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Готові списки джерел за темами / Muscle deoxygenation

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Добірка наукової літератури з теми "Muscle deoxygenation"

Автор: Grafiati

Опубліковано: 10 січня 2023

Оновлено: 28 січня 2023

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Статті в журналах з теми "Muscle deoxygenation"

1

Goulding, Richie P., Dai Okushima, Simon Marwood, David C. Poole, Thomas J. Barstow, Tze-Huan Lei, Narihiko Kondo, and Shunsaku Koga. "Impact of supine exercise on muscle deoxygenation kinetics heterogeneity: mechanistic insights into slow pulmonary oxygen uptake dynamics." Journal of Applied Physiology 129, no. 3 (September 1, 2020): 535–46. http://dx.doi.org/10.1152/japplphysiol.00213.2020.

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Анотація:

We show that supine exercise causes a greater degree of muscle deoxygenation in both deep and superficial muscle and increases the spatial heterogeneity of muscle deoxygenation. Therefore, this study suggests that any O2 delivery gradient toward deep versus superficial muscle is insufficient to mitigate impairments in oxidative function in response to reduced whole muscle O2 delivery. More heterogeneous muscle deoxygenation is associated with slower V̇o2 kinetics.

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2

Koga, Shunsaku, Yutaka Kano, Thomas J. Barstow, Leonardo F. Ferreira, Etsuko Ohmae, Mizuki Sudo, and David C. Poole. "Kinetics of muscle deoxygenation and microvascular Po2 during contractions in rat: comparison of optical spectroscopy and phosphorescence-quenching techniques." Journal of Applied Physiology 112, no. 1 (January 1, 2012): 26–32. http://dx.doi.org/10.1152/japplphysiol.00925.2011.

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Анотація:

The overarching presumption with near-infrared spectroscopy measurement of muscle deoxygenation is that the signal reflects predominantly the intramuscular microcirculatory compartment rather than intramyocyte myoglobin (Mb). To test this hypothesis, we compared the kinetics profile of muscle deoxygenation using visible light spectroscopy (suitable for the superficial fiber layers) with that for microvascular O2 partial pressure (i.e., PmvO2, phosphorescence quenching) within the same muscle region (0.5∼1 mm depth) during transitions from rest to electrically stimulated contractions in the gastrocnemius of male Wistar rats ( n = 14). Both responses could be modeled by a time delay (TD), followed by a close-to-exponential change to the new steady level. However, the TD for the muscle deoxygenation profile was significantly longer compared with that for the phosphorescence-quenching PmvO2 [8.6 ± 1.4 and 2.7 ± 0.6 s (means ± SE) for the deoxygenation and PmvO2, respectively; P < 0.05]. The time constants (τ) of the responses were not different (8.8 ± 4.7 and 11.2 ± 1.8 s for the deoxygenation and PmvO2, respectively). These disparate (TD) responses suggest that the deoxygenation characteristics of Mb extend the TD, thereby increasing the duration (number of contractions) before the onset of muscle deoxygenation. However, this effect was insufficient to increase the mean response time. Somewhat differently, the muscle deoxygenation response measured using near-infrared spectroscopy in the deeper regions (∼5 mm depth) (∼50% type I Mb-rich, highly oxidative fibers) was slower (τ = 42.3 ± 6.6 s; P < 0.05) than the corresponding value for superficial muscle measured using visible light spectroscopy or PmvO2 and can be explained on the basis of known fiber-type differences in PmvO2 kinetics. These data suggest that, within the superficial and also deeper muscle regions, the τ of the deoxygenation signal may represent a useful index of local O2 extraction kinetics during exercise transients.

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3

Lusina, Sarah-Jane C., Darren E. R. Warburton, Nicola G. Hatfield, and A. William Sheel. "Muscle deoxygenation of upper-limb muscles during progressive arm-cranking exercise." Applied Physiology, Nutrition, and Metabolism 33, no. 2 (April 2008): 231–38. http://dx.doi.org/10.1139/h07-156.

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Анотація:

The purpose of this study was to determine which upper-limb muscle exhibits the greatest change in muscle deoxygenation during arm-cranking exercise (ACE). We hypothesized that the biceps brachii (BB) would show the greatest change in muscle deoxygenation during progressive ACE to exhaustion relative to triceps brachii (TR), brachioradialis (BR), and anterior deltoid (AD). Healthy young men (n = 11; age = 27 ± 1 y; mean ± SEM) performed an incremental ACE test to exhaustion. Near-infrared spectroscopy (NIRS) was used to monitor the relative concentration changes in oxy- (O2Hb), deoxy- (HHb), and total hemoglobin (Hbtot), as well as tissue oxygenation index (TOI) in each of the 4 muscles. During submaximal arm exercise, we found that changes to NIRS-derived measurements were not different between the 4 muscles studied (p > 0.05). At maximal exercise HHb was significantly higher in the BB compared with AD (p < 0.05). Relative to the other 3 muscles, BB exhibited the greatest decrease in O2Hb and TOI (p < 0.05). Our investigation provides two new and important findings: (i) during submaximal ACE the BB, TR, BR, and AD exhibit similar changes in muscle deoxygenation and (ii) during maximal ACE the BB exhibits the greatest change in intramuscular O2 status.

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4

Chin, Lisa M. K., John M. Kowalchuk, Thomas J. Barstow, Narihiko Kondo, Tatsuro Amano, Tomoyuki Shiojiri, and Shunsaku Koga. "The relationship between muscle deoxygenation and activation in different muscles of the quadriceps during cycle ramp exercise." Journal of Applied Physiology 111, no. 5 (November 2011): 1259–65. http://dx.doi.org/10.1152/japplphysiol.01216.2010.

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Анотація:

The relationship between muscle deoxygenation and activation was examined in three different muscles of the quadriceps during cycling ramp exercise. Seven young male adults (24 ± 3 yr; mean ± SD) pedaled at 60 rpm to exhaustion, with a work rate (WR) increase of 20 W/min. Pulmonary oxygen uptake was measured breath-by-breath, while muscle deoxygenation (HHb) and activity were measured by time-resolved near-infrared spectroscopy (NIRS) and surface electromyography (EMG), respectively, at the vastus lateralis (VL), rectus femoris (RF), and vastus medialis (VM). Muscle deoxygenation was corrected for adipose tissue thickness and normalized to the amplitude of the HHb response, while EMG signals were integrated (iEMG) and normalized to the maximum iEMG determined from maximal voluntary contractions. Muscle deoxygenation and activation were then plotted as a percentage of maximal work rate (%WRmax). The HHb response for all three muscle groups was fitted by a sigmoid function, which was determined as the best fitting model. The c/d parameter for the sigmoid fit (representing the %WRmax at 50% of the total amplitude of the HHb response) was similar between VL (47 ± 12% WRmax) and VM (43 ± 11% WRmax), yet greater ( P < 0.05) for RF (65 ± 13% WRmax), demonstrating a “right shift” of the HHb response compared with VL and VM. The iEMG also showed that muscle activation of the RF muscle was lower ( P < 0.05) compared with VL and VM throughout the majority of the ramp exercise, which may explain the different HHb response in RF. Therefore, these data suggest that the sigmoid function can be used to model the HHb response in different muscles of the quadriceps; however, simultaneous measures of muscle activation are also needed for the HHb response to be properly interpreted during cycle ramp exercise.

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5

Ambrozic, Jana, Mitja Lainscak, and Matej Podbregar. "Use of Near Infrared Spectroscopy to Asses Remote Ischemic Preconditioning in Skeletal Muscle." Physiology Journal 2013 (February 21, 2013): 1–7. http://dx.doi.org/10.1155/2013/154327.

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Анотація:

Remote ischemic preconditioning (IPC) is a procedure during which brief periods of ischemia protect distant organ from ischemia-reperfusion injury. Appling IPC on an upper arm, this phenomenon has been demonstrated in several studies. Skeletal muscle tissue oxygenation at rest (StO2) and StO2 deoxygenation rate during vascular occlusion can be measured using near infrared spectroscopy (NIRS). We aimed to investigate the effects of remote upper arm IPC on StO2 and flow-mediated dilatation (FMD) in healthy male volunteers. In a randomized controlled crossover trial, resting StO2, StO2 deoxygenation rate, and FMD were measured on testing arm at baseline and after 60 minutes. After basal measurements IPC protocol on a contralateral arm was performed. StO2 deoxygenation rate was significantly lower after remote, the IPC cycles in comparison to deoxygenation rate at baseline (9.7±2.6 versus 7.5±2.5%, P=0.002). Comparison of deoxygenation rates showed a significant difference between the IPC and the control protocol (F=5.512, P=0.003). No differences were observed in FMD before and after remote IPC and in the control protocol. In healthy young adults, remote IPC reduces StO2 deoxygenation rate but has no significant impact on FMD. NIRS technique offers a novel approach to asses skeletal muscle adaptation in response to remote ischemic stimuli.

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6

Kime, Ryotaro, Joohee Im, Daniel Moser, Shoko Nioka, Toshihito Katsumura, and Britton Chance. "Nonuniform muscle deoxygenation in single muscle during bicycle exercise." Japanese Journal of Physical Fitness and Sports Medicine 54, no. 1 (2005): 74. http://dx.doi.org/10.7600/jspfsm.54.74.

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7

Niemeijer, Victor M., Ruud F. Spee, Thijs Schoots, Pieter F. F. Wijn, and Hareld M. C. Kemps. "Limitations of skeletal muscle oxygen delivery and utilization during moderate-intensity exercise in moderately impaired patients with chronic heart failure." American Journal of Physiology-Heart and Circulatory Physiology 311, no. 6 (December 1, 2016): H1530—H1539. http://dx.doi.org/10.1152/ajpheart.00474.2016.

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The extent and speed of transient skeletal muscle deoxygenation during exercise onset in patients with chronic heart failure (CHF) are related to impairments of local O2 delivery and utilization. This study examined the physiological background of submaximal exercise performance in 19 moderately impaired patients with CHF (Weber class A, B, and C) compared with 19 matched healthy control (HC) subjects by measuring skeletal muscle oxygenation (SmO2) changes during cycling exercise. All subjects performed two subsequent moderate-intensity 6-min exercise tests (bouts 1 and 2) with measurements of pulmonary oxygen uptake kinetics and SmO2 using near-infrared spatially resolved spectroscopy at the vastus lateralis for determination of absolute oxygenation values, amplitudes, kinetics (mean response time for onset), and deoxygenation overshoot characteristics. In CHF, deoxygenation kinetics were slower compared with HC (21.3 ± 5.3 s vs. 16.7 ± 4.4 s, P < 0.05, respectively). After priming exercise (i.e., during bout 2), deoxygenation kinetics were accelerated in CHF to values no longer different from HC (16.9 ± 4.6 s vs. 15.4 ± 4.2 s, P = 0.35). However, priming did not speed deoxygenation kinetics in CHF subjects with a deoxygenation overshoot, whereas it did reduce the incidence of the overshoot in this specific group ( P < 0.05). These results provide evidence for heterogeneity with respect to limitations of O2 delivery and utilization during moderate-intensity exercise in patients with CHF, with slowed deoxygenation kinetics indicating a predominant O2 utilization impairment and the presence of a deoxygenation overshoot, with a reduction after priming in a subgroup, indicating an initial O2 delivery to utilization mismatch.

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8

Anthierens, Agathe, Nicolas Olivier, André Thevenon, and Patrick Mucci. "Trunk Muscle Aerobic Metabolism Responses in Endurance Athletes, Combat Athletes and Untrained Men." International Journal of Sports Medicine 40, no. 07 (June 12, 2019): 434–39. http://dx.doi.org/10.1055/a-0856-7207.

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Анотація:

AbstractThis study investigated aerobic metabolism responses in trunk muscles during a prolonged trunk extension exercise in athletes and untrained young men. The aim was to analyze the adaptations induced by 2 types of sports: one involving intensive use of trunk muscles (i. e., judo), and one known to induce high aerobic capacity in the whole body (i. e., cycling). Eleven judokas, 10 cyclists and 9 healthy untrained young men performed trunk extension exercises on an isokinetic dynamometer. During the first session, muscle strength was assessed during maximal trunk extension. During a second session, a 5-min exercise was performed to investigate aerobic responses with regard to trunk muscles. The near infrared spectroscopy technique and a gas exchange analyzer were used continuously to evaluate mechanical efficiency, V̇O2 on-set kinetics, trunk muscle deoxygenation and blood volume. Judokas showed greater trunk strength and mechanical efficiency (p<0.05). Cyclists presented faster V̇O2 on-set kinetics (p<0.05) and greater muscle deoxygenation and blood volume compared to untrained men (p<0.001). These results suggest that practicing judo improves trunk extension efficiency whereas cycling accelerates aerobic pathways and enhances microvascular responses to trunk extension exercise. Sport practice improves aerobic metabolism responses in trunk extensor muscles differently, according to the training specificities.

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9

Okushima, Dai, David C. Poole, Harry B. Rossiter, Thomas J. Barstow, Narihiko Kondo, Etsuko Ohmae, and Shunsaku Koga. "Muscle deoxygenation in the quadriceps during ramp incremental cycling: Deep vs. superficial heterogeneity." Journal of Applied Physiology 119, no. 11 (December 1, 2015): 1313–19. http://dx.doi.org/10.1152/japplphysiol.00574.2015.

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Анотація:

Muscle deoxygenation (i.e., deoxy[Hb + Mb]) during exercise assesses the matching of oxygen delivery (Q̇o2) to oxygen utilization (V̇o2). Until now limitations in near-infrared spectroscopy (NIRS) technology did not permit discrimination of deoxy[Hb + Mb] between superficial and deep muscles. In humans, the deep quadriceps is more highly vascularized and oxidative than the superficial quadriceps. Using high-power time-resolved NIRS, we tested the hypothesis that deoxygenation of the deep quadriceps would be less than in superficial muscle during incremental cycling exercise in eight males. Pulmonary V̇o2 was measured and muscle deoxy[Hb + Mb] was determined in the superficial vastus lateralis (VL), vastus medialis (VM), and rectus femoris (RF-s) and the deep rectus femoris (RF-d). deoxy[Hb + Mb] in RF-d was significantly less than VL at 70% (67.2 ± 7.0 vs. 75.5 ± 10.7 μM) and 80% (71.4 ± 11.0 vs. 79.0 ± 15.4 μM) of peak work rate (WRpeak), but greater than VL and VM at WRpeak (87.7 ± 32.5 vs. 76.6 ± 17.5 and 75.1 ± 19.9 μM). RF-s was intermediate at WRpeak (82.6 ± 18.7 μM). Total hemoglobin and myoglobin concentration and tissue oxygen saturation were significantly greater in RF-d than RF-s throughout exercise. The slope of deoxy[Hb + Mb] increase (proportional to Q̇o2/V̇o2) in VL and VM slowed markedly above 70% WRpeak, whereas it became greater in RF-d. This divergent deoxygenation pattern may be due to a greater population of slow-twitch muscle fibers in the RF-d muscle and the differential recruitment profiles and vascular and metabolic control properties of specific fiber populations within superficial and deeper muscle regions.

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10

Goulding, Richie P., Simon Marwood, Dai Okushima, David C. Poole, Thomas J. Barstow, Tze-Huan Lei, Narihiko Kondo, and Shunsaku Koga. "Effect of priming exercise and body position on pulmonary oxygen uptake and muscle deoxygenation kinetics during cycle exercise." Journal of Applied Physiology 129, no. 4 (October 1, 2020): 810–22. http://dx.doi.org/10.1152/japplphysiol.00478.2020.

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Анотація:

Here we show that oxygen uptake (V̇o2) kinetics are slower in the supine compared with upright body position, an effect that is associated with an increased amplitude of skeletal muscle deoxygenation in the supine position. After priming in the supine position, the amplitude of muscle deoxygenation remained markedly elevated above that observed during upright exercise. Hence, the priming effect cannot be solely attributed to enhanced O2 delivery, and enhancements to intracellular O2 utilization must also be contributory.

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Більше джерел

Дисертації з теми "Muscle deoxygenation"

1

Ward, Aaron Tyler. "The Effect of Sequential Lower Body Positive Pressure on Forearm Blood Flow and Muscle Deoxygenation During Dynamic Handgrip Exercise." University of Toledo / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1461849449.

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2

Goodwin, Ashley. "Oxygen Uptake Kinetics in Skeletal Muscle Using Near-Infrared Spectroscopy (NIRS): Evaluating Healthy Responses of Muscle Deoxygenation." Thesis, 2021. https://doi.org/10.7916/d8-dn72-fw74.

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Анотація:

The purpose of this dissertation series was to examine oxygen uptake kinetics in skeletal muscle by evaluating responses of local muscle deoxygenation during incremental exercise in healthy individuals using near-infrared spectroscopy (NIRS). Metabolic activity in skeletal muscle, as part of the integrative responses of the cardiovascular, respiratory and neuromuscular systems, are major determinants of an individual’s physical capacity and function. The workings of these systems, called whole-body metabolism, affect the capability of an individual to engage in activities of daily living, to exercise, and participate in athletic performance. Thus, they have a strong impact on health as engagement in physical activity is well known to be effective in improving cardiorespiratory fitness and reducing the risks of chronic disease. At this time, the in vivo relationships between whole-body metabolism and local muscle metabolic activity are not well understood, but with the availability of NIRS technology this is possible. NIRS is a noninvasive optical technique used to continuously measure changes in muscle tissue oxygen saturation locally, allowing interrogation of the functional integration between muscle metabolism and the cardiovascular system in intact human beings, which is what the series of studies in this dissertation evaluate. Healthy adults and adolescents were enrolled as healthy control participants into an observational study evaluating changes in local muscle oxygen uptake in neuromuscular disease during exercise. Participants performed a maximal cardiopulmonary exercise test (CPET) on a recumbent cycle ergometer. Changes in muscle deoxygenation (HHb), reflecting local oxygen uptake, were measured using NIRS and whole-body metabolism was assessed synchronously via expired gas analysis. After an initial increase in HHb at exercise onset, a consistent pattern of plateau in HHb was observed in the healthy participants near the end of peak exercise. Despite increasing workload and oxygen uptake (VO2) in the final minutes of the test, it was unclear what mechanisms were contributing to this HHb response. It was hypothesized that the HHb-Workload relationship evaluated at the time of VO2peak would be non-linear, such that a greater maximum workload achieved at VO2peak would not be linearly matched by greater ΔHHb (i.e., greater total change from rest to VO2peak). First, a critical evaluation of the literature was conducted to explore this hypothesis. Chapter 2 provides the results of a scoping review that was performed in order to better understand the scientific evidence using NIRS that describes the relationships between indices of muscle oxygen saturation and workload during incremental exercise. This formed the basis to pursue the hypothesis-driven research presented in the subsequent chapters, interrogating the overarching question of this dissertation related to the HHb-Workload relationship. The review revealed there are three methodological approaches to examining changes in muscle oxygen saturation and workload, the least common of which was examination of HHb and workload at the VO2peak time point. Changes in muscle oxygen saturation and work have also been studied as the change in muscle oxygenation over the duration of exercise and at a certain time point or intensity during incremental exercise. Based on the literature, it was clear that there was a dearth of research examining the HHb plateau response in relation to work at VO2peak. Accordingly, chapter 3 provides the results of a pilot study that evaluated the relationship between change in HHb (ΔHHb) and the maximum workload (MW) achieved at VO2peak, where it was hypothesized that the relationship at this time point would be non-linear. A polynomial regression model was used to describe the relationship. The results of this study showed that at lower maximum workloads there were initial increases in ΔHHb with increasing maximum workload but at the highest maximum workloads, ΔHHb attenuated. A polynomial model including ΔHHb and MW, with VO2peak (an indicator of cardiorespiratory fitness) as a covariate, best characterized the relationship. Age was not significantly related to ΔHHb or MW, and VO2peak appeared to play a partial role as its inclusion as a covariate helped explain approximately a quarter of the variance, suggesting other factors may be contributing to the attenuated HHb response. From this pilot work it was hypothesized that the attenuation in ΔHHb at higher maximum workloads, and the HHb plateau observed during CPET, could be explained by muscle efficiency. If so, a longer duration and lesser slope of the HHb plateau in the minutes leading up to VO2peak occurs in muscles with higher metabolic efficiency. As muscle efficiency is defined as a ratio of external work accomplished to internal energy expended, the hypothesis, if true, would support a better matching of the internal work (VO2) to the external work (workload on the ergometer). Chapter 4 provides the results of a secondary analysis that sought to determine whether the observed plateau in HHb reflected muscular efficiency by comparing the slope of the HHb plateau (HHb[s]) to a commonly used method of assessing muscle efficiency, delta efficiency (DE). It was hypothesized that HHb[s] and DE would be inversely and significantly correlated, providing a potential mechanism for the attenuated HHb response and a noninvasive method for assessing muscle efficiency. In contrast to the hypothesis, HHb[s] and DE were not associated, suggesting that a mechanism other than muscle efficiency is contributing to the HHb plateau. Collectively, this series of studies demonstrate that there is a need to better understand the relationship between HHb and workload in healthy individuals, because of a paucity of evidence exploring the HHb-MW relationship at VO2peak, the finding that ΔHHb attenuates at higher maximum workloads, and that results suggest the HHb plateau phenomenon cannot be explained by muscle efficiency. Future work should seek to elucidate the mechanism that allows healthy individuals to achieve higher workloads (i.e., continue exercising at high intensity) without further increasing muscle oxygen uptake, in a larger more heterogeneous sample.

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3

Rodriguez-Anderson, Ramón F. "The Influence of Respiratory Muscle Work on Locomotor and Respiratory Muscle Oxygenation Trends in Repeated-Sprint Exercise." Thesis, 2018. https://vuir.vu.edu.au/37831/.

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Анотація:

This thesis investigated the role respiratory muscle work has on locomotor and respiratory muscle oxygen (O2) utilisation during multiple sprint work. To measure O2 delivery and uptake in real time, near-infrared spectroscopy (NIRS) can be used. However, there are inconsistent methods of smoothing and determining peaks and nadirs from the NIRS signal. Therefore, the aim of study 1 was to examine the effects of different methodologies commonly used in the literature on the determination of peaks and nadirs in the vastus lateralis deoxyhaemoglobin (HHbVL) signal. Means derived from predetermined windows, irrespective of length and data smoothing, underestimated the magnitude of peak and nadir [HHbVL] compared to a rolling mean approach. Based on the results, we suggest using a digital filter to smooth NIRS data, rather than an arithmetic mean, and a rolling approach to determine peaks and nadirs for accurate interpretation of muscle oxygenation trends. In the second study, the effects of heightened inspiratory muscle work on [HHbVL] and respiratory muscle deoxyhaemoglobin ([HHbRM]) trends were examined. In response to the heightened inspiratory muscle work, HHbRM was elevated across the sprint series. There were no clear differences in HHbVL trends between exercise conditions. The lack of difference in HHbVL between trials implies respiratory muscle O2 uptake does not limit locomotor oxygenation trends. Study 3 investigated the role of arterial hypoxemia on respiratory muscle oxygenation trends, and its implications on locomotor oxygenation. While exercising in hypoxia (14.5% O2), HHbVL was higher during the sprint and recovery phases of the repeated-sprint protocol compared to normoxia (21% O2). There were no clear differences in respiratory muscle oxygenation trends between conditions. The clear reduction in locomotor muscle O2 delivery (inferred from HHbVL) while respiratory muscle oxygenation was maintained, suggests preferential blood flow distribution to the respiratory muscle to compensate for arterial hypoxemia, which may explain in part compromise locomotor O2 delivery. The aim of the final study was to examine the role of respiratory muscle strength on locomotor and respiratory muscle oxygenation trends in repeated-sprint exercise. Inspiratory muscle training (IMT) was used to reduce the relative intensity of exercise hyperpnoea by strengthening the respiratory muscles. Repeat-sprint ability was again assessed in normoxia and hypoxia. After 4 weeks of training, there was a 35% increase of inspiratory muscle pressure in the IMT beyond the control group. Despite the substantial change in respiratory muscle strength, oxygenation trends were not affected in either normoxia or hypoxia. The findings of this thesis do not support the work of breathing as being a limiting factor in locomotor muscle oxygenation in normoxia. The intermittent nature of repeated-sprint activity is likely a key mediating factor for which O2 delivery can be maintained to both the locomotor and respiratory muscles. However, under conditions of arterial hypoxemia, locomotor muscle oxygenation may be compromised by preferential O2 delivery to the respiratory muscles.

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Частини книг з теми "Muscle deoxygenation"

1

Nioka, S., D. Moser, G. Lech, M. Evengelisti, T. Verde, B. Chance, and S. Kuno. "Muscle Deoxygenation in Aerobic and Anaerobic Exercise." In Oxygen Transport to Tissue XX, 63–70. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-4863-8_8.

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2

Takagi, Shun, Ryotaro Kime, Taishi Midorikawa, Masatsugu Niwayama, Shizuo Sakamoto, and Toshihito Katsumura. "Skeletal Muscle Deoxygenation Responses During Treadmill Exercise in Children." In Advances in Experimental Medicine and Biology, 341–46. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-0620-8_45.

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3

Kime, Ryotaro, Masatsugu Niwayama, Yasuhisa Kaneko, Shun Takagi, Sayuri Fuse, Takuya Osada, Norio Murase, and Toshihito Katsumura. "Muscle Deoxygenation and Its Heterogeneity Changes After Endurance Training." In Advances in Experimental Medicine and Biology, 275–81. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-38810-6_37.

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4

Takagi, Shun, Norio Murase, Ryotaro Kime, Masatsugu Niwayama, Takuya Osada, and Toshihito Katsumura. "Low Volume Aerobic Training Heightens Muscle Deoxygenation in Early Post-Angina Pectoris Patients." In Advances in Experimental Medicine and Biology, 255–61. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-38810-6_34.

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5

Kime, Ryotaro, Masatsugu Niwayama, Masako Fujioka, Kiyoshi Shiroishi, Takuya Osawa, Kousuke Shimomura, Takuya Osada, Norio Murase, and Toshihito Katsumura. "Unchanged Muscle Deoxygenation Heterogeneity During Bicycle Exercise After 6 Weeks of Endurance Training." In Advances in Experimental Medicine and Biology, 353–58. Boston, MA: Springer US, 2009. http://dx.doi.org/10.1007/978-1-4419-1241-1_51.

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6

Takagi, Shun, Ryotaro Kime, Masatsugu Niwayama, Takuya Osada, Norio Murase, Shizuo Sakamoto, and Toshihito Katsumura. "Sex-Related Difference in Muscle Deoxygenation Responses Between Aerobic Capacity-Matched Elderly Men and Women." In Advances in Experimental Medicine and Biology, 55–61. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3023-4_7.

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7

Takagi, Shun, Ryotaro Kime, Norio Murase, Masatsugu Niwayama, Shizuo Sakamoto, and Toshihito Katsumura. "Skeletal Muscle Deoxygenation and Its Relationship to Aerobic Capacity During Early and Late Stages of Aging." In Advances in Experimental Medicine and Biology, 77–82. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-48238-1_12.

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8

Bowen, T. Scott, Shunsaku Koga, Tatsuro Amano, Narihiko Kondo, and Harry B. Rossiter. "The Spatial Distribution of Absolute Skeletal Muscle Deoxygenation During Ramp-Incremental Exercise Is Not Influenced by Hypoxia." In Advances in Experimental Medicine and Biology, 19–26. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3023-4_2.

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9

Morishita, Shinichiro, Atsuhiro Tsubaki, Kazuki Hotta, Sho Kojima, Daichi Sato, Akihito Shirayama, Yuki Ito, and Hideaki Onishi. "Relationship Between the Borg Scale Rating of Perceived Exertion and Leg-Muscle Deoxygenation During Incremental Exercise in Healthy Adults." In Advances in Experimental Medicine and Biology, 95–99. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-48238-1_15.

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Тези доповідей конференцій з теми "Muscle deoxygenation"

1

Souza, V., M. Lafeta, M. Saldanha, F. Penido, T. Menezes, S. Tanni, A. Albuquerque, et al. "Dynamic matching of oxygen uptake kinetics and muscle deoxygenation in post-COVID-19." In ERS International Congress 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/13993003.congress-2022.2212.

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2

Reid, Wendy Darlene, Arjun Patel, Zacherie Bergeron, Andrew Ho, Tongyu Shi, Marcelle Campos, Ewan Goligher, and Laurent Brochard. "Deoxygenation recruitment patterns of abdominal and neck muscles during four bed exercises." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.269.

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3

Verdaguer-Codina, Joan, and Jaume A. Mirallas. "Deoxygenation and the blood volume signals in the flexor carpi ulnaris and radialis muscles obtained during the execution of the Mirallas's test of judo athletes." In BiOS Europe '96, edited by David A. Benaron, Britton Chance, and Gerhard J. Mueller. SPIE, 1996. http://dx.doi.org/10.1117/12.260843.

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