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Дисертації з теми "Smoking-related disease"

Автор: Grafiati

Опубліковано: 3 червня 2025

Оновлено: 31 липня 2025

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1

Parzych, Katarzyna. "The role of innate immune system receptors in smoking related disease." Thesis, Imperial College London, 2013. http://hdl.handle.net/10044/1/14371.

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Анотація:

Cigarette smoke induces inflammation, in part, by activation of Toll like receptor (TLR) 2 and TLR4. TLRs are present on most cells and recognize pathogen associated molecular patterns (PAMPs) and non-pathogenic molecules, known as danger associated molecular patterns (DAMPs). Activation of TLRs results in the propagation of a signalling pathway leading to the de novo synthesis of pro-inflammatory mediators that include CXCL8 and IL-1β. TLRs initiate inflammation and are involved in tissue repair processes in a number of diseases such as chronic obstructive pulmonary disease (COPD) and cancer.

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2

Wong, Chun-yam Fanny. "Is secondhand smoking related to stroke in old age in Hong Kong?" View the Table of Contents & Abstract, 2007. http://sunzi.lib.hku.hk/hkuto/record/B38479539.

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3

Doolan, Daniel M. "Age related differences in smoking cessation outcomes for women hospitalized with cardiovascular disease." Diss., Search in ProQuest Dissertations & Theses. UC Only, 2007. http://gateway.proquest.com/openurl?url_ver=Z39.88-2004&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&res_dat=xri:pqdiss&rft_dat=xri:pqdiss:3261241.

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4

Campbell, Joshua David. "Genome-wide characterization of microRNA and gene expression patterns in smoking-related lung disease." Thesis, Boston University, 2012. https://hdl.handle.net/2144/12307.

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Thesis (Ph.D.)--Boston University PLEASE NOTE: Boston University Libraries did not receive an Authorization To Manage form for this thesis or dissertation. It is therefore not openly accessible, though it may be available by request. If you are the author or principal advisor of this work and would like to request open access for it, please contact us at open-help@bu.edu. Thank you.<br>Smoking-related lung diseases such as chronic obstructive pulmonary disease (COPD) and lung cancer are significant public health concerns world-wide. High throughput genomic technologies have opened up a new re

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5

Wong, Chun-yam Fanny, and 黃峻崟. "Is secondhand smoking related to stroke in old age in Hong Kong?" Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B39724633.

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6

Emslie, Karen. "Periodontal disease and oral health-related quality of life : smoking cessation adventures in primary dental care." Thesis, University of Dundee, 2014. https://discovery.dundee.ac.uk/en/studentTheses/073d9a66-18e8-49ea-b048-71c3298e02cc.

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Much of the workload of primary dental care teams in remote-rural Scotland consists of treating periodontal disease, an inflammatory condition for which smoking is a proven risk factor. This thesis sought to ascertain the effectiveness of a smoking cessation intervention applied in a primary care dental setting and employed a narrative literature review to explore periodontal disease, smoking and smoking cessation interventions. This literature review informed the design of a randomised controlled trial which failed due to lack of recruitment. The Medical Research Council Framework for the Dev

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7

Crowther, Ann Jeannette Louise. "Glutathione, the redox sensitive transcription factors AP-1 and NF-κB, and early one adenoviral protein in human lung in smoking related lung disease". Thesis, University of Edinburgh, 2007. http://hdl.handle.net/1842/27843.

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NF-κB and AP-1 are both redox sensitive transcription factors, and are involved in the regulation of the gene transcription of many pro-inflammatory mediators. AP-1 and NF-κB have a close relationship with γ-glutamylcysteine synthetase (γ-GCS), the rate limiting enzyme in the synthesis of glutathione, with the γ-GCS gene containing various elements including an AP-1 binding site. Susceptibility to the effects of cigarette smoke is likely to explain why certain individuals develop COP and this susceptibility may arise from an earlier viral infection such as adenoviral infection that lies dorma

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8

Theakstone, Kirkham Andrew Jonathan. "Smoking and the use of lower tar yield cigarettes." Thesis, University of Surrey, 1988. http://epubs.surrey.ac.uk/844124/.

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Анотація:

The aim of this thesis was to investigate smoking behaviour in a population of habitual cigarette smokers. Measurements were made 5 times, at intervals of 4 weeks whilst smoking cigarettes with tar yields above 10 mg. After switching to lower yield cigarettes (reductions of at least 3 mg tar and 0.2 mg nicotine) a further 6 visits were made at intervals of 6 weeks. A group of subjects followed the same protocol but without switching. Tobacco smoke uptake was estimated by measuring pre and post-smoking plasma nicotine, cotinine, carboxyhaemoglobin (HbCO) and expired carbon monoxide (infinity).

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9

Mahmood, MQ. "Studies on epithelial mesenchymal transition (EMT) in 'smoking related' airway disease." Thesis, 2017. https://eprints.utas.edu.au/23879/1/Mahmood_whole_thesis.pdf.

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Анотація:

Background: Chronic obstructive pulmonary disease (COPD) is a global health problem, killing approximately 3 million people annually and emerging as the third largest cause of human mortality worldwide. COPD is a poorly reversible chronic, slowly progressive airway obstructive respiratory disease. It is mainly smoking-related and primarily constitutes small airway fibrosis and destruction, more generalized “chronic bronchitis” and in some but not all later development of emphysema. Tobacco smoking is the main causative factor for development of COPD, both in developed and developing countries.

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10

Tsuei, Ho, and 崔禾. "Exploring the smoking cessation behavior and it’s related factors among patients with chronic obstructive pulmonary disease." Thesis, 2011. http://ndltd.ncl.edu.tw/handle/37328707053707798656.

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碩士<br>國立臺北護理學院<br>護理研究所<br>99<br>The aim of the study was to explore the smoking cessation behavior and it’s related factors of adults with chronic obstructive pulmonary disease. A convenient sample of 100 adults diagnosed with chronic obstructive pulmonary disease was recruited from the pulmonary clinics in Northern Taiwan. After signing an inform consent, participants answered the questionnaire which included the smoking cessation stage, smoking cessation health belief, smoking cessation self-efficacy and Fagerstrom test for nicotine dependence. Data were collected from February to October i

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11

Binnington, Matthew John. "CYP2A6 and CYP2B6 Genetic Variation, and Tobacco Use Behaviours and Biomarkers in Alaska Natives." Thesis, 2011. http://hdl.handle.net/1807/30182.

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Анотація:

The impact of CYP2A6 and CYP2B6 genetic variation on nicotine metabolism, tobacco use behaviours, and nicotine biomarkers was investigated in a group of Alaska Natives (n = 400). CYP2A6 and CYP2B6 allele frequencies were unique and associations of CYP2A6 genotype and CYP2A6 activity (plasma and urine trans 3’-hydroxycotinine/cotinine (3HC/COT) ratios) were robust. Notably, this population possessed a more rapid rate of CYP2A6 activity (higher plasma 3HC/COT) when compared to CYP2A6 wild-type individuals in other ethnic groups (ANOVA P < 0.001). Also demonstrated was a significant difference in

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12

Edman, Kristina. "Epidemiological studies of Oral Health, development and influencing factors in the county of Dalarna, Sweden 1983–2013." Doctoral thesis, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-280131.

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The aim of this thesis is to describe the development of oral health and possible associations with socioeconomic and socio-behavioural factors, in an adult population over a period of 30 years. A further aim is to describe attitudes to, and demands of dental care, and the impact of oral conditions on quality of life. The study sample consisted of 787-1158 individuals, aged between 35-85 years, randomly selected from Dalarna’s population register in 1982, 2002, 2007 and 2012. The studies were carried out in 1983, 2003, 2008 and 2013, and the participants responded to a questionnaire and a clin

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13

Kusko, Rebecca. "Integrative transcriptomics in smoking related lung diseases." Thesis, 2015. https://hdl.handle.net/2144/15452.

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Chronic lung diseases including Chronic Obstructive Pulmonary Disease (COPD), Idiopathic Pulmonary Fibrosis (IPF) and lung cancer are major causes of morbidity and mortality in the United States due to high incidence and limited therapeutic options. In order to address this critical issue, I have leveraged RNA sequencing and integrative genomics to define disease-associated transcriptomic changes which could be potentially targeted to lead to new therapeutics. We sequenced the lung transcriptome of subjects with IPF (n=19), emphysema (n=19, a subtype of COPD), or neither (n=20). The expressi

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14

Lai, Yung-Han, and 賴永翰. "The Impact of Smoking Habits on Respiratory-Related Diseases Medical Utilization." Thesis, 2013. http://ndltd.ncl.edu.tw/handle/91048375310813550343.

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碩士<br>國立臺北大學<br>財政學系<br>101<br>Medical expenditures have rapidly increased since the implementation of National Health Insurance (NHI), from 106 billion points in 1995 to 375 billion points in 2011. Smoking behavior has been found causing the problem of health and leads to a considerable medical care use. Therefore, government set up the tobacco hazards prevention act in 1997 in order to further improve people health and indirectly relief the financial pressure of NHI. Since the respiratory organs is highly related to smoking habits. The objective of this study is to investigate whether smokin

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15

Pavel, Ana Brandusa. "Multi-omics data integration for the detection and characterization of smoking related lung diseases." Thesis, 2017. https://hdl.handle.net/2144/24073.

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Lung cancer is the leading cause of death from cancer in the world. First, we hypothesized that microRNA expression is altered in the bronchial epithelium of patients with lung cancer and that incorporating microRNA expression into an existing mRNA biomarker may improve its performance. Using bronchial brushings collected from current and former smokers, we profiled microRNA expression via small RNA sequencing for 347 patients with available mRNA data. We found that four microRNAs were under-expressed in cancer patients compared to controls (p<0.002, FDR<0.2). We explored the role of these m

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