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Статті в журналах з теми "Premalignant"

Автор: Grafiati

Опубліковано: 3 червня 2025

Оновлено: 31 липня 2025

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1

Namba, Ruria, Jeannie E. Maglione, Lawrence J. T. Young, et al. "Molecular Characterization of the Transition to Malignancy in a Genetically Engineered Mouse-Based Model of Ductal Carcinoma In situ." Molecular Cancer Research 2, no. 8 (2004): 453–63. http://dx.doi.org/10.1158/1541-7786.453.2.8.

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Abstract A transplantable model of human ductal carcinoma in situ that progresses to invasive carcinoma was developed from a genetically engineered mouse (GEM). Additional lines were established using early mammary premalignant lesions from transgenic MMTV-PyV-mT mice. These lines were verified to be premalignant and transplanted repeatedly to establish stable and predictable properties. Here, we report the first in-depth molecular analysis of neoplastic progression occurring in one premalignant transplantable GEM-derived line. Oligonucleotide microarrays showed that many genes are differentia

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2

Gündüz, Reyhan, Elif Ağaçayak, Gülcan Okutucu, Ulaş Alabalik, and Mehmet Sıddık Evsen. "Evaluation of definitive histopathological results of patients diagnosed with endometrial polyps: a tertiary care center experience." African Health Sciences 22, no. 1 (2022): 125–32. http://dx.doi.org/10.4314/ahs.v22i1.16.

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Objectives: To determine the premalignancy and malignancy prevalence in patients diagnosed with endometrial polyps and to investigate factors affecting premalignancy and malignancy. Methods: In our retrospective study, patients who were diagnosed with endometrial polyp with endometrial samples and who underwent polypectomy by hysteroscopy or hysterectomy within one year were included. Results: Premalignant / malignant histopathological results were detected in 7 (2.8%) patients. There were no statistically significant differences in histopathological results and endometrial samplin

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3

Blaisdell, Adam, Takiko Daikoku, Chin Siean Tay, Sudhansu Dey, and Adrian Erlebacher. "Neutrophil recruitment into premalignant lesions differentially influences tumor growth versus progression. (TUM4P.928)." Journal of Immunology 192, no. 1_Supplement (2014): 138.29. http://dx.doi.org/10.4049/jimmunol.192.supp.138.29.

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Abstract Neutrophils possess a diverse arsenal of enzymatic and chemical weapons that can inflict damage upon pathogens and host cells alike. Most tumors harbor neutrophils even from the earliest stages of progression (premalignancy), wherein these weapons could, in theory, either kill a tumor cell or enhance its malignant potential via mutagenesis. However, the interaction between neutrophils and premalignant tumor cells has not been well characterized. To this end, we utilized a mouse model of endometrial carcinoma with defined stages of premalignancy and malignancy that faithfully represent

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4

Stanton, Sasha E., Kristin G. Anderson, Tullia C. Bruno, et al. "SITC strategic vision: prevention, premalignant immunity, host and environmental factors." Journal for ImmunoTherapy of Cancer 13, no. 3 (2025): e010419. https://doi.org/10.1136/jitc-2024-010419.

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Cancer immunotherapy has improved the survival of a subset of patients by harnessing the power of the immune system to find and destroy malignant cells. The immune system also protects the host by destroying developing premalignant and malignant tumors. Advancing our knowledge of premalignant immunity and immune changes seen in lesions that develop into invasive cancer versus those that regress offers an exciting opportunity to leverage the immune system for immune prevention and immune interception of premalignancy. Understanding the immune environment of premalignant lesions and how chronic

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5

Ma, Peiwen, Pamela Beatty, John McKolanis, Robert Schoen, Randal Brand, and Olivera J. Finn. "Myeloid derived suppressor cells (MDSC) and anti-MUC1 immunosurveillance in pre-malignancy and cancer." Journal of Immunology 200, no. 1_Supplement (2018): 122.17. http://dx.doi.org/10.4049/jimmunol.200.supp.122.17.

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Abstract Recent work from our lab showed that in a colon cancer prevention trial of a tumor antigen MUC1 vaccine in patients with premalignant colon adenomas, those who did not make anti-MUC1 IgG, had higher pre-vaccination levels of circulating MDSC compared to vaccine responders. This was the first observation of increased MDSC in premalignancy. MDSC in premalignancy have not been compared to MDSC in cancer and our hypothesis is that there might be differences in the composition of various MDSC subpopulation and their immunosuppressive functions due to the shorter time of exposure to disease

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6

Spira, Avrum, Mary L. Disis, John T. Schiller, et al. "Leveraging premalignant biology for immune-based cancer prevention." Proceedings of the National Academy of Sciences 113, no. 39 (2016): 10750–58. http://dx.doi.org/10.1073/pnas.1608077113.

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Prevention is an essential component of cancer eradication. Next-generation sequencing of cancer genomes and epigenomes has defined large numbers of driver mutations and molecular subgroups, leading to therapeutic advances. By comparison, there is a relative paucity of such knowledge in premalignant neoplasia, which inherently limits the potential to develop precision prevention strategies. Studies on the interplay between germ-line and somatic events have elucidated genetic processes underlying premalignant progression and preventive targets. Emerging data hint at the immune system’s ability

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7

Nemakayala, Divyesh Reddy, Shilpa Tatineni, Ikponmwosa Enofe, Ling Wang, and Heather Laird-Fick. "Prevalence of gastric cancer and premalignant changes in a Michigan cohort, 2013-2017." Journal of Clinical Oncology 37, no. 15_suppl (2019): e15539-e15539. http://dx.doi.org/10.1200/jco.2019.37.15_suppl.e15539.

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e15539 Background: Gastric cancer (GC) is uncommon in the US. Incidence varies by geography, race/ethnicity, sex, age, and socioeconomic status (SES). H. pylori is a risk factor for premalignant lesions and GC. Prior studies estimate risk of progression from premalignant to GC at 0.1-0.5% per year. US prevalence of premalignant lesions is unclear. This study describes the prevalence of premalignant lesions and GC in a large community-based sample. Methods: Patients aged ≥18 years undergoing EGD with gastric biopsy in Sparrow Health System were identified. Pathology reports were abstracted for

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8

Zhou, Xian, Jianhao Zeng, Alexys T. Riddick, Victor H. Engelhard, and Hui Zong. "Premalignancy induced B cell-centered immune aggregates for immune-prevention against triple negative breast cancer." Journal of Immunology 210, no. 1_Supplement (2023): 88.10. http://dx.doi.org/10.4049/jimmunol.210.supp.88.10.

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Abstract Immune surveillance is believed to be a critical mechanism to eradicate premalignant mutant cells to prevent cancer. However, the cellular organization and mechanisms for immune surveillance remain largely unknown because premalignant mutant cells are “invisible” in patient samples and conventional animal models. To gain a glimpse into this process, our lab developed a genetically engineered mouse model recapitulating human basal-like breast cancer through unequivocal labeling of premalignant p53-Brca1 mutant cells with GFP. Using this model, we discovered prominent immune aggregates

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9

Tatineni, Shilpa, Divyesh Reddy Nemakayala, Ikponmwosa Enofe, Ling Wang, and Heather Laird-Fick. "Prevalence of esophageal malignant and premalignant lesions in a Michigan cohort, 2013-2017." Journal of Clinical Oncology 37, no. 15_suppl (2019): e15541-e15541. http://dx.doi.org/10.1200/jco.2019.37.15_suppl.e15541.

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e15541 Background: Esophageal cancer is diagnosed in roughly 4 per 100,000 US population. Older men are most frequently affected. Adenocarcinoma is most common and incidence is increasing. EGD with biopsy is important for diagnosis. Many patients present with metastases, limiting treatment options. EGD can identify Barrett’s esophagus, a precursor lesion for adenocarcinoma, but evaluation of biopsy specimens is difficult. This study describes findings from esophageal biopsies in a large community-based Michigan cohort. Methods: Patients aged ≥18 years undergoing EGD with esophageal biopsies in

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10

Huber, Michaell A. "PREMALIGNANT LESIONS." Journal of the American Dental Association 139, no. 4 (2008): 395–96. http://dx.doi.org/10.14219/jada.archive.2008.0172.

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11

TEJADA-BERGES, TREVOR. "Premalignant Lesions." Clinical Obstetrics and Gynecology 54, no. 1 (2011): 133–40. http://dx.doi.org/10.1097/grf.0b013e31820837d1.

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12

Siregar, Gontar Alamsyah, Ida Parwati, Tri Hanggono Achmad, and Yoni Fuadah Syukriani. "Association between VEGF-634G>C Gene Polymorphism with Gastric Premalignant Lesions and Serum VEGF Levels in Helicobacter pylori Gastritis Patients." Open Access Macedonian Journal of Medical Sciences 6, no. 8 (2018): 1328–34. http://dx.doi.org/10.3889/oamjms.2018.266.

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AIM: To evaluate the association between VEGF-634G>C gene polymorphism with premalignant gastric lesions as well as the level of VEGF.METHODS: This cross-sectional study included patients with H. pylori gastritis at Haji Adam Malik General Hospital, Permata Bunda General Hospital, and Universitas Sumatera Utara Hospital, Medan, Indonesia. Detection of H. pylori infection was made using positive results of 14C-UBT, rapid urease test, and/or immunohistochemistry. Gastric premalignant lesion diagnosis was made when one or more of the following were present: chronic atrophic gastritis, intestin

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13

Scrobota, Ioana, Grigore Baciut, Roxana Buzatu, et al. "Oxidative Stress Markers in Chemically Induced Oral Premalignant Lesions." Revista de Chimie 68, no. 6 (2017): 1211–15. http://dx.doi.org/10.37358/rc.17.6.5643.

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In our study we induced oral premalignant lesions using 4 nitro-quinoline-1-oxide (4NQO) topically applied on oral mucosa for 24 weeks. We detected an oxidative stress marker malondialdehyde (MDA) significantly increased (p[0.05) and an antioxidant/detoxifying activity marker, glutathione (GSH), significantly decreased (p[0.05) in the oral induced epithelial dysplasias. We appreciated that issue MDA and GSH could serve as diagnostic, screening and evaluation of response to therapy tools in oral premalignacy.

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14

Jani, Chinmay T., Arpita Joshi, Khushboo Jain, and D. V. Bala. "EPR19-068: Association of Oral Premalignant Lesions with the Consumption of Tobacco." Journal of the National Comprehensive Cancer Network 17, no. 3.5 (2019): EPR19–068. http://dx.doi.org/10.6004/jnccn.2018.7261.

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Introduction: Tobacco use is a major public health problem globally. According to the WHO, tobacco is the second most important cause of death in the world. Tobacco is used in different forms: smoke and smokeless. In India, people also consume beetle nut along with tobacco, causing more harmful effects. The present study was done to determine prevalence of tobacco use in industrial workers and associate it with the prevalence of oral premalignant lesions. Methodology: A cross-sectional observational study of 909 industrial workers working in 4 industries was undertaken from March 2014 to Octob

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15

Sadri, Maziar, Jeremy McMahon, and Andrew Parker. "Laryngeal dysplasia: aetiology and molecular biology." Journal of Laryngology & Otology 120, no. 3 (2005): 170–77. http://dx.doi.org/10.1017/s0022215105005360.

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Laryngeal premalignancy is a common clinical concern. While tobacco has long been established as the principal identifiable aetiological factor, the last two decades have seen publications investigating a potential role for gastroesophageal reflux, gastrectomy and human papilloma virus. Furthermore, there have been major advances in our understanding of the molecular biology of cancer and premalignancy. Accompanying this increased understanding, significant efforts have been made to correlate the expression of molecular markers with the clinical course of premalignant laryngeal lesions.This re

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16

Yadav, Manisha, Vipin Arora, Showket Hussain, et al. "Evaluation of extracellular signal regulated kinase 1 expression in premalignant lesions of oral cavity." International Surgery Journal 7, no. 12 (2020): 3965. http://dx.doi.org/10.18203/2349-2902.isj20204978.

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Background: Most oral squamous cell carcinomas are preceded by premalignant lesions like leukoplakia, erythroplakia etc. Identification of these lesions by molecular markers and intervention at an early stage can reduce mortality, morbidity and cost of treatment associated with oral cancer. Extracellular signal regulated kinase 1 (ERK 1) is one of the various markers involved in carcinogenesis.Methods: We studied the expression of ERK 1 in twenty-five oral premalignant lesion biopsies using immunohistochemistry (IHC) and western blot analysis.Results: Ninety-two percent of premalignant lesions

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17

Ziada, Mahmoud, Christopher B. Bunker, and Asif Muneer. "Premalignant penile lesions." Journal of Clinical Urology 9, no. 4 (2016): 216–22. http://dx.doi.org/10.1177/2051415816649822.

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18

William, William N. "Oral premalignant lesions." Current Opinion in Oncology 24, no. 3 (2012): 205–10. http://dx.doi.org/10.1097/cco.0b013e32835091bd.

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19

Acheson, N., R. Ganesan, and K. K. Chan. "Premalignant vulvar disorders." Current Obstetrics & Gynaecology 10, no. 1 (2000): 12–17. http://dx.doi.org/10.1054/cuog.2000.0057.

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20

Kirwan, J. M., and J. J. O. Herod. "Premalignant vulval disorders." Current Obstetrics & Gynaecology 12, no. 2 (2002): 90–96. http://dx.doi.org/10.1054/cuog.2001.0240.

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21

Schwartz, Robert A. "Premalignant keratinocytic neoplasms." Journal of the American Academy of Dermatology 35, no. 2 (1996): 223–42. http://dx.doi.org/10.1016/s0190-9622(96)90329-1.

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22

Iyengar, Sucheta, and Nigel Acheson. "Premalignant vulval conditions." Obstetrics, Gynaecology & Reproductive Medicine 18, no. 3 (2008): 60–63. http://dx.doi.org/10.1016/j.ogrm.2008.01.002.

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23

Scully, Crispian. "PD1. Premalignant lesions." Oral Oncology 47 (July 2011): S5. http://dx.doi.org/10.1016/j.oraloncology.2011.06.031.

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24

Amagasa, Teruo. "Oral premalignant lesions." International Journal of Clinical Oncology 16, no. 1 (2011): 1–4. http://dx.doi.org/10.1007/s10147-010-0158-2.

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25

Smith, Chandler. "Premalignant lesion terminology." Human Pathology 16, no. 7 (1985): 756. http://dx.doi.org/10.1016/s0046-8177(85)80167-2.

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26

Jordan, J. A. "Cervical premalignant disease." Current Obstetrics & Gynaecology 1, no. 3 (1991): 123. http://dx.doi.org/10.1016/0957-5847(91)90059-7.

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27

Dr., Indushree D. "Oral Premalignant Lesions." International Journal of Innovative Science and Research Technology 8, no. 1 (2023): 1206–15. https://doi.org/10.5281/zenodo.7605151.

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This paper provides an general overview of oral premalignant lesions and conditions , reasons and causes behind this premalignant lesions and conditions and how to identify these conditions and differentiate them from other non cancerous lesions and this paper also provides information on early signs and Symptoms along with signs and symptoms during the later stages and complications associated and also provides information on etiopathogenesis and as of how to diagnose these lesions clinically and by different laboratory tests and also gives an overview on management a of these lesions.

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28

Swain, Santosh Kumar. "Premalignant lesions of the oral cavity: current perspectives." International Journal of Research in Medical Sciences 9, no. 6 (2021): 1816. http://dx.doi.org/10.18203/2320-6012.ijrms20212259.

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Premalignant lesions of the oral cavity are also called as potentially malignant disorders. The common premalignant lesions of the oral cavity include leukoplakia, oral submucous fibrosis (OSMF) and oral erythroplakia. These lesions have a very high malignant transformation rate. The exact etiological factors for development of the premalignant lesions of the oral cavity are not fully understood till date. Use of tobacco, alcohol drinking, chewing betel quid containing areca nut and solar rays are important etiological factors for resulting the premalignant lesions of the oral cavity. Early di

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29

Budak, Emine, and Ahmet Yanarateş. "PET/CT Parameters are Useful in Discrimination of Incidental Benign, Premalignant and Malignant Colonic Lesions." Nuklearmedizin 59, no. 03 (2020): 235–40. http://dx.doi.org/10.1055/a-1084-4723.

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Abstract Aim The present study evaluates the role of PET parameters pertaining to incidentally detected foci of colorectal uptake (IFCU) on FDG PET/CT in the differentiation of benign, premalignant and malignant lesions. Methods This retrospective study included 74 patients. The colonoscopic and histopathological findings were regarded as the reference standards. The results were evaluated on a lesion basis by dividing the lesions into three groups as benign, premalignant and malignant. The maximum standardized uptake value (SUVmax), SUVmean, metabolic tumor volume(MTV) and total lesion glycol

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30

x, Shachita, and Shivbrat Sharma. "Oral Leukoplakia: A Premalignant Condition." International Journal of Science and Research (IJSR) 11, no. 1 (2022): 13–14. http://dx.doi.org/10.21275/sr211229111455.

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31

Clark, Anna-Maria, and M. Rita Young. "Administration of a vaccine composed of dendritic cells pulsed with premalignant oral lesion lysate results in an increase in CD4+ and CD8+ T cells and a decrease in Foxp3+ cells in tongues of mice bearing carcinogen-induced premalignant oral lesions. (131.24)." Journal of Immunology 184, no. 1_Supplement (2010): 131.24. http://dx.doi.org/10.4049/jimmunol.184.supp.131.24.

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Abstract Most studies with dendritic cell vaccines have focused on eliminating or preventing progression of established malignancies. This is complicated by the immune suppression often exhibited in the presence of solid carcinomas. The goal of this study is to determine if an immune response can be elicited by administering a DC vaccine during the premalignant stages of oral squamous cell carcinoma, prior to development of systemic immune suppression. Mice treated with the carcinogen 4NQO in drinking water develop premalignant oral lesions which progress to OSCC. As previous studies in our la

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32

Namasivayam, Vikneswaran, Calvin J. Koh, Stephen Tsao, et al. "Academy of Medicine, Singapore clinical guideline on endoscopic surveillance and management of gastric premalignant lesions." Annals of the Academy of Medicine, Singapore 51, no. 7 (2022): 417–35. http://dx.doi.org/10.47102/annals-acadmedsg.2021433.

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Gastric cancer (GC) has a good prognosis, if detected at an early stage. The intestinal subtype of GC follows a stepwise progression to carcinoma, which is treatable with early detection and intervention using high-quality endoscopy. Premalignant lesions and gastric epithelial polyps are commonly encountered in clinical practice. Surveillance of patients with premalignant gastric lesions may aid in early diagnosis of GC, and thus improve chances of survival. An expert professional workgroup was formed to summarise the current evidence and provide recommendations on the management of patients w

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33

De Costa, Anna-Maria, David Walker, Corinne Schuyler, and Rita Young. "Administration of a vaccine composed of dendritic cells pulsed with premalignant oral lesion lysate stimulates Th1 and Th17 immunity in mice bearing carcinogen-induced premalignant oral lesions. (165.13)." Journal of Immunology 186, no. 1_Supplement (2011): 165.13. http://dx.doi.org/10.4049/jimmunol.186.supp.165.13.

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Abstract The use of dendritic cell (DC) vaccines as treatment for malignancy is complicated by the immune suppression often exhibited in the presence of solid carcinomas such as head and neck squamous cell carcinoma (HNSCC). The goal of this study is to determine if an immune response can be elicited by administering a DC vaccine during the premalignant stages of HNSCC, prior to development of immune suppression. Mice treated with the carcinogen 4NQO in drinking water develop premalignant oral lesions which progress to HNSCC. As previous studies demonstrated that premalignant lesions and HNSCC

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34

Khachemoune, Amor, Hui Yu Juan, and Rohan Singh. "Shedding new light on actinic keratoses and squamous cell carcinoma in situ." JAAPA 37, no. 1 (2024): 1–6. http://dx.doi.org/10.1097/01.jaa.0000995696.23729.37.

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ABSTRACT Cutaneous squamous cell carcinoma can arise from various premalignant lesions such as actinic keratosis, Bowen disease, and premalignant genital squamous cell lesions. Identification and treatment can prevent malignant transformation and death. This article describes the causes, epidemiology, and characteristics of suspicious premalignant squamous cell lesions so that clinicians can identify these lesions and refer patients for specialist treatment as appropriate.

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Jaber, M. A., P. Diz Dios, E. Vazquez García, A. Cutando Soriano, and S. R. Porter. "Spanish dental students knowledge of oral malignancy and premalignancy." European Journal of Dental Education 1, no. 4 (1997): 167–71. http://dx.doi.org/10.1111/j.1600-0579.1997.tb00093.x.

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Background: In view of the gradual rise in oral malignancy in Europe, there is an increased need for undergraduate dental students to have some appropriate training in the recognition of the signs and symptoms of oral premalignancy and malignancy, and be aware of the appropriate early management of patients with such oral lesions. The present study outlines the results of a pilot study to determine the knowledge and awareness of a cohort of undergraduate dental students in Spain regarding the management of oral premalignancy and malignancy.Method: In March 1997 a self‐administered questionnair

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Thompson, Henry J., John N. McGinley, Pamela Wolfe, Nicole S. Spoelstra, and Katrina K. Knott. "Targeting Angiogenesis for Mammary Cancer Prevention: Factors to Consider in Experimental Design and Analysis." Cancer Epidemiology, Biomarkers & Prevention 13, no. 7 (2004): 1173–84. http://dx.doi.org/10.1158/1055-9965.1173.13.7.

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Abstract An experimental model developed to investigate premalignant stages of breast cancer was used to establish a rationale for designing experiments that target angiogenesis for cancer prevention. Blood vessels were identified via CD31 immunostaining, and all vessels that occurred in a 50 μm wide region circumscribing each pathology were counted using a digital imaging technique. The blood vessel density associated with terminal end buds was unaffected by carcinogen treatment, whereas vessel density was higher in intraductal proliferations and ductal carcinoma in situ than in terminal end

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37

Negovan, Anca, Mihaela Iancu, Valeriu Moldovan, Simona Mocan, and Claudia Banescu. "The Interaction between GSTT1, GSTM1, and GSTP1 Ile105Val Gene Polymorphisms and Environmental Risk Factors in Premalignant Gastric Lesions Risk." BioMed Research International 2017 (2017): 1–9. http://dx.doi.org/10.1155/2017/7365080.

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The study investigated the possible influence of GSTM1, GSTT1, and GSTP1 gene polymorphisms as predisposing factors for premalignant gastric lesions as well as their interaction with H. pylori infection, gastrotoxic drugs, smoking, and alcohol consumption. In this study, 270 patients with a complet set of gastric biopsies and successfully genotyped were finally included. The GSTM1 gene polymorphism had significant contribution in mild/severe endoscopic lesions (p=0.01) as well as in premalignant lesions (p=0.01). The GSTM1 null genotype increased the risk for mucosal defects in H. pylori-negat

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38

Beg, Ayesha, Mohammad Aminul Islam, Nibedita Roy, et al. "Cytological and colposcopic evaluation of abnormal cervix." Community Based Medical Journal 8, no. 1 (2019): 3–11. http://dx.doi.org/10.3329/cbmj.v8i1.55663.

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The second most common cancer among women worldwide is cancer of the cervix commonly develop due to abnormal cervix. To evaluate abnormal cervix by cytology and colposcopy, a prospective study was undertaken among 100 women attending at the colposcopy clinic in Mymensingh Medical College Hospital (MMCH) from January 2008 to July 2008. Cytological and colposcopic evaluation were done in all the patients attending the colposcopy clinic with various complaints like per vaginal discharge, post-coital bleeding, dyspareunia, irregular per vaginal bleeding, backache and abnormal cervix on examination

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Wang, Guangda, Liang Liu, Yang Zhao, Yan Lin, and Limian Er. "Comparative genomic analysis unveiling the mutational landscape associated with premalignant lesions and early-stage gastric cardia cancer." Medicine 104, no. 2 (2025): e40332. https://doi.org/10.1097/md.0000000000040332.

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This study enrolled 10 patients diagnosed with premalignant lesions and early-stage gastric cardia adenocarcinoma (GCA), confirmed through endoscopic examination. These patients were subjected to next-generation sequencing (NGS) using a customized 1123-gene panel to identify genetic alterations and signaling pathways. The results were compared to stage IIB to IV GCA samples from the cancer genome atlas (TCGA) and a cohort of Hong Kong patients. The study provides insights into the molecular drivers of GCA progression, with potential therapeutic implications. A total of 10 patients diagnosed wi

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Goswami, Arunima, Bharadwaj Bordoloi, Pinky Goswami, and Aanchal Tandon. "Efficacy of Photodynamic Therapy in the Treatment of Oral Premalignant Lesions: A Systematic Review." Journal of Primary Care Dentistry and Oral Health 4, no. 3 (2023): 64–70. http://dx.doi.org/10.4103/jpcdoh.jpcdoh_18_23.

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Abstract Photodynamic therapy (PDT) is considered a contemporary effective treatment option for oral premalignant lesions. This systematic review aims to evaluate the usefulness of PDT for the treatment of oral premalignant lesions. The focused question was “Is PDT effective in the treatment of oral premalignant lesions?” A literature search was made in PubMed/Medline and Google Scholar databases using different combinations of the following keywords: photodynamic therapy, oral premalignant lesions, oral leukoplakia (OL), erythroplakia, oral erythroleukoplakia (OEL), oral verrucous hyperplasia

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Porukala, Manisri, and P. K. Vinod. "Gene expression signatures of stepwise progression of Hepatocellular Carcinoma." PLOS ONE 18, no. 12 (2023): e0296454. http://dx.doi.org/10.1371/journal.pone.0296454.

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The molecular pathogenesis of Hepatocellular Carcinoma (HCC) is a complex process progressing from premalignant stages to cancer in a stepwise manner. Mostly, HCC is detected at advanced stages, leading to high mortality rates. Hence, characterising the molecular underpinnings of HCC from normal to cancer state through precancerous state may help in early detection and improve its prognosis and treatment. In this work, we analysed the transcriptomic profile of tumour and premalignant samples from HCC or chronic liver disease patients, who had undergone either total or partial hepatectomy. The

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Ohtomo, Kuni. "CIRRHOSIS AND PREMALIGNANT NODULES." Magnetic Resonance Imaging Clinics of North America 5, no. 2 (1997): 331–46. http://dx.doi.org/10.1016/s1064-9689(21)00199-9.

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Vora, RitaVipul, RochitRajesh Singhal, TrushaManoj Patel, and KiraAntony Pariath. "Premalignant male genital dermatoses." Indian Journal of Sexually Transmitted Diseases and AIDS 40, no. 2 (2019): 97. http://dx.doi.org/10.4103/ijstd.ijstd_106_17.

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Curtius, Kit, Nicholas A. Wright, and Trevor A. Graham. "Evolution of Premalignant Disease." Cold Spring Harbor Perspectives in Medicine 7, no. 12 (2017): a026542. http://dx.doi.org/10.1101/cshperspect.a026542.

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Epstein, Joel, Meir Gorsky, and Sharon Elad. "PREMALIGNANT LESIONS: Authors' response." Journal of the American Dental Association 139, no. 4 (2008): 396–98. http://dx.doi.org/10.14219/jada.archive.2008.0171.

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Davison, James M., and Jerry J. Marty. "Detecting Premalignant Cervical Lesions." Obstetrical & Gynecological Survey 49, no. 9 (1994): 627. http://dx.doi.org/10.1097/00006254-199409000-00019.

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Scully, C. "K-7 Premalignant lesions." Oral Oncology Supplement 1, no. 1 (2005): 29. http://dx.doi.org/10.1016/s1744-7895(05)80010-0.

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Horvai, Andrew, and K. Krishnan Unni. "Premalignant conditions of bone." Journal of Orthopaedic Science 11, no. 4 (2006): 412–23. http://dx.doi.org/10.1007/s00776-006-1037-6.

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Nagorney, David M. "Are Hepatic Adenomas Premalignant?" HPB Surgery 10, no. 1 (1996): 59–61. http://dx.doi.org/10.1155/1996/59629.

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Objective: To investigate clinical experience with the apparent malignant transformation of benign liver cell adenomas.Design: Retrospective review of personal experience and literature.Setting: University hospital and affiliated community hospitals.Patients: All patients diagnosed with liver cell adenomas over a 30-year period.Interventions: Liver resection and/or tumor biopsy.Main Outcome Measures: Gender, age, drug associations, alpha-fetoprotein levels, response to treatment, and survival.Results: Thirteen patients from personal experience and 26 patients from the reports of others had liv

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Collado, Manuel, Jesús Gil, Alejo Efeyan, et al. "Senescence in premalignant tumours." Nature 436, no. 7051 (2005): 642. http://dx.doi.org/10.1038/436642a.

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