Bibliografias temáticas / TRIM18

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Literatura científica selecionada sobre o tema "TRIM18"

Autor: Grafiati
Publicado: 1 de abril de 2023
Última modificação: 31 de julho de 2025

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Artigos de revistas sobre o assunto "TRIM18"

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Kimsa, Malgorzata W., B. Strzalka-Mrozik, M. C. Kimsa, et al. "Differential Expression of Tripartite Motif-Containing Family in Normal Human Dermal Fibroblasts in Response to Porcine Endogenous Retrovirus Infection." Folia Biologica 60, no. 3 (2014): 144–51. http://dx.doi.org/10.14712/fb2014060030144.

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Antiretroviral restriction factors may play an essential role in the safety of xenotransplantation. Therefore, the present study focused on investigation of the changes in the tripartite motif-containing family (TRIM) gene expression in normal human dermal fibroblasts with and without lipopolysaccharide stimulation in response to porcine endogenous retrovirus infection. Analysis of the expression profile of TRIMs was performed using oligonucleotide microarrays and QRT-PCR. Nine (TRIM1, TRIM2, TRIM5, TRIM14, TRIM16, TRIM18, TRIM22, TRIM27 and TRIM31) statistically significantly differentially e
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Panhwar, S. N., C. Huang, Q. Luo, et al. "OVERVIEW OF CLASS-I TRIPARTITE MOTIF (TRIM) PROTEINS SPECIFICALLY TRIM67." Pakistan Journal of Agriculture, Agricultural Engineering and Veterinary Sciences 40, no. 2 (2024): 108–19. https://doi.org/10.47432/2024.40.2.8.

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The TRIM family is a group of genes expressed during embryogenesis. This review emphasized data on Class-I TRIM genes, including TRIM1, TRIM9, TRIM36, TRIM46, TRIM67, TRIM18, and TRIM76. This article will discuss the structure and function of these genes and their roles in various physiological processes such as embryonic development, and immune regulation. Class-I TRIM proteins develop as E3 ubiquitin ligases and work with E2 ubiquitin-conjugating enzymes. TRIM9 is expressed in the stem cells of mice and linked to various neuronal functions and neurological diseases. TRIM18 is found in microt
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Yap, Melvyn W., Mark P. Dodding, and Jonathan P. Stoye. "Trim-Cyclophilin A Fusion Proteins Can Restrict Human Immunodeficiency Virus Type 1 Infection at Two Distinct Phases in the Viral Life Cycle." Journal of Virology 80, no. 8 (2006): 4061–67. http://dx.doi.org/10.1128/jvi.80.8.4061-4067.2006.

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ABSTRACT The Trim5α protein from several primates restricts retroviruses in a capsid (CA)-dependent manner. In owl monkeys, the B30.2 domain of Trim5 has been replaced by cyclophilin A (CypA) following a retrotransposition. Restriction of human immunodeficiency virus type 1 (HIV-1) by the resulting Trim5-CypA fusion protein depends on CA binding to CypA, suggesting both that the B30.2 domain might be involved in CA binding and that the tripartite RING motif, B-BOX, and coiled coil (RBCC) motif domain can function independently of the B30.2 domain in restriction. To investigate the potential of
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Tran, Pham-Tue-Hung, Mir Himayet Kabir, Naveed Asghar, et al. "Identification of TRIM21 and TRIM14 as Antiviral Factors Against Langat and Zika Viruses." Viruses 17, no. 5 (2025): 644. https://doi.org/10.3390/v17050644.

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Flaviviruses are usually transmitted to humans via mosquito or tick bites, whose infections may lead to severe diseases and fatality. During intracellular infection, they remodel the endoplasmic reticulum (ER) membrane to generate compartments scaffolding the replication complex (RC) where replication of the viral genome takes place. In this study, we purified the ER membrane fraction of virus infected cells to identify the proteins that were enriched during flavivirus infection. We found that tripartite motif-containing proteins (TRIMs) including TRIM38, TRIM21, and TRIM14 were significantly
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De La Cruz-Herrera, Carlos F., Michael H. Tatham, Umama Z. Siddiqi, et al. "Changes in SUMO-modified proteins in Epstein-Barr virus infection identifies reciprocal regulation of TRIM24/28/33 complexes and the lytic switch BZLF1." PLOS Pathogens 19, no. 7 (2023): e1011477. http://dx.doi.org/10.1371/journal.ppat.1011477.

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SUMO modifications regulate the function of many proteins and are important in controlling herpesvirus infections. We performed a site-specific proteomic analysis of SUMO1- and SUMO2-modified proteins in Epstein-Barr virus (EBV) latent and lytic infection to identify proteins that change in SUMO modification status in response to EBV reactivation. Major changes were identified in all three components of the TRIM24/TRIM28/TRIM33 complex, with TRIM24 being rapidly degraded and TRIM33 being phosphorylated and SUMOylated in response to EBV lytic infection. Further experiments revealed TRIM24 and T
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Sebastian, Sarah, Christian Grütter, Caterina Strambio de Castillia та ін. "An Invariant Surface Patch on the TRIM5α PRYSPRY Domain Is Required for Retroviral Restriction but Dispensable for Capsid Binding". Journal of Virology 83, № 7 (2009): 3365–73. http://dx.doi.org/10.1128/jvi.00432-08.

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ABSTRACT TRIM5α is a retrovirus restriction factor in the host cell cytoplasm that blocks infection before provirus establishment. Restriction activity requires capsid (CA)-specific recognition by the PRYSPRY domain of TRIM5α. To better understand the restriction mechanism, nine charge-cluster-to-triple-alanine mutants in the TRIM5α PRYSPRY domain were assessed for CA-specific restriction activity. Five mutants distributed along the TRIM5α PRYSPRY primary sequence disrupted restriction activity against N-tropic murine leukemia virus and equine infectious anemia virus. Modeling of the TRIM5α PR
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Margalit, Liad, Carmit Strauss, Ayellet Tal, and Sharon Schlesinger. "Trim24 and Trim33 Play a Role in Epigenetic Silencing of Retroviruses in Embryonic Stem Cells." Viruses 12, no. 9 (2020): 1015. http://dx.doi.org/10.3390/v12091015.

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Embryonic stem cells (ESC) have the ability to epigenetically silence endogenous and exogenous retroviral sequences. Trim28 plays an important role in establishing this silencing, but less is known about the role other Trim proteins play. The Tif1 family is a sub-group of the Trim family, which possess histone binding ability in addition to the distinctive RING domain. Here, we have examined the interaction between three Tif1 family members, namely Trim24, Trim28 and Trim33, and their function in retroviral silencing. We identify a complex formed in ESC, comprised of these three proteins. We f
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Toka, Felix N., Kiera Dunaway, Matylda Mielcarska, Felicia Smaltz, and Magdalena Bossowska-Nowicka. "Expression pattern of TRIM genes in bovine macrophages stimulated with PAMPs." Journal of Immunology 198, no. 1_Supplement (2017): 129.7. http://dx.doi.org/10.4049/jimmunol.198.supp.129.7.

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Abstract In mammals innate immune mechanisms form the first line of defence against invading pathogens. Detection of viruses early in infection relies on intracellular receptors that sense microbial molecular patterns, subsequently leading to gene transcription that eventually produces IFN type I and II. Type I and II IFNs act to prevent replication of viruses. Tripartite Motif-containing (TRIM) proteins belong to a superfamily of RING-domain E3 ubiquitin ligases. They represent a novel class of antiviral molecules involved in innate immunity. These enzymes function in a wide variety of import
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Rybakowska, Paulina, Nina Wolska, Arkadiusz Klopocki, et al. "Multiple TRIM proteins are targets of autoimmune response in lupus and Sjogren's syndrome. (HUM7P.308)." Journal of Immunology 192, no. 1_Supplement (2014): 184.17. http://dx.doi.org/10.4049/jimmunol.192.supp.184.17.

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Abstract TRIM21 belongs to the large family of tripartite motif containing proteins, and is often targeted by autoantibodies in lupus and Sjogren’s syndrome. Considering the significant protein domain homology between different TRIM proteins, we hypothesized that additional TRIM proteins are targets of autoimmunity. Based on the literature, in this study we investigated autoantibody responses to TRIM38. While 9% of lupus patients (n=149) had anti-TRIM38 antibodies, the incidence in Sjogren’s syndrome patients (n=150) was 12%, and in controls (n=50) it was 4%. With respect to TRIM21, the incide
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Agarwal, Neeraj, Sebastien Rinaldetti, Bassem B. Cheikh, et al. "TRIM28 is a transcriptional activator of the mutant TERT promoter in human bladder cancer." Proceedings of the National Academy of Sciences 118, no. 38 (2021): e2102423118. http://dx.doi.org/10.1073/pnas.2102423118.

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Bladder cancer (BC) has a 70% telomerase reverse transcriptase (TERT or hTERT in humans) promoter mutation prevalence, commonly at −124 base pairs, and this is associated with increased hTERT expression and poor patient prognosis. We inserted a green fluorescent protein (GFP) tag in the mutant hTERT promoter allele to create BC cells expressing an hTERT-GFP fusion protein. These cells were used in a fluorescence-activated cell sorting–based pooled CRISPR-Cas9 Kinome knockout genetic screen to identify tripartite motif containing 28 (TRIM28) and TRIM24 as regulators of hTERT expression. TRIM28
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Teses / dissertações sobre o assunto "TRIM18"

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FERNANDES, LAGES INES. "Identification of Deubiquitinases (DUBs) associated with TRIM18/MID1." Doctoral thesis, Università degli Studi di Trieste, 2023. https://hdl.handle.net/11368/3042038.

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Ubiquitination is a post-translation modification process crucial to control protein degradation, localization, and activity. Tripartite Motif (TRIM) proteins participate in the ubiquitination process behaving as E3 ubiquitin ligases, responsible for the specific recognition of the substrate to be ubiquitinated. In the opposite process, Deubiquitinating enzymes (DUBs) can deconjugate the ubiquitin from the protein target. The antagonism of DUBs and E3s is essential to maintain protein homeostasis and signaling in cells. In this project, we focused on TRIM18 (also named MID1) that when mutated
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ZANCHETTA, MELANIA EVA. "BRAF35 as target of MID1/TRIM18 E3 ligase activity." Doctoral thesis, Università degli Studi di Trieste, 2016. http://hdl.handle.net/11368/2908069.

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TRIM proteins are a family of ubiquitin E3 ligase enzymes characterized by the presence of a conserved N-terminus, the tripartite motif, which consists of a RING finger domain, two B-box motifs and an alpha-helical Coiled-coil region (RBCC). We focused our attention on TRIM18/MID1, the gene responsible for the X-linked form of Opitz G/BBB syndrome, a congenital disease characterized by defects in midline development and mental retardation. More recently, MID1 has also been found as overexpressed in some aggressive prostate cancers. The role of MID1 within the cell and the target(s) of its E3 u
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MASCARO, MARTINA. "Ruolo di TRIM18, gene che causa la sindrome di Opitz, nel controllo della ciliogenesi attraverso l'autofagia." Doctoral thesis, Università degli Studi di Trieste, 2021. http://hdl.handle.net/11368/2996095.

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TRIM18/MID1 è una E3-Ubiquitina ligasi appartenente alla famiglia di proteine TRIM che si localizza sui microtubuli. Le mutazioni con perdita di funzione nel gene MID1 sono state associate alla forma legata all'X della sindrome di Opitz, una malattia genetica caratterizzata da difetti nello sviluppo della linea mediana ventrale. All'interno della cellula, MID1 esiste come un complesso macromolecolare microtubulare, costituito da diversi interattori non tutti ancora identificati. Tra questi, α4 è una delle subunità regolatorie della proteina fosfatasi 2A (PP2A). È stato riportato che MID1 riduc
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Basu, Shrivastava Meenakshi. "Régulation de la stabilité de NFATc3 par SUMO et les E3 ubiquitine-ligases Trim39 et Trim17." Thesis, Montpellier, 2020. http://www.theses.fr/2020MONTT043.

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Les facteurs de transcription NFAT (facteur nucléaire des cellules T activées) jouent un rôle physiologique important dans le développement et le fonctionnement de nombreux organes, notamment dans le système immunitaire et le système nerveux. Par conséquent, leur dérégulation a été impliquée dans diverses maladies humaines telles que le cancer, les maladies neurodégénératives et les maladies auto-immunes. La régulation de l'activité de NFAT par translocation nucléo-cytoplasmique a été largement étudiée. En revanche, la régulation du niveau protéique de NFAT par le système ubiquitine-protéasome
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Buberl, Cilli Dana [Verfasser]. "Expressionsanalyse der putativen E3 Ligasen Trim23 und Trim7 in der frühen Neuralentwicklung von Xenopus laevis / Cilli Dana Buberl." Halle, 2017. http://d-nb.info/1137867698/34.

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Borle, Pawar Ankush [Verfasser], Norbert [Akademischer Betreuer] Frey, and Dennis [Gutachter] Schade. "Functional characterization of TRIM24 and TRIM32 proteins in the heart through their interaction with Dysbindin / Ankush Borle Pawar ; Gutachter: Dennis Schade ; Betreuer: Norbert Frey." Kiel : Universitätsbibliothek Kiel, 2019. http://d-nb.info/1220691259/34.

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Locke, Matthew. "TRIM32 in Genetic Disease." Thesis, University of Oxford, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.514962.

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VENUTO, SANTINA. "Dissecting the TRIM8 role in the pathogenesis of glioma." Doctoral thesis, Università degli Studi di Foggia, 2019. http://hdl.handle.net/11369/382357.

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I gliomi umani sono un gruppo eterogeneo di tumori cerebrali maligni primari, la cui patogenesi molecolare risulta ancora parzialmente sconosciuta. Pertanto, la comprensione dei meccanismi molecolari alla base della loro insorgenza e del loro decorso può portare a una migliore scelta di terapie appropriate e a migliori risultati prognostici, attraverso l'identificazione di nuovi geni specifici associati al glioma. Le proteine appartenenti alla famiglia “tripartite motif” (TRIM) sono coinvolte in diversi processi biologici, tra cui la regolazione trascrizionale, il controllo della progressione
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Crichton, Jennifer E. "The Role of the E3-ubiquitin Ligase Trim17 in the Mitochondrial Cell Death Pathway." Thèse, Université d'Ottawa / University of Ottawa, 2013. http://hdl.handle.net/10393/23715.

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The upregulation of apoptosis is a hallmark of several neurodegenerative disorders including ischemic stroke. In neurons, as in other cell types, Bax and tBid are critical regulators of the intrinsic pathway upstream of mitochondrial outer membrane permeabilization (MOMP) and caspase activation. The characterization of the molecular events that occur during the early stages is therefore extremely important from a therapeutic standpoint. Here I show that two independent genetic pilot screens looking for novel regulators of Bax activation identified a common hit in the E3 ubiquitin ligase Trim17
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Simpson, Shmona. "Genetic, structural, and functional exploration of the restrictive capacity of TRIM proteins against immunodeficiency viruses." Thesis, University of Oxford, 2017. https://ora.ox.ac.uk/objects/uuid:1af588ba-603a-4f39-9443-bb1a95d983f5.

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HIV-2 differs from HIV-1 in that many infected people experience normal survival, whilst only 20% progress rapidly to AIDS. Understanding mechanisms of delayed HIV-2 disease progression could provide new insights into HIV control. The Caio Community Cohort was established in Guinea-Bissau in the setting of high HIV-2 prevalence. This thesis investigates the role of polymorphic host restriction factors of the TRIM family in HIV-2 outcome. TRIM proteins are a family of E3 ubiquitin-ligases, where closely-related TRIM5α and TRIM22 are thought to inhibit HIV-1 transcription, uncoating
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Livros sobre o assunto "TRIM18"

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Sodums, Dzintars. Jauni trimdā. "Karogs", 1997.

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Odom, Joe. TRIMIX diver manual. 3rd ed. Technical Diving International, 1998.

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Hennessy, Mark. Trim. Royal Irish Academy, 2003.

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David, Cameron-Smith, ed. Professor Trim's ultimate food energy guide. Allen & Unwin, 2003.

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Astros, J. G. d'. Lo trimfe de la lengua gascoa. Per Noste, 2007.

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Berry, Nancy E. Architectural Trim. Rockport Publishers, 2009.

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DeKorne, Clayton. Trim carpentry. Taunton, 2007.

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Kossow, Greg. Trim complete. Taunton Press, 2007.

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Coppola, Alan C. Fit & trim. Im-Press Pub. Co., 1986.

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Haxell, Kate. Knit edgings & trims: 150 stitches. Interweave, 2009.

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Capítulos de livros sobre o assunto "TRIM18"

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Gooch, Jan W. "Trimer." In Encyclopedic Dictionary of Polymers. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_12131.

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Moss, Joel, and Martha Vaughan. "ARD1/TRIM23." In Encyclopedia of Signaling Molecules. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_644.

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Moss, Joel, Michaela U. Gack, and Martha Vaughan. "ARD1/TRIM23." In Encyclopedia of Signaling Molecules. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4614-6438-9_644-1.

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Biswas-Fiss, Esther E., Stephanie Affet, Malissa Ha, et al. "ARD1/TRIM23." In Encyclopedia of Signaling Molecules. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_644.

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Hall, Christine M., Amaka C. Offiah, Francesca Forzano, Mario Lituania, Gen Nishimura, and Valérie Cormier-Daire. "Odontochondrodysplasia, TRIP11-Related." In Fetal and Perinatal Skeletal Dysplasias, 2nd ed. CRC Press, 2024. http://dx.doi.org/10.1201/9781003166948-44.

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Templeton, J. L. "Tungsten Trimer Synthesis." In Inorganic Reactions and Methods. John Wiley & Sons, Inc., 2007. http://dx.doi.org/10.1002/9780470145296.ch64.

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Grosso, Monica, Ilias Cheimariotis, Marcin Stepniak, Chiara Lodi, and Alessandro Marotta. "The Role of TRIMIS as a Policy Support Tool." In Lecture Notes in Mobility. Springer Nature Switzerland, 2025. https://doi.org/10.1007/978-3-031-85578-8_103.

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Abstract TRIMIS is a transport policy support tool that provides open-access information on transport research and innovation. It supports the vision of a clean, connected, and competitive European transport system and exemplifies the European Commission’s commitment to open science. TRIMIS collects, curates, analyses, and disseminates European and non-European data on transport research and innovation. It also analyses technology trends and research and innovation capacities in the transport sector. TRIMIS maintains an open database of almost 9,000 transport research and innovation projects a
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Hirota, E., K. Kuchitsu, T. Steimle, J. Vogt, and N. Vogt. "39 C3H6F6 Difluoromethane trimer." In Molecules Containing Three or Four Carbon Atoms and Molecules Containing Five or More Carbon Atoms. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-41504-3_40.

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Demaison, J. "143 H6O3 Water trimer." In Symmetric Top Molecules. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-47532-3_145.

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Seong, Hongje, Seoung Wug Oh, Brian Price, Euntai Kim, and Joon-Young Lee. "One-Trimap Video Matting." In Lecture Notes in Computer Science. Springer Nature Switzerland, 2022. http://dx.doi.org/10.1007/978-3-031-19818-2_25.

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Trabalhos de conferências sobre o assunto "TRIM18"

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Kim, Jungheon, and Dongkun Shin. "Delayed TRIM for Reducing Trim Overhead." In 2024 13th Non-Volatile Memory Systems and Applications Symposium (NVMSA). IEEE, 2024. http://dx.doi.org/10.1109/nvmsa63038.2024.10693664.

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Liu, Zichuan, Ke Wang, Mingyuan Wu, Lantao Yu, Klara Nahrstedt, and Xin Lu. "I-Matting: Improved Trimap-Free Image Matting." In 2024 IEEE International Conference on Multimedia and Expo (ICME). IEEE, 2024. http://dx.doi.org/10.1109/icme57554.2024.10687689.

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Alfaqeeh, Mosab. "TriMod Fusion for Multimodal Named Entity Recognition in Social Media." In 2024 34th International Conference on Collaborative Advances in Software and COmputiNg (CASCON). IEEE, 2024. https://doi.org/10.1109/cascon62161.2024.10837944.

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Zamani, Lachin, and Reza Azmi. "TriMAE: Fashion Visual Search with Triplet Masked Auto Encoder Vision Transformer." In 2024 14th International Conference on Computer and Knowledge Engineering (ICCKE). IEEE, 2024. https://doi.org/10.1109/iccke65377.2024.10874812.

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Metzler, M., IJ Diets, J. Hoyer, et al. "TRIM28 haploinsufficiency predisposes to Wilms tumor." In 32. Jahrestagung der Kind-Philipp-Stiftung für pädiatrisch onkologische Forschung. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1687131.

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Duan, Wenyang, Hongsen Zhang, Limin Huang, et al. "Numerical Simulation of Trim Optimization on Resistance Performance Based on CFD Method." In ASME 2019 38th International Conference on Ocean, Offshore and Arctic Engineering. American Society of Mechanical Engineers, 2019. http://dx.doi.org/10.1115/omae2019-96181.

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Abstract In response to the gradually stringent carbon emission requirements of the International Maritime Organization (IMO), the energy-saving methods of the shipping industry have received increasing attention. Today how to reduce fuel consumptions so as to lower carbon emissions to improve the economic and environmental benefits of ships has become a hot topic. As one of the most easily implemented energy-saving methods, trim optimization has caught more and more researchers’ eyes. In this paper, a commercial CFD software STAR-CCM+ was adopted to analyze the influence of trim on the resist
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Saetti, Umberto, and Jonathan Rogers. "A Motion Primitive Perspective on Rotorcraft Regime Recognition." In Vertical Flight Society 76th Annual Forum & Technology Display. The Vertical Flight Society, 2020. http://dx.doi.org/10.4050/f-0076-2020-16266.

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An alternative approach to regime recognition that is based on the notion of motion primitives is developed. The algorithm developed is non-causal and leverages the ideas of maneuvers and trims as defined in a motion primitive context. The algorithm functions in three major steps. Given a state and control input time history obtained from flight data, the first step consists of classifying the state and control time history into trim and maneuver segments. The second step leverages the information in the trim state and control vectors to classify each trim segment into a particular trim condit
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Fong, Ka-Wing, Jonathan Zhao, Bin Zheng, and Jindan Yu. "Abstract 1521: Role of Trim28 in prostate cancer." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-1521.

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Kirkby, David R., and David T. Delpy. "An optoelectronic cross-correlator using a gain modulated avalanche photodiode for measurement of the tissue temporal point spread function." In Advances in Optical Imaging and Photon Migration. Optica Publishing Group, 1996. http://dx.doi.org/10.1364/aoipm.1996.trit108.

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A small and relatively inexpensive optoelectronic cross-correlator, using an avalanche photodiode (APD) detector and without any moving parts has been developed to measure the temporal point spread function (TPSF) of light in tissue. From the TPSF, it is in principle possible to determine the optical properties of the tissue. Currently a temporal response of 275 ps full width half maximum (FWHM) has been achieved. Measurements of the TPSF of a tissue phantom using a synchroscan streak camera and the cross-correlator show excellent agreement, although the achievable signal to noise ratio curren
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Zhang, Changming, Mukherjee Subhas, Tucker-Burden Carol, Monica Chau, Jun Kong, and Daniel Brat. "Abstract 2516: TRIM8 modulates stem-like cells in glioblastoma." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-2516.

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Relatórios de organizações sobre o assunto "TRIM18"

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Strathman, James. Extraboard Management: TriMet Case Study. Portland State University Library, 2012. http://dx.doi.org/10.15760/trec.4.

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Kalberer, Jennifer L., and Jennifer C. Spanich. Evaluation of the TRIMAX 280 System. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada405546.

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Trumble, E. F. TRIMPWR: A post processor for TRIMHX. Office of Scientific and Technical Information (OSTI), 1989. http://dx.doi.org/10.2172/6975898.

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Trumble, E. F. Validation and verification summary report for GRIMHX and TRIMHX. Office of Scientific and Technical Information (OSTI), 1990. http://dx.doi.org/10.2172/10156331.

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Trumble, E. F. Validation and verification summary report for GRIMHX and TRIMHX. Office of Scientific and Technical Information (OSTI), 1990. http://dx.doi.org/10.2172/5038892.

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Fries, Joseph. Helicopter Trim Analysis. Defense Technical Information Center, 1996. http://dx.doi.org/10.21236/ada310293.

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Brooks, Stephen. Integrated Fields of Permanent Magnet Dipole Trims. Office of Scientific and Technical Information (OSTI), 2021. http://dx.doi.org/10.2172/1895098.

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Minden, M. L., and T. R. O'Meara. Range-Doppler, Target-Referencing Imaging Systems (RD-TRIMS). Defense Technical Information Center, 1990. http://dx.doi.org/10.21236/ada360123.

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Strathman, James, Joseph Broach, and Steve Callas. Evaluation of Short Duration Unscheduled Absences Among Transit Operators: TriMet Case Study. Portland State University Library, 2009. http://dx.doi.org/10.15760/trec.141.

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Mills, John R. An Introduction To PC-TRIM. U.S. Department of Agriculture, Forest Service, Pacific Northwest Research Station, 1989. http://dx.doi.org/10.2737/pnw-rn-491.

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