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Готові списки джерел за темами / Nitric oxide Pathophysiology / Дисертації

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Дисертації з теми "Nitric oxide Pathophysiology"

Автор: Grafiati

Опубліковано: 4 червня 2021

Оновлено: 8 лютого 2022

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1

Groves, Peter H. "The influence of exogenous nitric oxide on the pathophysiology of angioplasty injury." Thesis, University of Newcastle Upon Tyne, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.308763.

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2

Evans, Kevin Andrew. "Hypoxia and vascular nitric oxide bioavailability : implications for the pathophysiology of high-altitude illness." Thesis, University of South Wales, 2009. https://pure.southwales.ac.uk/en/studentthesis/hypoxia-and-vascular-nitric-oxide-bioavailability(3cd64bcd-5fb9-4209-a6f3-ab219e906a17).html.

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Introduction: Nitric oxide (NO) is an integral molecule implicated in the control of vascular function. It has been suggested that vascular dysfunction may lead to the development of acute mountain sickness (AMS), high-altitude cerebral oedema (HACE) and high-altitude pulmonary oedema (HAPE), though data to date remains scarce. Therefore, there is a clear need for further work to address the role of NO in the pathogenesis of high-altitude illness. Aims: There were two primary aims of the current work: (1) To examine whether hypoxia mediated changes in systemic NO metabolism are related to the

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3

Hill, Anita. "The Role of Complement and Nitric Oxide in the Pathophysiology of Paroxysmal Nocturnal Haemoglobinuria." Thesis, University of Leeds, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.503289.

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4

Kerckx, Yannick. "Modeling nitric oxide production and transport in the human lung." Doctoral thesis, Universite Libre de Bruxelles, 2009. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/210324.

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Le travail présenté ici porte sur l’étude de la production et du transport du monoxyde d’azote (NO) dans le poumon humain. Le NO est une molécule dont l’implication dans des processus physiologiques n’a été mis en évidence qu’en 1987. Depuis, il a été démontré que le NO joue de nombreux rôles dans le corps humain. Le NO est un gaz labile (instable) dans les conditions physiologiques, il diffuse très facilement au travers des parois et il a une grande affinité pour l’hémoglobine. La production du NO est liée à 3 isoformes différentes de la protéine appelées synthases du NO ou NO synthases.<p>\<

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5

Collop, Natalie Chantel. "An investigation of the importance of the ATM protein in the endothelium and its role in the signalling pathways of NO production." Thesis, Stellenbosch : Stellenbosch University, 2015. http://hdl.handle.net/10019.1/97067.

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Thesis (MScMedSc)--Stellenbosch University, 2015.<br>ENGLISH ABSTRACT: Ataxia telangiectasia (AT) is a well-characterized neurodegenerative disease resulting from a genetic defect in the Atm gene causing an absence or very low expression of the ATM protein. As AT patients are prone to the development of insulin resistance and atherosclerosis, the aim or the current study was to investigate the importance of the ATM protein in the endothelium and its role in the signalling pathways of nitric oxide (NO) production. To accomplish this, the first objective was to establish an in-house endothelial

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6

Warner, Anke Sigrid. "The expression, regulation and effects of inducible nitric oxide synthase in hibernating myocardium." Title page, contents and summary only, 2002. http://web4.library.adelaide.edu.au/theses/09PH/09phw279.pdf.

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Amendments inserted at back. "May 2002" Includes bibliographical references (leaves 237-290) Experiments described in this thesis address the potential role of inducible nitric oxide synthase (iNOS) in hibernating myocardium. Specifically it was sought to establish a cellular model of hibernating myocardium and investigate the expression, regulation and effects of iNOS in this model. Experiments were performed using primary cultures of neonatal rat ventricular myocytes.

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7

Shukla, Nilima. "Pathophysiology and treatment of intimal hyperplasia and vein graft failure : a focus on risk factors, nitric oxide and oxidant stress." Thesis, Imperial College London, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.397800.

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8

Ahlers, Belinda A. "Regulated L-Arginine transport in heart failure." Monash University, Faculty of Medicine, 2003. http://arrow.monash.edu.au/hdl/1959.1/9521.

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9

Tolias, Christos. "Extracellular superoxide and nitric oxide : a real-time investigation in the role of free radicals in brain cell physiology and pathophysiology in vitro." Thesis, University of Warwick, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.484267.

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10

Beamer, Edward. "A kainic acid-induced status epilepticus model of epileptogenesis in the C57BL/6J mouse : interventions targeting nitric oxide and NMDA receptor-mediated pathophysiology." Thesis, University of Liverpool, 2012. http://livrepository.liverpool.ac.uk/11993/.

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In this thesis, the behavioral, electrographic and neurobiological effects of a period of kainic acid-induced status epilepticus (SE) on the C57BL/6J inbred mouse strain are characterised. The severity of epileptic behaviour was scored, used immunohistochemistry to investigate the anatomical distribution of c-Fos expression in the hippocampal formation following SE and recorded EEG during and after SE using an implantable, wireless telemetry device. Further to assessing the severity of SE, changes subsequent to seizures related to the emergence of chronic epilepsy were investigated, including

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11

Boulanger, Véronique. "Effects of proinflammatory agents on oxygen species production by bovine mammary epithelial and immune cells." Thesis, McGill University, 2000. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=30348.

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The purpose of this study was to investigate which type(s) of somatic cells release nitric oxide (NO) in response to Escherichia coli lipopolysaccharide (LPS) and cytokines in vitro and how NO affects superoxide anion (O2-) production by bovine neutrophils and blood monocytes. Mammary epithelial cell line (FbE) released NO after stimulation with recombinant bovine interleukin-1beta (rBoIL-1beta). Moreover, monocytes produced NO in response to recombinant bovine interferon gamma (rBoIFN-gamma) alone or in combination with LPS in a dose- and time-dependent manner. Nitric oxide production was dim

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12

Tiev, Kiet Phong. "Rôle du monoxyde d'azote dans la physiopathologie des atteintes pulmonaires de la sclérodermie systémique." Thesis, Paris Est, 2008. http://www.theses.fr/2008PEST0081.

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La pneumopathie interstitielle (PI) est devenue la principale cause de décès de la sclérodermie systémique (ScS). Au cours de PI, l’activation immunitaire déclenche une forte production du monoxyde d’azote (NO) et l’augmentation de la concentration de NO dans l’air expiré des patients atteints de ScS avec PI suggère que cette méthode pourrait détecter précocement l’alvéolite, afin de traiter à temps la PI pour éviter son évolution vers la fibrose pulmonaire. En utilisant le modèle à deux compartiments séparant le NO alvéolaire (CANO) du NO bronchique, nous avons montré que la CANO est : (1) au

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13

Kristjánsson, Guðjón. "Food Antigen Sensitivity in Coeliac Disease Assessed by the Mucosal Patch Technique." Doctoral thesis, Uppsala University, Department of Medical Sciences, 2005. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-6020.

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<p>A diagnosis of coeliac disease (CD) in adults relies on the presence of a structurally abnormal intestinal mucosa, followed by a clear clinical remission on a gluten-free diet. There is a clear need for a rapid, simple, safe and sensitive method to determine the type and intensity of inflammation in the gut mucosa in clinical practice. The overall aims of our studies were to develop and evaluate a new technique, “the mucosal patch technique”, to characterize rectal local inflammatory process after rectal food challenge in patients with CD<b>. In study 1</b> we evaluated the potential of the

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14

Angelo, Robert Michael. "Nitric Oxide in Health and Disease: Physiology, Pathophysiology, and Clinical Measurement." Diss., 2008. http://hdl.handle.net/10161/919.

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<p>Red blood cell-dependent hypoxic vasodilation is largely mediated via the delivery of NO through <em>S</em>-nitrosohemoglobin (Hb-SNO). Hb-SNO is regulated through allosteric and redox mechanisms that are not well understood. Part One of this dissertation explores the biochemical features of Hb micropopulations suggested to be involved in Hb-SNO synthesis. An NO-liganded mixed valency micropopulation was synthesized <em>in vitro</em> and identified spectroscopically as a ferric nitrosyl species (Fe(III)NO). Remarkably, this species was found to undergo a reaction that couples heme reduction

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15

"Studies of the aging patterns of nitric oxide synthase in rodent hippocampus." 1997. http://library.cuhk.edu.hk/record=b5896260.

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by Wong Ho Wai.<br>Thesis (M.Phil.)--Chinese University of Hong Kong, 1997.<br>Includes bibliographical references (leaves 107-129).<br>Abstract --- p.i<br>List of Abbreviations --- p.ii<br>Contents --- p.iii<br>Chapter Chapter 1. --- Introduction<br>Chapter 1.1 --- Introduction of aging in central nervous system --- p.1<br>Chapter 1.2 --- Introduction of hippocampus<br>Structure of the hippocampus --- p.4<br>Function of hippocampus --- p.6<br>Chapter 1.3 --- A literature review of aging in hippocampus<br>Cell loss in aging --- p.8<br>Ultrastructural changes in aging --- p.9<br>Changes

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16

Parajuli, Nirmal [Verfasser]. "The role of nitric oxide synthases in the pathophysiology of chronic obstructive pulmonary disease / by Nirmal Parajuli." 2009. http://d-nb.info/1000411672/34.

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17

"Role of nitric oxide (NO), NO synthases and soluble guanylyl cyclase/cGMP/protein kinase G signaling pathway in the regulation of apoptosis and cell proliferation in pancreatic islets and ovarian cancer cells." Thesis, 2006. http://library.cuhk.edu.hk/record=b6074229.

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In the studies about ovarian cancer cells, basal iNOS expression in the chemosensitive OV2008 cells was significantly higher than in the chemoresistant C13* cells. Cisplatin further increased iNOS expression in OV2008 cells, but had no effect in C13* cells. Furthermore, cisplatin dramatically reduced the expression levels of eNOS and nNOS, but again only in OV2008 cells. The data suggest that failure of cisplatin to upregulate iNOS and downregulate eNOS and nNOS in C13* cells could be an etiological factor in chemoresistance. Addition of exogenous NO at high levels, using SNAP, significantly i

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18

"Apoptotic DNA fragmentation in the brains of young and aged eNOS-, iNOS- and nNOS-knockout mice." Thesis, 2005. http://library.cuhk.edu.hk/record=b6074119.

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First study determined the effects of genetic deletion of nNOS on the levels of spontaneous apoptosis in brain of young-adult (2-3 months) and aged (12-18 months) mice, using nNOS-knockout mice with age-matched B6129SF2/J mice as wild-type control. The results indicate that aging resulted in 11-fold increase in levels of apoptotic-DNA-fragmentation in B6129SF2/J mouse brain. nNOS-knockout mice demonstrated dramatic (72-fold) increases in levels of apoptotic-DNA-fragmentation in young-adult, but not aged, brains. Aging resulted in decreased number of nNOS-positive cells, increased number of iNO

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19

Walther, Stefanie. "Mechanismen der Urocortin-II-induzierten Stimulation der NO-Produktion in isolierten Kaninchen-Ventrikelmyozyten." Doctoral thesis, 2009. http://hdl.handle.net/11858/00-1735-0000-0006-AF67-7.

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