Gotowe bibliografie tematyczne / Anti-atherosclerosis

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Gotowa bibliografia na temat „Anti-atherosclerosis”

Autor: Grafiati
Data publikacji: 3 czerwca 2025
Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "Anti-atherosclerosis"

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Näär, Anders M. "Anti-atherosclerosis or No Anti-atherosclerosis." Arteriosclerosis, Thrombosis, and Vascular Biology 33, no. 3 (2013): 447–48. http://dx.doi.org/10.1161/atvbaha.112.301021.

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Edwards, D. D. "Atherosclerosis KOs Anti-Spasm Fighter." Science News 132, no. 22 (1987): 342. http://dx.doi.org/10.2307/3971886.

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Bugger, Heiko, and Andreas Zirlik. "Anti-inflammatory Strategies in Atherosclerosis." Hämostaseologie 41, no. 06 (2021): 433–42. http://dx.doi.org/10.1055/a-1661-0020.

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AbstractAtherosclerotic vascular disease and its related complications are the major cause of mortality in Western societies. Atherosclerosis is a chronic inflammatory disease of the arterial wall triggered by traditional and nontraditional risk factors and mediated by inflammatory and immune responses. Recent clinical trials provided compelling evidence corroborating that atherosclerosis is an inflammatory disease and demonstrated efficacy of anti-inflammatory interventions in reducing cardiovascular events and mortality. Traditional risk factors drive vascular inflammation, further justifyin
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Kocyigit, Duygu. "Anti-inflammatory therapy in atherosclerosis." Frontiers in Bioscience 25, no. 2 (2020): 242–69. http://dx.doi.org/10.2741/4805.

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Moubayed, Sami P., Therese M. Heinonen, and Jean-Claude Tardif. "Anti-inflammatory drugs and atherosclerosis." Current Opinion in Lipidology 18, no. 6 (2007): 638–44. http://dx.doi.org/10.1097/mol.0b013e3282f0ee11.

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Bäck, Magnus, and Göran K. Hansson. "Anti-inflammatory therapies for atherosclerosis." Nature Reviews Cardiology 12, no. 4 (2015): 199–211. http://dx.doi.org/10.1038/nrcardio.2015.5.

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Dai, Zijian, Siqi Li, Yantong Meng, et al. "Capsaicin Ameliorates High-Fat Diet-Induced Atherosclerosis in ApoE−/− Mice via Remodeling Gut Microbiota." Nutrients 14, no. 20 (2022): 4334. http://dx.doi.org/10.3390/nu14204334.

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Capsaicin is a pungent alkaloid abundantly present in peppers with outstanding biological activities, including the anti-atherosclerosis effect. Previous studies revealed that gut microbiota played an important role in the beneficial effects of capsaicin, but whether it is essential for the anti-atherosclerosis effect of capsaicin is unclear. This study evaluated the anti-atherosclerosis effect of capsaicin in ApoE−/− mice and further explored the role of depleting gut microbiota in the improvement of atherosclerosis. The results showed that capsaicin administration could prevent the developme
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Cohen Arazi, H., and J. J. Badimon. "Anti-Inflammatory Effects of Anti-Platelet Treatment in Atherosclerosis." Current Pharmaceutical Design 18, no. 28 (2012): 4311–25. http://dx.doi.org/10.2174/138161212802481264.

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NASONOV, E. L., and T. V. POPKOVA. "Atherosclerosis: perspectives of anti-inflammatory therapy." Terapevticheskii arkhi 90, no. 5 (2018): 4–12. http://dx.doi.org/10.26442/terarkh20189054-12.

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Wang, Hui, Shutong Shen, Qinkao Xuan, Xiuzhi Wang, Xinli Li, and Xiangqing Kong. "Use of microRNAs as Anti-Atherosclerosis." Mini-Reviews in Medicinal Chemistry 15, no. 6 (2015): 452–58. http://dx.doi.org/10.2174/1389557515666150324123635.

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Rozprawy doktorskie na temat "Anti-atherosclerosis"

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Layne, Kerry Anne. "Anti-inflammatory effects of anti-platelet drugs : implications for atherosclerosis." Thesis, King's College London (University of London), 2018. https://kclpure.kcl.ac.uk/portal/en/theses/antiinflammatory-effects-of-antiplatelet-drugs(1da58105-e415-4241-a696-effb35badd21).html.

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Cardiovascular disease secondary to atherosclerosis is the leading cause of death worldwide. The pathophysiology of atherosclerosis is multifactorial and complex. This thesis offers insight into the regulation of several potential biomarkers of cardiovascular disease and explores the effects of anti-platelet therapy in modulating these pathways. CD14highCD16+ monocytes are elevated in patients with atherosclerosis and may be predictive of future cardiovascular events. The initial study in this PhD project assessed the effects of anti-platelet drugs on the phenotype of circulating monocytes in
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Nation, Daniel Addison. "The Oxytocinergic Anti-Inflammatory Pathway in Atherosclerosis." Scholarly Repository, 2009. http://scholarlyrepository.miami.edu/oa_dissertations/268.

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Background. Social deprivation or isolation accelerates the progression of atherosclerosis in several animal models of the disease. Conversely, stable social environment has been associated with reduction in the extent and severity of atherosclerosis. While positive social interactions are thought to be related to this protective effect, little is known about the physiological mechanisms responsible. Recently, the neurohypophyseal peptide, oxytocin (OT), has been found to play a role in both positive social interactions and cardiovascular homeostasis, suggesting that this neuropeptide ma
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Moss, Joe. "Anti-inflammatory actions of nutraceuticals : novel emerging therapies for atherosclerosis?" Thesis, Cardiff University, 2018. http://orca.cf.ac.uk/111849/.

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Background Cardiovascular disease (CVD)-related events such as myocardial infarction and stroke remain the world’s leading cause of death. The incidence of CVD-related events is expected to rise in the future due to the increase in the global prevalence of obesity and diabetes, in addition to less economically developed countries adopting a western style diet. Atherosclerosis is a chronic inflammatory disease which is the underlying cause of CVD and characterised by the build-up of fatty deposits within the walls of medium and large arteries. Macrophages play critical roles during the pathogen
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Khamis, Ramzi Yousef Jabra. "Anti-oxidised low density lipoprotein antibodies for the serology and imaging of atherosclerosis." Thesis, Imperial College London, 2014. http://hdl.handle.net/10044/1/45407.

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Atherosclerosis and its related major adverse cardiovascular events represent a major worldwide morbidity and mortality burden. The paradigm of identifying ‘vulnerable plaques’ leading to ‘vulnerable patients’ remains challenging despite recent advances in cardiovascular medicine. The project aimed to develop an antibody-based strategy for the translational development of imaging oxidised low density lipoprotein (LDL), a major constituent of vulnerable atherosclerotic plaque. It also aimed to simultaneously develop anti-oxidised LDL antibody-based serological assays for the stratification of i
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Clarke, S. C. "The effect of oestrogens and anti-oestrogens on plasma risk factors for coronary atherosclerosis and arterial flow-mediated vasodilation in patients with coronary atherosclerosis." Thesis, University of Cambridge, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.597727.

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Skeoch, Sarah. "Investigation of atherosclerosis and the effects of anti-inflammatory therapy on plaque morphology in rheumatoid arthritis." Thesis, University of Manchester, 2015. https://www.research.manchester.ac.uk/portal/en/theses/investigation-of-atherosclerosis-and-the-effects-of-anti-inflammatory-therapy-on-plaque-morphology-in-rheumatoid-arthritis(971c3727-54a8-49ff-aab7-8a6e0aa9d1ba).html.

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Introduction: Rheumatoid arthritis (RA) is a systemic autoimmune condition, characterised by an inflammatory arthritis. It is associated with a 50% increased risk of cardiovascular (CV) mortality. Chronic inflammation is thought to lead to accelerated atherosclerosis in RA. There is some evidence to suggest that patients have a more inflammatory, unstable atherosclerotic plaque phenotype. The impact of advances in RA treatment, on cardiovascular co-morbidity remains unclear. The aims of the current study were to employ non-invasive imaging techniques to test the hypothesis that RA patients hav
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Maurent, Kelly. "Synthèse de composés phénoliques de type diarylheptanoïde : évaluation de leurs propriétés antioxydantes et anti-inflammatoires." Thesis, Toulouse 3, 2017. http://www.theses.fr/2017TOU30255/document.

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L'athérosclérose est une des premières causes de mortalité dans les pays industrialisés. Elle est à l'origine de diverses pathologies telles que les infarctus du myocarde, les accidents vasculaires cérébraux et les maladies des artères coronaires. Deux facteurs clés sont à l'origine de l'athérosclérose : 1) un stress oxydant qui conduit à l'oxydation des lipoprotéines de basse densité dans la paroi artérielle et qui constitue la première étape du processus athérosclérotique ; 2) un processus inflammatoire auto-entretenu qui permet le développement des dernières étapes de l'athérogénèse conduis
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Chaston, Reve W. "SERUM ANTI-PHOSPHORYLCHOLINE AND ANTI-CARDIOLIPIN CONCENTRATIONS FOLLOWING PERIODONTAL SCALING AND ROOT PLANING." VCU Scholars Compass, 2006. http://scholarscompass.vcu.edu/etd/834.

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Atherosclerosis is an insidious disease with serious morbidity and mortality including ischemic heart disease, stroke, and myocardial infarction. This condition is progressive and can start early in life eventually leading to large plaques and arterial occlusion. Two key components of this process are the immune system and lipids; in particular, LDL which accumulates within the arterial walls and macrophages which recognize and engulf oxidized-LDL (oxLDL) to form foam cells. Knowing that certain antibodies directed against bacterial antigens such as phosphorylcholine (PC) and cardiolipin (CL)
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Ganzetti, G. S. "USE OF GENETICALLY MODIFIED MICE TO STUDY THE ANTI-INFLAMMATORY AND IMMUNOMODULATORY ROLE OF HIGH-DENSITY LIPOPROTEINS DURING ATHEROSCLEROSIS DEVELOPMENT." Doctoral thesis, Università degli Studi di Milano, 2017. http://hdl.handle.net/2434/482886.

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Introduction: High-density lipoproteins (HDLs) have several anti-atherosclerotic/anti-inflammatory properties and the apolipoprotein A-I (apoA-I), the main protein component of HDL, plays a major role. The aim of the present project was to investigate the impact of apoA-I on atherosclerosis development, phenotype and inflammation, through the use of genetically modified mice. Methods: This study was performed in C57Bl/6 wild-type mice, resistant to atherosclerosis development, and in three athero-prone mouse lines: apoEKO, apoEKO with the additional deletion of murine apoA-I (dKO) and dKO ove
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Youssouf, Latufa. "Fonctionnalisation de biopolymères extraits de macroalgues pour encapsuler des principes actifs hydrophobes." Thesis, La Réunion, 2016. http://www.theses.fr/2016LARE0030/document.

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Le diabète est caractérisé par une hyperglycémie chronique mais également par un stress oxydant et une inflammation chronique conduisant à des complications cardiovasculaires telles que l'athérosclérose. La curcumine est un polyphénol connu pour ses propriétés antioxydantes et antiinflammatoires. Elle est capable de neutraliser les radicaux libres et les médiateurs inflammatoires impliqués dans le développement des maladies métaboliques. De ce fait, la curcumine peut avoir une action préventive contre le diabète de type 2 et ses complications cardiovasculaires. Cependant la curcumine, comme de
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Książki na temat "Anti-atherosclerosis"

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Thornton, Clare, and Justin Mason. Vascular biology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0057.

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Vascular biology is the study of the physiology of the vasculature and how it may be the target for disease processes. An understanding of vascular biology is central to the study of rheumatic disease for three reasons: it is an integral part of a functioning immune system; it is the primary site of pathology in many conditions; and it is the site of the important secondary complications of chronic inflammation, endothelial dysfunction and atherosclerosis. Vascular biology requires a detailed knowledge of the anatomy and physiology of the vasculature and its constituent vessels. The multistep
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Thornton, Clare, and Justin Mason. Vascular biology. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199642489.003.0057_update_001.

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Vascular biology is the study of the physiology of the vasculature and how it may be the target for disease processes. An understanding of vascular biology is central to the study of rheumatic disease for three reasons: it is an integral part of a functioning immune system; it is the primary site of pathology in many conditions; and it is the site of the important secondary complications of chronic inflammation, endothelial dysfunction and atherosclerosis. Vascular biology requires a detailed knowledge of the anatomy and physiology of the vasculature and its constituent vessels. The multistep
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Klingenberg, Roland, and Ulf Müller-Ladner. Mechanisms of inflammation. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0270.

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This chapter provides a brief summary of the immune pathogenesis of atherosclerosis, highlighting shared features with inflammatory pathways in rheumatoid arthritis (RA) described in detail in Chapter 25.4. RA constitutes a prototype autoimmune disease primarily affecting the joints but also the heart and vessels associated with increased cardiovascular mortality. Recent years have produced a wealth of novel insights into the diversity of immune cell types which either propagate or dampen inflammation in atherogenesis. Expansion of this inherent anti-inflammatory component carried by regulator
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Yang, Zhihong, and Xiu-Fen Ming. Adventitia and perivascular adipose tissue—the integral unit in vascular disease. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0020.

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Obesity and obesity-associated metabolic disorders are highly associated with cardiovascular disease. Abnormal ectopic deposition and accumulation of adipose tissue in organs, including perivascular space (perivascular adipose tissue, PVAT) in obesity are emerging to contribute to vascular disease development through pathological paracrine and/or endocrine secretion of cytokines, namely adipokines, which are vasoactive factors including vascular relaxing and contracting factors, smooth muscle growth promoting and inhibiting factors, and pro- and anti-inflammatory factors. In obesity, productio
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Dewhurst, Alexander Timothy, and Brigitta Brandner. Intensive care management after vascular surgery. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0370.

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Vascular patients require admission to an intensive care unit at a number of stages during their hospital stay. They often have multiple co-morbidities and are at risk of major complications. Their management strategy requires a multidisciplinary approach with locally agreed pathways taking national frameworks into account. Vascular emergencies require immediate resuscitation and transfer to a tertiary cardiovascular centre. Vascular disease occurs throughout the arterial vascular tree, affecting both large and small vessels. The major cause is atherosclerosis. The management of vascular condi
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Części książek na temat "Anti-atherosclerosis"

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Autieri, Michael V. "Pro and Anti-Inflammatory Cytokine Networks in Atherosclerosis." In Atherosclerosis. John Wiley & Sons, Inc, 2015. http://dx.doi.org/10.1002/9781118828533.ch26.

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Deroissart, Justine, Florentina Porsch, Thomas Koller, and Christoph J. Binder. "Anti-inflammatory and Immunomodulatory Therapies in Atherosclerosis." In Prevention and Treatment of Atherosclerosis. Springer International Publishing, 2021. http://dx.doi.org/10.1007/164_2021_505.

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AbstractHypercholesterolemia is a major risk factor in atherosclerosis development and lipid-lowering drugs (i.e., statins) remain the treatment of choice. Despite effective reduction of LDL cholesterol in patients, a residual cardiovascular risk persists in some individuals, highlighting the need for further therapeutic intervention. Recently, the CANTOS trial paved the way toward the development of specific therapies targeting inflammation, a key feature in atherosclerosis progression. The pre-existence of multiple drugs modulating both innate and adaptive immune responses has significantly
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Orekhov, A. N., E. A. Ivanova, and Y. V. Bobryshev. "5. Cellular cholesterol retention in atherosclerosis and direct anti-atherosclerosis therapy." In Handbook of cholesterol. Wageningen Academic Publishers, 2016. http://dx.doi.org/10.3920/978-90-8686-821-6_5.

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Mackness, Bharti, and Mike Mackness. "Anti-Inflammatory Properties of Paraoxonase-1 in Atherosclerosis." In Advances in Experimental Medicine and Biology. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60761-350-3_13.

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Tahara, Nobuhiro, Atsuko Tahara, and Sho-ichi Yamagishi. "Clinical Feasibility and Monitoring of the Effects of Anti-inflammatory Therapy in Atherosclerosis." In Cardiovascular Imaging. Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-09268-3_16.

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Reaven, Peter D., and Wulf Palinski. "The Use of Animal Models to Study Diabetes and Atherosclerosis and Potential Anti-Atherosclerotic Therapies." In Diabetes and Cardiovascular Disease. Humana Press, 2005. http://dx.doi.org/10.1385/1-59259-908-7:259.

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Szekanecz, Zoltán, György Kerekes, and Yehuda Shoenfeld. "Anti-Inflammatory Therapeutic Approaches." In Inflammation and Atherosclerosis. Springer Vienna, 2011. http://dx.doi.org/10.1007/978-3-7091-0338-8_26.

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Vini, Ravindran, Sreeja Sreekumar, Juberiya M. Azeez, and Sreeja Sreeharshan. "Pomegranate Extract Protects Endothelial Cells from TNF-α Associated Damage." In Proceedings of the Conference BioSangam 2022: Emerging Trends in Biotechnology (BIOSANGAM 2022). Atlantis Press International BV, 2022. http://dx.doi.org/10.2991/978-94-6463-020-6_27.

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AbstractPomegranates are known for being rich in polyphenols and are considered to have immense therapeutic potential. The present study investigates the hypothesis that the Methanolic Extract of Pomegranate (PME), a rich source of antioxidants, may reverse the adverse effects of TNF-α in endothelial cells. This was done by pre-treating the endothelial cells EA.hy926 with PME (80 µg/ml) before subjecting them to apoptotic stimuli, which was TNF-α in combination with cyclohexamide. PME was found to rescue a population of cells from apoptosis induced by TNF-α modulating the levels of BCL2 and BA
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Agrawal, S. S. "Development of Transdermal and Transbuccal Drug Delivery Systems for Cardioactive Drugs with Special Reference to Anti- Hypertensive Agents." In Atherosclerosis, Hypertension and Diabetes. Springer US, 2003. http://dx.doi.org/10.1007/978-1-4419-9232-1_19.

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von Eckardstein, Arnold. "High Density Lipoproteins: Is There a Comeback as a Therapeutic Target?" In Prevention and Treatment of Atherosclerosis. Springer International Publishing, 2021. http://dx.doi.org/10.1007/164_2021_536.

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AbstractLow plasma levels of High Density Lipoprotein (HDL) cholesterol (HDL-C) are associated with increased risks of atherosclerotic cardiovascular disease (ASCVD). In cell culture and animal models, HDL particles exert multiple potentially anti-atherogenic effects. However, drugs increasing HDL-C have failed to prevent cardiovascular endpoints. Mendelian Randomization studies neither found any genetic causality for the associations of HDL-C levels with differences in cardiovascular risk. Therefore, the causal role and, hence, utility as a therapeutic target of HDL has been questioned. Howev
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Streszczenia konferencji na temat "Anti-atherosclerosis"

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Zheng, Guang, Junmei Zhao, Jiaqi Ru, and Hongtao Guo. "The Anti-atherosclerosis Protein Regulation Network Delivered by Onion Quercetin." In 2021 IEEE International Conference on Bioinformatics and Biomedicine (BIBM). IEEE, 2021. http://dx.doi.org/10.1109/bibm52615.2021.9669722.

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Sarker, Sunandita, Yiannis S. Chatzizisis, Srivatsan Kidambi, and Benjamin S. Terry. "Design and Development of a Novel Drug Delivery Catheter for Atherosclerosis." In 2018 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/dmd2018-6869.

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Atherosclerosis is a chronic progressive cardiovascular disease that results from plaque formation in the arteries. It is one of the leading causes of death and loss of healthy life in modern world. Atherosclerosis lesions consist of sub-endothelial accumulations of cholesterol and inflammatory cells [1]. However, not all lesions progress to the final stage to cause catastrophic ischemic cardiovascular events [2]. Early identification and treatment of high-risk plaques before they rupture, and precipitate adverse events constitutes a major challenge in cardiology today. Numerous investigations
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Erden, Abdulsamet, Uğur Canpolat, Oğuz Abdullah Uyaroğlu, et al. "SAT0372 CHANGE OF SUBCLINICAL ATHEROSCLEROSIS AFTER FIVE YEARS ANTI-TNF TREATMENTS IN PSORIATIC ARTHRITIS." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.6044.

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Cormier, J. F., A. Ko, L. P. Choo-Smith, et al. "Broadband coherent anti-Stokes Raman scattering (CARS): a potential tool for atherosclerosis diagnostic imaging." In Biomedical Optics (BiOS) 2007, edited by Nikiforos Kollias, Bernard Choi, Haishan Zeng, et al. SPIE, 2007. http://dx.doi.org/10.1117/12.701243.

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Tristantini, Dewi, M. Rizki Ramadhan, and Aisyah Hanifah. "Shelf life estimation of anti-atherosclerosis herbs using ASLT: Critical water content and sorption isotherms model." In THE 4TH BIOMEDICAL ENGINEERING’S RECENT PROGRESS IN BIOMATERIALS, DRUGS DEVELOPMENT, HEALTH, AND MEDICAL DEVICES: Proceedings of the International Symposium of Biomedical Engineering (ISBE) 2019. AIP Publishing, 2019. http://dx.doi.org/10.1063/1.5139350.

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Ortolan, A., M. Puato, G. Boschetti, et al. "AB0745 Subclinical atherosclerosis evolution in psoriatic arthritis patients treated with anti-tnf alpha: 5 years follow up." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.5545.

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Van der Heiden, K., H. C. Groen, P. C. Evans, et al. "Non-Invasive Molecular Imaging of Shear Stress-Induced Endothelial Activation and Atherosclerotic Plaque Vulnerability." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80515.

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Atherosclerosis is a lipid- and inflammation driven disease of the larger arteries and is found at specific locations in the arterial tree, i.e. at branches and bends where endothelial cells are exposed to low and low, oscillatory shear stress. Shear stress, the frictional force acting on the endothelial cells as a result of the blood flow, affects endothelial physiology. It determines the location of atherosclerotic lesion development as low and low, oscillatory shear stress induce pro-inflammatory transcription factors but reduce expression and/or activity of anti-inflammatory transcription
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Patel, Nisha S., and Alisa Morss Clyne. "A Computational Model of Fibroblast Growth Factor-2 Binding to Isolated and Intact Cell Surface Receptors: Effects of Fibroblast Growth Factor-2 Concentration, Flow and Delivery Mode." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80798.

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Fibroblast growth factor-2 (FGF2) plays an important role in both healthy vascular cell functions and pathogenesis in cancer, atherosclerosis and reduced perfusion in diabetes (1–4). FGF2 therapy and targeted drug delivery have great potential in the treatment of such diseases, but have had little clinical success. FGF2 binding kinetics to heparan sulfate proteoglycan (HSPG) and fibroblast growth factor receptors (FGFR) have been largely studied under static conditions (5), however FGF2 binding to endothelial cells occurs physiologically under fluid flow conditions. Understanding complex FGF2
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Hartley, Adam, Michelle Greene, Mikhail Caga-Anan, et al. "BS15 In vivo whole-body fluorescence molecular imaging of experimental atherosclerosis using anti-malondialdehyde-modified low-density lipoprotein humanised antibody fragment targeted nanoparticles." In British Cardiovascular Society Annual Conference, ‘100 years of Cardiology’, 6–8 June 2022. BMJ Publishing Group Ltd and British Cardiovascular Society, 2022. http://dx.doi.org/10.1136/heartjnl-2022-bcs.195.

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Hartley, Adam, Michelle Greene, Mikhail Caga-Anan, et al. "BS15 In vivo whole-body fluorescence molecular imaging of experimental atherosclerosis using anti-malondialdehyde-modified low-density lipoprotein humanised antibody fragment targeted nanoparticles." In British Cardiovascular Society Annual Conference, ‘100 years of Cardiology’, 6–8 June 2022. BMJ Publishing Group Ltd and British Cardiovascular Society, 2022. http://dx.doi.org/10.1136/heartjnl-2022-bcs.195.

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Raporty organizacyjne na temat "Anti-atherosclerosis"

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zheng, Nan, Jinyan Zhong, Longfu Jiang, and Xi Chen. Prophylactic efficacy and safety of anti-thrombotic regimens in patients with stable atherosclerosis coronary disease and (or) peripheral artery disease: A systematic review and network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.3.0049.

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