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Relevant bibliographies by topics / 5-a] pyrimidin-3-yl)phenol(PHTPP)

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Journal articles

Academic literature on the topic '5-a] pyrimidin-3-yl)phenol(PHTPP)'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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Journal articles on the topic "5-a] pyrimidin-3-yl)phenol(PHTPP)"

1

Zang, Kai-Kai, Xiao Xiao, Li-Qiang Chen, et al. "Distinct Function of Estrogen Receptors in the Rodent Anterior Cingulate Cortex in Pain-related Aversion." Anesthesiology 133, no. 1 (2020): 165–84. http://dx.doi.org/10.1097/aln.0000000000003324.

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Background Brain-derived estrogen is implicated in pain-related aversion; however, which estrogen receptors mediate this effect remains unclear. This study hypothesized that the different estrogen receptors in the rostral anterior cingulate cortex play distinct roles in pain-related aversion. Methods Formalin-induced conditioned place avoidance and place escape/avoidance paradigms were used to evaluate pain-related aversion in rodents. Immunohistochemistry and Western blotting were used to detect estrogen receptor expression. Patch-clamp recordings were used to examine N-methyl-d-aspartate–med

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Ben-Jonathan, Nira, Shenglin Chen, Joseph A. Dunckley, Christopher LaPensee та Sanjay Kansra. "Estrogen Receptor-α Mediates the Epidermal Growth Factor-Stimulated Prolactin Expression and Release in Lactotrophs". Endocrinology 150, № 2 (2009): 795–802. http://dx.doi.org/10.1210/en.2008-0756.

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Epidermal growth factor (EGF) is a potent regulator of cell function in many cell types. EGF-receptor (EGFR/ErbB1)-activated Erk1/2 has been reported to activate estrogen receptor (ER) in an estrogen (E2)-independent manner. In the pituitary lactotrophs, both EGF and E2 stimulate prolactin (PRL) release, but the nature of interactions between ErbB and ERα signaling is unknown. Our objectives were to 1) characterize EGF-induced PRL release, 2) determine whether this effect requires ERα, and 3) determine the molecular basis for cross talk between ErbB and ERα signaling pathways. Using GH3 cells,

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Chan, Karen Kar-Loen, Thomas Ho-Yin Leung, David Wai Chan та ін. "Targeting estrogen receptor subtypes (ERα and ERβ) with selective ER modulators in ovarian cancer". Journal of Endocrinology 221, № 2 (2014): 325–36. http://dx.doi.org/10.1530/joe-13-0500.

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Ovarian cancer cells express both estrogen receptor α (ERα) and ERβ, and hormonal therapy is an attractive treatment option because of its relatively few side effects. However, estrogen was previously shown to have opposite effects in tumors expressing ERα compared with ERβ, indicating that the two receptor subtypes may have opposing effects. This may explain the modest response to nonselective estrogen inhibition in clinical practice. In this study, we aimed to investigate the effect of selectively targeting each ER subtype on ovarian cancer growth. Ovarian cancer cell lines SKOV3 and OV2008,

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Hernández-Vega, Ana M., Aylin Del Moral-Morales, Carmen J. Zamora-Sánchez, Ana G. Piña-Medina, Aliesha González-Arenas, and Ignacio Camacho-Arroyo. "Estradiol Induces Epithelial to Mesenchymal Transition of Human Glioblastoma Cells." Cells 9, no. 9 (2020): 1930. http://dx.doi.org/10.3390/cells9091930.

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The mesenchymal phenotype of glioblastoma multiforme (GBM), the most frequent and malignant brain tumor, is associated with the worst prognosis. The epithelial–mesenchymal transition (EMT) is a cell plasticity mechanism involved in GBM malignancy. In this study, we determined 17β-estradiol (E2)-induced EMT by changes in cell morphology, expression of EMT markers, and cell migration and invasion assays in human GBM-derived cell lines. E2 (10 nM) modified the shape and size of GBM cells due to a reorganization of actin filaments. We evaluated EMT markers expression by RT-qPCR, Western blot, and

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Wang, Mengxia, Xiaoxia Yang, Qian Zhou, et al. "Neuroprotective Mechanism of Icariin on Hypoxic Ischemic Brain Damage in Neonatal Mice." Oxidative Medicine and Cellular Longevity 2022 (November 15, 2022): 1–32. http://dx.doi.org/10.1155/2022/1330928.

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Objective. Our previous results showed that icariin (ICA) could inhibit apoptosis and provide neuroprotection against hypoxic-ischemic brain damage (HIBD) in neonatal mice, but the specific mechanism of its neuroprotective effect remains unknown. This study aims at exploring whether ICA plays a neuroprotective role in apoptosis inhibition by regulating autophagy through the estrogen receptor α (ERα)/estrogen receptor β (ERβ) pathway in neonatal mice with HIBD. Methods. A neonatal mouse model of HIBD was constructed in vivo, and an oxygen and glucose deprivation (OGD) model in HT22 cells from t

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Grassi, Daniela, Maria Jose Bellini, Estefania Acaz-Fonseca, GianCarlo Panzica та Luis M. Garcia-Segura. "Estradiol and Testosterone Regulate Arginine-Vasopressin Expression in SH-SY5Y Human Female Neuroblastoma Cells Through Estrogen Receptors-α and -β". Endocrinology 154, № 6 (2013): 2092–100. http://dx.doi.org/10.1210/en.2012-2137.

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Abstract The expression of arginine-vasopressin (AVP) is regulated by estradiol and testosterone (T) in different neuronal populations by mechanisms that are not yet fully understood. Estrogen receptors (ERs) have been shown to participate in the regulation of AVP neurons by estradiol. In addition, there is evidence of the participation of ERβ in the regulation of AVP expression exerted by T via its metabolite 5α-dihydrotestosterone (5α-DHT) and its further conversion in the androgen metabolite and ERβ ligand 3β-diol. In this study we have explored the role of ERs in the regulation exerted by

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Bandyopadhyay, M., L. Seidman, E. O'Quinn, and B. Rohrer. "Estrogen Ameliorates Photoreceptor Cell Loss In Light-Induced Retinal Degenerated Balb/C Mice." October 3, 2017. https://doi.org/10.19070/2332-290X-1700069.

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Purpose: Estrogen or 17β-estradiol (17β-E2) is known to act as an antioxidant and has neuroprotective effects. The purpose of this study is to evaluate the effects of estrogen and estrogen receptor beta (ERβ) on photoreceptor cell structure and function in an experimental model for light-induced photoreceptor degeneration. Methods: 3-5 months old Balb/c mice were exposed to constant light of ~1500 lux. Mice were divided into four groups and treated via intraperitoneal injections with vehicle, 17β-E2, the ERβ inhibitor [4-(2-phenyl-5,7-bis(trifluoromethyl) pyrazolo[1,5-a]pyrimidin-3-yl)phenol,

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Banerjee, Samya, Apurba Mandal, Rohit Rai, Arif Ali Mandal, and Prodyut Dhar. "Vitamin B6 appended polypyridyl Co(III) complexes for photo‐triggered antibacterial activity." Chemistry – An Asian Journal, September 11, 2024. http://dx.doi.org/10.1002/asia.202400943.

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Three novel polypyridyl‐Co(III)‐vitamin B6 complexes viz., [Co(CF3‐phtpy)(SBVB6)]Cl (Co1), [Co(anthracene‐tpy)(SBVB6)]Cl (Co2), [Co(NMe2‐phtpy)(SBVB6)]Cl (Co3), where 4'‐(4‐(trifluoromethyl)phenyl)‐2,2':6',2''‐terpyridine = CF3‐phtpy, 4'‐(anthracen‐9‐yl)‐2,2':6',2''‐terpyridine = anthracene‐tpy;, 4‐([2,2':6',2''‐terpyridin]‐4'‐yl)‐N,N‐dimethylaniline = NMe2‐phtpy, (E)‐5‐(hydroxymethyl)‐4‐(((2‐hydroxyphenyl)imino)methyl)‐2‐methylpyridin‐3‐ol = H2SBVB6 was successfully developed for aPDT (antibacterial photodynamic therapy) applications. Co1‐Co3 exhibited an intense absorption band at ca. 435‐48

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SARI, JELLY MILINIA PUSPITA, SITI HERLINDA, ELFITA ELFITA, and SUWANDI SUWANDI. "Various secondary metabolites of endophytic fungi induced by a newly modified medium and their insecticidal activities." Biodiversitas Journal of Biological Diversity 26, no. 4 (2025). https://doi.org/10.13057/biodiv/d260437.

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Abstract. Sari JMP, Herlinda S, Elfita, Suwandi S. 2025. Various secondary metabolites of endophytic fungi induced by a newly modified medium and their insecticidal activities. Biodiversitas 26: 1881-1890. This research aimed to identify and increase the variation of secondary metabolites of Penicillium citrinum, Beauveria bassiana, and Metarhizium anisopliae from South Sumatra and to determine their insecticidal activities. The fungi were grown on a Glucose Yeast Extract Broth (GYB) medium enriched with cricket powder and a Potato Dextrose Broth (PDB) medium. Secondary metabolite compounds fr

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