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Książki na temat „Hepatitis B virus and hepatitis C virus”

Autor: Grafiati

Data publikacji: 2 czerwca 2025

Data aktualizacji: 31 lipca 2025

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1

1944-, Gerlich W. H., ed. Research in chronic viral hepatitis. Springer-Verlag, 1993.

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2

Association, Canadian Hospital, ed. Guide to policies for health care facilities and agencies: Bloodborne pathogens, especially Human Immunodeficiency Virus (HIV), Hepatitis B Virus, (HBV) and Hepatitis C Virus (HCV). Canadian Hospital Association, 1994.

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3

Guo, Haitao, and Andrea Cuconati, eds. Hepatitis B Virus. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6700-1.

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4

W, Reesink H., ed. Hepatitis C virus. 2nd ed. Karger, 1998.

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Guo, Haitao, and Andrea Cuconati, eds. Hepatitis B Virus. Springer US, 2024. http://dx.doi.org/10.1007/978-1-0716-4027-2.

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W, Reesink H., ed. Hepatitis C virus. Karger, 1994.

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7

Shiffman, Mitchell L., ed. Chronic Hepatitis C Virus. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-1192-5.

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8

Tang, Hong, ed. Hepatitis B Virus Infection. Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-13-9151-4.

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9

Law, Mansun, ed. Hepatitis C Virus Protocols. Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-8976-8.

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10

Jirillo, Emilio, ed. Hepatitis C Virus Disease. Springer New York, 2008. http://dx.doi.org/10.1007/978-0-387-71376-2.

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11

Miyamura, Tatsuo, Stanley M. Lemon, Christopher M. Walker, and Takaji Wakita, eds. Hepatitis C Virus I. Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-56098-2.

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12

Miyamura, Tatsuo, Stanley M. Lemon, Christopher M. Walker, and Takaji Wakita, eds. Hepatitis C Virus II. Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-56101-9.

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Chayama, Kazuaki, ed. Hepatitis C Virus Treatment. Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-2416-0.

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14

1968-, Zhang Weiying, and Ye Lihong, eds. Hepatitis B virus research focus. Nova Science Publishers, 2008.

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15

Sawyer, Forrest. Hepatitis C. Films for the Humanities & Sciences, 2003.

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16

Rajen, Koshy, and Caselmann Wolfgang H, eds. Hepatitis B virus: Molecular mechanisms in disease and novel strategies for therapy. Imperial College Press, 1998.

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17

Hirsh, Jennifer Lee. Hepatitis B virus and Hepatitis A virus in Kingston, Jamaica: Maternal-child immunity. s.n.], 1989.

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18

Kao, Jia-Horng, ed. Hepatitis B Virus and Liver Disease. Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-3615-8.

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19

Berenguer, Marina, ed. Hepatitis C Virus and Liver Transplantation. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-8438-7.

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20

Liaw, Yun-Fan, and Fabien Zoulim, eds. Hepatitis B Virus in Human Diseases. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-22330-8.

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21

Kao, Jia-Horng, and Ding-Shinn Chen, eds. Hepatitis B Virus and Liver Disease. Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-4843-2.

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22

Feitelson, Mark. Molecular Components of Hepatitis B Virus. Springer US, 1985. http://dx.doi.org/10.1007/978-1-4613-2573-4.

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23

S, Fiorino, ed. Hepatitis B virus and immune response. Nova Science Publishers, 2008.

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24

V, Agnello, ed. Immunopathology of hepatitis C virus infection. Springer-Verlag, 1997.

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25

1946-, Callea F., Zorzi M. 1920-, Desmet V, Associazione volontari italiani del sangue. Brescia Division., and Postgraduate Course on Viral Hepatitis (1985 : Brescia, Italy), eds. Viral hepatitis. Springer-Verlag, 1986.

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26

Alan, McLachlan, ed. Molecular biology of the hepatitis B virus. CRC Press, 1991.

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27

Agubokwu, Juliana. Hepatitis B Virus HBV and Hepatitis C Virus HCV: Nigerian-American Immigrants Awareness, Knowledge, and Attitudes. MedCrave Group, 2023.

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28

Wilson, Deanna. Hepatitis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0035.

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Hepatitis A (HAV) and E (HEV) viruses are spread via the fecal-oral route. Hepatitis B virus (HBV) exposure is via occupational or recreational activities. Hepatitis D virus (HDV; also spread parentally) can only coinfect or superinfect those with chronic HBV. Hepatitis C (HCV) transmission is predominantly parenteral; the highest risk group is injection drug users. Prodromal-period patients with acute hepatitis present with vague constitutional symptoms when serum transaminases peak, with elevated serum bilirubin and varying levels of hepatic protein synthesis impairment; during the icteric p

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29

Ihlenfeldt, Hans Georg. Die B- und T-Zell-Epitope des Hepatitis-C-Virus. 1995.

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30

Keshav, Satish, and Palak Trivedi. Viral hepatitis. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0212.

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Hepatitis means ‘inflammation of the liver’ and is manifest with symptoms that include malaise, anorexia, fever, flu-like symptoms, and pain in the right upper quadrant of the abdomen, with the pain being caused by swelling of the liver and its capsule. Elevations in circulating hepatic enzymes, particularly aspartate transaminase and alanine transaminase, are common, with jaundice occurring some time after the onset of other symptoms and signs. There are five viruses that primarily cause viral hepatitis: hepatitis A, B, C, D, and E viruses, abbreviated HAV, HBV, HCV, HDV, and HEV, respectivel

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31

Mohr, Leonhard. Nachweis von Hepatitis C-Virus-RNA im Serum von Patienten mit Posttransfusionshepatitis und chronischer Non-A, Non B Hepatitis. 1993.

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32

Küther, Doris. Die chronische Hepatitis Non-A, Non-B: Spontanverlauf, Frequenz der Hepatitis C Virus (HCV) Infektion und therapeutische Beeinflussung durch Interferon alpha. 1992.

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33

Iske, Ludger. Nachweis genomischer Sequenzen von Hepatitis-C-Virus mittels Polymerase-Kettenreaktion bei Patienten mit akuter und chronischer Hepatitis Non A Non B. 1993.

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34

Protocolo para estimar la mortalidad por cirrosis y por carcinoma hepatocelular atribuible a las hepatitis virales B y C. Pan American Health Organization, 2021. http://dx.doi.org/10.37774/9789275323762.

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Una de las metas a alcanzar para el 2030 de la Estrategia mundial del sector de la salud contra las hepatitis víricas 2016-202: hacia el fin de las hepatitis víricas es reducir la mortalidad por el virus de la hepatitis B (VHB) y el virus de la hepatitis C (VHC). Para medir este indicador y darle seguimiento, los países requieren poner en funcionamiento un proceso sistemático para generar estimaciones nacionales de mortalidad por hepatitis virales. Esta publicación está dirigida a las instituciones y ministerios encargados de monitorizar los avances en cada país. El objetivo principal de este

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35

Levy, Barry S. Liver Disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190662677.003.0030.

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This chapter describes occupational and environmental liver disorders. It describes the types of liver function and types of liver damage, and how these functions and this damage can be assessed. Workers in healthcare and solid waste management are at increased risk hepatitis B virus and hepatitis C virus infections. Occupational exposure to swine is associated with hepatitis E virus infection. More than 100 industrial chemicals can be acutely hepatotoxic in experimental animals or humans. Metabolic reactions may affect the hepatotoxicity of chemicals. Occupational exposure to organic solvents

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36

Franceschi, Silvia, Hashem B. El-Serag, David Forman, Robert Newton, and Martyn Plummer. Infectious Agents. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0024.

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Eleven infectious agents (seven viruses, three parasites, and one bacterium) have been classified by the International Agency for Research on Cancer as carcinogenic to humans for one or more cancer sites: hepatitis B virus; hepatitis C virus; thirteen types of human papillomavirus (HPV); human immunodeficiency virus type 1 (HIV-1); human T-cell leukemia virus type 1; Epstein-Barr virus; Kaposi sarcoma herpesvirus; Helicobacter pylori; Opisthorchis viverrini; Clonorchis sinensis; and Schistosoma haematobium. Other infectious agents, such as Merkel cell polyomavirus, Plasmodium falciparum, and c

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37

Russi, Mark. Biological Hazards. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190662677.003.0016.

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This chapter describes various biological hazards and their impact on workers and others. A major focus of the chapter is biological hazards in healthcare and laboratory settings, including exposure to bloodborne pathogens and prevention of diseases related to them. Sections deal with sharps injuries, HIV/AIDS, hepatitis B virus, hepatitis C virus, tuberculosis, and other infectious diseases that can be acquired in the work environment via direct contact, droplet or airborne spread, or fecal-oral transmission. In addition, infectious agents spread by animal contact or arthropod vectors in a br

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38

Bulterys, Marc, Julia Brotherton, and Ding-Shinn Chen. Prevention of Infection-Related Cancers. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0066.

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This chapter discusses primary prevention measures that disrupt transmission of oncogenic infections. It begins by discussing vaccination against hepatitis B virus (HBV) and human papillomavirus (HPV), two major causes of cancer for which safe and effective vaccines are currently available. It briefly discusses the importance of treatment and prophylaxis against human immunodeficiency virus type 1 (HIV-1), which potentiates the virulence of other viral infections as well as directly increasing the incidence of non-Hodgkin lymphoma. It does not discuss the treatment of HBV or hepatitis C virus

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39

Reesink, H. W., ed. Hepatitis C Virus. S. Karger AG, 1998. http://dx.doi.org/10.1159/isbn.978-3-318-00249-2.

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40

Kourtis, Athena P., Shruti Chandramouli, Gonzague Jourdain, and Marc Bulterys. Hepatitis B Virus. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0004.

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Hepatitis B virus (HBV) is the most common cause of chronic viral hepatitis and hepatocellular carcinoma in the world. Worldwide, more than 250 million people are chronically infected with HBV, causing nearly 780,000 deaths each year, and mother-to-child transmission (MTCT) accounts for more than one-third of chronic HBV infections. Universal vaccination in neonates is the most effective strategy for eliminating infections worldwide. Maternal antiviral treatment during the antepartum/postpartum period for mothers with high HBV viral loads is effective in preventing HBV MTCT. Full immunization

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41

Honegger, Jonathan R. Hepatitis C Virus. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0005.

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An estimated 185 million individuals have been infected with hepatitis C virus (HCV) worldwide. Although often clinically silent for decades, chronic HCV infection predisposes to late-onset complications, including liver cirrhosis and hepatocellular carcinoma. Mother-to-child transmission (MTCT) of HCV affects approximately 5% of children born to viremic mothers and is the primary route of HCV infection in young children. While some vertically acquired HCV infections are resolved during the first years of life, many persist indefinitely. Chronically infected children tend to be asymptomatic an

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42

Koshy, Rajen, and Wolfgang H. Caselmann. Hepatitis B Virus. PUBLISHED BY IMPERIAL COLLEGE PRESS AND DISTRIBUTED BY WORLD SCIENTIFIC PUBLISHING CO., 1998. http://dx.doi.org/10.1142/p005.

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Hepatitis C virus. Saunders, 2008.

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Bamia, Christina, Sherri Stuver, and Lorelei Mucci. Cancer of the Liver and Biliary Tract. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676827.003.0012.

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Primary liver cancer is one of the most commonly occurring cancers globally, and is the second most common cause of cancer death worldwide. There are two major histologic forms of primary liver cancer: hepatocellular carcinoma (HCC) and cholangiocarcinoma. It is a rapidly and almost uniformly fatal disease, yet there is already sufficient knowledge about its major risk factors, many of which are modifiable, to make primary prevention effective. Primary liver cancer is one of the first common human cancer that was found to have an infectious etiology, with hepatitis B virus (HBV) and hepatitis

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45

Thomas London, W., Jessica L. Petrick, and Katherine A. McGlynn. Liver Cancer. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0033.

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Primary liver cancer is the sixth most frequently occurring cancer in the world and the second most common in terms of cancer deaths. The global burden of liver cancer is borne principally by countries in East Asia and Africa, where 80% of liver cancer arises. Incidence rates of liver cancer, however, have begun to decline in Asia, while rates are increasing in low-rate areas such as Europe and North America. The dominant histology of liver cancer in almost all countries is hepatocellular carcinoma (HCC). The major risk factors for HCC—chronic infection with either hepatitis B virus (HBV) or h

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46

Law, Mansun. Hepatitis C Virus Protocols. Springer New York, 2018.

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47

Hepatitis C Virus Infection. Blackwell Publishing, 1992.

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Chronic Hepatitis B. W.B. Saunders Company, 2010.

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Cooke, Graham. Viral infection. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0308.

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Viral infection includes any clinical illness caused by a pathogenic virus. Acute viral infections are amongst the most common illnesses of humans and range from minor upper respiratory tract infections to viral haemorrhagic fever. The principles in diagnosing acute viral infection are, first, recognize the syndrome, then identify key features that might suggest a specific diagnosis, and, finally, consider laboratory investigations to elucidate the specific causative agent. The host–pathogen response determines different outcomes for specific viral infections. After infection with some viruses

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Van Calsteren, Kristel. Chronic maternal infections. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713333.003.0050.

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Pregnant women diagnosed with chronic infections are a worldwide problem. In developed countries, the most frequently encountered are hepatitis B and C, toxoplasmosis, syphilis, herpes simplex, and Cytomegalovirus infections. In developing countries, human immunodeficiency virus and malaria are also seen commonly in pregnant women. Maternal infections are associated with various complications in pregnant women, but also with congenital infections with or without structural anomalies and long-term sequelae, fetal growth restriction, preterm delivery, and perinatal mortality. Moreover, increasin

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