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Journal articles on the topic '06a061'

Author: Grafiati

Published: 25 July 2024

Last updated: 31 July 2025

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1

Džamonja, Mirna, and Katherine Thompson. "Universality Results for Well-Founded Posets." Sarajevo Journal of Mathematics 1, no. 2 (2024): 147–60. http://dx.doi.org/10.5644/sjm.01.2.01.

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In this paper it is shown that the univerality spectrum of well-founded posets is exactly the same as the spectrum of the class of well-orders. A universality result for a restricted class of well-founded posets under rank and order preserving embeddings is also proved. This is done using a club guessing method generalised by Kojman which demonstrates a surjective homomorphism with subsets of the reals of bounded size ordered by inclusion. 1991 Mathematics Subject Classification. 03E04, 06A05, 06A06

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AN, Bhavale, and Shelke DS. "Graceful Labeling of Posets." Annals of Mathematics and Physics 8, no. 1 (2025): 018–28. https://doi.org/10.17352/amp.000142.

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The concept of graph labeling was introduced in the mid-1960s by Rosa. In this paper, we introduce a notion of graceful labeling of a finite poset. We obtain graceful labeling of some postes such as a chain, a fence, and a crown. In 2002 Thakare, Pawar, and Waphare introduced the `adjunct' operation of two lattices with respect to an adjunct pair of elements. We obtain the graceful labeling of an adjunct sum of two chains with respect to an adjunct pair (0, 1). AMS Subject Classification 2020: 06A05, 06A06, 05C78

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Xu, L., B. T. Keenan, A. S. Wiemken, A. I. Pack, and R. J. Schwab. "0569 Soft Palate Fat Between Lean Adults with Obstructive Sleep Apnea and Healthy Control." Sleep 43, Supplement_1 (2020): A218. http://dx.doi.org/10.1093/sleep/zsaa056.566.

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Abstract Introduction Previous studies have shown that obese patients with obstructive sleep apnea (OSA) have a significantly greater percentage of fat tissue in soft palate than normal subjects. However, the influence of soft palate fat is not clear in non-obese adults with OSA. This study compared the volume of fat in the soft palate between lean adults with OSA and lean controls. Methods We examined soft palate fat in 21 lean OSA cases and 16 lean controls with body mass index (BMI) &lt;25 kg/m2. All subjects underwent a magnetic resonance imaging (MRI) with three-point Dixon scan. We u

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Kundel, V., P. Darko, P. Taweesedt, et al. "0966 Sleep-Wake Patterns in Mothers and Children in a Rural Community With Limited Access to Electricity: Results from the Ghana Randomized Air Pollution and Health Study." Sleep 43, Supplement_1 (2020): A367. http://dx.doi.org/10.1093/sleep/zsaa056.962.

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Abstract Introduction Studies measuring objective sleep duration in rural/indigenous populations are limited, showing sleep duration similar to that of industrialized countries. Little is known about sleep duration in women of reproductive age, and children within these populations. Our study is the first to objectively characterize sleep in mothers and children in an agrarian community with limited access to electricity, utilizing data from the Ghana Randomized Air Pollution and Heath Study (GRAPHS). Methods The GRAPHS cohort, a cluster-randomized trial, evaluated the efficacy of clean fuels

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Afangbedji, Nowah Kokou Apeadoufia, James G. Taylor, Sergei Nekhai, and Marina Jerebtsova. "Elevated Plasma Soluble Urokinase-Type Plasminogen Activator Receptor (suPAR) in Sickle Cell Disease - a Marker of Chronic Kidney Disease." Blood 138, Supplement 1 (2021): 968. http://dx.doi.org/10.1182/blood-2021-153938.

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Abstract Background: Sickle cell nephropathy (SCN) is one of the most common complications of SCD, leading in most cases to chronic kidney disease (CKD) and end-stage renal disease (ESRD). Despite the high prevalence of CKD in sickle cell disease (SCD) patients, there remains a poor understanding of the pathophysiological mechanism of SCN and a lack of biomarkers for early detection of SCD-associated CKD. Soluble urokinase-type plasminogen activator receptor (suPAR) is an emerging biomarker of CKD. suPAR is a member of the fibrinolytic system, which is dysregulated in SCD patients. Objective:

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HEIANZA, YORIKO, XUAN WANG, QIAOCHU XUE, LAWRENCE J. APPEL, FRANK SACKS, and LU QI. "114-OR: Genetic Variants in GIPR and Effects of Dietary Carbohydrate Quantity and Quality on Glycemic Responses to an Oral Glucose Load—The OmniCarb Trial." Diabetes 72, Supplement_1 (2023). http://dx.doi.org/10.2337/db23-114-or.

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The gut incretin hormone, glucose-dependent insulinotropic polypeptide (GIP), may play a pivotal role in mediating adverse effects of high glycemic index (GI) foods on metabolic diseases. GIP exerts its effects by binding to a receptor, GIPR; genome-wide association studies have identified variants in the GIPR region related to type 2 diabetes (T2D) and glycemic/GIP responses to glucose tolerance tests. We examined whether GIPR variants were related to impaired glycemic responses during oral glucose tolerance tests (OGTTs) after consuming 4 diets (each for 5 weeks) with different carbohydrate

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BACHOR, TOMAS P., KUSH ATTAL, EIRINI VAGENA, et al. "1522-P: Identification of AgRP Cells in the Murine Hindbrain That Drive Feeding." Diabetes 72, Supplement_1 (2023). http://dx.doi.org/10.2337/db23-1522-p.

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The central melanocortin system is essential for the regulation of food intake and body weight in humans and rodents. Agouti-related protein (AgRP) is the sole orexigenic component of the central melanocortin system and is conserved across mammalian species. AgRP is currently known to express exclusively in the mediobasal hypothalamus, and hypothalamic AgRP-expressing neurons are essential for feeding regulation. Here we describe a previously unknown population of AgRP cells in the area postrema (AP) and the adjacent commissural nucleus of the solitary tract (cNTS) of the causal brainstem. AgR

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Doyle, Brendan M., Sara M. F. Turner, Bobbi Johnson, et al. "Comparison of Hypoglossal Motoneuron Transgene Expression following Single vs Dual Tongue Injection of AAV9‐CBA‐GFP." FASEB Journal 30, S1 (2016). http://dx.doi.org/10.1096/fasebj.30.1_supplement.lb739.

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Retrograde transport of viral vectors following intramuscular injection allows for targeted gene delivery to motoneuron pools. Gene delivery to the hypoglossal (XII) motoneurons is of potential interest in disorders involving impaired tongue muscle control such as dysphagia, dysarthria, and possibly obstructive sleep apnea. Using a murine model of Pompe disease, our group has shown that a single intramuscular injection of 1.00e11 vector genomes (vg) of recombinant adeno‐associated virus serotype 9 (AAV9) into the base of the tongue produces sustained transgene expression in a subset of XII mot

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HEIANZA, YORIKO, QIAOCHU XUE, JENNIFER ROOD, GEORGE BRAY, FRANK SACKS, and LU QI. "48-LB: Changes in Plasma Levels of Nonnutritive Sweetener Erythritol Are Related to Two-Year Changes of Insulin Sensitivity in Response to Weight-Loss Diets—The POUNDS Lost trial." Diabetes 72, Supplement_1 (2023). http://dx.doi.org/10.2337/db23-48-lb.

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Evidence on the benefits of non-nutritive sweeteners for weight management and improving insulin sensitivity and glucose metabolism is inconsistent. Recently, high levels of circulating erythritol, a sugar alcohol used as one of the non-nutritive sweeteners, have been linked to increased risks of type 2 diabetes and its vascular complications. Whether changes in circulating erythritol levels are related to insulin sensitivity and glucose metabolism remains unknown. Here, we investigated whether changes in plasma erythritol induced by weight-loss diet interventions were related to improved insu

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PALANISAMY, SRIKANTH, UGUR PARLATAN, MEHMET OZEN, et al. "1712-P: Serum miRNA-1 Levels as a Potential Marker for Myocardial Steatosis in Normal Weight and Obese/Overweight Diabetic Individuals—A Pilot Study." Diabetes 72, Supplement_1 (2023). http://dx.doi.org/10.2337/db23-1712-p.

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Of the 37 million Americans with type II diabetes, a substantial proportion have a normal body mass index (BMI). These populations suffer a disproportionately high burden of mortality; the development of diabetic cardiomyopathy (DCM) is likely a contributor. Myocardial steatosis, a known consequence of diabetes, is a significant part of DCM's pathogenesis; unfortunately, there is no feasible way to screen patients for steatosis. Exosomal microRNAs (miRNAs) are a promising biomarker for this purpose. However, no study has evaluated miRNA signatures in normal-weight patients with diabetes. Serum

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11

Turner, Sara, Aaron Hoyt, Darin Falk, Barry Byrne, and David Fuller. "Genome‐wide Assessment of the Pompe (Gaa⁻^/⁻) Mouse Cervical Spinal Cord Confirms Widespread Neuropathology." FASEB Journal 30, S1 (2016). http://dx.doi.org/10.1096/fasebj.30.1_supplement.1285.2.

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Pompe disease, caused by deficiency of acid alpha‐glucosidase (GAA), leads to glycogen accumulation, disruption of cellular architecture and neuromuscular impairments. Respiratory and lingual motor systems often decline first, and respiratory motoneurons typically show profound histopathology and glycogen accumulation at an early age. Although neuropathology is now firmly established in Pompe tissues from both humans and animal models, the associated mechanisms have not been evaluated. Therefore, we studied the cervical spinal cord of adult Pompe (Gaa−/−) and wild‐type (129SVE) mice using hist

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Bertaux‐Skeirik, Nina, Mark Wunderlich, Emma Teal, et al. "CD44v9 Emerges in Response to Injury and Contributes to the Regeneration of the Gastric Epithelium." FASEB Journal 31, S1 (2017). http://dx.doi.org/10.1096/fasebj.31.1_supplement.893.1.

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Cluster‐of‐differentiation gene 44, in particular CD44 variant isoforms (CD44v), emerges during regeneration of the gastric epithelium in response to injury. In particular, CD44v9 is expressed within Spasmolytic Polypeptide/TFF2‐Expressing Metaplasia (SPEM) glands during gastric repair, but the function is unknown. Here we tested the hypothesis that CD44v9 marks a regenerative cell lineage that plays a functional role in gastric repair. Acetic acid injury was induced in CD44‐deficient (CD44KO) and C57BL/6 (BL6) mice. Mouse‐derived gastric organoids expressing CD44v9 were transplanted at the ul

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Øyen, Jannike, Lise Madsen, Anne Lise Brantsæter, Svetlana Ondrasova Skurtveit, and Grace Margrethe Egeland. "Lean Fish Intake Decreases the Risk of Type 2 Diabetes Mellitus in Norwegian Women (P18-036-19)." Current Developments in Nutrition 3, Supplement_1 (2019). http://dx.doi.org/10.1093/cdn/nzz039.p18-036-19.

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Abstract Objectives The potential impact of seafood consumption and long chain n-3 polyunsaturated fatty acids (LCn-3PUFA) supplement use on the development of insulin resistance and type 2 diabetes mellitus (T2DM) is not yet fully clarified. The aim of this large cohort study was to investigate the associations between prenatal intake of total seafood, lean fish, fatty fish and LCn-3PUFA supplement use and the risk of T2DM in women after pregnancy. Methods The study subjects (n = 60 831, median age 31 years) participates in the ongoing population-based Norwegian Mother and Child Cohort study

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Doyle, Brendan Michael, Sara M. F. Turner, Amy Poirier, et al. "Comparison of treatment efficacy in the hypoglossal motor system of AAV‐DES‐coGAA and novel vector AAV‐DES‐IGFIIcoGAA in Gaa^−/− mice." FASEB Journal 31, S1 (2017). http://dx.doi.org/10.1096/fasebj.31.1_supplement.873.10.

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Pompe disease is a neuromuscular disorder characterized by a systemic lack of the glycogen metabolizing enzyme acid‐alpha glucosidase. The result is extensive intracellular lysosomal glycogen accumulation and disruption of cellular architecture and function. The hypoglossal (XII) motor system is particularly susceptible to pathology in Pompe disease and both tongue and XII motoneurons show substantial pathology early in the disease progression. Patients often present with macroglossia and tongue weakness, and animal models show pathology in tongue muscles and XII motoneurons. Initial clinical

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15

Denaro, Frank, Chozha Rathinam, Sumiko Williams, and Joseph Bryant. "A Small Animal Model to Study Cardiac Fibrosis in Chronic HIV Infection." Physiology 40, S1 (2025). https://doi.org/10.1152/physiol.2025.40.s1.0811.

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The effect of HIV on the heart is complex and continues throughout the lifetime of the individual. Without antiviral treatment opportunistic infections can cause serious damage. With antivirals the effect of opportunistic infection is mitigated. However, the continued existence of viable HIV in the reservoirs creates a state of persistent inflammation which contributes to cardiac abnormalities. Typically, inflammatory monocytes and neutrophils are not present in the healthy heart but when there is tissue damage they can be found at those sites. Cardiac macrophages on the other hand reside in t

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Li, Yilang, Josue Zambrano-Carrasco, Yang Cui, Jie Li, and Huabo Su. "OTUD6B Regulates Ventricular Chamber Maturation and Is Required for Mouse Embryonic Development." Physiology 40, S1 (2025). https://doi.org/10.1152/physiol.2025.40.s1.0884.

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Deubiquitinases (DUBs) regulate protein stability and various cellular processes by removing ubiquitin from substrate proteins. OTUD6B, a member of the OTU DUB family, has been linked to intellectual disabilities and congenital heart defects through compound heterozygous or homozygous variants in the OTUD6B gene. However, its molecular and pathophysiological roles in the heart remain poorly understood. Here, we demonstrate that OTUD6B is essential for ventricular chamber development and postnatal survival. OTUD6B is ubiquitously expressed in mouse embryos. Hypomorphic Otud6b neo/neo mice, with

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Gendlina, Inessa, and Kami Kim. "Toxoplasma gondii Reprogram Metabolism of the Host During Infection." FASEB Journal 31, S1 (2017). http://dx.doi.org/10.1096/fasebj.31.1_supplement.lb204.

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Toxoplasma gondii is an obligate intracellular eukaryotic parasite able to infect mammals including humans. After initial infection, people can remain latently infected for many years, however, parasite reactivation in people who have AIDS or are otherwise immunocompromised, and during pregnancy, has dramatic deleterious consequences. Both host and parasite factors are responsible for disease progression, and in recent years it has become evident that the host‐parasite interplay is complex with significant amount of crosstalk. As an obligate intracellular parasite that is dependent on the host

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URBINA, ELAINE M., SCOTT ISOM, DANA DABELEA, et al. "102-OR: Association of Elevated Arterial Stiffness with Cardiac Target Organ Damage in Young Adults with Diabetes: The Search for Diabetes in Youth Study." Diabetes 71, Supplement_1 (2022). http://dx.doi.org/10.2337/db22-102-or.

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Arterial stiffness and echocardiograms were assessed in participants with youth-onset DM (N=399, 22.8 ± 5.1 years, duration 10.8 ± 3.2 years, 63% female, 45% non-Hispanic White; 33% non-Hispanic Black, 13.5% Hispanic, 8% Other, 44% type 2 DM (T2D)) . Cardiovascular risk factors (CVRF) , arterial stiffness and cardiac measures were compared between those with or without elevated arterial stiffness (stiff = Pulse Wave Velocity [PWV] ≥ 90th% for lean controls) . Association between PWV and cardiac parameters after adjusting for CVRF was assessed. Participants with high PWV were 4.6 yrs older, had

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Xu, Jiaxi, Srinivas Sriramula, and Eric Lazartigues. "Neuronal activation of ADAM17 by AT1A receptors contributes to neurogenic hypertension." FASEB Journal 30, S1 (2016). http://dx.doi.org/10.1096/fasebj.30.1_supplement.757.5.

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We previously reported that neurogenic hypertension is associated with a reduction of Angiotensin Converting Enzyme 2 (ACE2) activity and an increase in A Disintegrin And Metalloprotease 17 (ADAM17) activity in the hypothalamus. In addition, we showed that silencing ADAM17 or blocking Angiotensin (Ang)‐II type 1 (AT1) receptors in the central nervous system (CNS) prevented DOCA‐salt hypertension, confirming the pivotal role of AT1R and ADAM17 in neurogenic hypertension. However, the interaction between AT1 receptors, ADAM17 and ACE2 is still unclear. Since ADAM17 is known to be expressed in mu

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Gibson, Breanne, Stephanie Moore‐Lotridge, Nicholas Mignemi, Gregory Hawley, William Oelsner, and Jonathan Schoenecker. "The Consumption of Plasminogen Following Severe Burn and Its Implications in Muscle Calcification." FASEB Journal 31, S1 (2017). http://dx.doi.org/10.1096/fasebj.31.1_supplement.390.4.

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INTRODUCTIONBurn and blast injuries present with serious risk for complications including fibrosis and calcification of the soft tissues. Plasmin, the active form of plasminogen, is the key serine protease responsible for the degradation of fibrin (fibrinolysis). Our lab has previously shown that plasmin is essential for the prevention of muscle fibrosis and calcification following injury. We hypothesized that 1) plasminogen is consumed in response to severe burn, and 2) this transient plasminogen deficiency impairs proper muscle regeneration.METHODSTo test our first hypothesis in humans, we c

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Senarathna, Janaka, Julia Brill, Shruthi Bare, Devorah Vanness, David Linden, and Arvind Pathak. "Image-based “Neurosurveillance” Reveals How Seizures Disrupt the Brain’s Microcirculation." Physiology 40, S1 (2025). https://doi.org/10.1152/physiol.2025.40.s1.0628.

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Although many neurological diseases (e.g. Alzheimer’s disease, brain cancers etc.) are characterized by disruptions in the brain’s microcirculation, it has been challenging to continuously track such microcirculatory changes over hours/days. This is largely due to the limitations of current imaging systems (e.g. MRI/PET) that require animals to be anesthetized during imaging, which also precludes continuous long-term imaging over the duration of the neuropathology being studied. To circumvent these limitations and image microcirculatory dysfunction during disease progression, we developed a no

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Oliveira, Rebeca, Janaka Senarathna, Vu Dinh, Aleksander Popel, and Arvind Pathak. "An Image-informed Computational Model for Predicting Spatiotemporal Variations in Cerebral Blood Flow." Physiology 40, S1 (2025). https://doi.org/10.1152/physiol.2025.40.s1.0793.

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To better understand the control of cerebral blood flow (CBF) and the effect of different CNS pathologies such as seizures on brain hemodynamics, several computational models have been developed. However, many of these models do not recapitulate spatiotemporal CBF variations or pathology-related CBF changes. Recent technological advances in in vivo imaging have enabled us to more accurately account for the microstructural and functional features of the cerebral vasculature in such models and validate their hemodynamic predictions. Therefore, here we present a novel “image-informed model” of te

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Caramaschi, Doretta, James Jungius, Christian M. Page, et al. "Association of medically assisted reproduction with offspring cord blood DNA methylation across cohorts." Human Reproduction, June 17, 2021. http://dx.doi.org/10.1093/humrep/deab137.

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Abstract STUDY QUESTION Is cord blood DNA methylation associated with having been conceived by medically assisted reproduction? SUMMARY ANSWER This study does not provide strong evidence of an association of conception by medically assisted reproduction with variation in infant blood cell DNA methylation. WHAT IS KNOWN ALREADY Medically assisted reproduction consists of procedures used to help infertile/subfertile couples conceive, including ART. Due to its importance in gene regulation during early development programming, DNA methylation and its perturbations associated with medically assist

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