Готові списки джерел за темами / AMPK signalling

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Добірка наукової літератури з теми "AMPK signalling"

Автор: Grafiati
Опубліковано: 10 січня 2023
Оновлено: 31 липня 2025

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Статті в журналах з теми "AMPK signalling"

1

Baumann, Philipp, Sonja Mandl-Weber, Bertold Emmerich, Christian Straka, Daniel Franke, and Ralf Schmidmaier. "Myeloma Cell Proliferation Is Inhibitied by the Activation of Adenosine Monophosphate Activated Protein Kinase (AMPK)." Blood 108, no. 11 (2006): 5045. http://dx.doi.org/10.1182/blood.v108.11.5045.5045.

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Abstract In multiple myeloma (MM), a network of cytokines in the bone marrow microenvironment promotes myeloma cell proliferation. Consequent inhibition of intracellular signalling in the myeloma cells seems to be a promising strategy to encounter disease progression. The multiple myeloma cell lines U266, OPM-2, RPMI-8226 and NCI-H929 were incubated with the AMPK activators AICAr and D942. Basal and cytokine stimulated proliferation rates of myeloma cells were measured by the WST-1 assay. Alterations of the cell cycle were determined by flow cytometry after staining with propidium iodide. Intr
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2

Shukal, Dhaval K., Pooja B. Malaviya, and Tusha Sharma. "Role of the AMPK signalling pathway in the aetiopathogenesis of ocular diseases." Human & Experimental Toxicology 41 (January 2022): 096032712110631. http://dx.doi.org/10.1177/09603271211063165.

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Background AMP-activated protein kinase (AMPK) plays a precise role as a master regulator of cellular energy homeostasis. AMPK is activated in response to the signalling cues that exhaust cellular ATP levels such as hypoxia, ischaemia, glucose depletion and heat shock. As a central regulator of both lipid and glucose metabolism, AMPK is considered to be a potential therapeutic target for the treatment of various diseases, including eye disorders. Objective To review all the shreds of evidence concerning the role of the AMPK signalling pathway in the pathogenesis of ocular diseases. Method Scie
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3

Heidorn-Czarna, Malgorzata, Herbert-Michael Heidorn, Sanjanie Fernando, et al. "Chronic Activation of AMPK Induces Mitochondrial Biogenesis through Differential Phosphorylation and Abundance of Mitochondrial Proteins in Dictyostelium discoideum." International Journal of Molecular Sciences 22, no. 21 (2021): 11675. http://dx.doi.org/10.3390/ijms222111675.

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Mitochondrial biogenesis is a highly controlled process that depends on diverse signalling pathways responding to cellular and environmental signals. AMP-activated protein kinase (AMPK) is a critical metabolic enzyme that acts at a central control point in cellular energy homeostasis. Numerous studies have revealed the crucial roles of AMPK in the regulation of mitochondrial biogenesis; however, molecular mechanisms underlying this process are still largely unknown. Previously, we have shown that, in cellular slime mould Dictyostelium discoideum, the overexpression of the catalytic α subunit o
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4

Okada, Motohiro, Kouji Fukuyama, and Eishi Motomura. "Dose-Dependent Biphasic Action of Quetiapine on AMPK Signalling via 5-HT7 Receptor: Exploring Pathophysiology of Clinical and Adverse Effects of Quetiapine." International Journal of Molecular Sciences 23, no. 16 (2022): 9103. http://dx.doi.org/10.3390/ijms23169103.

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Recent pharmacological studies indicated that the modulation of tripartite-synaptic transmission plays important roles in the pathophysiology of schizophrenia, mood disorders and adverse reactions. Therefore, to explore the mechanisms underlying the clinical and adverse reactions to atypical antipsychotics, the present study determined the effects of the sub-chronic administration of quetiapine (QTP: 3~30 μM) on the protein expression of 5-HT7 receptor (5-HT7R), connexin43 (Cx43), cAMP level and intracellular signalling, Akt, Erk and adenosine monophosphate-activated protein kinase (AMPK) in c
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5

Lim, Chung Thong, Blerina Kola, and Márta Korbonits. "AMPK as a mediator of hormonal signalling." Journal of Molecular Endocrinology 44, no. 2 (2009): 87–97. http://dx.doi.org/10.1677/jme-09-0063.

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AMP-activated protein kinase (AMPK) is a key molecular player in energy homeostasis at both cellular and whole-body levels. AMPK has been shown to mediate the metabolic effects of hormones such as leptin, ghrelin, adiponectin, glucocorticoids and insulin as well as cannabinoids. Generally, activated AMPK stimulates catabolic pathways (glycolysis, fatty acid oxidation and mitochondrial biogenesis) and inhibits anabolic pathways (gluconeogenesis, glycogen, fatty acid and protein synthesis), and has a direct appetite-regulating effect in the hypothalamus. Drugs that activate AMPK, namely metformi
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6

Steinberg, Gregory, R. "Cytokine Regulation of AMPK signalling." Frontiers in Bioscience Volume, no. 14 (2009): 1902. http://dx.doi.org/10.2741/3350.

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Strembitska, Anastasiya, Sarah Mancini, Jonathan Gamwell, Timothy Palmer, George Baillie, and Ian Salt. "A769662 Inhibits Insulin-Stimulated Akt Activation in Human Macrovascular Endothelial Cells Independent of AMP-Activated Protein Kinase." International Journal of Molecular Sciences 19, no. 12 (2018): 3886. http://dx.doi.org/10.3390/ijms19123886.

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Protein kinase B (Akt) is a key enzyme in the insulin signalling cascade, required for insulin-stimulated NO production in endothelial cells (ECs). Previous studies have suggested that AMP-activated protein kinase (AMPK) activation stimulates NO synthesis and enhances insulin-stimulated Akt activation, yet these studies have largely used indirect activators of AMPK. The effects of the allosteric AMPK activator A769662 on insulin signalling and endothelial function was therefore examined in cultured human macrovascular ECs. Surprisingly, A769662 inhibited insulin-stimulated NO synthesis and Akt
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Ahwazi, Danial, Katyayanee Neopane, Greg R. Markby, et al. "Investigation of the specificity and mechanism of action of the ULK1/AMPK inhibitor SBI-0206965." Biochemical Journal 478, no. 15 (2021): 2977–97. http://dx.doi.org/10.1042/bcj20210284.

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SBI-0206965, originally identified as an inhibitor of the autophagy initiator kinase ULK1, has recently been reported as a more potent and selective AMP-activated protein kinase (AMPK) inhibitor relative to the widely used, but promiscuous inhibitor Compound C/Dorsomorphin. Here, we studied the effects of SBI-0206965 on AMPK signalling and metabolic readouts in multiple cell types, including hepatocytes, skeletal muscle cells and adipocytes. We observed SBI-0206965 dose dependently attenuated AMPK activator (991)-stimulated ACC phosphorylation and inhibition of lipogenesis in hepatocytes. SBI-
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9

Neumann, Dietbert, and Benoit Viollet. "AMP-Activated Protein Kinase Signalling." International Journal of Molecular Sciences 20, no. 3 (2019): 766. http://dx.doi.org/10.3390/ijms20030766.

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Carroll, Bernadette, and Elaine A. Dunlop. "The lysosome: a crucial hub for AMPK and mTORC1 signalling." Biochemical Journal 474, no. 9 (2017): 1453–66. http://dx.doi.org/10.1042/bcj20160780.

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Much attention has recently been focussed on the lysosome as a signalling hub. Following the initial discovery that localisation of the nutrient-sensitive kinase, mammalian target of rapamycin complex 1 (mTORC1), to the lysosome was essential for mTORC1 activation, the field has rapidly expanded to reveal the role of the lysosome as a platform permitting the co-ordination of several homeostatic signalling pathways. Much is now understood about how the lysosome contributes to amino acid sensing by mTORC1, the involvement of the energy-sensing kinase, AMP-activated protein kinase (AMPK), at the
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Дисертації з теми "AMPK signalling"

1

Mancini, Sarah J. "Regulation of inflammatory signalling in adipocytes by AMPK." Thesis, University of Glasgow, 2014. http://theses.gla.ac.uk/5130/.

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AMP-activated protein kinase (AMPK) has been proposed to be a potential therapeutic target for patients with Type 2 diabetes and the metabolic syndrome. While the role of AMPK in muscle and liver is relatively well-characterised, less is known about the role of AMPK in the other principal metabolic tissue, adipose. Obesity is associated with the chronic, sub-clinical inflammation of adipose tissue. Characteristic hypertrophic adipocytes and the elevated infiltration and activation of macrophages stimulate production of cytokines and chemokines, including tumour necrosis factor-alpha (TNF-a), i
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2

Ovens, Ashley John. "Moving towards isoform-specific AMPK activation." Phd thesis, Australian Catholic University, 2021. https://acuresearchbank.acu.edu.au/download/368ef3f9b6ef00c34227af8bea2a17693253138f442217dfd893f8986519d895/36925900/Ovens_2021_Moving_towards_isoform-specific_AMPK_activation.pdf.

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The AMP-activated protein kinase (AMPK) αβγ heterotrimer is a highly conserved serine/threonine protein kinase that acts as a metabolic fuel sensor and is crucial for maintaining cellular energy homeostasis. Mammalian AMPK forms complexes in a 1:1:1 ratio made of unique subunit isoform variations (α1, α2, β1, β2, γ1, γ2, γ3) that allow for 12 distinct AMPK complexes to form, where each complex is subject to a range of modifications such as phosphorylation. Each isoform exhibits distinct tissue-expression signatures, α2 and β2 are expressed in a range of tissues but display high expression in s
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3

Dandapani, Madhumita. "The AMPK signalling pathway in cancer and DNA damage." Thesis, University of Dundee, 2013. https://discovery.dundee.ac.uk/en/studentTheses/9a3ca91b-dd8b-4407-bed2-cd9dd4c00ca7.

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4

Narbonne, Patrick. "Roles of LKB1/AMPK signalling in the «C.elegans» dauer larva." Thesis, McGill University, 2009. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=32247.

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Many organisms can execute a dormant state or diapause to survive harsh environmental conditions for extended durations. When Caenorhabditis elegans larvae enter the dauer diapause, they completely arrest development and feeding, but remain active and motile, yet become stress-resistant and extremely long-lived. Entry into dauer is associated with a reduction in insulin-like signalling, the establishment of a generalized cell cycle arrest, the accumulation of nutritive resources and a concomitant global c
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5

Hinchy, Elizabeth. "How cellular ATP/ADP ratios and reactive oxygen species affect AMPK signalling." Thesis, University of Cambridge, 2017. https://www.repository.cam.ac.uk/handle/1810/270029.

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Mitochondria are key generators of cellular ATP, vital to complex life. Historically, mitochondrial generation of reactive oxygen species (ROS) was considered to be an unregulated process, produced by dysfunctional mitochondria. More recently, mitochondrial ROS generated by complex I, particularly by the process of reverse electron transfer (RET), has emerged as a potentially biologically relevant signal that is tightly-regulated and dependent on mitochondrial status. ROS production by RET is reported to play a role in the innate immune response and lifespan extension in fruit flies. One way i
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Speirs, Claire. "Examination of potential mechanisms linking AMPK to inhibition of IL-6 signalling." Thesis, University of Glasgow, 2017. http://theses.gla.ac.uk/8185/.

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Considerable recent evidence supports the role of AMP-activated protein kinase (AMPK) as an anti-inflammatory mediator, yet the mechanisms of its anti-inflammatory actions are only starting to be unravelled. Inappropriate cytokine stimulated Janus kinase (JAK)-signal transducer and activator of transcription (STAT) signalling is a key feature of many pro-inflammatory events, including atherogenesis. Previous unpublished studies in our group have investigated whether AMPK modifies cytokine stimulation of JAK-STAT signalling in HUVECs. These preliminary investigations demonstrated that pre-treat
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Raja, Erna. "Cross-regulation between TGFβ/BMP Signalling and the metabolic LKB1 pathway". Doctoral thesis, Ludwig Institute for Cancer Research, Faculty of Medicine, Uppsala University, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-178181.

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Анотація:
Cell signalling determines physiological responses to many cellular stimuli and environmental changes. The transforming growth factor-beta (TGFβ)/bone morphogenetic protein (BMP) signalling pathways begin by binding of ligand to the heterodimeric receptor complex, followed by activation of Smads that translocate to the nucleus to regulate transcription of genes that further mediate cellular physiology. The TGFβ/BMP pathways are very important for proper tissue development and homeostasis, thus precise spatial and temporal regulation of the signalling pathway is required and achieved by many po
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8

Bacon, Sandra. "Investigation of plant derived compounds mediating cell signalling effects on FOXO1a and AMPK." Thesis, University of Dundee, 2012. https://discovery.dundee.ac.uk/en/studentTheses/b6abc6a4-de82-430a-ad30-015e8f69c8e8.

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In recent years there has been a considerable increase in human obesity levels with an associated increase in the incidence of type 2 diabetes mellitus (T2DM), a disorder of glucose metabolism characterised by insulin resistance. It is unclear why obesity and insulin resistance should frequently exist comorbidly but the current prevalence of polypharmacy suggests the underlying mechanisms may be multifactorial in origin. This project has highlighted plant compounds and extracts with: Insulin-like properties in cell culture experiments in that they induce phosphorylation and therefore inactivat
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To, Truc. "AMPK Promotes Xenophagy Through ‘Priming’ of Autophagic Kinases upon Detection of Salmonella Outer Membrane Vesicles." Thesis, Université d'Ottawa / University of Ottawa, 2019. http://hdl.handle.net/10393/38764.

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The autophagy pathway is an essential component of the innate immune response, capable of rapidly targeting intracellular bacteria, which are subsequently degraded by lysosomal enzymes. Recent work has begun to elucidate the regulatory signalling for autophagy induction in response to pathogenic bacteria. However, the initial signalling regulating autophagy induction in response to the detection of pathogens remains largely unclear. Here we report that AMPK, an important upstream activator of the autophagy pathway, is rapidly stimulated upon detection of pathogenic bacteria, prior to bacteria
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Green, Charlotte. "Insulin sensitivity and fatty acid induced lipotoxicity : The role of PKB and AMPK signalling." Thesis, University of Dundee, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.510657.

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Книги з теми "AMPK signalling"

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AMP-Activated Protein Kinase Signalling. MDPI, 2019. http://dx.doi.org/10.3390/books978-3-03897-663-9.

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Частини книг з теми "AMPK signalling"

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Evans, A. Mark, Sophronia A. Lewis, Oluseye A. Ogunbayo, and Javier Moral-Sanz. "Modulation of the LKB1-AMPK Signalling Pathway Underpins Hypoxic Pulmonary Vasoconstriction and Pulmonary Hypertension." In Advances in Experimental Medicine and Biology. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-18440-1_11.

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2

Evans, A. Mark, Chris Peers, Christopher N. Wyatt, Prem Kumar, and D. Grahame Hardie. "Ion Channel Regulation by the LKB1-AMPK Signalling Pathway: The Key to Carotid Body Activation by Hypoxia and Metabolic Homeostasis at the Whole Body Level." In Advances in Experimental Medicine and Biology. Springer Netherlands, 2012. http://dx.doi.org/10.1007/978-94-007-4584-1_11.

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3

Menegollo, Michela, Isabella Tessari, Luigi Bubacco, and Gyorgy Szabadkai. "Determination of ATP, ADP, and AMP Levels by Reversed-Phase High-Performance Liquid Chromatography in Cultured Cells." In Calcium Signalling. Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9018-4_19.

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Duarte, Carlos B., Ana L. Carvalho, and Arsélio P. Carvalho. "Modulation of the Ampa/Kainate Receptors by Protein Kinase C." In Signalling Mechanisms — from Transcription Factors to Oxidative Stress. Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-79675-3_11.

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Evans, A. Mark, D. Grahame Hardie, Antony Galione, Chris Peers, Prem Kumar, and Christopher N. Wyatt. "AMP-Activated Protein Kinase Couples Mitochondrial Inhibition by Hypoxia to Cell-Specific Ca2+ Signalling Mechanisms in Oxygensensing Cells." In Signalling Pathways in Acute Oxygen Sensing. John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/9780470035009.ch18.

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Rasmussen, Howard, Carlos Isales, Shridar Ganesan, Roberto Calle, and Walter Zawalich. "Ca2+-Cyclic AMP Interactions in Sustained Cellular Responses." In Ciba Foundation Symposium 164 - Interactions Among Cell Signalling Systems. John Wiley & Sons, Ltd., 2007. http://dx.doi.org/10.1002/9780470514207.ch7.

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Grassie, Morag A., and Graeme Milligan. "The Use of Stably Transfected Cell Lines in the Analysis of Functional Interactions between Goα1 & Giα2 and the α2C10 Adrenoceptor." In Signalling Mechanisms — from Transcription Factors to Oxidative Stress. Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-79675-3_10.

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Piazza, Francesco, and Gianpietro Semenzato. "Signalling Molecules as Selective Targets for Therapeutic Strategies in Multiple Myeloma." In Cell Signaling & Molecular Targets in Cancer. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4614-0730-0_5.

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Rutherford, Claire, and Timothy M. Palmer. "Molecular Basis of Protective Anti-Inflammatory Signalling by Cyclic AMP in the Vascular Endothelium." In Systems Biology for Signaling Networks. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-5797-9_23.

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Santarelli, Enrico. "Asset Specificity, R&D Financing and the Signalling Properties of the Firm’s Finance Structure." In Finance and Technological Change. Palgrave Macmillan UK, 1995. http://dx.doi.org/10.1057/9780230375031_4.

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Тези доповідей конференцій з теми "AMPK signalling"

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Ahmad, Saad, Mariam Saeed, Juan M. Guerrero, et al. "DC Link Control for Trains with Onboard Energy Storage using Voltage Level Signalling." In 2024 Energy Conversion Congress & Expo Europe (ECCE Europe). IEEE, 2024. http://dx.doi.org/10.1109/ecceeurope62508.2024.10752013.

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Chang, Jae Won, Seung-Nam Jung, Lihua Liua та ін. "Abstract 843: CLDN1 mediates tumor invasion in HNSCC by regulation of EMT through AMPK-TGF-β signalling". У Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-843.

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Celestini, V., T. Tezil, L. Russo, et al. "PO-161 The AMPK and MEK/ERK signalling pathways regulate mitochondrial FOXO3A import through phosphorylation of serine 12 and serine 30." In Abstracts of the 25th Biennial Congress of the European Association for Cancer Research, Amsterdam, The Netherlands, 30 June – 3 July 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/esmoopen-2018-eacr25.683.

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Marriot, D. "Installation & testing of the signalling system." In 11th IET Professional Development Course on Railway Signalling and Control Systems. Institution of Engineering and Technology, 2006. http://dx.doi.org/10.1049/ic.2006.0695.

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Nicholson, T. J. "Train equipment & interfaces." In IET Professional Development Course on Railway Signalling and Control Systems (RSCS 2010). IET, 2010. http://dx.doi.org/10.1049/ic.2010.0097.

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Nicholson, T. J. "Train equipment & interfaces." In IET Professional Development Course on Railway Signalling and Control Systems (RSCS 2012). IET, 2012. http://dx.doi.org/10.1049/ic.2012.0053.

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Clark, G. E. "Supervision & operation of mass transit systems." In 11th IET Professional Development Course on Railway Signalling and Control Systems. Institution of Engineering and Technology, 2006. http://dx.doi.org/10.1049/ic.2006.0691.

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Nicholson, T. J. "Total automation: impacts & systems." In IET Professional Development Course on Railway Signalling and Control Systems (RSCS 2010). IET, 2010. http://dx.doi.org/10.1049/ic.2010.0105.

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Nicholson, T. J. "Total automation: impacts & systems." In IET Professional Development Course on Railway Signalling and Control Systems (RSCS 2012). IET, 2012. http://dx.doi.org/10.1049/ic.2012.0058.

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McKendrick, B. "Asset management standards & practice." In IET Professional Development Course on Railway Signalling and Control Systems (RSCS 2010). IET, 2010. http://dx.doi.org/10.1049/ic.2010.0087.

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Звіти організацій з теми "AMPK signalling"

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Luo, Fang, han-han Chen, Jun-Jie Feng, et al. Effect of acupuncture on insulin signalling pathway and mitochondrial AMPK pathway in animal model of type 2 diabetes mellitus:A systematic evaluation and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.11.0086.

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Yuval, Boaz, and Todd E. Shelly. Lek Behavior of Mediterranean Fruit Flies: An Experimental Analysis. United States Department of Agriculture, 2000. http://dx.doi.org/10.32747/2000.7575272.bard.

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The Mediterranean fruit fly, Ceratitis capitata (Diptera: Tephritidae), is a ubiquitous pest of fruit trees, causing significant economic damage both in the U.S. and in Israel. Control efforts in the future will rely heavily on the sterile insect technique (SIT). Success of such operations hinges on the competitive ability of released males. The mating system of the medfly is based on leks. These are aggregations of sexually signaling males that attract females (who then select and copulate a courting male). A major component of male competitiveness is their ability to join existing leks or es
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