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Добірка наукової літератури з теми "Immune health"

Автор: Grafiati

Опубліковано: 10 січня 2023

Оновлено: 28 січня 2023

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Статті в журналах з теми "Immune health"

Maharramova, Sevinc, Mahbuba Veliyeva, Mahira Amirova, Ulviyya Azizova, Ulker Majidova, Huseyn Abıyev, Gulnara Dashdamirova, and Farah Mammadova. "Surrounding Plants as Reliable Immune Boosters." Health 14, no. 11 (2022): 1105–13. http://dx.doi.org/10.4236/health.2022.1411078.

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Hoseinzadeh, Fatemeh, Porya Hassan Abadi, Mehdi Agheltar, Arvin Aghayinejad, Farnaz Torabian, Arash Akhavan Rezayat, Farzad Akbarzadeh, and Hamid Reza Rahimi. "The Role of Immune System in Depression Disorder." Health 08, no. 15 (2016): 1726–43. http://dx.doi.org/10.4236/health.2016.815167.

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Ruan, Tao, Lingjun Li, Xi Peng, and Bangyuan Wu. "Effects of Methionine on the Immune Function in Animals." Health 09, no. 05 (2017): 857–69. http://dx.doi.org/10.4236/health.2017.95061.

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Yan, Fang, and D. B. Polk. "Probiotics and immune health." Current Opinion in Gastroenterology 27, no. 6 (November 2011): 496–501. http://dx.doi.org/10.1097/mog.0b013e32834baa4d.

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Rani, Usha. "Breastfeeding: Importance in Early Development of the Immune System and Long-term Health." Journal of Communicable Diseases 52, no. 02 (June 30, 2022): 107–9. http://dx.doi.org/10.24321/0019.5138.202280.

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Breastfeeding supplies the baby and infant with unparalleled natural nutrients. Human breast milk also has several antimicrobial agents and may influence immune system development, as evidenced by prior research on newborn immunisation response and thymus gland development. Human milk is a dynamic supply of nutrients and bioactive ingredients and promotes the healthy growth and development of the human newborn. Infants are more susceptible to infection because their developing immune systems have a number of weaknesses. This review focuses on the direct effect of human milk on innate immunity in infants. Numerous new studies have made the multi-functionality of the bioactive components of human milk very clear. Our knowledge of the potential positive effects of human milk on infants has increased. These effects are not achievable with milk formulae. Human milk contains antimicrobial proteins and peptides that have a broader involvement in innate immune defence than previously thought. A complex combination of the anti-inflammatory and antioxidative substances that human milk supplies to the intestine results in a special environment of improved immune defence with reduced inflammation.

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Minton, Kirsty. "Linking immune and emotional health." Nature Reviews Immunology 13, no. 9 (August 23, 2013): 617. http://dx.doi.org/10.1038/nri3529.

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Liu, Baochi, Meng Wang, Jinsong Su, Yanzheng Song, Li Liu, and Lei Li. "Correlation analysis of compromised immune function with perioperative sepsis in HIV-positive patient." Health 04, no. 04 (2012): 190–95. http://dx.doi.org/10.4236/health.2012.44028.

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Wei, Xiangdong, Bin Zhang, and Baojun Pan. "MMP1 Is a Prognostic-Related Biomarker and Correlated with Immune Infiltration in Breast Cancer." Health 14, no. 02 (2022): 219–35. http://dx.doi.org/10.4236/health.2022.142017.

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Dietert, Rodney, and Judith Zelikoff. "Pediatric Immune Dysfunction and Health Risks Following Early-Life Immune Insult." Current Pediatric Reviews 5, no. 1 (February 1, 2009): 36–51. http://dx.doi.org/10.2174/157339609787587591.

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Chandok, Meena, Palanikumaran Sakthivel, Charles Chaney, Michael Mccormack, Kathryn Wagner, Gary Mire, John Newby, Anil Parwani, and Kathleen Renee. "Novel early detection test for breast cancer and its recurrence in blood through changes in immuno-biochemical signals (46.12)." Journal of Immunology 188, no. 1_Supplement (May 1, 2012): 46.12. http://dx.doi.org/10.4049/jimmunol.188.supp.46.12.

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Abstract We are developing an immune sensor for early detection of breast cancer and its recurrence in blood, based on identifying the earliest specific changes/alterations in the immuno-biochemical signals in different immune components that are distinctly different from those in a healthy immune system. The screening of different immuno-biochemical signals in their native and altered forms in different immune components/cells led to identification of a novel T cell population. Analysis of this T cell population and its immuno-biochemical signals on blood samples from the 4 groups - (i) healthy, (ii) recently diagnosed with invasive breast cancer stage 1{before the onset of treatment/surgery}, (iii) breast cancer treated individuals who had incidence of recurrence/relapse {BCR}, and (iv) breast cancer treated individuals who were disease free {BDF}- was done using multi-parameter flow cytometry. Sixteen molecules, assessed as the most likely candidates for sensing disruptions in the immune pathway, were used in developing a matrix. Care was taken to separate subjects that could confound the immuno-biochemical signals, potentially arising from other immune disorders. The method designed is different from conventional approaches and has the potential to be used as a cost-effective assessment to predict health status.

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Дисертації з теми "Immune health"

Shbat, Layla. "Immune modulation in cardiovascular disease." Thesis, McGill University, 2011. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=103617.

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The importance of the adaptive immune response in cardiovascular disease has been increasingly appreciated. However, limited information is available on immune modulation in the context of hypertension and atherosclerosis. In order to fill this knowledge gap, the amount of T regulatory (Treg) cells was determined by flow cytometry on cells from the spleen and the aorta in two murine models, namely angiotensin (Ang) II-induced hypertension (HT) and endothelin-1 (ET-1)-exacerbated high fat diet (HFD)-induced atherosclerosis in apolipoprotein E knockout (apoE-/-). Two groups of mice were studied. In the first study, 12-week old male C57BL/6 mice were infused with Ang II (1 µg/kg/min, s.c.) for 14 days via an osmotic pump or implanted with a dummy pump. In the second study, 8-week old C57BL/6 male transgenic mice with endothelium-restricted preproendothelin-1 (eET-1) overexpression, apoE-/-, eET-1/apoE-/- crosses, and wild type (WT) mice were fed a HFD or a normal diet (ND) for 8 weeks. A trend towards an increase in several T lymphocyte subpopulations including natural (CD4+CD25+Foxp3+) Tregs was observed in the spleen of mice infused with Ang II whereas in aorta natural Tregs tended to decrease. In atherosclerosis, an increase in classical (CD4+CD25+) Tregs was observed in the spleen of eET-1. HFD reduced the Treg content in the spleen of both WT and eET-1. In addition, HFD tended to increase natural Tregs in eET-1/apoE-/- crosses. In aorta, HFD increased classical Tregs and tended to increase natural Tregs in eET-1 whereas it tended to decrease natural Tregs in eET-1/apoE-/- crosses. The lack of significant change in the above studies limits drawing conclusions. However, the results suggest that ET-1 and HFD have an impact on Treg populations in the spleen and aorta. Additional animals and/or refinement in the techniques could lead to more definitive conclusions.
Le rôle de la réponse immunitaire adaptative dans l'hypertension et l'athérosclérose commence à être apprécié. Cependant, il n'est pas clair que les lymphocytes T régulateurs (Tregs) jouent un rôle dans ces deux pathologies. Dans le but d'éclaircir le rôle de ces lymphocytes, le contenu en Tregs a été déterminé à l'aide de cytométrie de flux dans la rate et l'aorte de deux modèles murins, l'hypertension induite par l'angiotensine (Ang) II et l'athérosclérose induite par une diète riche en gras (DRG) dans des souris knockout pour l'apolipoprotéine E (apoE-/-) exagérée par la surexpression de l'endothéline (ET)-1. Deux groupes de souris ont été étudiés. Dans le premier groupe, des souris mâles C57BL/6 de 12 semaines ont été infusées ou pas avec de l'Ang II (1 µg/kg/min, s.c.) pendant 2 semaines. Dans le second groupe, des souris mâles C57BL/6 de 8 semaines transgéniques surexprimant l'ET-1 dans les cellules endothéliales (eET-1), apoE-/-, eET-1/apoE-/- et sauvages (WT) ont été nourries avec une DRG ou une diète normale (DN) pendant 8 semaines. Les souris infusées avec l'Ang II présentaient une tendance à l'augmentation de plusieurs sous-populations de lymphocytes T incluant les Tregs naturels (CD4+CD25+Foxp3+) dans la rate. Par contre, au niveau de l'aorte les Tregs naturels tendaient à diminuer. Dans l'étude de l'athérosclérose, une augmentation des Tregs (CD4+CD25+) a été observée dans la rate des souris eET-1. La DRG a réduit le contenu de Tregs dans la rate des souris WT et eET-1 et tendait à accroître les Tregs naturels dans la rate des eET-1/apoE-/-. Au niveau de l'aorte, la DRG a augmenté les Tregs et tendait à accroître les Tregs naturels dans les eET-1 et tendait à diminuer ces lymphocytes dans les eET-1/apoE-/-. Le manque de changements significatifs limite la possibilité de tirer des conclusions. Cependant, les résultats suggèrent que l'ET-1 et la DRG ont un impact sur la population de Tregs dans la rate et l'aorte. Des animaux additionnels et/ou un raffinement des techniques pourraient donner des résultats plus définitifs.

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Liu, Yuhong. "Sigma Receptors and the Immune System /." The Ohio State University, 1995. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487930304687004.

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Esplin, Brandt L. "Replenishment of innate immune system in health and disease." Oklahoma City : [s.n.], 2009.

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Cho, Sungyoo. "Immune evasion of CD1d molecules and NKT cells in the innate immune response against viruses." [Bloomington, Ind.] : Indiana University, 2005. http://gateway.proquest.com/openurl?url_ver=Z39.88-2004&res_dat=xri:pqdiss&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&rft_dat=xri:pqdiss:3185405.

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Thesis (Ph.D.)--Indiana University, 2005.
Source: Dissertation Abstracts International, Volume: 66-08, Section: B, page: 4071. Chair: Randy R. Brutkiewicz. Title from dissertation home page (viewed Oct. 10, 2006).

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Hassan-Zahraee, Mina. "Anergy and the human skin immune system." Thesis, McGill University, 1996. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=42051.

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An initial study comparing cytokine gene expression in the skin of control vs. anergic patients lacking delayed type hypersensitivity reactivity revealed no difference; but disclosed an apparent absence of detectable CD3+ T cells in the skin of anergic individuals. To assess its significance for anergy, an investigation of the role of skin T cells in DTH-reactive healthy individuals was undertaken. To do so, the phenotypic and functional characteristics of T cells isolated from skin and blood were compared. Analysis by flow cytometry has shown that 74% of skin T cells expressed cell surface HLADR, 66% were positive for the IL-2 receptor CD25, and less than 43% have displayed the VLA integrin $ alpha$4 chain as compared to 28%, 7%, 79% for peripheral blood mononuclear cells respectively. The expression of a cutaneous lymphocyte antigen (CLA) was 61% in the former and 14% in the latter. Functionally, skin T cells failed to proliferate in response to all ligands including IL-2, anti-CD3, lectins and phorbol esters with ionomycin, as well as showed a reduced Ca++ flux to phytohemagglutinin. Skin tissue co-cultured with autochtonous PBL could inhibit its proliferative reaction. Despite their ability to proliferate, lymphocytes from skin were shown to be able to produce IFN$ gamma$ in response to PHA+IL-12 as well as anti-CD3+IL-2. Inhibition by anti-cytokine mAbs revealed that in both instances IL-12 was obligatory for this production. In an additional study it was established that a hitherto uncharacterized subset of T cells in blood which could secrete IFN$ gamma$ consisted of CLA+ cells. This observation established a functional link between these CLA+ skin-seeking T cells and the CLA+ T cells in skin.
A major difference between IFN$ gamma$-producing cells from blood and skin was found to be the tempo of synthesis: whereas, PBMC was first detected to contain IFN$ gamma$ 42 hours following activation, lasting for days, skin cells were positive after 2.5 hrs of activation, (or 16x faster) for a duration of only 90 minutes. These kinetics were confirmed using intact skin in culture. Experiments designed to reveal the mechanism of this fast action have shown that mRNA for IFN$ gamma$ is present in unstimulated isolated skin T cells as well as in intact skin, but not in PBMC, and its presence may be attributed to ongoing constitutive transcription. Activation of skin T cells, which has been shown to elicit prompt translation in IFN$ gamma$ synthesis has also been shown, at the same time, to terminate IFN$ gamma$ gene transcription in an apparently selective manner. Accordingly, it can be seen that the amount of IFN$ gamma$ synthesized in skin and the duration of its synthesis is preprogrammed. This mode of regulation may be unique to the skin, and unique for IFN$ gamma.$
The results presented are interpreted to indicate that r cells present in human skin may play an essential role in the DTH response, and provide evidence for "peripheral sensitization", or lymphocyte activation outside organized lymphoid tissue. Because of its speed, it may represent the antigen-specific component of a first line cutaneous host defence system. The absence of such T cells in the skin of anergic patients may indeed be responsible for a lack of DTH reactivity, and its clinical consequences.

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Singh, Rekha. "Cellular immune responses in HSV and CMV infections." Thesis, University of Ottawa (Canada), 2003. http://hdl.handle.net/10393/29036.

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The herpes simplex virus (HSV) and cytomegalovirus (CMV) are the prominent members of the Herpesviridae family collectively responsible for the majority of herpes-related morbidity and mortality in humans. These viruses are therefore the subjects of this study that was undertaken to improve our understanding of the nature of immune response and the mechanism of viral modulation leading to suppression of viral replication or evasion of the host immune response in vivo. The present study has examined the T helper responses to HSV-2 and murine CMV (MCMV) and provides new insights into the nature and the modulation of the T helper responses by these viruses in vivo. B7-1/B7-2 costimulation of T cells by antigen-presenting cells is essential for T cell activation by antigen. HSV-2 infection affects the expression of both B7-1 and B7-2 on monocytes, the key antigen presenting cells in vivo, potentially in two ways with opposing outcomes. It abrogates or diminishes the IFN-gamma-upregulation of B7-1 (in 8 out of 9 patients) and B7-2 (in 6 out of 9 patients) on monocytes. However, the infection also augments the expression of B7-1 and B7-2 on monocytes through an IFN-gamma-independent mechanism (in 9 out of 9 patients). Although the clinical significance of these opposing effects is presently unclear, these may be related to the immunological mechanism or strategy leading to recurrent disease in immunocompetent hosts. Like HSV-2, infections with MCMV in mice also led to a predominant Thl type immune response characterized by high levels of IFN-gamma and low IL-4 production. Studies with specific MCMV mutants, containing Tn3 transposon in the open reading frame of M25, M27, M43, or m09 gene, led to the identification of M43 gene that specifically suppresses IL-4 response. The Tn3 disruption of M43, but not M25, M27 or m09, gene of MCMV led to a strong IL-4 response (p = 0.0002) despite the presence of a dominant IFN-gamma response. These results provide insight into the possible role of a herpesvirus gene that can profoundly modulate the nature of T helper response in vivo. The presence of a homologous genetic element in other herpesvirus genomes may explain the dominant Th1 immune response triggered by HSV-2 and possibly other herpesviruses. The obvious importance of this finding, beyond herpesvirus immunopathogenesis, lies in the ability of M43 and homologous genes to globally modulate the nature of cytokine response in vivo and suppress Th2 cytokine-mediated diseases. (Abstract shortened by UMI.)

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Shey, Muki Shehu. "Determinants of innate immune responses to mycobacteria." Doctoral thesis, University of Cape Town, 2012. http://hdl.handle.net/11427/10986.

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Includes bibliographical references.
Innate cells such as macrophages, monocytes, myeloid dendritic cells and granulocytes recognise mycobacteria and initiate immune responses such as phagocytosis, cytokine production and expression of maturation markers. The type and magnitude of innate responses to mycobacteria may determine the subsequent adaptive responses generated. Our aims were to determine maturational changes in innate immune responses to mycobacteria over the first 9 months of life, and to assess effects of genetic variations in toll-like receptors on host responses to mycobacteria. This knowledge is important for designing rational strategies for vaccination against tuberculosis.

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Wechsler, Daniel Steven Gary. "Immune mechanisms of cure in Trypanosoma musculi infection." Thesis, McGill University, 1987. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=75348.

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Trypanosoma musculi is a protozoan parasite which produces a characteristic, self-limiting murine infection of approximately three weeks duration; the infection comprises a growth phase, a plateau phase and an elimination phase. Following clearance of parasitaemia, a mouse is cured and immune to reinfection. The present studies examine the immune mechanisms which operate during the elimination phase.
Passive transfer of plasma from an immune mouse to an infected recipient brings about rapid and complete clearance of parasitaemia in C57BL/6 mice. This curative activity is labile to heat treatment for 30 minutes at 56$ sp circ$C. A protein A- derived immunoglobulin fraction of immune plasma (IP) shares these properties. Further purification shows that the curative activity resides primarily in the IgG2a subclass, and that this antibody is intrinsically heat-labile. Complement component C3 (but not the lytic C5-C9 sequence) is necessary for antibody-mediated cure of infection. Cellular elements (macrophages) are also essential for elimination of parasitaemia to occur. The ultimate T. musculi effector mechanism thus requires the interaction of both humoral and cellular components.

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Aziz, Douglas C. "Molecular studies on murine acquired immune deficiency syndrome (MAIDS)." Thesis, McGill University, 1990. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=74558.

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The Duplan strain of Radiation Leukemia Virus (RadLV) induces an AIDS-like syndrome when injected intraperitoneally into adult C57BL/6 mice. The mice develop T cell depletion, polyclonal B cell proliferation (lymphadenopathy, splenomegaly), hypergammaglobulinemia, and immune deficiency. This syndrome has recently been designated MAIDS (murine acquired immunodeficiency syndrome). This virus preparation, passaged in vivo, is crude and known to contain different strains of retroviruses. To identify the etiologic agent of this disease, we first infected fibroblasts in vitro with this crude virus mixture. Supernatants of these cultures induced disease. Unintegrated viral DNA from fibroblasts newly-infected with this virus preparation was extracted by the Hirt supernatant method and analysed with various viral probes: 8.8 and 4.8 kb DNA species were detected and molecularly cloned in pUC 18 (clones Du9S and Du5H, respectively). An unique DNA sequence derived from the gag region of the defective Du5H genome did not hybridize to various other retroviral DNA's (Moloney MuLV, N-Cl-35 or Du9S). The restriction of Du9S is similar to the map of other RadLV's. The defective Du5H DNA was transfected into fibroblasts in the presence of the neomycin resistance gene and rescued with the non-leukemogenic ecotropic RadLV (G6T2). This virus complex, as well as the ecotropic virus alone, were inoculated into C57BL/6 mice to test their pathogenic potential. The rescued cloned defective virus caused MAIDS, whereas the helper virus (G6T2) alone did not cause disease. Sequence data shows that the defective virus has large deletions in pol and env, and that the gag region is conserved and harbors a novel p12 sequence. This mouse model may help in understanding some important mechanisms underlying the biology of retrovirus-induced immunodeficiency syndromes, such as AIDS.

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Tran, Elise H. "Immune invasion and glial activation in experimental autoimmune encephalomyelitis." Thesis, McGill University, 2000. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=36845.

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Leukocyte recruitment into tissues in response to infection or injury is a crucial event for the elimination of pathogens to protect the host. However, when leukocytes invade the central nervous system (CNS) and neuromflammatory disorders result, neurological function may be compromised. Infiltration of the CNS, predominantly by T cells and macrophages, characterizes Multiple Sclerosis and its animal counterpart, Experimental Autoimmune Encephalomyelitis (EAE).
Autoreactive T cells that initiate EAE produce Th1 cytokines (e.g., IFNgamma, TNFalpha). Nevertheless, previous studies also indicated an unnecessary or even protective role for IFNgamma in EAE. I have identified a novel role for IFNgamma in my studies using IFNgamma- or IFNgammaR-knockout mice. IFNgamma promotes the expression of the chemokines RANTES, MIP-1alpha, and MCP-1, which recruit mononuclear cells in the CNS to induce a non-lethal remitting EAE. Without IFNgamma, the chemokines MIP-2 and TCA-3, and polymorphonuclear leukocytes prevail, producing an unusually lethal EAE. MIP-1alpha is, however, dispensable in recruiting mononuclear cells, as EAE could still be induced in mice deficient in MIP-1alpha or its CCRS receptor.
To examine how much T cells depend on the cooperation with macrophages in the CNS to induce EAE, selective depletion of peripheral macrophages in mice was achieved by intravenous administration of clodronate-loaded liposomes. Treated mice showed no clinical signs of EAE following adoptive transfer of myelin-reactive T cells, but an altered distribution of leukocytes. These leukocytes were confined within the perivascular or meningeal space, not invading the CNS parenchyma. Levels of TNFalpha and inducible nitric oxide synthase (iNOS) in the CNS were reduced in these asymptomatic macrophage-depleted mice compared to untreated mice with EAE. In these asymptomatic mice, NOS expression was restricted to parenchymal astrocytes. In mice with EAE, however, both macrophages/microglia and astrocytes in infiltrates expressed NOS. Surprisingly, some astrocytes that were distant from infiltrates also expressed NOS, thus suggesting that astrocytes may modulate leukocyte infiltration via release of NO through their foot processes in the blood-brain barrier. Collectively, my data propose a model of a dynamic network in which the interplay among cytokines, chemokines and nitric oxide, may determine the magnitude, the composition, or the resolution of inflammatory infiltrates, as well as the clinical outcome of EAE.

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Книги з теми "Immune health"

Immune power: Health and the immune system. London: Optima, 1990.

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Li, Bin, and Fan Pan, eds. Immune Metabolism in Health and Tumor. Dordrecht: Springer Netherlands, 2017. http://dx.doi.org/10.1007/978-94-024-1170-6.

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Bueno, Valquiria, Janet M. Lord, and Thomas A. Jackson, eds. The Ageing Immune System and Health. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-43365-3.

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Greenberg, Sylvia S. Immunity and survival: Keys to immune system health. New York, N.Y: Human Sciences Press, 1989.

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Janeway, Charles A. Immunobiology: The immunesystem in health anddisease. Oxford: Blackwell, 1994.

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Stress, immune function, and health: The connection. New York: Wiley-Liss, 1999.

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Janeway, Charles. Immunobiology: The immune system in health and disease. 2nd ed. London: Current Biology, 1996.

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1956-, Travers Paul, ed. Immunobiology: The immune system in health and disease. London: Current Biology Limited, 1994.

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1956-, Travers Paul, ed. Immunobiology: The immune system in health and disease. 3rd ed. London: Current Biology, 1997.

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Janeway, Charles A. Immunobiology: The immune system in health and disease. London: Current Biology Limited, 1994.

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Частини книг з теми "Immune health"

Carr, John, Shih-Ping Chen, Joseph F. Connor, Roy Kirkwood, and Joaquim Segalés. "Immune System Disorders." In Pig Health, 255–75. Boca Raton : CRC Press, [2018]: CRC Press, 2018. http://dx.doi.org/10.1201/9781315157061-8.

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Conroy, Michelle E., and W. Allan Walker. "Intestinal Immune Health." In Nestlé Nutrition Workshop Series: Pediatric Program, 111–25. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000146255.

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Ng, Y. C., and J. A. Schifferli. "Complement, immune complexes and immune complex disease." In Complement in Health and Disease, 199–228. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-011-2214-6_7.

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Lee, Wang Jae. "Immune System." In Vitamin C in Human Health and Disease, 75–88. Dordrecht: Springer Netherlands, 2019. http://dx.doi.org/10.1007/978-94-024-1713-5_4.

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Nieman, David C. "Immune System." In Encyclopedia of Exercise Medicine in Health and Disease, 441–44. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_108.

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O’Shea, Daniel J. "Acquired Immune Deficiency Syndrome." In Encyclopedia of Immigrant Health, 154–58. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-5659-0_14.

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Hanamsagar, Richa, Sandra M. Cardona, Tammy Kielian, and Astrid E. Cardona. "Roles in Immune Responses." In Microglia in Health and Disease, 115–44. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1429-6_5.

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Böning, Dieter, Michael I. Lindinger, Damian M. Bailey, Istvan Berczi, Kameljit Kalsi, José González-Alonso, David J. Dyck, et al. "Adaptive Immune Cells." In Encyclopedia of Exercise Medicine in Health and Disease, 17. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_4022.

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Wang, Jing, Shuqin Liu, Guoping Li, and Junjie Xiao. "Exercise Regulates the Immune System." In Physical Exercise for Human Health, 395–408. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-1792-1_27.

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Mitterstiller, Anna-Maria, Laura von Raffay, and Manfred Nairz. "Iron Deficiency, Anemia, and the Immune System." In Nutrition and Health, 235–48. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-14521-6_18.

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Тези доповідей конференцій з теми "Immune health"

Na, Risha, and Ruilong Chen. "Differences in Immune Checkpoint Protein Expression among Immune Cells in Lung Carcinoma." In ICIMH 2021: 2021 the 3rd International Conference on Intelligent Medicine and Health. New York, NY, USA: ACM, 2021. http://dx.doi.org/10.1145/3484377.3484392.

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Iswati, Retno Setyo. "ANALYSIS OF THE RELATIONSHIP BETWEEN HISTORY OF EXCLUSIVE BREASTFEEDING WITH IMMUNITY STATUS OF INFANTS AGED 6 – 12 MONTHS DURING THE COVID-19 PANDEMIC." In International Conference on Public Health and Medical Sciences. Goodwood Conferences, 2022. http://dx.doi.org/10.35912/icophmeds.v1i1.22.

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Analyzing the relationship between a history of exclusive breastfeeding and the immune status of infants aged 6-12 months during the covid-19 pandemic. This study is a descriptive-analytic study with a cross-sectional approach. The sampling technique used was accidental sampling. The sample is 62 respondents. Primary data collection uses a questionnaire. Data analysis using Chi-Square with a significance of 0.05. The history of exclusive breastfeeding was 61.3%. The immune status of infants aged 6-12 months was mostly good, which was 56.4%. There is a relationship between a history of exclusive breastfeeding and the immune status of infants aged 6-12 months during the covid-19 pandemic with a p-value of 0.040 The results of the study cannot be generalized widely. Many factors affect the immune status of infants aged 6 – 12 months Providing information to mothers and the community about the importance of exclusive breastfeeding on the baby's immune status.

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Costin, Hariton, and Silviu Ioan Bejinariu. "Medical signal processing by means of immune algorithms." In 2013 E-Health and Bioengineering Conference (EHB). IEEE, 2013. http://dx.doi.org/10.1109/ehb.2013.6707386.

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Mokhtar, Maizura, and Joe Howe. "Safer Flying Using an Immune-Inspired Adaptive Health Monitoring System." In 2013 IEEE International Conference on Systems, Man and Cybernetics (SMC 2013). IEEE, 2013. http://dx.doi.org/10.1109/smc.2013.444.

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Chen, Bo, and Chuanzhi Zang. "Discovery of emerging patterns with immune network theory." In SPIE Smart Structures and Materials + Nondestructive Evaluation and Health Monitoring, edited by Masayoshi Tomizuka. SPIE, 2010. http://dx.doi.org/10.1117/12.847612.

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Nepomnyashchaya, Elina K., Elena N. Velichko, Evgenij Aksenov, and Tatyana Bogomaz. "Laser correlation spectroscopy as a powerful tool to study immune responses." In Biophotonics: Photonic Solutions for Better Health Care, edited by Jürgen Popp, Valery V. Tuchin, and Francesco S. Pavone. SPIE, 2018. http://dx.doi.org/10.1117/12.2307241.

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Chelly, Zeineb, and Zied Elouedi. "Further exploration of the hybrid Fuzzy-Rough Dendritic Cell immune classifier." In 2013 E-Health and Bioengineering Conference (EHB). IEEE, 2013. http://dx.doi.org/10.1109/ehb.2013.6707300.

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Sheng-li, Hou, Zhou Yang, Li Le-xi, and Xu Chang-kai. "Sensor fault detection based on self organizing immune network." In 2021 Global Reliability and Prognostics and Health Management (PHM-Nanjing). IEEE, 2021. http://dx.doi.org/10.1109/phm-nanjing52125.2021.9613031.

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Beatty, Gregory. "Abstract IA-05: Immune health and intratumoral immune heterogeneity: Barriers to the efficacy of immunotherapy in pancreatic cancer." In Abstracts: AACR Virtual Special Conference on Pancreatic Cancer; September 29-30, 2020. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.panca20-ia-05.

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Guo, Yuanlin. "Efficacy, Safety and Immune Reactions Associated with COVID-19 Vaccines." In International Conference on Health Big Data and Intelligent Healthcare. SCITEPRESS - Science and Technology Publications, 2022. http://dx.doi.org/10.5220/0011368800003438.

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Звіти організацій з теми "Immune health"

Romano, Tracy. Investigation of Neural-Immune Profiling, Transcriptomics and Proteomics and Clinical Tools in Assessing Navy Dolphin Health. Fort Belvoir, VA: Defense Technical Information Center, December 2007. http://dx.doi.org/10.21236/ada474990.

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Pai, Menaka, Allan Grill, Noah Ivers, Antonina Maltsev, Katherine J. Miller, Fahad Razak, Michael Schull, et al. Vaccine Induced Prothrombotic Immune Thrombocytopenia (VIPIT) Following AstraZeneca COVID-19 Vaccination. Ontario COVID-19 Science Advisory Table, March 2021. http://dx.doi.org/10.47326/ocsat.2021.02.17.1.0.

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This Science Brief provides information for health care professionals about Vaccine Induced Prothrombotic Immune Thrombocytopenia (VIPIT), a rare adverse event following the AstraZeneca vaccine. This brief describes the pathophysiology, presentation, diagnostic work-up and treatment of VIPIT. Figure 1 presents a decision tree for diagnosis and rule out of VIPIT.

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Ozano, Kim. Interventions Aimed at Preventing, Detecting and Treating Malaria, TB and HIV in Nigeria. Institute of Development Studies (IDS), January 2022. http://dx.doi.org/10.19088/k4d.2022.039.

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There is an abundance of available evidence on the effectiveness of interventions aimed at preventing, detecting and treating malaria, TB and HIV in Nigeria. The evidence suggests that future interventions concerning these three diseases should focus on health systems strengthening, considering each of the 6 WHO building blocks. Therefore, this review is structured around the building blocks for each disease, This is part of a series of reports looking into Epidemiology of Malaria, human immune deficiency virus (HIV) and tuberculosis (TB) across a set of African Nations.

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Chejanovsky, Nor, Diana Cox-Foster, Victoria Soroker, and Ron Ophir. Honeybee modulation of infection with the Israeli acute paralysis virus, in asymptomatic, acutely infected and CCD colonies. United States Department of Agriculture, December 2013. http://dx.doi.org/10.32747/2013.7594392.bard.

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Honey bee (Apis mellifera) colony losses pose a severe risk to the food chain. The IAPV (Israeli acute paralysis virus) was correlated with CCD, a particular case of colony collapse. Honey bees severely infected with IAPV show shivering wings that progress to paralysis and subsequent death. Bee viruses, including IAPV, are widely present in honey bee colonies but often there are no pathological symptoms. Infestation of the beehive with Varroa mites or exposure to stress factors leads to significant increase in viral titers and fatal infections. We hypothesized that the honey bee is regulating/controlling IAPV and viral infections in asymptomatic infections and this control is broken through "stress" leading to acute infections and/or CCD. Our aims were: 1. To discover genetic changes in IAPV that may affect tissue tropism in the host, and/or virus infectivity and pathogenicity. 2. To elucidate mechanisms used by the host to regulate/ manage the IAPV-infection in vivo and in vitro. To achieve the above objectives we first studied stress-induced virus activation. Our data indicated that some pesticides, including myclobutanil, chlorothalonil and fluvalinate, result in amplified viral titers when bees are exposed at sub lethal levels by a single feeding. Analysis of the level of immune-related bee genes indicated that CCD-colonies exhibit altered and weaker immune responses than healthy colonies. Given the important role of viral RNA interference (RNAi) in combating viral infections we investigated if CCD-colonies were able to elicit this particular antiviral response. Deep-sequencing analysis of samples from CCD-colonies from US and Israel revealed high frequency of small interfering RNAs (siRNA) perfectly matching IAPV, Kashmir bee virus and Deformed wing virus genomes. Israeli colonies showed high titers of IAPV and a conserved RNAi pattern of targeting the viral genome .Our findings were further supported by analysis of samples from colonies experimentally infected with IAPV. Following for the first time the dynamics of IAPV infection in a group of CCD colonies that we rescued from collapse, we found that IAPV conserves its potential to act as one lethal, infectious factor and that its continuous replication in CCD colonies deeply affects their health and survival. Ours is the first report on the dominant role of IAPV in CCD-colonies outside from the US under natural conditions. We concluded that CCD-colonies do exhibit a regular siRNA response that is specific against predominant viruses associated with colony losses and other immune pathways may account for their weak immune response towards virus infection. Our findings: 1. Reveal that preventive measures should be taken by the beekeepers to avoid insecticide-based stress induction of viral infections as well as to manage CCD colonies as a source of highly infectious viruses such as IAPV. 2. Contribute to identify honey bee mechanisms involved in managing viral infections.

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Bain, Luchuo Engelbert, and Darja Dobermann. Malaria, HIV and TB in the Democratic Republic of the Congo: Epidemiology, Disease Control Challenges and Interventions. Institute of Development Studies (IDS), March 2022. http://dx.doi.org/10.19088/k4d.2022.034.

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Malaria, human immune deficiency virus (HIV) and tuberculosis (TB) are leading causes of death and public health threat to millions in Democratic Republic of Congo (DRC). The DRC is the second most malaria affected sub-Saharan African country after Nigeria, with malaria being the leading cause of death in children under 5 years (Lechthaler et al., 2019). The HIV prevalence in the country in the adult population stands at 1%, with extensive variations by region (UNAIDS, 2021c). The DRC is considered a high burden country for TB and HIV infection (Linguissi et al., 2017). This rapid review emphasizes significant elements of the epidemiology of malaria, HIV, and TB in DRC, as well as limitations in prevention, detection, and treatment, and examines a few interventions that aim to address these limitations. Evidence utilised is a mixture of the most recent grey literature NGO (programme reports and related documents) literature supplemented by peer reviewed academic literature from the past five years and national survey data when available. Although the clinical disease aspects of malaria, HIV and TB are well-researched there is less research available on socio-demographic variation, disease control challenges and interventions targeting these in the DRC. This is part of a series of reports looking into Epidemiology of Malaria, human immune deficiency virus (HIV) and tuberculosis (TB) across a set of African Nations.

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Knibb, Rebecca, Lily Hawkins, and Dan Rigby. Food Sensitive Study: Wave Two Survey. Food Standards Agency, September 2022. http://dx.doi.org/10.46756/sci.fsa.nyx192.

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Food hypersensitivities (FH) include food allergy, food intolerance and coeliac disease. Food allergy and coeliac disease involve an immune mediated reaction to certain foods; food intolerance is caused by a non-immune mediated reaction (such as an enzymatic or pharmacological effect). Each of these FHs result in unpleasant symptoms if the food is eaten in sufficient quantity, with food allergic reactions sometimes resulting in life-threatening symptoms. Management of FH by an individual or members of their family therefore involves constant vigilance and risk assessment to determine if a food is safe to eat. Research over the last twenty years has demonstrated that this burden, along with the unpredictable nature of FH reactions, has an impact on quality of life (QoL). QoL encompasses our emotions, physical health, the environment we live in, our social networks and day-to-day activities. FH has been shown to have an impact on many of these areas, however there are still research gaps. In particular, many studies focus on children, adolescents or parents rather than the adult population and little is known about those with food intolerances. In order to make a comprehensive characterisation and evaluation of the burden caused by living with FH, the day-to-day management of FH and associated inconveniences, the FSA has commissioned this project, led by Aston University. The project is called the FoodSensitive study and this report relates to findings for workstream one, a survey to assess the impact of FH on QoL. This survey was carried out in two waves, one year apart. This report covers the second wave and a comparison of wave one and two for those participants who completed both waves.

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Zhenni, Mu, Le Lei, Shen Sinan, and Tang Li. Effectiveness of integrated Chinese herbal medicine Shoutai Pill and Western medicine in the treatment of recurrent pregnancy loss: A protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2021. http://dx.doi.org/10.37766/inplasy2021.10.0062.

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Review question / Objective: We provide a protocol to evaluate the efficacy of integrated Shoutai Pill and Western medicine to update the evaluation for the best available and security treatment for recurrent pregnancy loss(RPL). Condition being studied: Recurrent pregnancy loss (RPL) is a distinct disorder defined by two or more consecutive pregnancy failures before 20 gestational weeks infertile couples. The incidence of this disease accounts for about 1%-5% of women of reproductive age and seriously affects their physical and psychological health. At present, the known etiology of this disease mainly includes abnormal anatomic structures, genetic abnormality, endocrine disorders, prethrombotic status, abnormal immune function, infection, etc. Excluding the above factors, approximately 40-50% of RPL remain unexplained, known as unexplained recurrent pregnancy loss (URPL). At present, the main therapeutic methods of RPL are surgical therapy, preimplantation genetic diagnosis (PGD), hormone therapy, anti-infection therapy, anticoagulation, and immunoregulatory therapy, etc. However, there is no effective treatment has been identified for URPL. Therefore, we still need to investigate effective treatments to reduce pregnancy losses and maintain successful pregnancy preservation in these patients.

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Lan, Xi, John C. F. Hsieh, Carl J. Schmidt, Qing Zhu, and Susan J. Lamont. Heat Stress Alters Immune Pathways in Liver of Divergent Chicken Lines. Ames (Iowa): Iowa State University, January 2017. http://dx.doi.org/10.31274/ans_air-180814-335.

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Haider, Huma. Malaria, HIV and TB in Mozambique: Epidemiology, Disease Control and Interventions. Institute of Development Studies, January 2022. http://dx.doi.org/10.19088/k4d.2022.035.

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Malaria, HIV and tuberculosis (TB) are significant public health concerns in Mozambique. Malaria was the fourth leading cause of death in the country in 2019, accounting for 42% of deaths among children under 5 years of age (Mugabe et al., 2021; USAID, 2018). Mozambique is among the top eight countries with the highest HIV prevalence; with the second highest mother-to-child transmission (MTCT) rate in the world (Fuente-Soro et al., 2021; Nacarapa et al., 2021). The incidence of TB is rising, with pediatric TB cases almost tripling in recent years (WHO, 2020b; Nguenha et al., 2018; Orlando et al., 2018). Mozambique has one of the highest global incidence of malaria-HIV and TB-HIV co-infection, which raises the likelihood of poor clinical outcomes (Moon et al., 2019; USAID, 2018). This rapid literature review highlights key aspects of the epidemiology of malaria, HIV and TB in Mozambique and challenges in prevention, detection and treatment; and surveys select interventions that seek to address these challenges. This is part of a series of reports looking into Epidemiology of Malaria, human immune deficiency virus (HIV) and tuberculosis (TB) across a set of African Nations.

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Haider, Huma. Malaria, HIV and TB in Mozambique: Epidemiology, Disease Control and Interventions. Institute of Development Studies, January 2022. http://dx.doi.org/10.19088/k4d.2022.035.

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