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Готові списки джерел за темами / Shoulder muscle / Книги

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Книги з теми "Shoulder muscle"

Автор: Grafiati

Опубліковано: 10 січня 2023

Оновлено: 28 січня 2023

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1

Taoru 1-pon de ganko na katakori yōtsū o naosu: "shinsōkin: gyaku-sutoretchi kenkōhō. Tōkyō: PHP Kenkyūjo, 2011.

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2

Nakatsuji, Tadashi. 1 tiao mao jin 2 fen zhong jie tong: Ni shen zhan song ji mao jin cao : ji rou suo bi shen geng neng shu zhan! Xinbei Shi Zhonghe Qu: Ping guo wu chu ban she you xian gong si, 2015.

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3

American Academy of Orthopaedic Surgeons, ed. Disorders of the proximal biceps tendon: Evaluation and treatment. Rosemont, IL: AAOS, American Academy of Orthopaedic Surgeons, 2014.

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4

7 weeks to 50 pull-ups: Strengthen and sculpt your arms, shoulders, back, and abs by training to do 50 consecutive pull-ups. Berkeley, CA: Ulysses Press, 2011.

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5

Russell, Belinda. A study using surface electromyography to compare the activity of shoulder muscles used by individuals with paraplegia during wheelchair transfers with and without a transfer board. Oxford: Oxford Brookes University, 2002.

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6

Reliability of peak torque values for shoulder internal and external rotation during eccentric and concentric loading using the Kin-Com 125E isokinetic dynamometer. 1991.

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7

Reliability of peak torque values for shoulder internal and external rotation during eccentric and concentric loading using the Kin-Com 125E isokinetic dynamometer. 1992.

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8

Muscle force velocity adaptations to variations in long term physical training. 1988.

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9

Muscle force velocity adaptations to variations in long term physical training. 1985.

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10

Redhead, Lucy. The role of selected muscle activity in idiopathic shoulder instability. 2004.

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11

An isokinetic shoulder profile of collegiate baseball pitchers and its relation to throwing velocity. 1991.

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12

An isokinetic shoulder profile of collegiate baseball pitchers and its relation to throwing velocity. 1991.

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13

An isokinetic shoulder profile of collegiate baseball pitchers and its relation to throwing velocity. 1989.

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14

Eccentric peak torque and maximal repetition work percentages of the dominant external rotators in College Division I baseball players. 1991.

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15

Eccentric peak torque and maximal repetition work percentages of the dominant external rotators in college Division I baseball players. 1992.

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16

The effects of two types of overload training on upper body strength development in females. 1989.

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17

The effects of two types of overload training on upper body strength development in females. 1989.

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18

The effects of two types of overload training on upper body strength development in females. 1989.

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19

The reliability of an isokinetic measurement protocol for the posterior rotator cuff musculature. 1990.

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20

Strength gains through aquatic exercise. 1992.

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21

An isokinetic shoulder profile of collegiate baseball pitchers and its relation to throwing velocity. 1991.

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22

Cohen, Jeffrey A., Justin J. Mowchun, Victoria H. Lawson, and Nathaniel M. Robbins. A 61-Year-Old Male with Severe Shoulder and Cervical Pain. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190491901.003.0007.

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Анотація:

Severe shoulder pain in the absence of a clear orthopedic cause may be due to acute brachial plexitis. Numbness and tingling in association with weakness and muscle atrophy that cannot be accounted for by a single nerve or nerve root distribution suggests the diagnosis. Additional clues suggesting brachial plexitis include intensity of shoulder pain and antecedent events such as illness, vaccination, injury, unusual physical activity or surgery. The approach to diagnosis of plexitis/plexopathy and appropriate evaluation for etiology are discussed. Management of this condition is conservative, relating to pain control and judicious use of mobilization and strengthening with physical therapy. Prognosis is generally good with recovery of strength occurring in weeks to months.

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23

The effects of two selected training programs on the strength, flexibility, and ratio of internal to external rotation of the rotator cuff. 1992.

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24

The effects of two selected training programs on the strength, flexibility, and ratio of internal to external rotation of the rotator cuff. 1992.

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25

Bowker, Lesley K., James D. Price, Ku Shah, and Sarah C. Smith. Musculoskeletal system. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198738381.003.0017.

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This chapter provides information on osteoarthritis, management of osteoarthritis, osteoporosis, management of osteoporosis, polymyalgia rheumatica, giant cell arteritis, muscle symptoms, Paget’s disease, gout, pseudogout, contractures, cervical spondylosis and myelopathy, osteomyelitis, the elderly foot, the elderly hand, the painful hip, the painful back, and the painful shoulder.

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26

Isokinetic evaluation of the posterior rotator cuff musculature following a strengthening program utilizing rubber tubing. 1992.

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27

Rockhard, Gavin. Muscle Trade: The Swedish Smith's Shoulders. Independently Published, 2018.

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28

Wood, Alix. Heads, Shoulders, Muscles, and Bones. Rosen Publishing Group, 2022.

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29

Heads, Shoulders, Muscles, and Bones. Rosen Publishing Group, 2022.

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30

Wood, Alix. Heads, Shoulders, Muscles, and Bones. Rosen Publishing Group, 2022.

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31

Laurence, M. How To Build Massive Shoulders: 6 Week Workout for Huge Shoulders, Shocking the Muscles into Growth, Building Massive Traps, Build Huge Shoulders, 20 ... Muscle Building. CreateSpace Independent Publishing Platform, 2017.

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32

Shaibani, Aziz. Muscle Twitching. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190661304.003.0019.

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Анотація:

Muscle twitching is an involuntary movement of the muscles that is usually focal and transient. Patients confuse it with restlessness of the legs and jerking of the extremities unless they are specifically asked. Tremor is also commonly mixed with twitching, but its regular nature should be noticed. Fasciculation and rippling are the most important neuromuscular causes of twitching. Reproduction of the symptoms in the clinic, if possible, is very useful for the diagnosis. Otherwise, a video showing these twitching is equally sufficient to provide evidence of the condition.

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33

Shaibani, Aziz. Muscle Twitching. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199898152.003.0019.

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Анотація:

Muscle twitching is a pianless involuntary movement of muscles, usually focal short lived. Patients may confuse it with restlessness of the legs and jerking of extremities unless specifically asked. Tremor,especially of the tongue, is also commonly confused with twitching, but its regular nature should be noticed. Fasciculations and rippling are the most important neuromuscular causes of twitching. Reproduction of the symptoms in the clinic, if possible, is very useful for the diagnosis. Otherwise, a video taken by the patient or family members showing these twitchings is equally good. Fasciculations may be enhanced by tapping the affected muscle group and hyperventilation. Surprisingly, EMG evidence of fasciculations may be scarce despite their clinical predominance.

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34

Shaibani, Aziz. Muscle Atrophy and Hypertrophy. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190661304.003.0017.

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Muscle atrophy is usually caused by interruption of axonal flow [axonal neuropathies, motor neuron diseases (MNDs), etc.]. If weakness is out of proportion to atrophy, demyelinating neuropathy should be suspected. Chronic myopathies and immobility also may cause atrophy, but no electromyography (EMG) evidence of denervation or myopathy is found. The pattern of atrophy is often helpful to localize the lesions. Atrophy of the interossi and preservation of the bulk of the thenar muscles suggest ulnar neuropathy, but atrophy of both would suggest a C8 or plexus pathology. Muscle enlargement may be due to fatty replacement, which can be confirmed by EMG and magnetic resonance imaging (MRI), or due to real muscle hypertrophy from excessive discharges (neuromyotonia).

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35

Shaibani, Aziz. Muscle Atrophy and Hypertrophy. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199898152.003.0017.

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Muscle atrophy is usually caused by interruption of axonal flow (axonal neuropathies, motor neuron diseases, etc.). If weakness is out of proportion to atrophy, conduction block due to demyelinating neuropathy should be suspected. Chronic myopathies and immobility may also cause atrophy, but no EMG evidence of denervation or myopathy is respectively found. The pattern of atrophy is often helpful to localize the lesion. Atrophy of the interossi and preservation of the bulk of the thenar muscles suggest ulnar neuropathy, but atrophy of both would suggest a C8 or plexus pathology. Muscle enlargement may be due to tissue replacement (fatt, amyloid), which can be confirmed by EMG and MRI, or may be due to real muscle hypertrophy from excessive discharges (neuromyotonia).

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36

Shaibani, Aziz. Distal Arm Weakness. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199898152.003.0015.

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Distal arm weakness may be caused by involvement of the intrinsic hand muscles (interossi, thenar and hypothenar muscles, lumbricals) or extrinsic hand muscles (long finger flexors and extensors). ALS is typical for the former type, and IBM is typical for the latter type. Incoordination of skilled finger movement due to cerebellar disease is associated with normal strength. Poor mobility due to joints pain and swelling should not be confused with muscle weakness. Mononeuropathies such as ulnar, radial, median, and AIN,lesions usually cause differential loss of function. Myasthenia sometimes causes weakness of the wrist and finger extensors. A small but distinct group of distal hereditary myopathies should always be borne in mind. Progressive sensorimotor neuropathies are usually associated with sensory symptoms. Multifocal motor neuropathy can be a diagnostic challenge.

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37

The physiological effects observed from aerobic dance training performed with light-resistance wrist weights. 1985.

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38

The physiological effects observed from aerobic dance training performed with light-resistance wrist weights. 1987.

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39

Shaibani, Aziz. Distal Arm Weakness. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190661304.003.0015.

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Distal arm weakness may be caused by involvement of the intrinsic hand muscles (interossi, thenar and hypothenar muscles, lumbricals) or extrinsic hands muscles (long fingers flexors and extensors). Amyotrophic lateral sclerosis (ALS) is typical of the former type, and inclusion body myositis (IBM) is typical for the later type. Incoordination of skilled finger movement due to cerebellar disease is associated with normal strength. Poor mobility due to joint pain and swelling should not be confused with muscle weakness. Mononeuropathies such as ulnar, radial, median, and anterior interosseus nerve lesions usually cause differential loss of function. Myasthenia gravis sometimes causes weakness of the wrist and finger extensors. A small but distinct group of distal hereditary myopathies should always be kept in mind. Progressive sensorimotor neuropathies are usually associated with sensory symptoms. Multifocal motor neuropathy can be a challenging diagnosis.

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40

Shaibani, Aziz. Quadriceps Weakness. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199898152.003.0014.

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Quadriceps muscles extend the knees and are important for walking and balance. Knee buckling is the most common presentation of quadriceps weakness. Knee buckling is common in the elderly as it can also be caused by knee arthritis. Patients with quadriceps weakness often modify their lifestyle for years before they seek medical advice. Quadriceps muscles are very sensitive to immobility and they may lose 50% of their bulk within 2 weeks of immobility. On the other hand, they build mass quickly by exercises. Sometimes, quadriceps muscles are selectively and severely involved, leading to an early disability. Severe thigh pain, if continued for a few weeks, may lead to disuse atrophy. Severe neuropathic thigh pain and atrophy are typically seen in diabetic amyotrophy. Other causes of thigh pain include L3 radiculopathy, meralgia paresthetica, and muscle infarction. Examination of the knee extension should never be deleted from neuromuscular evaluation.

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41

Shaibani, Aziz. Quadriceps Weakness. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190661304.003.0014.

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Анотація:

Quadriceps muscles extend the knees and are important for walking and balance. Knee buckling (sudden giving away of the knees) is the most common presentation of quadriceps weakness. Knee buckling is common in the elderly, as it can also be caused by knee arthritis. Patients with quadriceps weakness often modify their lifestyle for years before they seek medical advice. Quadriceps muscles are very sensitive to immobility, and they may lose half their bulk within 2 weeks. On the other hand, they build mass quickly with exercise. Sometimes quadriceps muscles are selectively and severely involved, leading to early disability. Severe thigh pain, if it continues for a few weeks, may lead to disuse atrophy. Severe neuropathic thigh pain and atrophy are typically seen in diabetic amyotrophy. Other causes of thigh pain include L3 radiculopathy, meralgia paresthetica, and muscle infarction. Examination of the knee extension should always be part of neuromuscular evaluation.

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42

Gowda, Rangnath. How to Fix Muscle Imbalance: Muscle Imbalance Correction Guide for Uneven Pecs, Biceps, Traps and Lats, Uneven Shoulders, Rolled or Rounded Shoulders, Pelvic Lateral Tilt, Uneven Waist or Twisted Core. Independently Published, 2021.

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43

Shaibani, Aziz. Myotonia. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190661304.003.0021.

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Анотація:

Myotonia is a slow relaxation phase after normal contraction. Patients report dystonia as muscle stiffness and sometimes pain. They usually adapt to it well. Falls due to myotonia may lead to accidents. Examination for percussion myotonia should be part of neuromuscular examination. Percussion of the thenar muscles with the reflex hammer is the most productive method. Electrically silent myotonia is a sign of Brody myopathy. Myotonia may be incidentally discovered during electromyography (EMG). The most important task is to differentiate between myotonia from paramyotonia clinically and electrically. There has been a significant understanding of the underlying channelopathies lately. Severe myotonia respond well to mexiletine.

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44

Pollard, Brian J. Muscle relaxants in critical illness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0047.

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The place of neuromuscular blocking agents in the intensive care unit (ICU) has changed markedly over the last 20 years. Originally regarded as a mainstay of the process of ‘sedation’, they are now only used for specific indications. The principal disadvantage is probably the difficulty in neurological assessment when a muscle relaxant is used coupled with the increased risk of awareness, because inadequate sedation will be masked. Of the available agents, the intermediate acting ones are the most popular. The degree of relaxation can be readily controlled and they have few side effects. In the presence of renal and/or hepatic disease atracurium or cisatracurium are preferred. Succinylcholine is only used for securing the airway due to its very rapid onset of action. Rocuronium given in a higher dose also possesses a rapid onset in situations when succinylcholine might be contraindicated. When using a muscle relaxant, its effect should always be monitored with a simple train of four pattern of stimulation from a hand-held nerve stimulator. This will ensure that an adequate and not excessive block is secured. If a more rapid reversal is required then a dose of neostigmine with glycopyrrolate may be used. Alternatively, if rocuronium is the relaxant in use then the new agent sugammadex is effective.

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45

Lahouti, Arash H., and Lisa Christopher-Stine. Toxic myopathies. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0009.

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Анотація:

Toxic myopathy symptoms range from myalgia and muscle cramps to severe weakness, bearing similarities to a number of other muscle conditions. Thus, when evaluating patients with muscle symptoms, an iatrogenic muscle problem should always be considered, to be able to distinguish a toxic from any other myopathy early on, preventing further muscle damage and to potentially reverse muscle injury by withdrawal of the toxic agent. Various commonly prescribed medications, as well as illicit drugs, may cause muscle damage. These substances may cause muscle injury through direct myotoxic effects, or indirectly through various mechanisms, such as electrolyte abnormalities and triggering, or disinhibiting the immune system response.

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46

Stewart, Brett. 7 Weeks to 50 Pull-Ups: Strengthen and Sculpt Your Arms, Shoulders, Back, and Abs by Training to Do 50 Consecutive Pull-Ups. Ulysses Press, 2011.

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47

Mammen, Andrew L., and Jessica R. Nance. Evaluation of hyperCKaemia. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0007.

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Анотація:

Serum creatine kinase (CK) levels may be elevated in patients with muscle weakness or pain. In asymptomatic patients with CK elevations, the focus should be on identifying reversible causes, followed by investigation for inherited muscle diseases. In asymptomatic patients with an incidental finding of elevated CK, clinicians should look for reversible causes, then re-test the CK after 10 days of rest in the absence of potential triggers. If the CK remains markedly elevated and/or electromyography proves myopathic, a muscle biopsy should be considered. Women of childbearing age with elevation of serum CK should be evaluated for dystrophin mutation. Genetic causes of hyperCKaemia can be pursued with targeted gene sequencing, or whole exome or next generation sequencing. Patients with inherited skeletal muscle diseases may also have associated cardiac disease, so a cardiology evaluation should be considered in all patients with unexplained CK elevations.

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48

Lilleker, James B., and Mark E. Roberts. Metabolic myopathies. Edited by Hector Chinoy and Robert Cooper. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198754121.003.0005.

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Metabolic myopathies are caused by defects in the metabolic processes of energy storage and utilization, and can present with exercise intolerance, fatigue, muscle pain, and weakness. Metabolic myopathies are rare and can be difficult to diagnose. However, the clinical presentation can be similar to, and thus mimic, both the idiopathic inflammatory myopathies and other genetic muscle disorders including the muscular dystrophies. Careful enquiry about the nature and timing of muscle pain, as well as identification of other clinical ‘red-flags’, can highlight the possibility of a metabolic myopathy. The possibility of metabolic myopathy or muscular dystrophy mimicking myositis should be considered early in ‘treatment-resistant myositis’ or ‘seronegative myositis’. The diagnosis of metabolic myopathies depends on a multidisciplinary team, an awareness of the increasing availability of enzyme activity testing and the utility of expanding genetic technologies. In some cases, dietary manipulation and enzyme replacement therapies are useful treatments.

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49

Penman, Alan, Kimberley Crowder, and William M. Watkins. 50 Studies Every Ophthalmologist Should Know. Edited by Michael E. Hochman. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780190050726.001.0001.

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50 Studies Every Ophthalmologist Should Know succinctly summarizes the most important and clinically relevant studies published in the mainstream ophthalmology literature in the past forty years. Emphasis has been placed on landmark studies, that changed thinking and practice in the field, rather than studies that are most frequently cited. Most are randomized controlled trials that have helped shape current ophthalmology practice guidelines. However, some important observational (cohort, case-control, and descriptive) studies are also included. The book is divided into seven sections, corresponding to anatomical segment or disease of the eye (cornea, cataract, glaucoma, vitreoretinal, macula, uveal tract, and orbit/eyelids/extraocular muscles/optic nerve). Each chapter finishes with an illustrative clinical case, and includes a reference to the relevant section of the practice guidelines of the American Academy of Ophthalmology. The book is not aimed solely at ophthalmologists, however. Every practicing physician, no matter his or her specialty, should be familiar with the relation of the eye to the rest of the human body, and with the use and value of the ophthalmoscope. Ophthalmology has a particular relevance to physicians working in primary care, internal medicine, neurology, neurosurgery, pediatrics, and emergency medicine.

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50

Shaibani, Aziz. Myotonia. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199898152.003.0021.

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Анотація:

Myotonia is a slow relaxation phase of a muscle after normal contraction. Patients report myotonia as muscle stiffness and sometimes pain. They usually adapt to it well. Falls due to myotonia may lead to accidents. Checking for percussion and action myotonia should be part of neuromuscular examination. Electrically silent myotonia is a sign of Brody’s syndrome. Myotonia may be incidentally discovered during EMG. The most important task is to differentiate between myotonia and paramyotonia clinically and electromyographically. Most myotonic disorders are caused by mutations of sodium, and chloride channels. There has been a significant understanding of the underlying channelopathies recently. Severe myotonia respond well to Mexiletine.

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