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Готові списки джерел за темами / Myocardial infarction recovery / Статті в журналах

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Статті в журналах з теми "Myocardial infarction recovery"

Автор: Grafiati

Опубліковано: 7 червня 2025

Оновлено: 2 серпня 2025

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1

Ando, H., J. Tanaka, M. Hisahara, M. Umesue, and T. Shirota. "Effect of Coronary Bypass Grafting onto the Site of Old Myocardial Infarction and the Recovery of Cardiac Function." Cardiovascular Surgery 6, no. 5 (1998): 511–19. http://dx.doi.org/10.1177/096721099800600515.

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This study investigated whether or not revascularization to the site of old myocardial infarction can bring beneficial effects on postoperative cardiac function. Thirty-two patients without a history of old myocardial infarction and 71 with a history of old myocardial infarction were included. The mean number of grafts bypassed were 2.7 and 2.8. and the mean duration of aortic clamping was 99 min and 105 min in non-old myocardial infarction and old myocardial infarction patients, respectively. All the patients underwent simple coronary artery bypass grafting electively. The left ventricular st

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2

Fridlund, Bengt, Benkt Högstedt, Evy Lidell, and Pår A. Larsson. "Recovery after Myocardial Infarction." Scandinavian Journal of Caring Sciences 5, no. 1 (1991): 23–32. http://dx.doi.org/10.1111/j.1471-6712.1991.tb00078.x.

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3

Owen, Patricia M. "Recovery from myocardial infarction." Journal of Cardiovascular Nursing 2, no. 1 (1987): 75–85. http://dx.doi.org/10.1097/00005082-198711000-00010.

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4

Širmenis, Raimondas, Antanas Kraniauskas, Rasa Jarašienė, Daiva Baltriukienė, Audronė Kalvelytė, and Virginija Bukelskienė. "Recovery of Infarcted Myocardium in an In Vivo Experiment." Medicina 47, no. 11 (2011): 88. http://dx.doi.org/10.3390/medicina47110088.

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Acute myocardial infarction leads to the loss of functional cardiomyocytes and structural integrity. The adult heart cannot repair the damaged tissue due to inability of mature cardiomyocytes to divide and lack of stem cells. The aim of this study was to evaluate the efficiency of introduced autologous skeletal musclederived stem cells to recover the function of acutely infarcted rabbit heart in the early postoperative period. Material and Methods. As a model for myocardium restoration in vivo, experimental rabbit heart infarct was used. Autologic adult myogenic stem cells were isolated from s

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5

Ristic, Andjelka, Milorad Damjanovic, Branislav Baskot, and Radomir Matunovic. "Stunned myocardium." Vojnosanitetski pregled 62, no. 2 (2005): 165–69. http://dx.doi.org/10.2298/vsp0502165r.

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Background. Stunned myocardium is a state of delayed recovery of regional contractility after a transient period of ischemia followed by reperfusion. Case report. A 67-year-old patient was admitted to our hospital with acute anterior myocardial infarction, and treated using percutaneous transluminal coronary angioplasty (PTCA) within acute disease stage. Reversible myocardial dysfunction persisted after ischemia following the return of normal perfusion. Abnormal resting wall motion with augmentation of contractility at low and high doses of dobutamine characterizes the stunned myocardium and r

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6

Khvesyk, Larysa, and Yuliia Antonova-Rafi. "Analysis of traditional physical therapy programs after myocardial infarction using evidence-based medicine approaches." Ukrainian Scientific Medical Youth Journal 144, no. 1 (2024): 111–17. http://dx.doi.org/10.32345/usmyj.1(144).2024.111-117.

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myocardial infarction is heart muscle necrosis associated with coronary blood supply disorders, which occurs in the vast majority of cases as a result of atherothrombosis. Mechanical revascularization of the myocardium is a more effective and safer way to improve the condition of patients with myocardial infarction compared to long-term drug therapy, but the risk of early complications of cardiac surgery remains high. Considering this, non-pharmacological methods of treatment of myocardial infarction play a valuable role. Numerous publications testify to the fact that in everyday medical pract

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7

Wang, Mian, and Fan Wu. "PHYSICAL EXERCISE ON THE RECOVERY OF OBESITY MYOCARDIAL INFARCTION." Revista Brasileira de Medicina do Esporte 27, no. 8 (2021): 783–85. http://dx.doi.org/10.1590/1517-8692202127082021_0368.

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ABSTRACT Introduction: Myocardial infarction caused by human obesity can cause a decline in mobility and a decline in the quality of a healthy life. Sports training is beneficial to maintain early physical functions after myocardial infarction. Objective: This article deals with the effect of applying walking aerobic exercise in the rehabilitation treatment of patients with myocardial infarction. Methods: We enrolled 91 patients with myocardial infarction in the early exercise group and 90 patients in the control group. The control group received the routine nursing intervention, and the early

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8

Robson, Andrew. "Exosomes improve myocardial recovery after infarction." Nature Reviews Cardiology 17, no. 12 (2020): 758. http://dx.doi.org/10.1038/s41569-020-00460-w.

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9

Nijveldt, Robin, Aernout M. Beek, Alexander Hirsch, et al. "Functional Recovery After Acute Myocardial Infarction." Journal of the American College of Cardiology 52, no. 3 (2008): 181–89. http://dx.doi.org/10.1016/j.jacc.2008.04.006.

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10

Saito, Daiji, Teruo Shiraki, Takefumi Oka, Akio Kajiyama, and Toshiyuki Takamura. "Risk Factors Indicating Recurrent Myocardial Infarction After Recovery From Acute Myocardial Infarction." Circulation Journal 66, no. 10 (2002): 877–80. http://dx.doi.org/10.1253/circj.66.877.

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11

Jin, Jiyang, Min Chen, Yongjun Li, et al. "Detecting Acute Myocardial Infarction by Diffusion-Weighted versus T2-Weighted Imaging and Myocardial Necrosis Markers." Texas Heart Institute Journal 43, no. 5 (2016): 383–91. http://dx.doi.org/10.14503/thij-15-5462.

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We used a porcine model of acute myocardial infarction to study the signal evolution of ischemic myocardium on diffusion-weighted magnetic resonance images (DWI). Eight Chinese miniature pigs underwent percutaneous left anterior descending or left circumflex coronary artery occlusion for 90 minutes followed by reperfusion, which induced acute myocardial infarction. We used DWI preprocedurally and hourly for 4 hours postprocedurally. We acquired turbo inversion recovery magnitude T2-weighted images (TIRM T2WI) and late gadolinium enhancement images from the DWI slices. We measured the serum myo

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12

Bouvier, Frederic, Jonas Höjer, Bassem A. Samad, et al. "Delayed recovery of myocardial perfusion in acute myocardial infarction." Coronary Artery Disease 9, no. 7 (1998): 443–50. http://dx.doi.org/10.1097/00019501-199809070-00007.

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13

Liu, Kuo, Demin Liu, and Wei Cui. "Protective Effect and Mechanism of Traditional Chinese Medicine on Myocardial Ischemia Reperfusion Injury." Evidence-Based Complementary and Alternative Medicine 2022 (March 31, 2022): 1–8. http://dx.doi.org/10.1155/2022/6121407.

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After acute myocardial infarction, early restoration of myocardial perfusion by thrombolysis or percutaneous coronary intervention is the most effective way to reduce the size of myocardial infarction and improve clinical outcomes. However, recovery of blood flow to the ischemic myocardium may cause ischemia-reperfusion (I/R) injury, a phenomenon that instead reduces the efficacy of myocardial reperfusion. Traditional Chinese medicine (TCM) has long been used for the treatment of cardiovascular diseases and has shown remarkable efficacy. Many studies have shown that some TCMs and their active

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14

Badimon, Lina, and Maria Borrell. "Microvasculature Recovery by Angiogenesis After Myocardial Infarction." Current Pharmaceutical Design 24, no. 25 (2018): 2967–73. http://dx.doi.org/10.2174/1381612824666180629162726.

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Advances in early reperfusion therapies focused on the revascularization of the ischemic tissues, in the last decades, lead to reduced mortality in acute myocardial infarction (MI) patients. However, a large proportion of patients show inadequate myocardial perfusion because of dysfunction of the microcirculation. The high prevalence of microvascular dysfunction after reperfusion therapies and the negative prognostic of this procedure justify the search for therapeutic strategies that aim to restore the microvascular network. It is well known that the size of the initial infarct, the duration

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15

Albulescu, Nicolae, Vlad Sabin Ivan, Adrian Apostol, Gheorghe Iovanescu, Mihaela Viviana Ivan, and Adalbert Schiller. "Cardiac Electrical Vulnerability after Acute Myocardial Infarction Associated with Respiratory Infections." Revista de Chimie 70, no. 1 (2019): 207–10. http://dx.doi.org/10.37358/rc.19.1.6883.

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Heart rate variability and the presence of late potentials are independent predictor factors for cardiac death and electrical vulnerability of the ischemic myocardium, especially after myocardial infarction(MI). Respiratory infection are known to be associated with MI both through direct action of the pathogen and by altering the hemodynamic status, through tachycardia and a rise in myocardial oxigen demand. Our paper wants to highlight that respiratory infection during subacute and late recovery phase of myocardial infarction can aggravate the electrical vulnerability of the myocardium and in

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16

Haywood, L. Julian. "Social Class and Recovery from Myocardial Infarction." Annals of Internal Medicine 128, no. 8 (1998): 695. http://dx.doi.org/10.7326/0003-4819-128-8-199804150-00024.

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17

Ickovics, Jeannette R. "Social Class and Recovery from Myocardial Infarction." Annals of Internal Medicine 128, no. 8 (1998): 696. http://dx.doi.org/10.7326/0003-4819-128-8-199804150-00025.

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18

Hamilton, Glenys A. "Recovery from Acute Myocardial Infarction in Women." Cardiology 77, no. 2 (1990): 58–70. http://dx.doi.org/10.1159/000174654.

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19

Stevens, Sherri, and Sandra P. Thomas. "Recovery of Midlife Women From Myocardial Infarction." Health Care for Women International 33, no. 12 (2012): 1096–113. http://dx.doi.org/10.1080/07399332.2012.684815.

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20

Ell, Kathleen, and L. Julian Haywood. "Social Support and Recovery From Myocardial Infarction." Journal of Social Service Research 7, no. 4 (1985): 1–19. http://dx.doi.org/10.1300/j079v07n04_01.

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21

Mladenovic, Zorica, Andjelka Angelkov-Ristic, Dimitra Kalimanovska-Ostric, et al. "Early predictors of left ventricular function improvement late after myocardial infarction." Vojnosanitetski pregled 65, no. 1 (2008): 9–14. http://dx.doi.org/10.2298/vsp0801009m.

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Background/Aim. Prognosis after acute myocardial infarction (AIM) depends on the extent of irreversibly damaged myocardium and viable tissue due to stunning or hibernation. The aim of the study was to assess the prognostic significance of early echocardiographic parameters of myocardial viability in prediction of late recovery of regional and global ventricular function. Methods. The study prospectively included 40 patients after the first, uncomplicated univessel AIM treated with percutaneous coronary intervention (PCI). Low-dose dobutamine echocardiography (LDDE) was preformed 7-10 days afte

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22

Thompson, C. H., G. J. Kemp, B. Rajagopalan, and G. K. Radda. "Metabolic Abnormalities in Skeletal Muscle after Myocardial Infarction in the Rat." Clinical Science 87, no. 4 (1994): 403–6. http://dx.doi.org/10.1042/cs0870403.

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1. The effect of experimental myocardial infarction on exercise and recovery of rat skeletal muscle was studied using 31P n.m.r. 4 weeks post-operatively. 2. Myocardial infarction (12 ± 3% of left ventricular volume), insufficient to produce haemodynamic manifestations of heart failure, was without significant effect on exercise bioenergetics of skeletal muscle. 3. Citrate synthase activity was reduced by 17% in the infarcted animals and there was a marked slowing of the rate of phosphocreatine recovery after infarction (half-time 0.7 ± 0.1 min to 1.6 ± 0.2 min) in the absence of evidence of l

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23

Skalsky, Keren, Arthur Shiyovich, Alon Shechter, Harel Gilutz, and Ygal Plakht. "Recovery from Acute Kidney Injury and Long-Term Prognosis following Acute Myocardial Infarction." Biomedicines 12, no. 7 (2024): 1490. http://dx.doi.org/10.3390/biomedicines12071490.

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We investigated the recovery pattern from acute kidney injury (AKI) following acute myocardial infarction (AMI) and its association with long-term mortality. The retrospective study included AMI patients (2002–2027), who developed AKI during hospitalization. Creatinine (Cr) measurements were collected and categorized into 24 h timeframes up to 7 days from AKI diagnosis. The following groups of recovery patterns were defined: rapid (24–48 h)/no rapid and early (72–144 h)/no early recovery. Specific cut-off points for recovery at each AKI stage and timeframe were determined through receiver oper

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24

Strelnikov, A. G., A. B. Romanov, S. N. Artemenko, et al. "Correction of the effects of myocardial infarction by intramyocardial injection of autologous bone marrow mononuclear cells." Patologiya krovoobrashcheniya i kardiokhirurgiya 17, no. 2 (2015): 37. http://dx.doi.org/10.21688/1681-3472-2013-4-37-39.

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To assess the functional recovery and remodeling of postinfarction myocardium, a combined technology of single-photon emission computed tomography (SPECT) and endocardial electrophysiological 3D reconstruction of the heart in patients with left ventricular dysfunction after myocardial infarction was used.

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25

De Meyer, Simon F., Alexander S. Savchenko, Michael S. Haas, et al. "Protective anti-inflammatory effect of ADAMTS13 on myocardial ischemia/reperfusion injury in mice." Blood 120, no. 26 (2012): 5217–23. http://dx.doi.org/10.1182/blood-2012-06-439935.

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Abstract Coronary heart disease is a major cause of death in the western world. Although essential for successful recovery, reperfusion of ischemic myocardium is inevitably associated with reperfusion injury. To investigate a potential protective role of ADAMTS13, a protease cleaving von Willebrand factor multimers, during myocardial ischemia/reperfusion, we used a mouse model of acute myocardial infarction. We found that Adamts13−/− mice developed larger myocardial infarctions than wild-type control mice, whereas treatment of wild-type mice with recombinant human ADAMTS13 (rhADAMTS13) led to

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26

Mayawi, Mayawi, Nurhayati Nurhayati, Novita Serly Laamena, Ariestha Widyastuty Bustan, Munazat Salmin, and Taufan Talib. "PERBANDINGAN MODEL REGRESI WEIBULL DAN REGRESI COX PROPOSIONAL HAZARD." Science Map Journal 4, no. 2 (2022): 49–60. http://dx.doi.org/10.30598/jmsvol4issue2pp49-60.

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This study aims to determine the model of Acute Myocardial Infarction using the Weibull Regression and Cox Proportional Hazard Regression methods to determine the factors that significantly influence the length of time for recovery of acute myocardial infarction patients. The results of this study indicate that the factors that significantly influence the recovery time of acute myocardial infarction patients are age, onset, number of secondary diagnoses and duration of pain. Based on the AIC value, the Weibull regression model is the best regression model because the AIC value is smaller, name

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27

Gao, Ling, Lu Wang, Yuhua Wei, et al. "Exosomes secreted by hiPSC-derived cardiac cells improve recovery from myocardial infarction in swine." Science Translational Medicine 12, no. 561 (2020): eaay1318. http://dx.doi.org/10.1126/scitranslmed.aay1318.

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Cell therapy treatment of myocardial infarction (MI) is mediated, in part, by exosomes secreted from transplanted cells. Thus, we compared the efficacy of treatment with a mixture of cardiomyocytes (CMs; 10 million), endothelial cells (ECs; 5 million), and smooth muscle cells (SMCs; 5 million) derived from human induced pluripotent stem cells (hiPSCs), or with exosomes extracted from the three cell types, in pigs after MI. Female pigs received sham surgery; infarction without treatment (MI group); or infarction and treatment with hiPSC-CMs, hiPSC-ECs, and hiPSC-SMCs (MI + Cell group); with hom

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28

Swinburn, Jonathan M. A., Avijit Lahiri, and Roxy Senior. "Intravenous myocardial contrast echocardiography predicts recovery of dysynergic myocardium early after acute myocardial infarction." Journal of the American College of Cardiology 38, no. 1 (2001): 19–25. http://dx.doi.org/10.1016/s0735-1097(01)01317-1.

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29

Lisachenko, G. V., A. V. Budaev, and S. V. Bannih. "ALTERED CONTRACTILE FUNCTION IN DOGS AFTER CLINICAL DEATH FOLLOWING ACUTE MIOCARDIAL INFARCTION AND INTACT HEART." Complex Issues of Cardiovascular Diseases 7, no. 2 (2018): 121–28. http://dx.doi.org/10.17802/2306-1278-2018-7-2-121-128.

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Aim. To assess myocardial contractile function in dogs after clinical death following acute myocardial infarction and to determine its role in the development of hemodynamic derangements after cardiopulmonary resuscitation. Methods. 180 dogs included in the experiment received pentobarbital anesthesia to assess contractile function and systemic hemodynamics after a 5-min clinical death caused by myocardial infarction. Results Dogs had phase changes in the myocardial contractile function with its initial increase, subsequent depression and normalization in the early postresuscitation period aft

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30

Tang, Yuting, Xiaofang Lin, Cheng Chen, et al. "Nucleolin Improves Heart Function During Recovery From Myocardial Infarction by Modulating Macrophage Polarization." Journal of Cardiovascular Pharmacology and Therapeutics 26, no. 4 (2021): 386–95. http://dx.doi.org/10.1177/1074248421989570.

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Background: Nucleolin has multiple functions within cell survival and proliferation pathways. Our previous studies have revealed that nucleolin can significantly reduce myocardial ischemia-reperfusion injury by promoting myocardial angiogenesis and reducing myocardial apoptosis. In this study, we attempted to determine the role of nucleolin in myocardial infarction (MI) injury recovery and the underlying mechanism. Methods: Male BALB/c mice aged 6–8 weeks were used to set up MI models by ligating the left anterior descending coronary artery. Nucleolin expression in the heart was downregulated

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31

Imane ESSAKET, Mohcine NAIM, Imane KATIF, Mohammed EL JAMILI, and Mustapha ELHATTAOUI. "Dissection intramyocardial a rare complication of myocardial infraction: Case report." World Journal of Advanced Research and Reviews 19, no. 2 (2023): 268–72. http://dx.doi.org/10.30574/wjarr.2023.19.2.1551.

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Intramyocardial dissecting hematoma (IDH) after acute myocardial infarction (MI) is a rare form of subacute cardiac rupture, resulting from hemorrhagic separation of helical fibers within the myocardium, which has been described in most studies as an incomplete cardiac rupture or a specific type of cardiac rupture. The management of myocardial dissection is variable and poorly documented, with clinical outcomes ranging from asymptomatic recovery to fatal cardiac events. We present here a case of myocardial dissection diagnosed by echocardiography.

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32

Imane, ESSAKET, NAIM Mohcine, KATIF Imane, EL JAMILI Mohammed, and ELHATTAOUI Mustapha. "Dissection intramyocardial a rare complication of myocardial infraction: Case report." World Journal of Advanced Research and Reviews 19, no. 2 (2023): 268–72. https://doi.org/10.5281/zenodo.10830409.

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Intramyocardial dissecting hematoma (IDH) after acute myocardial infarction (MI) is a rare form of subacute cardiac rupture, resulting from hemorrhagic separation of helical fibers within the myocardium, which has been described in most studies as an incomplete cardiac rupture or a specific type of cardiac rupture. The management of myocardial dissection is variable and poorly documented, with clinical outcomes ranging from asymptomatic recovery to fatal cardiac events. We present here a case of myocardial dissection diagnosed by echocardiography. 

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33

García-Manzanares, María, Estefanía Tarazón, Ana Ortega, et al. "XPO1 Gene Therapy Attenuates Cardiac Dysfunction in Rats with Chronic Induced Myocardial Infarction." Journal of Cardiovascular Translational Research 13, no. 4 (2019): 593–600. http://dx.doi.org/10.1007/s12265-019-09932-y.

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AbstractTranscriptomic signature of XPO1 was highly expressed and inversely related to left ventricular function in ischemic cardiomyopathy patients. We hypothesized that treatment with AAV9-shXPO1 attenuates left ventricular dysfunction and remodeling in a myocardial infarction rat model. We induced myocardial infarction by coronary ligation in Sprague-Dawley rats (n = 10), which received AAV9-shXPO1 (n = 5) or placebo AAV9-scramble (n = 5) treatment. Serial echocardiographic assessment was performed throughout the study. After myocardial infarction, AAV9-shXPO1-treated rats showed partial re

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34

Mares, Razvan Gheorghita, Goran Marinkovic, Ovidiu Simion Cotoi, and Alexandru Schiopu. "Innate Immune Mechanisms in Myocardial Infarction - An Update." Revista Romana de Medicina de Laborator 26, no. 1 (2018): 9–20. http://dx.doi.org/10.1515/rrlm-2017-0031.

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Abstract Acute myocardial infarction (AMI) is a disease associated with high morbidity and mortality. Currently there are no available treatments specifically targeting the post-ischemic myocardial processes that lead to heart failure and recurrent coronary events. The innate immune system plays a central role in the two consecutive phases that follow an acute ischemic event: the inflammatory phase and the reparatory phase. The inflamatory phase involves a massive infiltration of neutrophils and inflammatory Ly6Chi monocytes into the injured myocardium. The reparatory phase is orchestrated by

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35

Steiger, Ruth, Noora Tuovinen, Agne Adukauskaite, et al. "Limbic Responses to Aversive Visual Stimuli during the Acute and Recovery Phase of Takotsubo Syndrome." Journal of Clinical Medicine 11, no. 16 (2022): 4891. http://dx.doi.org/10.3390/jcm11164891.

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The role of the limbic system in the acute phase and during the recovery of takotsubo syndrome needs further clarification. In this longitudinal study, anatomical and task-based functional magnetic resonance imaging of the brain was performed during an emotional picture paradigm in 19 postmenopausal female takotsubo syndrome patients in the acute and recovery phases in comparison to sex- and aged-matched 15 healthy controls and 15 patients presenting with myocardial infarction. Statistical analyses were performed based on the general linear model where aversive and positive picture conditions

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36

Brohi, Gul Hassan, Shahzeb Rasool Memon, Muhammad Yaqoob Shahani, Samreen Memon, and Umbreen Bano. "ACUTE MYOCARDIAL INFARCTION." Professional Medical Journal 25, no. 09 (2018): 1374–79. http://dx.doi.org/10.29309/tpmj/2018.25.09.120.

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Background: Death and morbidity are commonly occurred worldwide due toheart diseases and are becoming an increasingly important problem in the developing countriesincluding Pakistan. The left ventricular ejection fraction (LVEF) serves as a good assessmenttool to document beneficial effects of cardiac rehabilitation (CR) program in cardiac patients.Objectives: To evaluate the effect of short-term CR program on LVEF in patients with myocardialinfarction (MI). Study Design: Cross sectional and Case series study. Setting: Departments ofCardiology, Liaquat University Hospital, Hyderabad. Period: 1

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37

Erne, P., J. Wardle, K. Sanders, S. M. Lewis, and A. Maseri. "Mean Platelet Volume and Size Distribution and Their Sensitivity to Agonists in Patients with Coronary Artery Disease and Congestive Heart Failure." Thrombosis and Haemostasis 59, no. 02 (1988): 259–63. http://dx.doi.org/10.1055/s-0038-1642766.

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SummaryMean platelet volume was related to platelet count in patients with myocardial infarction (n = 55) and patients with congestive heart failure (n = 9). 18 patients with acute myocardial infarction were tested at admission and 4-7 days later, together with 13 patients with chronic stable angina and 10 patients with chest pain which was not related to coronary artery disease. In citrated blood a relative reduced frequency of large platelets (>13 fl) occured in patients with acute myocardial infarction at admission but was not seen during recovery or in patients with chronic stable angin

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38

Karakus, Alper, and Berat Uğuz. "An early echocardiographic prediction for functional myocardial recovery after ST elevation myocardial infarction." Kardiologiia 61, no. 1 (2021): 66–71. http://dx.doi.org/10.18087/cardio.2021.1.n1477.

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Goals Discrimination of the time course of functional recovery in early period following ST elevation myocardial infarction (STEMI) has prognostic importance. This study aims to establish the ability of the combined systolic-diastolic index (E / (Ea x Sa), for differentiating recoverable myocardium or persistent non-viability in subjects with STEMI.Material and methods 77 patients with first acute STEMI were enrolled to the study. Serial transthoracic echocardiography (TTE) examinations were performed at the time of the admission (immediately after reperfusion), hospital discharge (5±1 days) a

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Lithovskiy, Igor A., and A. V. Gordienko. "Cardiogenic shock: can the well-forgotten old become the new?" Clinical Medicine (Russian Journal) 94, no. 12 (2017): 941–48. http://dx.doi.org/10.18821/0023-2149-2016-94-11-941-948.

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Cardiogenic shock (CS) is considered to be the most severe variant of acute heart failure. It occurs against the background of myocardial infarction or non-coronary myocardial disease with the loss of 40-50% contractile ability by the left ventricular mass and acute heart overload of another genesis. The marked decrease of blood pressure is accompanied by generalized impairment of microcirculation in all organs. The acting recommendations for emergency assistance suggest to recover blood perfusion pressure in ischemic tissues by inotropic drugs vasopressors, crystalloids that eliminate hypovol

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Spath, N. B., D. M. L. Lilburn, G. A. Gray, et al. "Manganese-Enhanced T1 Mapping in the Myocardium of Normal and Infarcted Hearts." Contrast Media & Molecular Imaging 2018 (October 25, 2018): 1–13. http://dx.doi.org/10.1155/2018/9641527.

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Background. Manganese-enhanced MRI (MEMRI) has the potential to identify viable myocardium and quantify calcium influx and handling. Two distinct manganese contrast media have been developed for clinical application, mangafodipir and EVP1001-1, employing different strategies to mitigate against adverse effects resulting from calcium-channel agonism. Mangafodipir delivers manganese ions as a chelate, and EVP1001-1 coadministers calcium gluconate. Using myocardial T1 mapping, we aimed to explore chelated and nonchelated manganese contrast agents, their mechanism of myocardial uptake, and their a

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Hadi, Sabri Sh, and Khalida M. Khudur. "Impact DASH System on Patients' Dietary Pattern after Recovery from Myocardial Infarction." Journal of the Faculty of Medicine Baghdad 66, no. 3 (2024): 374–81. http://dx.doi.org/10.32007/jfacmedbaghdad.6632348.

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Background: One of the top three causes of death and morbidity is cardiovascular disease Myocardial infarction is a clinical illness defined by quickly growing severe myocardial ischemia. It is the most frequent health problem in the world and the main cause of mortality. Objectives: The study aims to assess the dietary pattern of food intake frequency for patients with post- myocardial infarction. Patients & Methods: The quasi-experimental design has been carried out to determine the impact of the Dietary Approaches to Stop Hypertension system on patients with post-myocardial infarction,

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Lagrand, W. K., F. Nijland, C. E. Hack, et al. "Interleukin-6 release predicts myocardial recovery (stunning) after acute myocardial infarction." Journal of the American College of Cardiology 31 (1998): 134. http://dx.doi.org/10.1016/s0735-1097(98)81224-2.

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Lagrand, W. "Interleukin-6 Release Predicts Myocardial Recovery (stunning) After Acute Myocardial Infarction." Journal of the American College of Cardiology 31, no. 2 (1998): 134A. http://dx.doi.org/10.1016/s0735-1097(97)84285-4.

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Parviz, Yasir, Mohammad Waleed, Sethumadhavan Vijayan, et al. "Cellular and molecular approaches to enhance myocardial recovery after myocardial infarction." Cardiovascular Revascularization Medicine 20, no. 4 (2019): 351–64. http://dx.doi.org/10.1016/j.carrev.2018.05.021.

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Mahmoudi, Michael, and Mark Mariathas. "Optimizing Myocardial Recovery Post-ST-Elevation Myocardial Infarction – An Unfulfilled Promise." Cardiovascular Revascularization Medicine 20, no. 4 (2019): 287–88. http://dx.doi.org/10.1016/j.carrev.2019.02.013.

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Lee, Jun Seong, Se-Jin Jeong, Sinai Kim, et al. "Conventional Dendritic Cells Impair Recovery after Myocardial Infarction." Journal of Immunology 201, no. 6 (2018): 1784–98. http://dx.doi.org/10.4049/jimmunol.1800322.

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Tod, Angela. "Exploring the meaning of recovery following myocardial infarction." Nursing Standard 23, no. 3 (2008): 35–42. http://dx.doi.org/10.7748/ns2008.09.23.3.35.c6672.

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Tod, Angela. "Exploring the meaning of recovery following myocardial infarction." Nursing Standard 23, no. 3 (2008): 35–42. http://dx.doi.org/10.7748/ns.23.3.35.s50.

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YAMAMOTO, Kazuhiro. "A New Prognostic Factor for Myocardial Infarction Recovery." Hypertension Research 31, no. 7 (2008): 1269–70. http://dx.doi.org/10.1291/hypres.31.1269.

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Woo, M.D, Kam S., and Harvey D. White, M.B. "FACTORS AFFECTING OUTCOME AFTER RECOVERY FROM MYOCARDIAL INFARCTION." Annual Review of Medicine 45, no. 1 (1994): 325–39. http://dx.doi.org/10.1146/annurev.med.45.1.325.

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