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Bibliografias temáticas / Osteoclasts

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Literatura científica selecionada sobre o tema "Osteoclasts"

Autor: Grafiati

Publicado: 4 de junho de 2021

Última modificação: 25 de julho de 2025

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Artigos de revistas sobre o assunto "Osteoclasts"

1

Combs, Charlotte E., Karen Fuller, Hashethra Kumar, et al. "Urocortin is a novel regulator of osteoclast differentiation and function through inhibition of a canonical transient receptor potential 1-like cation channel." Journal of Endocrinology 212, no. 2 (2011): 187–97. http://dx.doi.org/10.1530/joe-11-0254.

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This study investigated the role of urocortin (UCN), a member of the corticotrophin-releasing factor (CRF) family of peptides, in osteoclast maturation and function. We found that 10−7 M UCN significantly (P<0.05) suppressed osteoclast differentiation from bone marrow precursor cells in culture and reduced the expression of several osteoclastic markers. Furthermore, UCN potently suppressed osteoclast bone resorption, by significantly inhibiting both the plan area of bone resorbed by osteoclasts and actin ring formation within osteoclasts at 10−9 M (P<0.05), with complete inhibition at 10

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2

Alatalo, Sari L., Jussi M. Halleen, Teuvo A. Hentunen, Jukka Mönkkönen, and H. Kalervo Väänänen. "Rapid Screening Method for Osteoclast Differentiation in Vitro That Measures Tartrate-resistant Acid Phosphatase 5b Activity Secreted into the Culture Medium." Clinical Chemistry 46, no. 11 (2000): 1751–54. http://dx.doi.org/10.1093/clinchem/46.11.1751.

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Abstract Background: Osteoclasts secrete tartrate-resistant acid phosphatase (TRAP; EC 3.1.3.2) 5b into the circulation. We studied the release of TRAP 5b from osteoclasts using a mouse in vitro osteoclast differentiation assay. Methods: We developed and characterized a polyclonal antiserum in rabbits, using purified human osteoclastic TRAP 5b as antigen. The antiserum was specific for TRAP in Western analysis of mouse osteoclast culture medium and was used to develop an immunoassay. We cultured mouse bone marrow-derived osteoclast precursor cells for 3–7 days with or without clodronate in the

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3

Niida, Shumpei, Masato Kaku, Hitoshi Amano, et al. "Vascular Endothelial Growth Factor Can Substitute for Macrophage Colony-Stimulating Factor in the Support of Osteoclastic Bone Resorption." Journal of Experimental Medicine 190, no. 2 (1999): 293–98. http://dx.doi.org/10.1084/jem.190.2.293.

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We demonstrated previously that a single injection of recombinant human macrophage colony-stimulating factor (rhM-CSF) is sufficient for osteoclast recruitment and survival in osteopetrotic (op/op) mice with a deficiency in osteoclasts resulting from a mutation in M-CSF gene. In this study, we show that a single injection of recombinant human vascular endothelial growth factor (rhVEGF) can similarly induce osteoclast recruitment in op/op mice. Osteoclasts predominantly expressed VEGF receptor 1 (VEGFR-1), and activity of recombinant human placenta growth factor 1 on osteoclast recruitment was

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4

Fuller, K., and T. J. Chambers. "Localisation of mRNA for collagenase in osteocytic, bone surface and chondrocytic cells but not osteoclasts." Journal of Cell Science 108, no. 6 (1995): 2221–30. http://dx.doi.org/10.1242/jcs.108.6.2221.

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Osteoclasts resorb the extracellular matrix of bone by secreting protons and enzymes into a circumpherentially sealed compartment between the osteoclast and the bone surface. Although the lysosomal cysteine proteinases play a major role in matrix degradation by osteoclasts, collagenase (matrix metalloproteinase-1, EC 3.4.24.7) is also required for osteoclastic bone resorption, and may be directly involved in collagen degradation in the hemivacuole. We assessed the effects of inhibitors of cysteine proteinases and collagenase on bone resorption by osteoclasts isolated from rodent bone. We found

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Fuller, Karen, Brian Wong, Simon Fox, Yongwon Choi, and Tim J. Chambers. "TRANCE Is Necessary and Sufficient for Osteoblast-mediated Activation of Bone Resorption in Osteoclasts." Journal of Experimental Medicine 188, no. 5 (1998): 997–1001. http://dx.doi.org/10.1084/jem.188.5.997.

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TRANCE (tumor necrosis factor–related activation-induced cytokine) is a recently described member of the tumor necrosis factor superfamily that stimulates dendritic cell survival and has also been found to induce osteoclastic differentiation from hemopoietic precursors. However, its effects on mature osteoclasts have not been defined. It has long been recognized that stimulation of osteoclasts by agents such as parathyroid hormone (PTH) occurs through a hormonal interaction with osteoblastic cells, which are thereby induced to activate osteoclasts. To determine whether TRANCE accounts for this

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6

Fuller, Karen, Chiho Murphy, Barrie Kirstein, Simon W. Fox та Timothy J. Chambers. "TNFα Potently Activates Osteoclasts, through a Direct Action Independent of and Strongly Synergistic with RANKL". Endocrinology 143, № 3 (2002): 1108–18. http://dx.doi.org/10.1210/endo.143.3.8701.

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Abstract TNFα is pivotal to the pathogenesis of inflammatory and possibly postmenopausal osteolysis. Much recent work has clarified mechanisms by which TNFα promotes osteoclastogenesis, but the means by which it activates osteoclasts to resorb bone remain uncertain. We found that very low concentrations of TNFα promoted actin ring formation, which correlates with functional activation in osteoclasts, both in osteoclasts formed in vitro and extracted from newborn rats. TNFα was equipotent with RANKL for this action. Activation by TNFα was unaffected by blockade of RANKL by OPG, its soluble deco

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7

Yu, Anna Xiao-Dan, Jian Xiao, Shi-Zheng Zhao, et al. "Biological Evaluation and Transcriptomic Analysis of Corylin as an Inhibitor of Osteoclast Differentiation." International Journal of Molecular Sciences 22, no. 7 (2021): 3540. http://dx.doi.org/10.3390/ijms22073540.

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Corylin, a flavonoid isolated from the fruit of Psoralea corylifolia, has an osteogenic effect on osteoblasts in vitro and bone micromass ex vivo. However, the effect and mechanism of corylin in regulating osteoclastogenesis remain unknown. By using murine bone marrow macrophages as the osteoclast precursor, corylin was found to inhibit the receptor activator of nuclear factor (NF) κB ligand (RANKL)-induced osteoclast differentiation via down-regulating osteoclastic marker genes. In parallel, F-actin formation and osteoclast migration were diminished in corylin-treated cultured osteoclasts, an

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Nakamura, I., M. F. Pilkington, P. T. Lakkakorpi, et al. "Role of alpha(v)beta(3) integrin in osteoclast migration and formation of the sealing zone." Journal of Cell Science 112, no. 22 (1999): 3985–93. http://dx.doi.org/10.1242/jcs.112.22.3985.

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The alpha(v)beta(3) integrin is abundantly expressed in osteoclasts and has been implicated in the regulation of osteoclast function, especially in cell attachment. However, in vivo studies have shown that echistatin, an RGD-containing disintegrin which binds to alpha(v)beta(3), inhibits bone resorption without changing the number of osteoclasts on the bone surface, suggesting inhibition of osteoclast activity. The objective of this study was to examine how occupancy of alpha(v)beta(3) integrins inhibits osteoclast function, using primary rat osteoclasts and murine pre-fusion osteoclast-like c

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9

Kameda, Takashi, Hiroshi Mano, Tatsuhisa Yuasa, et al. "Estrogen Inhibits Bone Resorption by Directly Inducing Apoptosis of the Bone-resorbing Osteoclasts." Journal of Experimental Medicine 186, no. 4 (1997): 489–95. http://dx.doi.org/10.1084/jem.186.4.489.

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Estrogen deficiency causes bone loss, which can be prevented by estrogen replacement therapy. Using a recently developed technique for isolation of highly purified mammalian osteoclasts, we showed that 17 β-estradiol (E2) was able to directly inhibit osteoclastic bone resorption. At concentrations effective for inhibiting bone resorption, E2 also directly induced osteoclast apoptosis in a dose- and time-dependent manner. ICI164,384 and tamoxifen, as pure and partial antagonists, respectively, completely or partially blocked the effect of E2 on both inhibition of osteoclastic bone resorption an

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10

Fuller, K., J. M. Owens, and T. J. Chambers. "Macrophage inflammatory protein-1 alpha and IL-8 stimulate the motility but suppress the resorption of isolated rat osteoclasts." Journal of Immunology 154, no. 11 (1995): 6065–72. http://dx.doi.org/10.4049/jimmunol.154.11.6065.

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Abstract Cells of the osteoblastic lineage play a major role in the regulation of osteoclastic bone resorption. Recent studies have demonstrated production of chemokines by osteoblastic cells. Although these phagocyte-stimulating and proinflammatory cytokines act as chemoattractants and activators for other members of the hemopoietic lineage, their actions on osteoclasts have not been characterized. We found that macrophage inflammatory protein-1 alpha (MIP-1 alpha) and IL-8 inhibited bone resorption by rat osteoclasts, primarily through reduction in the proportion of osteoclasts resorbing bon

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Teses / dissertações sobre o assunto "Osteoclasts"

1

O'Brien, Elizabeth Ann. "Regulation of osteoclast activity : differential adhesion of osteoclasts to the bone surface." Thesis, University of Liverpool, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.343930.

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Rowlands, Marit-Naomi. "In vitro production of osteoclasts." Thesis, University of Liverpool, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.250270.

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Nesbitt, Stephen Anthony. "Collagen binding proteins in osteoclasts." Thesis, Open University, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.265344.

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4

Herrera, Bruno Schneider. "Óxido nítrico e periodontite experimental: caracterização de mediadores intracelulares da atividade osteoclastogênica, conseqüências locais e sistêmicas." Universidade de São Paulo, 2008. http://www.teses.usp.br/teses/disponiveis/42/42136/tde-11092008-154717/.

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A doença periodontal é a doença crônica mais prevalente nas doenças orais. Dentre os mediadores desse processo contam-se a Resolvina E1 (RvE1), um mediador pró-resolução da inflamação capaz de diminuir a perda óssea secundária á doença periodontal em coelhos, e o oxido nítrico (NO), o qual pode ser produzido em grandes quantidades pela ação de citocinas e estimular a diferenciação e atividade osteoclástica. O objetivo deste estudo foi investigar os efeitos da RvE1 em osteoclastos (OCs) em cultura e os mecanismos envolvidos, bem como o papel do NO na progressão da periodontite experimental em r

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5

Franco, Gilson Cesar Nobre. "Analise da farmacocinetica e dos indices PK/PD da doxiciclina no plasma, fluido gengival e saliva e avaliação de seu efeito sobre a osteoclastogenese mediada por RANKL." [s.n.], 2007. http://repositorio.unicamp.br/jspui/handle/REPOSIP/288516.

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Orientadores: Pedro Luiz Rosalen, Francisco Carlos Groppo, Toshihisa Kawai<br>Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba<br>Made available in DSpace on 2018-08-08T05:44:44Z (GMT). No. of bitstreams: 1 Franco_GilsonCesarNobre_D.pdf: 2005090 bytes, checksum: e5ea5ec57b82c8e2fa17e200ad139928 (MD5) Previous issue date: 2007<br>Resumo: Doxiciclina (Dox) é um antimicrobiano pertencente à família das tetraciclinas com um amplo espectro de ação contra bactérias Gram-positivas e Gram-negativas. Além de suas propriedades antimicrobianas, Dox é atualmen

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Cayana, Ezymar Gomes. "Efeito da administração intermitente do PTH (1-34) na periodontite experimental em ratas expostas à fumaça de cigarros." [s.n.], 2010. http://repositorio.unicamp.br/jspui/handle/REPOSIP/290845.

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Orientadores: Enilson Antônio Sallum, João Baptista César Neto<br>Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba<br>Made available in DSpace on 2018-08-16T09:18:22Z (GMT). No. of bitstreams: 1 Cayana_EzymarGomes_D.pdf: 2999544 bytes, checksum: 87318d3714c0dada68775562aa703c38 (MD5) Previous issue date: 2010<br>Resumo: O objetivo deste estudo foi investigar histológica e histoquimicamente a influência da inalação da fumaça de cigarros (IFC) e da administração intermitente de PTH 1-34 sobre a perda óssea alveolar na região de furca em ratas submeti

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7

Taylor, Adam. "The role of Rab GTPases in osteoclasts." Thesis, Available from the University of Aberdeen Library and Historic Collections Digital Resources, 2009. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=59017.

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Hussein, Osama. "Interaction of breast cancer cells with osteoclasts." Thesis, McGill University, 2011. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=103695.

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Breast cancer is a major health problem. Metastatic disease is generally incurable. In the majority of patients, the skeleton bears the major metastatic burden. Bony lesions of metastatic breast cancer are usually osteolytic. Osteolytic metastases are formed by the pathological activation of osteoclasts. Anti-osteoclastic drugs are standard of care for patients suffering from breast cancer metastases. We conducted this project to decipher the signalling mechanisms responsible for osteoclasts activation in response to exposure to mediators released from mammary carcinoma cells. We evaluated the

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9

Gray, A. "Isolation, generation and characterization of equine osteoclasts." Thesis, University of Cambridge, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.599624.

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a) Equine osteoclasts (OCs) were identified in <I>ex vivo</I> samples of developing long bone. They were particularly numerous in areas of active bone formation and remodelling such as at the longitudinal septum between cartilage and subchondral bone and in the spongiosum. Equine OCs were identified as being typically multinuclear and expressing very high levels of tartrate resistant acid phosphatase (TRAP) and cathepsin K. Only low levels of cathepsin B were detected in equine OCs. b) Techniques were developed to isolate equine OCs for the first time <I>in vitro</I>. The phenotype of these ce

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Ford, Lorna. "An investigation into the effects of endocannabinoids and the COX-2 metabolite of 2-Arachidonyl glycerol on bone cells." Thesis, Available from the University of Aberdeen Library and Historic Collections Digital Resources, 2009. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=33596.

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Livros sobre o assunto "Osteoclasts"

1

R, Rifkin Barry, and Gay Carol V, eds. Biology and physiology of the osteoclast. CRC Press, 1992.

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2

Yu, Hesheng. P2 purinoceptor-linked Ca2+ signaling and pH changes in osteoclasts. University of Toronto, Faculty of Dentistry], 1996.

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3

Rodionova, N. V. Funkt͡s︡ionalʹnai͡a︡ morfologii͡a︡ kletok v osteogeneze. Nauk. dumka, 1989.

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4

National Institute on Aging/National Institute of Dental Research Workshop on Human Models of Skeletal Aging (1994 Washington, D.C.). National Institute on Aging/National Institute of Dental Research Workshop on Human Models of Skeletal Aging: Washington, DC, March 1-2, 1994. Edited by Robey Pamela Gehron 1952-, Sherman Sherry, National Institute of Dental Research (U.S.), and National Institute on Aging. Springer International, 1995.

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5

Karhukorpi, Eeva-Kaisa. The mode of action of osteoclasts in bone resorption: An ultrastructural study of enzymes possibly involved. University of Oulu], Department of Anatomy, 1991.

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6

Maria, Bijvoet Olav Leonardus, Lipton Allan, and International Cancer Congress (15th : 1990 : Hamburg, Germany), eds. Osteoclast inhibition in the management of malignancy-related bone disorders: An international symposium held during the 15th International Cancer Congress, Hamburg, Germany, August 1990. Hogrefe & Huber, 1993.

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7

D, Rubens R., and European Conference on Clinical Oncology (5th : 1989 : London, England), eds. The Management of bone metastases and hypercalcaemia by osteoclast inhibition: An international symposium held during the 5th European Conference on Clinical Oncology (ECCO 5), London, September 1989. Hogrefe & Huber, 1990.

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8

Shorey, Seema. Differences in the degree to which osteoclasts from different parts of the skeleton employ cathepsin K and matrix metalloproteinases for bone resorption. National Library of Canada, 2002.

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9

Holt, Ian. Control of osteoclast activity. University of Manchester, 1996.

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10

H, Helfrich Miep, and Ralston Stuart, eds. Bone research protocols. Humana Press, 2003.

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Capítulos de livros sobre o assunto "Osteoclasts"

1

Baak, Marleen A., Bernard Gutin, Kim A. Krawczewski Carhuatanta, et al. "Osteoclasts." In Encyclopedia of Exercise Medicine in Health and Disease. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_2794.

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2

Crockett, Julie C., David J. Mellis, and Adam Taylor. "Transfection of Osteoclasts and Osteoclast Precursors." In Methods in Molecular Biology. Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-415-5_14.

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3

Roodman, G. David, Linda M. McManus, and Anne Demulder. "Pagetic Osteoclasts." In Medical Intelligence Unit. Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-662-22505-9_3.

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4

Győri, Dávid, and Attila Mócsai. "Osteoclasts in Inflammation." In Compendium of Inflammatory Diseases. Springer Basel, 2016. http://dx.doi.org/10.1007/978-3-7643-8550-7_155.

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Győri, Dávid, and Attila Mócsai. "Osteoclasts in Inflammation." In Encyclopedia of Inflammatory Diseases. Springer Basel, 2013. http://dx.doi.org/10.1007/978-3-0348-0620-6_155-1.

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Manolagas, Stavros C. "Osteoblasts and Osteoclasts." In Principles of Molecular Rheumatology. Humana Press, 2000. https://doi.org/10.1007/978-1-59259-018-6_18.

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Atkins, Samantha K., Farwah Iqbal, Johana Barrientos, Cecilia Giachelli, and Elena Aikawa. "Osteoclasts in Cardiovascular Calcification." In Contemporary Cardiology. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-46725-8_18.

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8

Rucci, Nadia, and Anna Teti. "Osteoclasts: Essentials and Methods." In Principles of Bone and Joint Research. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-58955-8_3.

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9

Takahashi, Naoyuki, Nobuyuki Udagawa, Yasuhiro Kobayashi, and Tatsuo Suda. "Generation of Osteoclasts In Vitro, and Assay of Osteoclast Activity." In Arthritis Research. Humana Press, 2007. http://dx.doi.org/10.1007/978-1-59745-401-8_18.

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Wynn, Robert, and Ansgar Schulz. "Inborn Errors of Metabolism and Osteopetrosis." In The EBMT Handbook. Springer International Publishing, 2024. http://dx.doi.org/10.1007/978-3-031-44080-9_91.

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AbstractInborn errors of metabolism (IEM) comprise a large group of inherited disease, some of which are disorders of lysosomal, peroxisomal, or mitochondrial function, and only some can be improved following HCT. The mechanism of action varies between the different metabolic disorders. In the lysosomal disorders, healthy donor cells deliver the enzyme (secretion) to residual enzyme-deficient host cells. This is a changing area of medicine, in which autologous stem cell gene therapy is changing BMT practice, and this is likely to accelerate in the immediate future.Osteopetrosis is a disorder o

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Trabalhos de conferências sobre o assunto "Osteoclasts"

1

Ravi, Vilupanur, Wilyson Wantah, Steven Castenada, et al. "Corrosion and Immune Response of Boron-Modified Titanium Alloys in Biological Environments." In CORROSION 2009. NACE International, 2009. https://doi.org/10.5006/c2009-09466.

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Abstract Titanium alloys containing boron additions have shown remarkable improvements in processing and mechanical behavior. Significant reductions in grain size are observed even with trace additions of boron (&lt; 0.1 wt% B) leading to benefits in processing. When the boron levels are increased to modest levels (~ 1 wt% B), the stiffness and strength of the Ti-6 Al-4 V base alloy increase by 20 – 30% with no loss in ductility. Keeping these advantages in mind, the aqueous corrosion behavior of these unique materials was investigated in this study to establish a baseline for performance

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2

McCann, Kristen, Travis Voorhees, Gamer Margoosian, Janice Miguel, and Vilupanur Ravi. "Electrochemical Evaluation of Titanium-Boron Alloys for Potential Biomedical Applications." In CORROSION 2016. NACE International, 2016. https://doi.org/10.5006/c2016-07842.

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Abstract Structural biomedical implant alloys are predominantly titanium-based with UNS R56400 (Ti-6Al-4V; Ti64) being the most commonly used. These alloys demonstrate excellent corrosion resistance and biocompatibility; however, there is an ongoing need to have longer lasting implants given the rapid increase in the longevity of the world’s population. A critical issue concerning the durability of the implants is aseptic loosening, a phenomenon initiated by the release of metallic cations from the alloy that alters the equilibrium between osteoclasts (bone-consuming cells) and osteoblasts (bo

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3

Bondarenko, Natalya, Natalya Slazhneva, Mariya Vorobyeva, Nadezhda Kropacheva, Maxim Korolev, and Vitaly Omelchenko. "An Influence of Anti-Sclerostin Oligonucleotide Aptamers on Viability and Proliferation of Osteoblasts and Osteoclasts in vitro." In 2024 IEEE International Multi-Conference on Engineering, Computer and Information Sciences (SIBIRCON). IEEE, 2024. http://dx.doi.org/10.1109/sibircon63777.2024.10758523.

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4

Reddy Manne, Sai Kumar, Brendan Martin, Tyler Roy, et al. "NOISe: Nuclei-Aware Osteoclast Instance Segmentation for Mouse-to-Human Domain Transfer." In 2024 IEEE/CVF Conference on Computer Vision and Pattern Recognition Workshops (CVPRW). IEEE, 2024. http://dx.doi.org/10.1109/cvprw63382.2024.00686.

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Ravi, Vilupanur A., Roviden Enriquez, Carlos Beecher, et al. "Stability of Advanced Titanium Alloys in Saline Solution and Characterization of Osteoblast Activation." In CORROSION 2012. NACE International, 2012. https://doi.org/10.5006/c2012-01709.

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Abstract Electrochemical characterization of titanium alloys with different levels of boron was carried out in phosphate buffered saline (PBS) at body temperature and in 0.9 wt% sodium chloride (saline) at room temperature. Two types of cyclic potentiodynamic polarization tests were conducted - one based on ASTM F 2129-08 and the other with a broader voltage scan with higher peak potentials that further distinguished the behavior of the different titanium alloys. Break-down (pitting) potentials for the alloys in the latter test were in the 4.9 – 6.5 V range. The susceptibility of osteoblast ce

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Suzuki, Keiko, Baoqian Zhu, Harvey A. Goldberg, et al. "INTRACELLULAR OSTEOPONTIN IN OSTEOCLASTS: IMPAIRED MIGRATION, CELL FUSION AND RESORPTION IN OSTEOCLASTS FROM OPN-/- AND CD44-/- MICE." In 3rd International Conference on Osteopontin and SIBLING (Small Integrin-Binding Ligand, N-linked Glycoprotein) Proteins, 2002. TheScientificWorld Ltd, 2002. http://dx.doi.org/10.1100/tsw.2002.263.

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LI, Xiaojuan, Xiaorui XIE, Jin REN, Liling REN, and Jian CAO. "Effects of Propranolol on Osteoclasts Cultured in Vitro." In International Conference on Biological Engineering and Pharmacy 2016 (BEP 2016). Atlantis Press, 2017. http://dx.doi.org/10.2991/bep-16.2017.6.

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Brunner, Julia S., Melanie Hofmann, Victoria Saferding, et al. "02.28 The role of arginase I in osteoclasts." In 37th European Workshop for Rheumatology Research 2–4 March 2017 Athens, Greece. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2016-211050.28.

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9

Nemoto, Atsuko, Toshimasa Uemura, Takashi Ushida, and Tetsuya Tateishi. "GRAVITY EFFECTS ON mRNA EXPRESSION OF MARKER ENZYMES IN OSTEOCLASTS." In Proceedings of the 12th International Symposium on Ceramics in Medicine. WORLD SCIENTIFIC, 1999. http://dx.doi.org/10.1142/9789814291064_0065.

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Muraoka, Sei, Kaichi Kaneko, Natsuko Kusunoki, Shinichi Kawai, and Toshihiro Nanki. "THU0046 FRACTALKINE PROMOTES DIFFERENTIATION INTO OSTEOCLASTS FROM HUMAN PERIPHERAL BLOOD MONOCYTES." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.3046.

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Relatórios de organizações sobre o assunto "Osteoclasts"

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Ampornaramveth, Ruchanee. IL-1β mediate cementoblasts and osteoclast precursors interaction. Chulalongkorn University, 2016. https://doi.org/10.58837/chula.res.2016.17.

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An injury of the periodontium followed by an inflammatory response often leads to root resorption. Resorption is accomplished by osteoclasts and their generation may depend on an interaction with the cells in direct contact with the root, the cementoblasts. Our study aimed to investigate the role of human cementoblasts in the formation of osteoclasts and the effect of IL-1β hereupon. Extracted teeth from healthy volunteers were subjected to sequential digestion by type I collagenase and trypsin. The effect of enzymatic digestion on the presence of cells on the root surface was analyzed by hist

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2

Giachelli, Cecilia, Bruce Sangeorzan, Susan Lund, Steven Bain, Cameron Rementer, and Dewayne Threet. Engineered Osteoclasts for the Treatment and Prevention of Heterotopic Ossification. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada612445.

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3

Alikhani, Mani, Sarah Alansari, Mohammed Al Jearah, et al. Osteoclasts: The Biological Knife In Sutural Responses To Mechanical Stimulation. CTOR Press, 2018. http://dx.doi.org/10.30771/2018.2.

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4

Alikhani, Mani, Sarah Alansari, Mohammed Al Jearah, et al. Osteoclasts: The biological knife in sutural responses to mechanical stimulation. CTOR Press, 2018. http://dx.doi.org/10.30771/2018.3.

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ภวสันต์, ประสิทธิ์, та ทัศนีย์ ยงชัยตระกูล. อิทธิพลของ TNF-Alfla ต่อการสร้าง MMP-13, RANKL และ M-CSF ในเซลล์เพาะเลี้ยงเอ็นยึดปริทันต์ : รายงานผลการวิจัย. จุฬาลงกรณ์มหาวิทยลัย, 2006. https://doi.org/10.58837/chula.res.2006.9.

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วัตถุประสงค์: เซลล์เอ็นยึดปริทันต์สามารถตอบสนองต่อเชื้อแบคทีเรียและไซโตไคน์ ในภาวะที่มีการอักเสบ โดยหลั่งเอนไซม์ เมทริกซ์เมแทโลโปรทีเนส-13 (MMP-13) เพิ่มขึ้น และสร้างโปรตีนนิวเคลียแฟคเตอร์แคปปาบีไลแกนด์ (RANKL) ซึ่งช่วยกระตุ้นการเกิดเซลล์สลายกระดูก (osteoclasts) ร่วมด้วย แต่การเกิดเซลล์สลายกระดูกจำเป็นต้องมี แมคโครฟาจ-โคโลนี สติมูเลติง แฟคเตอร์ (M-CSF) ร่วมด้วย เราจึงศึกษาว่า เซลล์เอ็นยึดปริทันต์สามารถสร้าง M-CSF และ MMP-13 ในการตอบสนองต่อไซโตไคน์ที่เกี่ยวกับการอักเสบในรอยโรคปริทันต์ คือ ทูเมอร์เนโครสิส แฟคเตอร์ แอลฟา (TNF[alpha]) หรือไม่ วิธีการวิจัย: กระตุ้นเซลล์เพาะเลี้ยงเอ็นยึดปริทันต์ด้วย

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6

Reddy, Sakamuri. Osteoclast Inhibitory Peptide-1 Therapy for Paget's Disease. Defense Technical Information Center, 2010. http://dx.doi.org/10.21236/ada539193.

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7

Reddy, Sakamuri V. Osteoclast Inhibitory Peptide-1 Therapy for Paget's Disease. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada567774.

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8

Reddy, Sakamuri. Osteoclast Inhibitory Peptide-1 Therapy for Paget's Disease. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada553287.

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9

Reddy, Sakamuri V. Measles Virus Nucleocapsid (MJVNP) Gene Expression and RANK Receptor Signaling in Osteoclast Precursors. Osteoclast Inhibitors Peptide Therapy for Pagets Disease. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada484715.

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Reddy, Sakamuri V. Measles Virus Nucleocapsid (MVNP) Gene Expression and RANK Receptor Signaling in Osteoclast Precursors, Osteoclast Inhibitors Peptide Therapy for Pagets Disease. Defense Technical Information Center, 2008. http://dx.doi.org/10.21236/ada500887.

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