Добірка наукової літератури з теми "Periodontal ligament"

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Статті в журналах з теми "Periodontal ligament":

1
Anneroth, G., K. H. Danielsson, H. Evers, K. G. Hedström, and Å. Nordenram. "Periodontal ligament injection." International Journal of Oral Surgery 14, no. 6 (December 1985): 538–43. http://dx.doi.org/10.1016/s0300-9785(85)80061-2.
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2
Lee, J.-H., B. A. Pryce, R. Schweitzer, M. I. Ryder, and S. P. Ho. "Differentiating zones at periodontal ligament-bone and periodontal ligament-cementum entheses." Journal of Periodontal Research 50, no. 6 (June 2015): 870–80. http://dx.doi.org/10.1111/jre.12281.
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3
Song, In Seok, Yoon Sic Han, Joo-Hee Lee, Soyoun Um, Hui Young Kim, and Byoung Moo Seo. "Periodontal Ligament Stem Cells for Periodontal Regeneration." Current Oral Health Reports 2, no. 4 (August 2015): 236–44. http://dx.doi.org/10.1007/s40496-015-0060-0.
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4
Cho, Moon-Il. "Periodontal Ligament and Cementum." Advances in Dental Research 9, 3_suppl (November 1995): 17. http://dx.doi.org/10.1177/0895937495009003s0801.
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5
Sehar, Dr Kousain. "Periodontal Ligament: Role of Fibroblast in Periodontal Healing." International Journal for Research in Applied Science and Engineering Technology 8, no. 6 (June 2020): 1494–503. http://dx.doi.org/10.22214/ijraset.2020.6244.
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6
Lund, Amy E. "PERIODONTAL LIGAMENT STEM CELLS ISOLATED." Journal of the American Dental Association 135, no. 9 (September 2004): 1236. http://dx.doi.org/10.14219/jada.archive.2004.0393.
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7
Mustafa, Manal, Ahmed Zarrough, Anne Isine Bolstad, Henning Lygre, Kamal Mustafa, Hatice Hasturk, Charles Serhan, Alpdogan Kantarci, and Thomas E. Van Dyke. "Resolvin D1 protects periodontal ligament." American Journal of Physiology-Cell Physiology 305, no. 6 (September 2013): C673—C679. http://dx.doi.org/10.1152/ajpcell.00242.2012.
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Resolution agonists are endogenous mediators that drive inflammation to homeostasis. We earlier demonstrated in vivo activity of resolvins and lipoxins on regenerative periodontal wound healing. The goal of this study was to determine the impact of resolvin D1 (RvD1) on the function of human periodontal ligament (PDL) fibroblasts, which are critical for wound healing during regeneration of the soft and hard tissues around teeth. Primary cells were cultured from biopsies obtained from three individuals free of periodontal diseases. Peripheral blood mononuclear cells were isolated by density gradient centrifugation from whole blood of healthy volunteers. PGE2, leukotriene B4 (LTB4), and lipoxin A4 (LXA4) in culture supernatants were measured by ELISA. The direct impact of RvD1 on PDL fibroblast proliferation was measured and wound closure was analyzed in vitro using a fibroblast culture “scratch assay.” PDL fibroblast function in response to RvD1 was further characterized by basic FGF production by ELISA. IL-1β and TNF-α enhanced the production of PGE2. Treatment of PDL cells and monocytes with 0.1–10 ng/ml RvD1 (0.27–27 M) reduced cytokine induced production of PGE2 and upregulated LXA4 production by both PDL cells and monocytes. RvD1 significantly enhanced PDL fibroblast proliferation and wound closure as well as basic FGF release. The results demonstrate that anti-inflammatory and proresolution actions of RvD1 with upregulation of arachidonic acid-derived endogenous resolution pathways (LXA4) and suggest resolution pathway synergy establishing a novel mechanism for the proresolution activity of the ω-3 docosahexaenoic acid-derived resolution agonist RvD1.
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Cuoghi, Osmar Aparecido, Pedro Marcelo Tondelli, Carlos Alberto Aiello, Marcos Rogério de Mendonça, and Silvano Cesar da Costa. "Importance of periodontal ligament thickness." Brazilian Oral Research 27, no. 1 (February 2013): 76–79. http://dx.doi.org/10.1590/s1806-83242013000100014.
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Kasugai, Shohei. "Characteristics of periodontal ligament and regeneration of periodontal tissue." Ensho Saisei 23, no. 1 (2003): 34–38. http://dx.doi.org/10.2492/jsir.23.34.
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10
Kaneda, T., M. Miyauchi, T. Takekoshi, S. Kitagawa, M. Kitagawa, H. Shiba, H. Kurihara, and T. Takata. "Characteristics of periodontal ligament subpopulations obtained by sequential enzymatic digestion of rat molar periodontal ligament." Bone 38, no. 3 (March 2006): 420–26. http://dx.doi.org/10.1016/j.bone.2005.08.021.
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Дисертації з теми "Periodontal ligament":

1
Winning, Lewis. "The osteogenic potential of periodontal ligament stem cells." Electronic Thesis or Dissertation, Queen's University Belfast, 2018. https://pure.qub.ac.uk/portal/en/theses/the-osteogenic-potential-of-periodontal-ligament-stem-cells(e5fdef0e-d55b-42b6-acb5-75a617b43edd).html.
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2
Miller, Eloise Peyton. "Building a synthetic periodontal ligament: collagen nanostructures on titanium." Text, Case Western Reserve University School of Graduate Studies / OhioLINK, 2006. http://rave.ohiolink.edu/etdc/view?acc_num=case1586422711694101.
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3
Nuersailike, Abuduwali [Verfasser]. "Characterization of Parathyroid Hormone 1 Receptor in Periodontal Ligament Cells / Abuduwali Nuersailike." Online-Ressource, Bonn : Universitäts- und Landesbibliothek Bonn, 2012. http://d-nb.info/1044080973/34.
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4
Wescott, David Clark, and n/a. "Osteogenic gene expression by human periodontal ligament cells under cyclic mechanical tension." University of Otago. School of Dentistry, 2008. http://adt.otago.ac.nz./public/adt-NZDU20081202.131453.
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Background and objectives: The most widely accepted tooth movement model is defined by the pressure-tension hypothesis. An orthodontic force applied to a tooth generates areas of compression and tension in the periodontal ligament (PDL), which are transmitted to the alveolar bone. Areas of tissue exposed to tensile strain undergo bone deposition, whereas areas of tissue exposed to compressive strain undergo bone resorption. We propose that human PDL cells in monolayer culture exposed to tensile mechanical strain would express multiple genes involved in osteogenesis. Materials and Methods: Human PDL cells were isolated and cultured from premolar teeth that were extracted for orthodontic reasons. These cells were plated on control and experimental Uniflex[TM] plates. Using a Flexercell FX4000 strain unit, PDL cells on experimental plates were exposed to a 12% uni-axial cyclic strain for 6 seconds out of every 90 seconds over a 24 hour period. RNA was extracted from the PDL cells at 6 hours, 12 hours and 24 hours. The differential expression of 78 genes implicated in osteoblast differentiation and bone metabolism was analysed using real-time reverse transcriptase polymerase chain reaction (RT-PCR) array technology. Results: Of the 78 genes tested, sixteen genes showed statistically significant (p<0.05) changes in expression in response to the mechanical strain regime. Eight genes were up-regulated (ALPL, BMP2, BMP6, COL2A1, ICAM1, PHEX, SOX9, and VEGFA) and eight genes were down-regulated (ANXA5, BMP4, COL11A1, COL3A1, EGF, ITGB1, MSX and SMAD1). Conclusions: This study has demonstrated that cultured human PDL cells express multiple osteogenic genes under tensile strain, which suggests that PDL cells may have a potential role in osseous remodeling during tooth movement. Key Words: Tooth movement, human PDL cells, tensile mechanical strain, osteogenic genes, real-time RT-PCR array, and Flexercell FX4000.
5
McCormack, Steven William. "Biomechanical function of the periodontal ligament in biting and orthodontic tooth movement." Electronic Thesis or Dissertation, University of Hull, 2016. http://hydra.hull.ac.uk/resources/hull:13630.
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Alveolar bone remodelling is vital for the success of dental implants and orthodontic treatments. However, the underlying biomechanical mechanisms, in particular the function of the periodontal ligament (PDL) in bone remodelling, are not well understood. The PDL is a soft fibrous connective tissue that joins the tooth root to the alveolar bone and plays a critical role in the transmission of loads from the teeth to the surrounding bone. However, due to its complex structure, small size and location within the tooth socket it is difficult to study in vivo. Finite element analysis (FEA) is an ideal tool with which to investigate the role of the PDL, but inclusion of the PDL in FE models is complex and time consuming and most FE models that include teeth do not consider the PDL. The aim of this study was to investigate the effects of including the PDL and its fibrous structure in mandibular finite element models. This research involved the development of a novel method to include the fibres of the PDL in FE models. A simplified single tooth model was developed to assess the effects of modelling fibrous PDL compared to the traditional approach of representing the PDL as a simple layer of solid material and to an absent PDL. The same study design was then applied to a high-resolution model of the human molar region, which is the first time that the fibrous structure of the PDL has been included in a model with realistic tooth and bone geometry. Finally, molar region models of five additional species (cat, cercocebus, pig, rabbit and sheep) were tested with and without a PDL. The results from the research showed that omission of the PDL creates a more rigid model, reducing the strains observed in the mandibular corpus for all six species studied. This suggests that the results obtained are not specific to the human molar region, but may be true for the mammalian mandible in general. Compared to a solid PDL, the fibrous PDL altered the strains in the models, in particular increasing the strains observed in the tooth socket. This may be important for the management of orthodontic treatment, as strains in this region are thought to play an important role in bone remodelling during orthodontic tooth movement.
6
Gaffey, Benjamin James, та n/a. "The effect of intermittent tensile strain on RANKL, OPG, M-CSF and IL-1β expression by periodontal ligament fibroblasts in vitro". University of Otago. School of Dentistry, 2007. http://adt.otago.ac.nz./public/adt-NZDU20071210.143246.
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Mechanical stress has been shown to play a role in bone remodelling during orthodontic tooth movement. Receptor activator of nuclear factor kβ - ligand (RANKL), osteoprotegerin (OPG), monocyte colony stimulating factor (M-CSF) and interleukin 1-β (IL-1β) play key roles in the regulation of bone remodelling, but the role of these cytokines in orthodontic tooth movement is poorly understood. Aim: The aim of this experiment was to examine the response of periodontal ligament (PDL) fibroblasts in monolayer culture to intermittent tensile stress as regards RANKL, OPG, M-CSF and IL-1β production. Methods: Human PDL fibroblasts were dissected from premolars extracted for orthodontic purposes. Explants were seeded out in 1cm wells and grown to confluence in Dulbecco�s modification of Eagle�s medium, containing 10% foetal calf serum and antibiotics, at 37�C in a humidified atmosphere of 5% CO₂/95% air. Upon reaching confluence, the cells were passaged into sequentially larger flasks. Fibroblasts were passaged 6 times. After reaching confluence in T175 flasks, the cells were detached and plated at a cell density of 10⁵/dish in 35mm Bioflex� Plates coated with type 1 collagen. The cells were placed under a continuous uni-axial strain of 12% for 6s of every 90s by a Flexercell FX 4000C[TM] for 0, 12, 24 and 48 hours. Cells were then detached and stored in RNAlater. Quantitative RT-PCR was used to determine the mRNA of the cytokines of interest. Results: Tensile force led to the down regulation of mRNA expression for OPG and IL-1β at 12 and 24 hours respectively, while M-CSF was up-regulated at 6 hours. RANKL was not detected at a significant level for quantification. Conclusion: This osteoclastic-type response indicates the complexity of mechanotransduction in an in vitro setting. Acknowledgments: This research was supported by the New Zealand Dental Research Foundation, the New Zealand Lottery Grants Board and the New Zealand Association of Orthodontists.
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Gaffey, Benjamin James, та n/a. "The effect of intermittent tensile strain on RANKL, OPG, M-CSF and IL-1β expression by periodontal ligament fibroblasts in vitro". University of Otago. School of Dentistry, 2007. http://adt.otago.ac.nz./public/adt-NZDU20071210.143246.
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Mechanical stress has been shown to play a role in bone remodelling during orthodontic tooth movement. Receptor activator of nuclear factor kβ - ligand (RANKL), osteoprotegerin (OPG), monocyte colony stimulating factor (M-CSF) and interleukin 1-β (IL-1β) play key roles in the regulation of bone remodelling, but the role of these cytokines in orthodontic tooth movement is poorly understood. Aim: The aim of this experiment was to examine the response of periodontal ligament (PDL) fibroblasts in monolayer culture to intermittent tensile stress as regards RANKL, OPG, M-CSF and IL-1β production. Methods: Human PDL fibroblasts were dissected from premolars extracted for orthodontic purposes. Explants were seeded out in 1cm wells and grown to confluence in Dulbecco�s modification of Eagle�s medium, containing 10% foetal calf serum and antibiotics, at 37�C in a humidified atmosphere of 5% CO₂/95% air. Upon reaching confluence, the cells were passaged into sequentially larger flasks. Fibroblasts were passaged 6 times. After reaching confluence in T175 flasks, the cells were detached and plated at a cell density of 10⁵/dish in 35mm Bioflex� Plates coated with type 1 collagen. The cells were placed under a continuous uni-axial strain of 12% for 6s of every 90s by a Flexercell FX 4000C[TM] for 0, 12, 24 and 48 hours. Cells were then detached and stored in RNAlater. Quantitative RT-PCR was used to determine the mRNA of the cytokines of interest. Results: Tensile force led to the down regulation of mRNA expression for OPG and IL-1β at 12 and 24 hours respectively, while M-CSF was up-regulated at 6 hours. RANKL was not detected at a significant level for quantification. Conclusion: This osteoclastic-type response indicates the complexity of mechanotransduction in an in vitro setting. Acknowledgments: This research was supported by the New Zealand Dental Research Foundation, the New Zealand Lottery Grants Board and the New Zealand Association of Orthodontists.
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Silva, Guilherme Ferreira da [UNESP]. "Reação histológica do periodonto, subjacente à região de furca perfurada e preenchida com diferentes materiais, em molares de ratos." PublishedVersion, Universidade Estadual Paulista (UNESP), 2006. http://hdl.handle.net/11449/90414.
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O sucesso do tratamento de dentes com perfurações de furca está diretamente relacionado ao tipo de material utilizado no preenchimento destas perfurações. Um material selador ideal deve possuir boas propriedades físicas, químicas e biológicas, sendo, o Mineral Trióxido Agregado (MTA) um dos materiais indicados. Assim, o objetivo desse estudo foi avaliar a resposta biológica de três materiais seladores: MTA-Angelus branco® (MTA), Endo-C.P.M.-Sealer® (CPM) e cimento de óxido de zinco e eugenol (OZE). Para isso, foram utilizados os primeiros molares superiores de 60 ratos; o primeiro molar superior do lado esquerdo hígido foi utilizado como controle e o do lado direito foi realizada a perfuração no assoalho da câmara pulpar com uma fresa nº ¼. Os materiais foram manipulados e inseridos na perfuração e a cavidade de acesso foi selada com ionômero de vidro. Decorrido os períodos de 07, 15, 30 e 60 dias, os animais foram sacrificados e os fragmentos das maxilas removidos e fixados em formaldeído. Após a descalcificação em EDTA, os fragmentos de maxila foram desidratados, diafanizados e incluídos em parafina. Os cortes foram corados com hematoxilina & eosina (H&E), tricrômico de Masson ou submetidos ao método do TRAP (“Tartrate Resistant Acid Phosphatase”) para realização das análises morfológica e morfométrica. A análise morfométrica foi realizada sob três parâmetros: espessura do ligamento periodontal (LP), número de células inflamatórias/mm2 (CI) e número de osteoclastos TRAP-positivos. Os resultados morfométricos revelaram aumento significante (p_0,05) no espaço do LP,em todos os períodos, em relação ao controle. Entretanto, aos 60 dias, este espaço foi menor em todos os grupos experimentais; o OZE foi o que provocou maior espessamento do LP, não havendo diferenças significantes...
The therapy success in the treatment of root perforations depends on the material used to fill them. The best sealing material should present good physical, chemical and biological properties. More recently, the mineral trioxide aggregate (MTA) has been indicated for sealing of root perforations. Thus, the aim of the present study was to evaluate the biological response of three sealing materials: White MTA-Angelus (MTA), Endo- C.P.M.-Sealer (CPM) and zinc-oxide and eugenol cement (OZE). Sixty adult rats weighing 250g were divided into three groups: MTA, CPM and OZE. Furcal perforations were performed in maxillary first molars with a round bur no. ¼; the contra-lateral maxillary first molars – without perforations - were used as control. The perforations were carried out and the materials were placed into these defects; access cavities were sealed with glass ionomer. After 07, 15, 30 and 60 days, the animals were killed, and fragments of maxilla containing first molars were removed and immediately immersed in 4% formaldehyde. After decalcification in EDTA, the specimens were processed and embedded in paraffin. Sections were stained with hematoxylin-eosin, Masson’s trichrome and submitted to TRAP (Tartrate Resistant Acid Phosphatase) reaction. The morphometric analysis, in the furcation region of the periodontal ligament, was performed considering three parameters: thickness of periodontal ligament (PL), number of inflammatory cells (IC) and number of TRAP-positive osteoclasts in the alveolar bone surface. The experimental groups showed, in all periods, significant increase (p_0.05) in the PL thickness in comparison to control group; this thickness was more accentuated in the OZE group. The periodontal space in the CPM group was less thick in all experimental periods in comparison to MTA and OZE groups, except in the period of 60 days; in this period, statistical differences in the periodontal space were not detected between CPM and MTA groups.
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Sousa, Hugo Alexandre de [UNESP]. "Avaliação microscópica do efeito de diferentes meios de conservação sobre o ligamento periodontal de dentes humanos extraídos cirurgicamente." PublishedVersion, Universidade Estadual Paulista (UNESP), 2004. http://hdl.handle.net/11449/101071.
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Pedrini, Denise. "Análise do conhecimento dos cirurgiões dentistas sobre plano de tratamento das injúrias do ligamento periodontal após traumatismo dentoalveolar /." Text, Araçatuba, 2008. http://hdl.handle.net/11449/106717.
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Resumo: O diagnóstico preciso e a elaboração de um plano de tratamento adequado podem constituir uma tarefa bastante complexa, especialmente nos traumatismos dentoalveolares, pois necessitam de uma abordagem multidisciplinar e conhecimento sobre o processo de reparo após o traumatismo. O objetivo do trabalho foi analisar o conhecimento dos cirurgiões dentistas sobre plano de tratamento das injúrias do ligamento periodontal após traumatismo dentoalveolar. Para tanto, a partir de um questionário, foram abordadas perguntas referentes ao perfil dos profissionais entrevistados e conduta frente às injúrias do ligamento periodontal (concussão, subluxação, luxação extrusiva, luxação lateral e luxação intrusiva) ocasionadas por traumatismo dentoalveolar. Seiscentos e noventa e três cirurgiões dentistas que participaram da 23ª Reunião Anual da SBPqO (2006) responderam o questionário e os dados obtidos foram submetidos à análise descritiva, enquanto o teste estatístico foi aplicado para demonstrar as freqüências e o nível de significância entre as variáveis (Teste qui-quadrado ou Teste Exato de Fisher). De acordo com os resultados obtidos, grandes dificuldades foram encontradas com relação ao plano de tratamento das luxações extrusiva, lateral e intrusiva. De maneira geral, a especialidade não influenciou na elaboração de planos adequados para as injúrias mais complexas. Foi possível concluir que os cirurgiões dentistas não apresentam conhecimento suficiente para tratar de maneira adequada as injúrias mais severas do ligamento periodontal após traumatismo dentoalveolar
Abstract: An accurate diagnosis and the establishment of an adequate treatment plan may constitute quite a complex task, particularly in cases of dentoalveolar trauma, which require a multidisciplinary approach and knowledge of the repair process in tooth injuries. The aim of this study was to analyze the dentists' knowledge of the treatment plan for periodontal ligament injuries after dentoalveolar trauma. For such purpose, a questionnaire was prepared with questions arguing about the profile of the interviewed professionals and their conduct facing periodontal ligament injuries (concussion, subluxation, extrusive luxation, lateral luxation and intrusive luxation) secondary to dentoalveolar trauma. Six hundred and ninety three dentists attending the 23rd Annual Meeting of the SBPqO (2006) filled out the questionnaire and the obtained data were subjected to descriptive analysis. Either chi-square test or Fisher's exact test was applied to determine the frequencies and the significance level among the variables. The results revealed great difficulties in establishing a treatment plan for extrusive, lateral and intrusive luxations. In general, the dental specialty of the participants did not influence the elaboration of adequate treatment plans for the most severe injuries. It could be concluded that dentists do not have sufficient knowledge to treat properly the most severe types of periodontal ligament injuries following a dentoalveolar trauma

Книги з теми "Periodontal ligament":

1
Loescher, Alison Ruth. Studies on the innervation of the periodontal ligament. Birmingham: Universityof Birmingham, 1989.
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2
Nguyen, Lan Phan. A critique of the paper "Collagen implants do not preserve periodontal ligament homeostasis in periodontal wounds". [Toronto: University of Toronto, Faculty of Dentistry], 1996.
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3
Lin, Deborah G. Storage conditions of avulsed teeth affect the phenotype of cultured human periodontal ligament cells. [Toronto: Faculty of Dentistry, University of Toronto, 1999.
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4
Whiteside, Leo A. Ligament Balancing. Berlin, Heidelberg: Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-642-18689-9.
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5
Camus, Emmanuel, and Luc Van Overstraeten, eds. Carpal Ligament Surgery. Paris: Springer Paris, 2013. http://dx.doi.org/10.1007/978-2-8178-0379-1.
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6
Rossi, Roberto, and Fabrizio Margheritini, eds. Knee Ligament Injuries. Milano: Springer Milan, 2014. http://dx.doi.org/10.1007/978-88-470-5513-1.
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7
Kinane, Denis F. Periodontal disease. Basel: Karger, 2012.
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Pattison, Anna Matsuishi. Periodontal instrumentation. 2nd ed. Norwalk, Conn: Appleton & Lange, 1991.
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Nagano, Keiji, and Yoshiaki Hasegawa, eds. Periodontal Pathogens. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-0939-2.
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10
Fanelli, MD, Gregory C., ed. Posterior Cruciate Ligament Injuries. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-12072-0.
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Частини книг з теми "Periodontal ligament":

1
Menicanin, Danijela, K. Hynes, J. Han, S. Gronthos, and P. M. Bartold. "Cementum and Periodontal Ligament Regeneration." In Engineering Mineralized and Load Bearing Tissues, 207–36. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-22345-2_12.
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2
Komaki, Motohiro. "Pericytes in the Periodontal Ligament." In Advances in Experimental Medicine and Biology, 169–86. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11093-2_10.
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3
Mark Bartold, P. "Bone and Tooth Interface: Periodontal Ligament." In Mineralized Tissues in Oral and Craniofacial Science, 217–30. West Sussex, UK: John Wiley & Sons, Inc.,, 2013. http://dx.doi.org/10.1002/9781118704868.ch26.
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4
Trubiani, Oriana, and Francesca Diomede. "Xeno-Free Culture of Human Periodontal Ligament Stem Cells." In Methods in Molecular Biology, 87–92. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/7651_2014_122.
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Elçin, Y. Murat, Bülend İnanç, and A. Eser Elçin. "Human Embryonic Stem Cell Differentiation on Periodontal Ligament Fibroblasts." In Methods in Molecular Biology, 269–81. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60761-369-5_14.
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Wilson, A. N., J. Middleton, G. N. Pande, and M. L. Jones. "Nonlinear Behaviour of the Periodontal Ligament a Numerical Approach." In Interfaces in Medicine and Mechanics—2, 309–18. Dordrecht: Springer Netherlands, 1991. http://dx.doi.org/10.1007/978-94-011-3852-9_32.
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Elçin, Y. Murat, Bülend İnanç, and A. Eser Elçin. "Differentiation of Human Embryonic Stem Cells on Periodontal Ligament Fibroblasts." In Methods in Molecular Biology, 223–35. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/7651_2014_130.
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Issaranggun Na Ayuthaya, Benjar, and Prasit Pavasant. "Influence of Exogenous IL-12 on Human Periodontal Ligament Cells." In Interface Oral Health Science 2016, 217–28. Singapore: Springer Singapore, 2016. http://dx.doi.org/10.1007/978-981-10-1560-1_18.
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9
Farag, Amro, Cédryck Vaquette, Dietmar W. Hutmacher, P. Mark Bartold, and Saso Ivanovski. "Fabrication and Characterization of Decellularized Periodontal Ligament Cell Sheet Constructs." In Methods in Molecular Biology, 403–12. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-6685-1_23.
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Saito, Masahiro. "Trends in Periodontal Regeneration Therapy: Potential Therapeutic Strategy of Extracellular Matrix Administration for Periodontal Ligament Regeneration." In Interface Oral Health Science 2014, 169–80. Tokyo: Springer Japan, 2015. http://dx.doi.org/10.1007/978-4-431-55192-8_14.
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Тези доповідей конференцій з теми "Periodontal ligament":

1
Maurtgi, Moustafa, Sharlina binti Mohamad, Siti Noor Fazliah Mohd Noor, and Norehan Mokhtar. "Effect of vitamin E on human periodontal ligament fibroblasts." In TRANSLATIONAL CRANIOFACIAL CONFERENCE 2016 (TCC 2016): Proceedings of the 1st Translational Craniofacial Conference 2016. Author(s), 2016. http://dx.doi.org/10.1063/1.4968874.
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2
Karam, Leandro Z., Maura S. Milczewski, and Hypolito J. Kalinowski. "Strain monitoring of the periodontal ligament in pig's mandibles." In OFS2012 22nd International Conference on Optical Fiber Sensor, edited by Yanbiao Liao, Wei Jin, David D. Sampson, Ryozo Yamauchi, Youngjoo Chung, Kentaro Nakamura, and Yunjiang Rao. SPIE, 2012. http://dx.doi.org/10.1117/12.975270.
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3
Kafel, N., J. Kolodziejski, M. Niemiera, and M. Reed. "Separation of the periodontal ligament for atraumatic tooth extraction." In 2014 40th Annual Northeast Bioengineering Conference (NEBEC). IEEE, 2014. http://dx.doi.org/10.1109/nebec.2014.6972830.
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4
Gao, Zhen, and Xiaoting Luo. "Biological Effect of Titanium's Surface Roughness on Periodontal Ligament Cells." In 2009 3rd International Conference on Bioinformatics and Biomedical Engineering (iCBBE). IEEE, 2009. http://dx.doi.org/10.1109/icbbe.2009.5162449.
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5
Menegotto, G. F., L. Grabarski, H. J. Kalinowski, and J. A. Simões. "Trial analysis of swine's periodontal ligament with Bragg grating sensors." In 20th International Conference on Optical Fibre Sensors, edited by Julian D. C. Jones. SPIE, 2009. http://dx.doi.org/10.1117/12.835231.
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6
Yang, Yu, Wencheng Tang, and Yao jun Wang. "Experimental Analysis of the Elastic Modulus of Periodontal Ligament in Nanoindentation." In ASME 2016 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. American Society of Mechanical Engineers, 2016. http://dx.doi.org/10.1115/detc2016-59040.
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Анотація:
The periodontal ligament (PDL) is a soft connective tissue which exhibits an inhomogeneous, nonlinear, and anisotropic material properties. and the elastic modulus of different positions on each section are not the same, analysis of the material properties of PDL enables a better understanding of biomechanical features for tooth movement. The aim of this study was to study the elastic modulus of different section of PDL in nanoindentation. Experimental results indicate that the average elastic modulus elastic modulus in midroot are lower than cervical margin and apex, and there is large change in the circumferential regions.
7
Rahimi, Masoud, and Afsaneh Mojra. "Numerical modeling of hyperfoam behavior of periodontal ligament in mechanical loading." In 2019 26th National and 4th International Iranian Conference on Biomedical Engineering (ICBME). IEEE, 2019. http://dx.doi.org/10.1109/icbme49163.2019.9030376.
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8
Zhurov, Alexei I., Sam L. Evans, Catherine A. Holt, and John Middleton. "A Nonlinear Compressible Transversely-Isotropic Viscohyperelastic Constitutive Model of the Periodontal Ligament." In ASME 2008 International Mechanical Engineering Congress and Exposition. ASMEDC, 2008. http://dx.doi.org/10.1115/imece2008-67949.
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Анотація:
The periodontal ligament may be treated as a transversely-isotropic viscohyperelastic fibre-reinforced compressible material which is subject to large deformations and has an essentially nonlinear behavior. Within these assumptions, a continuum constitutive model of the PDL was proposed recently [48], which involves a number of material parameters that have to be identified from experimental data. An optical motion analysis system was developed [26] to collect data on the deformation of the PDL. In the present paper, an advanced version of the model is suggested, which is based on the assumption of the existence of an additive strain-energy function dependent on a number of principal invariants. The sensitivity analysis of the material parameters is performed and a parameter identification technique is suggested.
9
Si-Eun Kim, Soo-Hyuk Uhm, Doo-Hoon Song, Chong-Kwan Kim, Kwang-Mahn Kim, Kyoung-Nam Kim, and Jeon-Geon Han. "Enhanced funtion of human periodontal ligament cells cultured on nanoporous titanium surfaces." In 2012 IEEE 39th International Conference on Plasma Sciences (ICOPS). IEEE, 2012. http://dx.doi.org/10.1109/plasma.2012.6383859.
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10
Wu, Bin, Wencheng Tang, and Bin Yan. "A Novel Finite Element Modeling Method for Periodontal Ligament of Impacted Maxillary Tooth." In 2009 3rd International Conference on Bioinformatics and Biomedical Engineering (iCBBE 2009). IEEE, 2009. http://dx.doi.org/10.1109/icbbe.2009.5162218.
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Звіти організацій з теми "Periodontal ligament":

1
Taylor, Dean C., and Richard C. Mather III. Anterior Cruciate Ligament (ACL) Reconstruction. Touch Surgery Simulations, May 2014. http://dx.doi.org/10.18556/touchsurgery/2014.s0022.
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2
Burkes, E. J., Greco Jr., Marbry G. W., Scruggs D. L., Crawford R. R., and J. J. Periodontal Stain Test Diagnosis Program. Fort Belvoir, VA: Defense Technical Information Center, January 1989. http://dx.doi.org/10.21236/ada247284.
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3
McDevitt, Cahir A. Acceleration of Ligament Healing with Cellular Attractants. Fort Belvoir, VA: Defense Technical Information Center, July 2008. http://dx.doi.org/10.21236/ada572654.
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4
Hanker, Jacob S., Beverly L. Giammara, E. J. Burkes, and G. W. Greco. Stain Test Modules for Periodontal Diagnosis. Fort Belvoir, VA: Defense Technical Information Center, October 1989. http://dx.doi.org/10.21236/ada247283.
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5
Andrews, Malcolm, and Dean Preston. TAB Models for Liquid Sheet and Ligament Breakup. Office of Scientific and Technical Information (OSTI), September 2014. http://dx.doi.org/10.2172/1154967.
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6
Yamamoto, Sota, Akinori Saito, Akinori Ishikawa, Koji Mizuno, and Eiichi Tanaka. Effects of Loading Direction on Antierior Cruciate Ligament Injury. Warrendale, PA: SAE International, May 2005. http://dx.doi.org/10.4271/2005-08-0067.
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7
Hutton, Stephen B. Preoperative Use of lntranasal Ketorolac Tromethamine (Sprix) in Periodontal Flap Surgery. Fort Belvoir, VA: Defense Technical Information Center, May 2015. http://dx.doi.org/10.21236/ad1012707.
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8
Lattaf, Sara, Lamiaa Abdallaoui, and Amal Bouziane. Effect of periodontal disease on Alzheimer’s disease: protocol of a systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2020. http://dx.doi.org/10.37766/inplasy2020.8.0033.
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9
Yotis, William W. Chemical and Biological Attributes of Selected Periodontopathogens as Potential Indicators of Periodontal Disease. Fort Belvoir, VA: Defense Technical Information Center, November 1992. http://dx.doi.org/10.21236/ada259063.
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10
Kapellas, Kostas. The association between periodontal disease and dementia: a systematic review and meta-analysis. Science Repository, April 2019. http://dx.doi.org/10.31487/j.dobcr.2019.01.005.
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