Relevant bibliographies by topics / 28-Homocastasterone

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Academic literature on the topic '28-Homocastasterone'

Author: Grafiati
Published: 3 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "28-Homocastasterone"

1

Elbert, Tomáš, Mahadeo R. Patil, and Aleš Marek. "Enantiospecific tritium labeling of 28-homocastasterone." Journal of Labelled Compounds and Radiopharmaceuticals 60, no. 3 (2017): 176–82. http://dx.doi.org/10.1002/jlcr.3488.

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2

Velan, Athithan. "In silico investigation of therapeutic potential of phytosteroids against SARS-CoV2 infection via inhibition of viral multiplication and attenuation of host cytokine storm." International Journal of Clinical Biochemistry and Research 11, no. 3 (2024): 162–70. http://dx.doi.org/10.18231/j.ijcbr.2024.025.

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SARS-CoV2 infection overcomes host cell membrane barrier, followed by utilization of host cellular processes for viral multiplication. Simultaneously, it triggers a cytokine storm within and around infected cells and tissues. Anti-inflammatory agents that can potentially inhibit viral penetration and multiplication within the host cells may be ideal drug candidates against COVID-19. Dietary phytosteroids have significant anti-inflammatory potential. Hence, the present study intends to investigate anti-viral potential of three dietary phytosteroids, namely, brassinolide, 28-homocastasterone and
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3

Panibrat, Olesya V., Vladimir N. Zhabinskii та Vladimir A. Khripach. "Effect of combination of cisplatin with brassinosteroids on the growth of cancer cells". Doklady of the National Academy of Sciences of Belarus 63, № 4 (2019): 437–44. http://dx.doi.org/10.29235/1561-8323-2019-63-4-437-444.

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In this work, the effect of brassinosteroids on the antitumor activity of classical cytostatic cisplatin in tumor cell lines A549 (human lung carcinoma) and Hep G2 (human hepatocellular carcinoma) was evaluated. Natural brassinosteroids 24-epibrassinolide and 28-homocastasterone, as well as their synthetic analogues (22S,23S)-24-epibrassinolide and (22S,23S)-28-homocastasterone were used. All four compounds with cisplatin inhibited the growth of cancer cells more effectively than cisplatin alone. Combinations with low concentrations of synthetic brassinosteroids were more effecient, and at 1 µM d
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4

Litvinovskaya, R. P., M. E. Raiman, and V. A. Khripach. "Synthesis of 22- and 23-oxoderivatives of 28-homocastasterone." Russian Journal of Organic Chemistry 44, no. 11 (2008): 1614–18. http://dx.doi.org/10.1134/s1070428008110080.

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5

Athithan, Velan, and Kotteazeth Srikumar. "PHYTOHORMONE INDUCED CHANGES ON BONE HISTOLOGY IN NORMAL AND DIABETIC MALE RAT." International Journal of Pharmacy and Pharmaceutical Sciences 9, no. 2 (2017): 287. http://dx.doi.org/10.22159/ijpps.2017v9i2.15770.

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<p><strong>Objective: </strong>To study the effect of brasslinolide keto isoform 28-homocastasterone in diabetic male rat bone marrow cells, bone histological changes, electrolytes and alkaline phosphatase activity in rat blood.</p><p><strong>Methods: </strong>Diabetes was induced in the group (n=6) of rats with a single peritoneal injection of streptozotocin (60 mg/kg bwt). With a treatment schedule of 15 consecutive days, control (n=6) and diabetic rats received 666µg/kg bwt of 28-homocastasterone. Circulating blood cell count, cell indices, bone mar
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Athithan, Velan, Ramasamy Ramesh, and Kotteazeth Srikumar. "28-HOMOCASTASTERONE: A NOVEL DIETARY PHYTO KETO OXYSTEROL MODULATING TESTICULAR STEROID METABOLISM AND LxR mRNA EXPRESSION IN DIABETIC RAT." International Journal of Pharmacy and Pharmaceutical Sciences 10, no. 2 (2018): 162. http://dx.doi.org/10.22159/ijpps.2018v10i2.23779.

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Objective: Present study aims to investigate 28-homocastasterone (28-HC) influences on testicular tissue in the normal and diabetic rat.Methods: Induction of diabetes was achieved by single peritoneal injection of streptozotocin (60 mg/kg b. wt) followed by 28-HC (100 µg/150 gm body weight) administration by oral gavage for 15 consecutive days to experimental rats. 3β-hydroxysteroid dehydrogenase, 17β-hydroxysteroid dehydrogenase activities, testosterone level, Liver X Receptor (LxR) mRNA expression, malondialdehyde (MDA), reduced glutathione (GSH) and testis histology was analysed.Results: In
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7

Pereira-Netto, A. B., M. M. C. Carvalho-Oliveira, J. A. Ramírez та L. R. Galagovsky. "Shooting Control in Eucalyptus Grandis × E. urophylla Hybrid: Comparative Effects of 28-Homocastasterone and a 5α-Monofluoro Derivative". Plant Cell, Tissue and Organ Culture 86, № 3 (2006): 329–35. http://dx.doi.org/10.1007/s11240-006-9124-0.

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8

Kotteazeth, Srikumar, and Athithan Velan. "28-Homocastasterone: A dietary plant keto oxysterol positively modulating hexokinase mRNA expression and catalytic activity in diabetic male rat." International Journal of Clinical and Experimental Physiology 5, no. 1 (2018): 48. http://dx.doi.org/10.4103/ijcep.ijcep_12_18.

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9

Athithan, Velan, and Kotteazeth Srikumar. "28-Homocastasterone down regulates blood glucose, cholesterol, triglycerides, SREBP1c and activates LxR expression in normal & diabetic male rat." Chemico-Biological Interactions 277 (November 2017): 8–20. http://dx.doi.org/10.1016/j.cbi.2017.08.010.

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10

Kolomeichuk, Liliya V., Ol’ga K. Murgan, Elena D. Danilova, et al. "Effects of Lactone- and Ketone-Brassinosteroids of the 28-Homobrassinolide Series on Barley Plants under Water Deficit." Plants 13, no. 10 (2024): 1345. http://dx.doi.org/10.3390/plants13101345.

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The aim of this work was to study the ability of 28-homobrassinolide (HBL) and 28-homocastasterone (HCS) to increase the resistance of barley (Hordeum vulgare L.) plants to drought and to alter their endogenous brassinosteroid status. Germinated barley seeds were treated with 0.1 nM HBL or HCS solutions for two hours. A water deficit was created by stopping the watering of 7-day-old plants for the next two weeks. Plants responded to drought through growth inhibition, impaired water status, increased lipid peroxidation, differential effects on antioxidant enzymes, intense proline accumulation,
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