Готові списки джерел за темами / Cholesterol control

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  2. Дисертації
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  5. Тези доповідей конференцій
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Добірка наукової літератури з теми "Cholesterol control"

Автор: Grafiati
Опубліковано: 4 червня 2021
Оновлено: 20 лютого 2023

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Статті в журналах з теми "Cholesterol control"

1

Kiberstis, P. A. "Idolizing Cholesterol Control." Science Signaling 2, no. 78 (July 7, 2009): ec232-ec232. http://dx.doi.org/10.1126/scisignal.278ec232.

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2

Fletcher, Gerald F. "Fat and Cholesterol Control." Journal of Cardiopulmonary Rehabilitation 12, no. 3 (May 1992): 162–63. http://dx.doi.org/10.1097/00008483-199205000-00002.

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3

Marecic, Maryfrances, and Robin Bagby. "Take Control of Cholesterol." Nutrition Today 23, no. 5 (September 1988): 47. http://dx.doi.org/10.1097/00017285-198809000-00011.

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4

Belavic, Jennifer. "Drugs to control cholesterol." Nursing 42, no. 3 (March 2012): 68. http://dx.doi.org/10.1097/01.nurse.0000398753.52565.6a.

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5

Haug, Anna, Arne T. Høstmark, Øystein Spydevold, and Einar Eilertsen. "Hypercholesterolaemia, hypotriacylglycerolaemia and increased lipoprotein lipase activity following orchidectomy in rats." Acta Endocrinologica 113, no. 1 (September 1986): 133–39. http://dx.doi.org/10.1530/acta.0.1130133.

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Abstract. Plasma lipoproteins, faecal cholesterol excretion, and activities of lecithin: cholesterol acyltransferase (LCAT) hepatic lipase (HL), and lipoprotein lipase (LPL) were determined in castrated rats, in rats treated with testosterone propionate after castration, and in sham-operated controls. Compared to control rats, whole-plasma total cholesterol (TC) rose, and triacylglycerols (TG) fell in castrated rats, but were normalized by androgen substitution. VLDL components tended to be reduced, whereas HDL2 components rose following castration. In general, testosterone substitution normal
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6

Khan, B., H. G. Wilcox, and M. Heimberg. "Cholesterol is required for secretion of very-low-density lipoprotein by rat liver." Biochemical Journal 258, no. 3 (March 15, 1989): 807–16. http://dx.doi.org/10.1042/bj2580807.

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To study potential effects of hepatic cholesterol concentration on secretion of very-low-density lipoprotein (VLDL) by the liver, male rats were fed on unsupplemented chow, chow with lovastatin (0.1%), or chow with lovastatin (0.1%) and cholesterol (0.1%) for 1 week. Livers were isolated from these animals and perfused in vitro, with a medium containing [2-14C]acetate, bovine serum albumin and glucose in Krebs-Henseleit buffer, and with an oleate-albumin complex. With lovastatin feeding, the hepatic concentrations of cholesteryl esters and triacylglycerols before perfusion were decreased, alth
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7

Anonymous. "Steps Overlooked in Cholesterol Control." Journal of Gerontological Nursing 20, no. 12 (December 1994): 47. http://dx.doi.org/10.3928/0098-9134-19941201-13.

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8

Florez, Hermes, Kelly J. Hunt, and Willy Marcos Valencia. "Reducing Disparities in Cholesterol Control." JAMA 328, no. 8 (August 23, 2022): 714. http://dx.doi.org/10.1001/jama.2022.13284.

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9

Björkhem, Ingemar. "Do oxysterols control cholesterol homeostasis?" Journal of Clinical Investigation 110, no. 6 (September 15, 2002): 725–30. http://dx.doi.org/10.1172/jci0216388.

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10

Kiberstis, P. A. "MiR-33 in Cholesterol Control." Science Signaling 3, no. 127 (June 22, 2010): ec189-ec189. http://dx.doi.org/10.1126/scisignal.3127ec189.

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Дисертації з теми "Cholesterol control"

1

Al-Seeni, Madeha N. "Control of hepatic cholesterol esterification." Thesis, University of Nottingham, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.293154.

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2

Sampson, William James. "The intracellular control of cholesterol metabolism." Thesis, University of Edinburgh, 1988. http://hdl.handle.net/1842/26913.

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The liver has a major role in the metabolism of cholesterol, being the main site of lipoprotein assembly and degradation and the only tissue where the metabolism of cholesterol to bile acids occurs. This provides the major pathway for the removal of cholesterol from the body. The results described in this thesis concern the use of specific enzyme inhibitors (58-035, Azacholesterol, Mevinolin) to determine the intracellular use of different sources of cholesterol in monolayers of rat hepatocytes. In particular, the fates of newly synthesized cholesterol from mevalonic acid and cholesterol deriv
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3

Veen, Jelske Nynke van der. "Nuclear receptors in control of cholesterol transport." [S.l. : Groningen : s.n. ; University Library Groningen] [Host], 2007. http://irs.ub.rug.nl/ppn/304675490.

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4

Dunn, Stuart Antony. "Hormone-sensitive lipase and the control of lipid metabolism in the human macrophage foam cell." Thesis, University of Newcastle Upon Tyne, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.311114.

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5

Sims, Helen M. "Transcriptional control of microsomal triglyceride transfer protein gene expression in the hamster." Thesis, University of Nottingham, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.366470.

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6

Martinic, Goran (Gary), of Western Sydney Hawkesbury University, of Science Technology and Environment College, and School of Environment and Agriculture. "Cyclodextrins as potential human anti-atherosclerotic agents." THESIS_CSTE_EAG_Martinic_G.xml, 2001. http://handle.uws.edu.au:8081/1959.7/129.

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Cyclodextrins (CDs) are naturally occurring cyclic oligosaccharides. Since it is believed that OxC blocks the removal of normal cholesterol from cells in the artery wall, it is possible that selective removal of OxC in the vessel wall in-vivo may prevent or reverse atherosclerosis.As a prelude to major studies, this research project was designed to answer two critical questions; 1/. What is the best route for delivery of CD. 2/. How do animals (apoE-/- mice) tolerate it. Pilot studies were established and results noted. These studies have provided valuable information in the apoE-/- mouse for
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7

Witsken, Colleen. "The Effect of Parental Control Over Child-Feeding on Compliance to Dietary Recommendations to Lower Blood Cholesterol." University of Cincinnati / OhioLINK, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1179758468.

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8

Dun, Alison. "Spatial and temporal control of regulated exocytosis by protein and lipid interactions." Thesis, University of Edinburgh, 2013. http://hdl.handle.net/1842/8087.

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Cellular communication requires the transport of chemical messengers between intracellular compartments and from cell to cell. The regulated exocytosis of a secretory vesicle at the plasma membrane involves the merger of two bilayers, with markedly different lipid composition, within a millisecond time scale. The spatial and temporal control of the protein and lipid complement at these fusion sites is essential. A highly conserved family of proteins are known to drive this fusion event; SNAP-25 and syntaxin-1 (t-SNAREs) associate at the plasma membrane in a 1:1 stoichiometry to provide a bindi
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9

Kangas-Kontio, T. (Tiia). "Genetic background of HDL-cholesterol and atherosclerosis:linkage and case-control studies in the Northern Finnish population." Doctoral thesis, Oulun yliopisto, 2011. http://urn.fi/urn:isbn:9789514295812.

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Abstract Coronary heart disease (CHD), a manifestation of atherosclerosis, is the leading single cause of death in Finland. CHD is affected by numerous genetic and environmental factors, their combined effects and interactions between them. Low HDL-cholesterol (HDL-C) is an independent risk factor for atherosclerosis and the most common dyslipidemia associated with early onset CHD, but the mechanisms regulating HDL-C levels and protecting from atherosclerosis are still not completely understood. Adiponectin is a hormone that is secreted by adipose tissue and has several anti-atherosclerotic ef
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10

Kranz, Eylath. "Dietary habits and life style in the etiology of cholesterol gallstone disease a matched case control study /." [S.l.] : [s.n.], 2002. http://deposit.ddb.de/cgi-bin/dokserv?idn=968492649.

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Книги з теми "Cholesterol control"

1

Diets to help control cholesterol. London: Thorsons, 1993.

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2

Harriet Roth's cholesterol-control cookbook. New York: New American Library, 1989.

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3

Roth, Harriet. Harriet Roth's cholesterol control cookbook. 2nd ed. New York: Plume, 2008.

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4

Roth, Harriet. Harriet Roth"s Cholesterol Control Cookbook. New York: A Plume Book, 1991.

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5

Royal Society of Medicine (Great Britain), ed. The cholesterol controversy. London: Royal Society of Medicine Press, 2008.

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6

Cholesterol control without diet!: The niacin solution. Scottdale, Ariz: Lilac Press, 1998.

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7

Bergasa, Ana María Lajusticia. Colesterol, triglicéridos y su control. Madrid: EDAF, 2002.

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8

Horovitz, Emmanuel. Cholesterol control made easy: How to lower your cholesterol for a healthier heart. Los Angeles, Calif: Health Trend Pub., 1990.

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9

Horovitz, Emmanuel. Cholesterol control made easy: How to lower your cholesterol for a healthier heart. Los Angeles: Health Trend Pub., 1990.

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10

Ulene, Art. Count out cholesterol. Berkeley, CA: Ulysses Press, 1994.

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Частини книг з теми "Cholesterol control"

1

Rittershaus, Charles W. "Inducing Autoreactivity to Control Cholesterol Metabolism." In Vascular Endothelium, 273. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4899-0133-0_35.

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2

Bosisio, E. "Dietary and Pharmacological Control of Cholesterol 7α-Hydroxylase." In Drugs Affecting Lipid Metabolism, 21–28. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71702-4_4.

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3

Luskey, K. L. "The Control of Cholesterol Homeostasis: Regulation of HMG CoA Reductase." In Receptor-Mediated Uptake in the Liver, 80–82. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-70956-2_15.

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4

Henrion, André. "In- and off-laboratory sources of uncertainty in the use of a serum standard reference material as a means of accuracy control in cholesterol determination." In Measurement Uncertainty in Chemical Analysis, 248–51. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-662-05173-3_44.

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5

Teramoto, Akio, Kazuto Yoshiba, Shigeru Matsushima, and Naotake Nakamura. "Helical Conformations of Conjugating Polymers and Their Control of Cholesteric Order." In ACS Symposium Series, 47–62. Washington, DC: American Chemical Society, 2005. http://dx.doi.org/10.1021/bk-2005-0916.ch005.

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6

Johnson, Heather M., and Patrick E. McBride. "Chapter 12High Blood Cholesterol." In Chronic Disease Epidemiology and Control. American Public Health Association, 2010. http://dx.doi.org/10.2105/9780875531922ch12.

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7

"Program on the Surgical Control of the Hyperlipidemias (POSCH) Trial: A Pivotal 25-Year Study." In Cholesterol-Lowering Therapy, 135–90. CRC Press, 1999. http://dx.doi.org/10.1201/b15444-7.

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8

Kubica, Krystian, and Joanna Balbus. "Mathematical modeling of cholesterol homeostasis." In Control Theory in Biomedical Engineering, 43–61. Elsevier, 2020. http://dx.doi.org/10.1016/b978-0-12-821350-6.00002-0.

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9

Purcell, John. "Cholesterol Oxidase for the Control of Boll Weevil." In Advances In Insect Control. CRC Press, 1997. http://dx.doi.org/10.1201/9780203211731.ch6.

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10

"Statins: Fermentation Products for Cholesterol Control in Humans." In Chemistry and Pharmacology of Naturally Occurring Bioactive Compounds, 457–66. CRC Press, 2013. http://dx.doi.org/10.1201/b13867-21.

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Тези доповідей конференцій з теми "Cholesterol control"

1

Pescador, R., R. Porta, R. Niada, M. Mantovani, and G. Prino. "DEFIBROTIDE DECREASES CHOLESTEROL CONTENT IN HYPERCHOLESTEROLEMIC RABBIT AORTA, WITH NO MODIFICATION OF PLASMA OR LIPOPROTEIN CHOLESTEROL." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643153.

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Defibrotide was shown to stimulate the production of endogenous PGI2 from rat and hamster aortic tissue, as well as the production of PGi2 frcm the coronary vascular bed in the platelet perfused heart model. This effect was only seen in presence of platelets. Durirg the infusion period with Defibrotide thromboxane release remained unaffected while platelet cAMP rised. Defibrotide, was also able to reduce the secretion of ATP from platelets as well as to deaggregate platelet cluTps. It has been postulated that long-term administration of stable PGI^ metabolites or analogues could be a more usef
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2

Verbeuren, T. J., M. J. Van Diest, and A. G. Herman. "CONTRACTIONS TO PLATELETS IN AORTAS OF CONTROL AND CHOLESTEROL-FED RABBITS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643799.

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Atherosclerotic aortas obtained from cholesterol-fed rabbits show a decreased responsiveness to noradrenaline, an increased responsiveness to low concentrations of serotoninand an unaltered responsiveness to prostaglandins. In vitro contractions induced by aggregating platelets are largely due to serotonin liberated during the aggregation. The present study was designed to compare the contractile responses to aggregating platelets inaortas obtained from control and cholesterol-fed rabbits.Male New Zealand rabbits were fed either a control or a 0.3% cholesterol diet during 16 weeks. Macroscopic
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3

Purwanti, Nunik, Rahmadiar Aditya Putri, and Siti Nurjanah. "Effectiveness of Sembung Tree Honey on Decreasing Cholesterol Level in Hypercolesterolemia." In The 7th International Conference on Public Health 2020. Masters Program in Public Health, Universitas Sebelas Maret, 2020. http://dx.doi.org/10.26911/the7thicph.05.22.

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ABSTRACT Background: Lifestyle changes, causing an increase in cholesterol. Honey has done a lot of research. The content of flavonoids can lower cholesterol. Many people consume Sembung tree honey, but not much research has been done, including its effectiveness in reducing cholesterol. This study aimed to analyze the effectiveness of sembung tree honey to reduce cholesterol levels in hypercholesterolemia. Subjects and Method: This was a quasi-experiment with pretest and posttest control group. A sample of 20 hypercholesterolemia sufferers in Kedensari Village was selected by purposive sampli
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4

Gunduz, Canan, Ozen K. Basoglu, Jan Hedner, Maria R. Bonsignore, Holger Hein, Richard Staats, Izoldi Bouloukaki, et al. "Hyperlipidemia Prevalence and Cholesterol Control in OSA: Data from European Sleep Apnea Database (ESADA)." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.oa3274.

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5

Badimon, J., L. Bodimon, and V. Fuster. "HIGH AND VERY HIGH DENSITY LIPOPROTEINS ADMINISTRATION INHIBITS PROGRESSION OF EXPERIMENTAL ATHEROSCLEROSIS IN THE RABBIT." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643748.

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Epidemiologic studies have shown an inverse relationship between HDL and coronary artery disease. We have previously demonstrated that in vivo administration of HDL-VHDL inhibits thedevelopment of atherosclerosis in cholesterol (cho)-fed rabbits. In the present study we have analyzed whether high levels of the physiological cholesterol acceptor, homologous HDL-VHDL,could inhibit the progression of established atherosclerotic lesions. Atherosclerosis was induced by feeding rabbits a 0.5% cho-rich diet for 2 months (140g/day). At that moment, a subgroup of animal (N=4) was sacrificed and their a
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6

Tamaoki, Nobuyuki, Yasuyuki Aoki, Masaya Moriyama, and Masatoshi Kidowaki. "Photochemical control of helical pitch of glass-forming dimeric cholesteric liquid crystals by isomerization of embedded di-mesogenic compounds with both cholesterol and azobenzene groups." In International Symposium on Optical Science and Technology, edited by Iam-Choon Khoo. SPIE, 2002. http://dx.doi.org/10.1117/12.453260.

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7

Wanner, C., M. Cooper, S. Inzucchi, B. Zinman, I. Zwiener, M. Eynatten, and A. Koitka-Weber. "Empagliflozin Improves Kidney Outcomes Irrespective of Control of Blood Pressure, Low-Density Lipoprotein Cholesterol and HbA1c." In Diabetes Kongress 2019 – 54. Jahrestagung der DDG. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1688342.

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8

Fitchett, D., J. Lee, JT George, HJ Woerle, and SE Inzucchi. "Empagliflozin reduces heart failure irrespective of control of blood pressure, low density lipoprotein cholesterol and HbA1c." In Diabetes Kongress 2018 – 53. Jahrestagung der DDG. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1641913.

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9

Fazil, Qurratu'Aini Aishah Ahmad, and Ummu Kulthum Jamaludin. "Investigation on the relationship between cholesterol and blood glucose levels using decision tree method in healthy subjects." In 2017 IEEE 2nd International Conference on Automatic Control and Intelligent Systems (I2CACIS). IEEE, 2017. http://dx.doi.org/10.1109/i2cacis.2017.8239051.

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10

Lobel, P., M. Palmer, and K. Schor. "CHRONIC ORAL DEFIBROTIDE STIMULATES VASCULAR PGI2 AND INHIBITS ATHEROSCLEROTIC PLAQUE FORMATION IN CHOLESTEROL-FED RABBITS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643150.

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Defibrotide (DEF) is a polydeoxyribonucleotide fraction from bovine lung, possessing profibrinolytic and PGI2 stimulating properties. Male rabbits were fed for 4 months a standard laboratory diet (150 g/day) without (A) or with (B) DEF or a cholesterol (1%) supplemented diet without (C) or with (D) DEF (60 mg/kg x day) was administered orally (drinking water) and withdrawn 24-36 h prior to the acute experiments.DEF did not change the elevated serum cholesterol: 18 ± 2 (C) vs. 26 ± 5 (D) mM but significantly reduced the plaque formation in the aorta from 4.5 ± 0.3 (C) to 3.3 ± 0.2 (D) (subjecti
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Звіти організацій з теми "Cholesterol control"

1

Gao, Hui, Chen Gong, Shi-chun Shen, Jia-ying Zhao, Dou-dou Xu, Fang-biao Tao, Yang Wang, and Xiao-chen Fan. A systematic review on the associations between prenatal phthalate exposure and childhood glycolipid metabolism and blood pressure: evidence from epidemiological studies. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2022. http://dx.doi.org/10.37766/inplasy2022.6.0111.

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Review question / Objective: The present systematic review was performed to obtain a summary of epidemiological evidence on the relationships of in utero exposure to phthalates with childhood glycolipid metabolism and blood pressure. Condition being studied: Childhood cardiovascular risk factors including blood pressure, lipid profile (e.g., triglycerides, total cholesterol, HDL−C, LDL−C) and glucose metabolism (e.g., insulin, insulin resistance, insulin sensitivity, glucose) were the interested outcomes. Eligibility criteria: In brief, epidemiological studies including cohort study, case-cont
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2

Chen, Jiankun, Yingming Gu, Lihong Yin, Minyi He, Na Liu, Yue Lu, Changcai Xie, Jiqiang Li, and Yu Chen. Network meta-analysis of curative efficacy of different acupuncture methods on obesity combined with insulin resistance. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2022. http://dx.doi.org/10.37766/inplasy2022.8.0075.

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Review question / Objective: Population：Patients diagnosed as obesity with insulin resistance. Obesity reference: Consensus of experts on the Prevention and treatment of adult obesity in China in 2011 and Consensus of Chinese experts on medical nutrition therapy for overweight/obesity in 2016 were developed by the Obesity Group of Chinese Society of Endocrinology(CSE); BMI≥28. IR reference: According to the Expert opinions on insulin resistance evaluation published by Chinese Diabetes Society, HOMA-IR≥2.68 is regarded as the standard for the diagnosis of IR. Regardless of age, gender and cours
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Також вас можуть зацікавити розширені добірки на тему "Cholesterol control" для конкретних типів джерел:
Статті в журналах Дисертації Книги
Ми пропонуємо знижки на всі преміум-плани для авторів, чиї праці увійшли до тематичних добірок літератури. Зв'яжіться з нами, щоб отримати унікальний промокод!

До бібліографії