Relevant bibliographies by topics / 3-iodothyronamine

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Academic literature on the topic '3-iodothyronamine'

Author: Grafiati
Published: 4 June 2021
Last updated: 14 March 2023

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Journal articles on the topic "3-iodothyronamine"

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Gachkar, Sogol, Rebecca Oelkrug, Beate Herrmann, et al. "N- and O-Acetylated 3-Iodothyronamines Have No Metabolic or Thermogenic Effects in Male Mice." European Thyroid Journal 9, no. 2 (2019): 57–66. http://dx.doi.org/10.1159/000504887.

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Introduction: Injection of 3-iodothyronamine into experimental animals profoundly affects their metabolism and body temperature. As 3-iodothyronamine is rapidly acetylated in vivo after injection, it was hypothesized that the metabolites N- or O-acetyl-3-iodothyronamines could constitute the active hormones. Methods: Adult male mice were injected once daily with one of the metabolites (5 mg/kg body weight intraperitoneally dissolved in 60% DMSO in PBS) or solvent. Metabolism was monitored by indirect calorimetry, body temperature by infrared thermography, and body composition by nuclear magnet
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Scanlan, Thomas S. "3-Iodothyronamine (T1AM): A New Player on the Thyroid Endocrine Team?" Endocrinology 150, no. 3 (2008): 1108–11. http://dx.doi.org/10.1210/en.2008-1596.

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3-Iodothyronamine (T1AM) is an endogenous compound with chemical features that are similar to thyroid hormone. T1AM has a carbon skeleton identical to that of T4 and contains a single carbon-iodine bond. Theoretically, T1AM could be produced from T4 by enzymatic decarboxylation and deiodination. Recent studies show that T1AM and higher iodinated thyronamines are subject to similar metabolic processing as iodothyronines such as T4, suggesting a biological linkage between iodothyronines and iodothyronamines. In addition, single doses of T1AM administered to rodents induce a hypometabolic state t
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Chiellini, G., S. Brogioni, S. Ghelardoni, et al. "3-Iodothyronamine modulates sarcoplasmic reticulum calcium release." Journal of Molecular and Cellular Cardiology 42, no. 6 (2007): S40. http://dx.doi.org/10.1016/j.yjmcc.2007.03.112.

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Brogioni, Simona, Grazia Chiellini, Matthew Hart, Thomas S. Scanlan, Elisabetta Cerbai, and Riccardo Zucchi. "3-Iodothyronamine affects calcium handling in isolated cardiomyocytes." Journal of Molecular and Cellular Cardiology 40, no. 6 (2006): 925. http://dx.doi.org/10.1016/j.yjmcc.2006.03.029.

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Ghelardoni, Sandra, Silvia Suffredini, Sabina Frascarelli, et al. "Modulation of cardiac ionic homeostasis by 3-iodothyronamine." Journal of Cellular and Molecular Medicine 13, no. 9b (2009): 3082–90. http://dx.doi.org/10.1111/j.1582-4934.2009.00728.x.

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Ianculescu, Alexandra G., Kathleen M. Giacomini, and Thomas S. Scanlan. "Identification and Characterization of 3-Iodothyronamine Intracellular Transport." Endocrinology 150, no. 4 (2009): 1991–99. http://dx.doi.org/10.1210/en.2008-1339.

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Saba, Alessandro, Grazia Chiellini, Sabina Frascarelli, et al. "Tissue Distribution and Cardiac Metabolism of 3-Iodothyronamine." Endocrinology 151, no. 10 (2010): 5063–73. http://dx.doi.org/10.1210/en.2010-0491.

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3-Iodothyronamine (T1AM) is a novel relative of thyroid hormone, able to interact with specific G protein-coupled receptors, known as trace amine-associated receptors. Significant functional effects are produced by exogenous T1AM, including a negative inotropic and chronotropic effect in cardiac preparations. This work was aimed at estimating endogenous T1AM concentration in different tissues and determining its cardiac metabolism. A novel HPLC tandem mass spectrometry assay was developed, allowing detection of T1AM, thyronamine, 3-iodothyroacetic acid, and thyroacetic acid. T1AM was detected
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Gencarelli, Manuela, Maura Lodovici, Lorenza Bellusci, Laura Raimondi, and Annunziatina Laurino. "Redox Properties of 3-Iodothyronamine (T1AM) and 3-Iodothyroacetic Acid (TA1)." International Journal of Molecular Sciences 23, no. 5 (2022): 2718. http://dx.doi.org/10.3390/ijms23052718.

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3-iodothyronamine (T1AM) and 3-iodothyroacetic acid (TA1) are thyroid-hormone-related compounds endowed with pharmacological activity through mechanisms that remain elusive. Some evidence suggests that they may have redox features. We assessed the chemical activity of T1AM and TA1 at pro-oxidant conditions. Further, in the cell model consisting of brown adipocytes (BAs) differentiated for 6 days in the absence (M cells) or in the presence of 20 nM T1AM (M + T1AM cells), characterized by pro-oxidant metabolism, or TA1 (M + TA1 cells), we investigated the expression/activity levels of pro- and a
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Gencarelli, Manuela, Annunziatina Laurino, Elisa Landucci, et al. "3-Iodothyronamine Affects Thermogenic Substrates’ Mobilization in Brown Adipocytes." Biology 9, no. 5 (2020): 95. http://dx.doi.org/10.3390/biology9050095.

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We investigated the effect of 3-iodothyronamine (T1AM) on thermogenic substrates in brown adipocytes (BAs). BAs isolated from the stromal fraction of rat brown adipose tissue were exposed to an adipogenic medium containing insulin in the absence (M) or in the presence of 20 nM T1AM (M+T1AM) for 6 days. At the end of the treatment, the expression of p-PKA/PKA, p-AKT/AKT, p-AMPK/AMPK, p-CREB/CREB, p-P38/P38, type 1 and 3 beta adrenergic receptors (β1–β3AR), GLUT4, type 2 deiodinase (DIO2), and uncoupling protein 1 (UCP-1) were evaluated. The effects of cell conditioning with T1AM on fatty acid m
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Ghelardoni, Sandra, Grazia Chiellini, Sabina Frascarelli, Alessandro Saba, and Riccardo Zucchi. "Uptake and metabolic effects of 3-iodothyronamine in hepatocytes." Journal of Endocrinology 221, no. 1 (2014): 101–10. http://dx.doi.org/10.1530/joe-13-0311.

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3-Iodothyronamine (T1AM) is an endogenous relative of thyroid hormone with profound metabolic effects. In different experimental models, T1AM increased blood glucose, and it is not clear whether this effect is entirely accounted by changes in insulin and/or glucagone secretion. Thus, in the present work, we investigated the uptake of T1AM by hepatocytes, which was compared with the uptake of thyroid hormones, and the effects of T1AM on hepatic glucose and ketone body production. Two different experimental models were used: HepG2 cells and perfused rat liver. Thyronines and thyronamines (T0AMs)
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Dissertations / Theses on the topic "3-iodothyronamine"

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Lavinia, Bandini. "3-iodothyronamine (T1AM) effects on glutamatergic postsynaptic signaling pathway." Doctoral thesis, Università di Siena, 2022. http://hdl.handle.net/11365/1195192.

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Thyroid hormones (TH), namely thyroxine (T4) and 3,5,3’-triiodothyronine (T3), are crucial regulators of multiple growth processes and control systems of energy metabolism. T4 and T3 undergo a complex metabolism in vivo, by several enzymes encompassing deiodinases, amine transferases, amine oxidases, decarboxylases and several classes of conjugating enzymes, particularly sulfotransferases and UDP-glucuronosyltransferases. T4 or T3 metabolites can produce significant functional effects when administered via interaction either with Thyroid Hormone Receptor (TR), or with other receptors. They ar
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White, Nicholas Edward. "The role of Relaxin-3 and 3-Iodothyronamine in the regulation of energy homeostasis." Thesis, Imperial College London, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.479156.

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Ianculescu, Alexandra G. "Investigations of 3-iodothyronamine as a novel regulator of thyroid endocrinology." Diss., Search in ProQuest Dissertations & Theses. UC Only, 2009. http://gateway.proquest.com/openurl?url_ver=Z39.88-2004&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&res_dat=xri:pqdiss&rft_dat=xri:pqdiss:3352462.

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Thesis (Ph.D.)--University of California, San Francisco, 2009.<br>Source: Dissertation Abstracts International, Volume: 70-04, Section: B, page: 2072. Advisers: Thomas S. Scanlan; Kathleen M. Giacomini.
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Lorenzini, Leonardo. "Effects of T3 and 3-iodothyronamine (T1AM) on cellular metabolism, and influence of serum proteins on T1AM assay." Doctoral thesis, Università di Siena, 2018. http://hdl.handle.net/11365/1046523.

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Thyroxine (T4) is the predominant form of thyroid hormone (TH). In target tissues, T4 is enzymatically deiodinated to 3,5,3′-triiodothyronine (T3), a high-affinity ligand for the nuclear TH receptors TRα and Trβ. T3 modulates genes transcription via activation of TRα and TRβ. Non-genomic effects have also been described. In 2004 the research groups of professors Scanlan Grandy and Zucchi discovered an endogenous thyroid hormone derivative called 3-iodothyronamine (T1AM). They proved that at nanomolar concentrations it can activate trace amine associated receptors 1 (TAARs)[1] and it may also
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Bräunig, Julia [Verfasser], Heike [Akademischer Betreuer] Biebermann, Roland [Gutachter] Lauster, Heike [Gutachter] Biebermann, and Juri [Gutachter] Rappsilbe. "3-Iodothyronamine and its role in aminergic G-protein coupled receptor signaling and neuromodulation / Julia Bräunig ; Gutachter: Roland Lauster, Heike Biebermann, Juri Rappsilbe ; Betreuer: Heike Biebermann." Berlin : Technische Universität Berlin, 2019. http://d-nb.info/1186130393/34.

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Sacripanti, Ginevra. "Thyroid hormone derivates T1AM and 3,5-T2: their effects on rat heart, NG108-15 and U-87 MG cell lines." Doctoral thesis, Università di Siena, 2020. http://hdl.handle.net/11365/1095534.

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The term “thyroid hormones”, classically referred to both 3,5,3′-triiodothyronine (T3) and thyroxine (T4), seems nowadays to be simplistic; some T3 and T4 metabolites, particularly 3,5diiodothyronine (3,5-T2) and 3-iodothyronamine (T1AM), are independent chemical messengers, with specific metabolic effects. In this PhD thesis we focused on the effects of these derivates on different tissues. In the first project we evaluated the effects of T1AM on the glutamatergic pathway, the main excitatory system in brain. A cancer hybrid cell line of mouse neuroblastoma and rat glioma (NG108-15) and a h
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Lehmphul, Ina. "Zelluläre Wirkung, Wirkmechanismen und Nachweisverfahren von Schilddrüsenhormonen und ihren Metaboliten." Doctoral thesis, Humboldt-Universität zu Berlin, Lebenswissenschaftliche Fakultät, 2015. http://dx.doi.org/10.18452/17434.

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Schilddrüsenhormone (TH) regulieren Metabolismus und Energiestoffwechsel. Der TH‐Metabolit (THM) 3,5‐T2 (3,5‐Diiod‐L‐Thyronin) aktiviert Fett‐Oxidation und mitochondriale Atmung. Der THM 3‐Iodothyronamin (3‐T1AM) beeinflusst zusätzlich glukoregulatorische Prozesse. THM können zur Reduktion von Körperfett beitragen. Um 3,5‐T2 im humanen Serum nachzuweisen sollte ein Immunoassay aufgebaut, validiert und angewendet werden. In intakten hepatozellulären (HepG2) sowie pankreatischen ß‐Zellen (MIN6) sollte untersucht werden ob THM durch Modulation der mitochondrialen Aktivität die zelluläre Substra
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MANNI, MARIA ELENA. "Pharmacological characterisation of behavioural and metabolic effects of 3-iodothyronamine (T₁AM) and 3-iodothyroacetic acid (TA1), endogenous end products of thyroid hormone metabolism." Doctoral thesis, 2015. http://hdl.handle.net/2158/1002323.

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Book chapters on the topic "3-iodothyronamine"

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Zucchi, Riccardo, Sandra Ghelardoni, and Grazia Chiellini. "Cardiac Functional Effects of 3-iodothyronamine, a New Endogenous Thyroid Hormone Derivative." In Thyroid and Heart Failure. Springer Milan, 2009. http://dx.doi.org/10.1007/978-88-470-1143-4_6.

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Laurino, A., and L. Raimondi. "3-Iodothyronamine, a New Chapter in Thyroid Story." In Trace Amines and Neurological Disorders. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-12-803603-7.00021-5.

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Chiellini, Grazia, Alessandro Saba, Maia Marchini, et al. "3-Iodothyronamine as a Novel Endogenous Chemical Messenger: Tissue Distribution and Catabolism." In The Endocrine Society's 92nd Annual Meeting, June 19–22, 2010 - San Diego. Endocrine Society, 2010. http://dx.doi.org/10.1210/endo-meetings.2010.part2.p12.p2-579.

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Kleinau, G., N. Khajavi, J. Köhrle, and H. Biebermann. "Differential Modulation of Adrenergic Receptor Signaling by Octopamine, Tyramine, Phenylethylamine, and 3-Iodothyronamine." In Trace Amines and Neurological Disorders. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-12-803603-7.00005-7.

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Pellegrini, Silvia, Erika Melissari, Veronica Mariotti, et al. "Administration of 3-Iodothyronamine (T1AM) Affects Gene Expression in the Rat Adipose Tissue Cells." In The Endocrine Society's 92nd Annual Meeting, June 19–22, 2010 - San Diego. Endocrine Society, 2010. http://dx.doi.org/10.1210/endo-meetings.2010.part2.p4.p2-157.

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Chiellini, Grazia, Julia Haviland, Hannah Reliand, et al. "New Insights into the Effects of 3-Iodothyronamine (T1AM) on Metabolism in Mice from Cavity Ring Down Spectroscopy (CRDS)." In BASIC - Hypothalamic-Pituitary-Thyroid Axis: Thyroid Hormone Metabolism, Cellular Uptake & Action. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part2.p16.p1-663.

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