Bibliografias temáticas / Thiamine deficiency

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Literatura científica selecionada sobre o tema "Thiamine deficiency"

Autor: Grafiati

Publicado: 4 de junho de 2021

Última modificação: 25 de julho de 2025

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Artigos de revistas sobre o assunto "Thiamine deficiency"

Ritorto, Giovanna, Sara Ussia, Rocco Mollace, et al. "The Pivotal Role of Thiamine Supplementation in Counteracting Cardiometabolic Dysfunctions Associated with Thiamine Deficiency." International Journal of Molecular Sciences 26, no. 7 (2025): 3090. https://doi.org/10.3390/ijms26073090.

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The isolation, structural elucidation, and synthesis of pure thiamin were achieved in 1936, marking a milestone in vitamin research. As an organic compound soluble in water, thiamin is essential for carbohydrate metabolism in plants and animals, and in its active form—as part of the thiamin pyrophosphate coenzyme—performs these functions. Thereby, thiamin represents an essential vitamin to human health and is involved in several pathways that regulate several pathophysiological mechanisms. Cardiovascular disease is significantly impacted by thiamine imbalance and its supplementation offers sub

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Olufunso, Adeoye Bayo, Nwachukwu Ijeoma Doris, Olanrewaju Okikiola, and Omobude Aisagbonhi Elizabeth. "Enzyme Thiaminase: A Known Anti-nutritional Enzyme with Unknown Therapeutic Potentials in Cancer Treatment." Cross Current International Journal of Medical and Biosciences 6, no. 06 (2024): 114–19. https://doi.org/10.36344/ccijmb.2024.v06i06.001.

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Thiaminase (EC 2.5.1.2) is an enzyme that cleaves thiamine into its pyrimidine and thiazole moieties resulting in thiamine deficiency in various organisms. It is classified into two main types: Thiaminase I and Thiaminase II defined by the nucleophile used in the mechanism by which the cleavage is accomplished. Thiaminase I employs a variety of nucleophiles including, amines and sulfhydryl compounds while thiaminase II exclusively uses water for hydrolysis of thiamine. The crystal structure of thiaminase I reveals a deep cleft that accommodates thiamine and highlighting important residues that

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Dr., G. Sri Manjula, D.V. Ramanjaneyulu, E. Muralinath, et al. "Identification of Early as well as Advanced Symptoms of B1 Deficiency Along with Neurological, Cardiac and Gastrointestinal Manifestations." Journal of Cardiovascular, Cardiopulmonary Rehabilitation Nursing 2, no. 2 (2024): 18–26. https://doi.org/10.5281/zenodo.13756478.

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Thiamine, also termed as thiamin and vitamin B1, is one of the 8 B-complex vitamins and is categorized as a water-soluble B vitamin. Thiamine happens naturally in specific diets, is included in food products, and is available in dietary supplements. Rich sources of thiamine include whole grains, brown rice, pig, chicken, soybeans, almonds, peas, dry beans, and fortified or enriched grain products, such as cereals, bread, and baby formulae. Multivitamins supplement an additional 1.5 mg of thiamine to a diet already excess in this essential vitami

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Pan, Xiaohua, Xuemei Nan, Liang Yang, Linshu Jiang, and Benhai Xiong. "Thiamine status, metabolism and application in dairy cows: a review." British Journal of Nutrition 120, no. 5 (2018): 491–99. http://dx.doi.org/10.1017/s0007114518001666.

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AbstractAs the co-enzyme of pyruvate dehydrogenase andα-ketoglutarate dehydrogenase, thiamine plays a critical role in carbohydrate metabolism in dairy cows. Apart from feedstuff, microbial thiamine synthesis in the rumen is the main source for dairy cows. However, the amount of ruminal thiamine synthesis, which is influenced by dietary N levels and forage to concentrate ratio, varies greatly. Notably, when dairy cows are overfed high-grain diets, subacute ruminal acidosis (SARA) occurs and results in thiamine deficiency. Thiamine deficiency is characterised by decreased ruminal and blood thia

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Richter, Catherine A., Allison N. Evans, Maureen K. Wright-Osment, et al. "Paenibacillus thiaminolyticus is not the cause of thiamine deficiency impeding lake trout (Salvelinus namaycush) recruitment in the Great Lakes." Canadian Journal of Fisheries and Aquatic Sciences 69, no. 6 (2012): 1056–64. http://dx.doi.org/10.1139/f2012-043.

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Thiamine (vitamin B1) deficiency is a global concern affecting wildlife, livestock, and humans. In Great Lakes salmonines, thiamine deficiency causes embryo mortality and is an impediment to restoration of native lake trout ( Salvelinus namaycush ) stocks. Thiamine deficiency in fish may result from a diet of prey with high levels of thiaminase I. The discoveries that the bacterial species Paenibacillus thiaminolyticus produces thiaminase I, is found in viscera of thiaminase-containing prey fish, and causes mortality when fed to lake trout in the laboratory provided circumstantial evidence imp

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Lonsdale, Derrick. "A Review of the Biochemistry, Metabolism and Clinical Benefits of Thiamin(e) and Its Derivatives." Evidence-Based Complementary and Alternative Medicine 3, no. 1 (2006): 49–59. http://dx.doi.org/10.1093/ecam/nek009.

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Thiamin(e), also known as vitamin B1, is now known to play a fundamental role in energy metabolism. Its discovery followed from the original early research on the ‘anti-beriberi factor’ found in rice polishings. After its synthesis in 1936, it led to many years of research to find its action in treating beriberi, a lethal scourge known for thousands of years, particularly in cultures dependent on rice as a staple. This paper refers to the previously described symptomatology of beriberi, emphasizing that it differs from that in pure, experimentally induced thiamine deficiency in human subjects.

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GOMES, Marleide da Mota, and Marcos Raimundo Gomes de FREITAS. "Probable first report of a motor deafferentation syndrome in the Paraguayan War." Arquivos de Neuro-Psiquiatria 79, no. 6 (2021): 554–56. http://dx.doi.org/10.1590/0004-282x-anp-2020-0479.

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ABSTRACT The Paraguayan War ended 150 years ago. Back then, there were outbreaks of combatants’ limb weakness and tingling related to "palustrian cachexia", not clearly funded at the time on nutritional deficiency, the use of native flora to feed troops, and alcoholism. We report a case of a soldier with ascending paralysis, mental confusion and finally tetraplegia with preserved oculomotricity. This would probably be a case of locked-in syndrome (LIS) due to Gayet-Wernicke's encephalopathy consequent to thiamine deficiency. The role of thiamine in the peripheral or central nervous system expr

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Bourassa, Megan, and Gilles Bergeron. "Advances from the International Thiamine Alliance." Current Developments in Nutrition 4, Supplement_2 (2020): 814. http://dx.doi.org/10.1093/cdn/nzaa053_019.

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Abstract Objectives Thiamine deficiency disorders, including beriberi, remain a pressing public health problem. Without rapid recognition of symptoms and treatment, it is often fatal, especially in infants, and can have lasting neurological effects for survivors. Thus our objective is to create an alliance of country representatives, public health professionals, physicians and researchers who can quantify the prevalence of thiamine deficiency disorders globally and create control and prevention strategies. Methods The first meeting of this international thiamine alliance was held in November 2

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Skelton, William P., and Nadine K. Skelton. "Thiamine deficiency neuropathy." Postgraduate Medicine 85, no. 8 (1989): 301–6. http://dx.doi.org/10.1080/00325481.1989.11700760.

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van Snippenburg, Wouter, Mariet G. J. Reijnders, Jose G. M. Hofhuis, Rien de Vos, Stephan Kamphuis, and Peter E. Spronk. "Thiamine Levels During Intensive Insulin Therapy in Critically Ill Patients." Journal of Intensive Care Medicine 32, no. 9 (2016): 559–64. http://dx.doi.org/10.1177/0885066616659429.

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Introduction: Thiamine is an essential cofactor in carbohydrate metabolism, and deficiency can therefore cause various organ dysfunctions. Little is known about the prevalence and possible worsening of thiamine deficiency in critically ill patients. In this study, we investigated the prevalence of thiamine deficiency at admission to the intensive care unit (ICU) and hypothesized that intensive insulin therapy, aimed at regulating glucose levels, increases thiamine utilization and therefore might cause or worsen deficiency in patients with limited thiamine stores. Materials and Methods: An obse

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Teses / dissertações sobre o assunto "Thiamine deficiency"

Hazell, Alan Stewart. "The pathophysiology of pyrithiamine-induced thiamine deficiency encephalopathy." Thesis, McGill University, 1995. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=28776.

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Pyrithiamine-induced thiamine deficiency (PTD) in the rat results in a metabolic disorder associated with selective cerebral vulnerability. The basis of the histological lesions that develop in areas that include the thalamus, medial geniculate, and inferior colliculus is undetermined but an underlying glutamate-mediated form of excitotoxicity may be responsible. This work examines the possibility that PTD leads to changes in extracellular glutamate concentration, membrane polarization status, and gene expression consistent with a decreased ability to maintain homeostatic integrity. At the

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Larkin, James Robert. "The role of the kidney in diabetic thiamine deficiency." Thesis, University of Warwick, 2010. http://wrap.warwick.ac.uk/34560/.

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Diabetes is a chronic epidemic compounded by a burden of vascular complications including diabetic nephropathy. Diabetic nephropathy affects ~40% of patients and is characterised by increased urinary albumin excretion and decreased glomerular filtration rate. Diabetic patients exhibit ~75% decreased plasma thiamine concentration, linked to increased renal thiamine clearance. In streptozotocin-induced diabetic rats, decreased plasma thiamine concentration was also associated with a reduction in expression and activity of transketolase. Transketolase is a thiamine pyrophosphate-dependent enzyme

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Molnar, Lydia. "Thiamine in a wet pet food application." Thesis, Kansas State University, 2017. http://hdl.handle.net/2097/35454.

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Master of Science Department of Grain Science and Industry Greg Aldrich Since 2010, there have been seven recalls related to thiamine deficiency in cat food products (FDA, 2017; FSA, 2017). Cats have a high requirement of thiamine (5.6 mg/kg), and deficiencies can lead to death within a month if not treated (AAFCO, 2017). A few studies have been published regarding the impact of retort processing on thiamine loss in canned pet food but no work has been reported on heat penetration in other containers (pouches and trays). Therefore, our objectives were to determine the effect of contai

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Navarro, Darren. "Region-selective effects of thiamine deficiency on cerebral metabolism in pyrithiamine-treated rats." Thesis, McGill University, 2008. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=115904.

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Pyrithiamine-induced thiamine deficiency in rats is a well-established animal model of Wernicke's Encephalopathy (WE). This thesis project, submitted as four articles, presents an examination of metabolic events that contribute to the selective neuronal lesions observed in the medial thalamus (MT) of thiamine-deficient (TD) rat. In addition, the phenomenon of glucose-precipitated worsening of neurological status in WE patients (Wallis et al., 1978; Watson et al., 1981) is explored. Lactate accumulation is known to occur selectively in regions of the TD brain, which ultimately express neuron

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Yates, Jodye. "Diazepam administration during acute thiamine deficiency attenuates subsequent neuropathology and spatial-memory deficits in rats." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape15/PQDD_0004/MQ39449.pdf.

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Kozel, Carrie L. "Early Feeding In Lake Trout Fry (salvelinus Namaycush) As A Mechanism For Ameliorating Thiamine Deficiency Complex." ScholarWorks @ UVM, 2017. http://scholarworks.uvm.edu/graddis/685.

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Recruitment failure of lake trout (Salvelinus namaycush) in the Great Lakes has been attributed in part to the consumption of alewife (Alosa pseudoharengus) by adult lake trout, leading to Thiamine Deficiency Complex (TDC) and early mortality in fry. The current understanding of thiamine deficiency in lake trout fry is based on information from culture and hatchery settings, which do not represent conditions fry experience in the wild and may influence the occurrence of TDC. In the wild, lake trout fry have access to zooplankton immediately following hatching; previous studies found that wild

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Alexander-Kaufman, Kimberley Louise. "Proteomics of the human alcoholic brain: Implications for the pathophysiology of alcohol-related brain damage." The University of Sydney, 2008. http://hdl.handle.net/2123/2692.

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Doctor of Philosophy (PhD) Proteomics is rapidly achieving recognition as a complimentary and perhaps superior approach to examine global changes in protein abundance in complex biological systems and the value of these techniques in neuropsychiatry is beginning to be acknowledged. Characterizing the brain’s regional proteomes provides a foundation for the detection of proteins that may be involved in disease-related processes. Firstly, optimal conditions were achieved for the application of two dimensional-gel electrophoresis (2D-GE)-based proteomics with postmortem human brain tissue. The

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Alexander-Kaufman, Kimberley Louise. "Proteomics of the human alcoholic brain: Implications for the pathophysiology of alcohol-related brain damage." Thesis, The University of Sydney, 2007. http://hdl.handle.net/2123/2692.

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Proteomics is rapidly achieving recognition as a complimentary and perhaps superior approach to examine global changes in protein abundance in complex biological systems and the value of these techniques in neuropsychiatry is beginning to be acknowledged. Characterizing the brain’s regional proteomes provides a foundation for the detection of proteins that may be involved in disease-related processes. Firstly, optimal conditions were achieved for the application of two dimensional-gel electrophoresis (2D-GE)-based proteomics with postmortem human brain tissue. These optimized techniques were t

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Soares, Susana Patrícia Veloso. "Paretic syndrome in gulls (Laridae) in the south of Portugal." Master's thesis, Universidade de Lisboa. Faculdade de Medicina Veterinária, 2014. http://hdl.handle.net/10400.5/6554.

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Dissertação de Mestrado Integrado em Medicina Veterinária RIAS, a Portuguese wildlife rehabilitation centre located in Algarve, has been admitting a substantial high number of seagulls, since its opening in October of 2009, with consistent clinical presentations pertaining to a paretic syndrome without cues of a particular disease. This preliminary study describes the clinical signs and microbiological, parasitological, toxicological and pathologic findings of paretic gulls received between 2009 and 2012. It tries to understand if there is an association between the manifestation of th

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Vindedzis, Sally Ann. "The relationship between low blood thiamin levels in diabetes to thiamin intake and diabetic control." Thesis, Curtin University, 2008. http://hdl.handle.net/20.500.11937/248.

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Mild thiamin deficiency is prevalent in diabetes, and high dose thiamin ameliorates some diabetic complications, but there are no definitive studies addressing thiamin intake, diabetes control and thiamin status in diabetes. Subjects were 113 people with diabetes (58 type 1, 55 type 2), 43 with and 70 without thiamin supplementation. Dietary thiamin was estimated by 24-hour recall, diabetes control by HbA1c. Age, BMI, albumin excretion, activity level and smoking status did not correlate with red cell thiamin (RCT) in either group. RCT correlated with serum thiamin (ST) (p < 0.01). In those un

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Livros sobre o assunto "Thiamine deficiency"

McCandless, David W. Thiamine deficiency and associated clinical disorders. Humana Press, 2010.

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McCandless, David W. Thiamine Deficiency and Associated Clinical Disorders. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60761-311-4.

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Carpenter, Kenneth J. Beriberi, white rice, and vitamin B: A disease, a cause, and a cure. University of California Press, 2000.

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Carpenter, Kenneth J. Beriberi, white rice, and vitamin B: A disease, a cause, and a cure. University of California Press, 2000.

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McCandless, David W. Thiamine Deficiency and Associated Clinical Disorders. Humana Press, 2012.

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Marrs, Chandler, and Derrick Lonsdale. Thiamine Deficiency Disease, Dysautonomia, and High Calorie Malnutrition. Elsevier Science & Technology Books, 2017.

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Marrs, Chandler, and Derrick Lonsdale. Thiamine Deficiency Disease, Dysautonomia, and High Calorie Malnutrition. Elsevier Science & Technology Books, 2017.

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Gluckman, Sir Peter, Mark Hanson, Chong Yap Seng, and Anne Bardsley. Vitamin B1 (thiamine) in pregnancy and breastfeeding. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198722700.003.0007.

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Vitamin B1 (thiamine) is involved in nervous system and muscle function and is essential for carbohydrate metabolism. Deficiency is common in Asia, where diets are often high in thiamine-depleted polished rice and can be low in other food sources. Pregnancy imposes an increasing requirement for thiamine over the course of gestation, and deficiency can lead to widespread metabolic disturbances affecting the placenta and fetus. Nutritional deficiency for thiamine is rare in people who consume a moderately varied diet that contains whole grains. However, excessive vomiting in pregnancy can cause

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Janssen, Mirian C. H., and Shamima Rahman. Pyruvate Dehydrogenase Complex Deficiency. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0006.

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Pyruvate dehydrogenase complex (PDHc) deficiency usually first manifests at a young age and is rarely diagnosed in adulthood. The clinical picture varies from neonatal death with overwhelming lactic acidosis to a relatively benign course early in life. The three main presentations are congenital lactic acidosis, Leigh syndrome, and episodic ataxia. Treatment consists of a ketogenic diet and cofactor supplementation with thiamine. Successful therapy is rare.

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Bâ, Abdoulaye. Maternal Alcohol Intake, Thiamine (vitamine B1) Deficiency and Central Nervous System Development. BAYSHOP (Generis Publishing), 2023.

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Capítulos de livros sobre o assunto "Thiamine deficiency"

Thurston, Jean Holowach, Richard E. Hauhart, John A. Dirgo, and David B. McDougal. "Thiamine Deficiency." In Cerebral Energy Metabolism and Metabolic Encephalopathy. Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-1209-3_15.

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Raj, Satish R., S. R. Wayne Chen, Robert S. Sheldon, et al. "Thiamine Deficiency." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_1733.

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McCandless, David W. "Early Thiamine Deficiency." In Thiamine Deficiency and Associated Clinical Disorders. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60761-311-4_2.

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Dias, Fernando Machado Vilhena, Aline Sanches Oliveira, Danilo Jorge da Silva, and Angela Maria Ribeiro. "Thiamine Deficiency and Poverty." In Handbook of Famine, Starvation, and Nutrient Deprivation. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-40007-5_83-1.

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McCandless, David W. "Thiamine Deficiency in Mammals." In Thiamine Deficiency and Associated Clinical Disorders. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60761-311-4_3.

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Misra, U. K., and J. Kalita. "Thiamine deficiency neurological disorders." In Neurological Consequences of Nutritional Disorders. CRC Press, 2021. http://dx.doi.org/10.1201/9780429316401-3.

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McCandless, David W. "Thiamine Deficiency in Serious Illness." In Thiamine Deficiency and Associated Clinical Disorders. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60761-311-4_10.

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McCandless, David W. "Early Chemistry." In Thiamine Deficiency and Associated Clinical Disorders. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60761-311-4_1.

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McCandless, David W. "World Health Concerns." In Thiamine Deficiency and Associated Clinical Disorders. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60761-311-4_11.

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Trabalhos de conferências sobre o assunto "Thiamine deficiency"

Umrani, Rohan, Hiral Amin, Ashley Pinckney, Charles Yang, and Daniel Tran. "Mixed Beri’s: High Output Heart Failure from Severe Anemia and Thiamine Deficiency." In 27th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2023. https://doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.12_2023.

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Background: Severe anemia and thiamine deficiency can independently result in high output heart failure (HOHF) through different mechanisms. Data on the threshold and timeframe at which these etiologies may precipitate HOHF is unclear. Case: A 63-year-old male with alcohol use disorder consuming 18 drinks/week presented with progressive shortness of breath and lower extremity edema for a few months. Physical exam revealed tachycardia, anasarca, jugular venous distension, Lancisi’s sign, and a midsystolic murmur. Echocardiogram demonstrated EF of 30-35%, dilated left ventricle, and cardiac inde

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Moura, Fernanda Maria Gonçalves de Sousa, Amanda Freitas Alves, João Paulo Barile, Sephora Sabrina Cândido de Almeida, Arthur da Veiga Kalil Coelho, and Marcele Schettini de Almeida. "Hallucinations and psychosis in Guillain-Barré syndrome: case report." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.394.

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Introduction: Guillain-Barré syndrome (GBS) is an acute inflammatory peripheral neuropathy that occurs after infection or immunization. Wernicke’s encephalopathy (WE) is caused by thiamine deficiency that classically presents with altered mental status, ataxic gait and ophthalmoplegia. Case report: Male, 64 years old, presented with diarrhea, vomiting and hyporexia. Three weeks later, he developed concomitant acute tetraparesis and cognitive impairment. On examination, he presented with persecutory delusions, hallucinations, tetraparesis with global areflexia. The cerebrospinal fluid analysis

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Shah, Avni, Karen Camero, Alvaro Serrano, Yair Erell, and John Li. "Infant with Thiamine Deficiency from a Rare Cause." In AAP National Conference & Exhibition Meeting Abstracts. American Academy of Pediatrics, 2021. http://dx.doi.org/10.1542/peds.147.3_meetingabstract.632-a.

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Mainra, A., and S. Thakur. "The Enigmatic Duo: Thiamine Deficiency Unveiled in Lactic Acidosis." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a3421.

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Uchel, T., and U. F. Sofi. "Refractory Lactic Acidosis Due to Thiamine Deficiency from Metformin Toxicity." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a2939.

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Costa, Virgínia Madureira, Iris Maria de Miranda Correia, Laís Michela Rodrigues Sales Arruda, et al. "The effectiveness of using thiamine in the Wernicke-Korsakoff syndrome." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.380.

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Introduction: The Wernicke-Korsakoff syndrome is a condition caused by thiamine (vitamin B1) deficiency in the brain, being a debilitating and potentially fatal factor. It is characterized by a classic triad: delirium, ophthalmoparesis and ataxia. Objectives: Analyze the possible effectiveness of using thiamine in the prognostic change of patients with the syndrome, as well as the ideal dose and identification of possible secondary outcomes of the use of thiamine. Methods: A systematic review made in March 2021, included studies published between 2011-2021. The descriptors selected according t

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Sweet, Rebecca, Lauren Lim та Jason Zastre. "Abstract 5419: Thiamine deficiency induces HIF-1 α stabilization and target gene expression." У Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-5419.

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Siqueira, Fernando, Vanessa Siqueira, Lucas Falcão, Arthur Bezerra, and Carlos Silva. "THE INFLUENCE BETWEEN ALZHEIMER’S DISEASE AND HEALTHY EATING: A SYSTEMATIC REVIEW." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda062.

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Background: Alzheimer’s Disease (AD) is a neurodegenerative disease responsible for neuronal losses that affect mainly the cortex and hippocampus, which begin to shrink in size, damaging cognitive functions. This process affects cholinergic neurons, influencing acetylcholine (ACH) levels, a memoryrelated neurotransmitter. Glucose metabolism and low thiamine levels appear to be affected by AD. Consequently, diabetes becomes a disease associated with AD and the thiamine deficiency levels depress the use of glucose by the brain. Thus, nutrition may have a role in preventing dementia through the t

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Thakur, S., and S. Whealon. "A Rare Case of Lactic Acidosis Associated With Severe Thiamine Deficiency in the United States." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a3398.

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Elyahu, A. Y., S. Huelskamp, and H. Poor. "Thiamine Deficiency Causing High-Output Heart Failure with Concomitant Vasoreactive Pulmonary Arterial Hypertension: A Case Report." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a7290.

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