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Relevant bibliographies by topics / 18F-Fluorodeprenyl-D2

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Academic literature on the topic '18F-Fluorodeprenyl-D2'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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Journal articles on the topic "18F-Fluorodeprenyl-D2"

1

Nag, Sangram, Patrik Fazio, Lutz Lehmann, et al. "In Vivo/In Vitro Characterisation of a Novel MAO-B Inhibitor Radioligand, Fluorine-18 Labeled Deuterated Fluorodeprenyl (18F-Fluorodeprenyl-D2)." Journal of Nuclear Medecine 57, no. 2 (2015): 315–20. https://doi.org/10.2967/jnumed.115.161083.

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INTRODUCTION: The aim of this study was to radiolabel a novel bis-deuterium substituted L-deprenyl analogue (<sup>18</sup>F-fluorodeprenyl-D<sub>2</sub>) with fluorine-18 and to evaluate its potential to visualize and quantify monoamine oxidase B (MAO-B) activity in vivo. METHODS: The precursor compound (5a + 5b) and reference standard (6) were synthesized in multi-step syntheses. Recombinant human MAO-B and MAO-A enzyme preparations were used in order to determine IC50 values. Radiolabeling was accomplished by nucleophilic substitution reaction. Whole hemisphere autoradiography (ARG) was perf

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2

Ko, Nare, Sang Ju Lee, Byung Seok Moon, Jae Seung Kim, and Seung Jun Oh. "A stable and robust production process of [18F]fluorodeprenyl-D2 using a cassette-type chemistry module." Nuclear Medicine and Biology 126-127 (November 2023): 108522. http://dx.doi.org/10.1016/j.nucmedbio.2023.108522.

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3

Tagnin, L., J. Dornreich, M. Zeinaki, et al. "Feasibility of Short Imaging Protocols for [18F]fluorodeprenyl-D2 PET in Autoimmune Encephalitis and Multiple System Atrophy." Nuklearmedizin - NuclearMedicine 64, no. 01 (2025): 83. https://doi.org/10.1055/s-0045-1804388.

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4

Rauchmann, Boris‐Stephan, Selim Üstün Guersel, Carolin Isabella Kurz, et al. "Investigating Reactive Astrocytosis in Alzheimer's Disease Using [18F]Fluorodeprenyl‐D2 PET Imaging." Alzheimer's & Dementia 20, S2 (2024). https://doi.org/10.1002/alz.092151.

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AbstractBackgroundAlzheimer's Disease (AD) is often accompanied by neuroinflammation, which manifests prior to significant cognitive decline. Reactive astrocytosis is a hallmark of such inflammation, potentially serving as an early biomarker for AD pathology. Our study employs [18F]fluorodeprenyl‐D2 ([18F]F‐DED) positron emission tomography (PET) imaging to in vivo quantify astrocytosis comparing AD with healthy controls and examines its assocciation with cognitive deterioration in AD.MethodOur cross‐sectional study involved 12 early‐stage Alzheimer's Disease (AD) patients and 11 healthy contr

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5

Ballweg, Anna, Carolin Klaus, Letizia Vogler, et al. "[18F]F-DED PET imaging of reactive astrogliosis in neurodegenerative diseases: preclinical proof of concept and first-in-human data." Journal of Neuroinflammation 20, no. 1 (2023). http://dx.doi.org/10.1186/s12974-023-02749-2.

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Abstract Objectives Reactive gliosis is a common pathological hallmark of CNS pathology resulting from neurodegeneration and neuroinflammation. In this study we investigate the capability of a novel monoamine oxidase B (MAO-B) PET ligand to monitor reactive astrogliosis in a transgenic mouse model of Alzheimer`s disease (AD). Furthermore, we performed a pilot study in patients with a range of neurodegenerative and neuroinflammatory conditions. Methods A cross-sectional cohort of 24 transgenic (PS2APP) and 25 wild-type mice (age range: 4.3–21.0 months) underwent 60 min dynamic [18F]fluorodepren

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