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Artykuły w czasopismach na temat „Long non-coding Heg RNA”

Autor: Grafiati

Data publikacji: 2 czerwca 2025

Data aktualizacji: 31 lipca 2025

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1

Christensen, Niels. "Regulation of TSH Receptor Autoantibodies by a long Non-Coding RNA (Heg) and Cdk1- A Review." British Journal of Medicine and Medical Research 3, no. 3 (2013): 508–16. http://dx.doi.org/10.9734/bjmmr/2013/2616.

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Wang, Danni, Danbo Wang, Ning Wang, Zaiqiu Long, and Xuemei Ren. "Long Non-Coding RNA BANCR Promotes Endometrial Cancer Cell Proliferation and Invasion by Regulating MMP2 and MMP1 via ERK/MAPK Signaling Pathway." Cellular Physiology and Biochemistry 40, no. 3-4 (2016): 644–56. http://dx.doi.org/10.1159/000452577.

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Background/Aims: Microarray screening had found BRAF-activated non-coding RNA (BANCR) was significantly upregulated in type 1 endometrial cancer (EC). This study aimed to assess the potential role of long non-coding RNA (lncRNA) BANCR in the pathogenesis and progression of type 1 EC. Methods: Quantitative reverse transcription polymerase chain reaction (qRT-PCR) was used to confirm the expression of BANCR in type 1 EC tissue, and analyze its clinical significance. In vitro, RNA interference (siRNA) was used to investigate the biological role of BANCR in type 1 EC. Results: qRT-PCR revealed tha

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3

Haque, Sulsal-Ul, Liang Niu, Damaris Kuhnell, et al. "Differential expression and prognostic value of long non-coding RNA in HPV-negative head and neck squamous cell carcinoma." Head & Neck 40, no. 7 (2018): 1555–64. http://dx.doi.org/10.1002/hed.25136.

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Mitsuhashi, Satomi, So Nakagawa, Mitsuru Sasaki-Honda, Hidetoshi Sakurai, Martin C. Frith, and Hiroaki Mitsuhashi. "Nanopore direct RNA sequencing detects DUX4-activated repeats and isoforms in human muscle cells." Human Molecular Genetics 30, no. 7 (2021): 552–63. http://dx.doi.org/10.1093/hmg/ddab063.

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Abstract Facioscapulohumeral muscular dystrophy (FSHD) is an inherited muscle disease caused by misexpression of the DUX4 gene in skeletal muscle. DUX4 is a transcription factor, which is normally expressed in the cleavage-stage embryo and regulates gene expression involved in early embryonic development. Recent studies revealed that DUX4 also activates the transcription of repetitive elements such as endogenous retroviruses (ERVs), mammalian apparent long terminal repeat (LTR)-retrotransposons and pericentromeric satellite repeats (Human Satellite II). DUX4-bound ERV sequences also create alt

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5

Lezirovitz, Karina, Gleiciele A. Vieira-Silva, Ana C. Batissoco, et al. "A rare genomic duplication in 2p14 underlies autosomal dominant hearing loss DFNA58." Human Molecular Genetics 29, no. 9 (2020): 1520–36. http://dx.doi.org/10.1093/hmg/ddaa075.

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Abstract Here we define a ~200 Kb genomic duplication in 2p14 as the genetic signature that segregates with postlingual progressive sensorineural autosomal dominant hearing loss (HL) in 20 affected individuals from the DFNA58 family, first reported in 2009. The duplication includes two entire genes, PLEK and CNRIP1, and the first exon of PPP3R1 (protein coding), in addition to four uncharacterized long non-coding (lnc) RNA genes and part of a novel protein-coding gene. Quantitative analysis of mRNA expression in blood samples revealed selective overexpression of CNRIP1 and of two lncRNA genes

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6

Cardoso, Ana M., Catarina M. Morais, Olinda Rebelo, et al. "Downregulation of long non-protein coding RNA MVIH impairs glioblastoma cell proliferation and invasion through an miR-302a-dependent mechanism." Human Molecular Genetics 30, no. 1 (2021): 46–64. http://dx.doi.org/10.1093/hmg/ddab009.

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Abstract Glioblastoma (GB) is the most frequent and malignant type of brain tumor, for which no effective therapy exists. The high proliferative and invasive nature of GB, as well as its acquired resistance to chemotherapy, makes this type of cancer extremely lethal shortly after diagnosis. Long non-protein coding RNAs (lncRNA) are a class of regulatory RNAs whose levels can be dysregulated in the context of diseases, unbalancing several physiological processes. The lncRNA associated with microvascular invasion in hepatocellular carcinoma (lncRNA-MVIH), overexpressed in several cancers, was de

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7

Treeck, Oliver, Florian Weber, Juergen Fritsch, et al. "DSCAM-AS1 Long Non-Coding RNA Exerts Oncogenic Functions in Endometrial Adenocarcinoma via Activation of a Tumor-Promoting Transcriptome Profile." Biomedicines 10, no. 7 (2022): 1727. http://dx.doi.org/10.3390/biomedicines10071727.

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Accumulating evidence suggests that lncRNA DSCAM-AS1 acts tumor-promoting in various cancer entities. In breast cancer, DSCAM-AS1 was shown to be the lncRNA being most responsive to induction by estrogen receptor α (ERα). In this study, we examined the function of DSCAM-AS1 in endometrial adenocarcinoma using in silico and different in vitro approaches. Initial analysis of open-source data revealed DSCAM-AS1 overexpression in endometrial cancer (EC) (p < 0.01) and a significant association with shorter overall survival of EC patients (HR = 1.78, p < 0.01). In EC, DSCAM-AS1 was associated

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8

Cardamone, Giulia, Elvezia M. Paraboschi, Giulia Soldà, et al. "Not only cancer: the long non-coding RNA MALAT1 affects the repertoire of alternatively spliced transcripts and circular RNAs in multiple sclerosis." Human Molecular Genetics 28, no. 9 (2018): 1414–28. http://dx.doi.org/10.1093/hmg/ddy438.

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AbstractLong non-coding RNAs (lncRNAs) are post-transcriptional and epigenetic regulators, whose implication in neurodegenerative and autoimmune diseases remains poorly understood. We analyzed publicly available microarray data sets to identify dysregulated lncRNAs in multiple sclerosis (MS), a neuroinflammatory autoimmune disease. We found a consistent upregulation in MS of the lncRNA MALAT1 (2.7-fold increase; meta-analysis, P = 1.3 × 10−8; 190 cases, 182 controls), known to regulate alternative splicing (AS). We confirmed MALAT1 upregulation in two independent MS cohorts (1.5-fold increase;

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9

Schmitt, Heather M., William M. Johnson, Inas F. Aboobakar, et al. "Identification and activity of the functional complex between hnRNPL and the pseudoexfoliation syndrome-associated lncRNA, LOXL1-AS1." Human Molecular Genetics 29, no. 12 (2020): 1986–95. http://dx.doi.org/10.1093/hmg/ddaa021.

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Abstract Individuals with pseudoexfoliation (PEX) syndrome exhibit various connective tissue pathologies associated with dysregulated extracellular matrix homeostasis. PEX glaucoma is a common, aggressive form of open-angle glaucoma resulting from the deposition of fibrillary material in the conventional outflow pathway. However, the molecular mechanisms that drive pathogenesis and genetic risk remain poorly understood. PEX glaucoma-associated single-nucleotide polymorphisms are located in and affect activity of the promoter of LOXL1-AS1, a long non-coding RNA (lncRNA). Nuclear and non-nuclear

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10

Bjeije, Hassan, Bahram Mohammad Soltani, Mehrdad Behmanesh, and Mohammad Reza Zali. "YWHAE long non-coding RNA competes with miR-323a-3p and miR-532-5p through activating K-Ras/Erk1/2 and PI3K/Akt signaling pathways in HCT116 cells." Human Molecular Genetics 28, no. 19 (2019): 3219–31. http://dx.doi.org/10.1093/hmg/ddz146.

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AbstractYWHAE gene product belongs to the 14-3-3 protein family that mediates signal transduction in plants and mammals. Protein-coding and non-coding RNA (lncRNA) transcripts have been reported for this gene in human. Here, we aimed to functionally characterize YWHAE-encoded lncRNA in colorectal cancer-originated cells. RNA-seq analysis showed that YWHAE gene is upregulated in colorectal cancer specimens. Additionally, bioinformatics analysis suggested that YWHAE lncRNA sponges miR-323a-3p and miR-532-5p that were predicted to target K-Ras 3′UTR sequence. Overexpression of YWHAE lncRNA result

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11

Guo, Zhongjie, Xuan Lin, and Xiaoxia Guo. "LncRNA TUG1 facilitates the development of endometrial cancer via interaction with FXR1." Tropical Journal of Pharmaceutical Research 20, no. 9 (2021): 1839–44. http://dx.doi.org/10.4314/tjpr.v20i9.9.

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Purpose: To investigate the potential influence of long non-coding RNA (lncRNA) TUG1 on the development of endometrial cancer (EC).Methods: A total of 24 paired EC species and paracancerous species were collected, and the differential expressions of TUG1 in them were determined. The regulatory effects of TUG1 on proliferative and migratory potential in Ishikawa and HEC-1A cells were assessed using cell counting kit-8 (CCK-8) and Transwell assay, respectively. Potential protein binding TUG1 was predicted by bioinformatics analysis and subsequently verified using RIP (RNA-Binding Protein Immunop

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12

Tirronen, Annakaisa, Krista Hokkanen, Taina Vuorio, and Seppo Ylä-Herttuala. "Recent advances in novel therapies for lipid disorders." Human Molecular Genetics 28, R1 (2019): R49—R54. http://dx.doi.org/10.1093/hmg/ddz132.

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Abstract The prevalence of lipid disorders is alarmingly increasing in the Western world. They are the result of either primary causes, such as unhealthy lifestyle choices or inherited risk factors, or secondary causes like other diseases or medication. Atypical changes in the synthesis, processing and catabolism of lipoprotein particles may lead to severe hypercholesterolemia, hypertriglyceridemia or elevated Lp(a). Although cholesterol-lowering drugs are the most prescribed medications, not all patients achieve guideline recommended cholesterol levels with the current treatment options, emph

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13

Wang, Wei, Liang Ge, Xiao-Juan Xu, et al. "LncRNA NEAT1 promotes endometrial cancer cell proliferation, migration and invasion by regulating the miR-144-3p/EZH2 axis." Radiology and Oncology 53, no. 4 (2019): 434–42. http://dx.doi.org/10.2478/raon-2019-0051.

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Abstract Background Endometrial cancer (EC) is one of the most common gynaecological tumours in the worldwide. Long non-coding RNA (lncRNA) nuclear enriched abundant transcript 1 (NEAT1) promotes cell proliferation, migration and invasion in EC cells. However, the molecular mechanisms of NEAT1 in EC have not been fully clarified. We conducted this study to reveal the function of NEAT1 in EC tissues and cell lines. Materials and methods Cancer and adjacent tissues were collected from EC patients. HEC-1A and Ishikawa cells were cultured in vitro. NEAT1 expression was downregulated by transfectin

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14

Zhang, Xiao-Yu, Zhuo-Chang Chen, Nan Li, et al. "Exosomal transfer of activated neutrophil-derived lncRNA CRNDE promotes proliferation and migration of airway smooth muscle cells in asthma." Human Molecular Genetics 31, no. 4 (2021): 638–50. http://dx.doi.org/10.1093/hmg/ddab283.

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Abstract Activated neutrophil-derived exosomes reportedly contribute to the proliferation of airway smooth muscle cells (ASMCs), thereby aggravating the airway wall remodeling during asthma; however, the specific mechanism remains unclear. Lipopolysaccharide (LPS)-EXO and si-CRNDE-EXO were extracted from the media of human neutrophils treated with LPS and LPS + si-CRNDE (a siRNA targets long non-coding RNA CRNDE), respectively. Human ASMCs were co-cultured with LPS-EXO or si-CRNDE-EXO, and cell viability, proliferation and migration were measured. The interplay of colorectal neoplasia differen

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15

Zakaria, Nur Atikah, Md Asiful Islam, Wan Zaidah Abdullah та ін. "Epigenetic Insights and Potential Modifiers as Therapeutic Targets in β–Thalassemia". Biomolecules 11, № 5 (2021): 755. http://dx.doi.org/10.3390/biom11050755.

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Thalassemia, an inherited quantitative globin disorder, consists of two types, α– and β–thalassemia. β–thalassemia is a heterogeneous disease that can be asymptomatic, mild, or even severe. Considerable research has focused on investigating its underlying etiology. These studies found that DNA hypomethylation in the β–globin gene cluster is significantly related to fetal hemoglobin (HbF) elevation. Histone modification reactivates γ-globin gene expression in adults and increases β–globin expression. Down-regulation of γ–globin suppressor genes, i.e., BCL11A, KLF1, HBG-XMN1, HBS1L-MYB, and SOX6

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16

Bedoni, Nicola, Mathieu Quinodoz, Michele Pinelli, et al. "An Alu-mediated duplication in NMNAT1, involved in NAD biosynthesis, causes a novel syndrome, SHILCA, affecting multiple tissues and organs." Human Molecular Genetics 29, no. 13 (2020): 2250–60. http://dx.doi.org/10.1093/hmg/ddaa112.

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Abstract We investigated the genetic origin of the phenotype displayed by three children from two unrelated Italian families, presenting with a previously unrecognized autosomal recessive disorder that included a severe form of spondylo-epiphyseal dysplasia, sensorineural hearing loss, intellectual disability and Leber congenital amaurosis (SHILCA), as well as some brain anomalies that were visible at the MRI. Autozygome-based analysis showed that these children shared a 4.76 Mb region of homozygosity on chromosome 1, with an identical haplotype. Nonetheless, whole-exome sequencing failed to i

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Toubiana, Shir, Gal Larom, Riham Smoom, et al. "Regulation of telomeric function by DNA methylation differs between humans and mice." Human Molecular Genetics 29, no. 19 (2020): 3197–210. http://dx.doi.org/10.1093/hmg/ddaa206.

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Abstract The most distal 2 kb region in the majority of human subtelomeres contains CpG-rich promoters for TERRA, a long non-coding RNA. When the function of the de novo DNA methyltransferase DNMT3B is disrupted, as in ICF1 syndrome, subtelomeres are abnormally hypomethylated, subtelomeric heterochromatin acquires open chromatin characteristics, TERRA is highly expressed, and telomeres shorten rapidly. In this study, we explored whether the regulation of subtelomeric epigenetic characteristics by DNMT3B is conserved between humans and mice. Studying the DNA sequence of the distal 30 kb of the

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18

Billakanti, Sindhu, Vanessa Shehu, Kathryn M. Farrell, et al. "Selective Globin Gene Regulation By the Non-Canonical Baf Chromatin Remodeling Complex." Blood 142, Supplement 1 (2023): 553. http://dx.doi.org/10.1182/blood-2023-179001.

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The regulation of the beta-type globin genes has served as a powerful model for developmental transcriptional control, with humans expressing embryonic (HBE), two fetal (HBG1/2) and two adult (HBB and HBD) versions at the corresponding developmental stages. Reactivation of HBG in adult erythroid cells is of benefit to patients with sickle cell disease and β-thalassemia. While past efforts focused on the identification of repressors of the HBG genes, including BCL11A, ZBTB7A, and NFI-A/X, to date no transcriptional activators have been identified with selectivity for any of the globin genes. Us

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19

Sung, Yun Ju, Lisa de las Fuentes, Thomas W. Winkler, et al. "A multi-ancestry genome-wide study incorporating gene–smoking interactions identifies multiple new loci for pulse pressure and mean arterial pressure." Human Molecular Genetics 28, no. 15 (2019): 2615–33. http://dx.doi.org/10.1093/hmg/ddz070.

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Abstract Elevated blood pressure (BP), a leading cause of global morbidity and mortality, is influenced by both genetic and lifestyle factors. Cigarette smoking is one such lifestyle factor. Across five ancestries, we performed a genome-wide gene–smoking interaction study of mean arterial pressure (MAP) and pulse pressure (PP) in 129 913 individuals in stage 1 and follow-up analysis in 480 178 additional individuals in stage 2. We report here 136 loci significantly associated with MAP and/or PP. Of these, 61 were previously published through main-effect analysis of BP traits, 37 were recently

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Kazimierczyk, Marek, and Jan Wrzesinski. "Long Non-Coding RNA Epigenetics." International Journal of Molecular Sciences 22, no. 11 (2021): 6166. http://dx.doi.org/10.3390/ijms22116166.

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Long noncoding RNAs exceeding a length of 200 nucleotides play an important role in ensuring cell functions and proper organism development by interacting with cellular compounds such as miRNA, mRNA, DNA and proteins. However, there is an additional level of lncRNA regulation, called lncRNA epigenetics, in gene expression control. In this review, we describe the most common modified nucleosides found in lncRNA, 6-methyladenosine, 5-methylcytidine, pseudouridine and inosine. The biosynthetic pathways of these nucleosides modified by the writer, eraser and reader enzymes are important to underst

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21

Stower, Hannah. "Long non-coding RNA stability." Nature Reviews Genetics 13, no. 5 (2012): 298. http://dx.doi.org/10.1038/nrg3234.

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Beylerli, O. A., and I. F. Gareev. "Long non-coding RNA — perspectives?" Profilakticheskaya meditsina 23, no. 2 (2020): 124. http://dx.doi.org/10.17116/profmed202023021124.

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Ma, Xiaoxia, Chaogang Shao, Yongfeng Jin, Huizhong Wang, and Yijun Meng. "Long non-coding RNAs." RNA Biology 11, no. 4 (2014): 373–90. http://dx.doi.org/10.4161/rna.28725.

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GAO, Yuan, Ning HUI, and Shan-rong LIU. "Long non-coding RNA: research progress." Academic Journal of Second Military Medical University 31, no. 7 (2011): 790–94. http://dx.doi.org/10.3724/sp.j.1008.2011.00790.

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Hauptman, Nina, and Damjan Glavač. "Long Non-Coding RNA in Cancer." International Journal of Molecular Sciences 14, no. 3 (2013): 4655–69. http://dx.doi.org/10.3390/ijms14034655.

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Zhang, J., X. Hao, M. Yin, T. Xu, and F. Guo. "Long non-coding RNA in osteogenesis." Bone & Joint Research 8, no. 2 (2019): 73–80. http://dx.doi.org/10.1302/2046-3758.82.bjr-2018-0074.r1.

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Saxena, Alka, and Piero Carninci. "Long non-coding RNA modifies chromatin." BioEssays 33, no. 11 (2011): 830–39. http://dx.doi.org/10.1002/bies.201100084.

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Aurilia, Cinzia, Gaia Palmini, Simone Donati, Irene Falsetti, Teresa Iantomasi, and Maria Luisa Brandi. "Long non coding RNA in osteoporosis." International Journal of Bone Fragility 2, no. 3 (2022): 102–5. http://dx.doi.org/10.57582/ijbf.220203.102.

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Osteoporosis (OP) is the most common skeletal disease, caused by a lack of balance between osteoclast and osteoblast activity. This results in erosion overriding the deposition of new bone matrix, consequently leading to low-quality bone and an increased risk of incurring fragility fractures. Dual energy X-ray absorptiometry is the gold standard for the diagnosis of OP, while anti-osteoporotic drugs are the gold standard for its treatment. However, due to limitations to their use, researchers have turned to epigenetics as a substantial source of molecules that could potentially be used as diag

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Jing, Fangyuan, Huicheng Jin, Yingying Mao, et al. "Genome-wide analysis of long non-coding RNA expression and function in colorectal cancer." Tumor Biology 39, no. 5 (2017): 101042831770365. http://dx.doi.org/10.1177/1010428317703650.

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Long non-coding RNAs (lncRNAs) are widely transcribed in the genome, but their expression profile and roles in colorectal cancer are not well understood. The aim of this study was to investigate the long non-coding RNA expression profile in colorectal cancer and look for potential diagnostic biomarkers of colorectal cancer. Long non-coding RNA microarray was applied to investigate the global long non-coding RNA expression profile in colorectal cancer. Gene ontology and Kyoto Encyclopedia of Genes and Genomes pathway analyses were performed using standard enrichment computational methods. The e

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Zhang, Tianzhu, Hui Hu, Ge Yan, et al. "Long Non-Coding RNA and Breast Cancer." Technology in Cancer Research & Treatment 18 (January 1, 2019): 153303381984388. http://dx.doi.org/10.1177/1533033819843889.

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Breast cancer, one of the most common diseases among women, is regarded as a heterogeneous and complicated disease that remains a major public health concern. Recently, owing to the development of next-generation sequencing technologies, long non-coding RNAs have received extensive attention. Numerous studies reveal that long non-coding RNAs are playing important roles in tumor development. Although the biological function and molecular mechanisms of long non-coding RNAs remain enigmatic, recent researchers have demonstrated that an array of long non-coding RNAs express abnormally in cancers,

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31

Liu, Xiu-Fen, Ji-Long Hao, Tian Xie, et al. "The BRAF activated non-coding RNA: A pivotal long non-coding RNA in human malignancies." Cell Proliferation 51, no. 4 (2018): e12449. http://dx.doi.org/10.1111/cpr.12449.

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Arun, Gayatri, Disha Aggarwal, and David L. Spector. "MALAT1 Long Non-Coding RNA: Functional Implications." Non-Coding RNA 6, no. 2 (2020): 22. http://dx.doi.org/10.3390/ncrna6020022.

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The mammalian genome is pervasively transcribed and the functional significance of many long non-coding RNA (lncRNA) transcripts are gradually being elucidated. Metastasis Associated Lung Adenocarcinoma Transcript 1 (MALAT1) is one of the most well-studied lncRNAs. MALAT1 is a highly conserved nuclear retained lncRNA that is abundantly expressed in cells and tissues and has been shown to play a role in regulating genes at both the transcriptional and post-transcriptional levels in a context-dependent manner. However, Malat1 has been shown to be dispensable for normal development and viability

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Cruz-Miranda, Gabriela, Alfredo Hidalgo-Miranda, Diego Bárcenas-López, et al. "Long Non-Coding RNA and Acute Leukemia." International Journal of Molecular Sciences 20, no. 3 (2019): 735. http://dx.doi.org/10.3390/ijms20030735.

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Acute leukemia (AL) is the main type of cancer in children worldwide. Mortality by this disease is high in developing countries and its etiology remains unanswered. Evidences showing the role of the long non-coding RNAs (lncRNAs) in the pathophysiology of hematological malignancies have increased drastically in the last decade. In addition to the contribution of these lncRNAs in leukemogenesis, recent studies have suggested that lncRNAs could be used as biomarkers in the diagnosis, prognosis, and therapeutic response in leukemia patients. The focus of this review is to describe the functional

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蔡, 雅莉. "Research Progress of Long Non-Coding RNA." International Journal of Psychiatry and Neurology 05, no. 03 (2016): 54–58. http://dx.doi.org/10.12677/ijpn.2016.53009.

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Han, Pei, and Ching-Pin Chang. "Long non-coding RNA and chromatin remodeling." RNA Biology 12, no. 10 (2015): 1094–98. http://dx.doi.org/10.1080/15476286.2015.1063770.

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Heaton, Nicholas S., and Bryan R. Cullen. "Viruses hijack a long non-coding RNA." Nature 552, no. 7684 (2017): 184–85. http://dx.doi.org/10.1038/d41586-017-07692-w.

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Diederichs, S. "52: Long non-coding RNA and cancer." European Journal of Cancer 50 (July 2014): S13. http://dx.doi.org/10.1016/s0959-8049(14)50052-4.

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Kosinska-Selbi, B., M. Mielczarek, and J. Szyda. "Review: Long non-coding RNA in livestock." Animal 14, no. 10 (2020): 2003–13. http://dx.doi.org/10.1017/s1751731120000841.

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Fathizadeh, Hadis, Seyed Mohammad Gheibi Hayat, Sounkalo Dao, et al. "Long non-coding RNA molecules in tuberculosis." International Journal of Biological Macromolecules 156 (August 2020): 340–46. http://dx.doi.org/10.1016/j.ijbiomac.2020.04.030.

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Cui, Ming, Lei You, Xiaoxia Ren, Wenjing Zhao, Quan Liao, and Yupei Zhao. "Long non-coding RNA PVT1 and cancer." Biochemical and Biophysical Research Communications 471, no. 1 (2016): 10–14. http://dx.doi.org/10.1016/j.bbrc.2015.12.101.

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Gibb, Ewan A., Emily A. Vucic, Katey S. S. Enfield, et al. "Human Cancer Long Non-Coding RNA Transcriptomes." PLoS ONE 6, no. 10 (2011): e25915. http://dx.doi.org/10.1371/journal.pone.0025915.

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Jin, J., J. Liu, H. Wang, L. Wong, and N. H. Chua. "PLncDB: plant long non-coding RNA database." Bioinformatics 29, no. 8 (2013): 1068–71. http://dx.doi.org/10.1093/bioinformatics/btt107.

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Cerase, Andrea, and Gian Gaetano Tartaglia. "Long non-coding RNA-polycomb intimate rendezvous." Open Biology 10, no. 9 (2020): 200126. http://dx.doi.org/10.1098/rsob.200126.

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The interaction between polycomb-repressive complexes 1/2 (PRC1/2) and long non-coding RNA (lncRNA), such as the X inactive specific transcript Xist and the HOX transcript antisense RNA (HOTAIR), has been the subject of intense debate. While cross-linking, immuno-precipitation and super-resolution microscopy argue against direct interaction of Polycomb with some lncRNAs, there is increasing evidence supporting the ability of both PRC1 and PRC2 to functionally associate with RNA. Recent data indicate that these interactions are in most cases spurious, but nonetheless crucial for a number of cel

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Yoshimoto, Rei, Akila Mayeda, Minoru Yoshida, and Shinichi Nakagawa. "MALAT1 long non-coding RNA in cancer." Biochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms 1859, no. 1 (2016): 192–99. http://dx.doi.org/10.1016/j.bbagrm.2015.09.012.

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Chen, Zhenyao, Tianyao Lei, Xin Chen, et al. "Long non-coding RNA in lung cancer." Clinica Chimica Acta 504 (May 2020): 190–200. http://dx.doi.org/10.1016/j.cca.2019.11.031.

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Luo, Lingli, Min Wang, Xianping Li, et al. "Long non-coding RNA LOC285194 in cancer." Clinica Chimica Acta 502 (March 2020): 1–8. http://dx.doi.org/10.1016/j.cca.2019.12.004.

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Cao, Yuepeng, Tian Tian, Weijian Li, et al. "Long non-coding RNA in bladder cancer." Clinica Chimica Acta 503 (April 2020): 113–21. http://dx.doi.org/10.1016/j.cca.2020.01.008.

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Zou, Yuanzhang, and Binghai Chen. "Long non-coding RNA HCP5 in cancer." Clinica Chimica Acta 512 (January 2021): 33–39. http://dx.doi.org/10.1016/j.cca.2020.11.015.

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Li, Chenyang, Lujia Cui, Siqiong Li, Minrui Li, and Xinpu Miao. "Long non‑coding RNA Mirt2 interacts with long non‑coding RNA IFNG‑AS1 to regulate ulcerative colitis." Experimental and Therapeutic Medicine 20, no. 5 (2020): 1. http://dx.doi.org/10.3892/etm.2020.9159.

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Zhang, Heping, Jingfang Wang, Taoyuan Yu, Jingmin Wang, Jun Lu, and Zongyang Yu. "Silencing LncRNA CASC9 inhibits proliferation and invasion of colorectal cancer cells by MiR-542-3p/ILK." PLOS ONE 17, no. 4 (2022): e0265901. http://dx.doi.org/10.1371/journal.pone.0265901.

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Colorectal cancer (CRC) ranks the third in cancers and the second in the reasons of cancer-related death. More evidence indicates that long non-coding RNA participates in tumor initiation and progression. It’s known that cancer susceptibility candidate 9 is an oncogenic long non-coding RNA in CRC. miR-542-3p is a negative regulator of CRC, while integrin-linked kinase could contribute to tumor progression and chemoresistance. However, the correlation among long non-coding RNA cancer susceptibility candidate 9, miR-542-3p and integrin-linked kinase in CRC is still unclear. We demonstrated long

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