Добірка наукової літератури з теми "Lungs Radiation injuries"

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Статті в журналах з теми "Lungs Radiation injuries":

1
Petricevic, A. "War injuries of the lungs." European Journal of Cardio-Thoracic Surgery 11, no. 5 (May 1997): 843–47. http://dx.doi.org/10.1016/s1010-7940(97)01163-9.
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Reeves, Glen I. "RADIATION INJURIES." Critical Care Clinics 15, no. 2 (April 1999): 457–73. http://dx.doi.org/10.1016/s0749-0704(05)70063-4.
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Shi, Chunmeng, and Shuliang Lu. "Radiation Injuries." International Journal of Lower Extremity Wounds 10, no. 3 (September 2011): 120–21. http://dx.doi.org/10.1177/1534734611418155.
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4
Saburova, A. S., M. V. Filimonova, V. V. Yuzhakov, L. I. Shevchenko, N. D. Yakovleva, L. N. Bandurko, A. E. Koretskaya, N. K. Fomina, V. O. Saburov та A. S. Filimonov. "The influence of nitric oxide synthases inhibitor Т1023 on the development of radiation pneumofibrosis in rats". Radiatsionnaya Gygiena = Radiation Hygiene 13, № 1 (березень 2020): 60–67. http://dx.doi.org/10.21514/1998-426x-2020-13-1-60-67.
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The purpose of the work was to study the ability of the NOS inhibitor T1023 to prevent late radiation injuries. Methods: the effects of T1023 (75 mg / kg, once i.p. 30 minutes before the irradiation) on the development of post-radiation pulmonitis and pneumofibrosis in rats with thoracic exposure to g-radiation at a dose of 12.5 Gy were studied histopathologically and morphometrically. The results of the studies showed that there wasn’t a significant objective effect of T1023 on the development of early radiation-induced lung injuries (9 weeks after irradiation). But it prevented late radiation induced lung injuaries (26 weeks after irradiation) – there were a significant lesser pathomorphological manifestations of post-radiation pulmonitis, proliferation of connective tissue and the development of fibrotic changes in the lung parenchyma. At this stage, the action of T1023 clearly contributed to the preservation of the normal histostructure of the lungs, reducing by 40% the content of compaction zones in the parenchyma. The ability of the NOS inhibitor T1023 to significantly limit the development of lungs late radiation reaction confirms the promise of further development of this compound as a means for prevention radiation therapy complications.
5
Taw, Irene K., William F. Hartsell, and David B. Rubin. "Radiation Toxicity of the Lungs." Clinical Pulmonary Medicine 2, no. 5 (September 1995): 295–302. http://dx.doi.org/10.1097/00045413-199509000-00006.
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6
Walker, Richard I. "Acute radiation injuries." Pharmacology & Therapeutics 39, no. 1-3 (January 1988): 9–12. http://dx.doi.org/10.1016/0163-7258(88)90033-2.
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Pandey, Manoj, and Balakrishnan Rajan. "Burn Injuries From Radiation." International Journal of Lower Extremity Wounds 3, no. 2 (June 2004): 96–99. http://dx.doi.org/10.1177/1534734604265534.
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Berry, RJ. "Treatment of Radiation Injuries." British Journal of Cancer 64, no. 3 (September 1991): 611. http://dx.doi.org/10.1038/bjc.1991.362.
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Friedstat, Jonathan, David A. Brown, and Benjamin Levi. "Chemical, Electrical, and Radiation Injuries." Clinics in Plastic Surgery 44, no. 3 (July 2017): 657–69. http://dx.doi.org/10.1016/j.cps.2017.02.021.
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10
Christensen, Doran M., Carol J. Iddins, and Stephen L. Sugarman. "Ionizing Radiation Injuries and Illnesses." Emergency Medicine Clinics of North America 32, no. 1 (February 2014): 245–65. http://dx.doi.org/10.1016/j.emc.2013.10.002.
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Дисертації з теми "Lungs Radiation injuries":

1
Scarboro, Sarah Brashear. "The use of a thyroid uptake system for assaying internal contamination following a radioactive dispersal event." Thesis, Georgia Institute of Technology, 2004. http://hdl.handle.net/1853/22639.
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Assaying internal contamination due to inhalation is a primary concern in developing emergency procedures related to Radioactive Dispersal Devices (RDD). One method of determining internal contamination makes use of a common medical instrument, a Thyroid Uptake System (TUS). The TUS used in this research has two collimators a thyroid uptake collimator and a bioassay collimator. Both collimators were considered and modeled in MCNP to be used in conjunction with six MIRD-type (Medical Internal Radiation Dose) phantoms. The collimators were placed in four positions on the phantoms the front right lung, the back right lung, the neck, and the thigh. Unit sources of Cs-137, Co-60, I-131, Ir-192, Am-241, and Sr/Y-90 were placed in the organs of the phantoms. MCNP particle tallies were performed over the detector crystal volume to determine the count-rate contributions from the unit source in each organ. Biokinetic modeling was performed using DCAL (Dose and Risk Calculation System) to generate coefficients to describe activity as a function of time in various organs. By folding the count-rate results with the organ concentrations, the detector response as a function of time after intake has been determined. This work was performed under funding provided by the Radiation Studies Branch of the Centers for Disease Control and Prevention.
2
Corsino, Betsy Ann 1962. "THE PULMONARY RESPONSE INDUCED BY GLASS FIBERS (INFLAMMATION, SILICOSIS, MURINE MODEL)." Thesis-Reproduction (electronic), The University of Arizona, 1986. http://hdl.handle.net/10150/291468.
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3
Chiu, Siu-hau, and 招兆厚. "A search for optimal radiation therapy technique for lung tumours stereotactic body radiation therapy (SBRT) : dosimetric comparison of 3D conformal radiotherapy, static gantry intensity modulated radiotherapy (IMRT) and volumetric-modulated arc therapy (VMAT) with flattening filter (FF) or flattening filter-free (FFF) beams." PG_Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2004. http://hdl.handle.net/10722/196549.
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Materials/Methods: Ten patients who underwent thoracic SBRT with primary stage I (T1/2N0) lung cancer or oligometastatic lung lesion, with PTV diameter ≤ 5cm were selected and were immobilized with Easyfoam or Vac-Lock. Planned/treated with inspiratory breath-hold (25 seconds, 70 to 80% of vital capacity) assisted with Active Breathing Control (ABC). Four treatment plans: non-coplanar 3DCRT, coplanar static gantry IMRT, coplanar VMAT (FF) and VMAT (FFF) were generated. Field arrangements, either static fields or partial arcs (duration=20 sec) were used to avoid direct beam entry to contralateral lung. All plans were compared in terms of dosimetric performance included dose to PTV or organs at risk (OAR), high/low dose spillage, integral dose (body and lungs), dose delivery efficiency (MU/Gy) and estimated beam-on time (BOT) with reference to the RTOG 0813 protocol. Results: All plans complied with RTOG 0813 protocol. VMAT (FF/ FFF) techniques improved target coverage and dose conformity, with the highest conformity number (CN > 0.91), compared to IMRT (0.88) and 3DCRT (0.85). The control of high dose spillage (NT>105% and CI) for IMRT (3.04% and 1.08) and VMAT (FF/ FFF) (1.08/ 1.06% and 1.03/ 1.04) techniques were comparable (p > 0.05) and significantly better than 3DCRT (4.22% and 1.11, p < 0.005) technique. In addition, VMAT (FF/ FFF) techniques performed the best in controlling low dose spillage (D2cm and R50%) compared with IMRT (reduction: 4.7%, p=0.036 and >5.9%, p = 0.009) and 3DCRT (reduction: > 16.3%, p < 0.001 and > 10%, p = 0.002). Benefits of rapid and isotropic dose fall-off were shown from superior tissue sparing (reduction ranges from 3.2% up to 67%) of ipsilateral brachial plexus, skin (0-5mm), great vessels and ribs. Also lung V10, V12.5, esophagus and heart tend to receive lower dose with VMAT technique. The relatively lower integral dose to whole body (> 3Gy∙L reduction, p < 0.013) and ipsilateral lung (0.65Gy∙L reduction, p = 0.025) compared with 3DCRT, were associated with lower risk of radiation induced cancers. The MU/Gy and BOT were substantial lower for VMAT (FF) (22.4% and 32.4%) compared with IMRT. Apart from higher (7%) maximum skin dose, dosimetric performance for VMAT (FFF) was comparable with VMAT (FF), with advantages of further reduction of MU/Gy (1.8% lesser), partial arc numbers (from 12-14 arcs down to 8 arcs) and BOT (35% shortened), owing to the increased dose output with flattening filter removal. Conclusions: VMAT (FF and FFF) plans maintained IMRT equivalent plan qualities, simultaneously enhanced the delivery efficiency with shortened BOT. VMAT (FFF) further reduced the required arcs number and BOT, significantly minimized the intra-fraction motions and more tolerable to patient with long SBRT treatment duration.
published_or_final_version
Medicine
Master
Master of Medical Sciences
4
Chakravarthy, Usha. "The effect of gamma radiation on intraocular cellular proliferation." Electronic Thesis or Dissertation, Queen's University Belfast, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.317046.
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5
Wong, Tony Po Yin, of Western Sydney Nepean University, and Faculty of Science and Technology. "Radiotherapy x-ray dosage distribution in lung and air cavities." THESIS_FST_XXX_Wong_T.xml, 1993. http://handle.uws.edu.au:8081/1959.7/360.
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The effect of lateral electron disequilibrium on patient dose has been investigated. This has been achieved by dosimetry in lung and air cavity phantoms at megavoltage x-ray energies. The scatter function photon beam models for tissue inhomogeneity, such as the ETAR correction algorithm, currently implemented in commercial treatment planning systems do not predict the dose distribution accurately in many situations where lateral electron equilibrium does not exist. The lung phantom is made up of solid water slabs and lung analogue slabs. Using a thimble ionization chamber, a Markus ionization chamber and TLDs the problems of central axis dose reduction and penumbral flaring in lung for x-rays have been investigated. It is found that the ETAR correction predicts the dose at mid lung with varying degrees of accuracy depending on the field size. It was found that internal body cavities, depending on their size, experience underdose or overdose in the distal surfaces of the cavities when compared with the results predicted by an ETAR correction algorithm. Therefore, this energy is not recommended for use in situations where cavities arise
Master of Science (Hons)
6
Tessem, May-Britt. "Metabolic effects of ultraviolet radiation on the anterior part of the eye." Doctoral thesis, comprehensive summary, Norwegian University of Science and Technology, Faculty of Medicine, 2006. http://urn.kb.se/resolve?urn=urn:nbn:no:ntnu:diva-742.
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Ultraviolet radiation (UV-R) is an environmental factor known to increase the risk of developing an irreversible opacification of the lens (cataract). Increased irradiance of UV-R to the earth because of depletion of stratospheric ozone is of current concern considering cataract formation. Detailed metabolic information from the cornea, lens and aqueous humour might give valuable knowledge on the biochcemical processes occurring in the eye after exposure to UV-R, and thereby a better understanding of the mechanisms by which UV-R induces cataractogenesis. The purpose of this thesis was to study metabolic effects of exposure to UV-R on the anterior part of the eye. Effects of UV-B (280-315 nm) and UV-A (315-400 nm) on the aqueous humour, cornea and the lens from animal models were investigated by 1H nuclear magnetic resonance (NMR) spectroscopy. Since the lens is composed of functionally distinct anatomical compartments, with different metabolic activity, biochemical changes in various compartments of the lens were analyzed.

Application of NMR-based metabonomics was effective to analyze metabolic changes in the anterior part of the eye after exposure to UV-R. High-resolution (HR) magic angle spinning (MAS) 1H NMR spectroscopy provided high quality spectra from intact tissue of cornea and lens, and provided important information about metabolic alteration occurring in these tissues after exposure to UV-R. The results from this thesis show that in vivo UV-B radiation affects metabolism of the anterior compartments of the eye. Metabolic changes were observed in aqueous humour, cornea, lens and in the different compartments of the lens. The antioxidants, glutathione and ascorbate, several amino acids, high energetic phosphates, and compounds important for membrane building and osmoregulation were substantially altered after exposure to UV-B radiation. Several biochemical effects such as oxidation, membrane disruption, osmoregulatory problems, lipid peroxidation, problems with cellular signalling and impairment of growth and protein synthesis were suggested. After UV-A exposure, no observable metabolic alterations were found in the anterior part of the eye in the present animal models.

7
Tessem, May-Britt. "Metabolic effects of ultraviolet radiation on the anterior part of the eye." Doctoral thesis, comprehensive summary, Norges teknisk-naturvitenskapelige universitet, Det medisinske fakultet, 2006. http://urn.kb.se/resolve?urn=urn:nbn:no:ntnu:diva-742.
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Ultraviolet radiation (UV-R) is an environmental factor known to increase the risk of developing an irreversible opacification of the lens (cataract). Increased irradiance of UV-R to the earth because of depletion of stratospheric ozone is of current concern considering cataract formation. Detailed metabolic information from the cornea, lens and aqueous humour might give valuable knowledge on the biochcemical processes occurring in the eye after exposure to UV-R, and thereby a better understanding of the mechanisms by which UV-R induces cataractogenesis. The purpose of this thesis was to study metabolic effects of exposure to UV-R on the anterior part of the eye. Effects of UV-B (280-315 nm) and UV-A (315-400 nm) on the aqueous humour, cornea and the lens from animal models were investigated by 1H nuclear magnetic resonance (NMR) spectroscopy. Since the lens is composed of functionally distinct anatomical compartments, with different metabolic activity, biochemical changes in various compartments of the lens were analyzed. Application of NMR-based metabonomics was effective to analyze metabolic changes in the anterior part of the eye after exposure to UV-R. High-resolution (HR) magic angle spinning (MAS) 1H NMR spectroscopy provided high quality spectra from intact tissue of cornea and lens, and provided important information about metabolic alteration occurring in these tissues after exposure to UV-R. The results from this thesis show that in vivo UV-B radiation affects metabolism of the anterior compartments of the eye. Metabolic changes were observed in aqueous humour, cornea, lens and in the different compartments of the lens. The antioxidants, glutathione and ascorbate, several amino acids, high energetic phosphates, and compounds important for membrane building and osmoregulation were substantially altered after exposure to UV-B radiation. Several biochemical effects such as oxidation, membrane disruption, osmoregulatory problems, lipid peroxidation, problems with cellular signalling and impairment of growth and protein synthesis were suggested. After UV-A exposure, no observable metabolic alterations were found in the anterior part of the eye in the present animal models.
8
Ayala, Marcelo. "Influence of exposure patterns and oxidation in UVR-induced cataract /." Stockholm : Karolinska institutet, 2005. http://diss.kib.ki.se/2005/91-7140-263-2/.
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9
Lam, Hoi-ching, and 林海清. "Dose modelling of the recoil effect of radon progeny attached aerosol in human respiratory tract by Monte Carlo method." PG_Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B45015570.
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Tell, Roger. "Description and prediction of clinical radiosensitivity : emphasis on normal tissue reactions /." Stockholm, 2004. http://diss.kib.ki.se/2003/91-7349-814-9.
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Книги з теми "Lungs Radiation injuries":

1
Consensus, Development Conference on the Treatment of Radiation Injuries (1st 1989 Washington D. C. ). Treatment of radiation injuries. New York: Plenum Press, 1990.
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2
Browne, Doris, Joseph F. Weiss, Thomas J. MacVittie, and Madhavan V. Pillai, eds. Treatment of Radiation Injuries. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4899-0864-3.
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3
Jahrestagung, Vereinigung Deutscher Strahlenschutzärzte. Strahlenreaktionen der Lunge: Hormesis : Richtlinie Strahlenschutz in der Medizin. Stuttgart: G. Fischer, 1994.
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4
Allman, Toney. Radiation sickness. Detroit: Lucent Books, a part of Gale, Cengage Learning, 2013.
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5
Scott, B. R. Models for pulmonary lethality and morbidity after irradiation from internal and external sources. Washington, DC: Division of Regulatory Applications, Office of Nuclear Regulatory Research, U.S. Nuclear Regulatory Commission, 1989.
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6
Jeremić, Branislav. Advances in radiation oncology in lung cancer. 2nd ed. Heidelberg: Springer, 2011.
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7
Jeremić, Branislav. Advances in radiation oncology in lung cancer. 2nd ed. Heidelberg: Springer, 2011.
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8
Inaba, Jirō, and Yūji Nakamura. Hōshasen ekigaku kenkyū no atarashii tenkai: Hōshasen no kenkō eikyō ni kansuru dētabēsu kōchiku ni mukete. Chiba-shi: Hōshasen Igaku Sōgō Kenkyūjo, 1997.
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9
Wilson, John W. Cellular repair/misrepair track model. Hampton, Va: Langley Research Center, 1991.
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10
Kongress, Gesellschaft fu r. Strahlenschutz Internationale. Die Wirkung niedriger Strahlendosen--im Kindes- und Jugendalter, in der Medizin, Umwelt und Technik, am Arbeitsplatz: Proceedings ; Internationaler Kongress Gesellschaft fu r Strahlenschutz e.V. (German Society for Radiation Protection), Westfa lische Wilhelms-Universita t Mu nster, 19.-21. Ma rz 1998. Bremen: Gesellschaft fu r Strahlenschutz, 2001.
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Частини книг з теми "Lungs Radiation injuries":

1
Knowlton, Christin A., Michelle Kolton Mackay, Tod W. Speer, Robyn B. Vera, Douglas W. Arthur, David E. Wazer, Rachelle Lanciano, et al. "Carcinoma of Lungs." In Encyclopedia of Radiation Oncology, 90. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-540-85516-3_1053.
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2
Kazzi, Ziad N. "Acute Radiation Injuries." In Critical Care Toxicology, 1–14. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-20790-2_32-1.
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3
Kazzi, Ziad N. "Acute Radiation Injuries." In Critical Care Toxicology, 605–17. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-17900-1_32.
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4
Gongora, R. "Accidental Radiation Injuries: Radiation Burns." In Emergency and Disaster Medicine, 347–54. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-69262-8_57.
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5
Bergentz, Sven-Erik, and David Bergqvist. "Radiation-induced Vascular Injuries." In Iatrogenic Vascular Injuries, 63–75. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74086-2_6.
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6
Cheng, Tianmin. "Combined Radiation–Burn Injuries." In Chinese Burn Surgery, 313–51. Dordrecht: Springer Netherlands, 2014. http://dx.doi.org/10.1007/978-94-017-8575-4_13.
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7
Gurelik, Gokhan, and Huseyin Baran Ozdemir. "Burns and Radiation Exposure." In Sports-related Eye Injuries, 85–93. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-13-9741-7_8.
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Schimpff, Stephen C. "Infections in Radiation Accidents." In Treatment of Radiation Injuries, 75–85. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4899-0864-3_8.
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9
Strambi, E. "Accidental Radiation Injuries: Whole-Body Radiation Syndromes." In Emergency and Disaster Medicine, 355–60. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-69262-8_58.
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10
Champlin, Richard. "Medical Assessment and Therapy in Bone Marrow Failure Due to Radiation Accidents." In Treatment of Radiation Injuries, 3–10. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4899-0864-3_1.
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Тези доповідей конференцій з теми "Lungs Radiation injuries":

1
Korotchenko, V. V. "Sports injuries." In Scientific trends: pedagogy and psychology. ЦНК МОАН, 2020. http://dx.doi.org/10.18411/sciencepublic-04-06-2020-11.
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2
Kobayashi, Yuya, Yoshiki Kawata, Noboru Niki, Keiji Umetani, Kurumi Saito, Toshihiro Okamoto, Hiroaki Sakai, Yasutaka Nakano, and Harumi Itho. "Bronchial based pulmonary acinus analysis in human lungs using a synchrotron radiation micro-CT." In Biomedical Applications in Molecular, Structural, and Functional Imaging, edited by Barjor Gimi and Andrzej Krol. SPIE, 2018. http://dx.doi.org/10.1117/12.2293428.
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3
Grushina, T. I. "Pharmacotherapy and magnetotherapy for early radiation damage to the lungs in cancer patients patients." In Arbat readings. Знание-М, 2020. http://dx.doi.org/10.38006/907345-01-0.2020.25.30.
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Rodionova, O., V. Voshedskiy, S. Vlasov, A. Solntseva, and M. Gusareva. "EP303 Combination treatment of radiation injuries of the rectum in patients with pelvic tumors." In ESGO Annual Meeting Abstracts. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/ijgc-2019-esgo.364.
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Sances, Anthony, Srirangam Kumaresan, David Daniels, and Keith Friedman. "Pediatric Airbag Injuries." In ASME 2002 International Mechanical Engineering Congress and Exposition. ASMEDC, 2002. http://dx.doi.org/10.1115/imece2002-32634.
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Анотація:
The advent of airbag technology has helped to reduce the injuries to belted occupants in motor vehicles during moderate to severe frontal and near frontal crashes [1–3]. Airbags have been in use since the early 1970s. As of July 2001, airbags have saved 7224 lives including 6066 drivers and 1158 front right passengers. However, the airbag deployments at low crash severity showed higher injury probability of occupants. The majority of airbag fatalities are associated with low speed impacts with deployments. As of July 2001, the National Highway Traffic Safety Administration (NHTSA) has reported 144 fatalities and serious life threatening injuries to children due to passenger airbags [4]. It is also reported that four children died and one child sustained life-threatening injury due to a driver side airbag. The publication from Transport Canada noted that the airbags increase the overall risk of injury of children under the age of 10 by approximately 21% [5]. Although the airbags have saved many lives, they are also responsible for fatalities and serious injuries during low speed severity collision. The present study reports pediatric airbag injuries sustained during low speed crashes.
6
Ohgami, Masatsugu, Nobuhiko Takai, Masahiko Watanabe, Koichi Ando, Akiko Uzawa, and Ryoichi Hirayama. "EFFECT OF N-METHYL-D-ASPARTATE RECEPTOR ANTAGONIST ON RADIATION-INDUCED GUT INJURIES IN MICE." In RAD Conference. RAD Association, 2017. http://dx.doi.org/10.21175/radproc.2017.02.
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7
Huelke, Donald F., Timothy W. Compton, and Charles P. Compton. "Lower Extremity Injuries in Frontal Crashes: Injuries, Locations, AIS and Contacts." In International Congress & Exposition. 400 Commonwealth Drive, Warrendale, PA, United States: SAE International, 1991. http://dx.doi.org/10.4271/910811.
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8
Liu, Xiao-Guang, Timon C. Liu, Jian-Ling Jiao, Cheng-Zhang Li, and Xiao-Yang Xu. "Photobiomodulation on sports injuries." In Third International Conference on Photonics and Imaging in Biology and Medicine, edited by Qingming Luo, Valery V. Tuchin, Min Gu, and Lihong V. Wang. SPIE, 2003. http://dx.doi.org/10.1117/12.546421.
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9
Johnson, Thomas E., and Ardith L. Wartick. "Laser injuries and deaths." In ILSC® 2003: Proceedings of the International Laser Safety Conference. Laser Institute of America, 2003. http://dx.doi.org/10.2351/1.5056520.
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10
Falcone, Giovanni, Raffaele Scurati, Francesca D'Elia, and Tiziana D'Isanto. "Basketball and ankle injuries." In Journal of Human Sport and Exercise - 2019 - Spring Conferences of Sports Science. Universidad de Alicante, 2019. http://dx.doi.org/10.14198/jhse.2019.14.proc4.79.
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Звіти організацій з теми "Lungs Radiation injuries":

1
Fine, Alan. Acute Lung Injury: Making Injured Lungs Perform Better and Rebuilding Healthy Lungs. Fort Belvoir, VA: Defense Technical Information Center, July 2010. http://dx.doi.org/10.21236/ada538317.
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2
Rogers, Peter H., Gary W. Caille, and Thomas N. Lewis. Response of the Lungs to Low Frequency Underwater Sound. Fort Belvoir, VA: Defense Technical Information Center, June 1994. http://dx.doi.org/10.21236/ada299456.
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3
Dartt, Darlene A. Molecular Solutions to Low Injuries Resulting from Battlefield Injuries. Fort Belvoir, VA: Defense Technical Information Center, May 2007. http://dx.doi.org/10.21236/ada472073.
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4
Robbins, E. S. Cellular morphometry of the bronchi of human and dog lungs. Office of Scientific and Technical Information (OSTI), September 1992. http://dx.doi.org/10.2172/6707808.
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5
Robbins, E. S. Cellular morphometry of the bronchi of human and dog lungs. Office of Scientific and Technical Information (OSTI), September 1991. http://dx.doi.org/10.2172/6262282.
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6
Traub, Richard J. Influence of Manufacturing Processes on the Performance of Phantom Lungs. Office of Scientific and Technical Information (OSTI), October 2008. http://dx.doi.org/10.2172/949146.
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7
Witten, Mark L. Research Training of the Effects of Toxic Substances on the Lungs. Fort Belvoir, VA: Defense Technical Information Center, May 1993. http://dx.doi.org/10.21236/ada267372.
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8
Witten, Mark L. Research Training of the Effects of Toxic Substances on the Lungs. Fort Belvoir, VA: Defense Technical Information Center, May 1995. http://dx.doi.org/10.21236/ada307408.
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9
Belkin, Michael, and N. Naveh. Laser Induced Retinal Injuries. Fort Belvoir, VA: Defense Technical Information Center, July 1991. http://dx.doi.org/10.21236/ada239046.
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10
Baum, Siegmund J. The Pathophysiology of Combined Radiation Injuries: A Review and Analysis of the Literature on Non-Human Research. Fort Belvoir, VA: Defense Technical Information Center, July 1991. http://dx.doi.org/10.21236/ada239981.
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