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Готові списки джерел за темами / Perfluorooctane sulphonate / Статті в журналах

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Статті в журналах з теми "Perfluorooctane sulphonate"

Автор: Grafiati

Опубліковано: 10 січня 2023

Оновлено: 28 січня 2023

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1

van Asselt, E. D., R. P. J. J. Rietra, P. F. A. M. Römkens, and H. J. van der Fels-Klerx. "Perfluorooctane sulphonate (PFOS) throughout the food production chain." Food Chemistry 128, no. 1 (September 2011): 1–6. http://dx.doi.org/10.1016/j.foodchem.2011.03.032.

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2

Borg, Daniel, Jasna Bogdanska, Maria Sundström, Stefan Nobel, Helen Håkansson, Ake Bergman, Joseph DePierre, Krister Halldin, and Ulrika Bergström. "Perinatal tissue distribution of perfluorooctane sulphonate (PFOS) in mice." Toxicology Letters 189 (September 2009): S147. http://dx.doi.org/10.1016/j.toxlet.2009.06.820.

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3

Rumsby, Paul C., Clare L. McLaughlin, and Tom Hall. "Perfluorooctane sulphonate and perfluorooctanoic acid in drinking and environmental waters." Philosophical Transactions of the Royal Society A: Mathematical, Physical and Engineering Sciences 367, no. 1904 (October 13, 2009): 4119–36. http://dx.doi.org/10.1098/rsta.2009.0109.

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Анотація:

Perfluorooctane sulphonate (PFOS) and perfluorooctanoic acid (PFOA) are chemicals that have been used for many years as surfactants in a variety of industrial and consumer products. Owing to their persistent, bioaccumulative and toxic (PBT) characteristics, PFOS has been phased out by its principal producer and the use of PFOA has been reduced. This PBT potential and a number of pollution incidents have led in recent years to an increase in studies surveying the concentrations of PFOS and PFOA in environmental waters worldwide. This paper reviews the results of these studies, as well as the monitoring that was conducted after the pollution incidents. The results of surveys suggest that PFOS and PFOA are found in environmental waters worldwide at low levels. In general, these levels are below health-based values set by international authoritative bodies for drinking water. There have been limited measurements of these chemicals in drinking water, but again these are below health-based values, except in some cases following pollution incidents. Monitoring studies suggested that where PFOS and PFOA were detected, they were at similar levels in both source and drinking water, suggesting that drinking water treatment does not remove these chemicals. However, new data show that PFOS and PFOA are effectively removed by granular activated carbon absorbers in practice. Further research is required on the newer perfluorinated chemicals that appear to be safer, but their degradation products have not as yet been fully studied.

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4

Umar Ijaz, Muhammad, Ayesha Rauf, Shama Mustafa, Hussain Ahmed, Asma Ashraf, Khalid Al-Ghanim, Satyanarayana Swamy Mruthinti, and S. Mahboob. "Pachypodol attenuates Perfluorooctane sulphonate-induced testicular damage by reducing oxidative stress." Saudi Journal of Biological Sciences 29, no. 3 (March 2022): 1380–85. http://dx.doi.org/10.1016/j.sjbs.2021.12.012.

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5

Zhang, Ling, Yuan-yuan Li, Tian Chen, Wei Xia, Yin Zhou, Yan-jian Wan, Zi-quan Lv, Geng-qi Li, and Shun-qing Xu. "Abnormal development of motor neurons in perfluorooctane sulphonate exposed zebrafish embryos." Ecotoxicology 20, no. 4 (February 5, 2011): 643–52. http://dx.doi.org/10.1007/s10646-011-0604-6.

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6

McLaughlin, Clare L., Simon Blake, Tom Hall, Mark Harman, Rakesh Kanda, Jim Foster, and Paul C. Rumsby. "Perfluorooctane sulphonate in raw and drinking water sources in the United Kingdom." Water and Environment Journal 25, no. 1 (February 9, 2011): 13–21. http://dx.doi.org/10.1111/j.1747-6593.2009.00183.x.

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7

Clara, M., S. Scharf, S. Weiss, O. Gans, and C. Scheffknecht. "Emissions of perfluorinated alkylated substances (PFAS) from point sources—identification of relevant branches." Water Science and Technology 58, no. 1 (July 1, 2008): 59–66. http://dx.doi.org/10.2166/wst.2008.641.

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Effluents of wastewater treatment plants are relevant point sources for the emission of hazardous xenobiotic substances to the aquatic environment. One group of substances, which recently entered scientific and political discussions, is the group of the perfluorinated alkylated substances (PFAS). The most studied compounds from this group are perfluorooctanoic acid (PFOA) and perfluorooctane sulphonate (PFOS), which are the most important degradation products of PFAS. These two substances are known to be persistent, bioaccumulative and toxic (PBT). In the present study, eleven PFAS were investigated in effluents of municipal wastewater treatment plants (WWTP) and in industrial wastewaters. PFOS and PFOA proved to be the dominant compounds in all sampled wastewaters. Concentrations of up to 340 ng/L of PFOS and up to 220 ng/L of PFOA were observed. Besides these two compounds, perfluorohexanoic acid (PFHxA) was also present in nearly all effluents and maximum concentrations of up to 280 ng/L werde measured. Only N-ethylperfluorooctane sulphonamide (N-EtPFOSA) and its degradation/metabolisation product perfluorooctane sulphonamide (PFOSA) were either detected below the limit of quantification or were not even detected at all. Beside the effluents of the municipal WWTPs, nine industrial wastewaters from six different industrial branches were also investigated. Significantly, the highest emissions or PFOS were observed from metal industry whereas paper industry showed the highest PFOA emission. Several PFAS, especially perfluorononanoic acid (PFNA), perfluorodecanoic acid (PFDA), perfluorododecanoic acid (PFDoA) and PFOS are predominantly emitted from industrial sources, with concentrations being a factor of 10 higher than those observed in the municipal WWTP effluents. Perfluorodecane sulphonate (PFDS), N-Et-PFOSA and PFOSA were not detected in any of the sampled industrial point sources.

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8

Xiao-Lan, Huang, Wu Hui-Qin, Huang Fang, Lin Xiao-Shan, and Zhu Zhi-Xin. "Determination of Perfluorooctane Sulphonate in Fabrics and Leathers using Liquid Chromatography-Mass Spectrometry." Chinese Journal of Analytical Chemistry 35, no. 11 (November 2007): 1591–95. http://dx.doi.org/10.1016/s1872-2040(07)60094-2.

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9

Zhuo, Qiongfang, Jiao Han, Junfeng Niu, and Junping Zhang. "Degradation of a persistent organic pollutant perfluorooctane sulphonate with Ti/SnO2–Sb2O5/PbO2-PTFE anode." Emerging Contaminants 6 (2020): 44–52. http://dx.doi.org/10.1016/j.emcon.2019.11.002.

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10

Li, Yin-Ming, and Fu-Shen Zhang. "Characterization of a cetyltrimethyl ammonium bromide-modified sorbent for removal of perfluorooctane sulphonate from water." Environmental Technology 35, no. 20 (May 5, 2014): 2556–68. http://dx.doi.org/10.1080/09593330.2014.912253.

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11

Jahja, Felicia Erika, and Muhammad Ilyas. "Literature Review: Cancers Among Workers Exposed To Perfluorooctane Sulfonate (PFOS)." Indonesian Journal of Community and Occupational Medicine 1, no. 3 (March 25, 2022): 179–88. http://dx.doi.org/10.53773/ijcom.v1i3.28.179-88.

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Introduction: Cancer is a disease with a high mortality rate in Indonesia. One of the chemical exposures that can cause cancer is perfluorooctanesulfonyl fluoride/perfluoro octane sulphonate (PFOS). PFOS is a chemical that is widely used due to its diverse functions, including use in fire extinguishers, waterproof materials in food boxes and furniture, and household hygiene products. Long-term exposure to workers affected by PFOS is suspected to be one of the risk factors for cancer. Methods: The author conducted a literature search of online search engines in 3 sources PubMed, Google Scholar, and JSTOR databases. The inclusion criteria in this study are articles in English, full text available, and conducted on the working population. The author has not restricted search within the last 5 years due to the limited reference of the topic. Results: Out of a total of 5 articles examined, the authors found the incidence of bladder cancer in workers as much as 1.28 times, there was even a death rate of 12.77 times in workers with PFOS exposure due to bladder cancer, although the results did not statistically significantly. Other studies have shown increased levels of PFOS in the blood but were not associated with changes in other laboratory examinations associated with health problems in workers. Conclusions: The results of the literature study found no significant association in cancer occurrence based on the length of PFOS exposure in workers. The literature search results also found no direct association between PFOS exposure to possible health problems.

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12

Völkel, W., O. Genzel-Boroviczény, H. Demmelmair, C. Gebauer, B. Koletzko, D. Twardella, U. Raab, and H. Fromme. "Perfluorooctane sulphonate (PFOS) and perfluorooctanoic acid (PFOA) in human breast milk: Results of a pilot study." International Journal of Hygiene and Environmental Health 211, no. 3-4 (July 2008): 440–46. http://dx.doi.org/10.1016/j.ijheh.2007.07.024.

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13

Karakas-Celik, Sevim, and Nurcan Aras. "An in-vitro investigation of the effect of perfluorooctane sulphonate on cell lines of embryonic origin." Molecular Biology Reports 41, no. 6 (February 18, 2014): 3755–59. http://dx.doi.org/10.1007/s11033-014-3240-4.

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14

Han, Rui, Mingxian Hu, Qiang Zhong, Chong Wan, Limin Liu, Fang Li, Fang Zhang та Wenjun Ding. "Perfluorooctane sulphonate induces oxidative hepatic damage via mitochondria-dependent and NF-κB/TNF-α-mediated pathway". Chemosphere 191 (січень 2018): 1056–64. http://dx.doi.org/10.1016/j.chemosphere.2017.08.070.

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15

Zheng, Xiao-qi, Ya-juan Shi, Yong-long Lu, and Xiang-bo Xu. "Growth inhibition and DNA damage in the earthworm (Eisenia fetida) exposed to perfluorooctane sulphonate and perfluorooctanoic acid." Chemistry and Ecology 32, no. 2 (January 6, 2016): 103–16. http://dx.doi.org/10.1080/02757540.2015.1116524.

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16

Zhao, Changwei, Guangshuo Hu, Deyin Hou, Ling Yu, Ying Zhao, Jun Wang, Aixin Cao, and Yonghong Zhai. "Study on the effects of cations and anions on the removal of perfluorooctane sulphonate by nanofiltration membrane." Separation and Purification Technology 202 (August 2018): 385–96. http://dx.doi.org/10.1016/j.seppur.2018.03.046.

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17

Kong, Xiangzhen, Wenxiu Liu, Wei He, Fuliu Xu, Albert A. Koelmans, and Wolf M. Mooij. "Multimedia fate modeling of perfluorooctanoic acid (PFOA) and perfluorooctane sulphonate (PFOS) in the shallow lake Chaohu, China." Environmental Pollution 237 (June 2018): 339–47. http://dx.doi.org/10.1016/j.envpol.2018.02.026.

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18

Sun, Menghan, Mingrui Cui, Yibo Wang, Xinfei Fan, and Chengwen Song. "Enhanced Permeability and Removal Efficiency for Phenol and Perfluorooctane Sulphonate by a Multifunctional CNT/Al2O3 Membrane with Electrochemical Assistance." Journal of Nanoscience and Nanotechnology 20, no. 9 (September 1, 2020): 5951–58. http://dx.doi.org/10.1166/jnn.2020.18554.

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Membrane separation is recognized to be a promising technology for addressing water crisis. Unfortunately, the emergence of membrane fouling and low removal efficiency makes it unattractive for practical application. Herein, an electrochemical multifunctional CNT/Al2O3 membrane was designed coupled multiple electrochemical functions with pore sieving, which could maintain high permeability and achieve good removal efficiency simultaneously, even for those molecules with size smaller than pore size. The multifunctional CNT/Al2O3 membrane possessing a pore size of 140 nm and pure water flux of 869.6 L · m-2 · h-1 · bar-1 was prepared. The results show that the multifunctional CNT/Al2O3 membrane exhibited a good anti-fouling properties for both bio-fouling and chemical fouling under electrochemical assistance with a permeability 3.6 and 1.5 times higher than those of CNT/Al2O3 membrane alone for the treatment of E. coli and humic acid, respectively. In addition, the CNT/Al2O3 membrane with electrochemical assistance also shows a high removal efficiency for the treatment of perfluorooctane sulphonate (PFOS) and phenol whose sizes are smaller than pore size. As for the treatment of surface water, it also presented a good performance. Finally, the regeneration of the membrane was investigated and the fouled membrane was reused through an electrochemical assisted back-wash method.

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19

Lertassavakorn, Teerapong, Nanthanit Pholphana, Nuchanart Rangkadilok, Tawit Suriyo, and Jutamaad Satayavivad. "Determination of perfluorooctane sulphonate and perfluorooctanoic acid in seafood and water from Map Ta Phut Industrial Estate area, Thailand." Food Additives & Contaminants: Part A 38, no. 8 (June 2, 2021): 1400–1415. http://dx.doi.org/10.1080/19440049.2021.1921281.

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20

Liao, Yanyan, Sijun Dong, Ryoiti Kiyama, Peng Cai, Liangpo Liu, and Heqing Shen. "Flos Lonicerae Extracts and Chlorogenic Acid Protect Human Umbilical Vein Endothelial Cells from the Toxic Damage of Perfluorooctane Sulphonate." Inflammation 36, no. 3 (February 8, 2013): 767–79. http://dx.doi.org/10.1007/s10753-013-9603-5.

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21

Zhao, Changwei, Tong Zhang, Guangshuo Hu, Jun Ma, Ruiping Song, and Jiding Li. "Efficient removal of perfluorooctane sulphonate by nanofiltration: Insights into the effect and mechanism of coexisting inorganic ions and humic acid." Journal of Membrane Science 610 (September 2020): 118176. http://dx.doi.org/10.1016/j.memsci.2020.118176.

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22

Caserta, Donatella, Francesca Ciardo, Giulia Bordi, Cristiana Guerranti, Emiliano Fanello, Guido Perra, Francesca Borghini, et al. "Correlation of Endocrine Disrupting Chemicals Serum Levels and White Blood Cells Gene Expression of Nuclear Receptors in a Population of Infertile Women." International Journal of Endocrinology 2013 (2013): 1–7. http://dx.doi.org/10.1155/2013/510703.

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Significant evidence supports that many endocrine disrupting chemicals could affect female reproductive health. Aim of this study was to compare the internal exposure to bisphenol A (BPA), perfluorooctane sulphonate (PFOS), perfluorooctanoic acid (PFOA), monoethylhexyl phthalate (MEHP), and di(2-ethylhexyl) phthalate (DEHP) in serum samples of 111 infertile women and 44 fertile women. Levels of gene expression of nuclear receptors (ERα, ERβ, AR, AhR, PXR, and PPARγ) were also analyzed as biomarkers of effective dose. The percentage of women with BPA concentrations above the limit of detection was significantly higher in infertile women than in controls. No statistically significant difference was found with regard to PFOS, PFOA, MEHP and DEHP. Infertile patients showed gene expression levels of ERα, ERβ, AR, and PXR significantly higher than controls. In infertile women, a positive association was found between BPA and MEHP levels and ERα, ERβ, AR, AhR, and PXR expression. PFOS concentration positively correlated with AR and PXR expression. PFOA levels negatively correlated with AhR expression. No correlation was found between DEHP levels and all evaluated nuclear receptors. This study underlines the need to provide special attention to substances that are still widely present in the environment and to integrate exposure measurements with relevant indicators of biological effects.

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23

Han, Rui, Fang Zhang, Chong Wan, Limin Liu, Qiang Zhong та Wenjun Ding. "Effect of perfluorooctane sulphonate-induced Kupffer cell activation on hepatocyte proliferation through the NF-κB/TNF-α/IL-6-dependent pathway". Chemosphere 200 (червень 2018): 283–94. http://dx.doi.org/10.1016/j.chemosphere.2018.02.137.

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24

Hartmann, Christina, Wolfgang Raffesberg, Sigrid Scharf, and Maria Uhl. "Research Article. Perfluoroalkylated substances in human urine: results of a biomonitoring pilot study." Biomonitoring 4, no. 1 (April 1, 2017): 1–10. http://dx.doi.org/10.1515/bimo-2017-0001.

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AbstractPerfluoroalkylated substances (PFASs) are a class of synthetic chemicals used in a wide range of processes and products due to their unique physicalchemical properties. Through intake of PFASs via food or several consumer products, humans can be exposed. Long-chain PFASs have been associated with adverse effects in laboratory animals, and there is also evidence for adverse health effects in humans. Although investigations of human exposure are mainly conducted in blood samples, some studies have shown that especially short-chain PFASs can be detected in human urine. In the present study, a sensitive analytical method was adapted for the measurement of 12 PFASs in human urine samples by HPLC-MS/MS. For verifying this method, concentrations in 11 male and female participants aged 25-46 years were analysed. In the study population, ranges of urinary PFASs concentrations were n.d.- 8.5 ng/l for perfluoropentanoic acid, <LOQ-3.0 ng/l for perfluorohexanoic acid, n.d.-1.8 ng/l for perfluorohexane sulphonate, n.d.-0.99 ng/l for perfluoroheptanoic acid, 0.79-5.1 ng/l for perfluorooctanoic acid, <LOQ-4.9 ng/l for perfluorooctane suphonate, and <LOQ-0.57 ng/l for perfluorononanoic acid. For the other investigated PFASs, no urinary exposure could be identified in any of the samples. The present study shows that several shortchain PFASs are detectable in human urine.

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25

Zeng, Huai-cai, Bi-qi Zhu, You-quan Wang, and Qing-zhi He. "ROS-Triggered Autophagy Is Involved in PFOS-Induced Apoptosis of Human Embryo Liver L-02 Cells." BioMed Research International 2021 (April 5, 2021): 1–9. http://dx.doi.org/10.1155/2021/6625952.

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The liver is the primary target organ for perfluorooctane sulphonate (PFOS), a recently discovered persistent organic pollutant. However, the mechanisms mediating hepatotoxicity remain unclear. Herein, we explored the relationship between reactive oxygen species (ROS) and autophagy and apoptosis induced by PFOS in L-02 cells, which are incubated with different concentrations of PFOS (0, 50, 100, 150, 200, or 250 μmol/L) for 24 or 48 hrs at 37°C. The results indicated that PFOS exposure decreased cell activities, enhanced ROS levels in a concentration-dependent manner, decreased mitochondrial membrane potential (MMP), and induced autophagy and apoptosis. Compared with the control, 200 μmol/L PFOS increased ROS levels; enhanced the expression of Bax, cleaved-caspase-3, and LC3-II; induced autophagy; decreased MMP; and lowered Bcl-2, p62, and Bcl-2/Bax ratio. The antioxidant N-acetyl cysteine (NAC) protected MMP against PFOS-induced changes and diminished apoptosis and autophagy. Compared with 200 μmol/L PFOS treatment, NAC pretreatment reversed the increase in ROS, Bax, and cleaved-caspase-3 protein caused by PFOS, lowered the apoptosis rate increased by PFOS, and increased the levels of MMP and Bcl-2/Bax ratio decreased by PFOS. The autophagy inhibitor 3-methyladenine and chloroquine decreased apoptosis and cleaved-caspase-3 protein level and increased the Bcl-2/Bax ratio. In summary, our results suggest that ROS-triggered autophagy is involved in PFOS-induced apoptosis in L-02 cells.

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26

Shen, Chensi, Jiaxin Ding, Chenye Xu, Long Zhang, Shuren Liu, and Yonghong Tian. "Perfluoroalkyl Mixture Exposure in Relation to Fetal Growth: Potential Roles of Maternal Characteristics and Associations with Birth Outcomes." Toxics 10, no. 11 (October 28, 2022): 650. http://dx.doi.org/10.3390/toxics10110650.

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Perfluoroalkyl substances (PFASs) exposure is suggested to interfere with fetal growth. However, limited investigations considered the roles of parity and delivery on PFASs distributions and the joint effects of PFASs mixture on birth outcomes. In this study, 506 birth cohorts were investigated in Hangzhou, China with 14 PFASs measured in maternal serum. Mothers with higher maternal ages who underwent cesarean section were associated with elevated PFASs burden, while parity showed a significant but diverse influence. A logarithmic unit increment in perfluorooctanoic acid (PFOA), perfluorooctane sulfonate (PFOS), and perfluorononane sulfonate (PFNS) was significantly associated with a reduced birth weight of 0.153 kg (95% confidence interval (CI): −0.274, −0.031, p = 0.014), 0.217 kg (95% CI: −0.385, −0.049, p = 0.012), and 0.137 kg (95% CI: −0.270, −0.003, p = 0.044), respectively. Higher perfluoroheptanoic acid (PFHpA) and perfluoroheptane sulphonate (PFHpS) were associated with increased Apgar-1 scores. PFOA (Odds ratio (OR): 2.17, 95% CI: 1.27, 3.71, p = 0.004) and PFNS (OR:1.59, 95% CI: 1.01, 2.50, p = 0.043) were also risk factors to preterm birth. In addition, the quantile-based g-computation showed that PFASs mixture exposure was significantly associated with Apgar-1 (OR: 0.324, 95%CI: 0.068, 0.579, p = 0.013) and preterm birth (OR: 0.356, 95% CI: 0.149, 0.845, p = 0.019). In conclusion, PFASs were widely distributed in the maternal serum, which was influenced by maternal characteristics and significantly associated with several birth outcomes. Further investigation should focus on the placenta transfer and toxicities of PFASs.

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27

Enevoldsen, Rasmus, and René K. Juhler. "Perfluorinated compounds (PFCs) in groundwater and aqueous soil extracts: using inline SPE-LC-MS/MS for screening and sorption characterisation of perfluorooctane sulphonate and related compounds." Analytical and Bioanalytical Chemistry 398, no. 3 (August 27, 2010): 1161–72. http://dx.doi.org/10.1007/s00216-010-4066-0.

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28

Ololade, I. "Hysteretic Desorption of Perfluorooctane Sulphonates within Sediment Components under Aerobic and Anoxic Conditions." International Research Journal of Pure and Applied Chemistry 4, no. 6 (January 10, 2014): 773–84. http://dx.doi.org/10.9734/irjpac/2014/10592.

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29

Zhang, Sheng, and David N Lerner. "Review of Physical and Chemical Properties of Perfluorooctanyl sulphonate (PFOS) with Respect to its Potential Contamination on the Environment." Advanced Materials Research 518-523 (May 2012): 2183–91. http://dx.doi.org/10.4028/www.scientific.net/amr.518-523.2183.

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Анотація:

Perfluorinated surfactants have emerged as priority environmental contaminants due to their detection in environmental and biological matrices as well as concerns regarding their persistence and toxicity. They have been found in groundwater, particularly at sites used for training firefighters. They do not biodegrade easily in groundwater, and are not retarded during transport. The most common chemical is Perfluorooctanyl Sulphonate (PFOS), which is mainly used in aqueous film forming foam (AFFF) to extinguish hydrocarbon-fuel fires. It is also used in many herbicide and insecticide formulations, cosmetics, greases and lubricants, paints, polishes, and adhesives. PFOS and related fluoro-organic chemicals have been used since the 1950s. A quantity of fluorosurfactants and related products are still in use all over the world. Intensive studies over the last few years discovered that PFOS and certain by-products were both ubiquitous in the environment and highly persistent. PFOS does not biodegrade in the environment and very limited degradation has been observed in wastewater treatment. The breakthrough curves of a single-well push-pull test indicated that there was no retardation for PFOS as well. It was detected in part-per-billion levels in blood samples obtained from blood banks in the United States, Japan, Europe, and China. There have been more and more reports on the accumulation and effect of PFOS in wild animals’ liver, serum and muscle as well. This suggests that PFOS can bioaccumulate to higher levels of the food chain.

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30

Zhang, De-Yong, Xiao-Lu Xu, Qin Ruan, Xiu-Ying Shen, and Yin Lu. "Subchronic Effects of Perfluorooctane Sulphonate on the Testicular Morphology and Spermatogenesis in Mice." Pakistan Journal of Zoology 51, no. 6 (2019). http://dx.doi.org/10.17582/journal.pjz/2019.51.6.2217.2223.

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31

Hassan, Masud, Jianhua Du, Yanju Liu, Ravi Naidu, Jin Zhang, Md Ariful Ahsan, and Fangjie Qi. "Magnetic biochar for removal of perfluorooctane sulphonate (PFOS): Interfacial interaction and adsorption mechanism." Environmental Technology & Innovation, April 2022, 102593. http://dx.doi.org/10.1016/j.eti.2022.102593.

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Hassan, Masud, Jianhua Du, Yanju Liu, Ravi Naidu, Jin Zhang, Md Ariful Ahsan, and Fangjie Qi. "Magnetic biochar for removal of perfluorooctane sulphonate (PFOS): Interfacial interaction and adsorption mechanism." Environmental Technology & Innovation, April 2022, 102593. http://dx.doi.org/10.1016/j.eti.2022.102593.

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Mandour, Dalia, ibrahim maher, Marwa Abd El – Aal, and Rania Said Moawad. "Quercetin Ameliorated Perfluorooctane Sulphonate-Induced Hepatic Toxicity in Adult Male Albino Rats (Biochemical, Histological and Immunohistochemical Study)." Egyptian Journal of Histology, July 5, 2022, 0. http://dx.doi.org/10.21608/ejh.2022.139170.1685.

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SANGYO EISEIGAKU ZASSHI 44, № 2 (2002): 79. http://dx.doi.org/10.1539/sangyoeisei.kj00002552738.

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