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Bibliografias temáticas / Macrophages

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Literatura científica selecionada sobre o tema "Macrophages"

Autor: Grafiati

Publicado: 4 de junho de 2021

Última modificação: 25 de julho de 2025

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Artigos de revistas sobre o assunto "Macrophages"

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Stojadinović, Marija. "Macrophage polarization and infectious diseases." Biologia Serbica 45, no. 2 (2023): 38–43. https://doi.org/10.5281/zenodo.10402369.

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<strong>Summary. </strong>Macrophages are a heterogeneous cell population present in most mammalian tissues with a wide range of functions. They are an essential component of optimal tissue homeostasis and an essential first line of defense against pathogens. Activated macrophages are typically divided into two phenotypes, M1 macrophages and M2 macrophages, which are influenced by microorganisms, the tissue microenvironment, and cytokine signals from physiological conditions to infections. The management of macrophage polarity is crucial for the prevention and treatment of infections and infla

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Rodriguez, Eric, Frederic Boudard, Michele Mallié, Jean-Marie Bastide, and Madeleine Bastide. "Murine macrophage elastolytic activity induced by Aspergillus fumigatus strains in vitro: evidence of the expression of two macrophage-induced protease genes." Canadian Journal of Microbiology 43, no. 7 (1997): 649–57. http://dx.doi.org/10.1139/m97-092.

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The interaction between Aspergillus fumigatus conidia and murine macrophages of various origins was investigated. Cocultures were carried out between A. fumigatus strains and freshly isolated murine pulmonary alveolar macrophages or two murine macrophage cell-lines: murine alveolar cell-line MALU and murine astrocytoma cell-line J774. By measuring the variation of elastolytic activity in the coculture supernatants with two elastin substrates, we demonstrated that either viable or fixed A. fumigatus or C. albicans yeasts or nonspecific particles induced significant macrophage elastolytic activi

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Lu, Yufei, Leiming Guo, and Gaofeng Ding. "PD1+ tumor associated macrophages predict poor prognosis of locally advanced esophageal squamous cell carcinoma." Future Oncology 15, no. 35 (2019): 4019–30. http://dx.doi.org/10.2217/fon-2019-0519.

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Aim: Tumor associated macrophages are the most abundant cancer immune cells. However, little was known about the identity of CD68+PD1+ macrophages as well as the contributions in the prognosis of esophageal squamous cell carcinoma (ESCC). Methods & methods: Immunofluorescence, flowcytometry and RT-PCR were used to analysis PD1+ macrophages in ESCC. Results: CD68+PD1+ macrophages which can express higher M2 markers in cancer tissues, increased about 4.2-times compared with para-cancer tissues. Additionally, PD1high macrophages were significantly correlated with more malignant phenotypes and

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Hargarten, Jessica C., Tyler C. Moore, Thomas M. Petro, Kenneth W. Nickerson, and Audrey L. Atkin. "Candida albicans Quorum Sensing Molecules Stimulate Mouse Macrophage Migration." Infection and Immunity 83, no. 10 (2015): 3857–64. http://dx.doi.org/10.1128/iai.00886-15.

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The polymorphic commensal fungusCandida albicanscauses life-threatening disease via bloodstream and intra-abdominal infections in immunocompromised and transplant patients. Although host immune evasion is a common strategy used by successful human fungal pathogens,C. albicansprovokes recognition by host immune cells less capable of destroying it. To accomplish this,C. albicanswhite cells secrete a low-molecular-weight chemoattractive stimulant(s) of macrophages, a phagocyte that they are able to survive within and eventually escape from.C. albicansopaque cells do not secrete this chemoattracti

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Yadav, Mahesh, та Jeffrey S. Schorey. "The β-glucan receptor dectin-1 functions together with TLR2 to mediate macrophage activation by mycobacteria". Blood 108, № 9 (2006): 3168–75. http://dx.doi.org/10.1182/blood-2006-05-024406.

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AbstractPattern recognition receptors (PRRs) play an essential role in a macrophage's response to mycobacterial infections. However, how these receptors work in concert to promote this macrophage response remains unclear. In this study, we used bone marrow–derived macrophages isolated from mannose receptor (MR), complement receptor 3 (CR3), MyD88, Toll-like receptor 4 (TLR4), and TLR2 knockout mice to examine the significance of these receptors in mediating a macrophage's response to a mycobacterial infection. We determined that mitogen-activated protein kinase (MAPK) activation and tumor necr

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Gallego, Carolina, Douglas Golenbock, Maria Adelaida Gomez, and Nancy Gore Saravia. "Toll-Like Receptors Participate in Macrophage Activation and Intracellular Control of Leishmania (Viannia) panamensis." Infection and Immunity 79, no. 7 (2011): 2871–79. http://dx.doi.org/10.1128/iai.01388-10.

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ABSTRACTToll-like receptors (TLRs) play a central role in macrophage activation and control of parasitic infections. Their contribution to the outcome ofLeishmaniainfection is just beginning to be deciphered. We examined the interaction ofLeishmania panamensiswith TLRs in the activation of host macrophages.L. panamensisinfection resulted in upregulation of TLR1, TLR2, TLR3, and TLR4 expression and induced tumor necrosis factor alpha (TNF-α) secretion by human primary macrophages at comparable levels and kinetics to those of specific TLR ligands. The TLR dependence of the host cell response was

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McKenzie, C. G. J., U. Koser, L. E. Lewis, et al. "Contribution of Candida albicans Cell Wall Components to Recognition by and Escape from Murine Macrophages." Infection and Immunity 78, no. 4 (2010): 1650–58. http://dx.doi.org/10.1128/iai.00001-10.

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ABSTRACT The pathogenicity of the opportunistic human fungal pathogen Candida albicans depends on its ability to escape destruction by the host immune system. Using mutant strains that are defective in cell surface glycosylation, cell wall protein synthesis, and yeast-hypha morphogenesis, we have investigated three important aspects of C. albicans innate immune interactions: phagocytosis by primary macrophages and macrophage cell lines, hyphal formation within macrophage phagosomes, and the ability to escape from and kill macrophages. We show that cell wall glycosylation is critically importan

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Wilson, Justin E., Bhuvana Katkere, and James R. Drake. "Francisella tularensis Induces Ubiquitin-Dependent Major Histocompatibility Complex Class II Degradation in Activated Macrophages." Infection and Immunity 77, no. 11 (2009): 4953–65. http://dx.doi.org/10.1128/iai.00844-09.

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ABSTRACT The intracellular bacterium Francisella tularensis survives and replicates within macrophages, ultimately killing the host cell. Resolution of infection requires the development of adaptive immunity through presentation of F. tularensis antigens to CD4+ and CD8+ T cells. We have previously established that F. tularensis induces macrophage prostaglandin E2 (PGE2) production, leading to skewed T-cell responses. PGE2 can also downregulate macrophage major histocompatibility complex (MHC) class II expression, suggesting that F. tularensis-elicited PGE2 may further alter T-cell responses v

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Careau, Éric, Léa-Isabelle Proulx, Philippe Pouliot, Annie Spahr, Véronique Turmel, and Élyse Y. Bissonnette. "Antigen sensitization modulates alveolar macrophage functions in an asthma model." American Journal of Physiology-Lung Cellular and Molecular Physiology 290, no. 5 (2006): L871—L879. http://dx.doi.org/10.1152/ajplung.00219.2005.

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We have previously demonstrated that adoptive transfer of alveolar macrophages from allergy-resistant rats to alveolar macrophage-depleted allergic rats prevents airway hyperresponsiveness development, suggesting an important role for alveolar macrophages in asthma pathogenesis. Given that ovalbumin sensitization can modulate alveolar macrophage cytokine production, we investigated the role of sensitized and unsensitized alveolar macrophages in an asthma model. Alveolar macrophages from unsensitized or sensitized Brown Norway rats were transferred to alveolar macrophage-depleted sensitized rat

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Shinonaga, Masamichi, Cha Cheng Chang, Noriyuki Suzuki, Masazumi Sato, and Takeo Kuwabara. "Immunohistological evaluation of macrophage infiltrates in brain tumors." Journal of Neurosurgery 68, no. 2 (1988): 259–65. http://dx.doi.org/10.3171/jns.1988.68.2.0259.

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✓ Peritumoral edema is one of the most serious complications of intracranial neoplasms; however, the exact pathogenesis of this condition is still unknown. To explore the effect of macrophages in brain tumors on the pathogenesis of peritumoral edema, 42 specimens of primary or metastatic brain tumors were studied. Frozen sections were examined by an immunoperoxidase staining technique with anti-Leu-M3 monoclonal antibody. Eight of 14 gliomas demonstrated Leu-M3-positive cell (macrophage) infiltration. The two glioblastomas showed a moderate or marked degree of macrophage infiltration. Twelve o

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Teses / dissertações sobre o assunto "Macrophages"

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Svensson, Ulf. "Macrophage activation by bacteria signalling to prostaglandin and cytokine responses /." Lund : Dept. of Medical & Physiological Chemistry, Lund University, 1994. http://books.google.com/books?id=sAhrAAAAMAAJ.

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Higuera, González Laura 1993. "Novel transcription regulators of tissue macrophages and alternative macrophage polarization." Doctoral thesis, TDX (Tesis Doctorals en Xarxa), 2021. http://hdl.handle.net/10803/672702.

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Macrophages play crucial roles in the defense of the organism against a wide range of pathogens. Macrophages can rapidly adapt to perturbations in the microenvironment due to the existence of a network of transcription factors that modulates their responses. While transcription factors that regulate macrophage identity have been widely described in the past decades, the role of transcription regulators that fine-tune tissue macrophage responses in homeostasis and infection is starting to be elucidated. Our group has previously identified transcription regulators of pro-inflammatory macrophag

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Tabata, Yasuhiko. "Macrophage phagocytosis of polymer microspheres and antitumor activation of macrophages." Kyoto University, 1987. http://hdl.handle.net/2433/74704.

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Raborn, Erinn Shenee. "Cannabinoid Modulation of Chemotaxis of Macrophages and Macrophage-like Cells." VCU Scholars Compass, 2007. http://hdl.handle.net/10156/1333.

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Grand-Perret, Thierry A. R. "Induction d'une activité anti-tumorale chez les macrophages péritonéaux murins." Paris 11, 1986. http://www.theses.fr/1986PA112301.

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Bouchareychas, Laura. "Implication des phagocytes mononuclées dans l'évolution de la plaque d'athérosclérose et relation avec l'homéostasie du cholestérol et des lipoprotéines." Thesis, Paris 6, 2014. http://www.theses.fr/2014PA066282/document.

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L'athérosclérose est un processus physiopathologique chronique impliqué dans la majorité des maladies cardio-vasculaires. Le développement des lésions d'athérosclérose est caractérisé par l'accumulation de lipides extra et intracellulaires dans la paroi artérielle à l'origine d'une forte réponse inflammatoire impliquant notamment les macrophages. Les macrophages sont considérés comme des acteurs clés dans le développement des plaques d'athérosclérose. En effet, de par leur capacité à métaboliser le cholestérol (captation, stockage, efflux), à réguler l'inflammation et à phagocyter les cellules

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Di, Maggio Paula. "Dietary lipids and inflammation : chylomicron remnants suppress pro-inflammatory pathways and activate antioxidant defence mechanisms in human macrophages." Thesis, Royal Veterinary College (University of London), 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.618287.

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Georges, George Tharwat. "Novel Characteristics of Murine Bone Marrow-Derived Macrophages and Human Macrophage-Like Cells." VCU Scholars Compass, 2006. http://scholarscompass.vcu.edu/etd/932.

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These studies provide evidence for novel properties of macrophages derived from bone marrow stem cells. In study 1, treatment of activated mouse bone marrow-derived macrophages (BMM) with either catecholamine synthesis inhibitors (α-methyl-para-tyrosine and fusaric acid) or the β2 adrenergic receptor antagonist ICI 118,551 demonstrated that BMM produce catecholamines. The catecholamines modulated macrophage cytokine production through autocrine actions on adrenergic receptors. In study II, undifferentiated human bone marrow cells were incubated in 30% mouse L929 fibroblast conditioned medium

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Awomoyi, Agnes Abiola Oluwatoyin. "Genetics of susceptibility to tuberculosis." Thesis, Open University, 2000. http://oro.open.ac.uk/58012/.

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Convincing evidence that activated macrophages play a critical role in control of mycobacterial diseases has been clearly established from animal and in-vitro studies. Macrophages produce a variety of molecules upon appropriate stimulation, which act in concert towards eventual killing of bacteria. People with SUb-optimal macrophage activation are more susceptible to infection with intracellular pathogens. My project aims to answer two questions relating to genetic regulation of macrophage activation in tuberculosis: do macrophage genes regulate microbial-induced responses and do macrophage ge

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Suñer, Navarro Clara. "CPEB4 function in macrophages." Doctoral thesis, Universitat de Barcelona, 2018. http://hdl.handle.net/10803/663483.

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As innate immune cells, macrophages sense endogenous and exogenous danger signals and respond orchestrating inflammatory processes. For the rapid induction and efficient resolution of inflammatory responses, macrophages induce the expression of pro- inflammatory and anti-inflammatory mediators, which cross-regulate each other through feedback loops. This process requires tightly controlled gene expression at multiple levels. Recently, the regulation of mRNA deadenylation has emerged as a key regulator of the strength and, critically, the duration of transient inflammatory responses. Cytoplasm

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Mais fontes

Livros sobre o assunto "Macrophages"

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Kloc, Malgorzata, ed. Macrophages. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-54090-0.

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Rousselet, Germain, ed. Macrophages. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7837-3.

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Lawrence, Toby, and Thorsten Hagemann, eds. Tumour-Associated Macrophages. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-0662-4.

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H, Heppner Gloria, and Fulton Amy M. 1950-, eds. Macrophages and cancer. CRC Press, 1988.

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Thorsten, Hagemann, and SpringerLink (Online service), eds. Tumour-Associated Macrophages. Springer Science+Business Media, LLC, 2012.

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6

David, Evered, Nugent Jonathan, O'Connor Maeve, Ciba Foundation, and Symposium on Biochemistry of Microphages (1985 : Ciba Foundation), eds. Biochemistry of macrophages. John Wiley, 1986.

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Mass, Elvira, ed. Tissue-Resident Macrophages. Springer US, 2024. http://dx.doi.org/10.1007/978-1-0716-3437-0.

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David, Evered, Nugent Jonathan, O'Connor Maeve, and Symposium on Biochemistry of Macrophages (1985 : Ciba Foundation), eds. Biochemistry of macrophages. Pitman, 1986.

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9

Reiner, Neil E., ed. Macrophages and Dendritic Cells. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-396-7.

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Horton, Michael A., ed. Macrophages and Related Cells. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4757-9534-9.

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Capítulos de livros sobre o assunto "Macrophages"

1

Kelly, Aoife, Aleksander M. Grabiec, and Mark A. Travis. "Culture of Human Monocyte-Derived Macrophages." In Macrophages. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7837-3_1.

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Ian Cumming, R., and Yen-Rei A. Yu. "Phenotyping Tumor-Associated Macrophages." In Macrophages. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7837-3_10.

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Dalby, Elizabeth. "Activating Murine Macrophages In Vitro." In Macrophages. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7837-3_11.

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Huang, Xuan, Yong Li, Mingui Fu, and Hong-Bo Xin. "Polarizing Macrophages In Vitro." In Macrophages. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7837-3_12.

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Roback, Linda, and Lisa P. Daley-Bauer. "Viral Replication Assay in Bone Marrow-Derived Macrophages." In Macrophages. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7837-3_13.

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Aribi, Mourad. "Macrophage Bactericidal Assays." In Macrophages. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7837-3_14.

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Montaño, Fernando, Sergio Grinstein, and Roni Levin. "Quantitative Phagocytosis Assays in Primary and Cultured Macrophages." In Macrophages. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7837-3_15.

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Mularski, Anna, Florence Marie-Anaïs, Julie Mazzolini, and Florence Niedergang. "Observing Frustrated Phagocytosis and Phagosome Formation and Closure Using Total Internal Reflection Fluorescence Microscopy (TIRFM)." In Macrophages. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7837-3_16.

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Rousselet, Germain. "Chromatin Immunoprecipitation in Macrophages." In Macrophages. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7837-3_17.

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Keller, Andrea-Anneliese, Marten B. Maeß, Michael Schnoor, Berith Scheiding, and Stefan Lorkowski. "Transfecting Macrophages." In Macrophages. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7837-3_18.

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Trabalhos de conferências sobre o assunto "Macrophages"

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Mahgoub, Yasmine, Rida Arif, and Susu Zughaier. "Pyocyanin pigment from Pseudomonas aeruginosa modulates innate immune defenses in macrophages." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2021. http://dx.doi.org/10.29117/quarfe.2021.0137.

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Background: Pseudomonas aeruginosa is a well-known opportunistic pathogen. The gram-negative bacillus, commonly associated with hospital-acquired infections, utilizes the host’s impaired immune responses to establish infection. Of its many virulence factors, pyocyanin is essential for P. aeruginosa to establish its full infectivity. Macrophages act as sentinels of the innate immune system, as well as play other roles in homeostasis, tissue remodeling, and bridging between the innate and adaptive immune systems. Aim: This study aimed to investigate the effects of pyocyanin on macrophage innate

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van Dam-Mieras, M. C. E., A. D. Muller, and G. Hornstra. "DIETARY LIPIDS, INFECTION AND MACROPHAGE PROCOAGULANT ACTIVITY." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643398.

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It is generally accepted that the type of dietary fat influences arterial thrombosis and atherosclerosis. Although it is still largely unknown how the dietary lipid composition influences the process of atherogenesis, it is evident that several cell types are involved. Morphological evidence for the involvement of monocyte/macropages has been given.We described before that the dietary lipid composition has striking effects on the procoagulant activity of macrophages. When macrophages were isolated from the spleens of healthy rats the procoagulant activity slightly decreased during the first fe

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McGee, Maria, and Henry Rothberger. "MECHANISMS OF PROCOAGULANT GENERATION BY ALVEOLAR MACROPHAGES DURING MATURATION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643168.

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During maturation in vivo and in vitro alveolar macrophages generate procoagulant(s) capable of activating the extrinsic pathway. It is generally agreed that at least part of the activity is due to TF (tissue factor). However, whether or not macrophages also generate functional factor VII or X is controversial. To characterize procoagulant activity increases, we measured kinetic parameters defining interactions between components of the TF-VII complex on membranes of alveolar macrophages either freshly isolated or cultured in serum free medium. In incubation mixtures with fixed concentrations

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Reinhard, Björn M., Hongyun Wang, and Linxi Wu. "Monitoring Cellular Trafficking of Nanoparticle Cargo in Murine Macrophages Through Plasmon Coupling Microscopy." In ASME 2013 2nd Global Congress on NanoEngineering for Medicine and Biology. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/nemb2013-93078.

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A detailed analysis of silver nanoparticle (NP) uptake and trafficking in the murine macrophage cell line J774A.1 through spectral analysis of the resonance wavelength of the metal NP cargo is presented. The NP spectra reveal a strong phenotypic variability in the NP uptake and processing on the single cell level. Cells containing non- or low-agglomerated NPs are found to coexist with cells containing NPs of varying degrees of NP agglomeration, clearly indicated by a spectral red-shift in the resonance wavelength. Pharmacological inhibition studies indicate that the observed differences in the

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Rocha, Aline Carvalho, and Victor Piana Andrade. "The role of tumor-associated macrophages in the prediction of sentinel lymph node involvement in breast cancer." In Brazilian Breast Cancer Symposium 2024. Mastology, 2024. http://dx.doi.org/10.29289/259453942024v34s1031.

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Objective: The purpose of this study was to evaluate the association between the TAM density of breast tumor stroma and sentinel lymph node involvement. Methodology: The cohort consisted of patients with histopathological diagnosis of early-stage invasive breast cancer submitted to mastectomy or quadrantectomy and sentinel lymph node biopsy between January 2007 and December 2012 at a Brazilian referral hospital (A.C.Camargo Cancer Center). Using tissue microarrays, 101 tumors were submitted to immunohistochemistry for total macrophages (CD68), M2 macrophages (CD163), M1 macrophages (HLA-DR), a

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Hobro, Alison J., Takeshi Sugiyama, Nicolas Pavillon, Takayuki Umakoshi, Prabhat Verma, and Nicholas Smith. "Label-free Raman imaging of saturated and unsaturated fatty acid uptake, storage, and return toward baseline levels in macrophages." In JSAP-Optica Joint Symposia. Optica Publishing Group, 2023. http://dx.doi.org/10.1364/jsapo.2023.19a_a602_1.

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Lipids play many important roles in the body including cell signaling and energy storage. The presence of excessive lipids, or disruption of normal lipid metabolic processes in the cell, has been linked to lifestyle diseases such as atherosclerosis and obesity. Where the affected cells are part of the innate immune system such dysregulation of lipids has also been implicated in impaired immune responses to infection. Therefore, understanding how macrophages are affected by the presence of fatty acids in their local environment is an important step in understanding lifestyle disease development

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Adany, R., A. Kiss, J. Kappelmayer, R. J. Ablin, and L. Muszbek. "EXPRESSION OF FACTOR XIII SUBUNIT A IN DIFFERENT TYPES OF HUMAN MACROPHAGES." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644651.

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In addition to plasma the presence of subunit a of blood coagulation Factor XIII (FXIIl) has been verified in platelets and megakariocytes. Most recently, we demonstrated that human peripheral blood monocytes also contain FXIII subunit a. The present study was designed 1/ to determine the stage in the maturation sequence of bone marrow monocytopoesis in which FXIII appears 2/ to establish if FXIII is retained during differentiation into macrophages 3/ to assess how general is the presence of FXIII subunit a in different types of macrophages. FXIII subunit a was immunomorphologically detected i

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Muszbek, L., and R. Adány. "CELLULAR DISTIBUTION OF FACTOR XIII IN HUMAN UTERUS AND PLACENTA." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644648.

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As spontaneous abortion is a frequent finding in females with Factor XIII (FXIIl) deficiency it has been presumed that the plasmatic or cellular form of this clotting factor is essential to normal fetus development. In this context it is of special interestthat FXIII subunit a has been demonstrated in the homoge-nate of human uterus and placenta by activity measurements and immunobiochemical methods. However, no information on its cellular distribution has beenpublished, so far. In the present study first FXIII subunit a was detected in paraformaldehyde-fixed, paraffin-embedded sections by imm

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Nikishina, M. A., S. P. Fedotov, E. V. Fedotov, and V. Fedotov. "STATISTICAL ANALYSIS OF NANOPARTICLE UPTAKE BY CELLS." In XI МЕЖДУНАРОДНАЯ КОНФЕРЕНЦИЯ МОЛОДЫХ УЧЕНЫХ: БИОИНФОРМАТИКОВ, БИОТЕХНОЛОГОВ, БИОФИЗИКОВ, ВИРУСОЛОГОВ, МОЛЕКУЛЯРНЫХ БИОЛОГОВ И СПЕЦИАЛИСТОВ ФУНДАМЕНТАЛЬНОЙ МЕДИЦИНЫ. IPC NSU, 2024. https://doi.org/10.25205/978-5-4437-1691-6-31.

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Experimental data on nanoparticle uptake by rat alveolar macrophages were analyzed. The distribution of pits on the surface of a single macrophage was statistically analyzed based on a limited sample of 100 and 200 subregions. It was shown that the negative binomial distribution provides a more accurate model for experimentally observed pits on the surface of a single cell.

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Belchamber, K., and E. Sapey. "S51 Hungry hungry macrophages: how multiple prey affects macrophage phagocytosis." In British Thoracic Society Winter Meeting, Wednesday 17 to Friday 19 February 2021, Programme and Abstracts. BMJ Publishing Group Ltd and British Thoracic Society, 2021. http://dx.doi.org/10.1136/thorax-2020-btsabstracts.56.

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Relatórios de organizações sobre o assunto "Macrophages"

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Havell, Edward A. Actions of Interferons on Macrophages. Defense Technical Information Center, 1985. http://dx.doi.org/10.21236/ada157006.

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2

Naftolin, Frederick. Macrophages, Estrogen and the Microenvironment in Breast Cancer. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada383077.

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3

Benson, J. M., K. J. Nikula, and R. A. Guilmette. Evidence for particle transport between alveolar macrophages in vivo. Office of Scientific and Technical Information (OSTI), 1995. http://dx.doi.org/10.2172/381362.

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4

Shpigel, Nahum, Raul Barletta, Ilan Rosenshine, and Marcelo Chaffer. Identification and characterization of Mycobacterium paratuberculosis virulence genes expressed in vivo by negative selection. United States Department of Agriculture, 2004. http://dx.doi.org/10.32747/2004.7696510.bard.

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Mycobacterium avium subsp. paratuberculosis (MAP) is the etiological agent of a severe inflammatory bowel disease (IBD) in ruminants, known as Johne’s disease or paratuberculosis. Johne’s disease is considered to be one of the most serious diseases affecting dairy cattle both in Israel and worldwide. Heavy economic losses are incurred by dairy farmers due to the severe effect of subclinical infection on milk production, fertility, lower disease resistance and early culling. Its influence in the United States alone is staggering, causing an estimated loss of $1.5 billion to the agriculture indu

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5

Yull, Fiona. NF-kappaB Activity in Macrophages Determines Metastatic Potential of Breast Tumor Cells. Defense Technical Information Center, 2010. http://dx.doi.org/10.21236/ada541379.

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6

Nelson, Corwin, Donald C. Beitz, Tim Reinhardt, and John Lippolis. Toll-Like Receptor Signaling in Bovine Macrophages Increases 1,25-Dihydroxyvitamin D3 Production. Iowa State University, 2008. http://dx.doi.org/10.31274/ans_air-180814-482.

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7

Yull, Fiona. NF-kappaB Activity in Macrophages Determines Metastatic Potential of Breast Tumor Cells. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada554014.

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8

Adiga, Umesh, Brian Bell, Larissa Ponomareva, et al. Automated Analysis and Classification of Infected Macrophages Using Bright-Field Amplitude Contrast Data. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada578711.

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9

Kim, Isaac. Neuroendocrine Differentiation in Prostate Cancer: Role of Bone Morphogenetic Protein-6 and Macrophages. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada555480.

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10

Splitter, Gary, and Menachem Banai. Microarray Analysis of Brucella melitensis Pathogenesis. United States Department of Agriculture, 2006. http://dx.doi.org/10.32747/2006.7709884.bard.

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Original Objectives 1. To determine the Brucella genes that lead to chronic macrophage infection. 2. To identify Brucella genes that contribute to infection. 3. To confirm the importance of Brucella genes in macrophages and placental cells by mutational analysis. Background Brucella spp. is a Gram-negative facultative intracellular bacterium that infects ruminants causing abortion or birth of severely debilitated animals. Brucellosis continues in Israel, caused by B. melitensis despite an intensive eradication campaign. Problems with the Rev1 vaccine emphasize the need for a greater understand

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