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Статті в журналах з теми "Phagocytic cell"

Автор: Grafiati

Опубліковано: 11 березня 2023

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1

Bondarenko, L. "The cell protection of weated pigs for probiotics." Tehnologìâ virobnictva ì pererobki produktìv tvarinnictva, no. 2(158) (November 24, 2020): 111–19. http://dx.doi.org/10.33245/2310-9289-2020-158-2-111-119.

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The immune system is central to ensuring the consistency of the body's homeostasis. The state of the body's natural resistance is determined by a set of non-specific protective mechanisms. Lymphocytes and phagocytes are actively involved in maintaining immunity. Lymphocytes recognize the antigens of pathogenic microorganisms, and phagocytes absorb and destroy the pathogens themselves. During the weaning of piglets from sows there is a decrease in the protective forces of their body. During this period, the natural resistance of the piglets is reduced due to the stressful situation caused by ch

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2

Jiang, Shuai, Zhihao Jia, Tao Zhang, et al. "Functional characterisation of phagocytes in the Pacific oyster Crassostrea gigas." PeerJ 4 (December 14, 2016): e2590. http://dx.doi.org/10.7717/peerj.2590.

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Invertebrates lack canonical adaptive immunity and mainly rely on innate immune system to fight against pathogens. The phagocytes, which could engulf and kill microbial pathogens, are likely to be of great importance and have to undertake significant roles in invertebrate immune defense. In the present study, flow cytometry combined with histological and lectin staining was employed to characterise functional features of phagocytes in the Pacific oyster Crassostrea gigas. Based on the cell size and cellular contents, haemocytes were categorised into three cell types, i.e., granulocytes, semigr

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3

QUIE, P. "Phagocytic cell dysfunction." Journal of Allergy and Clinical Immunology 77, no. 3 (1986): 387–98. http://dx.doi.org/10.1016/0091-6749(86)90169-7.

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4

Tussupbekova, Gulmira, Anar Rakhmetova, Gulnaziya Alshynbekova, et al. "Influence of Industrial Factors on Cytomorphological Indicators of Phagocytic Cells." Open Access Macedonian Journal of Medical Sciences 10, A (2022): 1207–10. http://dx.doi.org/10.3889/oamjms.2022.5939.

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AIM: The article has researched cell morphological changes of phagocytic system’s cell of experimented animals by influence of cool-rock dust phisical loading basis. METHODS: The phagocytic activity in combined operation of destructive changes in the cells and the lowering of vital capacity was investigated. RESULTS: The taken results let cosider the structure-functional condition of the phagocytal system’s cell in operation of unfavorable and industrial factors. CONCLUSION: Experimental animals with intratracheal administration of CRD have pronounced changes in phagocytic ac

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5

Lovewell, Rustin R., Sandra M. Hayes, George A. O'Toole, and Brent Berwin. "Pseudomonas aeruginosaflagellar motility activates the phagocyte PI3K/Akt pathway to induce phagocytic engulfment." American Journal of Physiology-Lung Cellular and Molecular Physiology 306, no. 7 (2014): L698—L707. http://dx.doi.org/10.1152/ajplung.00319.2013.

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Phagocytosis of the bacterial pathogen Pseudomonas aeruginosa is the primary means by which the host controls bacterially induced pneumonia during lung infection. Previous studies have identified flagellar swimming motility as a key pathogen-associated molecular pattern (PAMP) recognized by phagocytes to initiate engulfment. Correspondingly, loss of flagellar motility is observed during chronic pulmonary infection with P. aeruginosa, and this likely reflects a selection for bacteria resistant to phagocytic clearance. However, the mechanism underlying the preferential phagocytic response to mot

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6

Holzer, T. J., L. Kizlaitis, M. Vachula, C. W. Weaver, and B. R. Andersen. "Human phagocytic cell responses to Mycobacterium leprae and Mycobacterium bovis Bacillus Calmette-Guérin. An in vitro comparison of leprosy vaccine components." Journal of Immunology 141, no. 5 (1988): 1701–8. http://dx.doi.org/10.4049/jimmunol.141.5.1701.

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Abstract Components of current vaccines for Hansen's disease include Mycobacterium bovis Bacillus Calmette-Guérin (BCG) and killed Mycobacterium leprae. BCG infections in humans are rare and most often occur in immune-compromised individuals. M. leprae on the other hand, although not causing clinical disease in most exposed individuals, is capable of infecting and replicating within mononuclear phagocytes. Lymphocytes from patients with the lepromatous form of Hansen's disease exhibit defective lymphokine production when challenged in vitro with M. leprae. This may result in inefficient mononu

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7

Snipes, RG, KW Lam, RC Dodd, TK Gray, and MS Cohen. "Acid phosphatase activity in mononuclear phagocytes and the U937 cell line: monocyte-derived macrophages express tartrate-resistant acid phosphatase." Blood 67, no. 3 (1986): 729–34. http://dx.doi.org/10.1182/blood.v67.3.729.729.

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Abstract Tartrate-resistant acid phosphatase (TRAcP) is used as a marker for osteoclasts, which are believed to be derived from phagocytic cells or phagocyte stem cell precursors. To further investigate the relationship between monocytic phagocytes and osteoclasts, acid phosphatase (AcP) activity was measured by three different techniques in human peripheral blood monocytes, monocyte-derived macrophages, and the U937 cell line. We found that cytochemistry and gel electrophoresis led to similar results, but that the colorimetric assay was inconsistent. Normal human peripheral monocytes expresse

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8

Snipes, RG, KW Lam, RC Dodd, TK Gray, and MS Cohen. "Acid phosphatase activity in mononuclear phagocytes and the U937 cell line: monocyte-derived macrophages express tartrate-resistant acid phosphatase." Blood 67, no. 3 (1986): 729–34. http://dx.doi.org/10.1182/blood.v67.3.729.bloodjournal673729.

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Tartrate-resistant acid phosphatase (TRAcP) is used as a marker for osteoclasts, which are believed to be derived from phagocytic cells or phagocyte stem cell precursors. To further investigate the relationship between monocytic phagocytes and osteoclasts, acid phosphatase (AcP) activity was measured by three different techniques in human peripheral blood monocytes, monocyte-derived macrophages, and the U937 cell line. We found that cytochemistry and gel electrophoresis led to similar results, but that the colorimetric assay was inconsistent. Normal human peripheral monocytes expressed both ta

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9

Wilson, M. E., R. J. Genco, and R. Snyderman. "The Phagocytic Cell: Summary." Clinical Infectious Diseases 7, no. 3 (1985): 387–89. http://dx.doi.org/10.1093/clinids/7.3.387.

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10

Verhoef, J., and F. A. Waldvogel. "Testing phagocytic cell function." European Journal of Clinical Microbiology 4, no. 4 (1985): 379–81. http://dx.doi.org/10.1007/bf02148686.

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11

Guo, Feiye, Ying Ding, Nora Caberoy, et al. "ABCF1 extrinsically regulates retinal pigment epithelial cell phagocytosis." Molecular Biology of the Cell 26, no. 12 (2015): 2311–20. http://dx.doi.org/10.1091/mbc.e14-09-1343.

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Phagocytosis of shed photoreceptor outer segments (POSs) by retinal pigment epithelial (RPE) cells is critical to retinal homeostasis and shares many conserved signaling pathways with other phagocytes, including extrinsic regulations. Phagocytotic ligands are the key to cargo recognition, engulfment initiation, and activity regulation. In this study, we identified intracellular protein ATP-binding cassette subfamily F member 1 (ABCF1) as a novel RPE phagocytotic ligand by a new approach of functional screening. ABCF1 was independently verified to extrinsically promote phagocytosis of shed POSs

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12

Ren, Y., R. L. Silverstein, J. Allen, and J. Savill. "CD36 gene transfer confers capacity for phagocytosis of cells undergoing apoptosis." Journal of Experimental Medicine 181, no. 5 (1995): 1857–62. http://dx.doi.org/10.1084/jem.181.5.1857.

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Phagocyte recognition and ingestion of intact cells undergoing apoptosis are key events in this generally important program of cell death. Insufficient phagocyte capacity for apoptotic cells can result in failure to clear dying cells before membrane integrity is lost, resulting in leakage of noxious cell contents and severe tissue damage. However, no means has been available to increase phagocytic clearance of apoptotic cells. We now report that transfection of the macrophage adhesion molecule CD36 into human Bowes melanoma cells specifically conferred greatly increased capacity to ingest apop

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13

Patil, Mallikarjun, Sherin Saheera, Praveen K. Dubey, et al. "Novel Mechanisms of Exosome-Mediated Phagocytosis of Dead Cells in Injured Heart." Circulation Research 129, no. 11 (2021): 1006–20. http://dx.doi.org/10.1161/circresaha.120.317900.

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Rationale: After myocardial ischemic injury, improper phagocytic clearance of dying cardiac cells and the ensuing lack of inflammation resolution results in adverse cardiac remodeling and dysfunction that might lead to heart failure. Therefore, therapeutic strategies to ameliorate immune cell phagocytic function are critical for augmenting cardiac repair after injury. Objective: To determine whether mesenchymal stem cell–derived exosomes (MSC-Exos) act as opsonin for apoptotic cells or trigger eat-me phagocytic signaling in resident/recruited phagocytes after myocardial ischemic injury. Method

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14

Brill-Karniely, Yifat, Dvir Dror, Tal Duanis-Assaf, et al. "Triangular correlation (TrC) between cancer aggressiveness, cell uptake capability, and cell deformability." Science Advances 6, no. 3 (2020): eaax2861. http://dx.doi.org/10.1126/sciadv.aax2861.

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The malignancy potential is correlated with the mechanical deformability of the cancer cells. However, mechanical tests for clinical applications are limited. We present here a Triangular Correlation (TrC) between cell deformability, phagocytic capacity, and cancer aggressiveness, suggesting that phagocytic measurements can be a mechanical surrogate marker of malignancy. The TrC was proved in human prostate cancer cells with different malignancy potential, and in human bladder cancer and melanoma cells that were sorted into subpopulations based solely on their phagocytic capacity. The more pha

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15

Fibbe, WE, J. van Damme, A. Billiau, et al. "Interleukin-1 (22-K factor) induces release of granulocyte-macrophage colony-stimulating activity from human mononuclear phagocytes." Blood 68, no. 6 (1986): 1316–21. http://dx.doi.org/10.1182/blood.v68.6.1316.1316.

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Abstract An electrophoretically pure preparation of natural human interleukin-1 (IL-1) was shown to stimulate in vitro colony formation in human bone marrow cultures. Day 4 myeloid cluster-forming cells (CFC), as well as early (day 7) and late (day 10) granulocyte-macrophage colony-forming units (CFU-GM) were stimulated in a dose-dependent fashion. At optimal concentrations of IL-1, the number of day 4 CFC reached 72%, the number of day 7 CFU-GM reached 32%, and the number of day 10 CFU-GM reached 80% of the respective numbers of colonies obtained by addition of crude leukocyte-conditioned med

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16

Fibbe, WE, J. van Damme, A. Billiau, et al. "Interleukin-1 (22-K factor) induces release of granulocyte-macrophage colony-stimulating activity from human mononuclear phagocytes." Blood 68, no. 6 (1986): 1316–21. http://dx.doi.org/10.1182/blood.v68.6.1316.bloodjournal6861316.

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An electrophoretically pure preparation of natural human interleukin-1 (IL-1) was shown to stimulate in vitro colony formation in human bone marrow cultures. Day 4 myeloid cluster-forming cells (CFC), as well as early (day 7) and late (day 10) granulocyte-macrophage colony-forming units (CFU-GM) were stimulated in a dose-dependent fashion. At optimal concentrations of IL-1, the number of day 4 CFC reached 72%, the number of day 7 CFU-GM reached 32%, and the number of day 10 CFU-GM reached 80% of the respective numbers of colonies obtained by addition of crude leukocyte-conditioned medium (LCM)

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17

du Preez, Kelly, Yolandi Rautenbach, Emma H. Hooijberg, and Amelia Goddard. "Oxidative burst and phagocytic activity of phagocytes in canine parvoviral enteritis." Journal of Veterinary Diagnostic Investigation 33, no. 5 (2021): 884–93. http://dx.doi.org/10.1177/10406387211025513.

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Canine parvoviral enteritis (CPE) is a severe disease characterized by systemic inflammation and immunosuppression. The function of circulating phagocytes (neutrophils and monocytes) in affected dogs has not been fully investigated. We characterized the functional capacity of canine phagocytes in CPE by determining their oxidative burst and phagocytic activities using flow cytometry. Blood was collected from 28 dogs with CPE and 11 healthy, age-matched, control dogs. Oxidative burst activity was assessed by stimulating phagocytes with opsonized Escherichia coli or phorbol 12-myristate 13-aceta

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18

Alva-Murillo, Nayeli, Joel Edmundo López-Meza, and Alejandra Ochoa-Zarzosa. "Nonprofessional Phagocytic Cell Receptors Involved inStaphylococcus aureusInternalization." BioMed Research International 2014 (2014): 1–9. http://dx.doi.org/10.1155/2014/538546.

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Staphylococcus aureusis a successful human and animal pathogen. The majority of infections caused by this pathogen are life threatening, primarily becauseS. aureushas developed multiple evasion strategies, possesses intracellular persistence for long periods, and targets the skin and soft tissues. Therefore, it is very important to understand the mechanisms employed byS. aureusto colonize and proliferate in these cells. The aim of this review is to describe the recent discoveries concerning the host receptors of nonprofessional phagocytes involved inS. aureusinternalization. Most of the knowle

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19

Winstel, Volker, Dominique Missiakas, and Olaf Schneewind. "Staphylococcus aureustargets the purine salvage pathway to kill phagocytes." Proceedings of the National Academy of Sciences 115, no. 26 (2018): 6846–51. http://dx.doi.org/10.1073/pnas.1805622115.

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Staphylococcus aureuscolonizes large segments of the human population and causes invasive infections due to its ability to escape phagocytic clearance. During infection, staphylococcal nuclease and adenosine synthase A convert neutrophil extracellular traps to deoxyadenosine (dAdo), which kills phagocytes. The mechanism whereby staphylococcal dAdo intoxicates phagocytes is not known. Here we used CRISPR-Cas9 mutagenesis to show that phagocyte intoxication involves uptake of dAdo via the human equilibrative nucleoside transporter 1, dAdo conversion to dAMP by deoxycytidine kinase and adenosine

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20

O'Keeffe, Kate M., Mieszko M. Wilk, John M. Leech, et al. "Manipulation of Autophagy in Phagocytes Facilitates Staphylococcus aureus Bloodstream Infection." Infection and Immunity 83, no. 9 (2015): 3445–57. http://dx.doi.org/10.1128/iai.00358-15.

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The capacity for intracellular survival within phagocytes is likely a critical factor facilitating the dissemination ofStaphylococcus aureusin the host. To date, the majority of work onS. aureus-phagocyte interactions has focused on neutrophils and, to a lesser extent, macrophages, yet we understand little about the role played by dendritic cells (DCs) in the direct killing of this bacterium. Using bone marrow-derived DCs (BMDCs), we demonstrate for the first time that DCs can effectively killS. aureusbut that certain strains ofS. aureushave the capacity to evade DC (and macrophage) killing by

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21

Selsted, Michael E., and Andre J. Ouellette. "Defensins in granules of phagocytic and non-phagocytic cells." Trends in Cell Biology 5, no. 3 (1995): 114–19. http://dx.doi.org/10.1016/s0962-8924(00)88961-8.

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22

Wang, Huizhen, Haikun Wang, Weipeng Xiong, et al. "Evaluation on the phagocytosis of apoptotic spermatogenic cells by Sertoli cells in vitro through detecting lipid droplet formation by Oil Red O staining." Reproduction 132, no. 3 (2006): 485–92. http://dx.doi.org/10.1530/rep.1.01213.

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During spermatogenesis, more than half of the differentiating spermatogenic cells undergo apoptosis before they mature into spermatozoa. Ultrastructure studies showed that the formation of lipid droplets in Sertoli cells was associated with phagocytosis of residual bodies and apoptotic germ cells by Sertoli cells. Here, a relationship between the phagocytosis of apoptotic spermatogenic cells and lipid droplet formation in Sertoli cells was studiedin vitroby Oil Red O (ORO) staining. The results confirmed that the formation of lipid droplets was a result of phagocytosis of apoptotic spermatogen

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23

Robertson, Maura D., Anthony Seaton, and J. A. Raeburn. "Phagocytic cell responses toAspergillus fumigatus." FEMS Microbiology Letters 47, no. 5 (1989): 305–6. http://dx.doi.org/10.1111/j.1574-6968.1989.tb02401.x.

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24

Henricks, P. A. J., J. Verhoef, and F. P. Nijkamp. "Modulation of phagocytic cell function." Veterinary Research Communications 10, no. 1 (1986): 165–88. http://dx.doi.org/10.1007/bf02213979.

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25

Vomund, Anthony N., Bernd H. Zinselmeyer, Jing Hughes, et al. "Beta cells transfer vesicles containing insulin to phagocytes for presentation to T cells." Proceedings of the National Academy of Sciences 112, no. 40 (2015): E5496—E5502. http://dx.doi.org/10.1073/pnas.1515954112.

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Beta cells from nondiabetic mice transfer secretory vesicles to phagocytic cells. The passage was shown in culture studies where the transfer was probed with CD4 T cells reactive to insulin peptides. Two sets of vesicles were transferred, one containing insulin and another containing catabolites of insulin. The passage required live beta cells in a close cell contact interaction with the phagocytes. It was increased by high glucose concentration and required mobilization of intracellular Ca2+. Live images of beta cell–phagocyte interactions documented the intimacy of the membrane contact and t

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26

Couzinet, Sabine, Elisabeth Cejas, Johannes Schittny, Peter Deplazes, Rainer Weber, and Stefan Zimmerli. "Phagocytic Uptake of Encephalitozoon cuniculi by Nonprofessional Phagocytes." Infection and Immunity 68, no. 12 (2000): 6939–45. http://dx.doi.org/10.1128/iai.68.12.6939-6945.2000.

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ABSTRACT Encephalitozoon cuniculi is an obligate intracellular, spore-forming parasite belonging to the microsporidia that can cause disseminated infection in immunocompromised persons. E. cuniculi spores infect host cells by germination, i.e., by explosively everting the polar filament, through which the spore contents (sporoplasms) are subsequently injected into the cytoplasm. In addition, we observed intracellular, nongerminated spores in various nonprofessional phagocytes. In MRC5 cells, the number of internalized spores was approximately 10-fold higher than the number of injected sporopla

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27

Kono, Rena, Yuji Ikegaya, and Ryuta Koyama. "Phagocytic Glial Cells in Brain Homeostasis." Cells 10, no. 6 (2021): 1348. http://dx.doi.org/10.3390/cells10061348.

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Phagocytosis by glial cells has been shown to play an important role in maintaining brain homeostasis. Microglia are currently considered to be the major phagocytes in the brain parenchyma, and these cells phagocytose a variety of materials, including dead cell debris, abnormally aggregated proteins, and, interestingly, the functional synapses of living neurons. The intracellular signaling mechanisms that regulate microglial phagocytosis have been studied extensively, and several important factors, including molecules known as “find me” signals and “eat me” signals and receptors on microglia t

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28

Oliynyk, Zh. "LONG-TERM EFFECTS OF SHAM SURGERY ON PHAGOCYTE FUNCTIONS IN RATS." Biotechnologia Acta 15, no. 2 (2022): 37–46. http://dx.doi.org/10.15407/biotech15.02.037.

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Animal models of inflammatory disorders, including those of the nervous system are commonly used to explore the pathophysiological role of immune cell response in disease triggering and course and to develop biotechnology products for therapeutic use. Modeling some of these disorders, particularly neurodegenerative diseases, implies surgical manipulations for the intracerebral introduction of disease-initiating substances (toxins, amyloids etc.). Design of these experiments involves the use of sham-operated animals as a control of non-specific intrinsic side-effects elicited by surgical manipu

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29

Berge, Lillian Nordbø, Monika Østensen, and Arthur Revhaug. "Phagocytic cell activity in pre-eclampsia." Acta Obstetricia et Gynecologica Scandinavica 67, no. 6 (1988): 499–504. http://dx.doi.org/10.3109/00016348809029860.

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30

Gil-Lamaignere, C., A. Maloukou, J. L. Rodriguez-Tudela, and E. Roilides. "Human phagocytic cell responses toScedosporium prolificans." Medical Mycology 39, no. 2 (2001): 169–75. http://dx.doi.org/10.1080/mmy.39.2.169.175.

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31

Mege, J. L., C. Capo, B. Michel, J. L. Gastaut, and P. Bongrand. "Phagocytic cell function in aged subjects." Neurobiology of Aging 9 (January 1988): 217–20. http://dx.doi.org/10.1016/s0197-4580(88)80054-x.

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32

Etzioni, Amos. "Novel aspects of phagocytic cell disorders." Current Opinion in Allergy and Clinical Immunology 1, no. 6 (2001): 535–40. http://dx.doi.org/10.1097/00130832-200112000-00007.

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33

Ocon, P., J. M. Reguera, P. Morata, C. Juarez, A. Alonso, and J. D. Colmenero. "Phagocytic cell function in active brucellosis." Infection and Immunity 62, no. 3 (1994): 910–14. http://dx.doi.org/10.1128/iai.62.3.910-914.1994.

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34

Moschella, Samuel L., and Thomas G. Cropley. "Mononuclear phagocytic and dendritic cell systems." Journal of the American Academy of Dermatology 22, no. 6 (1990): 1091–97. http://dx.doi.org/10.1016/0190-9622(90)70158-e.

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35

Rezende, Gustavo L., Marcio Nakanishi, Shirley C. P. Couto, et al. "Alterations in innate immune responses of patients with chronic rhinosinusitis related to cystic fibrosis." PLOS ONE 17, no. 5 (2022): e0267986. http://dx.doi.org/10.1371/journal.pone.0267986.

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The role of phagocytes of children with cystic fibrosis (CF) associated with different phenotypes of chronic rhinosinusitis (CRS) is unclear. The aim of this study was to evaluate the phagocytic capacity of blood neutrophils and monocytes and production of superoxide anion by phagocytes in patients with CF with or without chronic rhinosinusitis and with or without nasal polyps (NP). This cross-sectional study was established in 2015–2017 in a tertiary reference center to the CF treatment, Brasilia, Brazil. Sample included 30 children volunteers with CRS related to CF (n = 16) and control subje

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36

Greenberg, Steven. "Diversity in phagocytic signalling." Journal of Cell Science 114, no. 6 (2001): 1039–40. http://dx.doi.org/10.1242/jcs.114.6.1039.

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37

Leslie, Mitch. "Rules of phagocytic attraction." Journal of Cell Biology 187, no. 5 (2009): 584. http://dx.doi.org/10.1083/jcb.1875iti3.

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38

Shklyar, Boris, Flonia Levy-Adam, Ketty Mishnaevski, and Estee Kurant. "Caspase Activity Is Required for Engulfment of Apoptotic Cells." Molecular and Cellular Biology 33, no. 16 (2013): 3191–201. http://dx.doi.org/10.1128/mcb.00233-13.

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Clearance of apoptotic cells by phagocytic neighbors is crucial for normal development of multicellular organisms. However, how phagocytes discriminate between healthy and dying cells remains poorly understood. We focus on glial phagocytosis of apoptotic neurons during development of theDrosophilacentral nervous system. We identified phosphatidylserine (PS) as a ligand on apoptotic cells for the phagocytic receptor Six Microns Under (SIMU) and report that PS alone is not sufficient for engulfment. Our data reveal that, additionally to PS exposure, caspase activity is required for clearance of

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39

Suresh, Madheswaran, Malarvizhi Gurusamy, and Natarajan Sudhakar. "MASKING ANTI-PHAGOCYTIC SIGNAL OF TUMOR BY PRO-PHAGOCYTIC SIGNAL-A KEY TO IMMUREMENT OF CANCER CELL." International Journal of Pharmacy and Pharmaceutical Sciences 8, no. 9 (2016): 323. http://dx.doi.org/10.22159/ijpps.2016v8i9.12990.

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<p>Immune surveillance is a mechanism where cells and tissues are watched constantly by ever alerted immune system. Most incipient cancer cells are recognized and eliminated by the immune surveillance mechanism, but still tumors have the ability to evade immune surveillance and immunological killing. One greater arm that tumor use to evade immune surveillance, is by expressing anti-phagocytic signal (CD47). Here we present a provocative hypothesis where cancer cells are removed alive by phagocytic cell (DC). That in turn will elicit effective and higher immunogenic condition. All this co

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Somersan, Selin, and Nina Bhardwaj. "Tethering and tickling." Journal of Cell Biology 155, no. 4 (2001): 501–4. http://dx.doi.org/10.1083/jcb.200110066.

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Several receptors are implicated in apoptotic cell (AC) uptake by phagocytic cells; however, their relative dominance in mammalian systems remains to be established. New studies shed light on the role of the phosphatidyl serine (PS) receptor (PSR). Ligation of PSR by PS on AC surfaces is considered essential for signaling uptake of ACs that are tethered to phagocytes via other receptors.

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Tsai, Wen-Hui, Shao-Chi Chang, Yu-Chieh Lin, and Hui-Chi Hsu. "CX3CL1(+) Microparticles-Induced MFG-E8 Enhances Apoptotic Cell Clearance by Alveolar Macrophages." Cells 10, no. 10 (2021): 2583. http://dx.doi.org/10.3390/cells10102583.

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During the resolution phase of acute lung injury, apoptotic cells release CX3CL1 as a “find-me” signal to attract alveolar macrophage transmigration toward apoptotic cells for phagocytosis. However, it is still not clear whether CX3CL1 has pro-phagocytic activity on alveolar macrophage. In this study, we investigated the role of apoptotic NB4 cells-derived CX3CL1(+) microparticles (apo-MP) on the phagocytic activity of NR8383 cells. We demonstrate that exogenous CX3CL1 and apo-MP enhanced the phagocytic activity of NR8383 cells in a CX3 CR1-dependent manner. The apo-MP-enhanced phagocytic acti

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Sateriale, Adam, Archana Vaithilingam, Liam Donnelly, Peter Miller, and Christopher D. Huston. "Feed-Forward Regulation of Phagocytosis by Entamoeba histolytica." Infection and Immunity 80, no. 12 (2012): 4456–62. http://dx.doi.org/10.1128/iai.00671-12.

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ABSTRACTThe parasitic protozoanEntamoeba histolyticais aptly named for its capacity to destroy host tissue. WhenE. histolyticatrophozoites invade the lamina propria of a host colon, extracellular matrices are degraded while host cells are killed and phagocytosed. The ability ofE. histolyticato phagocytose host cells correlates with virulencein vivo. In order to better understand the mechanism of phagocytosis, we used anE. histolyticaAffymetrix microarray chip to measure the total gene expression of phagocytic and nonphagocytic subpopulations. Using paramagnetic beads coated with a known host l

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Schreiner, G. F. "The mesangial phagocyte and its regulation of contractile cell biology." Journal of the American Society of Nephrology 2, no. 10 (1992): S74. http://dx.doi.org/10.1681/asn.v210s74.

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The mesangium constitutes the core of the renal glomerulus. It consists of the matrix, composed of mucopolysaccharides and glycoproteins, and two cell types. The predominant cell type is the mesangial cell, resembling a vascular smooth muscle cell. Up to 15% of the mesangial cell population additionally consists of resident mesangial phagocytes. These are derived from the bone marrow and belong to the family of mononuclear leukocytes. They are phagocytic, express Fc and C3 receptors, and display membrane Ia antigens. They syngeneically stimulate lymphocyte proliferation via antigen presentatio

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SkÖLd, C. M., A. Eklund, G. HalldÉN, and J. Hed. "Different cell surface and phagocytic properties in mononuclear phagocytes from blood and alveoli." APMIS 98, no. 1-6 (1990): 415–22. http://dx.doi.org/10.1111/j.1699-0463.1990.tb01052.x.

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Potoka, Douglas A., Sonshin Takao, Tetsuhiro Owaki, Gregory B. Bulkley, and Andrew S. Klein. "Endothelial Cells Potentiate Oxidant-Mediated Kupffer Cell Phagocytic Killing." Free Radical Biology and Medicine 24, no. 7-8 (1998): 1217–27. http://dx.doi.org/10.1016/s0891-5849(97)00453-x.

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Wendler, Astrid, Nicholas James, Michael H. Jones, and Christian Pernstich. "Phagocytosed Polyhedrin-Cytokine Cocrystal Nanoparticles Provide Sustained Secretion of Bioactive Cytokines from Macrophages." BioDesign Research 2021 (May 15, 2021): 1–12. http://dx.doi.org/10.34133/2021/9816485.

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Many cells possess the ability to engulf and incorporate particles by phagocytosis. This active process is characteristic of microorganisms as well as higher order species. In mammals, monocytes, macrophages, and microglia are among the so-called professional phagocytes. In addition, cells such as fibroblast and chondrocytes are classified as nonprofessional phagocytes. Professional phagocytes play important roles in both the innate and adaptive immune responses, wound healing, and tissue homeostasis. Consequently, these cells are increasingly studied as targets and vectors of therapeutic inte

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Damisah, Eyiyemisi C., Robert A. Hill, Anupama Rai, et al. "Astrocytes and microglia play orchestrated roles and respect phagocytic territories during neuronal corpse removal in vivo." Science Advances 6, no. 26 (2020): eaba3239. http://dx.doi.org/10.1126/sciadv.aba3239.

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Cell death is prevalent throughout life; however, the coordinated interactions and roles of phagocytes during corpse removal in the live brain are poorly understood. We developed photochemical and viral methodologies to induce death in single cells and combined this with intravital optical imaging. This approach allowed us to track multicellular phagocytic interactions with precise spatiotemporal resolution. Astrocytes and microglia engaged with dying neurons in an orchestrated and synchronized fashion. Each glial cell played specialized roles: Astrocyte processes rapidly polarized and engulfe

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Gaforio, José Juan, Elena Ortega, Ignacio Algarra, María José Serrano, and Gerardo Alvarez de Cienfuegos. "NK Cells Mediate Increase of Phagocytic Activity but Not of Proinflammatory Cytokine (Interleukin-6 [IL-6], Tumor Necrosis Factor Alpha, and IL-12) Production Elicited in Splenic Macrophages by Tilorone Treatment of Mice during Acute Systemic Candidiasis." Clinical and Vaccine Immunology 9, no. 6 (2002): 1282–94. http://dx.doi.org/10.1128/cdli.9.6.1282-1294.2002.

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ABSTRACT The participation of NK cells in the activation of splenic macrophages or in resistance to systemic candidiasis is still a matter of debate. We had previously reported that there is a correlation between natural killer cell activation and resistance to systemic candidiasis. In those experiments we had used tilorone to boost NK cell activity in mice. Here we show a mechanism elicited by tilorone in splenic macrophages which could explain their effect on mouse survival during acute disseminated Candida albicans infection. The results demonstrate that tilorone treatment elicits, by a dir

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Molinari, Beatriz L., Deborah R. Tasat, Mónica A. Palmieri, and Rómulo L. Cabrini. "Kinetics of MTT-formazan exocytosis in phagocytic and non-phagocytic cells." Micron 36, no. 2 (2005): 177–83. http://dx.doi.org/10.1016/j.micron.2004.08.002.

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Plekhova, N. G., L. M. Somova, and E. I. Drobot. "The metabolism of the innate immunity cells in bacterial infections." Biomeditsinskaya Khimiya 61, no. 1 (2015): 105–14. http://dx.doi.org/10.18097/pbmc20156101105.

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Metabolic activity of innate immunity cells infected by various doses of Gram-negative (Yersinia pseudotuberculosis, Salmonella enteritidis) and Gram-positive (Staphylococcus aureus, Listeria monocytogenes) bacteria has been investigated. Using various animal models we found that during the initial period (up to 2 days) changes of infection in cellular responses depend on the type of the pathogen. In response to infection caused by Gram-negative bacteria predominant neutrophil accumulation in the foci of inflammation was observed, while Gram-positive bacteria induced preferential accumulation

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