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Dissertations / Theses on the topic 'Β-hCG'

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1

MIZUOCHI, TSUGUO, MUNEHIRO NAKATA, IRVING BOIME та ін. "Alteration of N-linked oligosaccharide structures of human chorionic gonadotropin β -subunit by disruption of disulfide bonds". Thesis, Springer, 1997. http://hdl.handle.net/2237/16683.

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2

Giller, Lisa Konstanze [Verfasser], Thomas [Akademischer Betreuer] [Gutachter] Rink та Thomas [Gutachter] Vogl. "β-HCG-induzierte Hyperthyreose in der Schwangerschaft - Vermeidung von Fehlinterpretationen im Rahmen von Vorsorgeuntersuchungen / Lisa Konstanze Giller ; Gutachter: Thomas Rink, Thomas Vogl ; Betreuer: Thomas Rink". Frankfurt am Main : Universitätsbibliothek Johann Christian Senckenberg, 2017. http://d-nb.info/1136368132/34.

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3

Aleithe, Susanne. "Transkriptionelle Interaktionen morphogener Signalwege in der adulten Leber und im Hepatozellulären Karzinom." Doctoral thesis, Universitätsbibliothek Leipzig, 2015. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-171241.

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Die evolutionär konservierten morphogenen Signalwege Wnt/β-Catenin und Hedgehog (Hh) spielen vor allem in der Embryogenese, Zelldifferenzierung und Kanzerogenese eine große Rolle. Es ist bekannt, dass es zwischen Komponenten der beiden Signalkaskaden zu verschiedensten Wechselwirkungen und Hintergrundreaktionen in unterschiedlichsten Organismen und Geweben kommt. Ziel dieser Arbeit war die Aufklärung einzelner mole- kularer, transkriptioneller Prozesse hinter diesen Kreuzreaktionen in primären Hepatozyten und dem Hepatozellulären Karzinom. Dafür sind die beiden Signalwege durch verschiedenste
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4

Schäffer, Michael. "Eine neue Domäne der IL-6 Rezeptor β -Kette, gp130, vermittelt Zellwachstum und die Aktivierung der Kinasen Hck und Erk". Diss., lmu, 2000. http://nbn-resolving.de/urn:nbn:de:bvb:19-3594.

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5

Oliveira, Isabela Silva de. "Resposta imune celular de portadores de hepa tite C antes e na 12ª semana de tratamento com interferon - alfa e ribavirina." Universidade Federal da Bahia, 2015. http://repositorio.ufba.br/ri/handle/ri/22200.

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Submitted by Pós Imunologia (ppgimicsufba@gmail.com) on 2017-02-17T18:25:36Z No. of bitstreams: 1 Tese Isabela.pdf: 1865877 bytes, checksum: cbd1986b7bda4dc09057f53feeaa35b2 (MD5)<br>Approved for entry into archive by Delba Rosa (delba@ufba.br) on 2017-05-02T12:43:10Z (GMT) No. of bitstreams: 1 Tese Isabela.pdf: 1865877 bytes, checksum: cbd1986b7bda4dc09057f53feeaa35b2 (MD5)<br>Made available in DSpace on 2017-05-02T12:43:10Z (GMT). No. of bitstreams: 1 Tese Isabela.pdf: 1865877 bytes, checksum: cbd1986b7bda4dc09057f53feeaa35b2 (MD5)<br>A hepatite C crônica (HCC) é um problema de saúde mundi
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6

Barbaglia, Matteo. "Sex differences in hepatitis C virus infection: characterisation of 17,β-estradiol-modulated antiviral defences in an HCV cell culture system". Doctoral thesis, Università del Piemonte Orientale, 2021. http://hdl.handle.net/11579/127833.

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Chronic hepatitis C is one of the most widespread liver diseases in the world, with over 170 million individuals infected with the hepatitis C virus (HCV). Several clinical studies have observed a protective effect of female sex against HCV infection and HCV-related liver diseases. Our group have demonstrated that 17,β-estradiol (E2) is able to inhibit HCV life cycle in vitro but the antiviral mechanisms are still unknown. In the first part of the present study, we evaluated the E2-mediated activation of hepatocytes innate immunity. Several studies showed the relationship between hypoxia and E
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7

LOCATELLI, LUIGI. "Expression of aVB6 integrin by Pkhd1-defective cholangiocytes links enhanced ductal secretion of Macrophage chemokines to progressive portal fibrosis in Congenital Hepatic Fibrosis." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2013. http://hdl.handle.net/10281/41733.

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BACKGROUND AND AIMS: Congenital Hepatic Fibrosis (CHF) is caused by mutations in PKHD1, a gene encoding for fibrocystin, a protein of unknown function, expressed in cholangiocyte cilia and centromers. In CHF, biliary dysgenesis is accompanied by severe progressive portal fibrosis and portal hypertension. The mechanisms responsible for portal fibrosis in CHF are unclear. The αvβ6 integrin mediates local activation of TGFβ1 and is expressed by reactive cholangiocytes during cholestasis. To understand the mechanisms of fibrosis in CHF we studied the expression of αvβ6 integrin and its regulation
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8

Medeiros, Sebastiao Freitas de. "Physiological aspects of the β-core hCG fragment / Sebastiao Freitas de Medeiros". Thesis, 1991. http://hdl.handle.net/2440/21333.

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Bibliography: leaves 230-294.<br>xiii, 294 leaves : ill. ; 30 cm.<br>Analyses in detail a native small fragment of hCG/[beta] hCG subunit, the [beta]-core hCG fragment which is found in large amounts in urine and may be of clinical importance. The aims were to purify the fragment, analyse its protein and carbohydrate structure, develop direct assay methods for its measurement, and to examine it's distribution in body fluids and it's relationship with the intact hCG molecule during pregnancy.<br>Thesis (Ph.D.)--University of Adelaide, Dept. of Obstetrics and Gynaecology, 1993
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9

Ortiz, Velásquez Javier Urbano [Verfasser]. "Serumbestimmungen von freiem β-hCG [beta-hCG] und PAPP-A mittels Elektrochemilumineszenz-Immunoassays : Einfluss auf die diagnostische Trennschärfe beim Ersttrimester-Screening / Javier Urbano Ortiz Velásquez". 2010. http://d-nb.info/1008382728/34.

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10

Γκόγκος, Παναγιώτης. "Αξιολόγηση των επιπέδων της AFP και β-hCG στον ορό της εγκύου κατά το β' τρίμηνο τησς κύησης με σκοπό την πρόβλεψη δυσμενούς έκβασης της κύησης". Thesis, 2008. http://nemertes.lis.upatras.gr/jspui/handle/10889/1522.

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Σκοπός της παρούσας προοπτικής µελέτης είναι η διερεύνηση της σχέσης µεταξύ των επιπέδων της α-φετοπρωτείνης και της β-hCG στον ορό της µητέρας κατά το β' τρίµηνο της κύησης και της δυσµενούς έκβασης της κύησης σε τυχαίο δείγµα εγκύων γυναικών της Νοτιο-Δυτικής Ελλάδος. Υλικό - Μέθοδος: 130 υγιείς γυναίκες ελληνικής καταγωγής µε αυτόµατη έναρξη της εγκυµοσύνης µελετήθηκαν για τα επίπεδα της AFP και της β-hCG στον ορό τους µεταξύ της 13ης-24ης εβδοµάδας της κύησης και παρακολουθήθηκαν για δυσµενή έκβαση της εγκυµοσύνης. Επίπεδα AFP καθώς και επίπεδα β-hCG στον ορό της εγκύου 2 φορές µεγ
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11

Djeha, Améyo Xoxoabu. "Association entre la thérapie antirétrovirale et les biomarqueurs de la fonction placentaire pendant la grossesse." Thèse, 2018. http://hdl.handle.net/1866/21604.

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12

Enkhjargal, Ulziidemberel, та Ulziidemberel Enkhjargal. "Determination of the Median Levels of Maternal Serum Free β-hCG, PAPP-A and Fetal NT Thickness at 11-13+6 Weeks in Relation to co-variables in Mongolian". Thesis, 2013. http://ndltd.ncl.edu.tw/handle/09419373205560142207.

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碩士<br>亞洲大學<br>健康產業管理學系健康管理組<br>101<br>Background: Prenatal care, it is health concept for maternal with fetal and neonatal. Prenatal screening intends to identify at risk for carrying a fetus with a certain anomaly. Most countries perform that “triple test” which is the second-trimester and “combined test” is the first-trimester which is including serum PAPP-A marker and serum free β-hCG with ultrasound measurement fetal NT. World screening approach moving from 2nd to the 1st trimester, which may even pose some problems for the Mongolian maternity care system. Officially, Mongolia has been no
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13

Aleithe, Susanne. "Transkriptionelle Interaktionen morphogener Signalwege in der adulten Leber und im Hepatozellulären Karzinom." Doctoral thesis, 2014. https://ul.qucosa.de/id/qucosa%3A13350.

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Die evolutionär konservierten morphogenen Signalwege Wnt/β-Catenin und Hedgehog (Hh) spielen vor allem in der Embryogenese, Zelldifferenzierung und Kanzerogenese eine große Rolle. Es ist bekannt, dass es zwischen Komponenten der beiden Signalkaskaden zu verschiedensten Wechselwirkungen und Hintergrundreaktionen in unterschiedlichsten Organismen und Geweben kommt. Ziel dieser Arbeit war die Aufklärung einzelner mole- kularer, transkriptioneller Prozesse hinter diesen Kreuzreaktionen in primären Hepatozyten und dem Hepatozellulären Karzinom. Dafür sind die beiden Signalwege durch verschiedenste
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14

"The mechanism of HCO₃-induced insulin secretion in pancreatic β-cells and the involvement in synaptic plasticity". Thesis, 2011. http://library.cuhk.edu.hk/record=b6075143.

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Apart from CFRD, low cognitive skill index (CSI) was also found in CF patients and was attributed the lacking of vitamin E. Since it is known that insulin plays a role in the learning and memory, decreased plasma insulin level in CF patients is an alternative mechanism for impaired cognitive function. Although numerous studies have found that insulin can improve learning and memory, the mechanism of it is not well understood. In this study, we investigated the effect of insulin on the expression of hippocampal early-phase long-term potentiation (E-LTP) in the immature rats. Hippocampal brain s
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15

Hu, Chia-Chi, та 胡家齊. "Indoleamine 2,3-Dioxygenase Promotes HCC Cell Proliferation by Activating β-catenin Signaling Through Aryl Hydrocarbon Receptor Pathway". Thesis, 2017. http://ndltd.ncl.edu.tw/handle/99njnp.

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碩士<br>國立臺灣大學<br>生物化學暨分子生物學研究所<br>105<br>Hepatocellular carcinoma (HCC), the main type of primary liver cancer, is a serious healthcare problem worldwide. Tumor immune escape is involved in the initiation and progression of HCC, which may result from indoleamine 2,3-dioxygenase (IDO)-mediated metabolism of tryptophan to kynurenine (KYN). Activation of the aryl hydrocarbon receptor (AhR) by KYN, which is an endogenous ligand for AhR, has been implicated in liver carcinogenesis. Recently, it has been demonstrated that IDO was overexpressed in 35.5% of HCC resection samples and resulted in signifi
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16

Chen, Wei-Jen, та 陳為仁. "To investigate the regulation of Graptopetalun paraguayense in TGF-β pathway and lipogenesis in HCC via gene expression signature". Thesis, 2013. http://ndltd.ncl.edu.tw/handle/rwzytc.

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碩士<br>國立陽明大學<br>生物藥學研究所<br>102<br>Hepatocellular carcinoma (HCC) is an aggressive tumor with a poor prognosis and most common cause of cancer deaths in the world. However, sorafenib is the only target therapy of HCC approved by the FDA and prolongs patient’s survival for 2-3 months. Therefore, it is urgent to identify new drug candidates for the treatment of HCC. To identify the therapeutic agents, we previously found that one of the herb medicine, GP and it partially purified fraction HH-F3 have anti-HCC and anti-fibrosis effects. However, the mechanism of HH-F3 was not clear yet. In this stu
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17

Mei-Jyun та 范美桾. "Part-I β-catenin plays the role of metastasis factor in humanHepatocellular carcinomaPart-IIDown-regulation of the β-catenin gene transactivation and metastatic functions by ER-α in highly proliferative HA22T cell line of HCC". Thesis, 2005. http://ndltd.ncl.edu.tw/handle/88871899806320626910.

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碩士<br>中山醫學大學<br>生化暨生物科技研究所<br>93<br>Part-I The occurrence of HCC could be resulted from several factors associated with genetic alterations, such as the activation of oncogenes, the enhancement of c-jun and NF-κB caused by HBV, the inhibition or mutation of antitumor gene, p53. However, in the present, there is no good markers for diagnosis and metastasis which can be used as treatment for HCC. Therefore, analyzing the gene expressions in differently differentiated HCC cell lines, and in HCC tissues become necessary to find the markers for the diagnosis and anti-metastasis of HCC. Our previou
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18

Schäffer, Michael [Verfasser]. "Eine neue Domäne der IL-6-Rezeptor-β-Kette [IL-6-Rezeptor-beta-Kette], gp130, vermittelt Zellwachstum und die Aktivierung der Kinasen Hck und Erk / Michael Schäffer". 2000. http://d-nb.info/961463201/34.

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