Relevant bibliographies by topics / 895.6/12

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Academic literature on the topic '895.6/12'

Author: Grafiati
Published: 25 July 2024
Last updated: 31 July 2025

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Journal articles on the topic "895.6/12"

1

Littlejohn, James Blake, Emily Evans Grenn, Kristen T. Carter, et al. "INCREASED PLATELET MITOCHONDRIAL FUNCTION CORRELATES WITH CLOT STRENGTH IN A RODENT FRACTURE MODEL." Journal of Trauma and Acute Care Surgery, November 13, 2023. http://dx.doi.org/10.1097/ta.0000000000004204.

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Abstract Background Thromboelastographic measures of clot strength increase early after injury, portending higher risks for thromboembolic complications during recovery. Understanding the specific role of platelets is challenging due to a lack of clinically relevant measures of platelet function. Platelet mitochondrial respirometry may provide insight to global platelet function, but has not yet been correlated with functional coagulation studies. Methods Wistar rats underwent anesthesia and either immediate sacrifice for baseline values [n = 6] or (1) bilateral hindlimb orthopedic injury [n =
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Nakanishi, Nobuto, Yuko Ono, Yusuke Miyazaki, et al. "Sepsis causes neutrophil infiltration in muscle leading to muscle atrophy and weakness in mice." Frontiers in Immunology 13 (October 31, 2022). http://dx.doi.org/10.3389/fimmu.2022.950646.

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BackgroundSepsis-induced muscle atrophy leads to prolonged physical dysfunction. Although the interaction of muscle atrophy and macrophage has been reported in sepsis, the role of neutrophils in muscle atrophy has not been thoroughly investigated. This study sought to investigate the long-term changes in muscle-localized neutrophils after sepsis induction and their possible role in sepsis.MethodsSepsis was induced in seven-week-old male C57BL/6J mice 8-12 (cecal slurry [CS] model) via intraperitoneal injection of 1 mg/g cecal slurry. The percentage change in body weight and grip strength was e
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Hansen, Barbara Caleen, Zhigang Liang, Fengjiao Sun, et al. "Nonalcoholic Fatty Liver Disease (NAFLD) in Obese Rhesus Monkeys Provides the First Animal Model that Accurately Reflects the Human Condition." FASEB Journal 31, S1 (2017). http://dx.doi.org/10.1096/fasebj.31.1_supplement.895.6.

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None of the many previous animal models of NAFLD/NASH, whether genetic mouse models or aberrant dietary challenges, replicate or fully reflect either the metabolic pathophysiology or the histopathology of patients with NAFLD/NASH. We report for the first time the development and characterization of NASH in middle‐aged naturally overweight nonhuman primates. This colony has been maintained continuously on a healthy low fat high fiber diet and by all parameters, it is clear that these rhesus monkeys have developed the same disease as develops so commonly in humans.MethodsNAFLD/NASH histopatholog
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Books on the topic "895.6/12"

1

Kamo, Chōmei. Hōjōki to Kamo no Chōmei: Shinshiten, tettei tsuiseki. Bensei Shuppan, 2012.

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2

Kamo, Chōmei. Hōjōki. Tsurezuregusa. Shōgakkan, 1986.

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3

Kamo, Chōmei. Aufzeichnungen aus meiner Hütte. Insel, 1997.

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Kamo, Chōmei. Hōjōki. Tsurezuregusa. Shōbō genzō zuimonki. Tannishō. Shōgakkan, 1995.

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Kamo, Chōmei. Hōjōki: Zen. Kabushiki Kaisha Kadokawa Gakugei Shuppan, 2013.

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6

Kamo, Chōmei. Hojoki: Canto a la vida desde una choza = Supeingo de kanaderu Hōjōki. Taiseido, 2015.

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7

Kamo, Chōmei. Hōjōki. Shōgakkan, 1995.

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8

Kamo, Chōmei. Hōjōki: Hosshinshu. Shinchōsha, 2016.

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9

Kamo, Chōmei. Emaki de yomu Hojoki. Tokyo Bijutsu, 2022.

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Kamo, Chōmei. Hōjōki: Tsurezuregusa. Tannishō. Shōgakkan, 2007.

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