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Статті в журналах з теми "Maternal glucose levels"

Автор: Grafiati

Опубліковано: 3 червня 2025

Оновлено: 31 липня 2025

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1

WENDLING, PATRICE. "‘Normal’ Maternal Glucose Levels Up Adverse Outcomes." Family Practice News 37, no. 14 (2007): 1–2. http://dx.doi.org/10.1016/s0300-7073(07)70838-1.

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2

Hunt, Summer. "Maternal Glucose Levels and Congenital Heart Defects." Nursing for Women's Health 22, no. 2 (2018): 113. http://dx.doi.org/10.1016/s1751-4851(18)30092-8.

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3

Nordström, Lennart, S. Arulkumaran, Selina Chua, et al. "Continuous Maternal Glucose Infusion During Labor: Effects on Maternal and Fetal Glucose and Lactate Levels." American Journal of Perinatology 12, no. 05 (1995): 357–62. http://dx.doi.org/10.1055/s-2007-994496.

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4

Witek-Janusek, L. "Maternal ethanol ingestion: effect on maternal and neonatal glucose balance." American Journal of Physiology-Endocrinology and Metabolism 251, no. 2 (1986): E178—E184. http://dx.doi.org/10.1152/ajpendo.1986.251.2.e178.

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Liver glycogen availability in the newborn is of major importance for the maintenance of postnatal blood glucose levels. This study examined the effect of maternal ethanol ingestion on maternal and neonatal glucose balance in the rat. Female rats were placed on the Lieber-DeCarli liquid ethanol diet, an isocaloric liquid pair-fed diet, or an ad libitum rat chow diet at 3 wk before mating and throughout gestation. Blood and livers were obtained from dams and rat pups on gestational days 21 and 22. The pups were studied up to 6 h in the fasted state and up to 24 h in the fed state. Maternal etha

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5

Harwell, C. M., J. F. Padbury, R. S. Anand, et al. "Fetal catecholamine responses to maternal hypoglycemia." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 259, no. 6 (1990): R1126—R1130. http://dx.doi.org/10.1152/ajpregu.1990.259.6.r1126.

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The present studies were designed to determine the fetal catecholamine and metabolic responses to insulin-induced maternal hypoglycemia. Maternal hypoglycemia was induced by a primed constant infusion of insulin and glucose administered to pregnant ewes to maintain maternal glucose at 20-25 mg/dl. Maternal and fetal samples for measurement of catecholamine, glucose, and free fatty acid levels and arterial blood gas analysis were collected before insulin infusion and at intervals thereafter for 6 h. Maternal and fetal plasma catecholamine levels increased significantly in response to hypoglycem

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6

Farah, Nadine, Jennifer Hogan, Vicky O'Dwyer, Bernard Stuart, Mairead Kennelly, and Michael J. Turner. "Influence of Maternal Glycemia on Intrauterine Fetal Adiposity Distribution after a Normal Oral Glucose Tolerance Test at 28 Weeks Gestation." Experimental Diabetes Research 2011 (2011): 1–6. http://dx.doi.org/10.1155/2011/951203.

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Objective. To examine the relationship between maternal glucose levels and intrauterine fetal adiposity distribution in women with a normal oral glucose tolerance test (OGTT) at 28 weeks gestation.Study Design. We recruited 231 women with a singleton pregnancy. At 28 and 37 weeks gestation, sonographic measurements of fetal body composition were performed. Multiple regression analysis was used to study the influence of different maternal variables on fetal adiposity distribution.Results. Maternal glucose levels correlated with the fetal abdominal subcutaneous tissue measurements (; ) and with

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7

Renaud, Jennifer A., and Thomas R. Wigton. "Maternal Glucose Levels after a 75-g Oral Glucose Challenge and Neonatal Outcome." Obstetrics & Gynecology 99, Supplement (2002): 20S. http://dx.doi.org/10.1097/00006250-200204001-00038.

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8

RENAUD, J. "Maternal glucose levels after a 75-g oral glucose challenge and neonatal outcome." Obstetrics & Gynecology 99, no. 4 (2002): S20. http://dx.doi.org/10.1016/s0029-7844(02)01705-2.

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9

Eluzai, Gaddam Zion, and K. Poojitha. "Study of glucose levels in newborn with reference to hypoglycemia." International Journal of Contemporary Pediatrics 6, no. 6 (2019): 2661. http://dx.doi.org/10.18203/2349-3291.ijcp20194750.

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Background: Hypoglycemia is one of the common metabolic problems in neonatal medicine. Early diagnosis and treatment of neonatal hypoglycemia is important as many studies found that, hypoglycaemic episodes in neonates lead to neurodevelopmental and physical growth deficits. In this study, blood glucose levels at different time points were assessed and the influence of maternal blood glucose, mode of delivery, gestational age on neonatal blood glucose levels were studied.Methods: Blood glucose levels were low at 0 and 6th hour and maximum at 24th hour. The blood glucose levels ranged from 27 mg

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10

Lim, Ives Yubin, Xinyi Lin, Ai Ling Teh, et al. "Dichotomy in the Impact of Elevated Maternal Glucose Levels on Neonatal Epigenome." Journal of Clinical Endocrinology & Metabolism 107, no. 3 (2021): e1277-e1292. http://dx.doi.org/10.1210/clinem/dgab710.

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Abstract Context Antenatal hyperglycemia is associated with increased risk of future adverse health outcomes in both mother and child. Variations in offspring’s epigenome can reflect the impact and response to in utero glycemic exposure, and may have different consequences for the child. Objective We examined possible differences in associations of basal glucose status and glucose handling during pregnancy with both clinical covariates and offspring cord tissue DNA methylation. Research Design and Methods This study included 830 mother-offspring dyads from the Growing Up in Singapore Towards H

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11

Hoirisch-Clapauch, Silvia, Maria Amelia S. Porto, and Antonio E. Nardi. "May maternal lifestyle have an impact on neonatal glucose levels?" Medical Hypotheses 87 (February 2016): 80–86. http://dx.doi.org/10.1016/j.mehy.2015.11.017.

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12

Facco, Francesca, Douglas Scheftner, William Lowe, et al. "197: Circadian clock genes and maternal glucose levels in pregnancy." American Journal of Obstetrics and Gynecology 206, no. 1 (2012): S99—S100. http://dx.doi.org/10.1016/j.ajog.2011.10.215.

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13

Wang, Panchalli, Chung-Shing Wu, Chung-Yi Li, Chun-Pai Yang, and Mei-Chun Lu. "Seasonality of gestational diabetes mellitus and maternal blood glucose levels." Medicine 99, no. 41 (2020): e22684. http://dx.doi.org/10.1097/md.0000000000022684.

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14

Loy, See Ling, Tuck Seng Cheng, Marjorelee T. Colega, et al. "Predominantly night-time feeding and maternal glycaemic levels during pregnancy." British Journal of Nutrition 115, no. 9 (2016): 1563–70. http://dx.doi.org/10.1017/s0007114516000441.

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AbstractLittle is known about the influence of meal timing and energy consumption patterns throughout the day on glucose regulation during pregnancy. We examined the association of maternal feeding patterns with glycaemic levels among lean and overweight pregnant women. In a prospective cohort study in Singapore, maternal 24-h dietary recalls, fasting glucose (FG) and 2-h postprandial glucose (2HPPG) concentrations were measured at 26–28 weeks of gestation. Women (n 985) were classified into lean (BMI<23 kg/m2) or overweight (BMI≥23 kg/m2) groups. They were further categorised as predominan

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15

Guzzardi, Maria Angela, Elena Sanguinetti, Antonietta Bartoli, et al. "Elevated glycemia and brain glucose utilization predict BDNF lowering since early life." Journal of Cerebral Blood Flow & Metabolism 38, no. 3 (2017): 447–55. http://dx.doi.org/10.1177/0271678x17697338.

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Obesity and diabetes associate with neurodegeneration. Brain glucose and BDNF are fundamental in perinatal development. BDNF is related to brain health, food intake and glucose metabolism. We characterized the relationship between glycemia and/or brain glucose utilization (by 18FDG-PET during fasting and glucose loading), obesity and BDNF in 4-weeks old (pre-obese) and 12-weeks old (obese) Zucker fa/fa rats, and their age-matched fa/+ controls. In 75 human infants, we assessed cord blood BDNF and glucose levels, appetite regulating hormones, body weight and maternal factors. Young and adult fa

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16

Al-Ramahi, Osamah. "Gestational hyperglycemia: a comprehensive overview of multiple defects and neurological disorders." Journal of Neurology & Stroke 14, no. 6 (2024): 186–89. https://doi.org/10.15406/jnsk.2024.14.00606.

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Gestational hyperglycemia, characterized by elevated blood glucose levels during pregnancy, poses significant risks to fetal development, particularly in relation to neural tube defects (NTDs) and other neurological disorders. This overview synthesizes findings from literature to explore the pathophysiological mechanisms by which maternal hyperglycemia influences fetal neurodevelopment. The review focuses on studies that demonstrate how elevated maternal glucose levels lead to fetal hyperinsulinemia, oxidative stress, and impaired placental function, thereby contributing to the risk of congeni

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17

Hassan, Manal Matlab, and Risala A. Ali. "Evaluation of maternal serum prolactin in pregnant women with gestational Diabetes mellitus." Journal of University of Shanghai for Science and Technology 23, no. 10 (2021): 195–205. http://dx.doi.org/10.51201/jusst/21/10738.

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Maintenance of normal glucose metabolism during pregnancy is of particular importance. Prolactin may be a mediator in pathogenesis of impaired glucose metabolism. This study aimed to assess the levels of maternal serum prolactin in pregnant women with gestational Diabetes mellitus (GDM). This prospective observational study was done at AL– Emamain AL- Kadhemain Medical City/ Department of Obstetrics and Gynecology in Baghdad/ Iraq during the period from 1st March to 1st October 2018 on 150 pregnant women who were referred for OGTT for different indications. For all included women, maternal ser

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18

McGowan, Ciara A., and Fionnuala M. McAuliffe. "The influence of maternal glycaemia and dietary glycaemic index on pregnancy outcome in healthy mothers." British Journal of Nutrition 104, no. 2 (2010): 153–59. http://dx.doi.org/10.1017/s0007114510000425.

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Infant birth weight has increased in Ireland in recent years along with levels of childhood overweight and obesity. The present article reviews the current literature on maternal glycaemia and the role of the dietary glycaemic index (GI) and its impact on pregnancy outcomes. It is known that maternal weight and weight gain significantly influence infant birth weight. Fetal macrosomia (birth weight >4000 g) is associated with an increased risk of perinatal trauma to both mother and infant. Furthermore, macrosomic infants have greater risk of being obese in childhood, adolescence and adulthoo

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19

Jian Yun, Xu, Liang Zhaoxia, Chai Yun, Fang Qin, Chen Yuanyuan, and Chen Danqing. "Changes in Maternal Glucose Metabolism after the Administration of Dexamethasone for Fetal Lung Development." International Journal of Endocrinology 2012 (2012): 1–3. http://dx.doi.org/10.1155/2012/652806.

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Aims. Antenatal dexamethasone administration for fetal lung development may impair maternal glucose tolerance. In this study, we investigated whether glucose and insulin levels differed among singleton and twin pregnancies and pregnancies with impaired glucose tolerance (IGT) after treatment with dexamethasone.Methods. Singleton pregnancies, twin pregnancies, and pregnancies with IGT between 28 and 33 weeks of gestation whose mothers were treated with dexamethasone were enrolled in this study. Exclusion criteria included gestational hypertension, diabetes, renal disorders, and infectious disea

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20

Hani, Diab, Omar Altal, Ala Alhowary, et al. "The Perioperative Neonatal and Maternal Glycemic Response and APGAR Score During Elective Cesarean Section: Factors and Anesthetic Management." Medical Archives 79, no. 1 (2025): 34. https://doi.org/10.5455/medarh.2025.79.34-40.

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Background: The type of anesthesia and intensity of pain experienced during surgery are linked to the body’s stress response, as reflected in preoperative and postoperative glucose levels. Objective: This comparative study aims to assess the hyperglycemic stress response to cesarean sections performed under different types of anesthesia. Methods: This prospective study included 302 participants, divided into two groups: a general anesthesia group and a spinal anesthesia group. Our primary objective was to investigate the effects of general versus spinal anesthesia on pregnant women undergoing

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21

Romern, Yitzhak, Alva Loven, Galila Agam, and Joseph R. Leiberman. "The temporal relationship between maternal blood and amniotic fluid glucose levels." American Journal of Obstetrics and Gynecology 168, no. 2 (1993): 611–14. http://dx.doi.org/10.1016/0002-9378(93)90504-c.

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22

Zhao, Jia-Yi, Qing Yu, Xiao-Ming Yang, and Xiang-Xin Li. "The correlation between the prevalence of gestational diabetes mellitus and maternal age in Southern China." LaboratoriumsMedizin 46, no. 1 (2021): 33–37. http://dx.doi.org/10.1515/labmed-2021-0068.

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Abstract Objectives The present study aimed to survey the prevalence of gestational diabetes mellitus (GDM) in Southern China and further to analyze the correlation between the prevalence of GDM and maternal age. Methods A retrospective cross-sectional study was carried out at the Southern Medical University Affiliated Maternal & Child Health Hospital of Foshan, Foshan, China between January and April 2020. Oral glucose tolerance tests (OGTT) was performed, using a 75 g glucose load and venous samples were drawn at 0 h, 1 h and 2 h at 24–28 weeks of gestation. GDM was diagnosed by Internat

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23

Lesage, J., D. Hahn, M. Leonhardt, B. Blondeau, B. Breant, and JP Dupouy. "Maternal undernutrition during late gestation-induced intrauterine growth restriction in the rat is associated with impaired placental GLUT3 expression, but does not correlate with endogenous corticosterone levels." Journal of Endocrinology 174, no. 1 (2002): 37–43. http://dx.doi.org/10.1677/joe.0.1740037.

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Fetal intrauterine growth restriction (IUGR) is a frequently occurring and serious complication of pregnancy. Infants exposed to IUGR are at risk for numerous perinatal morbidities, including hypoglycemia in the neonatal period, as well as increased risk of later physical and/or mental impairments, cardiovascular disease and non-insulin-dependent diabetes mellitus. Fetal growth restriction most often results from uteroplacental dysfunction during the later stage of pregnancy. As glucose, which is the most abundant nutrient crossing the placenta, fulfills a large portion of the fetal energy req

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24

Saleh, Jumana, Lovina Machado, and Zahra Razvi. "2-Hour Postload Serum Glucose Levels and Maternal Blood Pressure as Independent Predictors of Birth Weight in “Appropriate for Gestational Age” Neonates in Healthy Nondiabetic Pregnancies." BioMed Research International 2013 (2013): 1–7. http://dx.doi.org/10.1155/2013/757459.

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Introduction. Increased neonatal birth weight (NBW), often associated with diabetic pregnancies, is a recognized indicator of childhood obesity and future metabolic risk. Predictors of NBW in healthy non-diabetic pregnancies are not yet established. Here, we investigated the association of maternal parameters of healthy non-diabetic mothers with NBW of their “appropriate-for-gestational age” neonates.Methods. The study involved 36 healthy mother/infant pairs. Examined parameters included NBW, maternal age, first and last trimester (BMI), weight gain, fasting serum lipids and glucose, 2-hour po

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25

Das, Utpala G., H. Farouk Sadiq, Michael J. Soares, William W. Hay, and Sherin U. Devaskar. "Time-dependent physiological regulation of rodent and ovine placental glucose transporter (GLUT-1) protein." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 274, no. 2 (1998): R339—R347. http://dx.doi.org/10.1152/ajpregu.1998.274.2.r339.

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To examine the in vivo and in vitro time-dependent effects of glucose on placental glucose transporter (GLUT-1) protein levels, we employed Western blot analysis using placenta from the short-term streptozotocin-induced diabetic pregnancy (STZ-D), uterine artery ligation-intrauterine growth restriction (IUGR) rat models, pregnant sheep exposed to chronic maternal glucose and insulin infusions, and the HRP.1 rat trophoblastic cell line exposed to differing concentrations of glucose. In the rat, 6 days of STZ-D with maternal and fetal hyperglycemia caused no substantive change, whereas 72 h of I

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26

Zöllkau, Janine, Laura Swiderski, Alexander Schmidt, et al. "The Relationship between Gestational Diabetes Metabolic Control and Fetal Autonomic Regulation, Movement and Birth Weight." Journal of Clinical Medicine 10, no. 15 (2021): 3378. http://dx.doi.org/10.3390/jcm10153378.

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(1) Background: Maternal metabolic control in gestational diabetes is suggested to influence fetal autonomic control and movement activity, which may have fetal outcome implications. We aimed to analyze the relationship between maternal metabolic control, fetal autonomic heart rate regulation, activity and birth weight. (2) Methods: Prospective noninterventional longitudinal cohort monitoring study accompanying 19 patients with specialist clinical care for gestational diabetes. Monthly fetal magnetocardiography with electro-physiologically-based beat-to-beat heart rate recording for analysis o

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27

Smith, F. G., and E. R. Lumbers. "Effects of maternal hyperglycemia on fetal renal function in sheep." American Journal of Physiology-Renal Physiology 255, no. 1 (1988): F11—F14. http://dx.doi.org/10.1152/ajprenal.1988.255.1.f11.

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The effects of maternal hyperglycemia on fetal renal function were investigated in 10 chronically catheterized fetal sheep after the infusion of 100 g of glucose into the ewe over 30 min. Fetal blood glucose levels rose (P less than 0.001) within 15 min of completing the glucose infusion from 15.75 +/- 2.8 to 195.4 +/- 18 (SE) mg/dl (n = 10). There was a significant increase in fetal glomerular filtration rate (P less than 0.05) from 2.73 +/- 0.41 to 3.65 +/- 0.40 (SE) ml/min (n = 10) within 1.5 h of the infusion of glucose into the ewe. Urine flow rate increased from 0.38 +/- 0.06 to 0.63 +/-

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28

Liu, Zhihua, Mingming Miao, Zhenjie Li, et al. "Effects of Phellinus vaninii Ljup Polysaccharide on the Diabetes Induced by Streptozotocin and Cigarette Smoking in Maternal Rats and Their Offspring." Journal of Biobased Materials and Bioenergy 16, no. 3 (2022): 526–31. http://dx.doi.org/10.1166/jbmb.2022.2196.

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This research was carried out to determine the influences of cigarette smoking and the hypoglycemic effects of Phellinus vaninii Ljup polysaccharide (PS) on STZ (streptozotocin)-induced severe hyperglycemia maternal rats, as well as the postnatal development of their offspring. The polysaccharide was administered orally and the cigarette smoke (CS) exposure was performed daily for four weeks. The food efficiency ratio, food intake, body weight gain, organ weights, and blood glucose level were tested for the maternal rats, and the body weight and glucose tolerance tests were evaluated for their

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29

Clapp III, James F. "Maternal carbohydrate intake and pregnancy outcome." Proceedings of the Nutrition Society 61, no. 1 (2002): 45–50. http://dx.doi.org/10.1079/pns2001129.

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Experimental evidence indicates that the primary maternal environmental factor that regulates feto–placental growth is substrate delivery to the placental site, which is the product of maternal substrate levels and the rate of placental-bed blood flow. Thus, maternal factors which change either substrate level or flow alter feto–placental growth rate. The best-studied substrate in human pregnancy is glucose, and there is a direct relationship between maternal blood glucose levels and size at birth. Altering the type of carbohydrate eaten (high- v. low-glycaemic sources) changes postprandial gl

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30

Liu, Gongshu, Nan Li, Shurong Sun, et al. "Maternal OGTT Glucose Levels at 26–30 Gestational Weeks with Offspring Growth and Development in Early Infancy." BioMed Research International 2014 (2014): 1–11. http://dx.doi.org/10.1155/2014/516980.

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Aims. We aim to evaluate the association of maternal gestational oral glucose tolerance test (OGTT) glucose concentrations with anthropometry in the offspring from birth to 12 months in Tianjin, China.Methods. A total of 27,157 pregnant women underwent OGTT during 26–30 weeks gestation, and their children had body weight/length measured from birth to 12 months old.Results. Maternal OGTT glucose concentrations at 26–30 gestational weeks were positively associated with Z-scores for birth length-for-gestational age and birth weight-for-length. Compared with infants born to mothers with normal glu

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31

Farida, Erye Frety Endyka, Andriyanti, and Atika. "The most influencing factors of the incidence of Preeclamsia at Public Health Center Sidosermo Surabaya." World Journal of Advanced Research and Reviews 23, no. 1 (2024): 738–45. https://doi.org/10.5281/zenodo.14786551.

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Maternal Mortality Rate in Indonesia is the highest in Southeast Asian countries. The main reason of maternal mortality in Indonesia is preeclampsia, which is the prevalention up to 5,3%. Aims : to analyze the risk factors of preeclampsia at Public Health Center Sidosermo Surabaya in 2023. Methods: This research method is analytic observational with case control design. The number of samples is 93 by using simple random sampling, which are 31 samples cases and 62 samples control. Independent variables in this research are age, BMI, frequency of ANC, hemoglobin levels in first trimester, high g

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32

Godang, Kristin, Kathrine Frey Frøslie, Tore Henriksen, Elisabeth Qvigstad, and Jens Bollerslev. "Seasonal variation in maternal and umbilical cord 25(OH) vitamin D and their associations with neonatal adiposity." European Journal of Endocrinology 170, no. 4 (2014): 609–17. http://dx.doi.org/10.1530/eje-13-0842.

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DesignNeonatal body fat is an important indicator of foetal energy supply and growth with potential importance for long-term health. In this study, we wanted to explore seasonal variation of 25-hydroxy-vitamin D (25(OH)D) in maternal and umbilical cord plasma (UCP) to examine whether maternal and foetal 25(OH)D levels were associated with maternal BMI and neonatal fat mass (FM), and to explore the relationship among maternal and neonatal 25(OH)D levels, maternal glucose/insulin levels and UCP C-peptide.MethodsAn observational, prospective study of determinants of foetal growth and birth weight

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Irene, Arockia Silviya, and Anthikat Sankaranarayanan Lata. "Deceptive Late Maternal Euglycaemia." Journal of the Endocrine Society 5, Supplement_1 (2021): A433—A434. http://dx.doi.org/10.1210/jendso/bvab048.884.

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Abstract Background: Strict glycemic control is the most critical factor determining outcome in Gestational Diabetes mellitus. But having capillary blood glucose readings within the target levels, in the late trimester may be deceptive. If fetal hyperinsulinaemia and fetopathy have already set in, maternal glucose may be spuriously normal. We support this hypothesis based on our experience with 4 GDM patients who apparently reported normal blood glucose in late trimester, yet ended up with neonatal complications. Materials and Methods: Records of mothers of four neonates (Infant of diabetic mo

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Zhao, Bai-Hui, Ying Jiang, Hong Zhu, et al. "Placental Delta-Like 1 Gene DNA Methylation Levels Are Related to Mothers’ Blood Glucose Concentration." Journal of Diabetes Research 2019 (December 11, 2019): 1–7. http://dx.doi.org/10.1155/2019/9521510.

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Purpose. We aim to identify the methylation status of delta-like 1 (DLK1) in the placenta and the correlation between DLK1 methylation and maternal serum glucose level and fetal birth weight. Methods. We analyzed the gene expression of DLK1 gene in both maternal and fetal sides of the placenta in a GDM group (n=15) and a control group (n=15) using real-time polymerase chain reaction. With MethylTargetTM technique, we detected the methylation status of DLK1 promotor in the placenta. Furthermore, Pearson’s correlation was used to confirm the association of methylation alteration of DLK1 promoter

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Li, Weijia. "Effect of Individualized Care for Pregnant Women with Diabetes Mellitus." Journal of Medicine and Health Science 2, no. 2 (2024): 1–5. http://dx.doi.org/10.62517/jmhs.202405201.

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Gestational diabetes mellitus (GDM) is the first occurrence of diabetes mellitus after pregnancy due to abnormal maternal glucose metabolism pregnant women with obvious clinical symptoms, but they are prone to maternal infection, polyhydramnios, macrosomia, etc. A series of adverse pregnancy outcomes cause physical and mental harm to pregnant women and their families. Individualized care refers to the development of a care plan tailored to the needs of the individual in order to improve the effectiveness and adherence of treatment. The effect of individualized care on maternal blood glucose le

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Sloboda, DM, and JP Newnham. "Repeated maternal glucocorticoid administration and the developing liver in fetal sheep." Journal of Endocrinology 175, no. 2 (2002): 535–43. http://dx.doi.org/10.1677/joe.0.1750535.

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Prenatal glucocorticoid exposure has been associated with a reduction in birth weight and postnatal alterations in glucose homeostasis and hypothalamic-pituitary-adrenal (HPA) axis function. The mechanisms underlying these responses are unknown, although changes in fetal hepatic development may play an important role. The fetal liver produces key regulators of fuel metabolism and of the developing HPA axis that are altered by glucocorticoids. The local availability of glucocorticoids is regulated, in part, by corticosteroid-binding protein (CBG), glucocorticoid receptors (GR) and by the enzyme

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Ovadia, Yaniv S., Dov Gefel, Yoel Toledano, et al. "Does Iodine Intake Modify the Effect of Maternal Dysglycemia on Birth Weight in Mild-to-Moderate Iodine-Deficient Populations? A Mother–Newborn Prospective Cohort Study." Nutrients 15, no. 13 (2023): 2914. http://dx.doi.org/10.3390/nu15132914.

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It is unclear how maternal glycemic status and maternal iodine status influence birth weight among individuals with mild-to-moderate iodine deficiency (ID). We studied the association between birth weight and both maternal glucose levels and iodine intake among pregnant women with mild-to-moderate ID. Glucose values were assessed using a glucose challenge test (GCT) and non-fasting glucose levels that were determined before delivery; individuals’ iodine statuses were assessed using an iodine food frequency questionnaire; and serum thyroglobulin (Tg) and urinary iodine concentrations (UIC) were

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Patel, Vaishali Nanranbhai, Toral M. Goswami, Neha H. Pandya, and Amita K. Mevada. "Maternal postprandial blood glucose levels and its relation with the pregnancy outcomes." International Journal of Biomedical and Advance Research 5, no. 3 (2014): 174. http://dx.doi.org/10.7439/ijbar.v5i3.702.

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Helle, Samuli, Toni Laaksonen, and Otso Huitu. "Sex-specific offspring growth according to maternal testosterone, corticosterone, and glucose levels." Behavioral Ecology 24, no. 1 (2012): 205–12. http://dx.doi.org/10.1093/beheco/ars155.

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Priest, James R., Wei Yang, Gerald Reaven, Joshua W. Knowles, and Gary M. Shaw. "Maternal Midpregnancy Glucose Levels and Risk of Congenital Heart Disease in Offspring." JAMA Pediatrics 169, no. 12 (2015): 1112. http://dx.doi.org/10.1001/jamapediatrics.2015.2831.

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Boggess, Kim A., Arielle Valint, Jerrie S. Refuerzo, et al. "Metformin Plus Insulin for Preexisting Diabetes or Gestational Diabetes in Early Pregnancy: The MOMPOD Randomized Clinical Trial." Obstetrical & Gynecological Survey 79, no. 6 (2024): 322–24. http://dx.doi.org/10.1097/01.ogx.0001025796.16668.73.

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(Abstracted from JAMA 2023;330(22):2182–2190) Preexisting type 2 diabetes in pregnant people increases the risk for adverse pregnancy outcomes, intrapartum complications, and neonatal hypoglycemia at birth. To achieve normal maternal glucose levels and optimize pregnancy outcomes, management includes maternal blood glucose monitoring, dietary management, and insulin therapy.

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Pickard, MR, AJ Leonard, LM Ogilvie, et al. "Maternal hypothyroidism in the rat influences placental and liver glycogen stores: fetal growth retardation near term is unrelated to maternal and placental glucose metabolic compromise." Journal of Endocrinology 176, no. 2 (2003): 247–55. http://dx.doi.org/10.1677/joe.0.1760247.

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Maternal hypothyroidism impairs fetal growth in the rat, but the mechanisms by which this occurs are unknown. Since the fetus derives its glucose supply from the mother, and maternal thyroidectomy may disturb maternal and placental glucose metabolism, we postulated that maternal and/or placental glucose metabolic compromise may contribute to fetal growth retardation in hypothyroid dams. Feto-placental growth, tissue glycogen stores and glucose levels in sera and amniotic fluid were determined in rat dams partially thyroidectomized (TX) before pregnancy and in euthyroid controls. Fetal body wei

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Stern, Christina, Sarah Schwarz, Gerit Moser, et al. "Placental Endocrine Activity: Adaptation and Disruption of Maternal Glucose Metabolism in Pregnancy and the Influence of Fetal Sex." International Journal of Molecular Sciences 22, no. 23 (2021): 12722. http://dx.doi.org/10.3390/ijms222312722.

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The placenta is an endocrine fetal organ, which secretes a plethora of steroid- and proteo-hormones, metabolic proteins, growth factors, and cytokines in order to adapt maternal physiology to pregnancy. Central to the growth of the fetus is the supply with nutrients, foremost with glucose. Therefore, during pregnancy, maternal insulin resistance arises, which elevates maternal blood glucose levels, and consequently ensures an adequate glucose supply for the developing fetus. At the same time, maternal β-cell mass and function increase to compensate for the higher insulin demand. These adaptati

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Jensen, Vivi F. H., Anne-Marie Mølck, Jette Nowak, et al. "Placental nutrient transporters adapt during persistent maternal hypoglycaemia in rats." PLOS ONE 17, no. 3 (2022): e0265988. http://dx.doi.org/10.1371/journal.pone.0265988.

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Maternal malnutrition is associated with decreased nutrient transfer to the foetus, which may lead to foetal growth restriction, predisposing children to a variety of diseases. However, regulation of placental nutrient transfer during decreased nutrient availability is not fully understood. In the present study, the aim was to investigate changes in levels of placental nutrient transporters accompanying maternal hypoglycaemia following different durations and stages of gestation in rats. Maternal hypoglycaemia was induced by insulin-infusion throughout gestation until gestation day (GD)20 or u

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Dingena, Cassy, Melvin Holmes, Eleanor Scott, and Michael Zulyniak. "Individualised PatieNt Care and Treatment FOR MatErnal Diabetes (INFORMED); Intensive Profiling of Dysglycemia in Maternal Diabetes – A Longitudinal Study Protocol." Current Developments in Nutrition 6, Supplement_1 (2022): 635. http://dx.doi.org/10.1093/cdn/nzac061.019.

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Abstract Objectives Women with pre-existing diabetes struggle to control glucose levels during pregnancy and are at high risk of pregnancy complications, compared to women without diabetes. Management of postprandial and nocturnal glucose levels are key targets to minimise risk of adverse events during pregnancy. This trial aims to investigate the effect of distinct nutrient patterns on 24-hr glucose levels in pregnant women with pre-existing diabetes to inform future strategies. Methods This single-blind randomised controlled trial will recruit 76 pregnant women with pre-existing Type 1 or Ty

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Nteeba, Jackson, Kaiyu Kubota, Wenfang Wang, et al. "Pancreatic prolactin receptor signaling regulates maternal glucose homeostasis." Journal of Endocrinology 241, no. 1 (2019): 71–83. http://dx.doi.org/10.1530/joe-18-0518.

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Prolactin (PRL) signaling has been implicated in the regulation of glucose homeostatic adaptations to pregnancy. In this report, the PRL receptor (Prlr) gene was conditionally disrupted in the pancreas, creating an animal model which proved useful for investigating the biology and pathology of gestational diabetes including its impacts on fetal and placental development. In mice, pancreatic PRLR signaling was demonstrated to be required for pregnancy-associated changes in maternal β cell mass and function. Disruption of the Prlr gene in the pancreas resulted in fewer insulin-producing cells, w

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Wei, Luxin, Xiaojing Li, and Kun He. "Clinical benefit of dexmedetomidine in combination with dezocine as epidural anesthesia during cesarean delivery for pregnant women with gestational diabetes mellitus." Tropical Journal of Pharmaceutical Research 21, no. 12 (2023): 2707–13. http://dx.doi.org/10.4314/tjpr.v21i12.28.

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Purpose: To investigate the clinical benefit of dexmedetomidine (DEX) when used in combination with dezocine as epidural anesthesia during cesarean delivery for puerperae with gestational diabetes mellitus (GDM).Methods: A total of 120 puerperae with GDM admitted to The Fourth Hospital of Shijiazhuang (Obstetrics and Gynecology Hospital Affiliated to Hebei Medical University, Shijiazhuang City, China) who underwent cesarean delivery from January 2019 to January 2020 were randomly assigned to groups A and B, with 60 patients per group. Epidural anesthesia with dezocine was used on patients in b

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Gleason, Brooke, Alan Kuang, James R. Bain, et al. "Association of Maternal Metabolites and Metabolite Networks with Newborn Outcomes in a Multi-Ancestry Cohort." Metabolites 13, no. 4 (2023): 505. http://dx.doi.org/10.3390/metabo13040505.

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The in utero environment is important for newborn size at birth, which is associated with childhood adiposity. We examined associations between maternal metabolite levels and newborn birthweight, sum of skinfolds (SSF), and cord C-peptide in a multinational and multi-ancestry cohort of 2337 mother–newborn dyads. Targeted and untargeted metabolomic assays were performed on fasting and 1 h maternal serum samples collected during an oral glucose tolerance test performed at 24–32 week gestation in women participating in the Hyperglycemia and Adverse Pregnancy Outcome (HAPO) Study. Anthropometric m

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Shi, Xiulin, Peiying Huang, Liying Wang, et al. "Maternal postload 1-hour glucose level during pregnancy and offspring’s overweight/obesity status in preschool age." BMJ Open Diabetes Research & Care 8, no. 1 (2020): e000738. http://dx.doi.org/10.1136/bmjdrc-2019-000738.

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BackgroundChildhood obesity is associated with adverse outcomes such as metabolic syndrome, diabetes, and cardiovascular diseases in adulthood. Identifying risk factors related to excessive adiposity in early childhood is of great importance for obesity intervention. The results of studies for associations between maternal with gestational diabetes and offspring obesity are conflicting. Nonetheless, the association of maternal glucose across a spectrum of glucose values with childhood adiposity outcomes is less clear.AimTo assess the association of maternal glucose across a spectrum of glucose

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Talton, Omonseigho O., Kathleen A. Pennington, Kelly E. Pollock, et al. "Maternal Hyperleptinemia Improves Offspring Insulin Sensitivity in Mice." Endocrinology 157, no. 7 (2016): 2636–48. http://dx.doi.org/10.1210/en.2016-1039.

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Maternal obesity and gestational diabetes are prevalent worldwide. Offspring of mothers with these conditions weigh more and are predisposed to metabolic syndrome. A hallmark of both conditions is maternal hyperleptinemia, but the role of elevated leptin levels during pregnancy on developmental programming is largely unknown. We previously found that offspring of hyperleptinemic mothers weighed less and had increased activity. The goal of this study was to determine whether maternal leptin affects offspring insulin sensitivity by investigating offspring glucose metabolism and lipid accumulatio

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