Gotowe bibliografie tematyczne / Allergic response

Spis treści

  1. Artykuły w czasopismach
  2. Rozprawy doktorskie
  3. Książki
  4. Części książek
  5. Streszczenia konferencji
  6. Raporty organizacyjne

Gotowa bibliografia na temat „Allergic response”

Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 5 lutego 2022

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Artykuły w czasopismach na temat "Allergic response"

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Martin, Lynn B., and Courtney A. C. Coon. "Photoperiod-driven variation in an allergic response is independent of allergen exposure." Canadian Journal of Zoology 90, no. 9 (September 2012): 1086–93. http://dx.doi.org/10.1139/z2012-075.

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Allergy prevalence and severity varies seasonally in humans, presumably due to intra-annual changes in allergen exposure. However, it is possible that seasonality of allergic responses is also influenced by seasonal changes in the immune system. Here, we asked whether extended exposure to different day lengths would alter allergic responses to pentadecylcatechol (PDC), an allergenic component of poison ivy ( Toxicodendron radicans (L.) Kuntze), in Siberian hamsters ( Phodopus sungorus (Pallas, 1773)), a species exhibiting extensive seasonal variation in immune functions. We found that contact
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Roger, Albert, Maria Basagana, Aina Teniente-Serra, Nathalie Depreux, Yanina Jurgens, Clara Padro, Sira Miquel, Carolina Elduque, and Eva M. Martinez-Caceres. "Immunotheraphy in Allergic Diseases." Current Pharmaceutical Design 24, no. 11 (June 27, 2018): 1174–94. http://dx.doi.org/10.2174/1381612824666180116094048.

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The prevalence of allergic diseases is increasing worldwide. It is estimated that more than 30% of the world population is now affected by one or more allergic conditions and a high proportion of this increase is in young people. The diagnosis of allergy is dependent on a history of symptoms on exposure to an allergen together with the detection of allergen-specific IgE. Accurate diagnosis of allergies opens up therapeutic options. Allergen specific immunotherapy is the only successful disease-modifying therapy for IgE-mediated allergic diseases. New therapeutic strategies have been developed
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Besedovsky, Luciana, Mona Benischke, Jörg Fischer, Amir S. Yazdi, and Jan Born. "Human sleep consolidates allergic responses conditioned to the environmental context of an allergen exposure." Proceedings of the National Academy of Sciences 117, no. 20 (May 4, 2020): 10983–88. http://dx.doi.org/10.1073/pnas.1920564117.

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Allergies are highly prevalent, and allergic responses can be triggered even in the absence of allergens due to Pavlovian conditioning to a specific cue. Here we show in humans suffering from allergic rhinitis that merely reencountering the environmental context in which an allergen was administered a week earlier is sufficient to trigger an allergic response—but only if participants had slept after allergen exposure. This context-conditioning effect was entirely absent when participants stayed awake the night after allergen exposure or were tested in a different context. Unlike in context con
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Jacquet, Alain. "Innate Immune Responses in House Dust Mite Allergy." ISRN Allergy 2013 (February 28, 2013): 1–18. http://dx.doi.org/10.1155/2013/735031.

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Sensitizations to house dust mites (HDM) trigger strong exacerbated allergen-induced inflammation of the skin and airways mucosa from atopic subjects resulting in atopic dermatitis as well as allergic rhinitis and asthma. Initially, the Th2-biased HDM allergic response was considered to be mediated only by allergen B- and T-cell epitopes to promote allergen-specific IgE production as well as IL-4, IL-5, and IL-13 to recruit inflammatory cells. But this general molecular model of HDM allergenicity must be revisited as a growing literature suggests that stimulations of innate immune activation p
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Dr. Mayank Surana, Dr. Vineeta Pande, Dr. Sharad Agarkhedkar, and Dr. Ajit Teegala. "Correlation between Total Serum Immunoglobulin E (IgE) and Absolute Eosinophil Count (AEC) in Allergic Diseases In Children." VIMS Health Science Journal 7, no. 1 (March 6, 2020): 5–9. http://dx.doi.org/10.46858/vimshsj.7102.

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Allergy, is a clinical expression of soluble factors like IgE, histamine or eosinophils found in serum or plasma of such patients. The products that are responsible for allergy are called as Allergens. Allergens normally induce IgE production which leads to type 1 hypersensitivity response on subsequent exposure to the same allergen. The target organs are mostly nose, lung, skin and gastrointestinal tract. Atopy is also considered as a triad of Atopic dermatitis, allergic rhinitis and bronchial asthma. Raised serum IgE and AEC are proven indicators of allergic phenomenon. Various studies show
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Deak, Peter E., Baksun Kim, Amina Abdul Qayum, Jaeho Shin, Girish Vitalpur, Kirsten M. Kloepfer, Matthew J. Turner, et al. "Designer covalent heterobivalent inhibitors prevent IgE-dependent responses to peanut allergen." Proceedings of the National Academy of Sciences 116, no. 18 (April 8, 2019): 8966–74. http://dx.doi.org/10.1073/pnas.1820417116.

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Allergies are a result of allergen proteins cross-linking allergen-specific IgE (sIgE) on the surface of mast cells and basophils. The diversity and complexity of allergen epitopes, and high-affinity of the sIgE–allergen interaction have impaired the development of allergen-specific inhibitors of allergic responses. This study presents a design of food allergen-specific sIgE inhibitors named covalent heterobivalent inhibitors (cHBIs) that selectively form covalent bonds to only sIgEs, thereby permanently inhibiting them. Using screening reagents termed nanoallergens, we identified two immunodo
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Simbirtsev, A. S. "Cytokines and their role in immune pathogenesis of allergy." Russian Medical Inquiry 5, no. 1 (2021): 32–37. http://dx.doi.org/10.32364/2587-6821-2021-5-1-32-37.

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Allergy is characterized by inadequate hyperimmune response to one or several antigens with the properties of allergens. A specific pattern of abnormal synthesis of some cytokines (principal molecular mediators of the initiation, development, and regulation of allergic inflammation) has an important pathogenic role in allergy. In recent years, it is generally accepted that allergic disorders are accounted for by impaired immune regulation resulting from increased imbalanced activation of allergen-specific T helper 2 cell clones. Allergic type of immune response is currently referred to as type
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Williams, Jesse W., Melissa Y. Tjota, and Anne I. Sperling. "The Contribution of Allergen-Specific IgG to the Development of Th2-Mediated Airway Inflammation." Journal of Allergy 2012 (October 21, 2012): 1–9. http://dx.doi.org/10.1155/2012/236075.

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In both human asthmatics and animal models of allergy, allergen-specific IgG can contribute to Th2-mediated allergic inflammation. Mouse models have elucidated an important role for IgG and Fc-gamma receptor (FcγR) signaling on antigen presenting cells (APC) for the induction of airway inflammation. These studies suggest a positive feedback loop between IgG produced by the adaptive B cell response and FcγR signaling on innate immune cells. Studies of IgG and FcγRs in humans with asthma or allergic lung disease have been more controversial. Some reports have identified associations between alle
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Lei, Dawn K., and Leslie C. Grammer. "An overview of allergens." Allergy and Asthma Proceedings 40, no. 6 (November 1, 2019): 362–65. http://dx.doi.org/10.2500/aap.2019.40.4247.

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Most allergens are proteins or glycoproteins that range in molecular weight from 5000 to 100,000 Da, although polysaccharides and low-molecular-weight substances may also be allergenic. Common allergens include pollens, fungal spores, house-dust mites, and animal epithelial materials but can also include drugs, biologic products, and insect venoms. The allergic response is dependent on the route of exposure. If the exposure is to an inhaled aeroallergen, then the allergic response will be respiratory in nature. Ingested or injected exposure gives rise to gastrointestinal, cutaneous, or anaphyl
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Gushchin, I. S. "Evolutionary background of allergic reactivity: mast cells, FcεRI, IgE - three components of the effector phase of the allergic response". Russian Journal of Allergy 15, № 4 (15 грудня 2018): 5–16. http://dx.doi.org/10.36691/rja131.

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The literature data on the evolution of the main obligatory participants in the effector phase of the IgE-mediated allergic response are presented: mast cells/basophils, immunoglobulin E, and high affinity receptor for the Fcε fragment (FcεRI). Allergic reactivity is considered as the most recent evolutionary immunologically-mediated acquisition of mammals. It is aimed at recognizing small amounts of allergen entering the body in a certain time regime, and organizing an allergen-specific inflammation that carries features of elimination function. The most biologically justified way to prevent
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Rozprawy doktorskie na temat "Allergic response"

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Buske-Kirschbaum, Angelika. "Cortisol Responses to Stress in Allergic Children: Interaction with the Immune Response." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2014. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-135731.

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Allergic manifestations are increasingly common in infants and children. Accumulating evidence suggests that the ‘epidemic’ increase of childhood allergy may be associated with environmental factors such as stress. Although the impact of stress on the manifestation and exacerbation of allergy has been demonstrated, the underlying mechanisms of stress-induced exacerbation are still obscure. A growing number of studies have suggested an altered hypothalamus-pituitary-adrenal (HPA) axis function to stress in allergic children. It is speculated that a dysfunctional HPA axis in response to stress m
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Buske-Kirschbaum, Angelika. "Cortisol Responses to Stress in Allergic Children: Interaction with the Immune Response." Karger, 2009. https://tud.qucosa.de/id/qucosa%3A27671.

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Allergic manifestations are increasingly common in infants and children. Accumulating evidence suggests that the ‘epidemic’ increase of childhood allergy may be associated with environmental factors such as stress. Although the impact of stress on the manifestation and exacerbation of allergy has been demonstrated, the underlying mechanisms of stress-induced exacerbation are still obscure. A growing number of studies have suggested an altered hypothalamus-pituitary-adrenal (HPA) axis function to stress in allergic children. It is speculated that a dysfunctional HPA axis in response to stress m
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Cameron, Elizabeth Anne. "Local isotype switching to IgE within allergic nasal mucosa in response to allergen exposure." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape4/PQDD_0035/NQ64528.pdf.

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Al, Hamdani A. "The measurement of muscle strength in allergic response." Thesis, University of Salford, 1990. http://usir.salford.ac.uk/14798/.

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The purpose of this thesis was to show a relationship between muscle strength and allergic reactions. Clinics have for many years relied on what have become traditional methods for testing for allergic reactions. These include such tests as the Patch test and the Skin-Prick test. Both these tests cause discomfort and irritation to sensitive patients. The historical development of allergies is outlined, showing that scientists and clinicians in the late 18th and early 19th century were aware of certain reactions which people showed to apparently normal non-reactive foods. The development of met
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Spada, Clayton Samuel. "Mediators of acute inflammation and their roles in modulating in vivo leukocyte infiltration and pathobiologic activity in the conjunctiva." Thesis, De Montfort University, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.278549.

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Elms, Joanne. "The immune functions of immunoglobulin D (IgD) and the allergic response." Thesis, University of Sheffield, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.370051.

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Zhu, Yan [Verfasser]. "Impact of gram-negative bacteria on the allergic immune response in a mouse model of allergen-induced eczema / Yan Zhu." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2010. http://d-nb.info/1025087461/34.

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Wang, Junji. "Therapeutic vaccines based on myobacterium vaccae for the treatment of an IgE response to ovalbumin in BALB/c mice." Thesis, University College London (University of London), 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.285207.

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McDermott, Jacqueline Ruth. "The influence of dendritic cells on the differentiation of T helper cells." Thesis, University of Surrey, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.326509.

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Koprich, James B. "Ontogeny of the fetal immune response to maternal smoking in relation to allergic asthma." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/MQ60854.pdf.

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Książki na temat "Allergic response"

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Hamdani, Alharith Al. The measurement of muscle strength in allergic response. Salford: University of Salford, 1990.

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Chadwick, Derek J., David Evered, and Julie Whelan, eds. Ciba Foundation Symposium 147 - IgE, Mast Cells and the Allergic Response. Chichester, UK: John Wiley & Sons, Ltd., 1989. http://dx.doi.org/10.1002/9780470513866.

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Allergy information for teens: Health tips about allergic reactions to food, pollen, mold, and other substances, including facts about diagnosing, treating, and preventing allergic responses and complications. Detroit, MI: Omnigraphics, 2013.

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Brown, Edgar. Your body's response. Dallas, Tex: Madison Avenue Pub. Co., 1985.

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Mak, Tak W. Handbook of immune response genes. New York: Plenum Press, 1998.

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Benjamini, Eli. Immunology: A short course. 3rd ed. New York: Wiley-Liss, 1996.

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Benjamini, Eli. Immunology: A short course. 2nd ed. New York: Wiley-Liss, 1991.

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Benjamini, Eli. Immunology: A short course. New York: Liss, 1988.

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I was poisoned by my body--I have a gut feeling you could be, too!: The odyssey of a doctor who reversed fibromyalgia, leaky gut syndrome, multiple allergic responses, naturally and her life 10 years after recovery. 2nd ed. Lancaster, OH: Lucky Press, 2007.

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Munster, C. Sorg. Cytokines Regulating the Allergic Response (Cytokines). S Karger Pub, 1989.

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Części książek na temat "Allergic response"

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Turner, Keven J. "Epidemiology of the Allergic Response." In Novartis Foundation Symposia, 205–29. Chichester, UK: John Wiley & Sons, Ltd., 2007. http://dx.doi.org/10.1002/9780470513866.ch13.

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Liu, G., A. Keane-Myers, D. Miyazaki, A. Tai, and S. J. Ono. "Molecular and Cellular Aspects of Allergic Conjunctivitis." In Immune Response and the Eye, 39–58. Basel: KARGER, 1999. http://dx.doi.org/10.1159/000058748.

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Flint, Kevin Charles. "Mast Cells and the Allergic Response." In The Bloomsbury Series in Clinical Science, 1–12. London: Springer London, 1987. http://dx.doi.org/10.1007/978-1-4471-1458-1_1.

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Gumowski, P. I., S. Dunoyer-Geindre, and J. P. Latgé. "Candida Cell Wall Antigens Inducing Allergic Symptoms." In Fungal Cell Wall and Immune Response, 383–98. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-76074-7_29.

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Nagler-Anderson, Cathryn. "Helminth-Induced Immunoregulation of an Allergic Response to Food." In Parasites and Allergy, 1–13. Basel: KARGER, 2005. http://dx.doi.org/10.1159/000088876.

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Brusasco, Vito, Emanuele Crimi, and Giovanni A. Rossi. "Airway Responsiveness, Late-Phase Response, and Inflammation in Allergic Asthma." In Asthma Treatment, 155–60. Boston, MA: Springer US, 1992. http://dx.doi.org/10.1007/978-1-4615-3446-4_13.

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Kalicki, Bolesław, Agnieszka Lipińska-Opałka, Katarzyna Kowalczyk, Katarzyna Mycroft, Joanna Królikowska, Agnieszka Rustecka, and Agata Wawrzyniak. "Excessive Body Weight and Immunological Response in Children with Allergic Diseases." In Advances in Experimental Medicine and Biology, 77–87. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/5584_2019_426.

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Gregory, Greg D., Allison Bickford, Michaela Robbie-Ryan, Mindy Tanzola, and Melissa A. Brown. "MASTering the Immune Response: Mast Cells in Autoimmunity." In Mast Cells and Basophils: Development, Activation and Roles in Allergic/Autoimmune Disease, 215–31. Chichester, UK: John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/9780470033449.ch18.

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Rieber, E. P., U. Pirron, N. Endres, and J. C. Prinz. "Human Fc ε RII/CD23 in the Regulation of the Allergic Immune Response." In New Trends in Allergy III, 82–91. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-46717-2_10.

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Abramson, J., E. A. Barbu, and I. Pecht. "Regulation of Mast Cell Secretory Response to the Type I Fcε Receptor: Inhibitory Elements and Desensitization." In Mast Cells and Basophils: Development, Activation and Roles in Allergic/Autoimmune Disease, 78–94. Chichester, UK: John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/9780470033449.ch6.

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Streszczenia konferencji na temat "Allergic response"

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SANJAR, S., D. SMITH, J. MORLEY, L. MAZZONI, and C. TAPPARELLI. "PAF ANTAGONISM AND THE RESPONSE TO ALLERGEN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644865.

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Intravenous infusion of platelet activating factor (PAF) causes platelets to aggregate and accumulate within the lung. A similar effect is observed when allergen is injected into sensitised animals. Since PAF is released in allergic reactions, it might be considered to be a mediator of this phenomenon. Intrathoracic accumulation of 111-Indium labelled platelets was detected by use of collimated sodium iodide crystal detectors as a part of an automated isotope monitoring system (AIMS 8000, Mumed ltd.). Intravenous infusion of PAF (600 ng/kg/h) caused progressive increase of the intrathoracic pl
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Harris, R. S., Jose G. Venegas, Vanessa J. Kelly, Tilo Winkler, Mamary Kone, Guido Musch, Marcos F. Vidal Melo, et al. "Ventilation, Perfusion And Airway Response To Segmental Allergen Challenge In Allergic Non-Asthmatic Subjects." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a2408.

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Shah, Sajeel, Yanira Riffo Vasquez, Clive Page, and Simon Pitchford. "Platelets roll and adhere to the vascular bed in response to allergen challenge in allergic mice." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.oa4845.

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Chen, Q. L., H. P. Ho, Y. K. Suen, S. K. Kong, Wen J. Li, and C. K. Wong. "Design and fabrication of centrifugal microfluidic disk for allergic response monitoring." In 2009 14th OptoElectronics and Communications Conference (OECC). IEEE, 2009. http://dx.doi.org/10.1109/oecc.2009.5215688.

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Pollak, M., M. Shaw, D. Wilson, and F. A. Ratjen. "Bronchodilator Response in Cystic Fibrosis Patients Diagnosed with Allergic Bronchopulmonary Aspergillosis." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a5351.

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Yuksel, H., E. Tayanc, O. Yilmaz, A. Yasar, and S. Inan. "Epithelial Barrier Dysfunction and Innate Immune System Interaction in Allergic Response." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a2198.

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Wheelock, Asa M., Susanna Lundstrom, Jun Yang, Jesper Z. Haeggstrom, Bruce D. Hammock, Johan Grunewald, Magnus Nord, Marianne van Hage, Anders Eklund, and Craig E. Wheelock. "Allergic Asthmatics Exhibit Altered Response In Oxylipin Profile As Compared To Healthy And Asthmatic Controls After Allergen Provocation." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a4447.

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Sadigov, Alizaman S. "Latent Tuberculosis Infection Suppresses Airway Allergic Response In Patients With Bronchial Asthma." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a1434.

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Kelly, JFC, ER Davies, ST Holgate, X. Xu, JA Whitsett, DE Davies, and HM Haitchi. "S89 Soluble adam33 augments the pulmonary immune response promoting allergic airway sensitivity." In British Thoracic Society Winter Meeting 2017, QEII Centre Broad Sanctuary Westminster London SW1P 3EE, 6 to 8 December 2017, Programme and Abstracts. BMJ Publishing Group Ltd and British Thoracic Society, 2017. http://dx.doi.org/10.1136/thoraxjnl-2017-210983.95.

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DiGiovanni, FA, R. Ellis, J. Wattie, S. Goncharova, M. Jordana, and MD Inman. "Ozone Exposure Can Facilitate Allergen Sensitization and Enhance the Allergic Response to House Dust Mite in a Mouse Model." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a2246.

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Raporty organizacyjne na temat "Allergic response"

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Dua, Shelley, Andrew Clark, Monica Ruiz-Garcia, Simon Bond, Stephen Durham, Ian Kimber, Clare Mills, et al. The effect of sleep deprivation and exercise on reaction threshold in peanut-allergic adults: a randomised controlled study. Food Standards Agency, May 2020. http://dx.doi.org/10.46756/sci.fsa.vjv675.

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This was a randomised cross-over trial that investigated whether common extrinsic factors, such as exercise and sleep deprivation can modulate the threshold of responses to allergenic foods in a representative group of adults from the peanut allergic population.
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Shibata, Yoshimi. Oral Administration of N-acetyl-D Glucosamine Polymer Particles Down-Regulates Airway Allergic Responses. Fort Belvoir, VA: Defense Technical Information Center, May 2008. http://dx.doi.org/10.21236/ada484241.

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Shibata, Yoshimi. Oral Administration of N-Acetyl-D Glucosamine Polymer Particles Down-Regulates Airway Allergic Responses. Fort Belvoir, VA: Defense Technical Information Center, March 2005. http://dx.doi.org/10.21236/ada442684.

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Shibata, Yoshimi. Oral Administration of N-Acetyl-D Glucosamine Polymer Particles Down-Regulates Airway Allergic Responses. Fort Belvoir, VA: Defense Technical Information Center, March 2006. http://dx.doi.org/10.21236/ada454070.

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Shibata, Yoshimi. Oral Administration of N-Acetyl-D-Glucosamine Polymer Particles Down-Regulates Airway Allergic Responses. Fort Belvoir, VA: Defense Technical Information Center, March 2007. http://dx.doi.org/10.21236/ada469207.

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Shibata, Yoshimi. Oral Administration of a N-Acetyl-D-Glucosamine Polymer Particles Down-Regulates Airway Allergic Responses. Fort Belvoir, VA: Defense Technical Information Center, March 2004. http://dx.doi.org/10.21236/ada424227.

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McPhedran, R., K. Patel, B. Toombs, P. Menon, M. Patel, J. Disson, K. Porter, A. John, and A. Rayner. Food allergen communication in businesses feasibility trial. Food Standards Agency, March 2021. http://dx.doi.org/10.46756/sci.fsa.tpf160.

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Background: Clear allergen communication in food business operators (FBOs) has been shown to have a positive impact on customers’ perceptions of businesses (Barnett et al., 2013). However, the precise size and nature of this effect is not known: there is a paucity of quantitative evidence in this area, particularly in the form of randomised controlled trials (RCTs). The Food Standards Agency (FSA), in collaboration with Kantar’s Behavioural Practice, conducted a feasibility trial to investigate whether a randomised cluster trial – involving the proactive communication of allergen information a
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