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Готові списки джерел за темами / Hemorrhagic conversion / Статті в журналах

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Статті в журналах з теми "Hemorrhagic conversion"

Автор: Grafiati

Опубліковано: 3 червня 2025

Оновлено: 31 липня 2025

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1

de Courten-Myers, Gabrielle M., Marla Kleinholz, Pat Holm, et al. "Hemorrhagic infarct conversion in experimental stroke." Annals of Emergency Medicine 21, no. 2 (1992): 120–26. http://dx.doi.org/10.1016/s0196-0644(05)80144-1.

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2

Dang, Jinfeng, Jieting Li, Haixia Li, Gaoyan Pian, and Zhizhuan Guo. "The Value of a Management Plan Based on Risk Factors for Cerebral Infarction Patients with Cerebral Hemorrhage." Journal of Healthcare Engineering 2022 (April 8, 2022): 1–6. http://dx.doi.org/10.1155/2022/8635487.

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Cerebral infarction is a neurological disease related to abnormal blood supply to brain tissue. Patients are mainly men between the ages of 50 and 60 years old. In order to explore the value of risk factor-based management programs for preventing hemorrhagic conversion in patients with cerebral infarction, this paper uses single and multifactor methods to analyze the risk factors of cerebral hemorrhage transformation after cerebral infarction and formulate risk factor-based management plans. 240 cases of cerebral infarction who were admitted to our hospital in the past 2 years were evenly divi

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3

Won, Soonmi, Jin Hwan Lee, Bushra Wali, Donald G. Stein, and Iqbal Sayeed. "Progesterone Attenuates Hemorrhagic Transformation after Delayed tPA Treatment in an Experimental Model of Stroke in Rats: Involvement of the VEGF–MMP Pathway." Journal of Cerebral Blood Flow & Metabolism 34, no. 1 (2013): 72–80. http://dx.doi.org/10.1038/jcbfm.2013.163.

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Tissue plasminogen activator (tPA) is the only FDA-approved treatment for acute stroke, but its use remains limited. Progesterone (PROG) has shown neuroprotection in ischemia, but before clinical testing, we must determine how it affects hemorrhagic transformation in tPA-treated ischemic rats. Male Sprague–Dawley rats underwent middle cerebral artery occlusion with reperfusion at 4.5 hours and tPA treatment at 4.5 hours, or PROG treatment intraperitoneally at 2 hours followed by subcutaneous injection at 6 hours post occlusion. Rats were killed at 24 hours and brains evaluated for cerebral hem

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4

Agarwal, Nitin, Giulio Zuccoli, Geoffrey Murdoch, Brian T. Jankowitz, and Stephanie Greene. "Developmental venous anomaly presenting as a spontaneous intraparenchymal hematoma without thrombosis." Neuroradiology Journal 29, no. 6 (2016): 465–69. http://dx.doi.org/10.1177/1971400916665387.

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Introduction Developmental venous anomalies (DVAs) are cited as the most common cerebral vascular malformations. Still, intracerebral hematomas are rarely thought to be caused by DVAs. In this report, the authors present a unique case of a DVA that hemorrhaged spontaneously, rather than hemorrhaging into a venous infarction following DVA thrombosis as has been more commonly reported. Clinical presentation A 22-year-old previously healthy male presented to the emergency department with a severe headache, confusion, and progressive hemiparesis. A computed tomography (CT) scan demonstrated a spon

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5

Khanna, Arjun, Brian P. Walcott, Kristopher T. Kahle, and J. Marc Simard. "Effect of glibenclamide on the prevention of secondary brain injury following ischemic stroke in humans." Neurosurgical Focus 36, no. 1 (2014): E11. http://dx.doi.org/10.3171/2013.10.focus13404.

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Cerebral edema and hemorrhagic conversion are common, potentially devastating complications of ischemic stroke and are associated with high rates of mortality and poor functional outcomes. Recent work exploring the molecular pathophysiology of the neurogliovascular unit in ischemic stroke suggests that deranged cellular ion homeostasis due to altered function and regulation of ion pumps, channels, and secondary active transporters plays an integral role in the development of cytotoxic and vasogenic edema and hemorrhagic conversion. Among these proteins involved in ion homeostasis, the ischemia

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6

Schneider, Christian P., Martin G. Schwacha, T. S. Anantha Samy, Kirby I. Bland та Irshad H. Chaudry. "Androgen-mediated modulation of macrophage function after trauma-hemorrhage: central role of 5α-dihydrotestosterone". Journal of Applied Physiology 95, № 1 (2003): 104–12. http://dx.doi.org/10.1152/japplphysiol.00182.2003.

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Androgens have been implicated as the causative factor for the postinjury immune dysfunction in males; however, it remains unknown whether androgens directly affect macrophages. To study this, male mice were sham operated or subjected to trauma (i.e., midline laparotomy) and hemorrhagic shock (mean arterial pressure, 30 ± 5 mmHg for 90 min and then resuscitated). The mice received the 5α-reductase inhibitor 4-hydroxyandrostenedione (4-OHA) before resuscitation. Plasma TNF-α, IL-6, and IL-10 levels were elevated after trauma-hemorrhage and normalized by 4-OHA. TNF-α and IL-6 production by splen

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7

Kim, Beom Joon, and Seung-Hoon Lee. "Silent Microbleeds and Hemorrhagic Conversion of an Embolic Infarction." Journal of Clinical Neurology 3, no. 3 (2007): 147. http://dx.doi.org/10.3988/jcn.2007.3.3.147.

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8

Dietzen, Anton, Anton Dietzen, Anjum Sayyad, and Susan Brady. "Anticoagulation: Hemorrhagic Conversion and VTE following Acute Ischemic Stroke." Archives of Physical Medicine and Rehabilitation 97, no. 10 (2016): e91. http://dx.doi.org/10.1016/j.apmr.2016.08.281.

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9

Modelska, K., M. A. Matthay, L. A. S. Brown, E. Deutch, L. N. Lu та J. F. Pittet. "Inhibition of β-adrenergic-dependent alveolar epithelial clearance by oxidant mechanisms after hemorrhagic shock". American Journal of Physiology-Lung Cellular and Molecular Physiology 276, № 5 (1999): L844—L857. http://dx.doi.org/10.1152/ajplung.1999.276.5.l844.

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Endogenous release of catecholamines is an important mechanism that can prevent alveolar flooding after brief but severe hemorrhagic shock. The objective of this study was to determine whether this catecholamine-dependent mechanism upregulates alveolar liquid clearance after prolonged hemorrhagic shock. Rats were hemorrhaged to a mean arterial pressure of 30–35 mmHg for 60 min and then resuscitated with a 4% albumin solution. Alveolar liquid clearance was measured 5 h later as the concentration of protein in the distal air spaces over 1 h after instillation of a 5% albumin solution into one lu

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10

Woodward, Keith, Scott Wegryn, Carla Staruk, and Eric M. Nyberg. "The Dotter method revisited: early experience with a novel method of rapid internal carotid artery revascularization in the setting of acute ischemic stroke." Journal of NeuroInterventional Surgery 8, no. 4 (2015): 360–66. http://dx.doi.org/10.1136/neurintsurg-2014-011587.

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BackgroundTandem occlusive disease in the setting of acute ischemic stroke involving cervical and cerebral arteries has been associated with poor neurological outcome and poses significant challenges to neurointerventionists. Previously described endovascular methods typically involve carotid revascularization with stent placement prior to or following intracranial thrombectomy. Stent-based approaches, however, require the use of antiplatelet therapy which may increase the risk of hemorrhagic transformation. We describe a novel modified Dotter technique which may be used for carotid revascular

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11

Core, Ashley, Nathan Pinner, Brittany Bethea, and Jessica A. Starr. "Timing of Anticoagulation in Atrial Fibrillation Status Post Cardioembolic Stroke." Journal of Pharmacy Practice 33, no. 5 (2019): 612–17. http://dx.doi.org/10.1177/0897190019825577.

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Background: Anticoagulation is the mainstay of secondary stroke prevention in patients with atrial fibrillation; however, few studies have assessed the optimal timing for initiation of anticoagulation post cardioembolic stroke. In the 2 weeks following an acute cardioembolic stroke, the risk of recurrent stroke is as high as 8%, but this risk must be balanced against the risk of hemorrhagic transformation with early initiation of anticoagulation. Purpose: This study described the time to initiation of anticoagulation and evaluated the in-hospital incidence of hemorrhagic and ischemic complicat

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12

Verkerk, Brittany S., Karen Berger, and Christine A. Lesch. "Aminocaproic Acid for the Reversal of Alteplase: A Case Series." Journal of Pharmacy Practice 33, no. 6 (2019): 919–25. http://dx.doi.org/10.1177/0897190019840095.

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Introduction: The evidence to support the use of aminocaproic acid for reversal of alteplase is limited to case reports. Current guidelines recommend cryoprecipitate as first-line treatment, or an antifibrinolytic if cryoprecipitate is unavailable or cannot be used. This case series describes the use of aminocaproic acid for alteplase-related hemorrhage. Materials and Methods: Patients who received aminocaproic acid within 48 hours of alteplase from January 1, 2014, to June 30, 2017 were included. Patients were excluded if aminocaproic acid was not administered for an alteplase-related hemorrh

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13

Dietzen, Anton N., Anjum Sayyad, Aidan Pettit, and Susan L. Brady. "Poster 38 Anticoagulation: Hemorrhagic Conversion and VTE following Acute Ischemic Stroke." PM&R 8, no. 9 (2016): S173. http://dx.doi.org/10.1016/j.pmrj.2016.07.081.

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14

Parrilla, G., J. Zamarro, M. Espinosa De Rueda, et al. "Brain Edema after Carotid Revascularization: Benign Syndrome or Prelude to Hemorrhage?" Neuroradiology Journal 22, no. 4 (2009): 464–70. http://dx.doi.org/10.1177/197140090902200418.

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Brain edema (BE) post carotid revascularization (CR) is considered a benign presentation of the hyperperfusion syndrome. Its physiopathology has been related to production of vasogenic edema that tends to progressive spontaneous resolution. Its relation with post-CR hemorrhage remains unclear. Three cases of BE post-CR are described and compared to controls. Clinical evolution, medical management, and plain-CT and perfusion-CT (PCT) results are analysed. Two cases of BE had a benign course, with a progressive resolution of edema and disappearance of neurologic deficits. Effective blood pressur

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15

Johnson, Derek R., and Norbert G. Campeau. "Dual-Energy CT Differentiates Contrast Staining From Hemorrhagic Conversion of Ischemic Stroke." Neurohospitalist 8, no. 1 (2017): 42–43. http://dx.doi.org/10.1177/1941874417704754.

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16

Strouse, John J., Joshua Field, Regina D. Crawford, and Sophie Lanzkron. "Antecedent Transfusion and Primary Hemorrhagic Stroke in Adults with Sickle Cell Disease." Blood 112, no. 11 (2008): 1437. http://dx.doi.org/10.1182/blood.v112.11.1437.1437.

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Abstract Primary hemorrhagic stroke is an uncommon but serious complication of sickle cell disease (SCD) with mortality from 20 to 65%. Proposed risk factors include previous ischemic stroke, aneurysms, low steady-state hemoglobin, high steady-state leukocyte count, acute chest syndrome, and transfusion. We performed a retrospective case-control study to evaluate risk factors for primary hemorrhagic stroke in adults (age &gt;18 years) with SCD from Johns Hopkins and Barnes- Jewish Hospitals and Duke University Medical Center from January 1989 to April 2008. Cases had SCD and intraparenchym

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17

Giri, Richa, Faim Ahamed, Saurabh Agarwal, and Lalit Kumar. "Assessment of serum ferritin and thyroid hormones level in acute ischemic stroke and their association with hemorrhagic conversion." International Journal of Advances in Medicine 8, no. 5 (2021): 672. http://dx.doi.org/10.18203/2349-3933.ijam20211474.

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Background: Acute ischemic stroke is a significant cause of mortality and the leading cause of long-term disability in the United States. Collective evidence suggests that low T3 levels and serum ferritin levels instantly following acute ischemic stroke are connected with greater stroke sternness, higher death rates, and poor functional outcomes. The aim of the study was to assess the serum ferritin and thyroid hormones level in acute ischemic stroke their association with hemorrhagic conversion.Methods: In this observational study, 60 acute ischemic stroke patients aged ≥ 18 years of both gen

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18

Kulesh, A. A., L. I. Syromyatnikova, and S. Krapivin. "Experience with non-immunogenic staphylokinase for intravenous thrombolysis in ischemic stroke: analysis of hospital registry data." Meditsinskiy sovet = Medical Council, no. 3 (April 10, 2023): 24–29. http://dx.doi.org/10.21518/ms2023-057.

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Introduction. Intravenous thrombolysis (IT) is the primary method of reperfusion therapy for ischemic stroke. Over the past couple of years, there has been a global trend towards the predominant use of the bolus forms of thrombolytic drugs that is caused by necessity to increase the reperfusion therapy rate to achieve its best outcomes.Aim. To evaluate the efficacy and safety of the use of non-immunogenic staphylokinase for IT in ischemic stroke in real-world clinical practice of the regional vascular center.Materials and methods. The clinical outcomes of the cohort of 50 patients who underwen

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19

Walsh, Michael, Hemant Parmar, Suresh K. Mukherji, and Alexander Mamourian. "Developmental venous anomaly with symptomatic thrombosis of the draining vein." Journal of Neurosurgery 109, no. 6 (2008): 1119–22. http://dx.doi.org/10.3171/jns.2008.109.12.1119.

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Developmental venous anomalies (DVAs) are generally considered a benign and asymptomatic finding on CT and MR imaging. The authors report 2 cases of spontaneous thrombosis of the draining vein of a DVA depicted on CT and MR imaging. One patient presented with a nonhemorrhagic transient ischemia, which was successfully treated with anticoagulant therapy. The second patient presented with ischemia complicated by hemorrhagic conversion.

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20

Kayan, Yasha, Josser E. Delgado Almandoz, Jennifer L. Fease, Anna M. Milner, Jill M. Scholz, and Maximilian Mulder. "Efficacy of a two-test protocol for achieving a therapeutic response to clopidogrel prior to elective endovascular intracranial aneurysm treatment and an ‘induced’ postoperative hyper-response." Journal of NeuroInterventional Surgery 9, no. 8 (2016): 792–96. http://dx.doi.org/10.1136/neurintsurg-2016-012409.

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IntroductionVariable response to clopidogrel can impact perioperative risk in elective endovascular intracranial aneurysm treatment. The present study aims to determine the efficacy of a two-test protocol in reaching in-range preoperative P2Y12 reaction units (PRU) of 60–240 and the rate of postoperative conversion to hyper-response.MethodsA 17-day two-test protocol (with tests on days 10 and 17) for patients starting clopidogrel in anticipation of elective endovascular intracranial aneurysm treatment was introduced in February 2013 at our institution. Records for patients started on this prot

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21

Karanjia, Nariman D., Surjait M. Singh, Vicki Porter- Fink, Adam L. Widdison, and Howard A. Reber. "A study of the time course of conversion of edematous to hemorrhagic pancreatitis." International Journal of Pancreatology 8, no. 2 (1991): 133–39. http://dx.doi.org/10.1007/bf02924427.

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22

Basil, Gregory, Timur Urakov, Margarete Grace Knudsen, and Jacques Morcos. "Jugular Tubercle Meningioma with Hemorrhagic Conversion Mimicking a Ruptured Thrombosed Giant Vertebrobasilar Aneurysm." World Neurosurgery 119 (November 2018): 108–12. http://dx.doi.org/10.1016/j.wneu.2018.07.159.

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23

Kamal, Haris, Bijal K. Mehta, Muhammad K. Ahmed, et al. "Laboratory factors associated with symptomatic hemorrhagic conversion of acute stroke after systemic thrombolysis." Journal of the Neurological Sciences 420 (January 2021): 117265. http://dx.doi.org/10.1016/j.jns.2020.117265.

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24

King, Benjamin T., Patrick D. Lawrence, Truman J. Milling, and Steven J. Warach. "Optimal delay time to initiate anticoagulation after ischemic stroke in atrial fibrillation (START): Methodology of a pragmatic, response-adaptive, prospective randomized clinical trial." International Journal of Stroke 14, no. 9 (2019): 977–82. http://dx.doi.org/10.1177/1747493019870651.

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Rationale An estimated 15% of all strokes are associated with untreated atrial fibrillation. Long-term secondary stroke prevention in atrial fibrillation is anticoagulation, increasingly with non-vitamin K oral anticoagulants. The optimal time to initiate anticoagulation following an atrial fibrillation-related stroke that balances hemorrhagic conversion with recurrent stroke is not yet known. Aims To determine if there is an optimal delay time to initiate anticoagulation after atrial fibrillation-related stroke that optimizes the composite outcome of hemorrhagic conversion and recurrent ische

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25

Gilbert, Brian W., Laxmi Dhakal, Alison Lacy, and Joel B. Huffman. "Hemorrhagic conversion after alteplase administration in a patient with vasculitis and acute ischemic stroke." American Journal of Health-System Pharmacy 76, no. 4 (2019): 211–13. http://dx.doi.org/10.1093/ajhp/zxy048.

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Abstract Purpose A case of alteplase administration to a patient with vasculitis and acute ischemic stroke (AIS) is reported. Summary A 38-year-old woman with a recent diagnosis of granulomatosis with polyangiitis (GPA) received alteplase for AIS and developed symptomatic hemorrhagic conversion. Published reports regarding the safety of thrombolytic therapy in patients with a high inflammatory burden are inconsistent. The current case adds to the literature on the topic. Conclusion More data regarding alteplase treatment in patients with GPA are needed to further establish the safety of this t

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26

Roger Thomas, G., Harold Thibodeaux, Carol J. Errett, et al. "Limiting Systemic Plasminogenolysis Reduces the Bleeding Potential for Tissue-type Plasminogen Activators but not for Streptokinase." Thrombosis and Haemostasis 75, no. 06 (1996): 915–20. http://dx.doi.org/10.1055/s-0038-1650394.

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SummaryClinical experience suggests that thrombolytic-induced bleeding is associated with systemic activation of the thrombolytic system. Using fibrin specific variants of tissue-type plasminogen activator (t-PA) and making use of the apparent fibrin specificity of streptokinase (SK) in the rabbit we tested the hypothesis that minimizing systemic plasmin production and fibrinogenolysis will decrease hemorrhages in models of peripheral bleeding and embolic stroke. t-PA consumed 51% of the available fibrinogen; caused cerebral bleeds and increased peripheral bleeding time. Fibrin-specific varian

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27

Macacari, Beatriz, Beatriz Roberta da Silva, Maria Eduarda Ferreira Pereira, et al. "Hematological Biomarkers Associated with Stroke Types: A Clinical Cross-Sectional Analysis." Journal of Vascular Diseases 4, no. 2 (2025): 20. https://doi.org/10.3390/jvd4020020.

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Background: Stroke is a major cause of morbidity and mortality worldwide, with distinct pathophysiological mechanisms between ischemic stroke (IS) and hemorrhagic stroke (HS). Hematological parameters, such as lymphocyte and erythrocyte count, have been implicated in stroke prognosis, but their predictive value remains uncertain. Objective: To evaluate the association between hematological biomarkers and stroke subtypes (ischemic stroke and hemorrhagic stroke), and transient ischemic attack. Methods: This cross-sectional study analyzed clinical, metabolic, and hematological parameters in patie

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28

Klein Nulend, Rowan, Andrew Tse, and Timothy Pollitt. "Cholecystitis causing rupture of the cystic artery and massive hemoperitoneum." Journal of Case Reports and Images in Surgery 10, no. 2 (2024): 1–4. http://dx.doi.org/10.5348/100139z12rn2024cr.

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Introduction: Cholecystitis is a common surgical pathology; however, it is uncommonly associated with hemorrhage. Cases of hemoperitoneum secondary to cholecystitis are usually in the context of cystic artery pseudoaneurysms; however, no reports describe rupture of a non-aneurysmal cystic artery. Case Report: We present a case of hemoperitoneum and hemorrhagic shock secondary to cholecystitis, with resultant rupture of a branch of the cystic artery, in the absence of a pseudoaneurysm. The patient was a middle-aged man who presented with severe right upper quadrant pain on a background of bilia

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29

Kamdar, Aditi, Natalie Rintoul, David F. Friedman, James T. Connelly, Holly Hedrick, and Leslie Raffini. "Comparative Effectiveness of Different Strategies for Monitoring Neonates on Extra Corporeal Membrane Oxygenation (ECMO)." Blood 128, no. 22 (2016): 1007. http://dx.doi.org/10.1182/blood.v128.22.1007.1007.

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Abstract Background: ECMO can be life-savingin patients with cardiac or respiratory failure. Anticoagulation, typically with unfractionated heparin (uFH), is necessary to prevent the ECMO circuit from clotting. However, titrating the intensity of anticoagulation to maintain the balance between preventing thrombotic and hemorrhagic complications remains a challenge for providers. This is particularly true in neonates who are at the highest risk of intracranial hemorrhage, and in whom titrating uFH is notoriously difficult. In order to improve titration of anticoagulation, we instituted Enhanced

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30

Camões- Barbosa, Alexandre, Inês Mendes Ribeiro, and Luisa Medeiros. "Contralateral Upper Limb Weakness Following Botulinum Toxin A Injection for Poststroke Spasticity." Acta Médica Portuguesa 33, no. 11 (2020): 761. http://dx.doi.org/10.20344/amp.11503.

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Botulinum toxin type A has been approved for spasticity management in poststroke patients. The adverse effects are generally of two types: those related to local injection; and those related to the systemic effects from spread of the toxin. Contralateral weakness after botulinum toxin A treatment is a rarely reported adverse effect. We report the case of a 33-year-old female who had been receiving regular injections of incobotulinum toxin A due to spasticity of the right limbs after a hemorrhagic stroke. A switch was made to abobotulinum toxin A with an overall conversion ratio of 1:3.83. The

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Prabhu, Sudesh, Tom Karl, Jim Morwood, and Nelson Alphonso. "Temporary Single Ventricle Palliation for Severe Diastolic Dysfunction in a Biventricular Heart." World Journal for Pediatric and Congenital Heart Surgery 8, no. 3 (2016): 404–7. http://dx.doi.org/10.1177/2150135116635145.

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We describe a neonate with severe left ventricular (LV) dysfunction manifesting soon after birth, who required extracorporeal life support (ECLS). Multiple attempts at separation from ECLS resulted in high left atrial pressures and hemorrhagic pulmonary edema. The inability to separate from ECLS after ten days prompted an unconventional treatment strategy. We performed a modified Norwood operation for LV rehabilitation on day 10 of ECLS. This was followed by successful separation from ECLS. Left ventricular function recovered, and the child underwent conversion to a biventricular circulation o

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Igor, B. Kovalenko* Zhanna Yu. Chefranova Nadezhda S. Zueva Vyacheslav D. Polyansky. "APPLICATION OF SELECTIVE THROMBOLYTIC THERAPY FOR ISCHEMIC STROKE TREATMENT." Indo American Journal of Pharmaceutical Sciences 04, no. 10 (2017): 3727–31. https://doi.org/10.5281/zenodo.1019479.

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The article deals with the review and analysis of the major study related to both applications of STLT and that compared to systemic thrombolysis which demonstrate good functional results after 90 days of STLT application and less number of intracranial hemorrhage during 24 hours. The comparative analysis of the outcomes of ischemic stroke treatment based on systemic and selective thrombolysis by Actilyse was conducted in Belgorod Regional Clinical Hospital. The procedure of selective thrombolysis was described. The analysis of the effectiveness and complications depending on medication dose a

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33

Prince, E. A., M. V. Jayaraman, T. Schirmang, and R. Haas. "Angiographically documented hemorrhagic conversion of a left middle cerebral artery embolic stroke during intra-arterial thrombolysis." Journal of NeuroInterventional Surgery 3, no. 3 (2010): 246–48. http://dx.doi.org/10.1136/jnis.2010.003459.

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34

Pergakis, Melissa B., Wan-Tsu W. Chang, Ali Tabatabai, et al. "Simulation-Based Assessment of Graduate Neurology Trainees' Performance Managing Acute Ischemic Stroke." Neurology 97, no. 24 (2021): e2414-e2422. http://dx.doi.org/10.1212/wnl.0000000000012972.

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Background and ObjectivesMultidisciplinary acute stroke teams improve acute ischemic stroke management but may hinder trainees' education, which in turn may contribute to poorer outcomes in community hospitals on graduation. Our goal was to assess graduate neurology trainee performance independently of a multidisciplinary stroke team in the management of acute ischemic stroke, tissue plasminogen activator (tPA)–related hemorrhage, and cerebral herniation syndrome.MethodsIn this prospective, observational, single-center simulation-based study, participants (subinterns to attending physicians) m

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35

Sweid, Ahmad, Batoul Hammoud, Kimon Bekelis, et al. "Cerebral ischemic and hemorrhagic complications of coronavirus disease 2019." International Journal of Stroke 15, no. 7 (2020): 733–42. http://dx.doi.org/10.1177/1747493020937189.

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Background The coronavirus disease 2019 is associated with neurological manifestations including stroke. Objectives We present a case series of coronavirus disease 2019 patients from two institutions with acute cerebrovascular pathologies. In addition, we present a pooled analysis of published data on large vessel occlusion in the setting of coronavirus disease 2019 and a concise summary of the pathophysiology of acute cerebrovascular disease in the setting of coronavirus disease 2019. Methods A retrospective study across two institutions was conducted between 20 March 2020 and 20 May 2020, fo

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Andreev, V. V., A. Yu Podunov, D. S. Lapin, et al. "Evaluation of hemostasis clinical and laboratory indicators of patients with stroke and coronavirus infection (COVID-19) on the basis of a multidisciplinary hospital in the period of its conversion to an infectious diseases hospital." Regional blood circulation and microcirculation 21, no. 3 (2022): 40–46. http://dx.doi.org/10.24884/1682-6655-2022-21-3-40-46.

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Introduction. The COVID-19 impact on hemostasis of stroke survivors with community-acquired pneumonia is an urgent problem. The aim of the study is to analyze the features of clinical and laboratory parameters in cerebral stroke combined with community-acquired pneumonia caused by the SARS-Co-V-2 virus. Materials and methods. The instrumental and laboratory examination results of 88 patients aged 73.0 (12.3) years in the acute period of stroke with community-acquired viral pneumonia symptoms were analyzed. The present study included 39.8 % (n=53) male and 60.2 % female (n=35) with duration of

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Ojeda, Chris, Nitya Chitravanshi, Patrick C. Reid, Charles J. Prestigiacomo, Ennis J. Duffis, and Chirag D. Gandhi. "Abstract TP132: Decompressive Hemicraniectomy Improves Survival in Malignant Middle Cerebral Artery Stroke Patients with Hemorhagic Conversion Admitted with a High Glasgow Coma Scale." Stroke 44, suppl_1 (2013). http://dx.doi.org/10.1161/str.44.suppl_1.atp132.

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Decompressive hemicraniectomy (DHC) has been shown to improve clinical outcome in cases of malignant infarct but its indications have not been well classified. This study focuses on patients who suffered malignant middle cerebral artery (MMCA) infarct with subsequent hemorrhagic conversion and were admitted with mild brain injury (initial Glasgow Coma Scale≥13). Survival rates of MMCA infarct patients with hemorrhagic conversion who underwent DHC were compared with those treated with medical management only. Hypothesis: Patients admitted for MMCA infarct with a Glasgow Coma Scale ≥13 who devel

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Burgess, Richard, Esteban Cheng Ching, Delora Wisco, et al. "Abstract 177: Absent Collateral Flow on CT Angiogram Predicts Acute Hemorrhagic Transformation in Ischemic Stroke." Stroke 44, suppl_1 (2013). http://dx.doi.org/10.1161/str.44.suppl_1.a177.

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Background: In patients with a large vessel occlusion, the degree of collateral vascular supply to an ischemic territory has been shown to be a predictor of stroke outcome. Prior studies have focused on the correlation between collateral flow measured on conventional digital subtraction angiography and outcome measures, including the presence of hemorrhagic conversion. CT/CTA is more widely available and more quickly accomplished than MR or conventional angiography. In this work we demonstrate that the absence of CT angiographic collaterals predicts hemorrhage transformation in acute ischemic

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Iwuchukwu, Ifeanyi, Tommy Chan, Arash Afshinnik, Harold Mcgrade, and Vivek Sabharwal. "Abstract T P62: The Presence of Cerebral Microhemorrhage is Not Associated With Hemorrhagic Conversion Following Post Thrombolysis for Acute Ischemic Stroke." Stroke 45, suppl_1 (2014). http://dx.doi.org/10.1161/str.45.suppl_1.tp62.

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Introduction: Hemorrhagic conversion remains an unpredictable complication of thrombolysis for acute ischemic stroke. Cerebral microhemorrhages (CMH) have been associated with intracerebral hemorrhage (ICH) and may serve as a biomarker for hemorrhagic conversion of acute ischemic stroke treated with tissue plasminogen activator. We sort to determine the relationship of the presence of CMH and hemorrhagic conversion of acute ischemic stroke treated with intravenous thrombolysis. Method: Using our institutional stroke registry, we identified patients with acute ischemic stroke treated with intra

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Zubair, Adeel S., and Kevin N. Sheth. "Hemorrhagic Conversion of Acute Ischemic Stroke." Neurotherapeutics, April 21, 2023. http://dx.doi.org/10.1007/s13311-023-01377-1.

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won, soonmi, Iqbal Sayeed, Bushra Wali, and Donald G. Stein. "Abstract TMP33: Progesterone Prevents Hemorrhagic Transformation after tPA Treatment in Experimental Stroke." Stroke 44, suppl_1 (2013). http://dx.doi.org/10.1161/str.44.suppl_1.atmp33.

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BACKGROUND: Tissue plasminogen activator (tPA) is the only FDA-approved treatment for acute stroke. Unfortunately, because of its narrow time window and high risk-to-benefit ratio it is used in less than 3.0% of stroke patients. Our group and others have shown progesterone (PROG) to be beneficial in several ischemic brain injury models. However, before PROG can be tested in stroke patients, how it will interact with tPA, and whether combination with PROG will increase or decrease the risk of bleeding and hemorrhagic conversion, should be determined. In the present study we examined whether PRO

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Bonaca, Marc P., Benjamin M. Scirica, Eugene Braunwald, et al. "Abstract 19144: New Ischemic Stroke and Outcomes with Vorapaxar vs. Placebo: Results from TRA 2°P-TIMI 50 Trial." Circulation 126, suppl_21 (2012). http://dx.doi.org/10.1161/circ.126.suppl_21.a19144.

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Vorapaxar is a potent platelet inhibitor that reduces thrombotic events in stable patients with a history of myocardial infarction (MI) or peripheral artery disease (PAD); however, this benefit is offset by increased bleeding including intracranial hemorrhage particularly in patients with established cerebrovascular disease (CVD). We investigated the outcomes in patients with MI or PAD experiencing a first ischemic stroke on vorapaxar. METHODS: The TRA2P-TIMI 50 trial was a randomized, double-blind, placebo controlled trial of vorapaxar 2.5 mg daily in 26,449 patients with established atherosc

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Ho, Huy, Ahmed Eddib, Mohammed Ahmed, Saramarina Sanchez, Suman Pasupuleti, and Nishant K. Nerella. "Abstract 295: Anti-coagulation Choice in Patient with Atrial Fibrillation and Acute Ischemic Stroke." Arteriosclerosis, Thrombosis, and Vascular Biology 43, Suppl_1 (2023). http://dx.doi.org/10.1161/atvb.43.suppl_1.295.

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To prevent embolic stroke, patients with atrial fibrillation are risk stratified, and started on anticoagulation accordingly. This poses a management challenge when these patients present to the hospital with ischemic stroke. Often, anticoagulation is held due to risk of hemorrhagic conversion. The optimal choice of anticoagulation to reduce the risk of hemorrhagic transformation of ischemic stroke in patients with atrial fibrillation is poorly studied. This study evaluates the risk of hemorrhagic conversion between different anticoagulation therapies in patients with atrial fibrillation who p

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Marquardt, Robert J., Sung-Min Cho, Lucy Zhang, Prateek Thatikunta, Ken Uchino, and Dolora Wisco. "Abstract TP223: Characterizing Ischemic Stroke and Hemorrhagic Conversion in Infectious Endocarditis." Stroke 48, suppl_1 (2017). http://dx.doi.org/10.1161/str.48.suppl_1.tp223.

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Introduction: Ischemic stroke is a common complication of infective endocarditis (IE) and can delay valve surgery. Identifying risk factors for acute ischemic stroke (AIS) and hemorrhagic conversion may help in perioperative risk assessment of these patients. Methods: Retrospective analysis was done on 116 consecutive patients with IE seen by stroke neurology at a tertiary center from January 2015 through July 2016. Clinical and radiographic characteristics were collected in a population whose initial evaluation was for acute stroke management or preoperative risk evaluation. Descriptive stati

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Dittmar, Erika, Thomas Wolfel, Lourdes Menendez, et al. "Conversion From Intravenous Alteplase to Tenecteplase for Treatment of Acute Ischemic Stroke Across a Large Community Hospital Health System." Annals of Pharmacotherapy, January 23, 2023, 106002802211494. http://dx.doi.org/10.1177/10600280221149409.

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Background: Recent evidence suggests tenecteplase at an intravenous dose of 0.25 mg/kg is as safe and efficacious as intravenous alteplase standard dose and demonstrates a more favorable pharmacokinetic profile for treatment of acute ischemic stroke. Objective: The purpose was to compare the safety and efficacy of alteplase versus tenecteplase for the treatment of acute ischemic stroke at a large community hospital health system following conversion in the preferred formulary thrombolytic. Methods: Prior to converting, medication safety and operationalization analyses were conducted. A multice

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Leykina, Liza A., Christine K. Fox, Nancy K. Hills, et al. "Abstract 89: Incidence and Characteristics of Hemorrhagic Stroke Among Post-STOP Participants." Stroke 51, Suppl_1 (2020). http://dx.doi.org/10.1161/str.51.suppl_1.89.

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Introduction: The Stroke Prevention Trial in Sickle Cell Anemia (STOP) changed standards of care for stroke screening and treatment of high-risk children with sickle cell anemia (SCA), reducing the risk of ischemic stroke. However, the incidence of hemorrhagic stroke in young patients with SCA in the post-STOP era remains poorly characterized. Methods: The Post-STOP multicenter cohort study collected follow-up data from prior participants (all with SCA) of the STOP or STOP II clinical trials. From 01/2012 – 05/2014, medical records analysts abstracted clinical, imaging and laboratory data coll

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Ghoshal, Shivani, and Nada El Husseini. "Accelerated mineralization as a mimic of hemorrhagic conversion." Neurology: Clinical Practice, October 30, 2019, 10.1212/CPJ.0000000000000764. http://dx.doi.org/10.1212/cpj.0000000000000764.

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Portela de Oliveira, Eduardo, Santanu Chakraborty, Mihilkumar Patel, Stefanos Finitsis, and Daniela Iancu. "Value of high-density sign on CT images after mechanical thrombectomy for large vessel occlusion in predicting hemorrhage and unfavorable outcome." Neuroradiology Journal, December 7, 2020, 197140092097525. http://dx.doi.org/10.1177/1971400920975259.

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Purpose Cerebral hyperdensities can appear on head computed tomography (CT) images performed early after endovascular treatment (EVT) in patients with acute ischemic stroke and may be secondary to contrast staining or hemorrhagic transformation. The aim of this study was to determine how the high-density sign on CT affects mortality and clinical outcome and whether CT parameters predict hemorrhagic conversion or unfavorable outcome. Methods We retrospectively reviewed a database of patients who underwent EVT with mechanical thrombectomy for acute ischemic stroke over 7 years. Included were acu

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Pham, Ly, Sydney O’Connor, Karen Yarbrough, et al. "Abstract TMP22: Intravenous Glyburide Treatment is Associated with Reduced Matrix Metalloproteinase-9 in Human Acute Stroke." Stroke 44, suppl_1 (2013). http://dx.doi.org/10.1161/str.44.suppl_1.atmp22.

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Background: Elevated matrix metalloproteinase-9 (MMP-9) following acute ischemic stroke is associated with blood-brain barrier breakdown and hemorrhagic conversion. Prior retrospective evidence suggests that sulfonylurea use may be associated with reduced risk of hemorrhagic conversion. We hypothesized that sulfonylureas may reduce MMP-9 level in stroke patients. Methods: Using serial plasma samples from six subjects in the Glyburide Advantage in Malignant Edema and Stroke Pilot trial (GAMES-Pilot), we evaluated the level of MMP-9 in human subjects presenting with large hemispheric stroke who

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Lin, Yu-Ting, and Min-Po Ho. "Tranexamic acid for treatment of tenecteplase related hemorrhagic conversion." American Journal of Emergency Medicine, July 2022. http://dx.doi.org/10.1016/j.ajem.2022.07.045.

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