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Дисертації з теми "Insulin resistance diabete"

Автор: Grafiati
Опубліковано: 9 червня 2023

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1

Capetini, Vinícius Cooper. "Efeito da suplementação com zinco na evolução da resistência à insulina induzida por dieta hiperlipídica em camundongos." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/42/42137/tde-10082016-154401/.

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O aumento da prevalência do diabete melito do tipo 2 (DM2) é intenso e implica ampla busca pela prevenção e tratamento da doença. Estudos têm mostrado a participação do zinco na síntese, secreção e via de sinalização da insulina e sobre o controle glicêmico. Este trabalho objetivou analisar o mecanismo de ação do zinco no controle da secreção de insulina e no controle glicêmico, de modo a entender se a suplementação com o zinco previne ou retarda a manifestação do DM2. O projeto foi aprovado pela CEUA-ICB (USP). Camundongos machos C57BL/6 foram divididos em 4 grupos experimentais: dieta contr
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2

Napolitano, Tiziana. "Confidentiel." Thesis, Université Côte d'Azur (ComUE), 2017. http://www.theses.fr/2017AZUR4141.

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3

GRANCINI, VALERIA. "RUOLO CENTRALE DELLA BETA-CELLULA NEL PROMUOVERE LA REGRESSIONE DEL DIABETE DOPO TRAPIANTO DI FEGATO IN PAZIENTI CON CIRROSI EPATICA." Doctoral thesis, Università degli Studi di Milano, 2019. http://hdl.handle.net/2434/658515.

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BACKGROUND & AIMS: Diabetes occurring as a direct consequence of loss of liver function is usually characterized by non-diabetic fasting plasma glucose (FPG) and haemoglobin A1c (HbA1c) levels and should regress after orthotopic liver transplantation (OLT). This observational, longitudinal study investigated the relationship between the time-courses of changes in all 3 direct determinants of glucose regulation, i.e., β-cell function, insulin clearance and insulin sensitivity, and diabetes regression after OLT. METHODS: Eighty cirrhotic patients with non-diabetic FPG and HbA1c levels und
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4

Pappalardo, Alessandro Orazio Giovanni. "Ruolo del recettore Gabaa nelle cellule alfa pancreatiche e suo coinvolgimento nelle fasi di desensibilizzazione indotta dalla lipotossicità nel diabete di tipo II." Doctoral thesis, Università di Catania, 2016. http://hdl.handle.net/10761/3991.

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Il GABA o acido gamma-aminobutirrico è il principale neurotrasmettitore inibitorio del sistema nervoso centrale (SNC) dei mammiferi. Circa il 20 - 50% di tutte le sinapsi utilizzano questo neurotrasmettitore. Esso è presente in quantità elevate, anche nello spazio interstiziale delle isole pancreatiche e nelle beta cellule pancreatiche dalle quali viene secreto. Il GABA oltre ad avere funzione trofica sulle beta cellule, attraverso il suo recettore (GABAA) controlla la riduzione della secrezione di glucagone insieme con lâ insulina. Nel nostro lavoro abbiamo studiato gli effetti dell'espo
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5

Leança, Camila Canteiro. "O metabolismo de lipoproteínas e a sensibilidade à insulina são distintamente modulados em indivíduos saudáveis com concentração alta ou baixa de HDL-colesterol." Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/5/5135/tde-27072012-143556/.

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A síndrome metabólica (SM) e o diabete melito (DM) caracterizam-se por uma série de alterações no metabolismo de lipoproteínas, entre elas a hipertrigliceridemia e a redução nas concentrações de HDL-colesterol (HDL-C). Em estudo prévio demonstramos que indivíduos saudáveis, não obesos, com concentração de HDL-C abaixo de 40mg/dL, quando comparados àqueles com concentração de HDL-C acima de 60mg/dL, apresentam, no plasma, esteróis marcadores de absorção intestinal de colesterol alimentar diminuídos, e de síntese de colesterol aumentados. Achados semelhantes foram descritos por outros autores em
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6

Kergoat, Micheline. "Secretion et mode d'action de l'insuline dans un modele de diabete non-insulinodependant chez le rat." Paris 7, 1988. http://www.theses.fr/1988PA077085.

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7

Pickavance, Lucy Cecilia. "Thiazolidinedione treatment in models of insulin resistance." Thesis, University of Liverpool, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.367553.

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8

GALLI, ANGELICA. "Ruolo dei PUFA OMEGA-3 nella regolazione della funzione endoteliale in parenti di primo grado di pazienti affetti da diabete mellito di tipo 2." Doctoral thesis, Università degli Studi di Roma "Tor Vergata", 2007. http://hdl.handle.net/2108/377.

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Obiettivi: La disfunzione endoteliale è il meccanismo iniziale nella patogenesi dell’aterosclerosi. Costituisce una delle alterazioni vascolari presenti in soggetti apparentemente sani parenti di primo grado di pazienti affetti da Diabete Mellito Tipo 2 (FDR). Gli acidi grassi polinsaturi n-3 (PUFA n-3), come l’acido eicosapentaenoico (EPA) e l’acido docosaesaenoico (DHA), hanno un’ampia serie di proprietà antinfiammatorie. Noi abbiamo voluto testare l’ipotesi che la somministrazione di PUFA n-3 potesse migliorare la funzione endoteliale in soggetti FDR, ad elevato rischio di sviluppare precoc
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9

Guarilha, Alessandra Lia Gasparetti. "Transdução do sinal da insulina em animais expostos ao frio : o papel do cross-talk entre o receptor 'beta' 3 - adrenergico e o receptor de insulina em tecido adiposo marrom." [s.n.], 2004. http://repositorio.unicamp.br/jspui/handle/REPOSIP/310365.

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Orientador: Licio Augusto Velloso<br>Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas<br>Made available in DSpace on 2018-08-04T02:29:02Z (GMT). No. of bitstreams: 1 Guarilha_AlessandraLiaGasparetti_D.pdf: 7883483 bytes, checksum: cefd0ee77fd363b470280f9b8a380ff9 (MD5) Previous issue date: 2004<br>Resumo: A exposição de animais homeotérmicos ao mo é utilizada como um método reprodutível para se obter um modelo animal de hipoinsulinemiaacompanhada por elevada mobilização periférica de glicose. No presente estudo, avaliaram-se as etapas iniciais e intermediár
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10

Lalli, Cristina Alba. "Efeito da rosiglitazona e da lovastatina na resistencia insulinica da dieta hiperlipidica." [s.n.], 2007. http://repositorio.unicamp.br/jspui/handle/REPOSIP/311224.

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Анотація:
Orientador: Mario Jose Abdalla Saad<br>Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas<br>Made available in DSpace on 2018-08-10T01:17:39Z (GMT). No. of bitstreams: 1 Lalli_CristinaAlba_D.pdf: 9125967 bytes, checksum: 5ab6be7c7ee8bef63c8acdec567c428b (MD5) Previous issue date: 2007<br>Resumo: A insulina é o principal hormônio anabólico, que atua através da ativação de transportadores, regulação de enzimas e expressão de genes que codificam enzimas envolvidas na captação e armazenamento de substratos. Para exercer suas ações, a insulina emprega duas vias pr
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11

Hale, P. J. "Insulin resistance in diabetes mellitus and obesity." Thesis, University of Oxford, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.371516.

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12

Eason, Robert C. "Treating type 2 diabetes through insulin resistance." Thesis, Aston University, 2002. http://publications.aston.ac.uk/10953/.

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Анотація:
Type 2 diabetes is an insidious disorder, with micro and/or macrovascular and nervous damage occurring in many patients before diagnosis. This damage is caused by hyperglycaemia and the diverse effects of insulin resistance. Obesity, in particular central obesity, is a strong pre-disposing factor for type 2 diabetes. Skeletal muscle is the main site of insulin-stimulated glucose disposal and appears to be the first organ that becomes insulin resistant in the diabetic state, with later involvement of adipose tissue and the liver. This study has investigated the use of novel agents to ameliorate
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13

Kershner, David. "Oral Glucose Insulin Secretion Test for Identifying Patients with Insulin Resistance." ScholarWorks, 2018. https://scholarworks.waldenu.edu/dissertations/5634.

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Insulin resistance is an increasing public health issue with the current literature, suggesting reduced sensitivity of insulin leads to adult onset diabetes and associated downstream pathologies that reduce life expectancy. The main objectives of this study were to evaluate the ability of the Oral Glucose Insulin Secretion Test (OGIST) to identify insulin resistance and examine differences in the insulin sensitivity based on gender, age, and ethnicity. This study was supported by the insulin resistance theory which focuses on the reduced ability of insulin to bind to the cellular insulin recep
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14

Wiggam, Malcolm Ivan. "Aspects of insulin resistance in essential hypertension and insulin-dependent diabetes mellitus." Thesis, Queen's University Belfast, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.387971.

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15

MAIORANA, ARIANNA. "A Method to isolate omental adipose stem cells in pediatric patients: perspectives in investigating the pathogenesis of metabolic syndrome." Doctoral thesis, Università degli Studi di Roma "Tor Vergata", 2008. http://hdl.handle.net/2108/516.

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Il tessuto adiposo è coinvolto nella regolazione di metabolismo glucidico e lipidico, omeostasi energetica, infiammazione e risposta immune. L’obesità addominale gioca un ruolo chiave nella regolazione dello sviluppo dell’ insulino-resistenza per l’elevata lipolisi del tessuto adiposo viscerale e la secrezione della adipochine. Gli individui nati con basso peso alla nascita tendono ad avere una redistribuzione centrale del tessuto adiposo e sono ad alto rischio di sviluppare sindrome metabolica, diabete di tipo 2 e patologie cardiovascolari. Diversi studi volti a caratterizzare le cellule stam
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16

Hunter, Steven J. "Insulin resistance : underlying mechanisms and influence of treatment." Thesis, Queen's University Belfast, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.337044.

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17

Frangioudakis, Georgia St Vincent's Clinical School UNSW. "Insulin signal transduction in vivo in states of lipid-induced insulin resistance." Awarded by:University of New South Wales. St Vincent's Clinical School, 2004. http://handle.unsw.edu.au/1959.4/27419.

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Insulin resistance is the major metabolic defect in obesity and Type 2 diabetes. Increased lipid accumulation is strongly associated with insulin resistance. A significant component of insulin resistance is thought to be a reduced ability of insulin to activate the cascade of phosphorylation events that lead to the metabolic effects of this hormone. The broad aims of this thesis were to examine the effect of high-fat diets containing different fat subtypes on in vivo insulin signalling, under conditions normally used to detect whole body insulin resistance, and to compare the effects of acute
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18

Vella, Sandro. "Pharmacological modulation of insulin resistance : benefits and harms." Thesis, University of Dundee, 2013. https://discovery.dundee.ac.uk/en/studentTheses/4428eca5-5018-4816-95d0-30c0a3043409.

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Aims: Thiazolidinediones have been advocated as second or third line insulin sensitizing agents in the management of type 2 diabetes (T2DM). Their widespread use has been hampered by concerns about their cardiovascular safety, including fluid retention. Metformin is established as first-line glucose-lowering pharmacotherapy in T2DM. It has also been suggested that it may have benefits in alleviating insulin resistance in type 1 diabetes (T1DM). This thesis examined: (i) cardiovascular, renal and metabolic differences between individuals with T2DM ‘tolerant’ or ‘intolerant’ of TZDs; (ii) risk f
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19

Song, Xiao Mei. "Insulin signal transduction in skeletal muscle : special consideration for insulin resistance and diabetes /." Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-4502-0/.

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20

Danielsson, Anna. "Insulin signalling in human adipocytes : mechanisms of insulin resistance in type 2 diabetes." Doctoral thesis, Linköping : Univ, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-10327.

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21

Lindmark, Stina. "Neurohormonal mechanisms in insulin resistance and type 2 diabetes." Doctoral thesis, Umeå : Univ, 2004. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-225.

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22

Reali, Federico. "Dynamical models for diabetes: insights into insulin resistance and type 1 diabetes." Doctoral thesis, Università degli studi di Trento, 2017. https://hdl.handle.net/11572/369184.

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This thesis summarizes my work in systems biology as a PhD student at The Microsoft Research - University of Trento Centre for Computational and Systems Biology (COSBI) and at the University of Trento, department of Mathematics. Systems biology is an interdisciplinary field that aims at integrating biology with computational and mathematical methods to gain a better understanding of biological phenomena [5, 6]. Among these methods, mathematical and dy- namical modeling have driven the discovery of mechanistic insights from the static representations of phenomena, that is, data. As a result, ma
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23

Reali, Federico. "Dynamical models for diabetes: insights into insulin resistance and type 1 diabetes." Doctoral thesis, University of Trento, 2017. http://eprints-phd.biblio.unitn.it/1962/1/Reali_PhDThesis.pdf.

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Анотація:
This thesis summarizes my work in systems biology as a PhD student at The Microsoft Research - University of Trento Centre for Computational and Systems Biology (COSBI) and at the University of Trento, department of Mathematics. Systems biology is an interdisciplinary field that aims at integrating biology with computational and mathematical methods to gain a better understanding of biological phenomena [5, 6]. Among these methods, mathematical and dy- namical modeling have driven the discovery of mechanistic insights from the static representations of phenomena, that is, data. As a result, m
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24

Beatty, Olivia Louise. "Aspects of insulin resistance, insulin secretion and microalbuminuria in diabetic, hypertensive and non-diabetic subjects." Thesis, Queen's University Belfast, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.484068.

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25

Ng, Foong Loo Yvonne Biotechnology &amp Biomolecular Sciences Faculty of Science UNSW. "Insulin action: unravelling AKT signalling in Adipocytes." Awarded by:University of New South Wales. Biotechnology & Biomolecular Sciences, 2009. http://handle.unsw.edu.au/1959.4/44628.

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The Ser/Thr kinase Akt plays an important role in many of insulin's actions including GLUT4 translocation to the plasma membrane (PM). However, there are several features of Akt's regulation of GLUT4 translocation that remain unclear. The goal of my thesis was to resolve some of the following questions: Is activation of Akt sufficient to stimulate GLUT4 translocation? What is the quantitative relationship in signal transmission between individual components within the Akt cascade? What is the role of Akt in insulin resistance? To determine if activation of Akt is sufficient to mediate GLUT4 tr
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26

Botteri, Gaia. "Novel potential determinants in endoplasmic reticulum stress, inflammation and insulin resistance: Apo CIII and sAPPβ". Doctoral thesis, Universitat de Barcelona, 2018. http://hdl.handle.net/10803/553241.

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Diabetes represents one of the biggest health challenges of the 21st century. Insulin resistance is the primary defect in the most common form of diabetes, type 2 diabetes mellitus (T2D), and is defined as a failure in the capacity of insulin to drive glucose into its target tissues. This condition both predicts and precedes the development of T2D. Loss of insulin sensitivity in skeletal muscle is the major defect in T2D and is believed to be critical in the pathogenesis of this disease. Elucidating new molecular mechanisms involved in insulin resistance in skeletal muscle may lead to the de
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27

Relton, Caroline L. "The identification and characterisation of genes associated with insulin resistance." Thesis, University of Newcastle Upon Tyne, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.264425.

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28

Al-Rifai, Sarah. "Rôle de la résistine hypothalamique dans l'installation de l’inflammation hypothalamique et l’insulino-résistance : impact de la consommation aigüe ou chronique d'un régime hyper lipidique." Thesis, Université Paris-Saclay (ComUE), 2019. http://www.theses.fr/2019SACLS095/document.

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La prévalence de l’obésité est en net progrès et constitue un problème majeur de santé publique. Cette pathologie est d’autant plus dangereuse qu’elle s’accompagne d’un cluster de désordres métaboliques dont l’inflammation chronique de bas grade et la résistance à l’insuline, principal facteur de risque du diabète de type 2. Les études montrent que la consommation d’un régime hyper lipidique (HFD) représente la cause majeure qui expose à l’obésité et aux pathologies qui lui sont associées. L’obésité induite par un régime HFD s’associe en effet à une inflammation hypothalamique ainsi qu’une alt
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29

Agbaje, Olorunsola Fatai. "Modelling methodologies to assess insulin resistance and insulin secretion in type 2 diabetes subjects." Thesis, City University London, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.397933.

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30

Clarke, Stephen. "Liver steatosis and insulin-resistance : reversal by Sutherlandia frutescens." Thesis, Nelson Mandela Metropolitan University, 2014. http://hdl.handle.net/10948/d1020788.

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Type 2 diabetes mellitus (T2DM) is rapidly emerging as one of the greatest global health issues of the 21st century. Insulin-resistance is a condition associated with T2DM and in the cell it is defined as the inadequate strength of insulin signalling from the insulin receptor downstream to the final substrates of insulin action involved in multiple metabolic, gene expression, and mitogenic aspects of cellular function. To investigate the potential mechanisms involved in the development of insulin-resistance, two in vitro liver cell models were established using palmitate or a combination of in
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31

Nowak, Christoph. "Insulin Resistance : Causes, biomarkers and consequences." Doctoral thesis, Uppsala universitet, Molekylär epidemiologi, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-316891.

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The worldwide increasing number of persons affected by largely preventable diseases like diabetes demands better prevention and treatment. Insulin is required for effective utilisation of circulating nutrients. Impaired responsiveness to insulin (insulin resistance, IR) is a hallmark of type 2 diabetes and independently raises the risk of heart attack and stroke. The pathophysiology of IR is incompletely understood. High-throughput measurement of large numbers of circulating biomarkers may provide new insights beyond established risk factors. The aims of this thesis were to (i) use proteomics,
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32

Karlsson, Håkan K. R. "Insulin signaling and glucose transport in insulin resistant human skeletal muscle /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-469-4/.

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33

Tobar, Natália. "Efeitos da diacereína sobre a resistência à insulina em modelo animal de obesidade e diabetes mellitus tipo 2." [s.n.], 2010. http://repositorio.unicamp.br/jspui/handle/REPOSIP/311205.

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Анотація:
Orientador: Mario Jose Abdalla Saad<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas<br>Made available in DSpace on 2018-08-17T01:01:56Z (GMT). No. of bitstreams: 1 Tobar_Natalia_M.pdf: 2047534 bytes, checksum: cd359ddf7e8c05aab24d1675313c8789 (MD5) Previous issue date: 2010<br>Resumo: As alarmantes estatísticas mundiais sobre a incidência de obesidade e diabetes mellitus tipo 2 (DM2) associadas aos seus índices de avanço já considerados epidêmicos permitem classificá-los como alguns dos mais desafiadores problemas de saúde pública da atualidade. A
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34

Calixto, Antonio Ramos 1976. "Obesidade central e hiperglicemia influênciam o aumento da concentração plasmática da enzima dipeptidil peptidase 4 (DPP-4)." [s.n.], 2012. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308482.

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Orientador: Bruno Geloneze Neto<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas<br>Made available in DSpace on 2018-08-21T14:11:14Z (GMT). No. of bitstreams: 1 Calixto_AntonioRamos_M.pdf: 1368625 bytes, checksum: 6dca3f38a1874198b0d47f5c57f139da (MD5) Previous issue date: 2012<br>Resumo: Objetivo: O principal objetivo deste estudo foi avaliar as relações das concentrações enzimáticas da DPP-4 com adiposidade e o grau de tolerância à glicose. Métodos: Foram avaliados 227 indivíduos (Mulheres: 64,8% Homens: 35,2%) de ambos os sexos, 73 indivíduos co
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35

Malvezzi, Maria Aparecida Pereira Nunes. "Efeito dose-resposta do fluoreto em parâmetros relacionados à resistência à insulina e na expressão de proteínas hepáticas e musculares em camundongos NOD." Universidade de São Paulo, 2018. http://www.teses.usp.br/teses/disponiveis/25/25149/tde-24012019-090227/.

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Enquanto alguns trabalhos relatam que a administração crônica de F causa resistência à insulina, estudo recente do nosso grupo revelou que em animais com diabetes previamente induzido por estreptozotocina, a administração crônica de baixas doses de F aumenta a sensibilidade à insulina, por interferir em vias metabólicas musculares e hepáticas. Entretanto, o diabetes induzido por estreptozotocina causa alterações metabólicas diferentes do diabetes tipo 1. Desta forma, seria interessante verificar se o aumento da sensibilidade à insulina induzido por baixas doses de F também ocorreria utilizando
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36

Marakis, Georgios. "Natural therapy for insulin-resistance syndrome and type II diabetes." Thesis, University of Reading, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.340013.

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37

Bagstaff, Stephanie M. "Hyperinsulinemia and insulin resistance in cultured human muscle cells." Thesis, University of Newcastle Upon Tyne, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.270177.

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38

Erickson, Andrea Rose. "The Association Between Dairy Consumption and Insulin Resistance." BYU ScholarsArchive, 2013. https://scholarsarchive.byu.edu/etd/4262.

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Background: A cross-sectional design was employed to ascertain the relationship between dairy consumption and insulin resistance (IR) in 272 middle-aged, nondiabetic women. Methods: Participants kept a seven-day weighed food record to report their diets, including consumption of dairy foods. IR was assessed using the homeostatic model assessment (HOMA), using the following formula: fasting plasma insulin (µU/ml) x fasting plasma glucose (mg/dL)/405. The Bod Pod was used to examine body fat percentage, and accelerometry over a seven-day period was used to assess physical activity. HOMA values w
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39

Tubbs, Emily. "Importance of intracellular Mitochondria-Associated endoplasmic reticulum Membranes (MAM) in insulin-resistance." Thesis, Lyon 1, 2014. http://www.theses.fr/2014LYO10210.

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Les mitochondries et le réticulum endoplasmique (RE) interagissent au niveau de points de contacts appelés « Mitochondria-Associated ER Membranes » (MAM), afin d'échanger du Ca2+ via le complexe TP3Rl/Grp75/VDACl et maintenir l'homéostasie énergétique. Bien que des dysfonctions mitochondriales, un stress du RE et des altérations de l'homéostasie du Ca2+ participent au développement de l'insulino-résistance, on ne sait pas si ce sont des facteurs indépendants ou s'ils sont inter-reliés par une altération des MAM. Mes travaux de thèse ont permis de mettre en évidence un nouveau rôle des MAM dans
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40

Talbot, Nicola A. "Obesity, inflammation and insulin resistance in skeletal muscle." Thesis, Royal Veterinary College (University of London), 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.618327.

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41

Crawford, Lynne Mary. "The regulation of triglyceride metabolism in the liver and adipose tissue." Thesis, University of Glasgow, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.263457.

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42

Higa, Karen Cristiane. "A influencia da periodontite na via de sinalização da insulina em musculo e figado de ratos machos wistar." [s.n.], 2006. http://repositorio.unicamp.br/jspui/handle/REPOSIP/311207.

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Orientador: Mario Jose Abdalla Saad<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas<br>Made available in DSpace on 2018-08-07T07:17:39Z (GMT). No. of bitstreams: 1 Higa_KarenCristiane_M.pdf: 2248937 bytes, checksum: 985d6be8df78440d12226577db008021 (MD5) Previous issue date: 2006<br>Resumo: Existem indicações de que há uma relação bidirecional entre diabetes mellitus e doença periodontal. Tanto o diabetes quanto a doença periodontal podem estimular a liberação crônica de citocinas pró-inflamatórias que têm um efeito deletério nos tecidos periodont
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43

Ribeiro, Daniele Lisboa. "Efeitos da resistencia periferica a insulina e do diabetes na prostata ventral de ratos." [s.n.], 2008. http://repositorio.unicamp.br/jspui/handle/REPOSIP/317901.

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Orientadores: Rejane Maira Goes, Sebastião Roberto Taboga<br>Tese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia<br>Made available in DSpace on 2018-08-11T04:14:32Z (GMT). No. of bitstreams: 1 Ribeiro_DanieleLisboa_D.pdf: 4151708 bytes, checksum: bbb2c893cf2f2f9dbfd24f8e30c01cd2 (MD5) Previous issue date: 2008<br>Resumo: O diabetes mellitus leva a complicações em diversos órgãos, incluindo as glândulas acessórias do sistema genital masculino. Na próstata, é bem estabelecido que essa doença acarreta atrofia epitelial, mas ainda não é claro o seu efeito sobre os compone
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44

Santos, Kelly Ferreira dos 1988. "Impacto da ingestão de proteínas do soro do leite pré-hidrolisadas na diabetes melitus induzida em ratos = Impact of pre-hydrolyzed milk whey protein intake on induced diabetes mellitus in rats." [s.n.], 2014. http://repositorio.unicamp.br/jspui/handle/REPOSIP/254460.

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Orientador: Jaime Amaya Farfán<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Engenharia de Alimentos<br>Made available in DSpace on 2018-08-25T04:18:39Z (GMT). No. of bitstreams: 1 Santos_KellyFerreirados_M.pdf: 2656997 bytes, checksum: ec6f74833977d9f20517fd00ec736d53 (MD5) Previous issue date: 2014<br>Resumo: O presente trabalho teve por objetivo avaliar a capacidade da proteína do soro do leite, sob suas duas formas (concentrada e hidrolisada) em auxiliar na prevenção do desenvolvimento de obesidade e diabetes em ratos Wistar diabéticos. Foram utilizados 40 r
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45

Izard-Sudres, Hélène. "Evaluation de la sensibilité à l'insuline et de l'insulinosécrétion selon la méthode du Minimal Model de Bergman dans une population de sujets obèses." Montpellier 1, 1996. http://www.theses.fr/1996MON11031.

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46

Fryer, Lee George Daniel. "Studies into the role of glucose transporter function in insulin resistance." Thesis, University College London (University of London), 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.265007.

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47

Järvi, Anette. "Carbohydrate-rich foods in the treatment of the insulin resistance syndrome : studies of the importance of the glycaemic index and dietary fibre /." Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2001. http://publications.uu.se/theses/91-554-5166-7/.

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48

Fornari, Adriana. "Avaliação de medidas de resistência e secreção de insulina em pacientes HCV positivos." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2008. http://hdl.handle.net/10183/15917.

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49

Campisi, Raffaele. "Insuline resistance and insuline sensibility in patients with obstructive sleep apnea." Doctoral thesis, Università di Catania, 2014. http://hdl.handle.net/10761/1596.

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Obstructive sleep apnea (OSA), also referred to as obstructive sleep apnea-hypopnea (OSAH), is a sleep disorder that involves cessation or significant decrease in airflow in the presence of breathing effort. It is the most common type of sleep disordered breathing (SDB) and is characterized by recurrent episodes of upper airway collapse during sleep. These episodes are associated with recurrent oxyhemoglobin desaturations and arousals from sleep. Despite being a common disease, OSAS is underrecognized by most primary care physicians; an estimated 80% of Americans with OSAS are not diagnosed.
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50

Andersen, Ditte K. "The role of microRNAs in skeletal muscle insulin resistance." Thesis, Royal Veterinary College (University of London), 2016. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.701676.

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