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Journal articles on the topic '-303'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 August 2024

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1

Gubaidullin, A. A., F. A. Gubaidullin, V. A. Iktisanov, and N. H. Musabirova. "Chages in parameters of deposits No. 301, 302, 303 of Romashkinskoye oil field in modeling reservoir conditions." Neftyanoe khozyaystvo - Oil Industry, no. 5 (2017): 18–20. http://dx.doi.org/10.24887/0028-2448-2017-5-18-20.

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2

Hesslink, R. L., and R. P. Adams. "303." Medicine & Science in Sports & Exercise 19, Supplement (April 1987): S51. http://dx.doi.org/10.1249/00005768-198704001-00303.

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3

Williamson, David, Sebastien Dupuis, Jacques-Alexandre Amiel, Mathieu Desgroseilliers, Marc Perreault, Zoe Thiboutot, Karim Serri, Pierre Marsolais, and Anne Julie Frenette. "303." Critical Care Medicine 40 (December 2012): 1–328. http://dx.doi.org/10.1097/01.ccm.0000424521.54317.db.

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4

Geiger, Krystina, Lindsay Arnold, and Kevin Horbowicz. "303." Critical Care Medicine 41 (December 2013): A71. http://dx.doi.org/10.1097/01.ccm.0000439448.80858.3d.

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5

Woods, Jeffrey A. "303." Medicine & Science in Sports & Exercise 41 (May 2009): 42–43. http://dx.doi.org/10.1249/01.mss.0000353001.31458.a2.

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6

Jamkhana, Zafar Akram, Brian Reichardt, John Mwangi, Nirav patel, and Aditya Uppalapati. "303." Critical Care Medicine 42 (December 2014): A1433. http://dx.doi.org/10.1097/01.ccm.0000457800.40815.fb.

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7

Hirsch, Nina, Jonathan Sevransky, William Checkley, and Greg Martin. "303." Critical Care Medicine 43 (December 2015): 77. http://dx.doi.org/10.1097/01.ccm.0000474131.75838.08.

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8

Fong, Isabel, Hershel Schaftel, Kanchan Koirala, and Ian Butler. "303." Critical Care Medicine 47 (January 2019): 133. http://dx.doi.org/10.1097/01.ccm.0000551057.35532.ed.

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Fredrickson, Sonja K., Karen A. Tisdel, Franklin J. Zieve, and Brandi Cummings. "303." Journal of Clinical Lipidology 1, no. 2 (May 2007): 163. http://dx.doi.org/10.1016/j.jacl.2007.03.035.

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10

Frazier, O. H., T. J. Myers, N. Palanichamy, C. Gemmato, I. D. Gregoric, R. M. Delgado, and B. Radovancevic. "303." Journal of Heart and Lung Transplantation 25, no. 2 (February 2006): S149. http://dx.doi.org/10.1016/j.healun.2005.11.315.

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11

Ang, C. "303." Journal of Minimally Invasive Gynecology 12, no. 5 (October 2005): 117. http://dx.doi.org/10.1016/j.jmig.2005.07.340.

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Davis, G., D. Milzman, M. Hui, T. Weiler, and J. Hill. "303." Annals of Emergency Medicine 48, no. 4 (October 2006): 92. http://dx.doi.org/10.1016/j.annemergmed.2006.07.764.

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13

Imani, Erinda Salma, and Ida Bagus Ketut Bayangkara. "Analisis Pelaporan Kinerja Lingkungan Pada Rumah Sakit Lavalette Tahun 2022." Journal of Trends Economics and Accounting Research 4, no. 2 (December 31, 2023): 498–508. http://dx.doi.org/10.47065/jtear.v4i2.1116.

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Lavalette Hospital Malang, located at Jl. WR. Supratman No.10 Malang City, can generate medical and hazardous waste, which can impact the environment. Many healthcare facilities still do not report their environmental performance with GRI standards as it is voluntary. However, to fulfill their environmental and social responsibilities, both external and internal, this environmental performance reporting is helpful. This research uses a qualitative method with a case study at Lavalette Hospital Malang to determine the management of responsibility and disclosure of environmental performance in accordance with GRI Standard 300 guidelines. The data used comes from documentation and interviews with environmental health staff and the Manager of Accounting, Finance, and IT at Lavalette Hospital. Then triangulation analysis was carried out. The results of the analysis show that Lavalette Hospital's environmental performance can be categorized into partial disclosure on GRI index 302: Energy, GRI 303: Water and Effluent, GRI 305: Emissions, GRI 306: Waste, GRI 307: Compliance, and GRI 308: Supplier environmental assessment.

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14

Stassinopoulos, Adonis, Mary Ann Schott, Grace M. Castro, and Lisa M. Turin. "Elimination of Immunoreactivity of Red Cells Treated with a Modified S-303 Pathogen Inactivation Process." Blood 104, no. 11 (November 16, 2004): 2703. http://dx.doi.org/10.1182/blood.v104.11.2703.2703.

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Abstract Background S-303 was developed to inactivate viruses, bacteria, protozoa, and leukocytes in red blood cell concentrates (RBC). S-303 is a modular FRALE compound (FRangible Anchor Linker Effector) designed to bind to nucleic acids with its Anchor, to react through its Effector, and to form cross-links. S-303 spontaneously decomposes to the non-reactive compound S-300 by hydrolysis of the Linker, to minimize protein adducts. Pathogen inactivation (PI) treatment utilized the co-addition of S-303 and unbuffered GSH to quench non-specific S-303 reactions. The treatment process was optimized to maintain RBC function and maximize PI. Pre-clinical dog and rabbit chronic transfusion studies with allogeneic S-303 RBC showed no detectable antibodies to S-303 RBC. In Phase 1 studies, transfusion of healthy subjects with autologous human S-303 RBC demonstrated acceptable post-transfusion recovery and life span. Repeated transfusion (n=5) of 28 healthy subjects with autologous S-303 RBC showed no detectable antibodies to S-303 RBC. In a Phase 3 trial evaluating chronic transfusion of allogeneic S-303 RBC to patients with thalassemia or sickle cell anemia, 2 of 26 patients developed low titer positive Indirect Antiglobulin Tests (IAT) to S-303 RBC (one of the 2 patients also had a direct reacting IgM agglutinin). For both patients Direct Antiglobulin Tests (DAT) were negative. However, pretreatment RBC from the same unit remained compatible. S-303-related Anchor derivatives inhibited the positive IAT. Following this observation, clinical trials of S-303 RBC were stopped, studies were initiated to define the immunologic response to S-303 RBC, and an improved S-303 treatment process was developed. Methods The original PI process utilized 200 μM S-303 and 2 mM unbuffered GSH. The process was modified to use 10-fold more neutralized GSH (20 mM) added to RBC 10 minutes prior to addition of S-303 (200μM). High titer anti-Anchor sera (RaS) were elicited by immunizing rabbits with a stable Anchor-KLH construct. A FACS assay to detect decoration of RBC with S-303 was developed using RaS and FITC goat anti-rabbit (GAR) IgG. IAT assays were performed with two methods: High titer RaS were tested with buffer gel cards (MTS), S-303 RBC suspended in low ionic strength solution (LISS) and GAR IgG. Reactive patient sera were tested with S-303 RBC and anti-IgG gel cards (MTS) Results S-303 RBC prepared with the original clinical process were positive for IAT by gel card for both the RaS (1:100) and for the patient sera (1:3). FACS analysis using RaS (1:100) with FITC GAR IgG (1:64) demonstrated a high level of labeling. Under the modified conditions (S-303m), S-303m RBC exhibited minimal labeling above background by FACS with RaS. Sera from the 2 patients with positive IAT against S-303 RBC were negative against S-303m RBC. In addition, high titer RaS were negative against S-303m RBC in IAT by gel card. Potent inactivation of bacteria in S303m RBC (S. epidermis, S. marcescens) and viruses (Vesicular stomatitis virus) was retained. Storage of S-303m RBC for 42 days exhibited hemolysis and K+ levels comparable to S303 RBC and higher ATP levels than S-303 RBC. Conclusions An improved PI process has been developed that significantly reduces RBC decoration by S-303 while maintaining PI and RBC in vitro function. The new process eliminated the positive IAT reactivity with sera from patients previously alloimmunized to S-303 RBC.

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15

Khamidulina, Khalidya Kh, Elena V. Tarasova, and Mikhail L. Lastovetsky. "Prediction of the biodegradation of chemicals using OECD QSAR Toolbox software." Toxicological Review 32, no. 1 (February 29, 2024): 20–30. http://dx.doi.org/10.47470/0869-7922-2024-32-1-20-30.

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Introduction. The development of integrated approaches to testing, assessment of the hazard and exposure risk of chemicals on human health and the environment is one of the priorities of preventive toxicology. An integrated approach involves various combinations of methods in silico, in chemico, in vitro, ex vivo, in vivo for hazard assessment. In the Russian Federation, in silico methods are often perceived with skepticism, mainly due to the lack of their legal status, weak methodological base and insufficient training of specialists. In order to expand the methodological base, the possibility of predicting the stability of chemicals in biotic conditions using the OECD QSAR Toolbox software was studied. Material and methods. The OECD QSAR Toolbox software version 4.4.1., OECD guidelines on the assessment of biodegradation of chemicals. Results. The world community and the OECD have developed and implemented a three-level system for chemicals biodegradation testing, which includes tests for readily biodegradation (OECD Guidelines 301 A, 301 B, 301 C, 301 D, 301 E, 301 F, 306, 310), tests to determine the potential biodegradability (OECD Guidelines 302 A, 302 B, 302 C, 304 A) and model systems tests (OECD Guidelines 303 A, 303 B). To test the capabilities of the program, 24 endpoints were selected. They are the determination of biodegradability (%) by BOD, DOC, CO2, CH4 releases in OECD tests 301 A, 301 B, 301 C, 301 D, 302 C, 302 B, biodegradability (%) in sediments and soil, bioconcentration factors for more than 100 organic chemicals of various structures. The parameters were calculated using the analog method, followed by mandatory interpretation of the data obtained by an expert. When predicting the biodegradability of chemicals, it is necessary to perform a series of calculations using different tests (OECD tests 301 A, 301 B, 301 C, 301 D, 301 E, 302 B are preferred) and grouping methods followed by a comprehensive assessment of the results obtained, taking into account not only the structural similarity of substances and analogues, but also the quality of the experimental data used. When predicting biodegradability by BOD values, it is advisable to use the OECD 301 C test. The proportion of tested substances whose biodegradability could be estimated by BOD values is about 50% in the OECD 301 C test, which is primarily due to the presence of a significant amount of experimental data. The calculation of bioconcentration factors seems to be promising. For about 45% of the tested substances, it was possible to calculate bioconcentration coefficients with good correlation with experimental data. Biodegradation in soil (% and half-life) can be predicted only for a very limited range of compounds (10% of the tested substances), which is due to the difficulty of selecting structurally similar analogues with experimental data. The method is not applicable for predicting the biodegradation of salts, organometallic compounds, polymer molecules and mixed products. Conclusion. The Russian Register of Potentially Hazardous Chemical and Biological Substances has developed a methodological guide for predicting the stability of chemicals in biotic conditions using the OECD QSAR Toolbox software. The document presents algorithms for calculating biodegradability (%) according to OECD tests 301, 302, 303; BOD, bioconcentration factors and biodegradability (%) in soil.

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16

Rakocevic, Natalie, Ohoud H. Alaslani, and Carlos H. Torres. "Case 303." Radiology 302, no. 3 (March 2022): 722–23. http://dx.doi.org/10.1148/radiol.203755.

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17

Iza, G. M., A. Iza, M. A. Grunauer, R. Yerovi, and S. Campos Miño. "ABSTRACT 303." Pediatric Critical Care Medicine 15 (May 2014): 71. http://dx.doi.org/10.1097/01.pcc.0000449029.66933.9e.

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18

Gurung, Anobha, and Michelle L. Bell. "P-303." Epidemiology 23 (September 2012): 1. http://dx.doi.org/10.1097/01.ede.0000417305.34382.9a.

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19

Peay, H. L., H. Scharff, B. B. Biesecker, B. S. Wilfond, J. Johnson, D. M. Escolar, K. Nagaraju, J. Piacentino, J. Bowie, and A. Tibben. "G.P.303." Neuromuscular Disorders 24, no. 9-10 (October 2014): 912–13. http://dx.doi.org/10.1016/j.nmd.2014.06.393.

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20

Borges, Vera T., Jose C. Peraçoli, Mariana Romão, Silmeia Zanati, Juliane R. Poiati, Ingrid C. Weel, and Maria Terezinha Peraçoli. "[303-POS]." Pregnancy Hypertension: An International Journal of Women's Cardiovascular Health 5, no. 1 (January 2015): 150. http://dx.doi.org/10.1016/j.preghy.2014.10.309.

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21

Rubina Ali, C., L. G. Nardo, G. Horne, and C. Fitzgerald. "P-303." Fertility and Sterility 86, no. 3 (September 2006): S247. http://dx.doi.org/10.1016/j.fertnstert.2006.07.657.

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22

Chaudhary, Arun Singh, S. P. Uniyal, and Pooja Pandey. "Evaluation of new genotypes of brinjal (Solacanum melongena L.) under tarai condition of Uttarakhand." Journal of Applied and Natural Science 9, no. 3 (September 1, 2017): 1840–43. http://dx.doi.org/10.31018/jans.v9i3.1449.

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In order to assess the performance of some new genotypes of brinjal (Solanum melongena L.) under tarai condition of Uttarakhand, an investigation was carried out at Vegetable Research Centre, G. B. Pant University of Agriculture and Technology, Pantnagar, during autumn-winter cropping season of 2012-13. The experiment was laid out in Randomized Complete Block Design with 4 replications and 9 treatments viz. PB-300, PB-301, PB-302, PB-303, PB-304, PB-305, Kashi Tarun, Punjab Sadabahar and Pant Samrat. To have comparative study, growth characters, per cent fruit infested by fruit borer, fruit yield and finally economics were also worked out. The findings revealed that none of the new genotypes in this study could supersede the local checks in respect to yield related attributes, per cent infested fruit by borer and economics. Amongst 6 genotypes and 3 commercial cultivars, variety Kashi Tarun proved the best with respect to fruit yield (490.73 q/ha) and B:C (2.43). It is also less infested by fruit borer. The per cent infested fruit by borer was 7.16 %. Variety pant samrat and pant bahar were also considered promising with 385.70 and 369.33 q/ha marketable fruit yield.

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23

Not Available, Not Available. "Erratum: Eur J Clin Pharmacol (1998) 54: 303-308." European Journal of Clinical Pharmacology 54, no. 7 (October 14, 1998): 577. http://dx.doi.org/10.1007/s002280050517.

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24

Лаврова, В. "СТРОКИ ОСКАРЖЕННЯ ПОВІДОМЛЕНЯ ПРО ПІДОЗРУ." Юридичний вісник, no. 2 (July 6, 2021): 166–76. http://dx.doi.org/10.32837/yuv.v0i2.2170.

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У статті досліджено колізію між строками подання скарги на рішення, дії чи бездіяльність слідчого, дізнавача чи прокурора, передбачені ч. 1 ст. 303 КПК, встановленими в ч. 1 ст. 304 КПК, та строками оскарження повідомлення про підозру, визначеними в п. 10 ч. 1 ст. 303 КПК. Проаналізовано норми п. 10 ч. 1 ст. 303 КПК та ч. 1 ст. 304 КПК із точки зору їх співвідношення як загальної та спеціальної норми. На основі проведеного аналізу визначено, що спеціальною нормою є п. 10 ч. 1 ст. 303 КПК, а загальною нормою є ч. 1 ст. 304 КПК, оскільки: 1) поява строків оскарження повідомлення про підозру є результатом диференціації (спеціалізації) правового регулювання, для досягнення найбільшої ефективності правового регулювання, а саме врахування особливостей повідомлення про підозру, як юридичного факту; 2) за критерієм об'єму правового регулювання п. 10 ч. 1 ст. 303 КПК є значно вужчою, оскільки стосується лише повідомлення про підозру, тоді як ч. 1 ст. 304 КПК поширюється на різні рішення, дії і бездіяльність; 3. п. 10 ч. 1 ст. 303 КПК є похідним від ч.1 ст. 304 КПК, яка є первинною щодо нього; 4. співвідношення між ч. 1 ст. 304 та п. 1 ч. 1 ст. 303 КПК характеризується співвідношенням понять «рід - вид»; 5) п. 10 ч. 1 ст. 303 КПК встановлено для зміни способу і меж регулювання оскарження повідомлення про підозру, а саме первинно впроваджено вказане оскарження (до цієї норми такого права не існувало) і встановлено його особливості. За результатами проведеного дослідження встановлено, що при вирішенні колізії між ч. 1 ст. 304 КПК та ч. 1 ст. 203 КПК слід керуватися загальними засадами кримінального провадження, а саме принципом верховенства права та принципом верховенства спеціальних норм, тобто колізійним правилом «спеціальний закон скасовує дію загального». Отже, п. 10 ч. 1 ст. 303 КПК має пріоритет в застосуванні перед ч. 1 ст. 304 КПК. Запропоновано викласти ч. 1 ст. 304 КПК у такй редакції: «1. Скарги на рішення, дії чи бездіяльність слідчого, дізнавача чи прокурора, передбачені частиною першою статті 303 цього Кодексу, за винятком повідомлення слідчого, дізнавача, прокурора про підозру, можуть бути подані особою протягом десяти днів з моменту прийняття рішення, вчинення дії або бездіяльності. Якщо рішення слідчого, дізнавача чи прокурора оформлюється постановою, строк подання скарги починається з дня отримання особою її копії».

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Torró Ferrero, G., D. F. J. Fernández Rego, and J. J. Agüera Arenas. "Abstract P-303." Pediatric Critical Care Medicine 19 (June 2018): 139. http://dx.doi.org/10.1097/01.pcc.0000537760.75047.9c.

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26

El-Khoury, Georges Y., Austin J. Corbett, and Thomas B. Summers. "Case report 303." Skeletal Radiology 13, no. 2 (February 1985): 164–68. http://dx.doi.org/10.1007/bf00352088.

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27

Rudolph, Robert I. "May iotaderma (#303)." Journal of the American Academy of Dermatology 80, no. 5 (May 2019): e135. http://dx.doi.org/10.1016/j.jaad.2018.11.011.

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28

Rudolph, Robert I. "May iotaderma (#303)." Journal of the American Academy of Dermatology 80, no. 6 (June 2019): e193. http://dx.doi.org/10.1016/j.jaad.2018.11.012.

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29

Teixeira, Ana Maria Fortaleza, and Margarita A. Villar Luis. "Distúrbios psiquiátricos, tentativas de suicídio, lesões e envenenamento em adolescentes atendidos em uma unidade de emergência, Ribeirão Preto, São Paulo, 1988-1993." Cadernos de Saúde Pública 13, no. 3 (September 1997): 517–25. http://dx.doi.org/10.1590/s0102-311x1997000300027.

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Trata-se de um estudo epidemiológico acerca da ocorrência de distúrbios psiquiátricos e outros relacionados, segundo o Código Internacional de Doenças, em adolescentes atendidos no setor de urgências psiquiátricas de um hospital-escola de Ribeirão Preto, São Paulo, Brasil, durante o período de 1988 a 1993. Os adolescentes representaram 23% dos atendimentos no período. No sexo feminino, predominaram os Transtornos Neuróticos (300), Suicídio e Lesões Auto-Infligidas (E950-E959) e Psicoses Esquizofrênicas (295). No sexo masculino, sobressaíram-se os Quadros Relacionados a Álcool e Drogas (291, 292, 303, 304, 305), Psicoses Esquizofrênicas (295) e Transtornos Neuróticos (300). Dentre os adolescentes, predominaram as faixas 20 a 24 anos e 15 a 19 anos, consecutivamente.

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30

Saltamavros, Alexandros D., George Adonakis, Sosanna Kritikou, Vasiliki Koika, Kleanthis Koufogiannis, Kostas Spyropoulos, George Kourounis, Christodoulos Flordellis, Venetsana Kyriazopoulou, and Neoklis A. Georgopoulos. "α2β adrenoreceptor 301–303 deletion polymorphism in polycystic ovary syndrome." Clinical Autonomic Research 17, no. 2 (March 16, 2007): 112–14. http://dx.doi.org/10.1007/s10286-007-0403-6.

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31

Yuan, Jun Ping, De Dong Chen, and Hai Rui Bo. "Nickel Release Rate of Type 303 Free Cutting Austenitic Stainless Steel." Advanced Materials Research 1096 (April 2015): 114–19. http://dx.doi.org/10.4028/www.scientific.net/amr.1096.114.

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Type 303 austenitic stainless steel has been applied in jewelry production, and its nickel release rate has become a concerned issue in the jewelry industry. In this paper, the commercial type 303 stainless steel was used as the test material, while 304 stainless steel as the contrast material; their nickel release rates and corrosion behaviors in artificial sweat were studied. The results show that the actually measured nickel release rate of 303 stainless steel reaches 2.06μg/cm2/week, nearly 25 times higher than that of 304 stainless steel, which exceeds the threshold specified in nickel release standard EN1811:2011 for jewelries coming into direct and prolonged contact with the skin (0.88μg/cm2/week), and its adjusted value also exceeds the threshold specified in Nickel Directive 2004/96/EC for puncture accessories (0.35μg/cm2/week). The high nickel release rate for 303 stainless steel is mainly caused by its high sulfur content and the inevitable formation of manganese sulfide inclusions, which will cause the pitting and exacerbate the material corrosion. Considering the risk of nickel sensitization of 303 stainless steel, it is not suggested to use 303 stainless steel as the jewelry material, especially for piercing jewelry.

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32

Honeyborne, Isobella, Francisco M. Codoñer, Alasdair Leslie, Gareth Tudor-Williams, Graz Luzzi, Thumbi Ndung'u, Bruce D. Walker, Philip J. Goulder, and Julia G. Prado. "HLA-Cw*03-Restricted CD8+ T-Cell Responses Targeting the HIV-1 Gag Major Homology Region Drive Virus Immune Escape and Fitness Constraints Compensated for by Intracodon Variation." Journal of Virology 84, no. 21 (August 25, 2010): 11279–88. http://dx.doi.org/10.1128/jvi.01144-10.

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ABSTRACT The potential importance of HLA-C-restricted CD8+ cytotoxic T lymphocytes (CTL) in HIV infection remains undetermined. We studied the dominant HLA-Cw*03-restricted CTL response to YVDRFFKTL296-304 (YL9), within the conserved major homology region (MHR) of the Gag protein, in 80 HLA-Cw*03-positive individuals with chronic HIV infection to better define the efficacy of the YL9 HLA-C-restricted response. The HLA-Cw*03 allele is strongly associated with HIV sequence changes from Thr-303 to Val, Ile, or Ala at position 8 within the YL9 epitope (P = 1.62 × 10−10). In vitro studies revealed that introduction of the changes T303I and T303A into the YL9 epitope both significantly reduced CTL recognition and substantially reduced the viral replicative capacity. However, subsequent selection of the Val-303 variant, via intracodon variation from Ile-303 (I303V) or Ala-303 (A303V), restored both viral fitness and CTL recognition, as supported by our in vivo data. These results illustrate that HLA-C-restricted CTL responses are capable of driving viral immune escape within Gag, but in contrast to what was previously described for HLA-B-restricted Gag escape mutants, the common Cw*03-Gag-303V variant selected resulted in no detectable benefit to the host.

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Meijer, G. "MO-DE-303-01." Medical Physics 42, no. 6Part28 (June 2015): 3558. http://dx.doi.org/10.1118/1.4925343.

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van der Heide, U. "MO-DE-303-02." Medical Physics 42, no. 6Part28 (June 2015): 3558. http://dx.doi.org/10.1118/1.4925344.

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Bowen, S. "MO-DE-303-03." Medical Physics 42, no. 6Part28 (June 2015): 3558. http://dx.doi.org/10.1118/1.4925345.

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36

Maiser, Samuel, Asish Kabir, David Sabsevitz, and Wendy Peltier. "Locked-In Syndrome #303." Journal of Palliative Medicine 19, no. 4 (April 2016): 460–61. http://dx.doi.org/10.1089/jpm.2016.0016.

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37

Langroudi, Reza Rezazadeh. "268-303/881-915." Studia Islamica 109, no. 2 (November 17, 2014): 191–207. http://dx.doi.org/10.1163/19585705-12341302.

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van Donk, R. N. "303 Wat Mij Opvalt ..." Zorg en Financiering 8, no. 3 (March 2009): 1. http://dx.doi.org/10.1007/bf03097972.

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Kline, M. P., and R. I. Morimoto. "Repression of the heat shock factor 1 transcriptional activation domain is modulated by constitutive phosphorylation." Molecular and Cellular Biology 17, no. 4 (April 1997): 2107–15. http://dx.doi.org/10.1128/mcb.17.4.2107.

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Heat shock transcription factor 1 (HSF1) is constitutively expressed in mammalian cells and negatively regulated for DNA binding and transcriptional activity. Upon exposure to heat shock and other forms of chemical and physiological stress, these activities of HSF1 are rapidly induced. In this report, we demonstrate that constitutive phosphorylation of HSF1 at serine residues distal to the transcriptional activation domain functions to repress transactivation. Tryptic phosphopeptide analysis of a collection of chimeric GAL4-HSF1 deletion and point mutants identified a region of constitutive phosphorylation encompassing serine residues 303 and 307. The significance of phosphorylation at serines 303 and 307 in the regulation of HSF1 transcriptional activity was demonstrated by transient transfection and assay of a chloramphenicol acetyltransferase reporter construct. Whereas the transfected wild-type GAL4-HSF1 chimera is repressed for transcriptional activity and derepressed by heat shock, mutation of serines 303 and 307 to alanine results in derepression to a high level of constitutive activity. Similar results were obtained with mutation of these serine residues in the context of full-length HSF1. These data reveal that constitutive phosphorylation of serines 303 and 307 has an important role in the negative regulation of HSF1 transcriptional activity at control temperatures.

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KLIMACEK, Mario, and Bernd NIDETZKY. "A catalytic consensus motif for d-mannitol 2-dehydrogenase, a member of a polyol-specific long-chain dehydrogenase family, revealed by kinetic characterization of site-directed mutants of the enzyme from Pseudomonas fluorescens." Biochemical Journal 367, no. 1 (October 1, 2002): 13–18. http://dx.doi.org/10.1042/bj20020932.

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Lys-295, Asn-300 and His-303 of d-mannitol 2-dehydrogenase from Pseudomonas fluorescens were mutated individually into alanine (K295A, N300A and H303A respectively). Purified mutants displayed catalytic efficiencies for NAD+-dependent oxidation of d-mannitol 300-fold (H303A), 1000-fold (N300A) and approx. 400000-fold (K295A) below the wild-type level. Comparison of primary kinetic isotope effects on kinetic parameters for d-fructose reduction by wild-type and mutants at pH10.0 demonstrate that Asn-300 has an auxiliary role in stabilization of the transition state of hydride transfer, and His-303 contributes to substrate positioning. The large solvent isotope effect of 11±1 on kcat for mannitol oxidation by K295A at pH(2H) 10.5 suggests a role for Lys-295 in general base enzymic catalysis. Positional conservation of Lys-295, Asn-300 and His-303 across a family of polyol-specific long-chain dehydrogenases suggests a unique catalytic signature: Lys-Xaa4-Asn-Xaa2-His (where ‘Xaa’ denotes ‘any amino acid').

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Jousseaume, Valérie. "303, numéro spécial, La Loire." Norois, no. 192 (September 1, 2004): 146. http://dx.doi.org/10.4000/norois.978.

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Rakocevic, Natalie, Ohoud H. Alaslani, and Carlos H. Torres. "Case 303: Delayed Posthypoxic Leukoencephalopathy." Radiology 304, no. 1 (July 2022): 241–44. http://dx.doi.org/10.1148/radiol.203756.

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Pantel, K. "303 INVITED Circulating Tumour Cells." European Journal of Cancer 47 (September 2011): S69. http://dx.doi.org/10.1016/s0959-8049(11)70518-4.

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Van Nieuwenhuizen, O. "303 Cerebral palsy in Europe." European Journal of Paediatric Neurology 3, no. 6 (January 1999): A13. http://dx.doi.org/10.1016/s1090-3798(99)91023-1.

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Kusdiana, Alchemi Putri Juliantika, Afdholiatus Syafaah, and Fetrina Oktavia. "RESISTENSI TANAMAN KARET KLON IRR SERI 300 TERHADAP PENYAKIT GUGUR DAUN CORYNESPORA." Jurnal Penelitian Karet, January 9, 2018, 115–28. http://dx.doi.org/10.22302/ppk.jpk.v35i2.374.

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Penyakit gugur daun Corynespora yang disebabkan oleh cendawan Corynespora cassiicola (C. cassiicola) merupakan salah satu penyakit daun penting yang dapat menyebabkan penurunan produksi lateks pada perkebunan karet. Salah satu tahapan penting untuk melepaskan klon karet baru adalah mengidentifikasi karakter sekunder seperti resistensi terhadap penyakit. Pengujian resistensi klon karet IRR seri 300 dilakukan di laboratorium dan rumah kaca dengan menggunakan Rancangan Acak Lengkap dua faktor yaitu faktor jenis (genotipe) klon (26 jenis klon) dan isolat C. cassiicola (3 isolat). Selain itu, pengamatan serangan penyakit secara langsung juga dilakukan pada tanaman belum menghasilkan di lapangan. Hasil pengujian menunjukkan semua isolat C. cassiicola berpengaruh nyata terhadap resistensi 26 klon IRR seri 300 baik di laboratorium maupun di rumah kaca. Hasil pengamatan pada tiga kegiatan menunjukkan bahwa 13 klon karet yaitu IRR 301, IRR 302, IRR 303, IRR 304, IRR 305, IRR 307, IRR 308, IRR 309, IRR 312, IRR 315, IRR 316, IRR 318, dan IRR 323 memiliki tingkat resistensi tinggi terhadap penyakit gugur daun Corynespora.

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"Gout (Abstracts 302-303)." Rheumatology 40, no. 90001 (April 1, 2001): 108. http://dx.doi.org/10.1093/rheumatology/40.suppl_1.108.

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"Pre-meeting Sunday Short Courses: July 30." Microscopy and Microanalysis 12, S1 (June 27, 2006): 26–28. http://dx.doi.org/10.1017/s1431927606160298.

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X-11: Scanning Cathodoluminescence Microscopy and Spectroscopy, Full Day: 9:00 AM–5:00 PM, Room 301/303.X-12: Digital Imaging 101: Scientific Imaging with Photoshop, Full day: 9:00 AM–5:00 PM, Room 302/304.X-13: Digital Imaging 102: Image Processing and Analysis, Full Day: 9:00 AM–5:00 PM, Room 306.X-14: Live Cell Imaging Using Fluorescence Methods, Full Day: 9:00 AM–5:00 PM, Room 305.X-15: What To Do with a Variable Pressure (VPSEM) or Environmental SEM (ESEM) And How To Do It (Or At Least How It Ought To Work), Full Day: 9:00 AM–5:00 PM, Room 307.X-16: 3-Dimensional Electron Microscopy (3DEM) in Life and Material Science—In-Depth Tutorial about Tomography—Basics and Methods, Full day: 9:00 AM–5:00 PM, Room 308.X-17 Failure Analysis and Evidence Preservation by Metallography, Half day: 9:00 AM–1:00 PM, Room 309/311.Special events: MSA and MAS presidential happenings, IMS Henry Clifton Sorby award and lecture, and art exhibit.

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Kusdiana, Alchemi Putri Juliantika, Afdholiatus Syafaah, and Sigit Ismawanto. "RESISTENSI TANAMAN KARET KLON IRR SERI 300 TERHADAP PENYAKIT GUGUR DAUN COLLETOTRICHUM DI SUMATERA SELATAN." Jurnal Penelitian Karet, November 29, 2018, 147–56. http://dx.doi.org/10.22302/ppk.jpk.v36i2.555.

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Penyakit gugur daun Colletotrichum merupakan salah satu penyakit penting yang dapat menyebabkan penurunan produksi lateks pada perkebunan karet. Pengendalian penyakit gugur daun Colletotrichum yang paling efektif adalah dengan penggunaan klon resisten. Pengujian resistensi klon karet IRR seri 300 dilakukan di laboratorium dan rumah kaca dengan menggunakan rancangan acak lengkap dua faktor yaitu jenis klon (26 jenis klon) dan isolat C. gloeosporioides (tiga isolat: CG-PR 303, CG-RRIM 600, dan CG-GT 1). Selain itu, pengamatan serangan penyakit secara langsung juga dilakukan pada tanaman belum menghasilkan di lapangan. Hasil pengujian menunjukkan bahwa semua isolat C. gloeosporioides memiliki pengaruh nyata terhadap tingkat ketahanan 22 klon IRR seri 300 baik di laboratorium maupun di rumah kaca. Isolat CG-PR-303 memiliki tingkat virulensi paling tinggi dibandingkan isolat lainnya. Berdasarkan hasil pengamatan pada tiga kegiatan dapat disimpulkan bahwa 13 klon karet yaitu IRR 300, IRR 301, IRR 302, IRR 307, IRR 308, IRR 309, IRR 310, IRR 311, IRR 313, IRR 315, IRR 316, IRR 318, dan IRR 321 memiliki tingkat resistensi yang tinggi terhadap penyakit gugur daun Colletotrichum.

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Kumar, Mode Girish, Umesha C., Gorla Prathyusha, and Pulibandla Avinash. "Performance of Timely Sown Wheat (Triticum aestivum L.) Genotypes under Restricted Irrigation Conditions in the Eastrern Regions of Uttar Pradesh." International Journal of Environment and Climate Change, July 20, 2022, 510–16. http://dx.doi.org/10.9734/ijecc/2022/v12i1131001.

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The field experiment was conducted during Rabi 2021-2022 genotypes at Wheat Breeding experimental Field, Naini Agriculture Institute, SHUATS, Prayagraj, Uttar Pradesh. The Soil of the experimental plots was sandy loam in texture with neutral soil reaction (pH 6.7). The experiment was laid out with six genotypes (NERI-301, NERI-302, NERI-303, NERI-304, NERI-305 and NERI-306) in a Randomized Block Design with four replications. Study revealed that growth parameters viz., higher plant height (105.26cm), number of effective tillers/hill (10.00/hill), and plant dry weight (22.13 g/plant) were recorded significantly higher for the genotype NERI-305.For yield attributing parameters viz., number of effective tillers (6.55/hill), number of grains/spike (63.07/spike),and test weight (49.38 g) were significantly highest for the genotype NERI-304, and yield parameters viz, grain yield (4.74 t/ha), harvest index (41.52%) were recorded significantly higher for the genotype NERI-304. spike length (13.35 cm) and straw yield (6.69t/ha) were r significantly highest for the genotype NERI-301. From the above findings gross return (1,18,500.00 INR/ha), net return (82,314.00 INR/ha) and Benefit cost ratio (2.27) were significantly highest for the genotype NERI-304.The genotype NERI-304 was found to be more potential as well as economically viable and productive over rest of the genotypes.

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Syafaah, Afdholiatus, Sigit Ismawanto, and Fetrina Oktavia. "PERTUMBUHAN TBM, KARAKTER FISIOLOGI, DAN KETAHANAN PENYAKIT KLON-KLON KARET IRR SERI 300 DI SUMATERA SELATAN." Jurnal Penelitian Karet, June 18, 2021, 1–10. http://dx.doi.org/10.22302/ppk.jpk.v39i1.774.

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Klon-klon karet IRR Seri 300 merupakan hasil persilangan yang dilakukan oleh Balai Penelitian Sungei Putih, Pusat Penelitian Karet pada tahun 1991 dan 1992. Pada tahun 2013 mulai dilakukan pengujian lanjutan di Kebun Percobaan Sembawa, Sumatera Selatan pada 22 klon-klon IRR seri 300 terpilih yang ditanam seluas 1 Ha masing-masing klon. Parameter pengamatan meliputi pertumbuhan tanaman, resistensi klon terhadap penyakit daun yang menyerang selama masa tanaman belum menghasilkan (TBM), karakter fisiologi awal, dan produksi tahun pertama. Hasil pengamatan pertumbuhan tanaman TBM pada 22 klon IRR Seri 300, terdapat enam klon yang mempunyai matang sadap pada umur 4,5 tahun yaitu IRR 300, IRR 301, IRR 302, IRR 307, IRR 309, dan IRR 310. Pengukuran lilit batang lanjutan sebelum penyadapan serentak dilakukan, terdapat 20 klon IRR seri 300 memiliki pertumbuhan TBM yang lebih baik daripada klon pembanding BPM 24 kecuali klon IRR 311 dan IRR 314. Selain itu, sebanyak delapan klon IRR seri 300 yang mempunyai lilit batang dan tebal kulit yang lebih baik daripada klon-klon lainnya (lilit batang > 45 cm dan tebal kulit > 5 cm), yaitu IRR 301, IRR 303, IRR 306, IRR 309, IRR 310, IRR 315, IRR 316, dan IRR 323. Kedelapan klon tersebut juga mempunyai tingkat resistensi tergolong resisten-sangat resisten terhadap penyakit gugur daun Corynespora dan Colletotrichum. IRR 306 dan IRR 310 merupakan salah satu klon harapan baru yang dapat dikembangkan di wilayah Sumatera Selatan. Pengamatan lanjutan masih perlu dilakukan untuk mendapatkan data pertumbuhan dan produksi klon-klon IRR Seri 300 pada masa tanaman menghasilkan (TM).

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