, John. Development of a Cement to Inhibit Alkali-Silica Reactivity (Research and Development Bulletin). Portland Cement Association, 1996.

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33

Schlag, G. Schlag: First Vienna Shock Forum-Pathophysiologi Cal Role of Med & Med Inhibit in Shock. John Wiley & Sons Inc, 1987.

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34

Cragin, Kim, e Peter Chalk. Terrorism and Development: Using Social and Economic Development to Inhibit a Resurgence of Terrorism. RAND Corporation, 2003. http://dx.doi.org/10.7249/mr1630.

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35

Barrere-Lemaire, Stéphanie, Sarawut Kumphune e Christophe Piot, eds. New Strategies to Inhibit Cell Death in Myocardial Ischemia-Reperfusion Injury: How to succeed? Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88976-653-6.

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36

Kavsak, Peter. Smurfs are E3 ubiquitin protein ligases that inhibit the transforming growth factor beta signalling pathway. 2001.

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37

Cragin, Kim. Terrorism and Development: Using Social and Economic Development Policies to Inhibit a Resurgence of Terrorism. RAND Corporation, 2003.

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38

Fleischmann, Roy. Signalling pathway inhibitors. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0081.

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Oral, small-molecule signalling pathway inhibitors, including ones that inhibit the JAK and SyK pathways, are currently in development for the treatment of rheumatoid arthritis (RA). Tofacitinib is an orally administered small-molecule inhibitor that targets the intracellular Janus kinase 3 and 1 (JAK1/3) molecules to a greater extent than JAK2 while baricitinib (formerly INCB028050) predominantly inhibits JAK1/2. Many of the proinflammatory cytokines implicated in the pathogenesis of RA utilize cell signalling that involves the JAK-STAT pathways and therefore inhibition of JAK-STAT signalling, by targeting multiple RA-associated cytokine pathways, has the potential to simultaneously reduce inflammation, cellular activation, and proliferation of key immune cells. Fostamatinib disodium is an orally available inhibitor of spleen tyrosine kinase (SyK), which is a cytoplasmic tyrosine kinase that is an important mediator of immunoreceptor signalling in mast cells, macrophages, neutrophils, and B cells. Interruption of SyK signalling may interrupt production of tumour necrosis factor (TNF) and metalloproteinase and therefore affect RA disease activity. Tofacitinib has been investigated in multiple phase 2 and phase 3 trials which have investigated its efficacy (clinical, functional, and radiographic) and safety in patients who have failed disease-modifying anti-inflammatory drugs (DMARDs) as monotherapy or in combination with DMARDs, compared to an inhibitor of tumour necrosis factor alpha (TNFα‎) and in patients who have failed TNFα‎ inhibitors. The efficacy of fostamatinib and baricitinib has been investigated in phase 2 trials; both are in large phase 3 clinical programmes. Each of these medications has demonstrated efficacy; their safety profile has been shown to be different from each other and from currently approved biological agents. This chapter discusses what is currently known and understood about their efficacy and safety.
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39

Nofzinger, Kirk Allen. The ability of drinking water contaminants to inhibit phorbol esters from binding to protein kinase C. 1987.

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40

Trachtman, Les. Don't Mess It Up: How Founders and Their Successors Can Avoid the Clichés That Inhibit Growth. River Grove Books, 2018.

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41

Addressing Marketing And Processing Constraints That Inhibit Agrifood Exports: A Guide for Policy Analysis And Planners. Food & Agriculture Org, 2006.

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42

Mease, Philip. Biologic treatments for psoriatic arthritis apart from TNF inhibition. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198737582.003.0030.

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Psoriatic arthritis (PsA) is an immunologically mediated inflammatory disease characterized by arthritis, enthesitis, dactylitis, spondylitis, and psoriasis. Prior to the introduction of targeted biologic medications, such as TNF inhibitors, the ability to control disease activity was limited, with only modest effects noted with traditional oral medications such as methotrexate and sulfasalazine. The introduction of TNF inhibitors substantially changed the outlook of PsA patients, yielding significant response in all relevant clinical domains and demonstrating the ability to inhibit progressive structural damage of joints. However, not all patients responded to these agents and many patients displayed initial response which waned over time, partly due to immunogenicity (development of antibodies which blocked full therapeutic effect of the biologic protein), or because of tolerability and side effect issues. Thus, it has been important to develop new medicines which target other key cytokines and immunologic pathways. Several medicines with a different mechanism of action have been approved or are in development for the treatment of PsA. Ustekinumab inhibits both IL12 and IL23 and thus is felt to work in both the TH1 and TH7 pathways of inflammation. The oral medicine apremilast inhibits phosphodiesterase 4, thus modulating the cyclic AMP pathway in immunologic cells, yielding an anti-inflammatory effect. Both of these medicines have been approved for the treatment of PsA as well as psoriasis. An emerging group of therapies, the IL17 inhibitors, has demonstrated significant effectiveness in psoriasis and PsA and one of these, Secukinumab, has been approved for psoriasis, PsA, and AS. Other medicines in development include the co-stimulatory blockade agent, abatacept, oral Janus Kinase (JAK) inhibitors, and an emerging group of therapies which inhibit IL23. As modulators of immune cell function, these agents have the potential to increase risk for infection, as well as other side effects. These must be discussed with the patient and considered when determining overall risk benefit analysis regarding their use. The emergence of medicines with a different mechanism of action than TNF inhibition has broadened and strengthened our ability to effectively treat PsA.
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43

Crass, Scott. Beloved Workhorses: How Not Pursuing Fame Did Not Inhibit U. S. House Members from Effectiveness and Likability. Xlibris Corporation LLC, 2021.

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44

Crass, Scott. Beloved Workhorses: How Not Pursuing Fame Did Not Inhibit U. S. House Members from Effectiveness and Likability. Xlibris Corporation LLC, 2021.

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45

Schleicher, Andreas. The Impact of Research in Education. Editado por Ben Levin, Jie Qi, Hilary Edelstein e Jacqueline Sohn. Bristol University Press, 2013. http://dx.doi.org/10.46692/9781447306214.

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This much-needed, original book analyzes efforts and systems in nine countries to mobilize research knowledge, describing the various factors that support or inhibit that work to provide an unprecedented view of the way education research is produced and shared.
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46

Ellison, David H., e Arohan R. Subramanya. Clinical use of diuretics. Editado por Robert Unwin. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0033.

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Diuretics are widely employed to treat extracellular fluid volume expansion caused by heart failure, cirrhosis of the liver, nephrotic syndrome, and chronic kidney disease. Major classes of diuretic inhibit sodium reabsorption along the proximal tubule, the loop of Henle, the distal convoluted tubule, and the connecting and collecting tubules. Loop diuretics have the highest ceiling of action and often form the cornerstones of diuretic treatment of oedema. Members of this class are short-acting drugs, with different bioavailabilities, the specifics of which contribute importantly to a rational and effective approach to their use. They are not filtered substantially because they are all protein bound. They enter tubules by secretion along the proximal tubule, thereby gaining access to the Na-K-2Cl cotransporter of the thick ascending limb. Their dose–response curves are sigmoidal and altered by several disease processes. Chronic administration can elicit adaptive processes along the nephron that limit their efficacy. Distal convoluted tubule diuretics, such as the thiazides, inhibit NaCl absorption along the distal convoluted tubule. While used predominantly to treat hypertension, they are also useful to treat oedema, especially when combined with loop diuretics. Drugs acting along the connecting tubule and collecting duct either inhibit Na+ channels directly or block mineralocorticoid receptors. These drugs are effective in states of very high aldosterone secretion, and can also be used to reduce the hypokalaemia caused by other classes of diuretics. An evidence-based approach to treating the oedematous patient is described.
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47

Shah, Idries. Overcoming Assumptions that Inhibit Spiritual Development : A Lecture Delivered Before a Live Audience, Plus Teaching Stories and Narratives. Hoopoe Books, 2000.

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48

Ellison, William. Ready, steady, go: The corridor's propects for growth look promising but land supply could inhibit some M62 towns. 1997.

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49

Jarrold, Christopher. Pretend play in autism: executive explanations. Oxford University Press, 2015. http://dx.doi.org/10.1093/med:psych/9780198523499.003.0004.

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This chapter discusses executive explanations of pretend play in autism. This includes problems with the metarepresentational-deficit account, if an executive dysfunction explanation is plausible, evidence for the role of executive deficits (failures to inhibit and to generate), generativity deficits, and how a generativity deficit fits with notions of executive dysfunction.
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50

Kabir, Mahfuz, Surendar Singh e Michael J. Ferrantino. The Textile-Clothing Value Chain in India and Bangladesh: How Appropriate Policies Can Promote (or Inhibit) Trade and Investment. World Bank, Washington, DC, 2019. http://dx.doi.org/10.1596/1813-9450-8731.

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