Добірка наукової літератури з теми "Periodontal disease Microbiology"

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Статті в журналах з теми "Periodontal disease Microbiology":

1
Moore, W. E. C. "Microbiology of periodontal disease." Journal of Periodontal Research 22, no. 5 (September 1987): 335–41. http://dx.doi.org/10.1111/j.1600-0765.1987.tb01595.x.
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2
Peros, William J., and Eugene D. Savitt. "The microbiology of periodontal disease." Clinical Microbiology Newsletter 11, no. 7 (April 1989): 49–51. http://dx.doi.org/10.1016/0196-4399(89)90009-3.
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3
Jenkinson, Howard F., and David Dymock. "The Microbiology of Periodontal Disease." Dental Update 26, no. 5 (June 1999): 191–97. http://dx.doi.org/10.12968/denu.1999.26.5.191.
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4
Riviere, George R. "Spirochetes in periodontal disease." Clinical Microbiology Newsletter 16, no. 19 (October 1994): 148–51. http://dx.doi.org/10.1016/0196-4399(94)90024-8.
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5
Aggarwal, Titiksha, Arundeep Kaur Lamba, Farrukh Faraz, and Shruti Tandon. "Viruses: Bystanders of periodontal disease." Microbial Pathogenesis 102 (January 2017): 54–58. http://dx.doi.org/10.1016/j.micpath.2016.11.019.
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6
Ohta, Hiroyuki, Susumu Kokegughi, Kazuhiro Fukui, and Keijiro Kato. "Actinobacillus (Haemophilus) actinomycetemcomitansin Periodontal Disease." Microbiology and Immunology 30, no. 7 (July 1986): 629–43. http://dx.doi.org/10.1111/j.1348-0421.1986.tb02990.x.
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7
Na, Hee Sam, and Jin Chung. "Link between Periodontal Disease and Diabetes." Journal of Bacteriology and Virology 46, no. 1 (2016): 52. http://dx.doi.org/10.4167/jbv.2016.46.1.52.
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8
Van Dyke, Thomas E. "Thwarting host immune responses in periodontal disease." Trends in Microbiology 6, no. 3 (March 1998): 88–89. http://dx.doi.org/10.1016/s0966-842x(98)01212-8.
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9
Tanner, A. C. R., and J. M. Goodson. "Sampling of microorganisms associated with periodontal disease." Oral Microbiology and Immunology 1, no. 1 (February 1986): 15–20. http://dx.doi.org/10.1111/j.1399-302x.1986.tb00310.x.
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10
Baker, Pamela J., and Derry C. Roopenian. "Genetic susceptibility to chronic periodontal disease." Microbes and Infection 4, no. 11 (September 2002): 1157–67. http://dx.doi.org/10.1016/s1286-4579(02)01642-8.
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Дисертації з теми "Periodontal disease Microbiology":

1
Tam, You-Cheuk. "The role of mucopolysaccharidase-producing anaerobic oral bacteria in the pathogenesis of inflammatory periodontal disease /." Electronic Thesis or Dissertation, McGill University, 1985. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=72044.
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Mucopolysaccharidase-producing oral bacteria may contribute to the pathogenesis of inflammatory periodontal disease in several ways. Bacterial mucopolysaccharidases, in the area of the gingival crevice, can destroy important components of the ground substance of connective tissue leading to periodontal destruction on the one hand and enhancing the spread of bacterial toxins on the other. Partial breakdown of proteoglycan due to penetration of small amounts of these enzymes may also expose cryptic antigenic determinants, resulting in destructive autoimmune reactions. Finally, mucopolysaccharidase-producing oral bacteria may interact symbiotically with other pathogens of the gingival sulcus by enzymatic breakdown of tissue to release fermentable substrates for these pathogens.
In the present investigation, it has been shown that anaerobic mucopolycaccharidase-producing bacteria are common inhabitants of gingival sulci of humans and that these microorganisms significantly increase in number in periodontal pockets of patients with inflammatory periodontal disease. Peptostreptococci probably are the most predominant producers of mucopolysaccharidases by virtue of their occurrence in subgingival plaque as well as the abundance of enzyme they produce. Peptostreptococci strains isolated from diseased periodontal pockets have been observed to convert from rough-colony-forming cells to smooth-colony-forming variants upon culturing in vitro. In dental plaque, hyaluronidase-producing peptostreptococci exist predominantly as the rough-colony-forming variants which produce higher amounts of hyaluronidase. Purified extracellular hyaluronidase from Peptostreptococcus strain 84H14S is different from previously reported bacterial hyaluronidases in several respects. It has different substrate specificity and optimum pH for activity. Also, the specific activity of this enzyme is much higher than any previously purified mucopolysaccharidases. Peptostreptococcus strain 84H14S is further shown to release potent cytotoxic factors into the culture medium, in addition to hyaluronidase, during its growth cycle. This may confer additional virulence to this bacterial genus in periodontal disease.
2
Sweet, Simon Paul. "Adherence, aggregation and hydrophobicity of oral bacteria : with particular reference to microorganisms implicated in periodontal disease." Electronic Thesis or Dissertation, University of Glasgow, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.236908.
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3
Ng, Kwai-sang Sam, and 吳桂生. "Psychological perspectives of periodontal disease." PG_Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B36918210.
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4
Abuaisha, Karim Belkais Faraj. "Prognostic biomarkers of periodontal disease." Electronic Thesis or Dissertation, King's College London (University of London), 2016. https://kclpure.kcl.ac.uk/portal/en/theses/prognostic-biomarkers-of-periodontal-disease(77882787-a695-42a3-b7e4-7caa8e9c4bde).html.
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Objectives: Previous studies in our laboratory have identified the antimicrobial proteins Human Neutrophil Protein 1 – 3 (HNP1-3), Myeloid Related Protein 8 (S100A8/MRP8) and LL-37 as putative periodontal salivary biomarkers. The aims of the studies reported in this thesis were to investigate the diagnostic and prognostic potential of these markers, together with Matrix Metalloproteinase- 8 (MMP-8), serum markers C - reactive protein (CRP) and Interleukin-6 (IL-6), on the initial outcome of nonsurgical periodontal treatment. Material & Methods: We carried out a cross sectional study which aimed to verify and establish a diagnostic threshold for a group of salivary biomarkers (MMP-8, HNP1-3, S100A8 and LL-37) and to test the validity of the diagnostic utility of these biomarkers. A total of 133 unstimulated saliva samples (46 with chronic periodontitis, 38 with aggressive periodontitis and 49 with gingivitis) were analysed by ELISA. In addition multiple markers were combined to give a single combined cut off point by normalising each biomarker to percentage of cut-off point value, (such that x + y = combined cut off point). These pre-determined cut-off points were applied to salivary AMPs levels in an independent cohort originally collected to investigate the effects of diabetes on periodontitis. To investigate prognostic potential a total of 66 participants were recruited to a longitudinal intervention study of patients with moderate–severe Chronic Periodontitis. 53 subjects completed the protocol and were included in the final analysis. Subjects (28 male, 25 female) age range (23-65 years) with were recruited, with 14 smokers and 3 with type II diabetes, and saliva and serum samples were collected prior to periodontal examination. Patients were then given a course of non-surgical periodontal therapy over 2 visits. 8-10 weeks post-operatively saliva/serum sampling and clinical examination were repeated. Salivary MMP-8, S100A8 and HNP1-3 concentrations were all determined by ELISAs. In addition we measured serum levels of CRP and Interleukin 6. Results: In the cross-sectional study the HNP1-3 and S100A8 could differentiate between gingivitis and chronic periodontitis with high specificity (around 90%) and around 75% sensitivity compared to MMP-8 which was able to discriminate between gingivitis and periodontitis (chronic and aggressive) with both high 3 specificity and sensitivity. LL37 showed no significant diagnostic potential. Within the independent cohort the application of pre-determined thresholds, either individual or combined cut-offs, were able to detect periodontitis with specificity of between 75 – 85 % but with very low sensitivity. In addition diabetic status was found to result in significantly increased MMP-8 and S100A8 concentrations in subjects with periodontal disease. In the intervention study, treatment resulted in reductions in the mean: a) number of deep sites (>4mm) (33.57 ± 20.75 vs 18.51 ± 13.87; mean ± SD, p<0.0001); b) probing pocket depths (5.92 ± 0.47 mm vs 4.74 ± 0.76 mm, p<0.0001); c) bleeding index (0.32 ± 0.20 vs 0.21 ± 0.16, p<0.0001); d) plaque index (0.46 ± 0.20 vs 0.37 ± 0.18, p=0.0003). Only the mean concentrations of MMP-8 and S100A8 showed significant reductions post-treatment (MMP-8: 355.4 ± 319.9 ng/ml vs 216.6 ± 217.2 ng/ml, p<0.0001), (S100A8: 1182 ± 1095 ng/ml vs 693.9 ± 719.6 ng/ml, p=0.0007). Only the change in concentrations of MMP-8 were strongly associated with magnitude of treatment response (MMP-8: r2= 0.1, p=0.02). In addition, the baseline levels of MMP-8 & S100A8 were also associated with treatment response (MMP-8: r2= 0.1, p=0.03; S100A8: r2=0.1, p=0.02). Overall, there were 13 out of 53 participants who did not respond to the treatment (24.5% of cases). MMP-8 baseline concentrations were significantly higher in responders (419.3 ± 343.1 ng/ml) than non-responders (158.8 ± 73.3 ng/ml) (p=0.009). MMP-8 concentrations at baseline that were above the cut-off (<182.8 ng/ml) predicted a good response to periodontal treatment with 77% sensitivity and 70% specificity. There was no effect of the single round of non-surgical periodontal treatment on the levels of systemic markers CRP & IL-6, and also there was no correlation between local and systemic markers. Conclusion: These results of both studies suggest that MMP-8, HNP1-3 and S100A8 may be useful to identify cases of periodontitis with good specificity and moderate sensitivity and may give superior results when combined. In addition the salivary MMP-8 and S100A8 showed promising periodontal prognostic ability to detect the likelihood of a good response to treatment, with MMP-8 showed the best results with moderate sensitivity and specificity. However, further validation studies would be useful in larger, non-diabetic cohorts.
5
Shub, Alexis. "Periodontal disease and adverse pregnancy outcomes." University of Western Australia. School of Women's and Infants' Health, 2007. http://theses.library.uwa.edu.au/adt-WU2007.0184.
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[Truncated abstract] Periodontal disease is a common and underdiagnosed disease in humans that may have adverse effects on pregnancy outcomes. The aim of this thesis was to investigate the effects of periodontal disease in pregnancy by means of two observational human studies and the development of animal models of fetal and uterine exposure to periodontopathic bacteria and lipopolysaccharide. I performed a prospective study examining the rates of preterm birth, small for gestational age neonates and neonatal inflammation in 277 women who had undergone a detailed antenatal periodontal examination and oral health questionnaire. Periodontal disease was associated with small for gestational age neonates, and increased CRP levels in umbilical cord blood, but no effect was seen on the rate of preterm birth. Maternal oral health symptoms predicted both periodontal disease and newborn biometry. In a retrospective case control study, I examined the role of periodontal disease in perinatal mortality. Participants included 53 women who had experienced a perinatal loss for which no cause could be found after thorough investigation, and 111 control women. Women who had experienced a perinatal loss were more than twice as likely as controls to have periodontal disease. The incidence of periodontal disease was even higher in women in whom the perinatal loss was due to extreme prematurity. In contrast to my prospective study, risks to the pregnancy could not be predicted by maternal oral health behaviours or oral health symptoms. In order to better understand the mechanisms regulating the associations described in the human studies, two animal models were developed; one to investigate acute exposure and the second to investigate long-term exposure to periodontal pathogens. The first study examined the effects of administration of a bolus of periodontopathic bacteria and lipopolysaccharide to the pregnant sheep. Injection of bacteria and lipopolysaccharide in the amniotic fluid of the pregnant preterm sheep caused a high rate of fetal lethality, disturbance of fetal acid base status and inflammation of the fetus and membranes. Given the circumstances of exposure to periodontopathic pathogens in human periodontal disease, a model investigating long-term exposure to periodontopathic lipopolysaccharide on pregnancy outcomes was developed. ... Overall, I have demonstrated that maternal periodontal disease is associated with adverse pregnancy outcomes including fetal growth restriction and possibly perinatal loss. Mechanisms regulating these effects are likely to be mediated by fetal adaptations to intrauterine inflammation resulting in altered fetal development, growth or survival. Randomised controlled trials that are currently in progress will provide further information on the effects of periodontal disease in human pregnancy, and the efficacy of treatment to reduce these adverse outcomes.
6
Mak, Yun-lok Raymond, and 麥潤樂. "Profile changes of putative periodontal pathogens after non-surgical periodontal treatment." PG_Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2002. http://hub.hku.hk/bib/B31954236.
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7
Cruz, Gabriela Alessandra da. "Raspagem e alisamento radicular realizado em uma unica sessão, em pacientes com diabetes melito, portadores de periodontite cronica : avaliações clinica e laboratorial." PublishedVersion, [s.n.], 2001. http://repositorio.unicamp.br/jspui/handle/REPOSIP/289998.
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Анотація:
Orientadores: Sergio de Toledo, Enilson Antonio Sallum
Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba
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Resumo: O objetivo deste estudo foi a avaliação clínica, hematológica e microbiológica do tratamento periodontal para ambos os grupos após realizar tratamento de raspagem e alisamento radicular em única sessão. Esse trabalho é um estudo prospectivo, paralelo e comparativo realizado em pacientes com doença periodontal crônica generalizada divididos em dois grupos, sendo 10 pacientes com diabetes melito insulino-dependente (DM) e 10 pacientes não diabéticos (NDM) acompanhados por 3 meses. Os dados obtidos nos períodos inicial e final foram divididos em parâmetros clínicos: índice de placa, Índice gengival, nível clínico de inserção, nível da margem gengival e profundidade de sondagem; parâmetros hematológicos: hemácias, hemoglobina, hematócrito, volume corpuscular médio, hemoglobina corpuscular média, concentração de hemoglobina corpuscular média, leucócitos, eosinófilos, metamielócitos, segmentados, linfócitos, monócitos, plaquetas, bastonetes, basófilos, glicemia, hemoglobina glicosilada (HbA1c), prova do laço, tempo de coagulação e sangramento e parâmetros microbiológicos, ¿pool¿ de bactéria por paciente, para os sítios com PS>5mm e regiões de bifurcação. As amostras foram obtidas com curetas tipo Gracey e analisadas através da técnica de reação de polimerase em cadeia ¿ PCR para verificação da freqüência de Actinobacillus actinomycetemcomitans, Porphyromonas gingivalis e Tannarella forsythensis. As avaliações clinicas e hematológicas não demonstraram diferenças estatísticas significantes entre grupos NDM e DM nos períodos iniciais e final. A avaliação microbiológica demonstrou diminuição de Tannarella forsythensis estatisticamente significativa apenas para o grupo NDM (p=0,0313) após 3 meses. Este estudo sugere que os pacientes com DM apresentaram respostas clinica e laboratorial similares ao grupo NDM após a terapia de raspagem e alisamento radicular realizados em única sessão após o período de acompanhamento de 3 meses
Abstract: The aim of this study was to compare the clinical, hematological and microbiological responses of periodontal treatment for both groups after full-mouth scaling and root planning. This study is a prospective, parallel, comparative, clinical, hematological and microbiological study performed in patients with generalized chronic periodontal disease separated in two groups, with 10 patients with diabetes mellitus insulin-dependent (DM) and 10 patients non-diabetics (NDM) attended by 3 months. The data from baseline and 3 months were separated in clinical parameters, plaque Index, gingival index, clinical attachment level, gingival margin level and probing depth; hematological parameters, red blood cell count, hemoglobin, hematocrit, mean corpuscular volume, mean corpuscular hemoglobin, mean corpuscular hemoglobin concentration, leucocytes, eosinophils, metamielocytes, segmenteds, linfocytes, monocytes, platelets, bastonets, basophil, glucose, HbA1c, bleeding time and coagulation time and microbiological parameters, pool of bacterial per patients that were obtained from sites with PD>5mm and furcation sites. The samples were obtained with Gracey curettes and analyzed by technical of polymerase chain reaction ¿ PCR, to verify the frequency of Actinobacillus actinomycetemcomitans, Porphyromonas gingivalis e Tannarella forsythensis. The clinical and hematological evaluations had no statistics significance changes between groups NDM and DM. The microbiological evaluation showed decrease of Tannarella forsythensis statistically significant for only NDM group (p=0,0313). This study suggest that diabetes mellitus patients respond to full-mouth scaling and root planning similar to non diabetics patients 3 months after therapy
Doutorado
Periodontia
Mestre em Clínica Odontológica
8
Martins, Angela Guimarães. "Avaliação clinica e microbiologica do uso de pontas sonicas diamantadas na descontaminação radicular (estudo longitudinal)." PublishedVersion, [s.n.], 2005. http://repositorio.unicamp.br/jspui/handle/REPOSIP/290413.
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Анотація:
Orientador: Antonio Wilson Sallum
Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba
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Resumo: O objetivo deste estudo foi avaliar longitudinalmente a efetividade de pontas sônicas diamantadas (RootplanerTM), na descontaminação de superfícies radiculares de dentes unirradiculares sob parâmetros clínicos (índice de placa, sangramento à sondagem, profundidade de sondagem, nível de inserção clínica relativo, e retração gengival) e microbiológicos (cultura microbiana e PCR), comparados a instrumentação com curetas. Esta pesquisa trata-se de um teste clínico controlado randomizado com um delineamento em parcela subdividida, envolvendo 12 pacientes com moderada a avançada periodontite crônica apresentando profundidade de sondagem ³ 5 mm. Os pacientes foram submetidos a tratamento inicial com sessões de remoção de placa e cálculo supragengival e instruções de higiene bucal. Posteriormente, retalho cirúrgico foi realizado e a superfície dos dentes instrumentada. Os parâmetros clínicos e microbiológicos foram avaliados no tempo inicial (T0), e em 30 dias, 6, 12 e 24 meses após a cirurgia. Os dados obtidos foram submetidos à análise estatística através de Análise de variância (ANOVA), teste de Tukey, realizados para todos os períodos (a=0.05), Friedman e Wilcoxon foram usados na avaliação não paramétrica das unidades formadoras de colônias. Os resultados mostraram que quando as pontas sônicas diamantadas foram comparadas a instrumentos manuais nenhuma diferença estatística foi encontrada em redução de profundidade de sondagem, índice de placa, sangramento a sondagem, ganho de inserção, recessão gengival. Bem como, nas reduções das contagens microbiológicas das UFCs e diminuição de A.a., P.g. e T.f.. Os resultados não mostraram diferença significativa entre os tratamentos, apenas entre os períodos de avaliação. Concluiu-se, dentro das limitações do estudo que as pontas sônicas diamantadas podem ser uma boa alternativa terapêutica para instrumentação radicular, mantendo a estabilidade da saúde dos tecidos periodontais em longo prazo
Abstract: The aim of this study was to evaluate the long term the effectiveness of diamond-coated sonic scaler insert (RootplanerTM), in the root surface debridement in single-rooted teeth on clinical (plaque index, bleeding on probing, probing depth, relative attachment level clinical, and gingival recession) and microbiological parameters (culture and PCR), compared to hand instruments. The investigation was a test-controlled randomized clinical trial with a splith-mouth design involving 12 patients with moderate to advanced chronic periodontites with pocket depth ³ 5 mm. The patients were submitted to initial treatment sessions of supragingival plaque and cauculus removal and were also instructed in oral hygiene techniques. After, flap surgery was carried out and the tooth surface instrumented. Clinical and microbiological parameters, were evaluated at baseline (0), in the 30 days, 6, 12 and 24 months after surgery. Analysis of variance and Tukey test was done goes all periods (a=0.05) and the results showed that when diamond-coated sonic scaler insert was compared to manual scalers, showed no statistical differences in probing depth reduction, plaque index, bleeding on probing, relative attachment level gain, gingival recession. As well as, reductions of the microbial counts and colony-forming units using Friedman and Wilcoxon test, and decrease of A.a, P.g. and T.f.. There were no significant differences between regard with treatments for all parameters at each re-examination. Within the limits of this study, it can be concluded that the use the of diamond-coated sonic scaler insert can be a good alternative for root debridement, maintaining the stability of the periodontal tissues in long term
Doutorado
Periodontia
Doutor em Clínica Odontológica
9
Moll, Robin John. "Detection of periodontal bacteria in atherosclerotic plaque." Electronic Thesis or Dissertation, University of Central Lancashire, 2016. http://clok.uclan.ac.uk/20464/.
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This study aims to examine the role infection plays in the pathogenesis of atherosclerosis through the molecular identification of latent bacterial species present in atherosclerotic plaque tissue from the carotid artery. Immunohistological examination of the atherosclerotic plaque tissue revealed widespread localisation of the bacterial cell wall constituent peptidoglycan (PGN). Targeted PCR amplification of the 16S rRNA gene permitted identification of a diverse collection of 16S rDNA sequences within atherosclerotic plaque samples. Thirteen of 21 (61.9%) plaques contained bacterial 16S rDNA relating to periodontal (P. gingivalis and T. forsythia), oral (Streptococcus spp.), and respiratory (Klebsiella pneumoniae and Mycobacterium tuberculosis) and commensal (Propionibacterium acnes and Staphylococcus epidermidis) species. Of the 160 isolates recovered P acnes (91%) was most frequently detected, followed by Lactobacillus spp. (3%), S. epidermidis (3%), S. mitis (3%) and S. sanguinis (1%). Challenge of THP-1 cells with a P. acnes isolate recovered from atherosclerotic plaque, resulted in significant temporal up-regulation the major cholesterogenic transcriptions factor, SREBP2 and its two target transcripts, LDLR and HMGR. Similarly, the cholesterol transporter gene ABCA1 was highly expressed in P. acnes-infected THP-1 cells in addition to inflammatory cytokines/chemokines TNFα, IL-1β, CCL3 (MIP-1α), cell adhesion molecule ICAM-1. Anti-apoptotic gene BCL2A1 showed the greatest increase in mRNA compared to all tested genes. Expression of the same panel of genes direct in atherosclerotic plaque tissue resulted in similar expression patterns.
10
Abukhres, Omar Mohamed Omar. "Microaerophilic gram-negative rods in periodontal disease." Electronic Thesis or Dissertation, University of Glasgow, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.320280.
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Книги з теми "Periodontal disease Microbiology":

1
Kinane, Denis F. Periodontal disease. Basel: Karger, 2012.
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2
Ekuni, Daisuke, Maurizio Battino, Takaaki Tomofuji, and Edward E. Putnins, eds. Studies on Periodontal Disease. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-9557-4.
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3
Nakano, Kazuhiko, Takashi Ooshima, and Atsuo Amano. Periodontal diseases in children and adolescents. New York: Nova Science Publishers, 2011.
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4
Berns, Joel M. Understanding periodontal diseases. 2nd ed. Carol Stream, Ill: Quintessence Books, 1993.
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5
Yamamoto, Sho L. Periodontal disease: Symptoms, treatment, and prevention. Hauppauge, N.Y: Nova Science, 2010.
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6
Dumitrescu, Alexandrina L., ed. Etiology and Pathogenesis of Periodontal Disease. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-03010-9.
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7
Buduneli, Nurcan. Biomarkers in Periodontal Health and Disease. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-37317-7.
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8
Linghorne, John. A new nonsurgical treatment of periodontal disease. Toronto: J. Linghorne, 1998.
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9
Dumitrescu, Alexandrina L. Genetic Variants in Periodontal Health and Disease. Berlin, Heidelberg: Springer-Verlag Berlin Heidelberg, 2010.
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Dumitrescu, Alexandrina L., and Junya Kobayashi. Genetic Variants in Periodontal Health and Disease. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-00680-7.
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Частини книг з теми "Periodontal disease Microbiology":

1
Dumitrescu, Alexandrina L., and Masaru Ohara. "Periodontal Microbiology." In Etiology and Pathogenesis of Periodontal Disease, 39–76. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-03010-9_2.
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2
Marsh, Philip, and Michael Martin. "Periodontal diseases." In Oral Microbiology, 167–97. Boston, MA: Springer US, 1992. http://dx.doi.org/10.1007/978-1-4615-7556-6_7.
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3
Taylor, Shelley Segrest. "Periodontal Disease." In Encyclopedia of Otolaryngology, Head and Neck Surgery, 2133. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-23499-6_200156.
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4
Gooch, Jan W. "Periodontal Disease." In Encyclopedic Dictionary of Polymers, 913. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_14452.
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5
Schäfer, Arne S. "Periodontal Disease." In Genomics, Personalized Medicine and Oral Disease, 145–66. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-17942-1_7.
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6
Scarfe, William C., Bruno Azevedo, Lucas Rodrigues Pinheiro, Menik Priaminiari, and Marcelo Augusto Oliveira Sales. "Periodontal Disease." In Maxillofacial Cone Beam Computed Tomography, 923–49. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-62061-9_23.
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7
Pandolfo, Ignazio, and Silvio Mazziotti. "The Periodontal Disease." In Orthopantomography, 121–38. Milano: Springer Milan, 2013. http://dx.doi.org/10.1007/978-88-470-5289-5_6.
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8
Braun-Falco, Markus, Henry J. Mankin, Sharon L. Wenger, Markus Braun-Falco, Stephan DiSean Kendall, Gerard C. Blobe, Christoph K. Weber, et al. "Periodontal Diseases." In Encyclopedia of Molecular Mechanisms of Disease, 1620–21. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_1396.
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9
Oates, Thomas W., and Namita Khandelwal. "Diabetes and Periodontal Disease." In Endocrinology, 451–71. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-44433-8_15.
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Oates, Thomas W., and Namita Khandelwal. "Diabetes and Periodontal Disease." In Endocrinology, 451–71. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-36694-0_15.
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Тези доповідей конференцій з теми "Periodontal disease Microbiology":

1
Weersink, Robert A. "Photodynamic therapy for periodontal disease." In Opto-Canada: SPIE Regional Meeting on Optoelectronics, Photonics, and Imaging, edited by John C. Armitage. SPIE, 2017. http://dx.doi.org/10.1117/12.2283905.
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2
Colston, Jr., Bill W., Matthew J. Everett, Luiz B. Da Silva, and Linda L. Otis. "OCT for diagnosis of periodontal disease." In BiOS '98 International Biomedical Optics Symposium, edited by Valery V. Tuchin and Joseph A. Izatt. SPIE, 1998. http://dx.doi.org/10.1117/12.306070.
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Albalat, Salvador E., Mariano L. Alcaniz-Raya, M. Carmen Juan, Vincente Grau Colomer, and Carlos Monserrat. "Automated system for periodontal disease diagnosis." In Medical Imaging 1997, edited by Kenneth M. Hanson. SPIE, 1997. http://dx.doi.org/10.1117/12.274099.
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4
Joo, Jaehan, Sinjin Jeong, Heetae Jin, Uhyeon Lee, Ji Young Yoon, and Suk Chan Kim. "Periodontal Disease Detection Using Convolutional Neural Networks." In 2019 International Conference on Artificial Intelligence in Information and Communication (ICAIIC). IEEE, 2019. http://dx.doi.org/10.1109/icaiic.2019.8669021.
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5
Aberin, Shannah Therese A., and Joel C. de Goma. "Detecting Periodontal Disease Using Convolutional Neural Networks." In 2018 IEEE 10th International Conference on Humanoid, Nanotechnology, Information Technology,Communication and Control, Environment and Management (HNICEM). IEEE, 2018. http://dx.doi.org/10.1109/hnicem.2018.8666389.
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Colston, Jr., Bill W., Matthew J. Everett, Luiz B. Da Silva, Linda L. Otis, and Howard Nathel. "Optical coherence tomography for diagnosing periodontal disease." In BiOS '97, Part of Photonics West, edited by Harvey A. Wigdor, John D. B. Featherstone, and Peter Rechmann. SPIE, 1997. http://dx.doi.org/10.1117/12.273591.
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7
Askarian, Behnam, Fatemehsadat Tabei, Grace Anne Tipton, and Jo Woon Chong. "Smartphone-Based Method for Detecting Periodontal Disease." In 2019 IEEE Healthcare Innovations and Point of Care Technologies (HI-POCT). IEEE, 2019. http://dx.doi.org/10.1109/hi-poct45284.2019.8962844.
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Giovani, Elcio M., Gilberto A. Noro-Filho, Bruno V. Caputo, Renato Casarin, Claudio Costa, Daniela Salgado, and Camila C. Santos. "PDT in periodontal disease of HAART resistance patients." In SPIE BiOS, edited by Michael R. Hamblin, James D. Carroll, and Praveen Arany. SPIE, 2016. http://dx.doi.org/10.1117/12.2212804.
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Colston, Jr., Bill W., Dora M. Gutierrez, Matthew J. Everett, Steve B. Brown, Kevin C. Langry, Weldon R. Cox, Paul W. Johnson, and Jeffrey N. Roe. "Intraoral fiber-optic-based diagnostic for periodontal disease." In BiOS 2000 The International Symposium on Biomedical Optics, edited by Tuan Vo-Dinh, Warren S. Grundfest, and David A. Benaron. SPIE, 2000. http://dx.doi.org/10.1117/12.384906.
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Vieru, Rozana D., Agafita Lefter, and Sonia Herman. "Using laser irradiation for the surgical treatment of periodontal disease." In Laser Florence 2001: a Window on the Laser Medicine World, edited by Leonardo Longo, Alfons G. Hofstetter, Mihail-Lucian Pascu, and Wilhelm R. A. Waidelich. SPIE, 2002. http://dx.doi.org/10.1117/12.486621.
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Звіти організацій з теми "Periodontal disease Microbiology":

1
Lattaf, Sara, Lamiaa Abdallaoui, and Amal Bouziane. Effect of periodontal disease on Alzheimer’s disease: protocol of a systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2020. http://dx.doi.org/10.37766/inplasy2020.8.0033.
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2
Yotis, William W. Chemical and Biological Attributes of Selected Periodontopathogens as Potential Indicators of Periodontal Disease. Fort Belvoir, VA: Defense Technical Information Center, November 1992. http://dx.doi.org/10.21236/ada259063.
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3
Kapellas, Kostas. The association between periodontal disease and dementia: a systematic review and meta-analysis. Science Repository, April 2019. http://dx.doi.org/10.31487/j.dobcr.2019.01.005.
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4
Yotis, William W. Chemical and Biological Attributes of Selected Periodontopathogens as Potential Indicators of Periodontal Disease. Fort Belvoir, VA: Defense Technical Information Center, August 1990. http://dx.doi.org/10.21236/ada226312.
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5
Burkes, E. J., Greco Jr., Marbry G. W., Scruggs D. L., Crawford R. R., and J. J. Periodontal Stain Test Diagnosis Program. Fort Belvoir, VA: Defense Technical Information Center, January 1989. http://dx.doi.org/10.21236/ada247284.
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6
Hanker, Jacob S., Beverly L. Giammara, E. J. Burkes, and G. W. Greco. Stain Test Modules for Periodontal Diagnosis. Fort Belvoir, VA: Defense Technical Information Center, October 1989. http://dx.doi.org/10.21236/ada247283.
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7
Spormann, Alfred. The Hopkins Microbiology Course. Office of Scientific and Technical Information (OSTI), July 2019. http://dx.doi.org/10.2172/1544817.
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Dolen, Virginia, Kenneth Bahk, Karen C. Carroll, Keith Klugman, and Nathan A. Ledeboer. Changing Diagnostic Paradigms for Microbiology. Chair Melissa B. Miller. American Society for Microbiology, 2017. http://dx.doi.org/10.1128/aamcol.17-18oct.2016.
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Marsh, Anne S. FAQ: Microbiology of Built Environments. American Society for Microbiology, September 2015. http://dx.doi.org/10.1128/aamcol.sept.2015.
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10
Carlson, Jake. Agronomy / Soil Microbiology - Purdue University. Purdue University Libraries, September 2011. http://dx.doi.org/10.5703/1288284314994.
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