Готові списки джерел за темами / Vasopressor

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Добірка наукової літератури з теми "Vasopressor"

Автор: Grafiati

Опубліковано: 3 червня 2025

Оновлено: 31 липня 2025

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Статті в журналах з теми "Vasopressor"

Guinot, Pierre-Grégoire, Audrey Martin, Vivien Berthoud, et al. "Vasopressor-Sparing Strategies in Patients with Shock: A Scoping-Review and an Evidence-Based Strategy Proposition." Journal of Clinical Medicine 10, no. 14 (2021): 3164. http://dx.doi.org/10.3390/jcm10143164.

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Despite the abundant literature on vasopressor therapy, few studies have focused on vasopressor-sparing strategies in patients with shock. We performed a scoping-review of the published studies evaluating vasopressor-sparing strategies by analyzing the results from randomized controlled trials conducted in patients with shock, with a focus on vasopressor doses and/or duration reduction. We analyzed 143 studies, mainly performed in septic shock. Our analysis demonstrated that several pharmacological and non-pharmacological strategies are associated with a decrease in the duration of vasopressor

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Van Haren, Robert M., Chad M. Thorson, Evan J. Valle, et al. "Vasopressor Use during Emergency Trauma Surgery." American Surgeon 80, no. 5 (2014): 472–78. http://dx.doi.org/10.1177/000313481408000518.

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Most evidence suggests early vasopressor use is associated with death after trauma, but no previous study has focused on patients requiring emergency operative intervention (OR). We test the hypothesis that vasopressors are harmful in this population. Records from 746 patients requiring OR from July 2009 to March 2013 were retrospectively reviewed and stratified based on vasopressor use (epinephrine [EPI], phenylephrine, ephedrine, norepinephrine, dobutamine, vasopressin) or no vasopressor use. Vasopressors were administered to 225 patients (30%) during OR; 59 patients (8%) received multiple v

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Hamzaoui, Olfa, Antoine Goury, and Jean-Louis Teboul. "The Eight Unanswered and Answered Questions about the Use of Vasopressors in Septic Shock." Journal of Clinical Medicine 12, no. 14 (2023): 4589. http://dx.doi.org/10.3390/jcm12144589.

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Septic shock is mainly characterized—in addition to hypovolemia—by vasoplegia as a consequence of a release of inflammatory mediators. Systemic vasodilatation due to depressed vascular tone results in arterial hypotension, which induces or worsens organ hypoperfusion. Accordingly, vasopressor therapy is mandatory to correct hypotension and to reverse organ perfusion due to hypotension. Currently, two vasopressors are recommended to be used, norepinephrine and vasopressin. Norepinephrine, an α1-agonist agent, is the first-line vasopressor. Vasopressin is suggested to be added to norepinephrine

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Knotzer, Hans, Bernhard Poidinger, and Axel Kleinsasser. "Pharmacologic Agents for the Treatment of Vasodilatory Shock." Current Pharmaceutical Design 25, no. 19 (2019): 2133–39. http://dx.doi.org/10.2174/1381612825666190704101907.

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: Vasodilatory shock is a life-threatening syndrome in critically ill patients and is characterized by severe hypotension and resultant tissue hypoperfusion. This shock state requires the use of vasopressor agents to restore adequate vascular tone. : Norepinephrine is still recommended as first-line vasopressor in the management of critically ill patients suffering from severe vasodilation. In the recent time, catecholaminergic vasopressor drugs have been associated with possible side effects at higher dosages. This so-called catecholamine toxicity has focused on alternative noncatecholaminerg

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Bissell, Brittany D., Carolyn Magee, Peter Moran, Melissa L. Thompson Bastin, and Alexander H. Flannery. "Hemodynamic Instability Secondary to Vasopressin Withdrawal in Septic Shock." Journal of Intensive Care Medicine 34, no. 9 (2017): 761–65. http://dx.doi.org/10.1177/0885066617716396.

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Rationale: Vasopressors such as norepinephrine are first line for support of mean arterial pressure (MAP) in the management of septic shock. Their use, however, is commonly associated with many adverse events. These detriments frequently trigger the use of alternative, noncatecholamine therapies, including vasopressin. Vasopressin deficiency is a known physiologic consequence of septic shock, and while guidelines recommend vasopressin in addition to norepinephrine, no consensus exists on the duration of deficiency or ideal time of cessation. Studies have suggested that vasopressin discontinuat

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Ramesh, Ambika, Akshith Doddi, Aisha Abbasi, Mohammad A. Al-Mamun, Ankit Sakhuja, and Khaled Shawwa. "Use of vasopressors in patients with acute kidney injury on continuous kidney replacement therapy." PLOS ONE 19, no. 12 (2024): e0315643. https://doi.org/10.1371/journal.pone.0315643.

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Objective To investigate whether the use of a specific vasopressor was associated with increased mortality or adverse outcomes in patients with acute kidney injury (AKI) receiving continuous kidney replacement therapy (CKRT). Methods Patients with AKI who underwent CKRT between 1/1/2012-1/1/2021 at a tertiary academic hospital were included. Cox proportional hazard model was used to assess the relationship between time-dependent vasopressor dose and in-hospital mortality. Results There were 641 patients with AKI that required CKRT. In-hospital mortality occurred in 318 (49.6%) patients. Those

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Gao, Hans, Samuel Garcia, Aidan Mullan, Daniel Fiterman Molinari, and Aaron Klassen. "From Ambulance to Admission." International Journal of Paramedicine, no. 10 (April 16, 2025): 37–54. https://doi.org/10.56068/ipna1247.

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OBJECTIVE: Vasopressors are critical for patients experiencing shock. This observational study aims to describe the usage of vasopressors by EMS, and to assess how vasopressor usage differs between transport and hospital environments. The information gathered in this study can help determine which vasopressors should be available in an EMS system and guide the management of patients requiring vasopressors during EMS transport. METHODS: A retrospective observation study was performed from December 2019 to December 2022. Inclusion criteria were adult patients who received vasopressor infusions d

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McHargue, Cody, Tam Pham, Julie Rizzo, and James Aden. "89 Vasopressor Use and Burn Resuscitation Outcomes at 5 Major US Burn Centers." Journal of Burn Care & Research 46, Supplement_1 (2025): S72. https://doi.org/10.1093/jbcr/iraf019.089.

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Abstract Introduction Prior research indicates that vasopressors are sometimes used during acute burn resuscitation, though significant concerns exist due to vasoconstrictive effects, reduced organ perfusion, and risks of organ failure and death. The purpose of this study was to examine the use of vasopressor infusions (to include epinephrine, norepinephrine, dopamine and vasopressin) during acute burn resuscitation in five major burn centers across the United States. Methods This was a secondary analysis of the Burn Navigator (BN) study, a multicenter observational trial that enrolled 300 pat

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Edward Gultom, Andrio Farel, Rommy F. Nadeak, Tasrif Hamdi, and Yuki Yunanda. "Perbandingan Vasopressin dan Norephinephrine sebagai Vasopressor pada Pasien Syok Sepsis." Jurnal Anestesi Perioperatif 12, no. 2 (2024): 97–108. http://dx.doi.org/10.15851/jap.v12n2.3739.

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Sepsis menyebabkan kematian terbesar, tingkat mortalitas sepsis tinggi dan dapat mencapai 50% pada syok sepsis. Tatalaksana resusitasi pada syok sepsis menggunakan vasopresor. norepinephrine saat ini adalah rekomendasi utama pada syok sepsis, vasopressin digunakan sebagai obat lini kedua untuk mengurangi efek samping yang disebabkan oleh obat seperti norephinephrine, dan juga membantu pada keadaan syok resisten-katekolamin. Penelitian ini merupakan penelitian uji klinis dengan metode acak tersamar ganda. Penelitian ini bertujuan untuk mengetahui perbandingan penggunaan vasopressin dan norephin

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Patel, Bhavesh M., Dean R. Chittock, James A. Russell, and Keith R. Walley. "Beneficial Effects of Short-term Vasopressin Infusion during Severe Septic Shock." Anesthesiology 96, no. 3 (2002): 576–82. http://dx.doi.org/10.1097/00000542-200203000-00011.

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Background Septic shock is associated with vasopressin deficiency and a hypersensitivity to its exogenous administration. The goal of the current study was to determine whether short-term vasopressin infusion in patients experiencing severe septic shock has a vasopressor sparing effect while maintaining hemodynamic stability and adequate end-organ perfusion. Methods Patients experiencing septic shock that required high-dose vasopressor support were randomized to a double-blinded 4-h infusion of either norepinephrine (n = 11) or vasopressin (n = 13), and open-label vasopressors were titrated to

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Дисертації з теми "Vasopressor"

Tabrizchi, Reza. "Different modes of vasopressor actions of angiotensin and non-selective or selective beta-adrenoceptor antagonists." Thesis, University of British Columbia, 1988. http://hdl.handle.net/2429/29439.

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Vasoconstriction can be initiated via the interaction of a number of chemicals with specific "receptive sites" known as the receptors. This thesis examines two distinctly different modes by which drugs initiate a contractile response, namely, (i) the interaction of angiotensin analogues with a heterogeneous population of angiotensin receptors in vascular smooth muscles, and (ii) the conditions whereby B-adrenoceptor antagonists interact with a-adrenoceptor antagonists thereby causing a pressor response. Conscious, unrestrained, instrumented-rats were used for the study. It has been suggested

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Wollan, Melissa. "Shock index as a predictor of vasopressor use in severe sepsis patients in the emergency department." [New Haven, Conn. : s.n.], 2008. http://ymtdl.med.yale.edu/theses/available/etd-12092008-172348/.

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Erkinaro, T. (Tiina). "Fetal and placental haemodynamic responses to hypoxaemia, maternal hypotension and vasopressor therapy in a chronic sheep model." Doctoral thesis, University of Oulu, 2006. http://urn.fi/urn:isbn:9514281659.

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Abstract Knowledge of the effects of maternally administered vasopressors on human fetal and placental haemodynamics is sparse and limited to elective Caesarean deliveries in uncomplicated pregnancies. We hypothesized that, after short-term fetal hypoxaemia, which activates fetal cardiovascular compensatory mechanisms, treatment of maternal hypotension with ephedrine or phenylephrine results in divergent responses in fetal and placental haemodynamics. Chronically instrumented near-term sheep fetuses with either normal placental function or increased placental vascular resistance following pl

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Daniels, Abigail Hanlise. "Maternal and cardiac output response to vasopressor therapy during spinal anaesthesia for Caesarean Section in severe preeclampsia." Master's thesis, University of Cape Town, 2017. http://hdl.handle.net/11427/27436.

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Background: The maternal haemodynamic responses to vasopressors during spinal anaesthesia for caesarean delivery in patients with severe preeclampsia, have not been accurately described. This study compared the haemodynamic effects of the vasopressors ephedrine and phenylephrine during spinal anaesthesia. Methods: Thirty nine women with treated severe preeclampsia presenting for spinal anaesthesia for caesarean section for a maternal indication, were studied. Baseline maternal haemodynamics were measured in the left lateral position, using minimal invasive cardiac output monitoring (LiDCOrapid

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Ngaka, Tshebeletso Christian. "The influence of body mass index on sensorimotor block and vasopressor requirement during spinal anaesthesia for elective caesarean section." Master's thesis, University of Cape Town, 2017. http://hdl.handle.net/11427/24485.

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Background: It has been suggested that the dose requirement for spinal anesthesia (SA) is lower in obese patients for cesarean delivery. In this prospective, observational, non-inferiority study we tested the hypothesis that obesity would not have a clinically important effect on vasopressor requirements or block height. Methods: Two groups of 25 parturients, Group O (BMI >40 kg/m²) and Group N (BMI <32 kg/m²) requiring elective cesarean delivery were recruited. All patients received 10 mg intrathecal hyperbaric bupivacaine co-administered with 10 μg fentanyl. Dermatomal levels were assessed a

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Hylands, Mathieu. "Première phase d’un programme de recherche sur l’utilisation de vasopresseurs en traumatologie : étude observationnelle et revue systématique." Mémoire, Université de Sherbrooke, 2016. http://hdl.handle.net/11143/9636.

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Résumé : Les réanimateurs ont recours à des interventions à la fois médicales et chirurgicales en contexte de choc traumatique. Le rôle des vasopresseurs dans cette prise en charge est controversé. Alors que les lignes directrices américaines considèrent que les vasopresseurs sont contre-indiqués, certains experts européens en encouragent l’utilisation pour diminuer le recours aux liquides intraveineux. Avant d’élaborer un essai clinique, il importe de comprendre la pratique actuelle à laquelle se comparera une intervention expérimentale, ainsi que de connaître le niveau d’incertitude dans la

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Schäfer, Andreas [Verfasser]. "Wirksamkeit der Vasopressoren Adrenalin und Arginin-Vasopressin während eines asphyktisch induzierten Herz-Kreislaufstillstandes am porcinen Tiermodell / Andreas Schäfer." Gießen : Universitätsbibliothek, 2014. http://d-nb.info/1068772867/34.

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Soares, André Vasconcelos. "Choque hemorrágico experimental em cães anestesiados com isofluorano, tratados com solução hipertônica e colóide associada a diferentes vasopressores." Universidade Federal de Santa Maria, 2010. http://repositorio.ufsm.br/handle/1/4055.

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Coordenação de Aperfeiçoamento de Pessoal de Nível Superior<br>The objective was to compare the hemodynamic and metabolic effects of treatment with hypertonic saline and colloid (expanders) associated with different vasoconstrictors in dogs subjected to experimental hemorrhagic shock. Twenty-four healthy adult mongrel dogs were included in the study, with mean body weight of 10.84±3.3kg, males and females. Following anesthetic induction by isoflurane inhalation, the animals were intubated and connected to a partial rebreathing system, and subjected to general inhalational anesthesia with isofl

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Sandgren, Jeremy Anton. "Vasopressin in preeclampsia." Diss., University of Iowa, 2019. https://ir.uiowa.edu/etd/6849.

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Preeclampsia is a devastating disorder of pregnancy characterized by high blood pressure, proteinuria, headache, renal glomerular endotheliosis, multi-organ system failure, and fetal and maternal demise. As reviewed in Chapter I, not much is known about the pathogenesis of preeclampsia, contributing to a lack of biomarkers and treatments for the disease. In Chapter II, we review arginine vasopressin, a circulating neuropeptide hormone with important fluid balance and cardiovascular actions. Vasopressin binds to numerous receptors throughout the body to elicit its effects and is associated with

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Londen, Liesbeth van. "Vasopressin in major depression /." Leiden : Universitair Facilitair Bedrijf, 2003. http://catalogue.bnf.fr/ark:/12148/cb39926386d.

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Книги з теми "Vasopressor"

Gash, Don Marshall, and Gerard J. Boer, eds. Vasopressin. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4615-8129-1.

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Kovács, L., and B. Lichardus. Vasopressin. Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-0449-1.

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M, Gash Don, and Boer Gerard J, eds. Vasopressin. Plenum, 1987.

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W, Schrier Robert, ed. Vasopressin. Raven Press, 1985.

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Ganten, Detlev, and Donald Pfaff, eds. Neurobiology of Vasopressin. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-68493-7.

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Zingg, Hans H., Charles W. Bourque, and Daniel G. Bichet, eds. Vasopressin and Oxytocin. Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-4871-3.

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1941-, Ganten D., Pfaff Donald W. 1939-, and Clarke G, eds. Neurobiology of vasopressin. Springer-Verlag, 1985.

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Inc, ebrary, ed. Perspectives on vasopressin. Imperial College Press, 2010.

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Arnost, Urban Ivan Jaroslav, Burbach, Johannes Peter Henri, 1954-, and Wied David de, eds. Advances in brain vasopressin. Elsevier, 1998.

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Gash, Don Marshall. Vasopressin: Principles and Properties. Springer US, 1987.

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Частини книг з теми "Vasopressor"

Davenport, Andrew, Todd W. Costantini, Raul Coimbra, et al. "Vasopressor Therapy." In Encyclopedia of Intensive Care Medicine. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_787.

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Singhal, Vinay, Puneet Chopra, and Vikas Sikri. "Vasopressor Drugs." In Principles and Practice of Neurocritical Care. Springer Nature Singapore, 2024. http://dx.doi.org/10.1007/978-981-99-8059-8_23.

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Frishman, William H., Angela Cheng-Lai, and Julie Chen. "Inotropic and Vasopressor Agents." In Current Cardiovascular Drugs. Current Medicine Group, 2000. http://dx.doi.org/10.1007/978-1-4615-6767-7_11.

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Lindner, K. H. "Vasopressor Therapy in Cardiopulmonary Resuscitation." In Update in Intensive Care and Emergency Medicine. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-84423-2_3.

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Obara, Shinju, and Masahiro Murakawa. "Controversy: Volume Load vs. Vasopressor." In Off-Pump Coronary Artery Bypass. Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-54986-4_7.

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Davenport, Andrew, Todd W. Costantini, Raul Coimbra, et al. "Vasopressor Drugs in Acute Kidney Injury." In Encyclopedia of Intensive Care Medicine. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_333.

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Archer, Thomas L. "Vasopressin, Used as a Vasopressor During Cystoscopy, Causes Non-reassuring Fetal Status." In Obstetric Anesthesia. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-26478-9_29.

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Ghosh, Supradip, and Garima Arora. "Fluid Management in Septic Shock." In Rational Use of Intravenous Fluids in Critically Ill Patients. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-42205-8_14.

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AbstractBoth relative (due to vasodilatation and leaky capillaries) and absolute hypovolemia are common in patients with septic shock, and fluid infusion remains the first-line resuscitation measure. However, fluid overload is a potential consequence of fluid infusion, especially left unmonitored. Early vasopressor infusion may be useful in specific situation. Every effort should be made to limit cumulative fluid balance in every stage of resuscitation.

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Krismer, Anette C., Norman A. Paradis, Volker Wenzel, and John Southall. "Use of Vasopressor Drugs in Cardiac Arrest." In Cardiopulmonary Resuscitation. Humana Press, 2005. http://dx.doi.org/10.1385/1-59259-814-5:305.

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De Backer, Daniel, and Pierre Foulon. "Vasopressor Support for Patients with Cardiopulmonary Failure." In Cardiopulmonary Monitoring. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-73387-2_49.

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Тези доповідей конференцій з теми "Vasopressor"

Rastogi, Anshul, and Dongjin Song Mentor. "Importance Sampling to Learn Vasopressor Dosage to Optimize Patient Mortality in an Interpretable Manner." In 2024 IEEE International Conference on Big Data (BigData). IEEE, 2024. https://doi.org/10.1109/bigdata62323.2024.10825148.

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Parsons, J., H. Hirzallah, and F. M. Zeid. "Vasopressor Induced Limb Ischemia." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6557.

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Bighamian, Ramin, Sadaf Soleymani, Andrew T. Reisner, Istvan Seri, and Jin-Oh Hahn. "Modeling and System Identification of Hemodynamic Responses to Vasopressor-Inotropes." In ASME 2013 Dynamic Systems and Control Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/dscc2013-3726.

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In an effort to establish an initial step towards the ultimate goal of developing an analytic tool to optimize the vasopressor-inotrope therapy through individualized dose-response relationships, we propose a phenomenological model intended to reproduce the hemodynamic response to vasopressor-inotropes. The proposed model consists of a cardiovascular model relating blood pressure to cardinal cardiovascular parameters (stroke volume and total peripheral resistance) and the phenomenological relationships between the cardinal cardiovascular parameters and the vasopressor-inotrope dose, in such a

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Lamontagne, Francois, Matthias Briel, Mark Duffett, et al. "Vasopressor Administration: A Survey Of Canadian Intensivists." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a4543.

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Bham, Z., and C. E. Manfredi. "High Dose Insulin for Vasopressor Refractory Cardiogenic Shock." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a3401.

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Shin, Sungtae, Andrew T. Reisner, Bryce Yapps, et al. "Forecasting Hypotension during Vasopressor Infusion via Time Series Analysis." In 2019 41st Annual International Conference of the IEEE Engineering in Medicine & Biology Society (EMBC). IEEE, 2019. http://dx.doi.org/10.1109/embc.2019.8857084.

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Meenach, C., A. Kelly, and E. Bensadoun. "Outcome of High Dose Vasopressor Therapy in Septic Shock." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6504.

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Shyu, D., N. E. Ingraham, C. Linke, and K. M. Pendleton. "Overview of Peripheral Vasopressor Usage in an Academic Health System." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a4781.

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Brown, Samuel, Michael J. Lanspa, Jason P. Jones, et al. "Outcomes After High-Dose Intravenous Vasopressor Administration In Patients With Shock." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a3169.

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Odish, M. F., S. Chicotka, C. Yi, et al. "Effect of Veno-Venous Extracorporeal Membrane Oxygenation (VV-ECMO) on Vasopressor Requirements." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a1590.

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Звіти організацій з теми "Vasopressor"

Zerbib, Olivier, Yaniv Hadi, Daniel Kovarsky, et al. Multiple Recurrent Pneumothoraces and Thoracic Drain Insertion in a Mechanically Ventilated Patient Suffering from Methadone Induced Cardiomyopathy. Science Repository, 2023. http://dx.doi.org/10.31487/j.jcmcr.2022.01.02.

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Objective: To describe the experience of a multimodal therapeutic approach in a patient with methadone-induced dilated cardiomyopathy who developed recurrent bilateral tension pneumothorax. Setting: Department of Intensive Care. Patient: A patient with methadone-induced cardiomyopathy and severe left ventricular dysfunction who after mechanical ventilation underwent bilateral tension pneumothorax and prolonged cardiovascular resuscitation (CPR). Interventions: Cardiac Angiography, Multiple counter–shock (defibrillator dose), Multiple Thoracic Drains. Case Report: A 56-year-old man with past IV

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Dallman, Mary F., and Daniel N. Darlington. Central Catecholaminergic Regulation of ACTH and Vasopressin. Defense Technical Information Center, 1988. http://dx.doi.org/10.21236/ada204418.

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North, William G. Vasopressin Gene-Related Products in the Management of Breast Cancer. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada383035.

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Yibchok-anun, Sirintorn, and Wara Panichkriangkrai. The role of arginine vasopressin in diabetes-associated glucagons secretion. Chulalongkorn University, 2002. https://doi.org/10.58837/chula.res.2002.63.

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The purpose of this study was to investigate the role of arginine-vasopressin (AVP) on glucagons secretion in both normal and diabetic rats. Diabetes was induced by intraperitoneal administration of alloxan HCI(200 mg/kg). Both glucagon and AVP were determined in the effluent of the perfused pancreas using radioimmunoassay. Diabetic rats had higher baseline glucagons concentrations than normal rats. AVP (1 pmol/L) failed to change glucagons secretion in normal rats, but significantly increased glucagons secretion in diabetic rats. AVP (10-100 pmol/L) increased glucagons secretion from both nor

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Leong, Trudy, Rephaim Mpofu, and Tamara Kredo. Intersectoral action: Public–private collaboration to improve equitable access to vasopressors for septic shock. World Evidence-based Healthcare Day, 2024. http://dx.doi.org/10.70253/kslg9525.

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Uyehara, Catherine F. Vasopressin Regulation and Renal Fluid and Electrolyte Handling in Rat Models of Acute and Chronic alcohol Exposure. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada436890.

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Uyehara, Catherine F. Vasopressin Regulation and Renal Fluid and Electrolyte Handling in Rat Models of Acute and Chronic Alcohol Exposure. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada398255.

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Uyehara, Catherine F. T. Vasopressin Regulation and Renal Fluid and Electrolyte Handling in Rat Model of Acute and Chronic Alcohol Exposure. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada412845.

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Noradrenaline confirmed as first-choice vasopressor for septic shock. National Institute for Health Research, 2015. http://dx.doi.org/10.3310/signal-000165.

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