Relevant bibliographies by topics / [18F]flutemetamol

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters

Academic literature on the topic '[18F]flutemetamol'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "[18F]flutemetamol"

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Kang, ShinWoo, Jinho Kim, Sang-Yoon Lee, Nobuyuki Okamura, and Keun-A. Chang. "MicroPET Imaging Assessment of Brain Tau and Amyloid Deposition in 6 × Tg Alzheimer’s Disease Model Mice." International Journal of Molecular Sciences 23, no. 10 (2022): 5485. http://dx.doi.org/10.3390/ijms23105485.

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Alzheimer’s disease (AD) is characterized by the deposition of extracellular amyloid plaques and intracellular accumulation of neurofibrillary tangles (NFT). Amyloid beta (Aβ) and tau imaging are widely used for diagnosing and monitoring AD in clinical settings. We evaluated the pathology of a recently developed 6 × Tg − AD (6 × Tg) mouse model by crossbreeding 5 × FAD mice with mice expressing mutant (P301L) tau protein using micro-positron emission tomography (PET) image analysis. PET studies were performed in these 6 × Tg mice using [18F]Flutemetamol, which is an amyloid PET radiotracer; [1
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Ossenkoppele, Rik, Ruben Smith, Tomas Ohlsson та ін. "Associations between tau, Aβ, and cortical thickness with cognition in Alzheimer disease". Neurology 92, № 6 (2019): e601-e612. http://dx.doi.org/10.1212/wnl.0000000000006875.

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ObjectiveTo examine the cross-sectional associations between regional tau, β-amyloid (Aβ), and cortical thickness and neuropsychological function across the preclinical and clinical spectrum of Alzheimer disease (AD).MethodsWe included 106 participants from the Swedish Biomarkers for Identifying Neurodegenerative Disorders Early and Reliably (BioFINDER) study, of whom 33 had preclinical AD (Aβ-positive cognitively normal individuals), 25 had prodromal AD (Aβ-positive mild cognitive impairment), and 48 had probable AD dementia. All underwent [18F]flortaucipir (tau) and structural MRI (cortical
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Hellberg, Sanna, Johanna Silvola, Heidi Liljenbäck, et al. "Amyloid-Targeting PET Tracer [18F]Flutemetamol Accumulates in Atherosclerotic Plaques." Molecules 24, no. 6 (2019): 1072. http://dx.doi.org/10.3390/molecules24061072.

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Atherosclerosis is characterized by the accumulation of oxidized lipids in the artery wall, which triggers an inflammatory response. Oxidized low-density lipoprotein (ox-LDL) presents amyloid-like structural properties, and different amyloid species have recently been recognized in atherosclerotic plaques. Therefore, we studied the uptake of the amyloid imaging agent [18F]Flutemetamol in atherosclerotic plaques. The binding of [18F]Flutemetamol to human carotid artery plaque was studied in vitro. In vivo uptake of the tracer was studied in hypercholesterolemic IGF-II/LDLR−/−ApoB100/100 mice an
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Müller, Ebba Gløersen, Trine Holt Edwin, Bjørn Heine Strand, Caroline Stokke, Mona Elisabeth Revheim, and Anne-Brita Knapskog. "Is Amyloid Burden Measured by 18F-Flutemetamol PET Associated with Progression in Clinical Alzheimer’s Disease?" Journal of Alzheimer's Disease 85, no. 1 (2022): 197–205. http://dx.doi.org/10.3233/jad-215046.

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Background: Patients with Alzheimer’s disease (AD) show heterogeneity in clinical progression rate, and we have limited tools to predict prognosis. Amyloid burden from 18F-Flutemetamol positron emission tomography (PET), as measured by standardized uptake value ratios (SUVR), might provide prognostic information. Objective: We investigate whether 18F-Flutemetamol PET composite or regional SUVRs are associated with trajectories of clinical progression. Methods: This observational longitudinal study included 94 patients with clinical AD. PET images were semi-quantified with normalization to pons
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Hammers, Dustin B., Taylor J. Atkinson, Bonnie C. A. Dalley, et al. "Amyloid Positivity Using [18F]Flutemetamol-PET and Cognitive Deficits in Nondemented Community-Dwelling Older Adults." American Journal of Alzheimer's Disease & Other Dementiasr 32, no. 6 (2017): 320–28. http://dx.doi.org/10.1177/1533317517698795.

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Little research exists examining the relationship between beta-amyloid neuritic plaque density via [18F]flutemetamol binding and cognition; consequently, the purpose of the current study was to compare cognitive performances among individuals having either increased amyloid deposition (Flute+) or minimal amyloid deposition (Flute−). Twenty-seven nondemented community-dwelling adults over the age of 65 underwent [18F]flutemetamol amyloid-positron emission tomography imaging, along with cognitive testing using the Repeatable Battery for the Assessment of Neuropsychological Status (RBANS) and sel
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Walker, Z., F. Inglis, C. Sadowsky, et al. "Reproducibility of [18f]flutemetamol pet amyloid image interpretation." Journal of the Neurological Sciences 333 (October 2013): e352. http://dx.doi.org/10.1016/j.jns.2013.07.1294.

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Heeman, Fiona, Maqsood Yaqub, Janine Hendriks, et al. "Parametric imaging of dual-time window [18F]flutemetamol and [18F]florbetaben studies." NeuroImage 234 (July 2021): 117953. http://dx.doi.org/10.1016/j.neuroimage.2021.117953.

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Ikonomovic, Milos D., Christopher J. Buckley, Eric E. Abrahamson та ін. "Post-mortem analyses of PiB and flutemetamol in diffuse and cored amyloid-β plaques in Alzheimer’s disease". Acta Neuropathologica 140, № 4 (2020): 463–76. http://dx.doi.org/10.1007/s00401-020-02175-1.

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Abstract Specificity and sensitivity of positron emission tomography (PET) radiopharmaceuticals targeting fibrillar amyloid-β (Aβ) deposits is high for detection of neuritic Aβ plaques, a mature form of Aβ deposits which often have dense Aβ core (i.e., cored plaques). However, imaging-to-autopsy validation studies of amyloid PET radioligands have identified several false positive cases all of which had mainly diffuse Aβ plaques (i.e., plaques without neuritic pathology or dense amyloid core), and high amyloid PET signal was reported in the striatum where diffuse plaques predominate in Alzheime
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Yeo, Jing Ming, Briony Waddell, Zubair Khan, and Suvankar Pal. "A SYSTEMATIC REVIEW & META-ANALYSIS OF F-18-LABELLED AMYLOID IMAGING IN ALZHEIMER'S DISEASE." Journal of Neurology, Neurosurgery & Psychiatry 86, no. 11 (2015): e4.142-e4. http://dx.doi.org/10.1136/jnnp-2015-312379.51.

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IntroductionThere has been recent interest in the use of fluorine-18-labelled (18F) tracers in amyloid imaging as they have longer half-lives compared to 11C-labelled Pittsburgh compound-B (11C-PIB). This systematic review and meta-analysis aims to assess the sensitivity and specificity of 18F tracers florbetapir, florbetaben and flutemetamol in diagnosing Alzheimer's disease (AD).MethodsWe systematically searched MEDLINE and EMBASE for relevant studies published from January 1980 to March 2014. We pooled the studies comparing imaging findings in AD and normal controls (NC) in a meta-analysis,
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Fiona, Heeman, Yaqub Maqsood, Lopes Alves Isadora, et al. "Simulating the effect of cerebral blood flow changes on regional quantification of [18F]flutemetamol and [18F]florbetaben studies." Journal of Cerebral Blood Flow & Metabolism 41, no. 3 (2020): 579–89. https://doi.org/10.1177/0271678X20918029.

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<strong>Abstract</strong> Global and regional changes in cerebral blood flow (CBF) can result in biased quantitative estimates of amyloid load by PET imaging. Therefore, the current simulation study assessed effects of these changes on amyloid quantification using a reference tissue approach for [<sup>18</sup>F]flutemetamol and [<sup>18</sup>F]florbetaben. Previously validated pharmacokinetic rate constants were used to simulate time-activity curves (TACs) corresponding to full dynamic and dual-time-window acquisition protocols. CBF changes were simulated by varying the tracer delivery (<em>K<
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Dissertations / Theses on the topic "[18F]flutemetamol"

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Lilja, Johan. "[18F]Flutemetamol PET image processing, visualization and quantification targeting clinical routine." Doctoral thesis, Uppsala universitet, Radiologi, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-317688.

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Alzheimer’s disease (AD) is the leading cause of dementia and is alone responsible for 60-70% of all cases of dementia. Though sharing clinical symptoms with other types of dementia, the hallmarks of AD are the abundance of extracellular depositions of β-amyloid (Aβ) plaques, intracellular neurofibrillary tangles of hyper phosphorylated tau proteins and synaptic depletion. The onset of the physiological hallmarks may precede clinical symptoms with a decade or more, and once clinical symptoms occur it may be difficult to separate AD from other types of dementia based on clinical symptoms alone.
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Heurling, Kerstin. "Characterization of [18F]flutemetamol binding properties : A β-amyloid PET imaging ligand". Doctoral thesis, Uppsala universitet, Radiologi, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-262019.

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The criteria for diagnosing Alzheimer’s disease (AD) have recently been revised to include the use of biomarkers for the in vivo presence of β-amyloid, one of the neuropathological hallmarks of AD. Examples of such biomarkers are positron emission tomography (PET) β-amyloid specific ligands, including [18F]flutemetamol. The aim of this thesis was to characterize the binding properties of [18F]flutemetamol from a tracer kinetic perspective as well as by validating binding measures through comparison with tissue pathology assessments. The applicability of previously developed kinetic models of t
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Book chapters on the topic "[18F]flutemetamol"

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Ballinger, James R. "18F-Flutemetamol." In PET Radiopharmaceuticals. Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-10271-4_46.

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