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Готові списки джерел за темами / And persistent inflammation

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Добірка наукової літератури з теми "And persistent inflammation"

Автор: Grafiati

Опубліковано: 7 червня 2025

Оновлено: 2 серпня 2025

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Статті в журналах з теми "And persistent inflammation"

1

Gentile, Lori F., Alex G. Cuenca, Philip A. Efron, et al. "Persistent inflammation and immunosuppression." Journal of Trauma and Acute Care Surgery 72, no. 6 (2012): 1491–501. http://dx.doi.org/10.1097/ta.0b013e318256e000.

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2

Tsirpanlis, George, Pantelis Bagos, Dimitris Ioannou, et al. "Exploring Inflammation in Hemodialysis Patients: Persistent and Superimposed Inflammation." Kidney and Blood Pressure Research 27, no. 2 (2004): 63–70. http://dx.doi.org/10.1159/000075809.

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3

Babaoglu, Hakan, Berkan Armagan, Erdal Bodakci, et al. "Predictors of persistent inflammation in familial Mediterranean fever and association with damage." Rheumatology 60, no. 1 (2020): 333–39. http://dx.doi.org/10.1093/rheumatology/keaa378.

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Abstract Objective Persistent inflammation is an insidious and less studied feature of FMF. We investigated clinical determinants of persistent inflammation and its associations with individual damage items. Methods This is a cross-sectional analysis of 917 FMF patients, who fulfilled the Tel Hashomer criteria and had at least 6 months’ follow-up. Patients were stratified based on whether they had persistent inflammation. We used logistic regression analysis to investigate independent predictors of persistent inflammation and the associated individual damage items. Results One hundred and fort

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4

Arias de la Rosa, I., M. L. Ladehesa Pineda, C. López-Medina, et al. "POS0432 CHARACTERIZATION OF THE CARDIOVASCULAR DISEASE PROTEOMIC PROFILE IN SPONDYLOARTHRITIS PATIENTS: POTENTIAL BIOMARKERS FOR PERSISTENT INFLAMMATION." Annals of the Rheumatic Diseases 82, Suppl 1 (2023): 472.1–472. http://dx.doi.org/10.1136/annrheumdis-2023-eular.2573.

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Backgroundpatients with Spondyloarthritis (SpA) have an increased risk of cardiovascular disease. Taking into account the strong relationship between inflammation and CVD, there is an urgent need to identify different molecular drivers of CVD signs and their association with inflammation.Objectivesto investigate the alteration of CVD-related proteins in the plasma of SpA patients, their association with clinical features, and to evaluate their potential role as biomarkers for the identification of persistent inflammation.Methodsa cross-sectional study including 120 patients with SpA and 30 age

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5

Barrado, Julia, and Vicente Estrada. "Inflammation in HIV: a persistent challenge." Enfermedades infecciosas y microbiologia clinica (English ed.) 42, no. 9 (2024): 467. http://dx.doi.org/10.1016/j.eimce.2024.09.007.

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Barrado, Julia, and Vicente Estrada. "Inflammation in HIV: A persistent challenge." Enfermedades Infecciosas y Microbiología Clínica 42, no. 9 (2024): 467. http://dx.doi.org/10.1016/j.eimc.2024.09.007.

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7

Nomellini, Vanessa, Lewis J. Kaplan, Carrie A. Sims, and Charles C. Caldwell. "Chronic Critical Illness and Persistent Inflammation." SHOCK 49, no. 1 (2018): 4–14. http://dx.doi.org/10.1097/shk.0000000000000939.

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8

Canonica, G., and Ilaria Baiardini. "Minimal Persistent Inflammation in Allergic Rhinitis." Allergy & Clinical Immunology International - Journal of the World Allergy Organization 18, no. 04 (2006): 169–71. http://dx.doi.org/10.1027/0838-1925.18.4.169.

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9

JATAKANON, ANON, CARINA UASUF, WAZIM MAZIAK, SAM LIM, KIAN FAN CHUNG, and PETER J BARNES. "Neutrophilic Inflammation in Severe Persistent Asthma." American Journal of Respiratory and Critical Care Medicine 160, no. 5 (1999): 1532–39. http://dx.doi.org/10.1164/ajrccm.160.5.9806170.

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10

Yoshida, Masaki, Shunji Itoh, Ikuji Hatamura, and Nobuo Nagai. "Persistent inflammation in photo-aged skin." Journal of Dermatological Science 86, no. 2 (2017): e81-e82. http://dx.doi.org/10.1016/j.jdermsci.2017.02.239.

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Дисертації з теми "And persistent inflammation"

1

Inglis, Julia Jane. "Characterisation of novel nociceptive mechanisms during persistent inflammation." Thesis, Queen Mary, University of London, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.413580.

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Lajunen, T. (Taina). "Persistent Chlamydia pneumoniae infection, inflammation and innate immunity." Doctoral thesis, University of Oulu, 2008. http://urn.fi/urn:isbn:9789514289965.

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Abstract Chlamydia pneumoniae is an obligatory intracellular pathogen that causes upper and lower respiratory tract infections. Like other Chlamydial species, also C. pneumoniae has a tendency to cause persistent infections, which have been associated with different cardiovascular, neurological, and respiratory diseases. In addition, a few studies have reported an association between C. pneumoniae seropositivity and an elevated body mass index (BMI), and it has been shown that C. pneumoniae is capable of infecting preadipocytes and adipocytes. The main aims of this study were to study if certa

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3

Khurana, Shruti. "Lung physiology & airway inflammation in COPD patients with persistent sputum production." Thesis, University of Manchester, 2013. https://www.research.manchester.ac.uk/portal/en/theses/lung-physiology-and-airway-inflammation-incopd-patients-with-persistent-sputum-production(b0f3374e-9a67-4ff9-a51e-d94eefa2f13c).html.

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Background: The clinical and pathological presentation of COPD is heterogeneous. ‘Chronic bronchitis’ is a phenotype of COPD, which is a clinical diagnosis of a productive cough of ≥ 3 months for ≥ 2 consecutive years. Chronic bronchitis is associated with worse lung function, frequent exacerbations, recurrent hospitalisations and premature death in patients with COPD. Chronic bronchitis sufferers can be further subphenotyped into those who produce sputum during exacerbation or during winter months only and those who are ‘persistent sputum producers,’ who experience mucous hypersecretion throu

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4

Gao, Helen Guoyi Li. "INCREASED FIBROGENIC PROTEINS FOLLOWING PERSISTENT LOW-GRADE INFLAMMATION IN A RAT MODEL OF LONG-TERM OVERUSE." Master's thesis, Temple University Libraries, 2013. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/238810.

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Biomedical Sciences<br>M.S.<br>We examined the relationship between grip strength declines and muscle-tendon responses induced by long-term performance of a high-repetition, low-force (HRLF) reaching task in rats. We hypothesized that grip strength declines would correlate with inflammation, fibrosis and degradation in flexor digitorum muscles and tendons. Grip strength declined after training, and further in weeks 18 and 24, in reach limbs of HRLF rats. Flexor digitorum tissues of reach limbs showed low-grade increases in inflammatory cytokines: IL-1beta after training and in week 18, IL-1alp

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5

Glaros, Trevor Griffiths. "Molecular Mechanisms Governing Persistent Induction of Pro-Inflammatory Genes by Lipopolysaccharide." Diss., Virginia Tech, 2011. http://hdl.handle.net/10919/73001.

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Low dose endotoxemia is caused by several health conditions including smoking, alcohol abuse, high fat diets, and aging. Several studies have correlated low dose endotoxemia with increased risks of atherosclerosis, diabetes, and Parkinson's disease. Unlike high doses of endotoxin which induce a strong but transient induction of pro-inflammatory mediators, low doses of endotoxin result in a mild but chronic induction of pro-inflammatory genes. The central hypothesis of our study was that if low doses of endotoxin are capable of inducing mild prolonged inflammation, then a unique signaling ci

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6

Roberts, Jill Marie. "The Role of Glial Activation in Descending Facilitation from the Rostroventromedial Medulla (RVM) in Models of Persistent Pain." Diss., The University of Arizona, 2009. http://hdl.handle.net/10150/194471.

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Substantial evidence shows that activation of glial cells in the spinal cord may promote central sensitization and enhancement of pain. Descending facilitation from the rostroventromedial medulla (RVM) is also recognized as a critical component in the maintenance of chronic pain states, although the precise mechanisms driving this activity are unclear. Here, we investigated the possibility that glial activation in the RVM could promote descending facilitation from the RVM in states of enhanced pain. Peripheral inflammation was induced with carrageenan injected into the plantar aspect of the hi

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Mahdavi, Jafar. "Helicobacter pylori adhesion and patho-adaptation : the role of BabA and SabA adhesins in persistent infection and chronic inflammation." Doctoral thesis, Umeå : Univ, 2004. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-271.

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Pitcher, Mark H. "The effects of persistent peripheral inflammation on the ultrastructural localization of spinal cord dorsal horn group I metabotropic glutamate receptors /." Thesis, McGill University, 2005. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=98770.

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Persistent peripheral inflammation is thought to induce functional plasticity of spinal dorsal horn neurons, and may produce changes in glutamate receptor expression. Alterations in expression and cellular localization of group I metabotropic glutamate receptors (mGluR1alpha and mGIuR5) is important in various neuronal systems, and these receptors are also known to modulate nociceptive neurotransmission in the spinal dorsal horn. The aim of the present study is to determine whether persistent inflammation produces alterations in ultrastructural localization of mGluR1alpha and mGluR5 in the dor

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Myre, Maxine. "Exploring the Independent and Combined Effects of Persistent Organic Pollutants and Hypoxia on Human Adipocyte Functions." Thèse, Université d'Ottawa / University of Ottawa, 2014. http://hdl.handle.net/10393/30416.

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Persistent organic pollutants (POPs) and adipose tissue hypoxia have been shown to independently affect adipocyte functions. The goals of this study were to (1) determine the effect of PCB-77, PCB-153, and DDE on the differentiation of human preadipocytes, and (2) investigate the cross-talk between PCB-77 and hypoxia in differentiated human adipocytes. First, human preadipocytes were exposed to PCB-77, PCB-153, or DDE during the entire 14-day differentiation period. We found no effect of low POP levels on lipid accumulation. Second, differentiated human adipocytes were exposed to a combination

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Gascon, Merlos Mireia 1984. "Persistent organic pollutants, bisphenol A, phthalates and respiratory and immune health in childhood." Doctoral thesis, Universitat Pompeu Fabra, 2014. http://hdl.handle.net/10803/145922.

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Persistent organic pollutants (POPs), bisphenol A (BPA) and pthalates may increase the risk of respiratory infections and allergic diseases in infants and the effects might last until, at least, adolescence. Cytokines and biomarkers of inflammation can provide information of the mechanisms behind such associations. Data from the “Infancia y Medioambiente” (INMA) population-based birth cohort project and from six other existing European birth cohort studies have been used in the present thesis, which also includes a systematic review. Results of the present work, which includes five scientific

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Книги з теми "And persistent inflammation"

1

Hoenigl, Martin, Sara Gianella, and Harald H. Kessler, eds. HIV-Associated Immune Activation and Persistent Inflammation. Frontiers Media SA, 2020. http://dx.doi.org/10.3389/978-2-88963-438-5.

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Carrero, Juan Jesús, and Peter Stenvinkel. The role of inflammation in chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0110.

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Low-grade persistent inflammation is a common feature of chronic kidney disease. This chapter provides an overview of the pathogenesis and clinical consequences of elevated pro-inflammatory cytokines in the uraemic milieu with an emphasis on dialysis stages. It reviews the multifactorial dialysis- and non-dialysis-related causes of inflammation and its purported role in the development of protein energy wasting, vascular calcification, endocrine disorders, and depression. The chapter also discusses the use and the need of monitoring C-reactive protein levels regularly in the clinical setting a

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3

Virna, Jessica. Anti Inflammatory Diet: What the Healthcare Industry Doesn’t Want You to Know! Cure Autoimmune Diseases and Persistent Inflammation for Life ... Arthritis, Heart Diseases, Joint Pain,). CreateSpace Independent Publishing Platform, 2015.

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4

Annane, Djillali, and B. Jérôme Aboab. Management of carbon monoxide poisoning. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0328.

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CO poisoning is the commonest cause of toxic death. Carbon monoxide is colourless, odourless, and tasteless, and is produced under various conditions. When people inhale CO, the gas diffuses rapidly to the body and replaces oxygen at the level of haemoglobin, myoglobin, and other oxygen carriers. Subsequently, CO causes oxygen deprivation of all body tissues. CO also induces oxidative stress and systemic inflammation. After CO poisoning a broad variety of symptoms may occur. Survivors of CO poisoning often present with persistent neurological sequels or develop delayed neurological symptoms. T

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5

Rosenblatt, Elizabeth. Peritonitis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0032.

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Peritonitis, or inflammation of the serosal membranes lining the abdominal cavity, is used predominately to describe primary peritonitis (spontaneous bacterial peritonitis [SBP]) and secondary peritonitis—two conditions with distinct pathophysiologies that require different diagnostic and therapeutic approaches. Tertiary peritonitis is characterized by persistent symptoms or signs of infection despite appropriate treatment of primary or secondary peritonitis. Patients undergoing peritoneal dialysis are at risk for catheter-associated peritonitis, which is sometimes considered an additional cat

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6

Mason, Peggy. Somatosensation. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190237493.003.0017.

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Under normal circumstances, the somatosensory system contributes more to shaping movements than to perception. Yet damage to the somatosensory system can result in spontaneous pain and other abnormal somatic perceptions. An exploration of the mechanisms and pathways involved in touch perception is slanted toward understanding the contribution of the dorsal column–medial lemniscus pathway to the generation of paresthesia and dysesthesia. Peripheral somatosensory afferents that contribute to the perception of sharp or aching pain, temperature, and itch are described. The properties of transient

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Candido, Kenneth D., Tatiana Tverdohleb, and Nebojsa Nick Knezevic. Postlaminectomy Syndrome. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190271787.003.0024.

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Postlaminectomy syndrome is persistent or recurrent back pain after otherwise anatomically successful lumbar spine surgery. A dramatic increase in the number of low back surgeries has been observed since 1997, with an increased incidence of pain after low back surgery in the range of 5% to 74.6%. The mechanisms contributing to back pain are muscle damage during surgery, muscle spasm, and inflammation, with subsequent development of myofascial pain syndrome as well as other typical and atypical back pain generators. Diagnosis is based primarily on history and physical examination, as well as re

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8

Ferguson, Robert, and Karen Gillock. Memory and Attention Adaptation Training. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780197521526.001.0001.

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Memory and Attention Adaptation Training (MAAT) is a cognitive-behavioral therapy (CBT) designed to help cancer survivors self-manage and mitigate the late and long-term effects of cancer and cancer therapy on memory function. Cancer-related cognitive impairment (CRCI) is a set of mild to moderate memory and attention impairments that can have an adverse influence on quality of life. CRCI symptoms tend to present during active treatment, but for some individuals cognitive changes can persist for years. While the exact prevalence of CRCI is unknown, review of the literature estimates that nearl

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9

Deighton, Chris. Rheumatoid arthritis—management. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0112.

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Influential guidelines on rheumatoid arthritis (RA) management agree on most key recommendations. Early diagnosis of persistent synovitis, and identification of poor prognostic markers, is essential. Rapid intervention is vital with drugs to suppress inflammation, slow down damaging disease components, and prevent disability. The label of RA covers a broad spectrum of disease severity, and there is controversy on: • whether the same interventions are needed for all patients • whether monotherapy or combination treatment is appropriate • the role of steroids in RA • the appropriate introduction

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10

Dalbeth, Nicola. Gout. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0141.

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Gout is a common and treatable disorder of purine metabolism. Gout typically presents as recurrent self-limiting episodes of severe inflammatory arthritis affecting the foot. In the presence of persistent hyperuricaemia, tophi, chronic synovitis, and joint damage may develop. Diagnosis of gout is confirmed by identification of monosodium urate (MSU) crystals using polarizing light microscopy. Hyperuricaemia is the central biochemical cause of gout. Genetic variants in certain renal tubular urate transporters including SLC2A9 and ABCG2, and dietary factors including intake of high-purine meats

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Частини книг з теми "And persistent inflammation"

1

Osher, Robert H. "Persistent Postoperative Inflammation." In What I Say. CRC Press, 2024. http://dx.doi.org/10.1201/9781003526926-63.

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2

Efron, P. A., F. A. Moore, and S. C. Brakenridge. "Persistent Inflammation, Immunosuppression and Catabolism after Severe Injury or Infection." In Annual Update in Intensive Care and Emergency Medicine 2018. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-73670-9_3.

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3

Ogawa, Rei. "Ideal Wound Closure Methods for Minimizing Scarring After Surgery." In Textbook on Scar Management. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-44766-3_21.

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AbstractWound-healing phenomena are the result of a cascade of complex biochemical events that can be categorized into four general overlapping phases: coagulation, inflammation, proliferation, and remodeling. Significantly, all four phases of wound healing are influenced by both intrinsic and extrinsic mechanical forces. These mechanical forces provoke chronic inflammation of the dermis, namely, the unceasing influx and activation of inflammatory cells, the persistent generation of blood vessels and nerve fibers, and the constant production of collagen by the activated fibroblasts. This chron

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4

Li, Zhi Jian, and John J. Huang. "How Do I Evaluate and Treat a Patient With Persistent Inflammation Following Intraocular Surgery?" In Curbside Consultation in Uveitis. CRC Press, 2024. http://dx.doi.org/10.1201/9781003523710-45.

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5

Maltsev, Dmytro. "Results of valacyclovir, valganciclovir, artesunate for the treatment of reactivated EBV-, HHV-6-, HHV-7-infections in children with autism spectrum disorders associated with genetic deficiency of the folate cycle." In IMMUNODIAGNOSTICS AND IMMUNOTHERAPY OF NEUROPSYCHIATRIC DISORDERS IN CHILDREN. TECHNOLOGY CENTER PC, 2025. https://doi.org/10.15587/978-617-8360-21-4.ch8.

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Autism spectrum disorders (ASD), which currently affect at least 1 % of the modern child population and continue to increase in prevalence, are a global problem that requires urgent attention. However, the FDA has not yet approved any drug to modify the course of ASD in children or to treat mental illness. The results of 5 recent meta-analyses and systematic reviews of randomized controlled trials demonstrate the association of ASD with genetic deficiencies of folic acid cycle enzymes (GDFC), which sheds light on the pathogenetic pathways of the formation of a state of transmethylation disorde

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6

Maltsev, Dmytro. "Neuroradiological signs of encephalopathy in children with autism spectrum disorders associated with genetic deficiency of the folate cycle." In IMMUNODIAGNOSTICS AND IMMUNOTHERAPY OF NEUROPSYCHIATRIC DISORDERS IN CHILDREN. TECHNOLOGY CENTER PC, 2025. https://doi.org/10.15587/978-617-8360-21-4.ch7.

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One of the important advances in psychiatry and neurology in recent years is the elucidation of the association between genetic deficiency of the folate cycle (GDFC) and autism spectrum disorders (ASD) in children. The evidence for such an association is based on the results of at least five meta-analyses of randomized controlled clinical trials and a number of additional randomized controlled trials, the results of which are still not systematized. It has been shown that GDFC leads to the development of a number of typical biochemical disorders, which cause a state of a special form of immuno

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Maltsev, Dmytro. "Results of the search for laboratory signs of autoimmune reactions to brain and extracerebral autoantigens in children with autism spectrum disorders associated with genetic deficiency of the folate cycle." In IMMUNODIAGNOSTICS AND IMMUNOTHERAPY OF NEUROPSYCHIATRIC DISORDERS IN CHILDREN. TECHNOLOGY CENTER PC, 2025. https://doi.org/10.15587/978-617-8360-21-4.ch5.

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Genetic deficiency of the folate cycle (GDFC) is an important associated factor in autism spectrum disorders (ASD) in children, as evidenced by the accumulated evidence base from meta-analyses of randomized controlled trials. Biochemical abnormalities caused by GDFC have been shown to lead to immune system damage with induction of immunodeficiency and associated immune dysregulation. Data from a systematic review by H. K. Hughes et al. clearly outline a range of representative pathological changes in the immune status in children with ASD, in particular, a pronounced cytokine imbalance with a

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Maltsev, Dmytro. "Efficacy of rituximab in autism spectrum disorders associated with genetic deficiency of the folate cycle with signs of antineuronal autoimmunity." In IMMUNODIAGNOSTICS AND IMMUNOTHERAPY OF NEUROPSYCHIATRIC DISORDERS IN CHILDREN. TECHNOLOGY CENTER PC, 2025. https://doi.org/10.15587/978-617-8360-21-4.ch12.

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Advances in genetics, molecular biology, and immunology over the past decades have significantly changed our understanding of the etiology and pathogenesis of autism spectrum disorders (ASD) in children. One of the key advances in this direction is the elucidation of the association of genetic deficiency of the folate cycle (GDFC) with ASD, evidence for which is based on the results of at least 5 meta-analyses of randomized controlled clinical trials and a number of additional controlled trials, the data of which have not yet been properly summarized. It has been established that GDFC leads to

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Kato, Akane, and Masayuki Hanaoka. "Pathogenesis of COPD (Persistence of Airway Inflammation): Why Does Airway Inflammation Persist After Cessation of Smoking?" In Respiratory Disease Series: Diagnostic Tools and Disease Managements. Springer Singapore, 2016. http://dx.doi.org/10.1007/978-981-10-0839-9_4.

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Spickett, Gavin. "Theories of causation." In ME/CFS and Long Covid. Oxford University PressOxford, 2025. https://doi.org/10.1093/med/9780198962137.003.0005.

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Abstract Chapter 5 covers the main theories for the development of fatigue, including, genetic, immunological, infective, autonomic, endocrine, mitochondrial, clotting, gut microbiome and virome, psychiatric, and psychological. It also provides a tentative synthesis of current thinking, focused on possible viral persistence in the brain and consequent chronic neuroinflammation. Many research studies into ME/CFS and Long Covid are hampered by insufficient numbers, lack of appropriate control groups, and inappropriate patient groups (longstanding and new patients combined). The exact pathways ca

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Тези доповідей конференцій з теми "And persistent inflammation"

1

Khoshnevis, Sepideh, and Kenneth R. Diller. "Cryosurgery Causes Profound and Persistent Ischemia." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80635.

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Cryotherapy has been used in the treatment of soft tissue trauma and other ailments since the time of Hippocrates. Currently it is commonly applied in conjunction with surgical procedures and by athletic trainers and physical therapists to control pain, swelling, bleeding, and inflammation. Localized cooling also results in slowing of the nerve conduction velocity and reduced muscle spasm and secondary hypoxic injury1,2. Cooling is commonly used for soft tissue injuries in combination with rest, compression and elevation to minimize inflammation.

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2

Kumar, A. J., X. Q. Wang, S. Seelye, et al. "Persistent Liver Dysfunction and Inflammation in Sepsis Survivors with Obesity." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a1481.

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3

Allinne, Jeanne, Wei Keat Lim, George Scott, et al. "IL-33 blockade prevents exacerbations in a model of chronic airway inflammation by blunting persistent inflammation and remodeling." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa3876.

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4

Velten, Markus, Kirk R. Hutchinson, Pamela Lucchesi, and Lynette K. Rogers. "Persistent Ventricular Dysfunction In Mice Exposed To Systemic Maternal Inflammation And Neonatal Hyperoxia." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a1856.

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5

Satoh, Hideyuki, Akiko Ochiai, Gaku Ikegami, Naoto Fueki, and Hironori Sagara. "Effects Of Switching Treatment In Patients Who Have Asthma With Persistent Airway Inflammation." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a1283.

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6

Klück, V., T. Crisan, M. Cleophas, et al. "SAT0045 Dna methylation of socs3 as a possible mechanism for persistent urate induced inflammation." In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.6281.

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7

Velten, Markus, Kathryn M. Heyob, Lynette K. Rogers, and Stephen E. Welty. "Systemic Maternal Inflammation And Neonatal Hyperoxia Induces Persistent Alterations In Pulmonary Structure And Function." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a3897.

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8

Guler, S. A., A. Sarbu, O. Stadler, et al. "Persistent inflammation in systemic sclerosis is strongly associated with mortality: a EUSTAR database analysis." In ERS International Congress 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/13993003.congress-2022.1912.

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9

Essayas, H., J. Kim, J. Mcgovern, et al. "Toll-like Receptor-9 Activation Promotes Persistent Inflammation in the Lung During Influenza Infection." In American Thoracic Society 2023 International Conference, May 19-24, 2023 - Washington, DC. American Thoracic Society, 2023. http://dx.doi.org/10.1164/ajrccm-conference.2023.207.1_meetingabstracts.a5608.

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10

Babaoglu, Hakan, Berkan Armagan, Erdal Bodakci, et al. "THU0562 PREVALENCE, UNDERLYING FACTORS AND DAMAGE ASSOCIATED WITH CHRONIC PERSISTENT INFLAMMATION IN PATIENTS WITH FMF." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.4766.

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Звіти організацій з теми "And persistent inflammation"

1

Thaxton, Jessica E. Chemotherapy Necessitates Increased Immune Control of HHVs: A Cause of Persistent Inflammation Enabling Protracted Fatigue in Breast Cancer Survivors. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada591462.

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2

Thaxton, Jessica E. Chemotherapy Necessitates Increased Immune Control of HHVs: A Cause of Persistent Inflammation Enabling Protracted Fatigue in Breast Cancer Survivors. Defense Technical Information Center, 2015. http://dx.doi.org/10.21236/ada621832.

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3

Bhowmik, Srishti, Anurag Mishra, Chandra Bhushan Singh, Sarmista Roy, and Vibha Singh. Malignant Transformation of Post-Nephrectomy Enterocutaneous Fistula: A Review of Literature. Science Repository, 2024. http://dx.doi.org/10.31487/j.ajscr.2024.02.01.

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Background: Enterocutaneous fistula is an aberrant epithelialized communication between the bowel and the skin occurring mostly as a complication of abdominal surgery. Enterocutaneous fistula may present as chronically discharging sinus and like any other chronic non healing wound, malignant transformation has rarely been reported in such gastrointestinal fistula. Herein we report an additional case of malignant transformation of enterocutaneous fistula leading to duodenal mass. Case Presentation: A 50-year diabetic gentleman presented with a persistent draining sinus from his surgical site of

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4

Czerwaty, Katarzyna, Karolina Dżaman, Krystyna Maria Sobczyk, and Katarzyna Irmina Sikrorska. The Overlap Syndrome of Obstructive Sleep Apnea and Chronic Obstructive Pulmonary Disease: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.11.0077.

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Review question / Objective: To provide the essential findings in the field of overlap syndrome of chronic obstructive pulmonary disease and obstructive sleep apnea, including prevalence, possible predictors, association with clinical outcomes, and severity compared to both chronic obstructive pulmonary disease and obstructive sleep apnea patients. Condition being studied: OSA is characterized by complete cessation (apnea) or significant decrease (hy-popnea) in airflow during sleep and recurrent episodes of upper airway collapse cause it during sleep leading to nocturnal oxyhemoglobin desatura

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5

Persisting Cryptococcus yeast species Vishniacozyma victoriae and Cryptococcus neoformans elicit unique airway inflammation in mice following repeated exposure (dataset). U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, 2023. http://dx.doi.org/10.26616/nioshrd-1061-2023-0.

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