Academic literature on the topic '2-Ethylhexanol'

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Journal articles on the topic "2-Ethylhexanol"

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Keith, Y., M. C. Cornu, P. M. Canning, J. Foster, J. C. Lhuguenot, and C. R. Elcombe. "Peroxisome proliferation due to di (2-ethylhexyl) adipate, 2-ethylhexanol and 2-ethylhexanoic acid." Archives of Toxicology 66, no. 5 (May 1992): 321–26. http://dx.doi.org/10.1007/bf01973626.

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Miura, Motofumi, Masaharu Toriyama, and Shigeyasu Motohashi. "Novel Synthesis of Optically Active 2‐Ethylhexanoic Acid, 2‐Ethylhexanol, and 2‐Ethylhexylamine via the Asymmetric Favorskii Rearrangement." Synthetic Communications 36, no. 3 (January 2006): 259–64. http://dx.doi.org/10.1080/00397910500374740.

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Ritter, E. J., W. J. Scott, J. L. Randall, and J. M. Ritter. "Teratogenicity of di(2-ethylhexyl) phthalate, 2-ethylhexanol, 2-ethylhexanoic acid, and valproic acid, and potentiation by caffeine." Teratology 35, no. 1 (February 1987): 41–46. http://dx.doi.org/10.1002/tera.1420350107.

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Bui, Linh M., Marie W. Taubeneck, Joel F. Commisso, Janet Y. Uriu-Hare, Willem D. Faber, and Carl L. Keen. "Altered zinc metabolism contributes to the developmental toxicity of 2-ethylhexanoic acid, 2-ethylhexanol and valproic acid." Toxicology 126, no. 1 (February 1998): 9–21. http://dx.doi.org/10.1016/s0300-483x(97)00171-6.

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Bhutada, S. R., and V. G. Pangarkar. "Esterification of phthalic anhydride with 2-ethylhexanol." Journal of Chemical Technology & Biotechnology 36, no. 2 (April 24, 2007): 61–66. http://dx.doi.org/10.1002/jctb.280360204.

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Nalli, Sandro, Owen J. Horn, Adam R. Grochowalski, David G. Cooper, and Jim A. Nicell. "Origin of 2-ethylhexanol as a VOC." Environmental Pollution 140, no. 1 (March 2006): 181–85. http://dx.doi.org/10.1016/j.envpol.2005.06.018.

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Liang, Ning, Xiaolong Zhang, Hualiang An, Xinqiang Zhao, and Yanji Wang. "Direct synthesis of 2-ethylhexanol via n-butanal aldol condensation–hydrogenation reaction integration over a Ni/Ce-Al2O3 bifunctional catalyst." Green Chemistry 17, no. 5 (2015): 2959–72. http://dx.doi.org/10.1039/c5gc00223k.

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Darracq, G., A. Couvert, C. Couriol, A. Amrane, and P. Le Cloirec. "Absorption and biodegradation of hydrophobic volatile organic compounds: determination of Henry's constants and biodegradation levels." Water Science and Technology 59, no. 7 (April 1, 2009): 1315–22. http://dx.doi.org/10.2166/wst.2009.124.

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Biodegradation of three volatile organic compounds (VOCs) was studied. Toluene, dimethylsulphide (DMS), and dimethyldisulphide (DMDS) were introduced into flasks filled with emulsions of Di-2-EthylHexylAdipate (DEHA) in water, containing biomass (activated sludge). The VOC concentrations were analysed in the gas, organic and aqueous phases, and compared to the initial VOC quantities introduced in order to deduce their consumption by biomass. Toluene and DMDS were completely consumed, and then removed from the gas and the organic phases, except when DEHA and water are in the same volume ratio, which appears to be extreme environmental conditions for bacterial growth. The high DMS volatility resulted in an important gas loss, leading to a lower amount of DMS available for activated sludge growth. For all the VOC experiments, some components, characteristics of the DEHA degradation, including 2-ethylhexanal, 2-ethylhexanol, 2-ethylhexanoic acid and adipic acid, were identified.
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Keith, Y., P. M. Canning, J. C. Lhuguenot, and C. R. Elcombe. "Peroxisome Proliferation due to di-(2-Ethylhexyl)adipate and 2-Ethylhexanol." Human Toxicology 4, no. 5 (September 1985): 551–52. http://dx.doi.org/10.1177/096032718500400540.

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KELLER, B., D. LIANG, and R. THURMAN. "2-Ethylhexanol uncouples oxidative phosphorylation in rat liver mitochondria." Toxicology Letters 57, no. 1 (June 1991): 113–20. http://dx.doi.org/10.1016/0378-4274(91)90125-p.

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Dissertations / Theses on the topic "2-Ethylhexanol"

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Ellis, Shawn Alfred. "2-ethylhexanol, a potential biological indicator of occupational exposure to the plasticizer di(2-ethylhexyl)phthalate." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp04/mq28742.pdf.

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Dewidar, Assem A. "Impact of Biosurfactants on Biodegradation of a Binary Mixture of Hydrophilic and Hydrophobic VOCs in Trickle Bed Air Biofilter." University of Cincinnati / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1563272543717587.

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Dutescu, Ralf Michael [Verfasser], and H. G. [Akademischer Betreuer] Wahl. "Expressionsanalyse der nukleären Rezeptoren PPAR-α/γ-1/γ-2 [PPAR-alpha, gamma-1, gamma-2] und der Transkriptionsfaktoren T-bet und GATA-3 nach Stimulation von dermalen Endothelzellen mit den Weichmacher, Di(2-ethylhexyl)phthalat-Metaboliten-2-Ethylhexanol und 4-Heptanon / Ralf Michael Dutescu. Betreuer: H. G. Wahl." Marburg : Philipps-Universität Marburg, 2011. http://d-nb.info/1013288459/34.

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Costa, Daniela Filipa de Oliveira. "Hepatic differentiation of skin-derived stem cells and their application within in vitro toxicity testing." Master's thesis, 2016. http://hdl.handle.net/10451/34619.

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Trabalho Final de Mestrado Integrado, Ciências Farmacêuticas, Universidade de Lisboa, Faculdade de Farmácia, 2016
Steatosis is the hepatic accumulation of lipids. Despite being the least severe stage of Non-Alcoholic fatty liver disease, evaluation of steatosis is a mechanistic assay for toxin-induced liver injury, mandatory for the development of new chemical entities. Toxin-induced liver injury represent a major concern to the pharmaceutical industry. The discontinuation of the development of new compounds, in addition with the withdrawal of market drugs, represent an immense economic impact, prompting an immense pressure to develop a predictable human-based in vitro hepatic model. HepG2 and HepaRG cell lines are the most used in vitro models for hepatotoxic evaluation. Nevertheless, this cell models neglect to represent the human hepatic metabolism. Alternatively, stem-cells derived hepatic cells, specifically human skin-derived precursors differentiated into hepatic cells (hSKP-HPC), are emerging as a new potential hepatotoxic in vitro study system. So as to compare the potential of hSKP-HPC, to the other two hepatic cell lines, cells were incubated with three widely known steatogenic compounds, oleic acid, tetracycline and 2-ethylhexanol, and cell viability, lipid accumulation and gene expression analysis was performed. Preliminary results revealed significant difference in sensitivity of the three cell lines to hepatotoxins. Indeed, HepaRG revealed the lowest accumulation of neutral lipids, where hSKP-HPC was shown to be exceptionally sensitive. Moreover, different gene expression profiles between HepaRG and hSKP-HPC revealed distinct steatosis induction mechanisms. While tetracycline decreased efflux of FAs and increased de novo lipogenesis in hSKP-HPC, further contributing for steatosis phenotype, a compensatory mechanisms was found in HepaRG cells. A similar finding was made in HepaRG cells incubated with 2-ethylhexanol, where an increase uptake of FAs was perceived, while the opposite was found in hSKP-HPC cells. Despite these differences, some parallelisms in key pathogenic mechanisms were detected. Ultimately, our preliminary results demonstrated that hSKP-HPC are a possible in vitro model for hepatotoxic evaluation.
A esteatose é definida como a acumulação hepática de lípidos. Apesar de ser a condição mais benigna dentro do espectro do fígado gordo não alcoólico, a esteatose é um mecanismo de lesão hepática induzida por toxinas, de análise obrigatória no desenvolvimento de novas entidades químicas. Lesões hepáticas induzidas por toxinas são uma grande preocupação da indústria farmacêutica. A descontinuação do estudo de novos compostos, assim como a retirada de medicamentos do mercado representam um enorme impacto económico, originando uma enorme pressão para o desenvolvimento de um modelo in vitro hepático humano fiável. As linhas celulares HepG2 e HepaRG são os modelos in vitro para avaliação hepatotóxica mais comuns. No entanto, estes modelos falham em representar fielmente o metabolismo hepático humano. Em alternativa, células hepáticas derivadas de células estaminais, especificamente células hepáticas diferenciadas de precursores derivados da pele humana (hSKP-HPC), estão a surgir como um novo sistema in vitro para o estudo de hepatotóxicos. De forma a comparar a potencialidade deste sistema, as linhas celulares anteriormente referenciadas foram incubadas com três compostos esteatogénicos: ácido oleico, tetraciclina, 2-etilhexanol, e a viabilidade celular, acumulação lipídica e análise de expressão genética foi realizada. Resultados preliminares revelam uma diferença significativa na sensibilidade das três linhas celulares às hepatotoxinas. De facto, as células HepaRG revelaram pouca acumulação de lípidos neutros, enquanto hSKP-HPC demostrou ser um modelo celular particularmente sensível. Adicionalmente, diferentes padrões de expressão genética entre as culturas celulares HepaRG e hSKP-HPC sugerem a ativação de diferentes mecanismos de indução de esteatose. Enquanto a incubação com tetraciclina originou uma diminuição dos níveis de mRNA de proteínas envolvidas com o efluxo de ácidos gordos e um aumento da lipogénese nas células hSKP-HPC, que contribuem para o fenótipo de esteatose, um mecanismo de feedback negativo foi visualizado nas células HepaRG. Uma observação também encontrada aquando o tratamento com 2-etilhexanol, onde um aumento do uptake de ácidos gordos foi considerado um mecanismo de indução de esteatose, enquanto o oposto foi encontrado nas células hSKP-HPC. Apesar destas diferenças, alguns paralelismos em mecanismos chave na indução de esteatose foram detetados. Em última analise, resultados preliminares demostram que hSKP-HPC é um modelo in vitro com potencial para avaliação hepatotóxica.
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YE, WUN-DE, and 葉文德. "Isobaric vapor-liquid equilibrium for binary mixtures containing of n-octanol、2-Ethyl-1-hexanol、Octanoic acid、2-Ethylhexanoic acid and 2-Ethyl-2-hexenal." Thesis, 2017. http://ndltd.ncl.edu.tw/handle/59560869594737355402.

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Books on the topic "2-Ethylhexanol"

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Environment, Alberta Alberta. Assessment report on 2-ethylhexanol for developing ambient air quality objectives. [Edmonton]: Alberta Environment, 2004.

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2-ethylhexanol: A potential biological indicator of occupational exposure to the plasticizer di(2-ethylhexyl)phthalate. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1999.

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Book chapters on the topic "2-Ethylhexanol"

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van Lierop, Ben, Laurence Castle, Alexandre Feigenbaum, and Achim Boenke. "2-Ethylhexanoic acid." In Spectra for the Identification of Additives in Food Packaging, 185–89. Dordrecht: Springer Netherlands, 1998. http://dx.doi.org/10.1007/978-94-011-5222-8_36.

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Hong, Chua, Gek Sim Yap Miranda, and Jem Ng Wun. "A Mathematical Model for the Anaerobic Degradation of 2-Ethylhexanoic Acid." In Biochemical Engineering for 2001, 800–803. Tokyo: Springer Japan, 1992. http://dx.doi.org/10.1007/978-4-431-68180-9_213.

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"2-Ethylhexanol." In Toxicological Evaluations, 181–205. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-84195-8_11.

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"2-Ethylhexanol." In Toxicological Evaluations, 103–57. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-642-59835-7_7.

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Grzesik, M., and T. Gumula. "Kinetics models for esterification of levulinic acid with 2-ethylhexanol using different catalysts." In Reaction Kinetics and the Development and Operation of Catalytic Processes, Proceedings of the 3rd International Symposium, 547–51. Elsevier, 2001. http://dx.doi.org/10.1016/s0167-2991(01)82010-3.

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"Table IV: Volatile compounds identified in the dust of swine confinement units Hydrocarbons Ketones Hexane 1) Acetone 4) «t-Pinen 1) Butanone 4) Limonen 1) Pentanone 4) 3,7,7-Trimethyl -Octanone 4) bicyclo (3,1,1)-l-0ctene-3-one 4) 2-Hepten 1) Benzene 1) Acids Toluene 1) Acetic 4)5)6) Alcohols Propionic 4)5)6) i-Butyric 1)5)6) 1-Pentanol 1) Butyric 4)5)6) 1-Heptanol 1) 1-Valeric 5)6) 4-Methylcyclo-Valeric 4)5)6) hexanol 1) Hexanoic 4)5) 2-Ethylhexanol 1) Heptanoic 4) Octanoic 4) Phenols Nonanoic 4) Decanoic 4) Phenol 1)3)6) Undecanoic 4) p-Cresol 3)4)6) Dodecanoic 4) o-Cresol 1) Laurie 3) p-Ethylphenol 3)6) Tridecanoic 4) o-Ethylphenol 4) Tetradecanoic 4) m-Ethylphenol 4) Benzoic 4) Phenyl acetic 3)4) Indoles 3-Phenyl propionic 3) Hydrocinnamic 4) Indole 2)6) Skatole 2)3)6) Miscellaneous Compounds Aldehydes 2-Pentylfuran 3) Vanillin 3) Butanal 4) 2-Butenal 4) Pentanal 4) 2-Pentenal 4) Hexanal 1)3)4) 1) = WEURMAN (13) 2-Hexenal 4) 2) = TRAVIS and ELLIOTT (31) Heptanal 1)3) 2-Heptenal 4) 3) = HAMMOND et al. (30) 2.4-Heptadienal 3)4) 4) = HAMMOND et al. (40) Nonanal 3) 2-Nonenal 3) 5) = AENGST (33) 2.4-Nonadienal 3)4) 6) = HARTUNG (34) Decanal 4) 2.4-Decadienal 3)4) Benzaldehyde 1)4)." In Odour Prevention and Control of Organic Sludge and Livestock Farming, 344. CRC Press, 1986. http://dx.doi.org/10.1201/9781482286311-138.

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Conference papers on the topic "2-Ethylhexanol"

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Purdham, J., A. Sass-Kortsak, P. Bozek, and S. Ellis. "186. Feasibility Study on the Use of 2-Ethylhexanol as a Biological Indicator of Di-(2-Ethylhexyl)Phthalate Exposure." In AIHce 1999. AIHA, 1999. http://dx.doi.org/10.3320/1.2763023.

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Liu, Mingyan, Kwanghoon Baek, Fransico Argüelles Vivas, Gayan Aruna Abeykoon, and Ryosuke Okuno. "Effects of Surfactant Partitioning Coefficient and Interfacial Tension on the Oil Displacement in Low-Tension Polymer Flooding." In SPE Annual Technical Conference and Exhibition. SPE, 2021. http://dx.doi.org/10.2118/206220-ms.

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Abstract Complex surfactant formulations have been applied to generate an ultra-low interfacial tension (IFT) (e.g., 10-3 dyne/cm) between the displacing water phase and the displaced oil phase in chemical enhanced oil recovery (CEOR), where the residual oil after waterflooding can be largely recovered as an oil bank. This paper is concerned with a simpler, lower-cost CEOR, in which a sole additive of surface active solvent (SAS) makes low-tension displacement fronts in polymer flooding (e.g., 10-2 dyne/cm) without involving ultra-low IFT microemulsion phase behavior. The main objective of this research is to technically verify such low-tension polymer (LTP) flooding for a secondary-mode oil displacement through a sandpack of 9.5 Darcy. Previous research found that 2-ethylhexanol-7PO-15EO (2-EH-7PO-15EO, or "7-15") as SAS was able to reduce the IFT between polymer solution and the reservoir oil from 15.8 dyne/cm to 0.025 dyne/cm. In this research, the effect of SAS partition coefficient on LTP flooding was studied as an additional factor for SAS optimization. In particular, the comparison between two SAS species, 2-EH-4PO-15EO (4-15) and 2-EH-7PO-25EO (7-25), was important, because they had similar IFT values, but markedly different partition coefficients. The IFT was 0.18 dyne/cm with 4-15 and 0.20 dynes/cm with 7-25; and the partition coefficients were 1.61 with 4-15 and 0.68 with 7-25 at the experimental temperature, 61°C. These two SAS species were compared in secondary-mode LTP flooding with a slug of 0.5 wt% SAS for 0.5 pore-volumes injected (PVI). The oil recovery factor at 1.0 PVI was 65% with 4-15 and 67% with 7-25. At 5.0 PVI, it was 74% with 4-15 and 84% with 7-25. Although these two SAS species gave comparable IFT values, their oil-displacement efficiencies were quite different because 7-25 propagated more efficiently in the sandpack with the smaller partition coefficient. The smaller partition coefficient helped the SAS flow more efficiently in the aqueous phase with less retention in the remaining oil. Optimization of SAS likely requires taking a balance between lowering the partition coefficient and lowering the IFT. The SAS recovery at the effluent was 61% for the 4-15 SAS and 78% for the 7-25 SAS. The propagation of the 4-15 SAS was retarded approximately by 1.0 PVI in comparison to that of the 7-25 SAS. The adsorption of the 4-15 and 7-25 SAS were 0.019 mg/g sandpack and 0.020 mg/g sandpack. With a similar IFT reduction, the SAS with a smaller partition coefficient (i.e., 7-25) resulted in less retention, less retardation, and more oil production for a given amount of injection.
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