Relevant bibliographies by topics / 11C-Deuterium-L-deprenyl

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Academic literature on the topic '11C-Deuterium-L-deprenyl'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "11C-Deuterium-L-deprenyl"

1

Logan, Jean, Joanna S. Fowler, Nora D. Volkow, Gene-Jack Wang, Robert R. MacGregor, and Colleen Shea. "Reproducibility of repeated measures of deuterium substituted [11C]L-deprenyl ([11C]L-deprenyl-D2) binding in the human brain." Nuclear Medicine and Biology 27, no. 1 (2000): 43–49. http://dx.doi.org/10.1016/s0969-8051(99)00088-8.

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2

Rodriguez-Vieitez, Elena, Stephen F. Carter, Konstantinos Chiotis, et al. "Comparison of Early-Phase 11C-Deuterium-L-Deprenyl and 11C-PiB PET for Assessing Brain Perfusion in Alzheimer's Disease." Journal of Nuclear Medecine 57, no. 7 (2016): 1071–7. https://doi.org/10.2967/jnumed.115.168732.

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The PET tracer <sup>11</sup>C-deuterium-L-deprenyl (<sup>11</sup>C-DED) has been used to visualize activated astrocytes in vivo in patients with Alzheimer&#39;s disease (AD). In this multitracer PET study, early-phase <sup>11</sup>C-DED and <sup>11</sup>C-Pittsburgh compound-B (<sup>11</sup>C-PiB) (eDED and ePiB, respectively) were compared as surrogate markers of brain perfusion, and the extent to which <sup>11</sup>C-DED binding is influenced by brain perfusion was investigated. METHODS:<sup> 11</sup>C-DED, <sup>11</sup>C-PiB and <sup>18</sup>F-fluorodeoxyglucose (FDG) dynamic PET scans were
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3

Vilaplana, Eduard, Elena Rodriguez-Vieitez, Daniel Ferreira, et al. "Cortical microstructural correlates of astrocytosis in autosomal-dominant Alzheimer disease." Neurology 94, no. 19 (2020): e2026-e2036. http://dx.doi.org/10.1212/wnl.0000000000009405.

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ObjectiveTo study the macrostructural and microstructural MRI correlates of brain astrocytosis, measured with 11C-deuterium-L-deprenyl (11C-DED)–PET, in familial autosomal-dominant Alzheimer disease (ADAD).MethodsThe total sample (n = 31) comprised ADAD mutation carriers (n = 10 presymptomatic, 39.2 ± 10.6 years old; n = 3 symptomatic, 55.5 ± 2.0 years old) and noncarriers (n = 18, 44.0 ± 13.7 years old) belonging to families with mutations in either the presenilin-1 or amyloid precursor protein genes. All participants underwent structural and diffusion MRI and neuropsychological assessment, a
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4

Farid, Karim, Stephen F. Carter, Elena Rodriguez-Vieitez, et al. "Case Report of Complex Amyotrophic Lateral Sclerosis with Cognitive Impairment and Cortical Amyloid Deposition." Journal of Alzheimer's Disease 47, no. 3 (2015): 661–67. https://doi.org/10.3233/JAD-141965.

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Amyotrophic lateral sclerosis (ALS), a fatal disease of unknown origin, affects motor neurons in the primary motor cortex, brainstem, and spinal cord. Cognitive impairment may occur before the motor symptoms. We present a patient who was initially diagnosed with mild cognitive impairment (MCI) due to Alzheimer&rsquo;s disease (AD) but who developed ALS-like symptoms during follow-up and died shortly thereafter. A 60-year-old subject with cognitive impairment underwent neuropsychological testing, cerebrospinal fluid (CSF) analysis, structural imaging (computed tomography and magnetic resonance
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5

Rodriguez-Vieitez, E., S. F. Carter, K. Chiotis, et al. "Comparison of Early-Phase 11C-Deuterium-L-Deprenyl and 11C-Pittsburgh Compound B PET for Assessing Brain Perfusion in Alzheimer Disease." Journal of Nuclear Medicine 57, no. 7 (2016): 1071–77. http://dx.doi.org/10.2967/jnumed.115.168732.

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6

Rodriguez-Vieitez, Elena, Ruiqing Ni, Balázs Gulyás, et al. "Astrocytosis precedes amyloid plaque deposition in Alzheimer APPswe transgenic mouse brain: a correlative positron emission tomography and in vitro imaging study." Eur J Nucl Med Mol Imaging. 42, no. 7 (2015): 1119–32. https://doi.org/10.1007/s00259-015-3047-0.

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PURPOSE: Pathological studies suggest that neuroinflammation is exacerbated by increased beta-amyloid (A&beta;) levels in the brain early in Alzheimer&#39;s disease (AD). The time course and relationships between astrocytosis and A&beta; deposition were examined using multitracer in vivo positron emission tomography (PET) imaging in an AD transgenic mouse model, followed by postmortem autoradiography and immunohistochemistry analysis. METHODS: PET imaging with the amyloid plaque tracer (11)C-AZD2184 and the astroglial tracer (11)C-deuterium-L-deprenyl ((11)C-DED) was carried out in APPswe mice
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7

Carter, S. F., M. Scholl, O. Almkvist, et al. "Evidence for Astrocytosis in Prodromal Alzheimer Disease Provided by 11C-Deuterium-L-Deprenyl: A Multitracer PET Paradigm Combining 11C-Pittsburgh Compound B and 18F-FDG." Journal of Nuclear Medicine 53, no. 1 (2012): 37–46. http://dx.doi.org/10.2967/jnumed.110.087031.

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8

Rodriguez-Vieitez, Elena, Karen Butina, Stephen F. Carter, and Agneta Nordberg. "P3-214: COMPARISON OF MEASUREMENTS OF CEREBRAL BLOOD FLOW BY EARLY FRAMES OF 11C-DEUTERIUM-L-DEPRENYL (11C-DED) AND 11C-PIB PET TRACERS AT DIFFERENT STAGES OF ALZHEIMER'S DISEASE." Alzheimer's & Dementia 10 (July 2014): P709. http://dx.doi.org/10.1016/j.jalz.2014.05.1305.

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9

Fowler, J. S., A. P. Wolf, R. R. Macgregor, et al. "Probing the biochemical basis of the PET image. Mechanistic PET studies of [11C]L-deprenyl binding using the deuterium isotope effect." Journal of Labelled Compounds and Radiopharmaceuticals 26, no. 1-12 (1989): 236–37. http://dx.doi.org/10.1002/jlcr.25802601107.

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10

Rodriguez-Vieitez, Elena, Karen Butina, Stephen F. Carter, and Agneta K. Nordberg. "IC-03-03: COMPARISON OF MEASUREMENTS OF CEREBRAL BLOOD FLOW BY EARLY FRAMES OF 11C-DEUTERIUM-L-DEPRENYL (11C-DED) AND 11C-PIB PET TRACERS AT DIFFERENT STAGES OF ALZHEIMER'S DISEASE." Alzheimer's & Dementia 10 (July 2014): P7. http://dx.doi.org/10.1016/j.jalz.2014.04.032.

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