Bibliografias temáticas / Intestine, Small Immunology

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Literatura científica selecionada sobre o tema "Intestine, Small Immunology"

Autor: Grafiati
Publicado: 4 de junho de 2021
Última modificação: 28 de janeiro de 2023

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Artigos de revistas sobre o assunto "Intestine, Small Immunology"

1

Bland, P. W., and M. Bailey. "Immunology of the small intestine." Transplantation Proceedings 30, no. 6 (September 1998): 2560–61. http://dx.doi.org/10.1016/s0041-1345(98)00725-8.

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Kunisawa, Jun, Yosuke Kurashima, Morio Higuchi, Masashi Gohda, Izumi Ishikawa, Ikuko Ogahara, Namju Kim, Miki Shimizu, and Hiroshi Kiyono. "Small and large intestinal intraepithelial T lymphocytes show distinct dependency on sphingosine 1-phosphate (42.11)." Journal of Immunology 178, no. 1_Supplement (April 1, 2007): S35. http://dx.doi.org/10.4049/jimmunol.178.supp.42.11.

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Abstract It is known that the composition of intraepithelial T lymphocyte (IEL) differs between small and large intestines, but the mechanism underlying that difference remains obscure. Here, we show that sphingosine 1-phosphate (S1P) plays a key role in regulating intestinal IEL trafficking into the small and large intestines. High levels of type 1 S1P receptor (S1P1) expression was noted on naïve IELs expressing CD4 or CD8αβ, which leads to their preferential migration into the large intestine. In contrast, recent thymic emigrants (RTEs), double-positive thymocytes, and double-negative thym
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Brandl, Katharina, George Plitas, Ronald P. DeMatteo, Laura V. Hooper та Eric G. Pamer. "MyD88-mediated signals induce in vivo production of the bactericidal lectin RegIIIγ and protect against intestinal Listeria monocytogenes infection (44.4)". Journal of Immunology 178, № 1_Supplement (1 квітня 2007): S48. http://dx.doi.org/10.4049/jimmunol.178.supp.44.4.

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Abstract Listeria monocytogenes (L. monocytogenes) is an intracellular bacterium that causes systemic infections after traversing the intestinal mucosa. To test the hypothesis that the Toll-like receptor pathway is involved in defense against intestinal L. monocytogenes infection, we analyzed the role of the common intracellular adaptor molecule myeloid differentiation primary-response protein 88 (MyD88) following oral infection with L. monocytogenes. We found that MyD88 deficient mice have increased susceptibility to intestinal L. monocytogenes infection with higher bacterial burden in spleen
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Chowers, Y., W. Holtmeier, J. Harwood, E. Morzycka-Wroblewska, and M. F. Kagnoff. "The V delta 1 T cell receptor repertoire in human small intestine and colon." Journal of Experimental Medicine 180, no. 1 (July 1, 1994): 183–90. http://dx.doi.org/10.1084/jem.180.1.183.

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V delta 1 bearing T cells comprise the major population of gamma/delta T cells in the human intestinal tract. To gain insight into mechanisms involved in the generation of these cells and the diversity of their repertoire, we have characterized the junctional sequences of V delta 1 T cell receptor transcripts in the human small intestine and colon. Mucosal biopsies obtained from defined regions along the length of the small intestine or colon contained a high frequency of either one or a few identical in frame V delta 1 sequences. Less abundant sequences were also detected repeatedly throughou
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Beagley, K. W., K. Fujihashi, A. S. Lagoo, S. Lagoo-Deenadaylan, C. A. Black, A. M. Murray, A. T. Sharmanov, M. Yamamoto, J. R. McGhee, and C. O. Elson. "Differences in intraepithelial lymphocyte T cell subsets isolated from murine small versus large intestine." Journal of Immunology 154, no. 11 (June 1, 1995): 5611–19. http://dx.doi.org/10.4049/jimmunol.154.11.5611.

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Abstract Intraepithelial lymphocytes (IELs) have been extensively studied in the murine small intestine. However, to date no studies have assessed IEL in the large intestine, despite the marked differences in function and lumenal environment. In the present study, we isolated IEL from both small and large intestine of three mouse strains (BALB/c, C3H/HeN, C57BL/6) and determined the frequency of CD2, CD4, and CD8 expression on CD3+ IEL, as well as the frequency of alpha beta and gamma delta TCR usage and V beta distribution. Higher numbers of IEL/unit length were always isolated from the small
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Allenspach, Karin. "Clinical Immunology and Immunopathology of the Canine and Feline Intestine." Veterinary Clinics of North America: Small Animal Practice 41, no. 2 (March 2011): 345–60. http://dx.doi.org/10.1016/j.cvsm.2011.01.004.

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Limon, Natalie M. "The Effects of Childhood, Adolescent and Adult Obesity on Epithelial T Cell Homeostasis in the Intestine." Journal of Immunology 198, no. 1_Supplement (May 1, 2017): 211.10. http://dx.doi.org/10.4049/jimmunol.198.supp.211.10.

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Abstract According to the CDC, 30% of adults in the U.S population are obese, while 17% of children and adolescents are obese. Obesity has resulted in a wide array of complications including disruption of barrier permeability as well as problems with tissue repair. The epithelial layer of the intestines contains intraepithelial intestinal lymphocytes (IEL) that are important in maintaining epithelial homeostasis and repairing tissue. To outline the mechanism by which obesity disrupts intestinal epithelial function among different age groups, childhood, adolescent, and adult mice were placed in
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Wang, Jian, Fengqi Li, Haiming Wei, Zhe-Xiong Lian, Rui Sun, and Zhigang Tian. "Respiratory influenza virus infection induces intestinal immune injury via microbiota-mediated Th17 cell–dependent inflammation." Journal of Experimental Medicine 211, no. 12 (November 3, 2014): 2397–410. http://dx.doi.org/10.1084/jem.20140625.

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Influenza in humans is often accompanied by gastroenteritis-like symptoms such as diarrhea, but the underlying mechanism is not yet understood. We explored the occurrence of gastroenteritis-like symptoms using a mouse model of respiratory influenza infection. We found that respiratory influenza infection caused intestinal injury when lung injury occurred, which was not due to direct intestinal viral infection. Influenza infection altered the intestinal microbiota composition, which was mediated by IFN-γ produced by lung-derived CCR9+CD4+ T cells recruited into the small intestine. Th17 cells m
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Hong, Chun Pyo, Bo Gie Yang, Jung-Hwan Kim, Min Seong Jang, Eun-Jung Lee, Eun Ji Jeun, Chan Kim, Ju-Young Seoh, and Myoung Ho Jang. "High fat diet-induced obesity affects CD4+ T cell differentiation in the small intestine (P3176)." Journal of Immunology 190, no. 1_Supplement (May 1, 2013): 61.13. http://dx.doi.org/10.4049/jimmunol.190.supp.61.13.

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Abstract Obesity-induced metabolic diseases are caused by the excess infiltration of pro-inflammatory cells to metabolic tissues including adipose and liver. However, systematic understanding of correlation between obesity and alterations of gut immunity is unclear. We hypothesized that immune cells in small intestine may be affected substantially upon high-fat feeding since dietary lipids are absorbed at luminal surface of small intestine. Here we found that small intestinal CD4+ and CD8+ T cells but not B cells were decreased in obese state. In CD4+ T cell subsets, the proportion of TH1 cell
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Vidal, Jorge E., Bruce A. McClane, Juliann Saputo, Jaquelyn Parker, and Francisco A. Uzal. "Effects of Clostridium perfringens Beta-Toxin on the Rabbit Small Intestine and Colon." Infection and Immunity 76, no. 10 (July 14, 2008): 4396–404. http://dx.doi.org/10.1128/iai.00547-08.

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ABSTRACT Clostridium perfringens type B and type C isolates, which produce beta-toxin (CPB), cause fatal diseases originating in the intestines of humans or livestock. Our previous studies demonstrated that CPB is necessary for type C isolate CN3685 to cause bloody necrotic enteritis in a rabbit ileal loop model and also showed that purified CPB, in the presence of trypsin inhibitor (TI), can reproduce type C pathology in rabbit ileal loops. We report here a more complete characterization of the effects of purified CPB in the rabbit small and large intestines. One microgram of purified CPB, in
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Teses / dissertações sobre o assunto "Intestine, Small Immunology"

1

Masjedi, Mohsen. "Physiological inflammation of the small intestine during weaning in the rat /." Title page, table of contents and summary only, 1998. http://web4.library.adelaide.edu.au/theses/09PH/09phm3973.pdf.

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Thompson, Fiona Marie. "Activation of the mucosal immune system and growth of the small intestine at weaning /." Title page, abstract and contents only, 1994. http://web4.library.adelaide.edu.au/theses/09PH/09pht4677.pdf.

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Randrian, Violaine. "Role of myosin IIA in the small intestine immunosurveillance by dendritic cells." Thesis, Sorbonne Paris Cité, 2017. http://www.theses.fr/2017USPCB038/document.

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Plusieurs méthodes de capture antigénique ont été décrites dans l’intestin grêle, surtout en cas d’infection: échantillonnage direct par les cellules dendritiques (DC), capture par les macrophages qui délivrent ensuite l’antigène aux DC du stroma, passage des antigènes à travers les cellules caliciformes. Des travaux antérieurs in vitro dans le laboratoire ont montré l’importance de la myosine IIA dans la coordination de la migration des DC avec la capture et de l’apprêtement antigénique. L’objectif de ma thèse était de combiner plusieurs méthodes d’imagerie telle que la microscopie intravital
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Moghaddami, Mahin. "Characterization of isolated lymphoid aggregations in the mucosa of the small intestine /." Title page, abstract and contents only, 1999. http://web4.library.adelaide.edu.au/theses/09PH/09phm6959.pdf.

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Thesis (Ph.D.)--University of Adelaide, Dept. of Microbiology and Immunology, 1999.<br>Errata & addenda tipped in behind back end paper. Copies of author's previously published articles in pocket on back end-paper. Bibliography: leaves 147-194.
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Guo, Weihong, and 郭衛紅. "The immune mechanisms and novel immunosuppressive approaches in experimental small bowel transplantation." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2001. http://hub.hku.hk/bib/B3124175X.

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Li, Xiaosong. "The mechanism study of novel approaches to control chronic allograft rejection in rat orthotopic small bowel transplantation." Click to view the E-thesis via HKUTO, 2006. http://sunzi.lib.hku.hk/hkuto/record/B36395778.

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Li, Xiaosong, and 李小松. "The mechanism study of novel approaches to control chronic allograft rejection in rat orthotopic small bowel transplantation." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2006. http://hub.hku.hk/bib/B36395778.

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Lemmey, Andrew Bruce. "Effects of insulin-like growth factors (IGFS) on recovery from gut resection in rats : a thesis submitted to the University of Adelaide, South Australia for the degree of Doctor of Philosophy." 1992, 1993. http://web4.library.adelaide.edu.au/theses/09PH/09phl554.pdf.

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Includes bibliographical references (leaves 159-213) Shows that IGF-I peptides are effective in diminishing post-surgical catabolism and enhancing adaptive gut hyperplasia in rats recovering from massive small bowel resection.
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9

Ou, Gangwei. "Human intestinal epithelial cells in innate immunity : interactions with normal microbiota and pathogenic bacteria." Doctoral thesis, Umeå : Umeå University, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-18388.

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Galvao, Flávio Henrique Ferreira. "Modelo experimental de doença do enxerto versus hospedeiro após transplante de intestino delgado." Universidade de São Paulo, 1998. http://www.teses.usp.br/teses/disponiveis/5/5132/tde-13072011-171433/.

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A doença do enxerto versus hospedeiro (DEVH) é uma grave complicação do transplante de órgãos sólidos, com alta mortalidade. Seu estudo tem sido limitado pela carência de modelos experimentais apropriados. Descreve-se um modelo de DEVH baseado no aumento do quimerismo, sua evolução clínica, histopatológica, do número das células quiméricas, do perfil das citocinas e da tolerância imunológica. Ratos Lewis (LEW) foram submetidos a transplante simultâneo de intestino delgado e medula óssea provenientes de ratos ACI (grupo de estudo - E) ou LEW (grupo controle - C), tratados com FK-506 (1 mg/Kg/di
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Mais fontes

Livros sobre o assunto "Intestine, Small Immunology"

1

N, Marsh Michael, ed. Immunopathology of the small intestine. Chichester [West Sussex]: Wiley, 1987.

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2

R, Grant David, and Wood Richard F. M, eds. Small bowel transplantation. London: E. Arnold, 1994.

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3

Small Bowel Transplantation (Hodder Arnold Publication). A Hodder Arnold Publication, 1996.

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4

E, Deltz, Thiede Arnulf, and Hamelmann H, eds. Small-bowel transplantation: Experimental and clinical fundamentals. Berlin: Springer-Verlag, 1986.

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5

Deltz, Eberhard, and Arnluf Thiede. Small-Bowel Transplantation: Experimental and Clinical Fundamentals. Springer-Verlag, 1987.

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6

Deltz, Eberhard. Small-Bowel Transplantation: Experimental and Clinical Fundamentals. Springer, 2011.

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7

Deltz, Eberhard, Arnulf Thiede, and Horst Hamelmann. Small-Bowel Transplantation: Experimental and Clinical Fundamentals. Springer London, Limited, 2012.

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8

Effects of immune cells and inflammation on smooth muscle and enteric nerves. Boca Raton, Fla: CRC Press, 1991.

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Capítulos de livros sobre o assunto "Intestine, Small Immunology"

1

Judge, Thomas. "The Small Bowel in Immunology." In Clinical Imaging of the Small Intestine, 29–37. New York, NY: Springer New York, 1999. http://dx.doi.org/10.1007/978-0-387-21565-5_3.

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2

Esses, Steven J., and Lloyd Mayer. "Mucosal Immunology of the Intestine." In Practical Gastroenterology and Hepatology: Small and Large Intestine and Pancreas, 23–27. Oxford, UK: Wiley-Blackwell, 2010. http://dx.doi.org/10.1002/9781444328417.ch4.

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Greney, Ph, E. Candolfi, and T. T. Kien. "Specific IgA response in small intestine during experimental toxoplasmosis." In Advances in Mucosal Immunology, 829–30. Dordrecht: Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-1848-1_261.

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Cummins, A. G., F. M. Thompson, L. Spargo, and G. Mayrhofer. "Maturation of the small intestine at weaning in the nude hypothymic rat." In Advances in Mucosal Immunology, 481–82. Dordrecht: Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-1848-1_142.

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Mowat, A. Mcl, A. J. Edwards, and I. N. Crispe. "T cell receptor expression by CD8+ intraepithelial lymphocytes from mouse small intestine." In Advances in Mucosal Immunology, 83–85. Dordrecht: Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-1848-1_21.

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Simister, N. E., and K. E. Mostov. "Functional expression of an Fc receptor cloned from neonatal rat small intestine." In Advances in Mucosal Immunology, 293–97. Dordrecht: Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-1848-1_83.

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Ferguson, Anne, and T. T. MacDonald. "Effects of Local Delayed Hypersensitivity on the Small Intestine." In Ciba Foundation Symposium 46 - Immunology of the Gut, 305–27. Chichester, UK: John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470720288.ch15.

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Trejdosiewicz, L. K., G. Malizia, S. Badr-el-Din, C. J. Smart, D. J. Oakes, J. Southgate, P. D. Howdle, G. Janossy, L. W. Poulter, and M. S. Losowsky. "T Cell and Mononuclear Phagocyte Populations of the Human Small and Large Intestine." In Recent Advances in Mucosal Immunology, 465–73. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-5344-7_54.

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Felstein, M. V., and A. McI Mowat. "Induction of Proliferative and Destructive Graft-Versus-Host Reactions in the Small Intestine." In Recent Advances in Mucosal Immunology, 653–60. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-5344-7_77.

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Nemirovsky, M. S., and N. Després. "Immunological and Immunopathological Characterization of a Mucosal Antigen from Guinea Pig Small Intestine." In Recent Advances in Mucosal Immunology, 783–89. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-5344-7_91.

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