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Готові списки джерел за темами / Systemic lupus erythematosus Immunological aspects / Дисертації

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Дисертації з теми "Systemic lupus erythematosus Immunological aspects"

Автор: Grafiati

Опубліковано: 4 червня 2021

Оновлено: 12 лютого 2022

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1

Li, Hei Philip, and 李曦. "Subphenotype stratification in systemic lupus erythematosus." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hub.hku.hk/bib/B48334765.

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Subsets of systemic lupus erythematosus (SLE) patients with distinct patterns of disease manifestations and autoantibody production have been reported, but seldom have these two phenomena been analysed together. Cluster analysis was performed on 1928 Chinese SLE patients based on autoantibody profile and the frequencies of various clinical manifestations were compared between each cluster. Separate association analyses between individual autoantibodies and clinical manifestations, as well as between clinical manifestations, were also performed. This study identifies three separate autoantib

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2

Chan, Tak-mao Daniel, and 陳德茂. "Pathogenesis of systemic lupus erythematosus: interactions between anti-DNA antibodies and vascular endothelialcells." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1994. http://hub.hku.hk/bib/B31981574.

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3

Yan, Sheng, and 晏晟. "Plasmacytoid dendritic cells : their functional abnormalities and regulatory mechanisms in the development of systemic lupus erythematosus." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/193512.

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Systemic lupus erythematosus (SLE) is a chronic multi-organ autoimmune disease that is characterised by diverse clinical manifestations. Immunologically, SLE features a prominent “interferon (IFN) signature” which is marked by an elevated expression of type I IFN-regulated genes in blood and tissue cells of patients with this condition. Plasmacytoid dendritic cells (pDCs), also known as the most potent type I IFN-producing cells, are therefore considered the major culprit in SLE pathogenesis. Previous studies from our group have demonstrated abnormalities in circulating and bone marrow (BM)-de

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4

Ling, Guangsheng, and 寧珖聖. "Mechanic assessments of autoimmune responses induced by dendritic cells upon interactions with dying cells: therole of IL-10." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B4308591X.

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5

Kam, Siu-kei Christy, and 甘笑琪. "Detection of anti-nuclear antibody responses induced by dendritic cells that have captured dying cells in mouse models." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2003. http://hub.hku.hk/bib/B31971842.

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6

Halpern, Melissa Dale. "The in vivo and in vitro effects of diethyldithiocarbamate on autoimmune New Zealand Black/White F₁ hybrid, MRL/Mp-lpr/lpr and related and normal murine strains." Diss., The University of Arizona, 1989. http://hdl.handle.net/10150/184940.

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New Zealand Black/White F₁ hybrid (NZB/W) and MRL/Mp-lpr/lpr (MRL/lpr) mice spontaneously develop a Systemic Lupus Erythematosus-like autoimmune disease. While the primary immunologic defect in the NZB/W is due to B cells, in the MRL/lpr it is a result of T cell abnormalities. Diethyldithiocarbamate (DTC), an agent suggested to enhance T cell function, was used to treat both strains. Weekly treatment of NZB/W mice with 25 mg/kg DTC had no significant effect upon survival or autoantibody levels but did induce changes in cell surface antigen expression. MRL/lpr mice treated with DTC displayed no

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7

Niemela-Waller, Kirsi (Kirsi M. ). "Cognitive Dysfunction in Systemic Lupus Erythematosus." Thesis, University of North Texas, 1997. https://digital.library.unt.edu/ark:/67531/metadc278952/.

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The purpose of the study was to determine the point prevalence of cognitive dysfunction in patients with systemic lupus erythematosus (SLE) and to investigate its association with corticosteroids and depression. The severity of dysfunction and the pattern of cognitive changes were examined. This study hypothesized that cognitive dysfunction is common in SLE and many previous studies have underestimated its prevalence, partially due to using limited neuropsychological batteries and insensitive test instruments. It was further hypothesized that the pattern of cognitive changes in SLE patients wi

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8

Fan, Hiu-yi Rosie, and 范曉怡. "Cost of systemic lupus erythematosus in Hong Kong." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B45012817.

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9

Cederholm, Anna. "Novel immunological mechanisms and factors in systemic lupus erythematosus-related cardiovascular disease /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-679-4/.

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10

葉偉基 and Wai-kee Eddie Ip. "Mannose-binding lectin and systemic lupus erythematosus: molecular studies." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1998. http://hub.hku.hk/bib/B31220952.

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11

Chong, Wai-po, and 莊偉波. "Association of interleukin 10 promoter polymorphisms with systemic lupus erythematosus." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2003. http://hub.hku.hk/bib/B29295981.

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12

Leung, Wai-nor, and 梁慧娜. "Stress and coping of patients with systemic lupus erythematosus." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1994. http://hub.hku.hk/bib/B3124984X.

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13

Ng, Man-wai, and 吳雯慧. "Association of B lymphocyte stimulator (BLyS) polymorphisms with systemic lupus erythematosus (SLE)." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B32053022.

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14

Chang, Yuk-kwan, and 張玉君. "Genetic susceptibility for systemic lupus erythematosus from genome-wide association studies-BANK1 as an example." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B46599812.

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15

Zhang, Jing, and 张婧. "Identification of susceptibility loci for systemic lupus erythematosus (SLE) in Asians and functional characterization of the related genes." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/200342.

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Анотація:

SLE is a complicated autoimmune disease with a strong genetic basis. Owing to the successful application of genome-wide association study (GWAS) on complex traits, significant progresses have been made in identifying susceptible loci for SLE. So far, more than 30 loci have been shown to have robust association with the disease, significantly improving our understanding of this disease. On the other hand, these loci only confer relatively small increments in disease risk, leaving a large amount of disease heritability unexplained. Several limitations may lead to the issue of “missing heritab

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16

Zhang, Yan, and 张彦. "Association studies of systemic lupus erythematosus (SLE) : from novel susceptibility loci to gene-gene interaction." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hdl.handle.net/10722/211557.

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Анотація:

Systemic lupus erythematosus (SLE) is characterized as an autoimmune disorder with unclear etiology. To identify the genetic effect of SLE, a genome wide association study (GWAS) and its further replication were conducted on SLE patients in Asian populations and ethnically matched controls. Before this study, most of the confirmed association loci were identified by GWAS studies in European populations. Apart from the established associations, we identified a SLE susceptible single nucleotide polymorphisms (SNP) located in ETS1 (rs1128334, P=2.3E-11, OR=1.29) in four different cohorts. This

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17

Hassan, Adla Bakri. "Mixed connective tissue disease, myositis and systemic lupus erythematosus : immunological and genetic studies in three related rheumatic autoimmune diseases /." Stockholm, 2002. http://diss.kib.ki.se/2002/91-7349-388-0.

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18

Eriksson, Catharina. "Immunological mechanisms in systemic autoimmunity : autoantibodies and chemokines in systemic lupus erythematosus and during treatment with TNF inhibitors in rheumatoid arthritis." Doctoral thesis, Umeå universitet, Klinisk immunologi, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-42954.

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Background. Rheumatoid Arthritis (RA) is an autoimmune inflammatory disease that, without powerful treatment, may lead to irreversible joint damage. During the past decade, anti-cytokine therapy has become available, e.g., infliximab, a chimeric antibody targeting the pro-inflammatory cytokine TNF that has a central role in the inflammatory process in RA patients. Systemic Lupus Erythematosus (SLE) is a systemic autoimmune disease that may affect all organs and is characterized by a massive antibody production. Chemokines, chemokine receptors and lipoprotein receptor-related protein 1(CD91) ar

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19

Siu, Ho-on, and 蕭可安. "The role of interferon regulatory factor 5 gene polymorphisms in systemic lupus erythematosus." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B39558393.

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20

Ng, Ping, and 吳萍. "STAT4 and BLK, but not PXK, are associated with Systemic lupus erythematosus (SLE) in Hong Kong Chinese." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B4150852X.

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21

Nicolaou, Stella A. "K⁺ Channel Trafficking in the Immunological Synapse of Human T Cells in Health and Autoimmunity." University of Cincinnati / OhioLINK, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1194547989.

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22

Nie, Yingjie, and 聶瑛潔. "Defective dendritic cells and mesenchymal stromal cells in systemic lupus erythematosus and the potential of mesenchymal stromal cells ascell-therapy." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B43278681.

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23

Bjarnegård, Niclas. "Aspects on wall properties of the brachial artery in man : with special reference to SLE and insulin-dependent diabetes mellitus /." Linköping : Department of Medical and Health Sciences, Linköping University, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-11273.

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24

Sally, Benjamin Andrew. "Failure to process chromatin on apoptotic microparticles in the absence of deoxyribonuclease 1 like 3 drives the development of systemic lupus erythematosus." Thesis, 2017. https://doi.org/10.7916/D8XG9WSQ.

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Анотація:

Systemic lupus erythematosus is an autoinflammatory disorder driven by the development of autoantibodies to self-nucleic acids, in particular to DNA and to chromatin. Loss-of-function mutations of the secreted deoxyribonuclease DNASE1L3 have been implicated in the development of aggressive familial lupus. In addition, recent genome-wide association studies have linked a hypomorphic variant of DNASE1L3 to sporadic lupus. Studies in the lab determined that Dnase1l3-deficient mice develop rapid autoantibody responses against dsDNA and chromatin, and at older ages this leads to a lupus-like inflam

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25

Inês, Luís Pedro Bolotinha de Sousa. "Predictors of outcome and immunological markers in patients with Systemic lupus erythematosus." Doctoral thesis, 2018. http://hdl.handle.net/10400.6/6312.

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Background Systemic lupus erythematosus (SLE) is the prototypic multisystem autoimmune disease, with a broad spectrum of clinical presentations encompassing almost all organs and a chronic course, which can vary from mild to life-threatening. There is a major unmet need for outcome prediction enabling tailoring management and therapeutic interventions for SLE patients. Objectives To improve outcome prediction in SLE, the specific aims of this thesis were: (1) to evaluate the performance of the ACR and the SLICC classification criteria sets for SLE; (2) to evaluate the effect of the cla

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26

Marinho, António Joaquim dos Santos Pereira Sá. "Vitamin D deficiency and resistance state association with clinical and immunological deregulation in systemic lupus erythematosus." Tese, 2017. https://repositorio-aberto.up.pt/handle/10216/90347.

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27

Marinho, António Joaquim dos Santos Pereira Sá. "Vitamin D deficiency and resistance state association with clinical and immunological deregulation in systemic lupus erythematosus." Doctoral thesis, 2017. https://repositorio-aberto.up.pt/handle/10216/90347.

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