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Статті в журналах з теми "Postnatal overfeeding"

Автор: Grafiati

Опубліковано: 20 квітня 2024

Оновлено: 31 липня 2025

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1

Sousa, Diana, Mariana Rocha, Andreia Amaro, et al. "Exposure to Obesogenic Environments during Perinatal Development Modulates Offspring Energy Balance Pathways in Adipose Tissue and Liver of Rodent Models." Nutrients 15, no. 5 (2023): 1281. http://dx.doi.org/10.3390/nu15051281.

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Obesogenic environments such as Westernized diets, overnutrition, and exposure to glycation during gestation and lactation can alter peripheral neuroendocrine factors in offspring, predisposing for metabolic diseases in adulthood. Thus, we hypothesized that exposure to obesogenic environments during the perinatal period reprograms offspring energy balance mechanisms. Four rat obesogenic models were studied: maternal diet-induced obesity (DIO); early-life obesity induced by postnatal overfeeding; maternal glycation; and postnatal overfeeding combined with maternal glycation. Metabolic parameter

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2

Castellano, Juan M., Agnete H. Bentsen, Miguel A. Sánchez-Garrido, et al. "Early Metabolic Programming of Puberty Onset: Impact of Changes in Postnatal Feeding and Rearing Conditions on the Timing of Puberty and Development of the Hypothalamic Kisspeptin System." Endocrinology 152, no. 9 (2011): 3396–408. http://dx.doi.org/10.1210/en.2010-1415.

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Kiss1 neurons have recently emerged as a putative conduit for the metabolic gating of reproduction, with leptin being a regulator of hypothalamic Kiss1 expression. Early perturbations of the nutritional status are known to predispose to different metabolic disorders later in life and to alter the timing of puberty; however, the potential underlying mechanisms remain poorly defined. Here we report how changes in the pattern of postnatal feeding affect the onset of puberty and evaluate key hormonal and neuropeptide [Kiss1/kisspeptin (Kp)] alterations linked to these early nutritional manipulatio

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3

You, Su, Franziska Götz, W. Rohde, and G. Dörner. "Early Postnatal Overfeeding and Diabetes Susceptibility." Experimental and Clinical Endocrinology & Diabetes 96, no. 06 (2009): 301–6. http://dx.doi.org/10.1055/s-0029-1211023.

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4

Mićić, Bojana, Ana Djordjevic, Nataša Veličković, et al. "AMPK Activation as a Protective Mechanism to Restrain Oxidative Stress in the Insulin-Resistant State in Skeletal Muscle of Rat Model of PCOS Subjected to Postnatal Overfeeding." Biomedicines 11, no. 6 (2023): 1586. http://dx.doi.org/10.3390/biomedicines11061586.

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Polycystic ovary syndrome (PCOS) is a common endocrinopathy in women of reproductive age, often associated with obesity and insulin resistance. Childhood obesity is an important predisposing factor for the development of PCOS later in life. Being particularly interested in the interplay between prepubertal obesity and hyperandrogenemia, we investigated the effects of early postnatal overfeeding, accomplished by reducing litter size during the period of suckling, on energy sensing and insulin signaling pathways in the gastrocnemius muscle of a rat model of PCOS-induced by 5α-dihydrotestosterone

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5

Kappeler, Laurent, Carlos De Magalhaes Filho, Patricia Leneuve, et al. "Early Postnatal Nutrition Determines Somatotropic Function in Mice." Endocrinology 150, no. 1 (2008): 314–23. http://dx.doi.org/10.1210/en.2008-0981.

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Increasing evidence suggests a developmental origin for a number of human diseases, notably after intrauterine or postnatal nutrient deprivation. Nutritional changes readily translate into alterations of somatic growth. However, whereas intrauterine growth retardation often shows postnatal catch-up growth, recovery from food restriction immediately after birth is limited. Therefore, we investigated whether early postnatal nutrition (undernutrition and overfeeding) modifies plasticity of growth through developmental control of the somatotropic hormone axis. We used cross-fostering in mice to in

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6

Josse, Marie, Eve Rigal, Nathalie Rosenblatt-Velin, et al. "Influence of postnatal overfeeding on postnatal heart development in juvenile mice." Archives of Cardiovascular Diseases Supplements 15, no. 2 (2023): 192. http://dx.doi.org/10.1016/j.acvdsp.2023.03.029.

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7

Du, Susu, Xiaolei Zhu, Nan Zhou, Wen Zheng, Wei Zhou, and Xiaonan Li. "Curcumin alleviates hepatic steatosis by improving mitochondrial function in postnatal overfed rats and fatty L02 cells through the SIRT3 pathway." Food & Function 13, no. 4 (2022): 2155–71. http://dx.doi.org/10.1039/d1fo03752h.

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Postnatal overfeeding damaged mitochondrial biogenesis and antioxidant response, and increased hepatic lipids and the severity of high-fat-induced NAFLD, while curcumin alleviated hepatic steatosis, at least partially, by enhancing mitochondrial function through SIRT3.

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8

Mićić, Bojana, Ana Teofilović, Ana Djordjevic, Nataša Veličković, Djuro Macut, and Danijela Vojnović Milutinović. "AMPK Activation Is Important for the Preservation of Insulin Sensitivity in Visceral, but Not in Subcutaneous Adipose Tissue of Postnatally Overfed Rat Model of Polycystic Ovary Syndrome." International Journal of Molecular Sciences 23, no. 16 (2022): 8942. http://dx.doi.org/10.3390/ijms23168942.

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Polycystic ovary syndrome (PCOS) is a well-known reproductive syndrome usually associated with obesity, insulin resistance, and hyperinsulinemia. Although the first signs of PCOS begin early in adolescence, it is underexplored whether peripubertal obesity predisposes women to PCOS metabolic disturbances. To highlight that, we examined the impact of postnatal overfeeding-induced obesity, achieved by litter size reduction during the suckling period, on metabolic disturbances associated with visceral and subcutaneous adipose tissue (VAT and SAT) function in the 5α-dihydrotestosterone (5α-DHT)-ind

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9

Boubred, Farid, Laurent Daniel, Christophe Buffat, et al. "Early postnatal overfeeding induces early chronic renal dysfunction in adult male rats." American Journal of Physiology-Renal Physiology 297, no. 4 (2009): F943—F951. http://dx.doi.org/10.1152/ajprenal.90704.2008.

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Low birth weight is associated with an increased risk of hypertension and renal dysfunction at adulthood. Such an association has been shown to involve a reduction of nephron endowment and to be enhanced by accelerated postnatal growth in humans. However, while low-birth-weight infants often undergo catch-up growth, little is known about the long-term vascular and renal effects of accelerated postnatal growth. We surimposed early postnatal overfeeding (OF; reduction of litter size during the suckling period) to appropriate-birth-weight (NBW+OF) and intrauterine growth restriction (IUGR; IUGR+O

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10

Josse, Marie, Eve Rigal, Nathalie Rosenblatt-Velin, et al. "Programming of Cardiovascular Dysfunction by Postnatal Overfeeding in Rodents." International Journal of Molecular Sciences 21, no. 24 (2020): 9427. http://dx.doi.org/10.3390/ijms21249427.

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Nutritional environment in the perinatal period has a great influence on health and diseases in adulthood. In rodents, litter size reduction reproduces the effects of postnatal overnutrition in infants and reveals that postnatal overfeeding (PNOF) not only permanently increases body weight but also affects the cardiovascular function in the short- and long-term. In addition to increased adiposity, the metabolic status of PNOF rodents is altered, with increased plasma insulin and leptin levels, associated with resistance to these hormones, changed profiles and levels of circulating lipids. PNOF

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11

Habbout, Ahmed. "247 Metabolic and cardiovascular consequences of postnatal overfeeding in mice." Archives of Cardiovascular Diseases Supplements 3, no. 1 (2011): 81. http://dx.doi.org/10.1016/s1878-6480(11)70249-x.

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12

Rigal, E., C. Greco, A. Méloux, et al. "Long-term impact of postnatal overfeeding on myocardial protective pathways." Archives of Cardiovascular Diseases Supplements 10, no. 2 (2018): 188–89. http://dx.doi.org/10.1016/j.acvdsp.2018.02.031.

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13

Habbout, Ahmed, Stéphanie Delemasure-Chalumeau, Carole Richard, Luc Rochette, and Catherine Vergely. "Metabolic, Oxidative and Cardiovascular Consequences of Postnatal Overfeeding in Mice." Free Radical Biology and Medicine 49 (January 2010): S42. http://dx.doi.org/10.1016/j.freeradbiomed.2010.10.090.

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14

Juvet, C., U. Simeoni, C. Yzydorczyk, et al. "Effect of early postnatal nutrition on chronic kidney disease and arterial hypertension in adulthood: a narrative review." Journal of Developmental Origins of Health and Disease 9, no. 6 (2018): 598–614. http://dx.doi.org/10.1017/s2040174418000454.

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AbstractIntrauterine growth restriction (IUGR) has been identified as a risk factor for adult chronic kidney disease (CKD), including hypertension (HTN). Accelerated postnatal catch-up growth superimposed to IUGR has been shown to further increase the risk of CKD and HTN. Although the impact of excessive postnatal growth without previous IUGR is less clear, excessive postnatal overfeeding in experimental animals shows a strong impact on the risk of CKD and HTN in adulthood. On the other hand, food restriction in the postnatal period seems to have a protective effect on CKD programming. All the

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15

MohanKumar, S. M. J., T. D. Rajendran, A. K. Vyas, et al. "Effects of prenatal bisphenol-A exposure and postnatal overfeeding on cardiovascular function in female sheep." Journal of Developmental Origins of Health and Disease 8, no. 1 (2016): 65–74. http://dx.doi.org/10.1017/s204017441600057x.

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Bisphenol-A (BPA) is a widely used endocrine-disrupting chemical. Prenatal exposure to BPA is known to affect birth weight, but its impact on the cardiovascular system has not been studied in detail. In this study, we investigated the effects of prenatal BPA treatment and its interaction with postnatal overfeeding on the cardiovascular system. Pregnant sheep were given daily subcutaneous injections of corn oil (control) or BPA (0.5 mg/kg/day in corn oil) from day 30 to day 90 of gestation. A subset of female offspring of these dams were overfed to increase body weight to ~30% over that of norm

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16

Sánchez-Garrido, M. A., J. M. Castellano, F. Ruiz-Pino, et al. "Metabolic Programming of Puberty: Sexually Dimorphic Responses to Early Nutritional Challenges." Endocrinology 154, no. 9 (2013): 3387–400. http://dx.doi.org/10.1210/en.2012-2157.

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Body energy stores and metabolic cues influence the onset of puberty. However, the pubertal impact of early nutritional challenges has been only fragmentarily addressed. We evaluated here the consequences, in terms of pubertal timing and hormonal markers, of various nutritional manipulations during pre- or postnatal maturation in rats of both sexes. Males and females were submitted to gestational undernutrition (UNG) or peripubertal (SUB) subnutrition or were raised in large (LL; underfeeding) or small (SL; overfeeding) litters. In addition, groups of UNG, LL, and SL rats were fed on a high-fa

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17

Melnik, Bodo C., Ralf Weiskirchen, Swen Malte John, et al. "White Adipocyte Stem Cell Expansion Through Infant Formula Feeding: New Insights into Epigenetic Programming Explaining the Early Protein Hypothesis of Obesity." International Journal of Molecular Sciences 26, no. 10 (2025): 4493. https://doi.org/10.3390/ijms26104493.

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Prolonged breastfeeding (BF), as opposed to artificial infant formula feeding (FF), has been shown to prevent the development of obesity later in life. The aim of our narrative review is to investigate the missing molecular link between postnatal protein overfeeding—often referred to as the “early protein hypothesis”—and the subsequent transcriptional and epigenetic changes that accelerate the expansion of adipocyte stem cells (ASCs) in the adipose vascular niche during postnatal white adipose tissue (WAT) development. To achieve this, we conducted a search on the Web of Science, Google Schola

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18

SCHMIDT, INGRID, CORINNA SCHOELCH, THOMAS ZISKA, DARIUS SCHNEIDER, ECKHART SIMON, and ANDREAS PLAGEMANN. "Interaction of genetic and environmental programming of the leptin system and of obesity disposition." Physiological Genomics 3, no. 2 (2000): 113–20. http://dx.doi.org/10.1152/physiolgenomics.2000.3.2.113.

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Schmidt, Ingrid, Corinna Schoelch, Thomas Ziska, Darius Schneider, Eckhart Simon, and Andreas Plagemann. Interaction of genetic and environmental programming for disturbances of the leptin system and for obesity. Physiol Genomics 3: 113–120, 2000.—Possible adverse interactions between an usually inconspicuous genetic trait and early environmental factors favoring the development of obesity were investigated in rats heterozygous for the leptin receptor defect “ fatty” ( fa). Pups were exposed to early postnatal overfeeding by reducing litter size from normally 10–12 to only 4. Rearing +/+ and +

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19

Amaro, Andreia, Diana Sousa, Mariana Sá-Rocha, et al. "Postnatal Overfeeding in Rodents Induces a Neurodevelopment Delay and Anxious-like Behaviour Accompanied by Sex- and Brain-Region-Specific Synaptic and Metabolic Changes." Nutrients 15, no. 16 (2023): 3581. http://dx.doi.org/10.3390/nu15163581.

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Nutritional disturbances during the early postnatal period can have long-lasting effects on neurodevelopment and may be related to behavioural changes at adulthood. While such neuronal connection disruption can contribute to social and behaviour alterations, the dysregulation of the neuroendocrine pathways involved in nutrient-sensing balance may also cause such impairments, although the underlying mechanisms are still unclear. We aimed to evaluate sex-specific neurodevelopmental and behavioural changes upon postnatal overfeeding and determine the potential underpinning mechanisms at the centr

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20

Boubred, Farid, Christophe Buffat, Jean-Marc Feuerstein, et al. "Effects of early postnatal hypernutrition on nephron number and long-term renal function and structure in rats." American Journal of Physiology-Renal Physiology 293, no. 6 (2007): F1944—F1949. http://dx.doi.org/10.1152/ajprenal.00141.2007.

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Various antenatal events impair nephrogenesis in humans as well as in several animal models. The consecutive low nephron endowment may contribute to an increased risk for cardiovascular and renal diseases in adulthood. However, little knowledge is available on the influence of the postnatal environment, especially nutrition, on nephrogenesis. Moreover, the consequences of early postnatal nutrition in late adulthood are not clear. We used a model of early postnatal overfeeding (OF) induced by reduction of litter size (3 pups/litter) in rats. Systolic blood pressure (SBP; plethysmography), glome

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21

Lenon Klein, John, Sander Martinho Adams, Amanda Farias De Moura, et al. "Efecto de la nutrición en el último tercio de la gestación de vacas de carne sobre el desarrollo de la progenie." Revista Mexicana de Ciencias Pecuarias 13, no. 3 (2022): 658–73. http://dx.doi.org/10.22319/rmcp.v13i3.6015.

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The restriction of nutrient intake by beef cows during pregnancy may influence the progeny postpartum growth potential, therefore, the objective of the present study was to evaluate the effects of nutritional restriction and adequate nutrition or overfeeding during the final third of gestation of the crossbred cows (Charolais x Nellore) kept in the Pampa biome, on the productive performance of the progeny up to 15 mo of age. Eighty-three (83) cows were divided into: control cows on natural pasture under nutritional restriction (RES); Supplementation to meet 100 % of requirements (REQ); Supplem

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22

Josse, Marie, Léa Longin, Eve Rigal, et al. "Early postnatal overfeeding influence on cardiac composition and metabolism in juvenile mice." Archives of Cardiovascular Diseases 118, no. 6-7 (2025): S223—S224. https://doi.org/10.1016/j.acvd.2025.03.113.

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23

Costa, V. M. G., A. E. Andreazzi, M. Bolotari, C. G. Lade, M. O. Guerra, and V. M. Peters. "Effect of postnatal overfeeding on the male and female Wistar rat reproductive parameters." Journal of Developmental Origins of Health and Disease 10, no. 6 (2019): 667–75. http://dx.doi.org/10.1017/s2040174419000163.

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AbstractOverweight/obesity has become a worldwide epidemic, and factors such as a sedentary lifestyle and inadequate eating habits directly contribute to the development of this condition. Studies indicate that rapid weight gain at critical development stages, such as the lactation period, is associated with the development of obesity, cardiovascular diseases, and diabetes in the long term. In addition to metabolic changes during adulthood, overweight/obesity may influence reproductive function of the population. In this context, the present study aimed to evaluate postnatal overfeeding effect

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24

Dai, Yanyan, Nan Zhou, Fan Yang, et al. "Effects of postnatal overfeeding and fish oil diet on energy expenditure in rats." Pediatric Research 83, no. 1 (2017): 156–63. http://dx.doi.org/10.1038/pr.2017.207.

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25

Clarke, M. A., A. Stefanidis, and S. J. Spencer. "Postnatal Overfeeding Leads to Obesity and Exacerbated Febrile Responses to Lipopolysaccharide Throughout Life." Journal of Neuroendocrinology 24, no. 3 (2012): 511–24. http://dx.doi.org/10.1111/j.1365-2826.2011.02269.x.

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26

Rodrigues, Ananda L., Érica P. G. De Souza, Simone V. Da Silva, et al. "Low expression of insulin signaling molecules impairs glucose uptake in adipocytes after early overnutrition." Journal of Endocrinology 195, no. 3 (2007): 485–94. http://dx.doi.org/10.1677/joe-07-0046.

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Experimental and clinical studies have demonstrated that early postnatal overnutrition represents a risk factor for later obesity and associated metabolic and cardiovascular disturbance. In the present study, we assessed the levels of glucose transporter 4 (GLUT-4), GLUT-1, insulin receptor (IR), IR substrate 1 (IRS-1), phosphatidylinositol 3-kinase (PI3K) and Akt expression, as well as insulin-stimulated glucose transport and Akt activity in adipocytes from adult rats previously raised in small litters (SL). The normal litter (NL) served as control group. We also investigated glycemia, insuli

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27

Yzydorczyk, Catherine, Na Li, Eve Rigal, et al. "Calorie Restriction in Adulthood Reduces Hepatic Disorders Induced by Transient Postnatal Overfeeding in Mice." Nutrients 11, no. 11 (2019): 2796. http://dx.doi.org/10.3390/nu11112796.

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Impaired early nutrition influences the risk of developing metabolic disorders in later life. We observed that transient postnatal overfeeding (OF) in mice induces long-term hepatic alterations, characterized by microsteatosis, fibrosis associated with oxidative stress (OS), and stress-induced premature senescence (SIPS). In this study, we investigated whether such changes can be reversed by moderate calorie restriction (CR). C57BL/6 male mice pups were maintained during lactation in litters adjusted to nine pups in the normal feeding (NF) group and three pups in the transient postnatal OF gro

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28

Boubred, F., M. Saint-Faust, C. Buffat, I. Ligi, I. Grandvuillemin, and U. Simeoni. "Developmental Origins of Chronic Renal Disease: An Integrative Hypothesis." International Journal of Nephrology 2013 (2013): 1–12. http://dx.doi.org/10.1155/2013/346067.

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Cardiovascular diseases are one of the leading causes of mortality. Hypertension (HT) is one of the principal risk factors associated with death. Chronic kidney disease (CKD), which is probably underestimated, increases the risk and the severity of adverse cardiovascular events. It is now recognized that low birth weight is a risk factor for these diseases, and this relationship is amplified by a rapid catch-up growth or overfeeding during infancy or childhood. The pathophysiological and molecular mechanisms involved in the “early programming” of CKD are multiple and partially understood. It h

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29

Peel, R. K., G. J. Eckerle, and R. V. Anthony. "Effects of overfeeding naturally-mated adolescent ewes on maternal, fetal, and postnatal lamb growth1." Journal of Animal Science 90, no. 11 (2012): 3698–708. http://dx.doi.org/10.2527/jas.2012-5140.

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30

Davidowa, Helga, and Andreas Plagemann. "Inhibition by insulin of hypothalamic VMN neurons in rats overweight due to postnatal overfeeding." Neuroreport 12, no. 15 (2001): 3201–4. http://dx.doi.org/10.1097/00001756-200110290-00012.

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31

Boullu-Ciocca, S., V. Tassistro, A. Dutour, and M. Grino. "Pioglitazone in adult rats reverses immediate postnatal overfeeding-induced metabolic, hormonal, and inflammatory alterations." Endocrine 50, no. 3 (2015): 608–19. http://dx.doi.org/10.1007/s12020-015-0657-z.

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32

Junior, Marcos Divino Ferreira, Keilah Valéria Naves Cavalcante, Lucas Araújo Ferreira, et al. "Postnatal early overfeeding induces cardiovascular dysfunction by oxidative stress in adult male Wistar rats." Life Sciences 226 (June 2019): 173–84. http://dx.doi.org/10.1016/j.lfs.2019.04.018.

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33

Tejera-Muñoz, Antonio, Lucía Guerra-Menéndez, Sara Amor, Daniel González-Hedström, Ángel Luis García-Villalón, and Miriam Granado. "Postnatal Overfeeding during Lactation Induces Endothelial Dysfunction and Cardiac Insulin Resistance in Adult Rats." International Journal of Molecular Sciences 24, no. 19 (2023): 14443. http://dx.doi.org/10.3390/ijms241914443.

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Early overnutrition is associated with cardiometabolic alterations in adulthood, likely attributed to reduced insulin sensitivity due to its crucial role in the cardiovascular system. This study aimed to assess the long-term effects of early overnutrition on the development of cardiovascular insulin resistance. An experimental childhood obesity model was established using male Sprague Dawley rats. Rats were organized into litters of 12 pups/mother (L12-Controls) or 3 pups/mother (L3-Overfed) at birth. After weaning, animals from L12 and L3 were housed three per cage and provided ad libitum acc

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34

Govoni, Kristen E., Sarah A. Reed, and Steven A. Zinn. "CELL BIOLOGY SYMPOSIUM: METABOLIC RESPONSES TO STRESS: FROM ANIMAL TO CELL: Poor maternal nutrition during gestation: effects on offspring whole-body and tissue-specific metabolism in livestock species1,2." Journal of Animal Science 97, no. 7 (2019): 3142–52. http://dx.doi.org/10.1093/jas/skz157.

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Abstract Poor maternal nutrition, both restricted-feeding and overfeeding, during gestation can negatively affect offspring growth, body composition, and metabolism. The effects are observed as early as the prenatal period and often persist through postnatal growth and adulthood. There is evidence of multigenerational effects demonstrating the long-term negative impacts on livestock production. We and others have demonstrated that poor maternal nutrition impairs muscle growth, increases adipose tissue, and negatively affects liver function. In addition to altered growth, changes in key metabol

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35

Fabianová, Kamila, Janka Babeľová, Dušan Fabian, et al. "Maternal High-Energy Diet during Pregnancy and Lactation Impairs Neurogenesis and Alters the Behavior of Adult Offspring in a Phenotype-Dependent Manner." International Journal of Molecular Sciences 23, no. 10 (2022): 5564. http://dx.doi.org/10.3390/ijms23105564.

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Obesity is one of the biggest and most costly health challenges the modern world encounters. Substantial evidence suggests that the risk of metabolic syndrome or obesity formation may be affected at a very early stage of development, in particular through fetal and/or neonatal overfeeding. Outcomes from epidemiological studies indicate that maternal nutrition during pregnancy and lactation has a profound impact on adult neurogenesis in the offspring. In the present study, an intergenerational dietary model employing overfeeding of experimental mice during prenatal and early postnatal developme

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36

Achard, Vincent, Caroline Sanchez, Virginie Tassistro, Monique Verdier, Marie-Christine Alessi, and Michel Grino. "Immediate Postnatal Overfeeding in Rats Programs Aortic Wall Structure Alterations and Metalloproteinases Dysregulation in Adulthood." American Journal of Hypertension 29, no. 6 (2015): 719–26. http://dx.doi.org/10.1093/ajh/hpv183.

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37

Plagemann, A., I. Heidrich, F. Götz, W. Rohde, and G. Dörner. "Obesity and Enhanced Diabetes and Cardiovascular Risk in Adult Rats due to Early Postnatal Overfeeding." Experimental and Clinical Endocrinology & Diabetes 99, no. 03 (2009): 154–58. http://dx.doi.org/10.1055/s-0029-1211159.

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Rodrigues, Ananda Lages, Egberto Gaspar de Moura, Magna Cottini Fonseca Passos, et al. "Postnatal early overfeeding induces hypothalamic higher SOCS3 expression and lower STAT3 activity in adult rats." Journal of Nutritional Biochemistry 22, no. 2 (2011): 109–17. http://dx.doi.org/10.1016/j.jnutbio.2009.11.013.

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Habbout, Ahmed, Na Li, Luc Rochette, and Catherine Vergely. "Postnatal Overfeeding in Rodents by Litter Size Reduction Induces Major Short- and Long-Term Pathophysiological Consequences." Journal of Nutrition 143, no. 5 (2013): 553–62. http://dx.doi.org/10.3945/jn.112.172825.

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Li, Na, Eve Rigal, Charles Guenancia, Luc Rochette, and Catherine Vergely. "0229: Alterations of cardiac function induced by postnatal overfeeding can be reversed by moderate diet restriction." Archives of Cardiovascular Diseases Supplements 6 (April 2014): 44. http://dx.doi.org/10.1016/s1878-6480(14)71382-5.

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Conceição, Ellen Paula Santos, Egberto Gaspar Moura, Elaine Oliveira, et al. "Dietary calcium supplementation in adult rats reverts brown adipose tissue dysfunction programmed by postnatal early overfeeding." Journal of Nutritional Biochemistry 39 (January 2017): 117–25. http://dx.doi.org/10.1016/j.jnutbio.2016.09.013.

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Santos-Marcos, Jose A., Alexia Barroso, Oriol A. Rangel-Zuñiga, et al. "Interplay between gonadal hormones and postnatal overfeeding in defining sex-dependent differences in gut microbiota architecture." Aging 12, no. 20 (2020): 19979–20000. http://dx.doi.org/10.18632/aging.104140.

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Habbout, Ahmed, Stephanie Delemasure, Françoise Goirand, et al. "Postnatal overfeeding in rats leads to moderate overweight and to cardiometabolic and oxidative alterations in adulthood." Biochimie 94, no. 1 (2012): 117–24. http://dx.doi.org/10.1016/j.biochi.2011.09.023.

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Dai, Yanyan, Fan Yang, Nan Zhou та ін. "A post-weaning fish oil dietary intervention reverses adverse metabolic outcomes and 11β-hydroxysteroid dehydrogenase type 1 expression in postnatal overfed rats". British Journal of Nutrition 116, № 9 (2016): 1519–29. http://dx.doi.org/10.1017/s0007114516003718.

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Анотація:

AbstractEarly life is considered a critical period for determining long-term metabolic health. Postnatal over-nutrition may alter glucocorticoid (GC) metabolism and increase the risk of developing obesity and metabolic disorders in adulthood. Our aim was to assess the effects of the dose and timing of a fish oil diet on obesity and the expression of GC-activated enzyme 11β-hydroxysteroid dehydrogenase type 1 (HSD1) in postnatal overfed rats. Litter sizes were adjusted to three (small litter (SL)) or ten (normal litter) rats on postnatal day 3 to induce overfeeding or normal feeding. The SL rat

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Veiga-Lopez, Almudena, Jacob Moeller, Rohit Sreedharan, et al. "Developmental programming: interaction between prenatal BPA exposure and postnatal adiposity on metabolic variables in female sheep." American Journal of Physiology-Endocrinology and Metabolism 310, no. 3 (2016): E238—E247. http://dx.doi.org/10.1152/ajpendo.00425.2015.

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Among potential contributors for the increased incidence of metabolic diseases is the developmental exposure to endocrine-disrupting chemicals such as bisphenol A (BPA). BPA is an estrogenic chemical used in a variety of consumer products. Evidence points to interactions of BPA with the prevailing environment. The aim of this study was to assess the effects of prenatal exposure to BPA on postnatal metabolic outcomes, including insulin resistance, adipose tissue distribution, adipocyte morphometry, and expression of inflammatory markers in adipose tissue as well as to assess whether postnatal o

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Morris, M. J. "060. OVER FEEDING EARLY IN LIFE AND RISK OF OBESITY: INSIGHT FROM THE RODENT." Reproduction, Fertility and Development 21, no. 9 (2009): 15. http://dx.doi.org/10.1071/srb09abs060.

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While adult lifestyle factors undoubtedly contribute to the incidence of obesity and its attendant disorders, mounting evidence suggests that programming of obesity may occur following over-nutrition during development. As hypothalamic control of appetite and energy expenditure is set early in life and can be perturbed by certain exposures such as under-nutrition and altered metabolic and hormonal signals, in utero exposure to maternal obesity related changes may contribute to programming of obesity in offspring. Data from animal studies indicate both intrauterine and postnatal environments ar

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Ferreira, Lucas Araújo, Marcos Divino Ferreira-Junior, Keytiane de Jesus Viana Amaral, et al. "Maternal postnatal early overfeeding induces sex-related cardiac dysfunction and alters sexually hormones levels in young offspring." Journal of Nutritional Biochemistry 103 (May 2022): 108969. http://dx.doi.org/10.1016/j.jnutbio.2022.108969.

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Koneva, L. A., A. K. Vyas, R. C. McEachin, et al. "Developmental programming: Interaction between prenatal BPA and postnatal overfeeding on cardiac tissue gene expression in female sheep." Environmental and Molecular Mutagenesis 58, no. 1 (2017): 4–18. http://dx.doi.org/10.1002/em.22071.

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Rinaldi, Wilson, Rodrigo Gomes, Dionízia Scomparin, et al. "Low-intensity and moderate exercise training improves autonomic nervous system activity imbalanced by postnatal early overfeeding in rats." Journal of the International Society of Sports Nutrition 11, no. 1 (2014): 25. http://dx.doi.org/10.1186/1550-2783-11-25.

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Li, Yuzhen, Andreas Plagemann, and Helga Davidowa. "Increased inhibition by agouti-related peptide of ventromedial hypothalamic neurons in rats overweight due to early postnatal overfeeding." Neuroscience Letters 330, no. 1 (2002): 33–36. http://dx.doi.org/10.1016/s0304-3940(02)00722-x.

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