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Добірка наукової літератури з теми "OCCLUSIONE PORTALE"

Автор: Grafiati

Опубліковано: 11 березня 2023

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Статті в журналах з теми "OCCLUSIONE PORTALE"

1

Bridwell, Rachel E., Sean Clerkin, Nathaniel R. Walker, Brit Long, and Sarah Goss. "Portal Venous Thrombosis in a Special Operations Paratrooper: A Case Report." Military Medicine 187, no. 1-2 (2021): 256–58. http://dx.doi.org/10.1093/milmed/usab387.

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ABSTRACT Portal vein thrombosis is the thrombotic occlusion of the extrahepatic portal system, which can propagate towards the vena caval system. Although rare, it occurs primarily in those with cirrhosis, intra-abdominal infections, malignancy, or hypercoagulable disorders. This report describes the first reported case of a soldier within special operations without identifiable risk factors who was found to have a completely occlusive portal vein thrombosis after approximately 10 days of insidious abdominal pain. This case emphasizes the importance of considering this rare but dangerous patho

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2

Iqbal, Nabeela, Syed Khalid Shah, and Shamima Haneef. "Polycythemia Vera Complicated by Portal Vein Thrombosis and Budd-Chiari Syndrome:." Journal of Bahria University Medical and Dental College 10, no. 02 (2021): 163–65. http://dx.doi.org/10.51985/jbumdc2020019.

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Polycythemia vera is a medical condition characterized by raised hematocrit. Owing to increased viscosity, the blood flow in the vessels become sluggish leading to the clinical features of polycythemia such as headache, blurring of vision, red skin, dizziness, raised blood pressure, itching and more serious medical events like vaso occlusion, thrombosis and strokes. In this case report, polycythemia vera presenting unusually with heamatemesis, melena and abdominal distension. Physical examination of this case revealed massive ascites with dilated veins around the umbilicus. The diagnosis of po

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3

Strbe, Sanja, Slaven Gojkovic, Ivan Krezic, et al. "Over-Dose Lithium Toxicity as an Occlusive-like Syndrome in Rats and Gastric Pentadecapeptide BPC 157." Biomedicines 9, no. 11 (2021): 1506. http://dx.doi.org/10.3390/biomedicines9111506.

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Due to endothelial impairment, high-dose lithium may produce an occlusive-like syndrome, comparable to permanent occlusion of major vessel-induced syndromes in rats; intracranial, portal, and caval hypertension, and aortal hypotension; multi-organ dysfunction syndrome; brain, heart, lung, liver, kidney, and gastrointestinal lesions; arterial and venous thrombosis; and tissue oxidative stress. Stable gastric pentadecapeptide BPC 157 may be a means of therapy via activating loops (bypassing vessel occlusion) and counteracting major occlusion syndromes. Recently, BPC 157 counteracted the lithium

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4

Wang, L. Q., B. G. Persson, and S. Bengmark. "The Influence of Portal Deviation on the Effect of Repeat Dearterializations of a Transplantable Adenocarcinoma to the Rat Liver." HPB Surgery 8, no. 1 (1994): 37–41. http://dx.doi.org/10.1155/1994/68212.

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As liver tumours receive some of their blood supply from the portal vein, we wanted to illustrate the influence of portal blood flow in combination with dearterialization in the treatment of liver tumours. Forty male, inbred Wistar/Furth rats with an adenocarcinoma transplanted to the liver were treated with various inflow occlusions repeated daily for 5 days. Deviation of the portal blood flow alone with an end-side porto-caval shunt did not alter the tumour growth (p = 0.089). Thirty min of repeat dearterializations was potentiated by portal deviation so that tumour growth was delayed (p = 0

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5

Marn, C. S., and I. R. Francis. "CT of portal venous occlusion." American Journal of Roentgenology 159, no. 4 (1992): 717–26. http://dx.doi.org/10.2214/ajr.159.4.1326882.

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6

Shibamoto, Toshishige, Sen Cui, Zonghai Ruan, Wei Liu, Hiromichi Takano, and Yasutaka Kurata. "Hepatic venoconstriction is involved in anaphylactic hypotension in rats." American Journal of Physiology-Heart and Circulatory Physiology 289, no. 4 (2005): H1436—H1441. http://dx.doi.org/10.1152/ajpheart.00368.2005.

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We determined the roles of liver and splanchnic vascular bed in anaphylactic hypotension in anesthetized rats and the effects of anaphylaxis on hepatic vascular resistances and liver weight in isolated perfused rat livers. In anesthetized rats sensitized with ovalbumin (1 mg), an intravenous injection of 0.6 mg ovalbumin caused not only a decrease in systemic arterial pressure from 120 ± 9 to 43 ± 10 mmHg but also an increase in portal venous pressure that persisted for 20 min after the antigen injection (the portal hypertension phase). The elimination of the splanchnic vascular beds, by the o

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7

Terada, N., S. Koyama, J. Horiuchi, and T. Takeuchi. "Participation of adrenoceptors in liver blood flow regulation in anesthetized dogs." American Journal of Physiology-Heart and Circulatory Physiology 253, no. 5 (1987): H1053—H1058. http://dx.doi.org/10.1152/ajpheart.1987.253.5.h1053.

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We evaluated involvement of adrenergic receptors in the responses of the hepatic vasculature to reduction either of portal venous flow or hepatic arterial inflow. Portal vein occlusion caused an increase in hepatic arterial blood flow (HAF) and decreases in hepatic arterial pressure (HAP) and hepatic arterial vascular resistance (HAR) in the intact group. After pretreatment with either yohimbine or prazosin, but not propranolol, occlusion of the portal vein produced a greater decrease in HAP as compared with that in the intact group. No significant changes in HAF, HAR, or hepatic tissue blood

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8

Kamikado, Chiaki, Toshishige Shibamoto, Minoru Hongo, and Shozo Koyama. "Effects of Hct and norepinephrine on segmental vascular resistance distribution in isolated perfused rat livers." American Journal of Physiology-Heart and Circulatory Physiology 286, no. 1 (2004): H121—H130. http://dx.doi.org/10.1152/ajpheart.01136.2002.

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We studied the effects of blood hematocrit (Hct), blood flow, or norepinephrine on segmental vascular resistances in isolated portally perfused rat livers. Total portal hepatic venous resistance ( Rt) was assigned to the portal ( Rpv), sinusoidal ( Rsinus), and hepatic venous ( Rhv) resistances using the portal occlusion (Ppo) and the hepatic venous occlusion (Phvo) pressures that were obtained during occlusion of the respective line. Four levels of Hct (30%, 20%, 10%, and 0%) were studied. Rpv comprises 44% of Rt, 37% of Rsinus, and 19% of Rhv in livers perfused at 30% Hct and portal venous p

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9

Bayraktar, Yusuf, Abdurrahman Kadayifci, Ferhun Balkanci, Burhan Kayhan, David H. Van Thiel, and Sevket Ruacan. "Splenic Artery Occlusion Masking Portal Hypertension." Journal of Clinical Gastroenterology 22, no. 4 (1996): 326–28. http://dx.doi.org/10.1097/00004836-199606000-00021.

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10

Cheng, Yu Fan, Chien Fu Hung, Yang Han Liu, Koon Kwan Ng, and Chung Chueng Tsai. "Hepatic actinomycosis with portal vein occlusion." Gastrointestinal Radiology 14, no. 1 (1989): 268–70. http://dx.doi.org/10.1007/bf01889213.

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