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Books on the topic '90-90-90 Target'

Author: Grafiati

Published: 5 June 2025

Last updated: 24 June 2025

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1

Davenport, Steven. Harden target: Defensive living for the 90's : a guide to services, self-protection & self-defense. Shield Pub. Co., 1992.

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2

Newcastle upon Tyne (England). City Council. Medium term plan 1989/90 - 1992/3: Statement of aims, objectives and targets. City Council, 1989.

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3

ṿeha-tarbut, Israel Miśrad ha-ḥinukh, ред. ha-Hotsaʾah ha-leʾumit le-tarbut, le-vidur ṿeli-sporṭ, 1984/85-1989/90. ha-Lishkah ha-merkazit li-sṭaṭisṭiḳah, 1993.

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4

Sepowski, Stephen J., ed. The Ultimate Hint Book. The Ultimate Game Club Ltd., 1991.

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5

Chase, Miss Allison. Target Language Toolkit: 90 Ideas To Get Your Language Learners Using More Target Language. Createspace Independent Publishing Platform, 2015.

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6

Staff, International Monetary Fund. Working Paper 90/106; the Simplest Test of Target Zone Credibility. International Monetary Fund, 1990.

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7

Fund, International Monetary. Working Paper 90/106; the Simplest Test of Target Zone Credibility. International Monetary Fund, 1990.

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8

Staff, International Monetary Fund. Working Paper 90/106; the Simplest Test of Target Zone Credibility. International Monetary Fund, 1990.

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9

Toward 90+% Target Language Use: Practical Tips from an Award-Winning Foreign Language Teacher. Independently Published, 2021.

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10

Community, Engineer. Daily Target Just 5 Mun Befor Starting Woork: Daily Planner Journal, to Do List Notebook, Daily Organizer, Targets Deffined , 90 Pages. Independently Published, 2020.

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11

News, World Spaceflight. 21st Century U.S. Army Joint Theater Missile Target Development Field Manual (FM 90-43) - Multiservice Procedures, Tactics, Techniques, Cruise Missiles, Theater Ballistic Missiles. Progressive Management, 2003.

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12

Gifford, Lorri. 90 Days to Learning the Tarot: No Memorization Required! Schiffer Publishing, Limited, 2014.

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13

Staff, International Monetary Fund. Working Paper 90/41; Targets, Indicators and Instruments of Monetary Policy. International Monetary Fund, 1990.

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14

Staff, International Monetary Fund. Working Paper 90/41; Targets, Indicators and Instruments of Monetary Policy. International Monetary Fund, 1990.

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15

Fund, International Monetary. Working Paper 90/41; Targets, Indicators and Instruments of Monetary Policy. International Monetary Fund, 1990.

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16

Dixon, Bradley P., J. Christopher Kingswood, and John J. Bissler. Tuberous sclerosis complex renal disease. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0330.

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Tuberous sclerosis complex (TSC) is an autosomal dominant genetic disorder affecting almost all organs. It has wider phenotypic variation than often appreciated, with less than half showing the combination of characteristic facial angiofibromas, epilepsy, and mental retardation. Renal angiomyolipomata or cysts are found in 90% and renal failure was historically a common mode of adult death from the disease. Pulmonary lymphangioleiomyomatosis is restricted to females. Angiomyolipomata or cystic disease, or both, may cause renal failure. Angiomyolipomata may also haemorrhage, especially from larger lesions. Manifestations of brain involvement substantially complicate management of many patients with TSC. The causative genes TSC1 and TSC2 encode tuberin and hamartin which are involved in control of the mammalian target of rapamycin pathway. Inhibitors of that pathway, such as sirolimus and everolimus, are therefore logical approaches to therapy and have been shown to be effective in reducing angiomyolipomata volume. It remains to be seen whether they can protect renal function.

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17

Lawlor, Peter. Multicomponent Intervention to Prevent Delirium in Hospitalized Older Patients (DRAFT). Edited by Nathan A. Gray and Thomas W. LeBlanc. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190658618.003.0011.

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This chapter, reports on a prospectively matched controlled trial in which Inouye et al. examined the comparative effectiveness of a targeted multicomponent strategy for reducing the risk of delirium with that of usual standard care. The six targeted baseline risk factors in delirium-free patients admitted to a medical service in a teaching hospital were cognitive impairment, sleep deprivation, immobility, visual impairment, hearing impairment, and dehydration. Using standardized interventions for each of these when present, 42/426 (9.9%) of those in the intervention group had a first-incident episode of delirium compared to 64/426 (15%) in the usual care group, representing a statistically significant reduction of approximately 33% in first-incident episodes of delirium. The number of patient-days with delirium (105 vs. 161) and delirium episodes (62 vs. 90) were significantly lower in the intervention group. Primary prevention of delirium was effective.

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18

Banerjee, Amitava, and Kaleab Asrress. Prevention of cardiovascular disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0343.

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The global scale of the cardiovascular disease epidemic is unquestionable, with cardiovascular disease causing a greater burden of mortality and morbidity than any other disease, regardless of country or population. With demographic change and ageing populations, the prevalence of cardiovascular disease and its risk factors is set to increase. The commonest cardiovascular diseases are atherosclerotic, affecting all arterial territories. The ‘burden of disease’ approach has highlighted the fact that cardiovascular disease and non-communicable diseases are not simply diseases of affluence but affect people of all countries, with enormous costs in terms of public health, healthcare, and overall economies. Coronary artery disease is the leading cause of mortality in all regions of the world apart from sub-Saharan Africa, followed by cerebrovascular disease. It should be noted, however, that there has been a major decline in cardiovascular disease mortality in Western Europe, the US, and Japan over the past 40 years. There are multiple factors underlying these favourable trends but understanding the epidemiology and characterizing individual risk factors for cardiovascular disease has been central in formulating preventive and treatment strategies. The INTERHEART study showed that 90% of cardiovascular risk can be explained by nine easily identifiable risk factors; an awareness of these, and the discovery of novel factors, will continue to serve in the fight to reduce the burden of cardiovascular disease. Geoffrey Rose first championed population-wide approaches versus strategies which target only high-risk individuals. Prevention aims to ‘catch the disease’ upstream, therefore delaying, reducing, or eliminating the risk of coronary artery disease. Surrogate markers for coronary artery disease have emerged in efforts to detect disease at earlier stages, and in order to better understand the pathophysiology. For example, coronary artery calcium scoring is emerging as a marker of future risk of coronary artery disease. Risk stratification scores are increasingly used as tools to individualize a person’s future risk of coronary artery disease in order to better target treatment and prevention strategies.

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19

Julien, Mic, Rachel McFadyen, and Jim Cullen, eds. Biological Control of Weeds in Australia. CSIRO Publishing, 2012. http://dx.doi.org/10.1071/9780643104204.

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Biological control of weeds has been practised for over 100 years and Australia has been a leader in this weed management technique. The classical example of control of prickly pears in Australia by the cactus moth Cactoblastis cactorum, which was imported from the Americas, helped to set the future for biocontrol of weeds in many countries. Since then there have been many projects using Classical Biological Control to manage numerous weed species, many of which have been successful. Importantly, there have been no serious negative non-target impacts – the technique, when practised as it is in Australia, is safe and environmentally friendly. Economic assessments have shown that biocontrol of weeds in Australia has provided exceedingly high benefit-to-cost ratios. This book reviews biological control of weeds in Australia to 2011, covering over 90 weed species and a multitude of biological control agents and potential agents. Each chapter has been written by practising biological control of weeds researchers and provides details of the weed, the history of its biological control, exploration for agents, potential agents studied and agents released and the outcomes of those releases. Many weeds were successfully controlled, some were not, many projects are still underway, some have just begun, however all are reported in detail in this book.  Biological Control of Weeds in Australia will provide invaluable information for biological control researchers in Australia and elsewhere. Agents used in Australia could be of immense value to other countries that suffer from the same weeds as Australia. The studies reported here provide direction to future research and provide examples and knowledge for researchers and students.

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20

Ugarte-Gil, Manuel F., and Graciela S. Alarcón. History of systemic lupus erythematosus. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198739180.003.0001.

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The first description of cutaneous ulcerations consistent with systemic lupus erythematosus (SLE) has been attributed to Hippocrates. The term lupus first appeared in English literature in the tenth century. Until the nineteenth century, however, this term was used to describe different conditions. Osler first recognized that organ involvement may occur with or without skin involvement. With the discovery of LE cells and autoantibodies, the use of lupus murine models, and the recognition of familial aggregation and the importance of genetic factors, the pathogenesis of SLE started to be unravelled and allowed the definition of classification criteria. In parallel, the discovery of cortisone, the use of immunosuppressive drugs and antimalarials, the control of hypertension, and the availability of renal replacement therapy improved the prognosis of SLE from a 4-year survival of 51% to a 5-year survival >90%. Advances in genetics and targeted therapies will lead to better intermediate and long-term outcomes.

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21

Newpower, Anthony. Iron Men and Tin Fish. Greenwood Publishing Group, Inc., 2006. http://dx.doi.org/10.5040/9798400672774.

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From the American entry into World War II until September 1943, U.S. submarines experienced an abnormally high number of torpedo failures. These failures resulted from three defects present in the primary torpedo of the day, the Mark XIV. These defects were a tendency to run deeper than the set depth, the frequent premature detonation of the Mark 6 magnetic influence exploder, and the failure of the contact exploder when hitting a target at the textbook ninety-degree angle. Ironically, despite using a completely independent design, the Germans experienced the same three defects. The Germans, however, fixed their defects in six months, while it took the Americans twenty-two months. Much of the delay on the American side resulted from the denial of senior leaders in the operational forces and in the Navy's Bureau of Ordnance (BuOrd) that the torpedo itself was defective. Instead, they blamed crews for poor marksmanship or lack of training. In the end, however, the submarine force itself overcame the bureaucratic inertia and correctly identified and fixed the three problems on their own, proving once again the industry of the average American soldier or sailor. From the American entry into World War II until September 1943, U.S. submarines experienced an abnormally high number of torpedo failures. These failures resulted from three defects present in the primary torpedo of the day, the Mark XIV. These defects were a tendency to run deeper than the set depth, the frequent premature detonation of the magnetic influence exploder, and the failure of the contact exploder when hitting a target at the textbook 90-degree angle. Ironically, despite using a completely independent design, the Germans experienced the same three defects. The Germans, however, fixed their defects in six months, while it took the Americans 22 months. Much of the delay on the American side resulted from the denial of senior leaders in the operational forces and in the Navy's Bureau of Ordnance (BuOrd) that the torpedo itself was defective. Instead, they blamed crews for poor marksmanship or lack of training. In the end, however, the submarine force itself overcame the bureaucratic inertia and correctly identified and fixed the three problems on their own, proving once again the industry of the average American soldier or sailor. Contrary to the interpretations of most submarine historians, this book concludes that BuOrd did not sit idly by while torpedoes failed on patrol after patrol. BuOrd acknowledged problems from early in the war, but their processes and their tunnel vision prevented them from realizing that the weapon sent to the fleet was grossly defective. One of World War II's forgotten heroes, Admiral Lockwood drove the process for finding and fixing the three major defects. This is first book that deals exclusively with the torpedo problem, building its case out of original research from the archives of the Bureau of Ordnance, the Chief of Naval Operations, Vice Admiral Lockwood's personal correspondence, and records from the British Admiralty at the National Archives of the United Kingdom. These sources are complemented by correspondence and interviews with men who actually participated in the events.

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22

Ferro, Charles J., and Khai Ping Ng. Recommendations for management of high renal risk chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0099.

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Poorer renal function is associated with increasing morbidity and mortality. In the wider population this is mainly as a consequence of cardiovascular disease. Renal patients are more likely to progress to end-stage renal disease, but also have high cardiovascular risk. Aiming to reduce both progression of renal impairment and cardiovascular disease are not contradictory. Focusing on the management of high-risk patients with proteinuria and reduced glomerular filtration rates, it is recommended that blood pressure should be kept below 140/90, or 130/80 if proteinuria is > 1 g/24 h (protein:creatinine ratio (PCR) >100 mg/mmol or 0.9 g/g). These targets may be modified according to age and other factors. Angiotensin-converting enzyme inhibitors (ACEIs) or angiotensin receptor antagonists should form part of the therapy for patients with proteinuria > 0.5 g/24 h (PCR > 50 mg/mmol or 0.45 g/g). Use of ACEIs or angiotensin receptor blockers in patients with lower levels of proteinuria may be indicated in some patient groups even in the absence of hypertension, notably in diabetic nephropathy. Evidence that other agents that reduce proteinuria bring additional benefits is weak at present. The best studies of ‘dual-blockade’ with various combinations of ACEIs, ARBs, and renin inhibitors have shown additional hazard with little evidence of additional benefit. Hyperlipidaemia—regardless of lipid levels, statin therapy is indicated in secondary cardiovascular prevention, and in primary prevention where cardiovascular risk is high, noting that current risk estimation tools do not adequately account for the increased risk of patients with CKD. There is not substantial evidence that lipid lowering therapy impacts on average rates of loss of GFR in progressive CKD. Non-drug lifestyle interventions to reduce cardiovascular risk, including stopping smoking, are important for all. Acidosis—in more advanced CKD it is justified to treat acidosis with oral sodium bicarbonate. Diet—sodium restriction to < 100 mmol/day (6 g/day) and avoidance of excessive dietary protein are justified in early to moderate CKD. Recommendations to limit levels of protein to 0.8 g/kg body weight are suggested by some, but additional protective effects of this are likely to be slight in patients who are otherwise well managed. Low-protein diets may carry some risk. Lower-protein diets may however be used to prevent symptoms in advanced CKD not treated by dialysis.

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