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Готові списки джерел за темами / Calcification, Physiologic / Статті в журналах

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Статті в журналах з теми "Calcification, Physiologic"

Автор: Grafiati

Опубліковано: 4 червня 2021

Оновлено: 3 лютого 2022

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1

Rutsch, Frank, and Robert Terkeltaub. "Deficiencies of physiologic calcification inhibitors and low-grade inflammation in arterial calcification: lessons for cartilage calcification." Joint Bone Spine 72, no. 2 (March 2005): 110–18. http://dx.doi.org/10.1016/j.jbspin.2004.05.014.

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2

Herrmann, Jaqueline, Milen Babic, Markus Tölle, Markus van der Giet, and Mirjam Schuchardt. "Research Models for Studying Vascular Calcification." International Journal of Molecular Sciences 21, no. 6 (March 23, 2020): 2204. http://dx.doi.org/10.3390/ijms21062204.

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Calcification of the vessel wall contributes to high cardiovascular morbidity and mortality. Vascular calcification (VC) is a systemic disease with multifaceted contributing and inhibiting factors in an actively regulated process. The exact underlying mechanisms are not fully elucidated and reliable treatment options are lacking. Due to the complex pathophysiology, various research models exist evaluating different aspects of VC. This review aims to give an overview of the cell and animal models used so far to study the molecular processes of VC. Here, in vitro cell culture models of different

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3

Scott, J. E., and M. Haigh. "Is dermatan sulfate proteoglycan the physiologic inhibitor of type I collagen calcification?" Bone 7, no. 2 (January 1986): 154. http://dx.doi.org/10.1016/8756-3282(86)90717-9.

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4

Doyle, Anthony James, and Graeme D. Anderson. "Physiologic Calcification of the Pineal Gland in Children on Computed Tomography: Prevalence, Observer Reliability and Association with Choroid Plexus Calcification." Academic Radiology 13, no. 7 (July 2006): 822–26. http://dx.doi.org/10.1016/j.acra.2006.04.004.

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5

Al-Zaghal, Abdullah, Siavash Mehdizadeh Seraj, Thomas J. Werner, Oke Gerke, Poul F. Høilund-Carlsen, and Abass Alavi. "Assessment of Physiologic Intracranial Calcification in Healthy Adults Using 18F-NaF PET/CT." Journal of Nuclear Medicine 60, no. 2 (July 12, 2018): 267–71. http://dx.doi.org/10.2967/jnumed.118.213678.

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6

Caliskan, Emine, and Mehmet Ozturk. "Evaluation of physiologic pineal gland calcification via computed tomography in the pediatric population." Annals of Medical Research 26, no. 10 (2019): 2391. http://dx.doi.org/10.5455/annalsmedres.2019.06.338.

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7

Nemes, Attila, and Tamás Forster. "Functional vascular alterations associated with aortic valve stenosis." Orvosi Hetilap 152, no. 25 (June 2011): 993–99. http://dx.doi.org/10.1556/oh.2011.29145.

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Degenerative changes, atherosclerotic process and calcification of valvular leaflets are mostly responsible for valvular aortic valve stenosis, but congenital bicuspid aortic valve and rheumatic fever in history are also known predisposing factors. Aortic valve stenosis is frequently associated with different functional vascular alterations. The aim of this review is to demonstrate these vascular alterations evaluated by non-invasive methods and underlying physiologic and pathophysiologic processes. Orv. Hetil., 2011, 152,993–999.

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8

Rezvova, M. A., E. A. Ovcharenko, T. V. Glushkova, Yu A. Kudryavtseva, and L. S. Barbarash. "Evaluation of calcification resistance of xenopericardium treated with polyhydroxy compounds." Russian Journal of Transplantology and Artificial Organs 23, no. 1 (April 10, 2021): 75–83. http://dx.doi.org/10.15825/1995-1191-2021-1-75-83.

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Calcification of biomaterials used in prosthetic heart valves has been a challenging issue in cardiovascular surgery. The objective of this work is to compare the efficiency of polyvinyl alcohol (PVA) and tannic acid (TA) modification of xenomaterials, pre-stabilized with glutaraldehyde (GA) and ethylene glycol diglycidyl ether (EGDE), in reducing calcification. Analysis of mechanical properties evaluated under uniaxial tension, showed a significant increase in the tensile strength of the test samples compared to the control (unmodified) samples (p < 0.05). Additional treatment of GA-fixed

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9

Fukai, Atsushi, Naohiro Kawamura, Taku Saito, Yasushi Oshima, Toshiyuki Ikeda, Fumitaka Kugimiya, Akiro Higashikawa, et al. "Akt1 in murine chondrocytes controls cartilage calcification during endochondral ossification under physiologic and pathologic conditions." Arthritis & Rheumatism 62, no. 3 (February 25, 2010): 826–36. http://dx.doi.org/10.1002/art.27296.

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10

Terkeltaub, Robert. "Physiologic and pathologic functions of the NPP nucleotide pyrophosphatase/phosphodiesterase family focusing on NPP1 in calcification." Purinergic Signalling 2, no. 2 (June 2006): 371–77. http://dx.doi.org/10.1007/s11302-005-5304-3.

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11

Pasch, Andreas, Willi Jahnen-Dechent, and Edward R. Smith. "Phosphate, Calcification in Blood, and Mineral Stress: The Physiologic Blood Mineral Buffering System and Its Association with Cardiovascular Risk." International Journal of Nephrology 2018 (September 2, 2018): 1–5. http://dx.doi.org/10.1155/2018/9182078.

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Phosphate is an important cardiovascular risk factor and lowering elevated blood phosphate concentrations is a main therapeutic target in kidney patients. Phosphate is subject to the blood mineral buffering system which controls the precipitation of calcium and phosphate. Calciprotein particles (CPP), self-assembling complexes of calcium phosphate and serum proteins, are the nanomorphological correlates of this system. CPP1 are spherical, 50-100 nm in diameter, and contain amorphous mineral. CPP2 are oblongated, 100-200nm in the long axis, and they contain a crystalline mineral core. The relat

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12

Som, Panchali, Rajarshi Roy, Sumit Datta, Asis Kumar Ghosal, Anubha Saha, and Subhajit Halder. "Physiological Intracranial Calcification in Eastern Indian Population-A CT Scan Study." National Journal of Clinical Anatomy 06, no. 01 (January 2017): 059–70. http://dx.doi.org/10.1055/s-0039-1700723.

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Abstract Background and Aims:Knowledge of physiological calcification in brain parenchyma is essential to avoid misinterpretation during radiological evaluation. The calcifications are commonly seen in pineal gland, falx cerebri, tentorium cerebelli and choroid plexus. Objective:To determine the incidence of physiological intracranial calcification and its relationship to age and sex in eastern Indian population. Materials and Method: A cross sectional descriptive study of CT scan brain was performed in age group between 20-80 yrs in eastern India. The study was conducted on 64 Slice MDCT PHIL

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13

Speelman, Lambert, Ajay Bohra, E. Marielle H. Bosboom, Geert Willem H. Schurink, Frans N. van de Vosse, Michel S. Makaroun, and David A. Vorp. "Effects of Wall Calcifications in Patient-Specific Wall Stress Analyses of Abdominal Aortic Aneurysms." Journal of Biomechanical Engineering 129, no. 1 (July 27, 2006): 105–9. http://dx.doi.org/10.1115/1.2401189.

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It is generally acknowledged that rupture of an abdominal aortic aneurysm (AAA) occurs when the stress acting on the wall over the cardiac cycle exceeds the strength of the wall. Peak wall stress computations appear to give a more accurate rupture risk assessment than AAA diameter, which is currently used for a diagnose. Despite the numerous studies utilizing patient-specific wall stress modeling of AAAs, none investigated the effect of wall calcifications on wall stress. The objective of this study was to evaluate the influence of calcifications on patient-specific finite element stress compu

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14

Scissons, Robert P., Abraham Ettaher, and Sophia Afridi. "Likelihood of Normal ABI Increases With Physiologic Testing Referrals From Rural Primary Care Physicians." Journal for Vascular Ultrasound 43, no. 3 (August 29, 2019): 123–27. http://dx.doi.org/10.1177/1544316719870070.

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Disparities in diagnostic capabilities have been noted between rural and urban health care facilities. We believe the clinical evaluation of peripheral arterial disease (PAD) by rural physicians may be similarly affected. Patients referred for arterial physiologic testing in an urban and rural regional health care network for a consecutive 7-month period were reviewed. Patients were classified into 3 groups based on referring physician specialty: (1) vascular surgeon or vascular medicine specialist (Vasc), (2) urban primary care physician (Urban), and (3) rural primary care physician (Rural).

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15

Olesen, Ping, Kirsten Nguyen, Lise Wogensen, Thomas Ledet, and Lars Melholt Rasmussen. "Calcification of human vascular smooth muscle cells: associations with osteoprotegerin expression and acceleration by high-dose insulin." American Journal of Physiology-Heart and Circulatory Physiology 292, no. 2 (February 2007): H1058—H1064. http://dx.doi.org/10.1152/ajpheart.00047.2006.

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Arterial medial calcifications occur often in diabetic individuals as part of the diabetic macroangiopathy. The pathogenesis is unknown, but the presence of calcifications predicts risk of cardiovascular events. We examined the effects of insulin on calcifying smooth muscle cells in vitro and measured the expression of the bone-related molecule osteoprotegerin (OPG). Human vascular smooth muscle cells (VSMCs) were grown from aorta from kidney donors. Induction of calcification was performed with β-glycerophosphate. The influence of insulin (200 μU/ml or 1,000 μU/ml) on calcification was judged

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16

Beattie, D., C. Xu, R. Vito, S. Glagov, and M. C. Whang. "Mechanical Analysis of Heterogeneous, Atherosclerotic Human Aorta." Journal of Biomechanical Engineering 120, no. 5 (October 1, 1998): 602–7. http://dx.doi.org/10.1115/1.2834750.

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An experimental technique was developed to determine the finite strain field in heterogeneous, diseased human aortic cross sections at physiologic pressures in vitro. Also, the distributions within the cross sections of four histologic features (disease-free zones, lipid accumulations, fibrous intimal tissue, and regions of calcification) were quantified using light microscopic morphometry. A model incorporating heterogeneous, plane stress finite elements coupled the experimental and histologic data. Tissue constituent mechanical properties were determined through an optimization strategy, and

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17

Friedland, Robert P., Jay S. Luxenberg, and Elisabeth Koss. "A Quantitative Study of Intracranial Calcification in Dementia of the Alzheimer Type." International Psychogeriatrics 2, no. 1 (March 1990): 37–43. http://dx.doi.org/10.1017/s104161029000028x.

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Abnormalities in calcium homeostasis have been reported in Alzheimer's disease (AD) and in the neurofibrillary tangle disorders of amyotrophic lateral sclerosis and parkinsonism-dementia occurring in the Pacific. In order to more fully evaluate calcium physiology in AD, we analyzed the size of pineal and choroid plexus calcifications, using X-ray computed tomography, in 23 patients with probable AD and 18 healthy age-matched control subjects. The area occupied by calcification was measured from hard copies of the data by two independent observers who were blind to the diagnosis. There were no

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18

Bazin, Dominique, Arnaud Dessombz, Christelle Nguyen, Hang Korng Ea, Frédéric Lioté, John Rehr, Christine Chappard, et al. "The status of strontium in biological apatites: an XANES/EXAFS investigation." Journal of Synchrotron Radiation 21, no. 1 (November 2, 2013): 136–42. http://dx.doi.org/10.1107/s1600577513023771.

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Osteoporosis represents a major public health problem through its association with fragility fractures. The public health burden of osteoporotic fractures will rise in future generations, due in part to an increase in life expectancy. Strontium-based drugs have been shown to increase bone mass in postmenopausal osteoporosis patients and to reduce fracture risk but the molecular mechanisms of the action of these Sr-based drugs are not totally elucidated. The local environment of Sr2+cations in biological apatites present in pathological and physiological calcifications in patients without such

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19

Bäck, Magnus, and Jean-Baptiste Michel. "From organic and inorganic phosphates to valvular and vascular calcifications." Cardiovascular Research 117, no. 9 (February 9, 2021): 2016–29. http://dx.doi.org/10.1093/cvr/cvab038.

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Abstract Calcification of the arterial wall and valves is an important part of the pathophysiological process of peripheral and coronary atherosclerosis, aortic stenosis, ageing, diabetes, and chronic kidney disease. This review aims to better understand how extracellular phosphates and their ability to be retained as calcium phosphates on the extracellular matrix initiate the mineralization process of arteries and valves. In this context, the physiological process of bone mineralization remains a human model for pathological soft tissue mineralization. Soluble (ionized) calcium precipitation

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20

Bazin, D., M. Daudon, Ch Chappard, J. J. Rehr, D. Thiaudière, and S. Reguer. "The status of strontium in biological apatites: an XANES investigation." Journal of Synchrotron Radiation 18, no. 6 (September 16, 2011): 912–18. http://dx.doi.org/10.1107/s0909049511032651.

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Osteoporosis represents a major public health problem and increases patient morbidity through its association with fragility fractures. Among the different treatments proposed, strontium-based drugs have been shown to increase bone mass in postmenopausal osteoporosis patients and to reduce fracture risk. While the localization of Sr2+cations in the bone matrix has been extensively studied, little is known regarding the status of Sr2+cations in natural biological apatite. In this investigation the local environment of Sr2+cations has been investigated through XANES (X-ray absorption near-edge s

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21

De Maré, Annelies, Stuart Maudsley, Abdelkrim Azmi, Jhana O. Hendrickx, Britt Opdebeeck, Ellen Neven, Patrick C. D’Haese, and Anja Verhulst. "Sclerostin as Regulatory Molecule in Vascular Media Calcification and the Bone–Vascular Axis." Toxins 11, no. 7 (July 21, 2019): 428. http://dx.doi.org/10.3390/toxins11070428.

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Sclerostin is a well-known inhibitor of bone formation that acts on Wnt/β-catenin signaling. This manuscript considers the possible role of sclerostin in vascular calcification, a process that shares many similarities with physiological bone formation. Rats were exposed to a warfarin-containing diet to induce vascular calcification. Vascular smooth muscle cell transdifferentiation, vascular calcification grade, and bone histomorphometry were examined. The presence and/or production of sclerostin was investigated in serum, aorta, and bone. Calcified human aortas were investigated to substantiat

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22

Kolotilov, Nikolay. "Pineal gland calcifications in patients with benign and malignant tumors of the larynx, pharynx, paranasal sinuses and auditory nerve." Radiation Diagnostics, Radiation Therapy, no. 2 (2020): 18–27. http://dx.doi.org/10.37336/2707-0700-2020-2-2.

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Calcifications of the brain – pineal gland, choroid plexuses of the lateral ventricles – are a long-known phenomenon. Calcification in such functionally important organs as the pineal gland and choroid plexuses of the lateral ventricles cannot be physiological (it is enough to recall the formation of stones in the gallbladder, kidneys, prostate gland, calcinosis of the heart valves and coronary vessels) or relatively neutral process in the body and not affect the activity of these organs. The purpose of the investigation is to study the nosological predisposition of calcifications and the inci

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23

Harpa, Marius Mihai, Ionela Movileanu, Leslie Neil Sierad, Ovidiu Simion Cotoi, Horatiu Suciu, Carmen Sircuta, Terezia Preda, et al. "Pulmonary heart valve replacement using stabilized acellular xenogeneic scaffolds; effects of seeding with autologous stem cells." Revista Romana de Medicina de Laborator 23, no. 4 (December 1, 2015): 415–30. http://dx.doi.org/10.1515/rrlm-2015-0046.

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Abstract Background: We hypothesized that an ideal heart valve replacement would be acellular valve root scaffolds seeded with autologous stem cells. To test this hypothesis, we prepared porcine acellular pulmonary valves, seeded them with autologous adipose derived stem cells (ADSCs) and implanted them in sheep and compared them to acellular valves. Methods: Fresh porcine pulmonary valve roots were decellularized with detergents and enzymes. ADSCs were isolated from subdermal fat and injected within the acellular cusps. Valves were then implanted in an extra-anatomic pulmonary position as RV

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24

Witteman, Jacqueline CM. "Coronary calcification detected by electron-beam computed tomography and non-coronary measures of atherosclerosis:." Circulation 103, suppl_1 (March 2001): 1344. http://dx.doi.org/10.1161/circ.103.suppl_1.9999-4.

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0004 Coronary calcification as detected by electron-beam tomography (EBT) is a measure of coronary atherosclerosis. We investigated whether coronary artery calcium is associated with non-coronary measures of atherosclerosis, using data from the population-based Rotterdam Study. Participants in the third phase of the study were invited to undergo EBT scanning for detection of coronary calcifications. EBT scans were obtained from 2263 participants (47% men, mean age (SD), 71.3 (5.7)). Quantification of coronary calcifications was performed using Agatston’s method, and resulted in a calcium score

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25

Schinzari, Francesca, Manfredi Tesauro, Aldo Bertoli, Alessia Valentini, Augusto Veneziani, Umberto Campia, and Carmine Cardillo. "Calcification biomarkers and vascular dysfunction in obesity and type 2 diabetes: influence of oral hypoglycemic agents." American Journal of Physiology-Endocrinology and Metabolism 317, no. 4 (October 1, 2019): E658—E666. http://dx.doi.org/10.1152/ajpendo.00204.2019.

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Vascular aging in obesity and type 2 diabetes (T2D) is associated with progressive vascular calcification, an independent predictor of morbidity and mortality. Pathways for vascular calcification modulate bone matrix deposition, thus regulating calcium deposits. We investigated the association between biomarkers of vascular calcification and vasodilator function in obesity or T2D, and whether antidiabetic therapies favorably impact those markers. Circulating levels of proteins involved in vascular calcification, such as osteopontin (OPN), osteoprotegerin (OPG), regulated on activation, normal

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26

Tong, Wenjuan, Xiaoling Zhang, Jia Luo, Fushun Pan, Jinyu Liang, Hui Huang, Manying Li, et al. "Value of multimodality imaging in the diagnosis of breast lesions with calcification: A retrospective study." Clinical Hemorheology and Microcirculation 76, no. 1 (October 15, 2020): 85–98. http://dx.doi.org/10.3233/ch-200877.

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PURPOSE: To assess the value of conventional ultrasound (US), contrast-enhanced ultrasound (CEUS) and mammography in the diagnosis of breast lesions with calcifications. METHODS: A total of 87 breast lesions with calcification were subjected to US, CEUS and mammography and divided into 3 groups: Group A (all cases), Group A1 (31 cases who underwent US and CEUS first followed by mammography), and Group A2 (56 cases who underwent mammography first followed by US and CEUS). A receiver operating characteristic (ROC) curve analysis was performed to evaluate the diagnostic efficacy of different meth

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27

Ross, Claire L., Verena Schoepf, Thomas M. DeCarlo, and Malcolm T. McCulloch. "Mechanisms and seasonal drivers of calcification in the temperate coral Turbinaria reniformis at its latitudinal limits." Proceedings of the Royal Society B: Biological Sciences 285, no. 1879 (May 23, 2018): 20180215. http://dx.doi.org/10.1098/rspb.2018.0215.

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High-latitude coral reefs provide natural laboratories for investigating the mechanisms and limits of coral calcification. While the calcification processes of tropical corals have been studied intensively, little is known about how their temperate counterparts grow under much lower temperature and light conditions. Here, we report the results of a long-term (2-year) study of seasonal changes in calcification rates, photo-physiology and calcifying fluid (cf) chemistry (using boron isotope systematics and Raman spectroscopy) for the coral Turbinaria reniformis growing near its latitudinal limit

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28

Zare, Ahmad, and Sara Choudhry. "Bilateral Basal Ganglia Calcification; An Unusual Initial Presentation of Pseudohypoparathyroidism." Journal of the Endocrine Society 5, Supplement_1 (May 1, 2021): A184—A185. http://dx.doi.org/10.1210/jendso/bvab048.373.

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Abstract Physiological intracranial calcification occurs in about 0.3–1.5% of cases. Hypoparathyroidism and pseudohypoparathyroidism are the most common causes of pathological basal ganglia calcification. A 21-year-old female who was initially evaluated by neurology team for headache and diplopia, underwent MRI of brain which revealed Calcifications involving the bilateral basal ganglia, thalami, dentate nuclei as well as juxtacortical frontal lobes. She reported Fatigue and muscle pain, usually in her arms especially after playing sports which had been going on for many years. She had no hist

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29

Boughner, D. R., M. Thornton, J. Dunmore-Buyze, and D. W. Holdsworth. "The radiographic quantitation of aortic valve calcification: implications for assessing bioprosthetic valve calcificationin vitro." Physiological Measurement 21, no. 3 (August 1, 2000): 409–16. http://dx.doi.org/10.1088/0967-3334/21/3/306.

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Atkinson, Jeffrey. "Age-related medial elastocalcinosis in arteries: mechanisms, animal models, and physiological consequences." Journal of Applied Physiology 105, no. 5 (November 2008): 1643–51. http://dx.doi.org/10.1152/japplphysiol.90476.2008.

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With age, the calcium content of the arterial wall increases. Calcification occurs at two main levels: intimal plaques and the medial elastic fiber network. The latter has been referred to as medial elastocalcinosis and is the subject of this review. The mechanisms involved in elastocalcinosis are complex and involve polar, apolar, and active processes. Vascular calcification may be species specific to humans. As laboratory animals, such as the rat, grow old, they suffer from only very mild arterial calcification. Different animal models of induction of massive arterial calcification by pharma

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31

Calabrò, Rocco Salvatore, Letteria Spadaro, Angela Marra, and Placido Bramanti. "Fahr’s Disease Presenting with Dementia at Onset: A Case Report and Literature Review." Behavioural Neurology 2014 (2014): 1–3. http://dx.doi.org/10.1155/2014/750975.

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Fahr’s disease (FD) is characterized by sporadic or familiar idiopathic calcification of the basal ganglia, dentate nuclei of the cerebellum, and centrum semiovale, mainly presenting with movement disorder, dementia, and behavioral abnormalities. We described a rare case of Fahr’s disease presenting at onset only with behavioral and neuropsychological alterations, whose diagnosis was supposed only after a brain CT, which showed extensive bilateral calcifications in the dentate nuclei of the cerebellum and basal ganglia. Since the onset of Fahr’s disease may be a dysexecutive syndrome with beha

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32

Peralta-Ramírez, A., A. Montes de Oca, A. I. Raya, C. Pineda, I. López, F. Guerrero, E. Diez, et al. "Vitamin E protection of obesity-enhanced vascular calcification in uremic rats." American Journal of Physiology-Renal Physiology 306, no. 4 (February 15, 2014): F422—F429. http://dx.doi.org/10.1152/ajprenal.00355.2013.

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This study aimed to determine the extent of extraskeletal calcification in uremic Zucker rats, by comparing obese and lean phenotypes, and to evaluate the influence of vitamin E (VitE) on the development of calcifications in both uremic rats and human vascular smooth muscle cells (HVSMCs) cultured in vitro. Zucker rats of lean and obese phenotypes with normal renal function [control (C); C-lean and C-obese groups] and with uremia [5/6 nephrectomy (Nx); Nx-lean and Nx-obese groups] and uremic rats treated with VitE (Nx-lean + VitE and Nx-obese + VitE groups) were studied. Uremic groups were sub

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33

Gori, Andrea, Christine Ferrier-Pagès, Sebastian J. Hennige, Fiona Murray, Cécile Rottier, Laura C. Wicks, and J. Murray Roberts. "Physiological response of the cold-water coralDesmophyllum dianthusto thermal stress and ocean acidification." PeerJ 4 (February 2, 2016): e1606. http://dx.doi.org/10.7717/peerj.1606.

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Rising temperatures and ocean acidification driven by anthropogenic carbon emissions threaten both tropical and temperate corals. However, the synergistic effect of these stressors on coral physiology is still poorly understood, in particular for cold-water corals. This study assessed changes in key physiological parameters (calcification, respiration and ammonium excretion) of the widespread cold-water coralDesmophyllum dianthusmaintained for ∼8 months at two temperatures (ambient 12 °C and elevated 15 °C) and two pCO2conditions (ambient 390 ppm and elevated 750 ppm). At ambient temperatures

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34

Sakai, H., and N. Nakamura. "Physiological calcification of the septum pellucidum." Neuroradiology 28, no. 2 (March 1986): 173. http://dx.doi.org/10.1007/bf00327893.

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Korff, Susanne, Frank Schoensiegel, Nora Riechert, Dieter Weichenhan, Hugo A. Katus, and Boris T. Ivandic. "Fine mapping of Dyscalc1, the major genetic determinant of dystrophic cardiac calcification in mice." Physiological Genomics 25, no. 3 (May 16, 2006): 387–92. http://dx.doi.org/10.1152/physiolgenomics.00010.2006.

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Calcification of severely dystrophic muscle is occasionally observed in targeted mouse models of muscular dystrophy and cardiomyopathy. Intracellular calcium deposition occurs in necrotic myocytes in the absence of plasma calcium and phosphate imbalances. In the heart, this recessive trait is referred to as dystrophic cardiac calcinosis (DCC). We identified previously Dyscalc1, a major genetic determinant of DCC, in a 15.2-Mbp region on proximal chromosome 7. We report now further steps toward the identification of the Dyscalc1 gene by reverse genetics. Transferring the Dyscalc1 locus from sus

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Chapperon, Coraline, Jacques Clavier, Clément Dugué, Erwan Amice, Manon Le Goff, and Sabine Roussel. "Seasonal and diurnal variability in carbon respiration, calcification and excretion rates of the abalone Haliotis tuberculata L." Journal of the Marine Biological Association of the United Kingdom 99, no. 2 (March 12, 2018): 393–402. http://dx.doi.org/10.1017/s0025315418000097.

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Abalone (Haliotis spp.) are commercially important marine shellfish species worldwide. Knowledge about the physiology of abalone that impacts life-history traits is important for a better understanding of the biology of the species and the impact of stressful husbandry procedures at different seasons. The present study quantified the seasonal and diurnal variations in four physiological parameters of the European species Haliotis tuberculata, i.e. carbon aerial and aquatic respiration, calcification and excretion rates, and the effect of prolonged aerial exposure upon abalone aerial respiratio

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Opdebeeck, Britt, Isabel R. Orriss, Ellen Neven, Patrick C. D’Haese, and Anja Verhulst. "Extracellular Nucleotides Regulate Arterial Calcification by Activating Both Independent and Dependent Purinergic Receptor Signaling Pathways." International Journal of Molecular Sciences 21, no. 20 (October 15, 2020): 7636. http://dx.doi.org/10.3390/ijms21207636.

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Arterial calcification, the deposition of calcium-phosphate crystals in the extracellular matrix, resembles physiological bone mineralization. It is well-known that extracellular nucleotides regulate bone homeostasis raising an emerging interest in the role of these molecules on arterial calcification. The purinergic independent pathway involves the enzymes ecto-nucleotide pyrophosphatase/phosphodiesterases (NPPs), ecto-nucleoside triphosphate diphosphohydrolases (NTPDases), 5′-nucleotidase and alkaline phosphatase. These regulate the production and breakdown of the calcification inhibitor—pyr

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Findlay, H. S., H. L. Wood, M. A. Kendall, J. I. Spicer, R. J. Twitchett, and S. Widdicombe. "Calcification, a physiological process to be considered in the context of the whole organism." Biogeosciences Discussions 6, no. 1 (February 24, 2009): 2267–84. http://dx.doi.org/10.5194/bgd-6-2267-2009.

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Abstract. Marine organisms that produce calcium carbonate structures are predicted to be most vulnerable to a decline in oceanic pH (ocean acidification) based on the understanding that calcification rates will decrease as a result of changes in the seawater carbonate chemistry thereby reducing carbonate ion concentration (and associated saturation states). Coastal seas are critical components of the global carbon cycle yet little research has been conducted on acidification impacts on coastal benthic organisms. Here, a critical appraisal of calcification in six benthic species showed, contrar

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Lohbeck, Kai T., Ulf Riebesell, and Thorsten B. H. Reusch. "Gene expression changes in the coccolithophore Emiliania huxleyi after 500 generations of selection to ocean acidification." Proceedings of the Royal Society B: Biological Sciences 281, no. 1786 (July 7, 2014): 20140003. http://dx.doi.org/10.1098/rspb.2014.0003.

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Coccolithophores are unicellular marine algae that produce biogenic calcite scales and substantially contribute to marine primary production and carbon export to the deep ocean. Ongoing ocean acidification particularly impairs calcifying organisms, mostly resulting in decreased growth and calcification. Recent studies revealed that the immediate physiological response in the coccolithophore Emiliania huxleyi to ocean acidification may be partially compensated by evolutionary adaptation, yet the underlying molecular mechanisms are currently unknown. Here, we report on the expression levels of 1

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Wall, C. B., R. A. B. Mason, W. R. Ellis, R. Cunning, and R. D. Gates. "Elevated p CO 2 affects tissue biomass composition, but not calcification, in a reef coral under two light regimes." Royal Society Open Science 4, no. 11 (November 2017): 170683. http://dx.doi.org/10.1098/rsos.170683.

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Ocean acidification (OA) is predicted to reduce reef coral calcification rates and threaten the long-term growth of coral reefs under climate change. Reduced coral growth at elevated p CO 2 may be buffered by sufficiently high irradiances; however, the interactive effects of OA and irradiance on other fundamental aspects of coral physiology, such as the composition and energetics of coral biomass, remain largely unexplored. This study tested the effects of two light treatments (7.5 versus 15.7 mol photons m −2 d −1 ) at ambient or elevated p CO 2 (435 versus 957 µatm) on calcification, photopi

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Thomsen, J., K. Haynert, K. M. Wegner, and F. Melzner. "Impact of seawater carbonate chemistry on the calcification of marine bivalves." Biogeosciences 12, no. 14 (July 17, 2015): 4209–20. http://dx.doi.org/10.5194/bg-12-4209-2015.

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Abstract. Bivalve calcification, particularly of the early larval stages, is highly sensitive to the change in ocean carbonate chemistry resulting from atmospheric CO2 uptake. Earlier studies suggested that declining seawater [CO32−] and thereby lowered carbonate saturation affect shell production. However, disturbances of physiological processes such as acid-base regulation by adverse seawater pCO2 and pH can affect calcification in a secondary fashion. In order to determine the exact carbonate system component by which growth and calcification are affected it is necessary to utilize more com

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de Nooijer, L. J., G. Langer, G. Nehrke, and J. Bijma. "Physiological controls on seawater uptake and calcification in the benthic foraminifer <i>Ammonia tepida</i>." Biogeosciences Discussions 6, no. 4 (July 16, 2009): 7083–102. http://dx.doi.org/10.5194/bgd-6-7083-2009.

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Abstract. During the last decades conceptual models describing the calcification pathway of foraminifera and its physiological controls have been developed. These models are derived by combining data of tracer experiments and microscopic observations obtained from different species. Although vital for understanding their calcitic isotopic and trace elemental composition, direct observational evidence on e.g. seawater vacuolization and intracellular Ca-cycling is lacking for most species. To analyse the relation between seawater uptake and calcification, we incubated juveniles of the cosmopolit

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Chen, Qiang, Ze-Yang Wang, Li-Yuan Chen, and Hou-Yuan Hu. "Roles of High Mobility Group Box 1 in Cardiovascular Calcification." Cellular Physiology and Biochemistry 42, no. 2 (2017): 427–40. http://dx.doi.org/10.1159/000477591.

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Calcific disease of the cardiovascular system, including atherosclerotic calcification, medial calcification in diabetes and calcific aortic valve disease, is an important risk factor for many adverse cardiovascular events such as ischemic cardiac events and subsequent mortality. Although cardiovascular calcification has long been considered to be a passive degenerative occurrence, it is now recognized as an active and highly regulated process that involves osteochondrogenic differentiation, apoptosis and extracellular vesicle release. Nonetheless, despite numerous studies on the pathogenesis

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Thomsen, J., K. Haynert, K. M. Wegner, and F. Melzner. "Impact of seawater carbonate chemistry on the calcification of marine bivalves." Biogeosciences Discussions 12, no. 2 (January 22, 2015): 1543–71. http://dx.doi.org/10.5194/bgd-12-1543-2015.

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Анотація:

Abstract. Bivalve calcification, particular of the early larval stages is highly sensitive to the change of ocean carbonate chemistry resulting from atmospheric CO2 uptake. Earlier studies suggested that declining seawater [CO32−] and thereby lowered carbonate saturation affect shell production. However, disturbances of physiological processes such as acid-base regulation by adverse seawater pCO2 and pH can affect calcification in a secondary fashion. In order to determine the exact carbonate system component by which growth and calcification are affected it is necessary to utilize more comple

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Meng, Fanxing, Yonggang Zhao, Bing Wang, Bingwei Li, Youming Sheng, Mingming Liu, Hongwei Li, and Ruijuan Xiu. "Endothelial Cells Promote Calcification in Aortic Smooth Muscle Cells from Spontaneously Hypertensive Rats." Cellular Physiology and Biochemistry 49, no. 6 (2018): 2371–81. http://dx.doi.org/10.1159/000493837.

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Background/Aims: Vascular calcification and hypertension are intimately linked, and the progression of hypertension is closely correlated with endothelial dysfunction. However, the role of endothelial cells (ECs) in vascular calcification of hypertension remains unclear. Therefore, the present study explored the effects of ECs on calcification of smooth muscle cells (SMCs) from aortas of spontaneously hypertensive rats (SHR). Methods: Aortic ECs and SMCs were isolated from SHR and Wistar rats, respectively. The roles of ECs in the regulation of SMCs calcification were investigated by co-cultur

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Carracedo, Miguel, Gonzalo Artiach, Anna Witasp, Joan Clària, Mattias Carlström, Andres Laguna-Fernandez, Peter Stenvinkel, and Magnus Bäck. "The G-protein coupled receptor ChemR23 determines smooth muscle cell phenotypic switching to enhance high phosphate-induced vascular calcification." Cardiovascular Research 115, no. 10 (December 28, 2018): 1557–66. http://dx.doi.org/10.1093/cvr/cvy316.

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Abstract Aims Vascular calcification, a marker of increased cardiovascular risk, is an active process orchestrated by smooth muscle cells. Observational studies indicate that omega-3 fatty acids protect against vascular calcification, but the mechanisms are unknown. The G-protein coupled receptor ChemR23 transduces the resolution of inflammation induced by the omega-3-derived lipid mediator resolvin E1. ChemR23 also contributes to osteoblastic differentiation of stem cells and bone formation, but its role in vascular calcification is unknown. The aim of this study was to establish the role of

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Bayrak, Seval, Duygu Göller Bulut, Emine Şebnem Kurşun Çakmak, and Kaan Orhan. "Cone Beam Computed Tomographic Evaluation of Intracranial Physiologic Calcifications." Journal of Craniofacial Surgery 30, no. 2 (2019): 510–13. http://dx.doi.org/10.1097/scs.0000000000004918.

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Lee, Chi H. "Physiological variables involved in heart valve substitute calcification." Expert Opinion on Biological Therapy 9, no. 8 (June 26, 2009): 1031–42. http://dx.doi.org/10.1517/14712590903085091.

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Shuvy, Mony, Suzan Abedat, Ronen Beeri, Michael Valitsky, Sameh Daher, Miriam Kott-Gutkowski, Anca Gal-Moscovici, Jacob Sosna, Nalini M. Rajamannan, and Chaim Lotan. "Raloxifene attenuates Gas6 and apoptosis in experimental aortic valve disease in renal failure." American Journal of Physiology-Heart and Circulatory Physiology 300, no. 5 (May 2011): H1829—H1840. http://dx.doi.org/10.1152/ajpheart.00240.2010.

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Renal failure is associated with aortic valve calcification. Using our rat model of uremia-induced reversible aortic valve calcification, we assessed the role of apoptosis and survival pathways in that disease. We also explored the effects of raloxifene, an estrogen receptor modulator, on valvular calcification. Gene array analysis was performed in aortic valves obtained from three groups of rats ( n = 7 rats/group): calcified valves obtained from rats fed with uremic diet, valves after calcification resolution following diet cessation, and control. In addition, four groups of rats ( n = 10 ra

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Bobryshev, Yuri V., Reginald S. A. Lord, and Dinh Tran. "Chlamydia pneumoniaein foci of “early” calcification of the tunica media in arteriosclerotic arteries: an incidental presence?" American Journal of Physiology-Heart and Circulatory Physiology 290, no. 4 (April 2006): H1510—H1519. http://dx.doi.org/10.1152/ajpheart.01055.2005.

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Only a few previous works investigated the involvement of Chlamydia pneumoniae ( Chlamydophila pneumoniae) in arterial calcification. The present study investigated a possible association between C. pneumoniae and medial calcification. Carotid artery segments obtained by endarterectomy from 60 patients were examined by PCR and immunohistochemistry to identify the presence of C. pneumoniae. Arterial specimens showing double-positive ( n = 17), double-negative ( n = 22), and single-positive results ( n = 21) were further analyzed by a combination of histology, immunohistochemistry, and electron

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