Gotowe bibliografie tematyczne / MiRNA-10b

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Gotowa bibliografia na temat „MiRNA-10b”

Autor: Grafiati

Data publikacji: 2 czerwca 2025

Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "MiRNA-10b"

Xu, Dong, Chiang-Ching Huang, Akadia Kachaochana, et al. "MicroRNA-10a Regulation of Proinflammatory Mediators: An Important Component of Untreated Juvenile Dermatomyositis." Journal of Rheumatology 43, no. 1 (2015): 161–68. http://dx.doi.org/10.3899/jrheum.141474.

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Objective.To identify differentially expressed microRNA (miRNA) in muscle biopsies (MBx) from 15 untreated children with juvenile dermatomyositis (JDM) compared with 5 controls.Methods.Following MBx miRNA profiling, differentially expressed miRNA and their protein targets were validated by quantitative real-time PCR (qRT-PCR) and immunological assay. The association of miRNA-10a and miRNA-10b with clinical data was evaluated, including Disease Activity Score (DAS), von Willebrand factor antigen (vWF:Ag), nailfold capillary end row loops, duration of untreated disease, and tumor necrosis factor

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Łosiewicz, Katarzyna, Małgorzata Chmielewska-Krzesińska, Piotr Socha, Anna Jakimiuk, and Krzysztof Wąsowicz. "MiRNA-21, miRNA-10b, and miRNA-34a Expression in Canine Mammary Gland Neoplasms." Bulletin of the Veterinary Institute in Pulawy 58, no. 3 (2014): 447–51. http://dx.doi.org/10.2478/bvip-2014-0068.

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Abstract The expression of miRNA-21, miRNA-10b, and miRNA-34a in malignant and benign tumours and non-neoplastic lesions in canine mammary gland, using real-time PCR with TaqMan probes was determined. The expression in normal tissues was compared to neoplastic and non-neoplastic lesions using one-way ANOVA test. Significant changes in miRNA expression in neoplastic tissues, as compared to normal ones, were demonstrated. In all neoplastic tissues, the miRNA-21 expression increased while in non-neoplastic lesions slightly decreased in comparison to normal ones. MiRNA-10b expression in malignant

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Chehkun, V. F., T. Borikun, and N. Yu Lukianova. "EFFECT OF 5-AZACYTIDINE ON miRNA EXPRESSION IN HUMAN BREAST CANCER CELLS WITH DIFFERENT SENSITIVITY TO CYTOSTATICS." Experimental Oncology 38, no. 1 (2016): 26–30. http://dx.doi.org/10.31768/2312-8852.2016.38(1):26-30.

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Aim: To analyze expression of miRNA in human breast cancer cells, sensitive and resistant to cisplatin and doxorubicin, and to explore possible modification of drug sensitivity via treatment of cells with 5-azacytidine (5-aza), a demethylating agent. Materials and Methods: The study was performed on wild-type MCF-7 cell line (MCF-7/S) and its two sublines MCF-7/Dox and MCF-7/DDP resistant to doxorubicin and cisplatin, respectively. Cells were treated with 5-aza, cisplatin, doxorubicin and their combinations. Relative expression levels of miRNA-221, -200b, -320a, -10b, -34a, -122 and -29b were

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Abdallah, Mays, Ismail H. Aziz, and Ahmed Zuhair Alsammarraie. "Assessment of miRNA-10b Expression Levels as a Potential Precursor to Metastasis in Localized and Locally Advanced/Metastatic Breast Cancer among Iraqi Patients." International Journal of Breast Cancer 2024 (February 28, 2024): 1–8. http://dx.doi.org/10.1155/2024/2408355.

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Breast cancer (BC) stands as the most prevalent form of carcinoma among women, ranking as the second leading cause of cancer-related mortality in the female population. The objective of this study is to assess the expression of miR-10b and determine its diagnostic and prognostic significance in breast cancer patients across various disease stages. The investigation was carried out in Baghdad at the Oncology Teaching Hospital within Baghdad Medical City and the Oncology Unit at Al-Yarmouk Teaching Hospital. A total of 150 samples were included and divided into two groups: the blood group consis

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Niedźwiecki, Sebastian, Janusz Piekarski, Bożena Szymańska, Zofia Pawłowska, and Arkadiusz Jeziorski. "Expression level of serum circulating miRNA-21, miRNA-10b and miRNA-200c in breast cancer patients with sentinel lymph node metastasis*." Postępy Higieny i Medycyny Doświadczalnej 73 (June 25, 2019): 325–32. http://dx.doi.org/10.5604/01.3001.0013.2562.

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MicroRNAs (miRNAs) act a role in regulation numerous processes crucial for oncogenesis. The aim of the study was to compare the blood serum concentrations of selected microRNAs (miRNA-21, miRNA-10b and miRNA-200c) between breast cancer patients without sentinel lymph node metastasis (Group 1) and those with metastasis (Group 2). The serum levels of miRNA-21, miRNA-10b and miRNA-200c were measured with using TaqMan PCR assays performed on a 7900HT Fast Real-Time PCR System in two groups of breast cancer patients: Group 1 – without sentinel lymph node metastasis (32 patients) and Group 2 – with

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Jurj, Maria-Ancuta, Michael A. Attathikhun, Meng Chen, et al. "Abstract 1186: microRNA-targeted small molecule inhibitors in metastatic cancers." Cancer Research 85, no. 8_Supplement_1 (2025): 1186. https://doi.org/10.1158/1538-7445.am2025-1186.

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Background: Gastric carcinoma is a highly heterogeneous disease with diverse subtypes, each with distinct histopathological and clinical characteristics, complicating prognosis and treatment. High mortality is driven by molecular and microenvironmental changes, often leading to peritoneal carcinomatosis. MicroRNAs (miRs), particularly miR-10b, play key roles in gastric cancer by promoting drug resistance, migration, and invasiveness. Targeting miR-10b with Small Molecule Inhibitors of miRNAs (SMIRs) offers a novel approach to disrupting RNA-small molecule interactions, presenting promising str

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Juwita, Juwita. "MICRORNA-21, MICRORNA-155 DAN MICRORNA-10B: BAGAIMANA PERANNYA PADA KANKER PAYUDARA?" Jurnal Kedokteran Syiah Kuala 17, no. 2 (2017): 119–25. http://dx.doi.org/10.24815/jks.v17i2.8991.

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Abstrak. Kanker payudara merupakan jenis kanker yang masih banyak diderita perempuan, baik di negara maju maupun berkembang. Perubahan genetik pada gen regulator utama menyebabkan perubahan ekspresi protein sehingga memicu terjadinya kanker. Saat ini diketahui bahwa microRNA berperan sebagai regulator utama gen manusia dan juga terlibat dalam karsinogenesis kanker payudara. MicroRNA-21 (miR-21), microRNA-155 (miR-155) dan microRNA-10b (miR-10b) merupakan oncogenic miRNA yang memicu perkembangan kanker payudara melalui inhibisi protein tumorsuppressor. Tinjauan ini membahas mengenai peran, gen

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Al-Mawlah, Yasir Haider, Asma’a H. Mohamed, Ali Mohammad Abd-Alameer, et al. "Assessment of the Specificity and Stability of Micro-RNAs as a Forensic Gene Marker." Biomedical and Biotechnology Research Journal (BBRJ) 7, no. 4 (2023): 569–76. http://dx.doi.org/10.4103/bbrj.bbrj_174_23.

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Abstract Background: Forensic investigations depend on bodily fluid analysis to identify the perpetrators. Identifying perpetrators requires knowledge about suspects’ body fluids. Due to their durability and tissue-specific expression patterns, miRNAs may be forensic indicators. However, miRNA expression patterns in various bodily fluids are seldom compared. This study examined miR-372, miR-135p, miR-124-3p, miR-16, and miR-10b expression in seminal fluids, blood stains, and vaginal secretions using quantitative PCR using SNORD-47 as a reference gene. This research compared miRNA expression le

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Zhang, Zhang, Shen, and Sun. "Novel MicroRNA Biomarkers for Colorectal Cancer Early Diagnosis and 5-Fluorouracil Chemotherapy Resistance but Not Prognosis: A Study from Databases to AI-Assisted Verifications." Cancers 12, no. 2 (2020): 341. http://dx.doi.org/10.3390/cancers12020341.

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Colorectal cancer (CRC) is one of the major causes of cancer death worldwide. In general, early diagnosis for CRC and individual therapy have led to better survival for the cancer patients. Accumulating studies concerning biomarkers have provided positive evidence to improve cancer early diagnosis and better therapy. It is, however, still necessary to further investigate the precise biomarkers for cancer early diagnosis and precision therapy and predicting prognosis. In this study, AI-assisted systems with bioinformatics algorithm integrated with microarray and RNA sequencing (RNA-seq) gene ex

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Yang, Rui-qi, Zhe-zhu Jin, San-ya Jiang, and Yong-jun Jin. "LncRNA GAS5 Interacts with MicroRNA-10b to Inhibit Cell Proliferation and Migration and Induces Apoptosis in Colorectal Cancer." Computational and Mathematical Methods in Medicine 2022 (January 22, 2022): 1–16. http://dx.doi.org/10.1155/2022/4996870.

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Objective. The purpose of this study was to study the effects of the GAS5/microRNA-10b (miR-10b) axis on proliferation, migration, and apoptosis of colorectal cancer (CRC). Methods. The expression levels of GAS5 and miR-10b in CRC tissues and cells were detected by quantitative real-time polymerase chain reaction (qRT-PCR). Wound healing experiment was used to detect the effects of GAS5 and miR-10b on the migration of CRC cells. The luciferase reporter gene experiment was used to verify miRNA targets. Immunohistochemical assay was used to detect the expression of proteins related to metastasis

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Części książek na temat "MiRNA-10b"

Mustafov, Denis, Sara Seriah, Roozba Malik, and Maria Braoudaki. "MicroRNA Biomarkers in Primary Brain Malignancies." In Epigenetics - Regulation and New Perspectives [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.108386.

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Despite the concerted efforts within the management of brain malignancies over the past few decades, primary brain cancers remain an obscure challenge with unfavourable outcomes for the patients. Glioblastomas (GBM) and medulloblastomas afford the most prevalent brain tumours and account for markedly high mortality rates within affected patients. The unmet clinical requirements for an early diagnostic biomarker and effective treatment have shed light onto microRNAs (miRNAs). These are small, endogenous noncoding RNAs involved in a wide spectrum of biological processes, such as post-translation

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Streszczenia konferencji na temat "MiRNA-10b"

Pal, Rekha, and Stephanie Greene. "Abstract 3082: miRNA-10b promotes medulloblastoma proliferation and survival via Bcl-2." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-3082.

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