Gotowe bibliografie tematyczne / Metabolic syndrome mechanism

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Gotowa bibliografia na temat „Metabolic syndrome mechanism”

Autor: Grafiati

Data publikacji: 2 czerwca 2025

Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "Metabolic syndrome mechanism"

Ogawa, Yoshihiro. "2. Molecular Mechanism of Metabolic Syndrome." Nihon Naika Gakkai Zasshi 105, Suppl (2016): 101b—102a. http://dx.doi.org/10.2169/naika.105.101b.

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Ogawa, Yoshihiro. "2. Molecular Mechanism of Metabolic Syndrome." Nihon Naika Gakkai Zasshi 105, no. 9 (2016): 1632–36. http://dx.doi.org/10.2169/naika.105.1632.

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McNamara, Anthony. "Metabolic syndrome." InnovAiT: Education and inspiration for general practice 12, no. 10 (2019): 582–88. http://dx.doi.org/10.1177/1755738019864615.

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Metabolic syndrome is the name given to a combination of cardiovascular risk factors. These include hyperinsulinaemia, impaired glucose tolerance, hypertension, central obesity and dyslipidaemia. People with metabolic syndrome have a high risk of developing type 2 diabetes, non-alcoholic fatty liver disease, and vascular conditions including coronary artery disease, peripheral vascular disease, and stroke. Central obesity can predispose to sleep apnoea and restrictive lung disease. For many years, it has been taught that the underlying mechanism of metabolic syndrome is insulin resistance seco

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Khairutdinov, V. R., and A. V. Samtsov. "Features of treatment of patients with psoriasis with metabolic syndrome." Vestnik dermatologii i venerologii 94, no. 4 (2018): 68–72. http://dx.doi.org/10.25208/0042-4609-2018-94-4-68-72.

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The article provides information on the prevalence of metabolic syndrome in patients with psoriasis. The criteria for the diagnosis of metabolic syndrome are presented. The General mechanisms of inflam - mation development in psoriasis and diseases forming the metabolic syndrome are described. Systemic subclinical inflammatory process developing in psoriasis and metabolic syndrome is considered as the main pathogenetic mechanism of their mutual negative influence. The difficulties of treatment of patients with psoriasis with metabolic syndrome are largely associated with the choice of a safe a

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Shimabukuro, Michio. "Clinical Diagnosis of Metabolic Syndrome 2. Lipotoxicity as a Mechanism of the Metabolic Syndrome." Internal Medicine 46, no. 16 (2007): 1285. http://dx.doi.org/10.2169/internalmedicine.46.1904.

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Hashim, Khairun-Nisa, Kok-Yong Chin, and Fairus Ahmad. "The Mechanism of Honey in Reversing Metabolic Syndrome." Molecules 26, no. 4 (2021): 808. http://dx.doi.org/10.3390/molecules26040808.

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Metabolic syndrome is a constellation of five risk factors comprising central obesity, hyperglycaemia, dyslipidaemia, and hypertension, which predispose a person to cardiometabolic diseases. Many studies reported the beneficial effects of honey in reversing metabolic syndrome through its antiobesity, hypoglycaemic, hypolipidaemic, and hypotensive actions. This review aims to provide an overview of the mechanism of honey in reversing metabolic syndrome. The therapeutic effects of honey largely depend on the antioxidant and anti-inflammatory properties of its polyphenol and flavonoid contents. P

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Ferraioli, Anthony, Kara Lee Shirley, and Panakkal David. "The Role of Atypical Antipsychotics in Glucose/Insulin Dysregulation and the Evolving Role of the Psychiatrist in a New Era of Drug Treatment Options." CNS Spectrums 9, no. 11 (2004): 849–61. http://dx.doi.org/10.1017/s1092852900002261.

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ABSTRACTThis article examines the issue of atypical antipsychotics, glucose/insulin, and other metabolic derangements (ie, metabolic syndrome), including a general introduction to the health, concerns of our patients, a review of the literature, possible mechanisms of antipsychotic induced glucose dysregulation, monitoring approaches, and management and prevention of metabolic syndrome. Literature review leads to mechanism hypotheses and risk estimations, leading to guidelines for monitoring and treatment. The patient population suffers from a higher degree of baseline metabolic dysregulation

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Canale, Maria Paola, Simone Manca di Villahermosa, Giuliana Martino, et al. "Obesity-Related Metabolic Syndrome: Mechanisms of Sympathetic Overactivity." International Journal of Endocrinology 2013 (2013): 1–12. http://dx.doi.org/10.1155/2013/865965.

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The prevalence of the metabolic syndrome has increased worldwide over the past few years. Sympathetic nervous system overactivity is a key mechanism leading to hypertension in patients with the metabolic syndrome. Sympathetic activation can be triggered by reflex mechanisms as arterial baroreceptor impairment, by metabolic factors as insulin resistance, and by dysregulated adipokine production and secretion from visceral fat with a mainly permissive role of leptin and antagonist role of adiponectin. Chronic sympathetic nervous system overactivity contributes to a further decline of insulin sen

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Sharonova, L. A., S. V. Bulgakova, S. A. Burakshaev, et al. "Relationship between the main components of metabolic syndrome and oral diseases." Experimental and Clinical Gastroenterology, no. 8 (February 3, 2025): 131–42. https://doi.org/10.31146/1682-8658-ecg-228-8-131-142.

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The main components of the metabolic syndrome - obesity, insulin resistance and carbohydrate metabolism disorders, dyslipidemia, arterial hypertension - are comorbid, having a single pathogenetic mechanism for the formation of conditions. The prevalence of oral diseases exceeds the combined incidence of the five major non-communicable diseases. The article presents data on the relationship between oral diseases and the main components of metabolic syndrome. The relationship between adipose tissue dysfunction, chronic inflammation, oxidative stress, insulin resistance, atherogenesis, and the ro

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Mansilla, Eduardo, Vanina Díaz Aquino, Daniel Zambón, et al. "Could Metabolic Syndrome, Lipodystrophy, and Aging Be Mesenchymal Stem Cell Exhaustion Syndromes?" Stem Cells International 2011 (2011): 1–10. http://dx.doi.org/10.4061/2011/943216.

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One of the most important and complex diseases of modern society is metabolic syndrome. This syndrome has not been completely understood, and therefore an effective treatment is not available yet. We propose a possible stem cell mechanism involved in the development of metabolic syndrome. This way of thinking lets us consider also other significant pathologies that could have similar etiopathogenic pathways, like lipodystrophic syndromes, progeria, and aging. All these clinical situations could be the consequence of a progressive and persistent stem cell exhaustion syndrome (SCES). The main ou

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Rozprawy doktorskie na temat "Metabolic syndrome mechanism"

Morris, Tiffany J. "Investigating the molecular mechanisms of the metabolic syndrome." Thesis, University of Cambridge, 2010. https://www.repository.cam.ac.uk/handle/1810/237703.

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This thesis aims to highlight molecular mechanisms that have been altered by prenatal undernutrition and may be involved in the metabolic syndrome. Two separate studies were conducted both using a rat model developed through manipulation of the maternal diet to provoke the key features of the metabolic syndrome in adult offspring. Microarray technology was used to detect changes in gene expression in target tissues between offspring of control (normally fed, AD) and undernourished (UN) mothers to obtain a broader picture of the cellular functions and genetic pathways that may be implicated in

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Crendal, Edward. "Myocardial mechanics in metabolic syndrome and aging populations." Thesis, Australian Catholic University, 2013. https://acuresearchbank.acu.edu.au/download/bdb5ee628a2c89f8e75680d9240bdfaaaf14763b565cd0e9e523c93185e5364e/3074279/CRENDAL_EDWARD_2013.pdf.

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Cardiovascular disease (CVD) is the number one cause of death worldwide. The clustering of metabolic risk factors, defined as metabolic syndrome (MetS), and aging are two separate factors that significantly increase the risk of CVD. Both aging and MetS influence CVD by impairing heart tissue (myocardial) function. However, the exact understanding of mechanisms and evolution of these impairments are incomplete in MetS and aging populations. One of the most accessible and utilised techniques for assessing myocardial function is conventional transthoracic echocardiography, and more recently speck

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Rajkhowa, Madhurima. "Insulin resistance in polycystic ovary syndrome mechanisms and the metabolic consequences." Thesis, Keele University, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.242286.

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Malan, Leoné. "Cardiovascular dysfunction and specific coping mechanisms in Africans / L. Malan." Thesis, North-West University, 2005. http://hdl.handle.net/10394/767.

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Motivation: Cardiovascular dysfunction and hypertension are some of the leading causes of morbidity and mortality in the African population. According to the World Health Organisation the increases in these diseases are escalating in developing countries. Apart from the contributory role of genetics towards the incidence of hypertension, evidence regarding lifestyle as a determinant or marker of cardiovascular diseases in this group is not well known. The interaction of psychological and physiological mechanisms can contribute towards a broader scope of behavioural physiology in the higher pre

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Palacios, Verdú María Gabriela 1983. "Analysis of the evolving clinical phenotype and molecular mechanisms in Williams-Beuren syndrome." Doctoral thesis, Universitat Pompeu Fabra, 2015. http://hdl.handle.net/10803/324646.

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Williams-Beuren syndrome is a neurocognitive disorder with multisystemic manifestations of variable expressivity caused by a 1.55-1.83 Mb at chromosomal band 7q11.23. In this thesis project we have characterized the 7q11.23 deletion to refine the breakpoint location of the deletion, identify hospots and search for putative cis and trans-acting mechanisms involved. We have also characterized novel and previously reported metabolic alterations in a cohort of patients and explored its association with putative candidate genes using different mouse models and gene association analyses. Finally, we

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Iglesias, Miguel Angel University of New South Wales /. Garvan Institute of Medical Research Physiology &amp Pharmacology UNSW. "Mechanisms of amelioration of lipid-induced insulin resistance: role of AMP-activated protein kinase." Awarded by:University of New South Wales / Garvan Institute of Medical Research. Physiology and Pharmacology, 2004. http://handle.unsw.edu.au/1959.4/22391.

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Insulin resistance is an early marker of Type II diabetes. Excessive lipid accumulation in muscle and liver leads to insulin resistance, and lowering tissue lipids causes an enhancement of insulin action. The enzyme AMP-activated protein kinase (AMPK) is activated when cellular energy levels are compromised, such as during exercise; this enhances fuel oxidation and inhibits energy consuming processes. The hypothesis in this thesis was that activating AMPK in a lipid-induced insulin resistant state leads to tissue lipid reduction and improved insulin sensitivity. Insulin resistant high-fat fed

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Thomas, Amandine. "Hypoxie intermittente et homéostasie glucidique : étude des mécanismes d'action cellulaire A hybrid model to study pathological mutations of the human ADP/ATP carriers Visceral white fat remodeling contributes to intermittent hypoxia-induced atherogenesis The insulin sensitizing effect of topiramate involves KATP channel activation in the central nervous system The Impact of Sleep Disorders on Glucose Metabolism: Endocrine and Molecular Mechanisms Endoplasmic reticulum stress as a novel inducer of hypoxia inducible factor-1 activity: its role in the susceptibility to myocardial ischemia-reperfusion induced by chronic intermittent hypoxia Chronic intermittent hypoxia improves whole-body glucose tolerance by activating skeletal muscle AMP-activated protein kinase in mice Prolyl-4-hydroxylase 1 (PHD1) deficiency impairs whole-body glucose tolerance and insulin sensitivity in mice but does not worsen high-fat diet-induced metabolic dysfunctions Specific transcriptomic signature in response to intermittent hypoxia exposure in liver and fat tissue." Thesis, Université Grenoble Alpes (ComUE), 2015. http://www.theses.fr/2015GREAV044.

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L'hypoxie intermittente (HI), induite par les apnées du sommeil, conduit à des altérations de la sensibilité à l'insuline et de l'homéostasie glucidique mais les mécanismes impliqués restent mal connus. L'objectif de ce travail était d'étudier les effets et les mécanismes sous jacents d'une exposition chronique à l'HI sur l'homéostasie glucidique. L'HI induit une résistance à l'insuline à la fois systémique et tissulaire, ainsi qu'une amélioration de la tolérance au glucose associée à une activation de l'AMPK musculaire. L'HI cause également des altérations du foie et du tissu adipeux associée

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Dharmalingam, Backialakshmi [Verfasser]. "Immune mediated disturbances of bone, connective tissue and vascular metabolism in Complex Regional Pain Syndrome (CRPS) : a new pathogenic mechanism of therapeutic relevance / Backialakshmi Dharmalingam." Gießen : Universitätsbibliothek, 2015. http://d-nb.info/1076980287/34.

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Nduhirabandi, Frederic. "The role of melatonin in cardioprotection : an investigation into the mechanisms involved in glucose homeostasis, microvascular endothelial function and mitochondrial function in normal and insulin resistant states." Thesis, Stellenbosch : Stellenbosch University, 2014. http://hdl.handle.net/10019.1/86332.

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Thesis (PhD)-- Stellenbosch University, 2014.<br>ENGLISH ABSTRACT: Introduction: The cardioprotective actions of the hormone melatonin against myocardial ischaemiareperfusion injury (IRI) are well-established. It has recently been shown to prevent the harmful effects of hyperphagia-induced obesity on the susceptibility of the heart to IRI as well as many of the harmful effects of obesity and insulin resistance. However, the exact mechanism whereby it exerts its beneficial action is still unknown. The aims of this study were to determine the effects of relatively short-term melatonin tre

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Mesquita, Maria do Céu Vinhas Sérgio Vilão. "Síndrome metabólica em infetados pelo vírus da imunodeficiência humana." Master's thesis, [s.n.], 2014. http://hdl.handle.net/10284/4841.

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Projeto de Pós-Graduação/Dissertação apresentado à Universidade Fernando Pessoa como parte dos requisitos para obtenção do grau de Mestre em Ciências Farmacêuticas<br>A Síndrome metabólica consiste num conjunto de fatores de risco (obesidade abdominal, hipertriglicemia, diminuição do colesterol de alta densidade, hipertensão e hiperglicemia) que predispõem o indivíduo para doenças do foro cardiovascular. O facto da literatura frequentemente associar os indivíduos infetados com o vírus da imunodeficiência humana a um risco aumentado de hipertensão, hiperlipidemia e diabetes, torna importante i

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Książki na temat "Metabolic syndrome mechanism"

C, Hansen Barbara, and Bray George A, eds. The metabolic syndrome: Epidemiology, clinical treatment, and underlying mechanisms. Humana Press, 2008.

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Gandhi, Tejal, and Anita Mehta, eds. Biochemical Mechanisms for Metabolic Syndrome. Springer Nature Switzerland, 2024. https://doi.org/10.1007/978-3-031-75686-3.

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Mantzoros, Christos S. Nutrition and metabolism: Underlying mechanisms and clinical consequences. Edited by SpringerLink (Online service). Humana, 2009.

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Lester, Packer, and Sies H. 1942-, eds. Oxidative stress and inflammatory mechanisms in obesity, diabetes, and the metabolic syndrome. CRC Press, 2008.

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Takao, Kumazawa, Kruger Lawrence, and Mizumura Kazue, eds. The polymodal receptor: A gateway to pathological pain. Elsevier, 1996.

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Metabolic syndrome: Underlying mechanisms and drug therapies. John Wiley & Sons, 2011.

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Bray, George A., and Barbara C. Hansen. Metabolic Syndrome : : Epidemiology, Clinical Treatment, and Underlying Mechanisms. Humana Press, 2010.

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Bray, George A., and Barbara C. Hansen. The Metabolic Syndrome : : Epidemiology, Clinical Treatment, and Underlying Mechanisms. Humana, 2010.

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The Metabolic Syndrome:: Epidemiology, Clinical Treatment, and Underlying Mechanisms (Contemporary Endocrinology). Humana Press, 2008.

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Mukhopadhyay, Satinath, and Sunetra Mondal. Metabolic Syndrome: From Mechanisms to Interventions. Elsevier Science & Technology, 2022.

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Części książek na temat "Metabolic syndrome mechanism"

Marar, Devan, Shobha H. Ganji, Vaijinath S. Kamanna, and Moti L. Kashyap. "Mechanism of Action of Niacin: Implications for Atherosclerosis and Drug Discovery." In Metabolic Syndrome. John Wiley & Sons, Inc., 2011. http://dx.doi.org/10.1002/9780470910016.ch9.

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Wang, Minghan. "Current Antidiabetic Therapies and Mechanisms." In Metabolic Syndrome. John Wiley & Sons, Inc., 2011. http://dx.doi.org/10.1002/9780470910016.ch10.

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Wang, Minghan. "Mechanisms and Complications of Metabolic Syndrome." In Metabolic Syndrome. John Wiley & Sons, Inc., 2011. http://dx.doi.org/10.1002/9780470910016.ch7.

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Burcelin, Remy, Cendrine Cabou, Christophe Magnan, and Pierre Gourdy. "GLP-1 Biology, Signaling Mechanisms, Physiology, and Clinical Studies." In Metabolic Syndrome. John Wiley & Sons, Inc., 2011. http://dx.doi.org/10.1002/9780470910016.ch11.

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Oette, Mark, Marvin J. Stone, Hendrik P. N. Scholl, et al. "Metabolic Syndrome." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_1295.

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Ben-Shmuel, Sarit, Ran Rostoker, Eyal J. Scheinman, and Derek LeRoith. "Metabolic Syndrome, Type 2 Diabetes, and Cancer: Epidemiology and Potential Mechanisms." In Metabolic Control. Springer International Publishing, 2015. http://dx.doi.org/10.1007/164_2015_12.

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Lumeng, Carey. "Immune-Mediated Mechanisms of the Metabolic Syndrome." In Encyclopedia of Medical Immunology. Springer New York, 2014. http://dx.doi.org/10.1007/978-0-387-84828-0_179.

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Ferreira, Isabel, Roel J. J. van de Laar, and Coen D. A. Stehouwer. "Obesity, Metabolic Syndrome, Diabetes and Smoking." In Blood Pressure and Arterial Wall Mechanics in Cardiovascular Diseases. Springer London, 2014. http://dx.doi.org/10.1007/978-1-4471-5198-2_33.

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Opresko, Patricia L., Jeanine A. Harrigan, Wen-Hsing Cheng, Robert M. Brosh, and Vilhelm A. Bohr. "Proposed Biological Functions for the Werner Syndrome Protein in DNA Metabolism." In Molecular Mechanisms of Werner’s Syndrome. Springer US, 2004. http://dx.doi.org/10.1007/978-1-4419-9032-7_8.

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Rahimlou, Mehran, and Azadeh Dehghani. "Functional Food and Obesity and Metabolic Syndrome." In Molecular Mechanisms of Action of Functional Foods and Nutraceuticals for Chronic Diseases. CRC Press, 2023. http://dx.doi.org/10.1201/9781003325642-5.

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Streszczenia konferencji na temat "Metabolic syndrome mechanism"

Aragon, Jose Gerald, Mohammed A. Rattu, Alex Gechlik, Nicholas Coan, James A. Espinosa, and Alan Lucerna. "Brief Review: The Refeeding Syndrome." In 28th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2024. https://doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.88_2024.

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Here we briefly review the definition, epidemiology, pathophysiology, clinical risk scenarios, prevention and treatment of the refeeding syndrome. Refeeding syndrome is a condition which arises when a severely malnourished individual has been restarted with increased nutrition in a relatively short period of time. The refeeding process can lead to shifts in a number of fluids and electrolytes. The hallmark feature is hypophosphatemia. The true incidence of refeeding syndrome is unknown. This is said to be partly due to the lack of a universally accepted definition. Populations that are likely

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Grech, Vasiliki Sofia, Kleomenis Lotsaris, Ioanna Grech, Vasiliki Kefala, and Efstathios Rallis. "Semaglutide (Ozempic) and obesity. A comprehensive guide for aestheticians." In 1st Conference of the Hellenic Scientific Society of Aesthetics. PHARMAKON-Press, 2024. http://dx.doi.org/10.61873/rjdb1796.

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Obesity is a complex interplay of biological, genetic, behavioural, and environmental factors. Going beyond the con- ventional Body Mass Index (BMI) evaluation, the complex relationship between obesity and skin diseases unveils the evolving role of aesthetics in health promotion. Adipose tissue, traditionally seen as an energy reservoir, is unveiled as a dynamic endocrine organ, playing a crucial role in the pathophysiological mechanisms of insulin resistance and metabolic syndrome. This article navigates the skin-deep impact of obesity and unravelling its influence on dermato- logical challen

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Sharobeem, Mariam. "Investigating the Efficacy of Myo-Inositol in Adolescents with Polycystic Ovary Syndrome: A Scoping Review." In 28th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2024. http://dx.doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.196_2024.

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Polycystic ovary syndrome (PCOS) affects approximately 6 to 13% of adolescent girls in the United States.6 PCOS poses difficulty with diagnosing and treatment with adolescents as its symptoms are heterogenous and are often rationalized with other diagnoses. Key diagnostic features of PCOS include irregular menstruation, infertility, obesity, hirsutism, increased risk of diabetes mellitus, hypertension, lipid abnormalities and metabolic syndrome.12 Currently, there are no pharmacological therapies approved by the FDA to treat PCOS.5 Alternatives include pharmacological management of symptoms wi

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Lammi, Carmen. "From the bench to the bedside: the history of lupin bioactive peptides as useful ingredient for the prevention of metabolic syndrome." In 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/bwgm4089.

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Proteins derived from lupin seeds are gaining attention as a source of bioactive peptides. In fact, several pieces of evidence highlight the biological activities of lupin protein hydrolysates and peptides, including hypocholesterolemic, hypoglycemic, antimicrobial, anti-inflammatory, and immunomodulatory effects. The health-promoting activities of lupin peptides have been tested in different animal models and clinical trials. Briefly, diets containing protein from L. albus or L. angustifolius significantly reduce both total cholesterol and low-density lipoprotein (LDL) cholesterol (LDL-C) lev

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Senhorinha, Gláucia Maria, Arlys Emanuel Mendes da Silva Santos, and Douglas Daniel Dophine. "The role of metabolic syndrome in Alzheimer’s disease." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.319.

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Background: Metabolic syndrome (MS) leads to the deposits formation of insoluble protein aggregates, neuroinflammation, oxidative stress, neuronal insulin resistance, progressive insulin resistance, desensitization and β-amyloid amyloidosis in the brain, besides direct ischemic effects which are closely associated with Alzheimer’s disease (AD).1 Objectives: The present study seeks to understand the role of the metabolic syndrome in the pathophysiology of Alzheimer’s disease and to describe preventive and therapeutic interventions. Methods: PUBMED and Web of Science were the databases used, the

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Kadhim AL-SHEMERY, Maryam, Rusul Ali AL-MASAOODI, Fadhel Y KHUDHEYER, and Fatema M.Ali AL-KHAFAGE. "THE PREVALENCE OF METABOLIC SYNDROME DURING QUARANTINE PERIOD DUE COVID-19 PANDEMIC." In VI.International Scientific Congress of Pure,Applied and Technological Sciences. Rimar Academy, 2022. http://dx.doi.org/10.47832/minarcongress6-17.

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Obesity, dyslipidemia, and insulin resistance are a cluster of Metabolic syndrome. Also, stress and In Iraqi, the percentage of metabolic syndrome increased due to the risk of depression. The aim of this study was early prediction and diagnosis of the metabolic syndrome, in addition, to determine the effect of quarantine due to the CORONA-19 pandemic on metabolic syndrome in Iraq. We investigated the physiological origins of this clinical observation linking metabolic syndrome with severity and adverse outcome of COVID-19. This cross-sectional study was conducted during the period from April t

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Parikh, Devashri, Maftuna Kurbonnazarova, Ravij Upadhyay, and Andrea Iannuzzelli. "Health Benefits of Saccharomyces boulardii as a Probiotic." In 28th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2024. http://dx.doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.194_2024.

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Saccharomyces boulardii (SB), a budding yeast, within the Saccharomyces genus. It commonly used as a probiotic that has been isolated from lychee and mangosteen fruit. S. boulardii is not known to acquire resistant genes and does not last in the intestine after 3-5 days of discontinuing the ingestion. The clinical efficacy of this probiotic yeast is known to improve various diarrhea such as pediatric diarrhea, antibiotic-associated diarrhea, acute diarrhea, and traveler’s diarrhea. Additionally, when used as an adjuvant to treatment for Helicobacter pylori and Clostridium difficile infections,

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Bayer, A., E. Zrenner, H. J. Thiel, S. Ried, and D. Schmidt. "Early detection of anticonvulsant drug toxicity by colour vision tests." In Noninvasive Assessment of the Visual System. Optica Publishing Group, 1991. http://dx.doi.org/10.1364/navs.1991.md10.

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Phenytoin and Carbamazepine, especially in combination or after repeated exposure to toxic blood concentrations, can produce neurological and mental syndroms. Possible mechanisms of impaired neurological and mental function include neuronal damage, or disturbance of folic acid, monoamine or hormonal metabolism. Phenytoin and Carbamazepine may also impair the ocular system. The most common symptoms are diplopia, nystagmus and blurred vision. Cerebellar or vestibulo-ocular dysfunction is suspected as mechanism of these ocular adverse effects.

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Peng, Suyuan, Yadan Fan, Liwei Wang, et al. "Leveraging Association Rule Mining to Detect Pathophysiological Mechanisms of Chronic Kidney Disease Complicated by Metabolic Syndrome." In 2018 IEEE International Conference on Bioinformatics and Biomedicine (BIBM). IEEE, 2018. http://dx.doi.org/10.1109/bibm.2018.8621226.

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Zhu, Lin, and Grace X. Ma. "Abstract PO-173: Racial/ethnic differences and underlying behavioral mechanisms in the association between metabolic syndrome and cancer." In Abstracts: AACR Virtual Conference: Thirteenth AACR Conference on the Science of Cancer Health Disparities in Racial/Ethnic Minorities and the Medically Underserved; October 2-4, 2020. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7755.disp20-po-173.

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Raporty organizacyjne na temat "Metabolic syndrome mechanism"

Czerwaty, Katarzyna, Karolina Dżaman, Krystyna Maria Sobczyk, and Katarzyna Irmina Sikrorska. The Overlap Syndrome of Obstructive Sleep Apnea and Chronic Obstructive Pulmonary Disease: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.11.0077.

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Review question / Objective: To provide the essential findings in the field of overlap syndrome of chronic obstructive pulmonary disease and obstructive sleep apnea, including prevalence, possible predictors, association with clinical outcomes, and severity compared to both chronic obstructive pulmonary disease and obstructive sleep apnea patients. Condition being studied: OSA is characterized by complete cessation (apnea) or significant decrease (hy-popnea) in airflow during sleep and recurrent episodes of upper airway collapse cause it during sleep leading to nocturnal oxyhemoglobin desatura

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