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Journal articles on the topic 'ABO antigens'

Author: Grafiati

Published: 4 June 2021

Last updated: 11 February 2022

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1

Anani, Waseem Q., Heather E. Ashwood, Anna Schmidt, Robert T. Burns, Gregory A. Denomme, and Karin M. Hoffmeister. "Predictive modeling of complex ABO glycan phenotypes by lectin microarrays." Blood Advances 4, no. 16 (2020): 3960–70. http://dx.doi.org/10.1182/bloodadvances.2020002051.

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Abstract Serological classification of individuals as A, B, O, or AB is a mainstay of blood banking. ABO blood groups or ABH antigens, in addition to other surface glycans, act as unique red blood cell (RBC) signatures and direct immune responses. ABO subgroups present as weakened, mixed field, or unexpected reactivity with serological reagents, but specific designations remain complex. Lectins detect glycan motifs with some recognizing ABH antigens. We evaluated a 45-probe lectin microarray to rapidly analyze ABO blood groups and associated unique glycan signatures within complex biological s

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2

Martinez, Aline Amorim, Alessandra Midori Kuniyoshi, Mihoko Yamamoto, et al. "Loss of red cell ABH Antigens and Hypermethylation of the ABO Gene Promoter region is frequent in Myeloid Malignancies." Blood 114, no. 22 (2009): 3151. http://dx.doi.org/10.1182/blood.v114.22.3151.3151.

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Abstract Abstract 3151 Poster Board III-88 Background Loss of red blood cell (RBC) antigens may occur in solid tumors and in hematological diseases; however the mechanisms involved in these changes are not fully understood. Aberrant DNA hypermethylation is thought to be involved in acute myeloid leukemia (AML) as well as in myelodysplastic syndromes (MDS), and the methylation of cytosines residues in the dinucleotide CpG may account for the expression patterns of the ABO genes. In this study we investigated loss of RBC ABH antigens and the possible role of DNA methylation in the ABO promoter g

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3

Glik, Ami, Lilach Bonstein, Nardeen Atwah, et al. "Stable Blood Group Chimerism in Recipients of ABO Incompatible Allogeneic Stem Cell Transplantation." Blood 124, no. 21 (2014): 1553. http://dx.doi.org/10.1182/blood.v124.21.1553.1553.

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Abstract Background: The ability to express the ABH antigens in oral mucosa and saliva is present in about 80% of the general population (hence called secretors) and is regulated by the Sec gene on chromosome 19q13.3, which encodes for the enzyme fut2 (α(1,2)Fucosyltransferase), resulting in the expression of blood types in body fluids, including saliva. The gene encoding for the ABH antigens that determines ABO blood type, is located on chromosome 9q34, and transcribes to the enzyme fut1. While fut1 is mainly expressed in erythroid tissue, fut2is expressed in secretory cells. The aim of the p

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4

Dunstan, RA, MB Simpson, RW Knowles, and WF Rosse. "The origin of ABH antigens on human platelets." Blood 65, no. 3 (1985): 615–19. http://dx.doi.org/10.1182/blood.v65.3.615.615.

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Abstract ABH antigens are present on platelets from individuals of the corresponding red cell phenotype, but the extent to which these antigens are intrinsic or adsorbed remains undefined. To evaluate platelets for intrinsic H substance, an IgM mouse monoclonal antibody against type 2H chain (the intrinsic H structure found on erythrocytes) was labeled with 125I and incubated with platelets from donors of different ABO type. The antibody showed dose-response saturation curves, and binding to platelets paralleled that of the red cell ABO type, with O greater than B greater than A1 greater than

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5

Dunstan, RA, MB Simpson, RW Knowles, and WF Rosse. "The origin of ABH antigens on human platelets." Blood 65, no. 3 (1985): 615–19. http://dx.doi.org/10.1182/blood.v65.3.615.bloodjournal653615.

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ABH antigens are present on platelets from individuals of the corresponding red cell phenotype, but the extent to which these antigens are intrinsic or adsorbed remains undefined. To evaluate platelets for intrinsic H substance, an IgM mouse monoclonal antibody against type 2H chain (the intrinsic H structure found on erythrocytes) was labeled with 125I and incubated with platelets from donors of different ABO type. The antibody showed dose-response saturation curves, and binding to platelets paralleled that of the red cell ABO type, with O greater than B greater than A1 greater than A1B great

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6

Dunbar, Nancy M. "Does ABO and RhD matching matter for platelet transfusion?" Hematology 2020, no. 1 (2020): 512–17. http://dx.doi.org/10.1182/hematology.2020000135.

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Abstract Platelets express ABO antigens and are collected in plasma, which contains ABO antibodies as would be consistent with the donor ABO group. Platelet ABO antigens that are incompatible with recipient ABO antibodies may have accelerated clearance from circulation and result in lower count increments. ABO antibodies that are passively transferred from donor plasma may result in hemolysis of recipient red blood cells. Although platelets do not express Rh antigens, they contain small numbers of intact red blood cells or fragments, which can lead to alloimmunization in the recipient. Alloimm

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7

Dabelsteen, E., and S. Gao. "ABO Blood-group Antigens in Oral Cancer." Journal of Dental Research 84, no. 1 (2005): 21–28. http://dx.doi.org/10.1177/154405910508400103.

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Tumor progression is often associated with altered glycosylation of the cell-surface proteins and lipids. The peripheral part of these cell-surface glycoconjugates often carries carbohydrate structures related to the ABO and Lewis blood-group antigens. The expression of histo-blood-group antigens in normal human tissues is dependent on the type of differentiation of the epithelium. In most human carcinomas, including oral carcinoma, a significant event is decreased expression of histo-blood-group antigens A and B. The mechanisms of aberrant expression of blood-group antigens are not clear in a

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8

Neil, Stuart J. D., Áine McKnight, Kenth Gustafsson, and Robin A. Weiss. "HIV-1 incorporates ABO histo-blood group antigens that sensitize virions to complement-mediated inactivation." Blood 105, no. 12 (2005): 4693–99. http://dx.doi.org/10.1182/blood-2004-11-4267.

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Abstract ABO histo-blood group antigens have been postulated to modify pathogen spread through the action of natural antibodies and complement. The antigens are generated by a polymorphic glycosyl-transferase encoded by 2 dominant active and a recessive inactive allele. In this study we investigated whether ABO sugars are incorporated into the envelope of HIV-1 virions. HIV vectors derived from cells expressing ABO antigens displayed sensitivity to fresh human serum analogous to ABO incompatibility, and ABO histo-blood group sugars were detected on the viral envelope protein, glycoprotein 120

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9

Onsten, Tor Gunnar Hugo, Sidia Maria Callegari-Jacques, and Luciano Zubaran Goldani. "The Higher Frequency of Blood Group B in a Brazilian Population with HIV Infection." Open AIDS Journal 7, no. 1 (2013): 47–50. http://dx.doi.org/10.2174/1874613601307010047.

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Objective:To analyze the frequency of and odds for and against HIV infection based on ABO blood type in a large sample of blood donors.Background:Coevolution between pathogens and hosts may explain the ABO system of polymorphisms. HIV-infected cells add ABO(H) blood group antigens to the viral envelope. Naturally occurring antibodies against ABO(H) antigens that are present in normal human sera are able to neutralize ABO-expressing HIVin vitro. Blood donors are ideal for studying blood groups and HIV infectionin vivobecause all donors are routinely typed and tested.Methods:All blood donors who

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10

Arthur, Connie M., Harold Clifford Sullivan, Christian Gerner-Smidt, et al. "Microbial Exposure Regulates the Development of Anti-Blood Group Antibodies." Blood 128, no. 22 (2016): 20. http://dx.doi.org/10.1182/blood.v128.22.20.20.

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Abstract Introduction: Anti-ABO antibodies represent the earliest recognized immunological barrier to transfusion and transplantation. However, despite Landsteiner's discovery of ABO blood group antigens over a century ago, the factors that regulate anti-ABO antibody formation remain relatively unknown. Anti-ABO antibodies develop spontaneously within the first few months of life, in the absence of a known antigenic exposure. However, antibody levels vary considerably between individuals suggesting that differences in exposure to environmental triggers may regulate anti-ABO antibody formation.

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11

Joshi, Bishal, Sanjit Kumar Kar, Shankar Yadav, et al. "Distribution of Blood Groups in Medical Students: A Comparative Study." Journal of Universal College of Medical Sciences 8, no. 1 (2020): 42–46. http://dx.doi.org/10.3126/jucms.v8i1.29837.

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INTRODUCTION: Blood groups depend on antigens present on the surface of red blood cells. Scientists have discovered at least 30 common antigens and hundreds of rare antigens causing antigen-antibody reaction in human red blood cells. These antigens are genetically determined and are developed in fetal life and remain unchanged till death. Many blood group systems are identified but ABO and Rh blood groups are more antigenic. MATERIAL AND METHODS: In the present study, we observed ABO and Rh blood groups of 3057 students who studied in Universal College of Medical Sciences, Bhairahawa, Ne

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12

Ogasawara, K., J. Ueki, M. Takenaka, and K. Furihata. "Study on the expression of ABH antigens on platelets." Blood 82, no. 3 (1993): 993–99. http://dx.doi.org/10.1182/blood.v82.3.993.993.

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Abstract We recently examined a case of refractoriness to HLA-matched, ABO- incompatible platelet transfusions. The transfused platelets that were rapidly cleared from the circulation of the recipient expressed an amount of B antigen more than 20 times that expressed by the blood group B platelets that were successfully transfused to the recipient. These observations led us to conduct enzyme-linked immunosorbent assay (ELISA) and immunoblotting studies of the amount of blood A and B antigens expressed on the surface of platelets from randomly selected donors. The donors were clearly classified

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13

Ogasawara, K., J. Ueki, M. Takenaka, and K. Furihata. "Study on the expression of ABH antigens on platelets." Blood 82, no. 3 (1993): 993–99. http://dx.doi.org/10.1182/blood.v82.3.993.bloodjournal823993.

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We recently examined a case of refractoriness to HLA-matched, ABO- incompatible platelet transfusions. The transfused platelets that were rapidly cleared from the circulation of the recipient expressed an amount of B antigen more than 20 times that expressed by the blood group B platelets that were successfully transfused to the recipient. These observations led us to conduct enzyme-linked immunosorbent assay (ELISA) and immunoblotting studies of the amount of blood A and B antigens expressed on the surface of platelets from randomly selected donors. The donors were clearly classified, accordi

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14

Matsui, Taei, Taketo Shimoyama, Masanori Matsumoto, et al. "ABO Blood Group Antigens on Human Plasma von Willebrand Factor After ABO-Mismatched Bone Marrow Transplantation." Blood 94, no. 8 (1999): 2895–900. http://dx.doi.org/10.1182/blood.v94.8.2895.420a03_2895_2900.

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von Willebrand factor (vWF) is synthesized exclusively by endothelial cells and megakaryocytes, and stored in the intracellular granules or constitutively secreted into plasma. ABO blood group antigens are covalently associated with asparagine-linked sugar chains of plasma vWF. The effect of ABO-mismatched bone marrow transplantation (BMT) or blood stem cell transplantation (BSCT) on the expression of ABO blood group antigens on the vWF was examined to obtain information on the origin of these antigens. In ABO-mismatched (HLA-matched) groups, 8 cases of BMT and 4 cases of BSCT were examined. I

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15

Fatima, Sabeen, Mona Aziz, Sindhu Rehman, Maliha Asif, Naseem Akhtar, and Yasmeen Batool. "KELL BLOOD GROUP ANTIGENS IN THE BLOOD DONORS ATTENDING BLOOD BANKS OF TERTIARY CARE HOSPITALS OF LAHORE, PAKISTAN." Professional Medical Journal 26, no. 07 (2019): 1167–71. http://dx.doi.org/10.29309/tpmj/2019.26.07.3792.

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Introduction: Among the complications of blood transfusion, Hemolytic transfusion reactions (HTRs) and Hemolytic disease of the newborn (HDN) are particularly important. Literature reports frequency of HTRs and related mortality up to 1/76,000 and 1/1.8 million units transfused respectively. These hemolytic reactions are caused by incompatibility between the donor and recipient blood and in cases of HDN, due to feto-maternal incompatibility due to maternal antibodies attacking the fetal red cells. Anti-K antibody is the most common antibody encountered in blood banks after the ABO and Rh antib

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16

Osby, Melanie, and Ira A. Shulman. "Phenotype Matching of Donor Red Blood Cell Units for Nonalloimmunized Sickle Cell Disease Patients: A Survey of 1182 North American Laboratories." Archives of Pathology & Laboratory Medicine 129, no. 2 (2005): 190–93. http://dx.doi.org/10.5858/2005-129-190-pmodrb.

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Abstract Context.—The transfusion of donor red blood cell units (RBCs) that lack certain red cell antigens (such as C, E, and K) when the corresponding antigens are absent from the recipient's red cells has been shown to reduce the risk of red cell alloimmunization in sickle cell disease patients. However, data are limited regarding the extent to which transfusion services routinely perform red cell antigen phenotype testing of nonalloimmunized sickle cell disease patients, and then use that information to select donor RBCs lacking 1 or more of the red cell antigens that the patient's red cell

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17

Crainic, K., M. Durigon, and R. Oriol. "ABO tissue antigens of egyptian mummies." Forensic Science International 43, no. 2 (1989): 113–24. http://dx.doi.org/10.1016/0379-0738(89)90130-8.

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18

Zarrinpar, Ali, and Ronald W. Busuttil. "Evading antigens—ABO-incompatible liver transplantation." Nature Reviews Gastroenterology & Hepatology 12, no. 12 (2015): 676–78. http://dx.doi.org/10.1038/nrgastro.2015.193.

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19

Kronstein-Wiedemann, Romy, Laura Schmidt, Jörn Lausen, Erhard Seifried, and Torsten Tonn. "Inhibition of the Transcription Factor RUNX1 Causes Glycosyltransferase a Repression." Blood 130, Suppl_1 (2017): 925. http://dx.doi.org/10.1182/blood.v130.suppl_1.925.925.

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Abstract Background: The ABO blood group system is unequivocally the most important in clinical transfusion medicine. Furthermore ABO is implicated in the development of a number of human diseases. The ABO antigens are not confined to RBCs but are widely expressed in a variety of human cells and tissues. Thus, ABO matching is critical not only in blood transfusion but also in cell, tissue and organ transplantation. The molecular genetic basis of the ABO system has been known since 1990. However, despite extensive investigations about regulation of ABO blood group receptor expression, the mecha

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20

Ngassaki-Yoka, Christ-Dominique, Jophrette Mireille Ntsame Ndong, and Cyrille Bisseye. "ABO, Rhesus blood groups and transfusion-transmitted infections among blood donors in Gabon." Sudan Journal of Medical Sciences 13, no. 1 (2018): 12. http://dx.doi.org/10.18502/sjms.v13i1.1685.

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Background: Few studies focused on the study of blood groups in Gabon. This study aimed to determine the phenotypic frequency of ABO and Rhesus antigens in blood donors of Libreville and to assess the association between ABO blood groups and transfusion-transmitted infections.Materials and Methods: The study of ABO and Rhesus blood groups concerned 4,744 blood donors. ABO and Rhesus phenotyping were obtained using monoclonal monospecific antisera: anti-A, anti-B, anti-AB, anti-D, anti-E, anti-C, anti-c, and anti-e with an automate (QWALYS® 3, DIAGAST, France) or a card gel (ID Card, BIO-RAD) a

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21

Lee, Jasmin, and Carol Guo. "Inconsistent trends regarding the association between ABO blood groups and susceptibility to SARS-CoV-2 infections." Journal of Undergraduate Life Sciences 15, no. 1 (2021): 6. http://dx.doi.org/10.33137/juls.v15i1.36956.

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ABO antigens, produced from the ABO gene, are known to impact host interactions with various viruses. One characteristic is the host’s susceptibility to viral infections. Host interaction with viral particles is altered by the blood type-determined combination of ABO antigens on the cellular surface. SARS-CoV-2 is a novel strain of the coronavirus family known to have structural similarities with SARS-CoV. Considering ABO antigens’ association with SARS-CoV, studies have examined their relationship with SARS-CoV-2 as well. We reviewed current perspectives on the relationship between host susce

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Prakobphol, Akraporn, Hakon Leffler, and Susan J. Fisher. "The High-Molecular-Weight Human Mucin Is the Primary Salivary Carrier of ABH, Lea, and Leb Blood Group Antigens." Critical Reviews in Oral Biology & Medicine 4, no. 3 (1993): 325–33. http://dx.doi.org/10.1177/10454411930040031001.

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Because many bacteria interact with the carbohydrate portions of receptor molecules, factors controlling glycosylation probably influence the ability of salivary components to mediate bacterial adherence/clearance. Important sources of diversity in glycosylation are the ABO, secretor, and Lewis genes, which code for glycosyltransferases that add specific sugar sequences to the termini of carbohydrate chains of glycolipids and glycoproteins. We identified, by Western blotting, salivary glycoproteins carrying the ABH and Le a or Leb antigens. Samples of whole, unstimulated saliva were obtained f

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23

Barragan, Antonio, Peter G. Kremsner, Mats Wahlgren, and Johan Carlson. "Blood Group A Antigen Is a Coreceptor inPlasmodium falciparum Rosetting." Infection and Immunity 68, no. 5 (2000): 2971–75. http://dx.doi.org/10.1128/iai.68.5.2971-2975.2000.

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ABSTRACT The malaria parasite Plasmodium falciparum utilizes molecules present on the surface of uninfected red blood cells (RBC) for rosette formation, and a dependency on ABO antigens has been previously shown. In this study, the antirosetting effect of immune sera was related to the blood group of the infected human host. Sera from malaria-immune blood group A (or B) individuals were less prone to disrupt rosettes from clinical isolates of blood group A (or B) patients than to disrupt rosettes from isolates of blood group O patients. All fresh clinical isolates and laboratory strains exhibi

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Houngkamhang, Nongluck, Apirom Vongsakulyanon, Patjaree Peungthum, et al. "Serum ABO Blood Typing by Surface Plasmon Resonance Technique." Advanced Materials Research 1131 (December 2015): 71–74. http://dx.doi.org/10.4028/www.scientific.net/amr.1131.71.

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Aim of this work is to develop a surface plasmon resonance (SPR) technique for detection of anti-A and anti-B which are the antibodies specific to ABO blood group in serum based on solid phase immobilization of antigens on the surface. Synthetic antigens A and B were immobilized on the carboxymethyldextran (CMD) surface to measure the antibodies. The immobilized synthetic antigen surface were tested function by injection of monoclonal anti-A and anti-B. Total 20 plasma samples at 1:10 dilution were measured by SPR techniuqe and the result were concordant to standard agglutination technique. Th

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25

TAKEDA, KATSUHIRO, and KOUICHI KIRAIWA. "ABO blood group antigens of human spermatozoa." Tohoku Journal of Experimental Medicine 147, no. 3 (1985): 267–72. http://dx.doi.org/10.1620/tjem.147.267.

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26

Kelton, JG, and G. Bebenek. "Granulocytes do not have surface ABO antigens." Transfusion 25, no. 6 (1985): 567–69. http://dx.doi.org/10.1046/j.1537-2995.1985.25686071432.x.

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27

Gylmiyarova, F. N., V. M. Radomskaya, O. A. Gusyakova, et al. "Modeling role of pyruvate in the processes of protein-protein interaction." Biomeditsinskaya Khimiya 61, no. 1 (2015): 132–40. http://dx.doi.org/10.18097/pbmc20156101132.

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Using the ABO antibody-antigen model the influence of natural metabolite pyruvate on the antibody interaction with of erythrocyte antigens, defining their group specificity has been investigated. Before agglutination reaction erythrocytes of A(II)-AB(IV) blood groups, monoclonal anti-A and anti-B antibodies were incubated with sodium pyruvate. Visualization of agglutinates was performed by means of flow cytometry and laser scanning confocal microscopy. Computer-aided prediction of the spectrum of biological activity of pyruvate by a PASS program proposed major regulatory pathways, in which pyr

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28

Spalter, Sergio H., Srini V. Kaveri, Emmanuelle Bonnin, Jean-Claude Mani, Jean-Pierre Cartron, and Michel D. Kazatchkine. "Normal Human Serum Contains Natural Antibodies Reactive With Autologous ABO Blood Group Antigens." Blood 93, no. 12 (1999): 4418–24. http://dx.doi.org/10.1182/blood.v93.12.4418.

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Abstract It is widely accepted that the serum of healthy individuals contains natural antibodies only against those blood group A or B antigens that are not expressed on the individual’s red blood cells. The mechanisms involved in tolerance to autologous blood group antigens remain unclear. In the present study, we show that IgM and IgG antibodies reactive with autologous blood group antigens are present in the immunoglobulin fraction of normal human serum. Natural IgG anti-A antibodies purified by affinity chromatography from IgG of individuals of blood group A exhibited an affinity for A tri

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Spalter, Sergio H., Srini V. Kaveri, Emmanuelle Bonnin, Jean-Claude Mani, Jean-Pierre Cartron, and Michel D. Kazatchkine. "Normal Human Serum Contains Natural Antibodies Reactive With Autologous ABO Blood Group Antigens." Blood 93, no. 12 (1999): 4418–24. http://dx.doi.org/10.1182/blood.v93.12.4418.412k20_4418_4424.

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It is widely accepted that the serum of healthy individuals contains natural antibodies only against those blood group A or B antigens that are not expressed on the individual’s red blood cells. The mechanisms involved in tolerance to autologous blood group antigens remain unclear. In the present study, we show that IgM and IgG antibodies reactive with autologous blood group antigens are present in the immunoglobulin fraction of normal human serum. Natural IgG anti-A antibodies purified by affinity chromatography from IgG of individuals of blood group A exhibited an affinity for A trisaccharid

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30

NasrEldin, Eman, Safaa A. A. Khaled, Nada O. Abdelhameed, et al. "Genotyping versus phenotyping of non-ABO erythrocyte antigens in patients with the Mediterranean hemopathic syndromes: Effect of transfusion therapy." PLOS ONE 16, no. 7 (2021): e0251576. http://dx.doi.org/10.1371/journal.pone.0251576.

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The Mediterranean hemopathic syndromes (MHS) are the most prevalent hemoglobinopathies in the Mediterranean basin. Transfusion therapy is the main therapy for these disorders, particularly for severe forms of the disease. Currently, pre-transfusion serological typing of erythrocyte antigens is the standard tool for reducing complications of transfusion in those patients. This study compared genotyping with phenotyping of non-ABO erythrocyte antigens in patients with MHS and assessed the effect of transfusion therapy on their results. One-hundred ninety-eight MHS patients were recruited, screen

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Maslov, Andrey A., Nailya Guskova, Ekaterina Guskova, et al. "The rates of ABO, Rh, and Kell antigens in children with cancer." Journal of Clinical Oncology 35, no. 15_suppl (2017): e22007-e22007. http://dx.doi.org/10.1200/jco.2017.35.15_suppl.e22007.

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e22007 Background: The purpose of the study was to analyze phenotypic characteristics of red blood cells by the AVBO, Rh and Kell systems in children with cancer. Methods: ABO and Rh blood groups were determined and erythrocyte antigens (D, С, с, Сw, Е, е, К, k) were typed (AutoVue Innova, USA) in blood samples of 114 children with solid tumors. Results: ABO blood groups distribution was as follows: A(II) > O(I) > B(III) > AB(IV) with A(II) prevalence. Rh(D)-positive phenotype was observed in 82 (71.9%) patients of 114: 47 (57.3%) boys and 35 (42.6%) girls. 32 (28.1%) patients of 114

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Venneker, Gerard T., Leo P. De Waal, Wiete Westerhof, Joe D'Amaro, Gezina M. Th Schreuder, and Syed S. Asghar. "ABO Genotyping of Complete Hydatidiform Moles." Disease Markers 11, no. 4 (1993): 187–90. http://dx.doi.org/10.1155/1993/356874.

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This study chanlcterizes the HLA class I and class II antigens in a group of patients with vitiligo and a control group, both of Dutch descent. Earlier reports had shown a significant positive association with DR4 and a significant negative association with DR3. We found that, after correction for the broad antigens studied, only Cw7 and DR6 were significantly associated with vitiligo. The significant positive association of DR6 with vitiligo is interesting since vitiligo has an autoimmune component in its pathogenesis and DR6 may be a marker for high immune responsiveness.

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Meloncelli, Peter J., and Todd L. Lowary. "Synthesis of ABO Histo-Blood Group Type V and VI Antigens." Australian Journal of Chemistry 62, no. 6 (2009): 558. http://dx.doi.org/10.1071/ch09058.

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The ABO histo-blood group antigens have long been of interest to chemists, biochemists, and evolutionary biologists. However, to date, a complete synthesis of all ABO histo-blood group antigens has not been conducted, despite the potential for such a panel to provide a more detailed understanding of the biological roles of these glycan motifs. Here we report the chemical synthesis of the A, B, and H type V and VI antigens in multi-milligramme quantities as part of an overall goal to prepare all 18 A, B, and H antigens. The A and B type V and VI antigens were prepared with a 7-octen-1-yl linker

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Curtis, Brian R., Janice G. McFarland, Andrea Fick, Andrew J. Lochowicz, Robert H. Ball, and Aster H. Richard. "Neonatal Alloimmune Thrombocytopenia (NATP) Associated with Maternal-Fetal Incompatibility for Blood Group B." Blood 106, no. 11 (2005): 955. http://dx.doi.org/10.1182/blood.v106.11.955.955.

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Abstract In most individuals, A and B blood group antigens are weakly expressed on platelets, allowing ABO incompatible platelets to be tolerated when transfused. However, in a minority of normal subjects (high expressers, H-Exp), platelets carry 10–20 times the usual number of A or B epitopes (up to 20,000/platelet, Blood2000;96:1574). Post-transfusion survival of incompatible H-Exp platelets has not been systematically studied. We recently encountered a family in which NATP in two infants appears to have been caused by maternal anti-B reacting with H-Exp fetal platelets inherited from a fath

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35

Morelli, Vania, Marieke de Visser, Nico van Tilburg, et al. "ABO blood group genotypes, plasma von Willebrand factor levels and loading of von Willebrand factor with A and B antigens." Thrombosis and Haemostasis 97, no. 04 (2007): 534–41. http://dx.doi.org/10.1160/th06-09-0549.

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SummaryABO blood group is a genetic determinant of von Willebrand factor (VWF) levels. We investigated the effect of ABO genotypes on VWF and factor VIII (FVIII) levels and on the degree to which VWF is loaded with A- and B-antigens, expressed as normalized ratios, nA-ratio and nB-ratio, respectively, in the Leiden Thrombophilia Study, a large case-control study on venous thrombosis. We found that the ABO locus had an allele-specific, dosage dependent effect on VWF and FVIII levels and on the loading of VWF with A-antigen and B-antigen. The highest mean nA- and nB-ratios were found in A 1 A 1

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Kishino, Koji, Kazuo Muroi, Yoko Nakaki, et al. "Analysis of ABH antigens in erythrocytes after ABO-Incompatible bone marrow transplantation." Journal of the Japan Society of Blood Transfusion 48, no. 4 (2002): 335–41. http://dx.doi.org/10.3925/jjtc1958.48.335.

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37

Murray, Glenn P., Steven R. Post, and Ginell R. Post. "ABO blood group is a determinant of von Willebrand factor protein levels in human pulmonary endothelial cells." Journal of Clinical Pathology 73, no. 6 (2019): 347–49. http://dx.doi.org/10.1136/jclinpath-2019-206182.

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ABO blood group antigens are expressed on von Willebrand factor (VWF) and glycosylation patterns influence circulating VWF levels. The aim of this study was to examine the effect of ABO blood type on tissue-associated VWF protein levels. We selected 35 formalin-fixed paraffin-embedded pulmonary tissue blocks obtained at autopsy from decedents who died from pulmonary embolism with known ABO blood groups (O, A, B and AB phenotypes), prepared tissue microarrays (TMAs) and stained TMAs with antibodies to VWF and platelet/endothelial cell adhesion marker-1 (PECAM-1) as a marker of endothelial cells

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Sequi-Canet, Jose Miguel, Jose Miguel Sequi-Sabater, Jose Ignacio Collar-Castillo, and Nelson Orta-Sibu. "Are ABO Blood Groups or Rh Antigen Perinatal Factors Affecting the Pass Rate of Transient Otoacoustic Emissions Screening Tests in Healthy Newborns during the First 48 h of Life?" International Journal of Neonatal Screening 5, no. 1 (2019): 4. http://dx.doi.org/10.3390/ijns5010004.

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Most hospitals recommend performing neonatal hearing screening. Transient evoked otoacoustic emission (TEOAE) tests represent an ideal technique for conducting this process. Previous studies have related the influence of ABO blood group and Rhesus antigens (Rh) on the susceptibility to various pathologies. However, available data about the potential relationship between ABO blood groups, Rh, and TEOAE pass rates are sparse. Recently, several authors concluded that O blood group and Rh+ are possible influential factors of TEOAE pass rates. Significantly different TEOAE amplitude response betwee

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Wang, Michael M., Soo Jung Lee, Jisu Kim, Jennifer J. Majersik, Mila Blaivas, and Jimo Borjigin. "ABO blood antigens define human cerebral endothelial diversity." NeuroReport 24, no. 2 (2013): 79–83. http://dx.doi.org/10.1097/wnr.0b013e32835c93a2.

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�rntoft, T. F. "S16.1 ABO and lewis antigens in bladder epithelium." Glycoconjugate Journal 10, no. 4 (1993): 322. http://dx.doi.org/10.1007/bf01210118.

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Chan, Esther HL, Thiow Christofer, Susan TT Lim, Lip Kun Tan, and Michelle Poon. "Evaluation of Blood Group Conversion Following ABO-Incompatible Hematopoietic Stem Cell Transplantation (HCT)." Blood 124, no. 21 (2014): 1557. http://dx.doi.org/10.1182/blood.v124.21.1557.1557.

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Abstract Title: Evaluation of blood group conversion following ABO-incompatible Hematopoietic Stem Cell Transplantation (HCT). Background: While there have been a number of reports concerning the influence of AB0 incompatibility on hematopoietic transplantation (HCT) outcomes, data evaluating post-HCT blood group conversions following ABO incompatible transplants remains extremely limited. Methods: We performed a single centre retrospective analysis of 97 patients undergoing major or minor or bidirectional ABO-mismatched bone marrow (BM), peripheral blood (PB), and cord blood (CB) hematopoieti

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Muellner, Sarah, Elliott Haut, Michael Streiff, John Holcomb, and Bryan Cotton. "ABO blood group as a potential risk factor for venous thromboembolism in acutely injured patients." Thrombosis and Haemostasis 105, no. 01 (2011): 5–13. http://dx.doi.org/10.1160/th10-08-0504.

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SummaryVenous thromboembolism (VTE) is a major health problem that results in a significant burden on hospitals and patients. VTE screening and prophylaxis protocols in trauma patients vary significantly among hospitals and providers. In addition, many patients develop VTE even in the absence of “high-risk” categories. Therefore, more research is needed to better understand and prevent VTE in these patients. ABO blood group has long been recognised as a risk factor for VTE, but its contribution to VTE risk in the trauma setting is poorly studied. This paper reviews the literature describing th

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Morais, Linduarte Varela, Aldair Sousa Paiva, Valéria SF Sales, and Geraldo Barroso Cavalcanti. "Genotypic and Alelicas Frequencies of the ABO and Rh Systems in the State of Rio Grande Do Norte, Northeast of Brazil." Blood 134, Supplement_1 (2019): 4990. http://dx.doi.org/10.1182/blood-2019-123802.

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Objective: To determine the immunophenotyping of a population of blood donors, intended to build a database for transfusion medicine. 2) To make available to patients with chronic diseases that are systematically dependent on blood transfusions as compatible as possible in ABH, Rh (DCcEe) and Kell 1 antigen systems. Method: A cross-sectional study was carried out on 11,664 blood donors for the ABH system typing and of these, 1255 blood donors were selected randomly for the determination of blood group antigens of the Rh system and Kell antigen. Blood centrifugation methods, centrifuge hemolysi

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Matsui, T., Y. Fujimura, S. Nishida, and K. Titani. "Human plasma alpha 2-macroglobulin and von Willebrand factor possess covalently linked ABO(H) blood group antigens in subjects with corresponding ABO phenotype." Blood 82, no. 2 (1993): 663–68. http://dx.doi.org/10.1182/blood.v82.2.663.663.

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Abstract We recently identified ABO(H) blood group structures in Asn-linked sugar chains of human von Willebrand factor (vWF) purified from factor VIII concentrates (J Biol Chem 267:8723, 1992). We surveyed plasma glycoproteins carrying ABO(H) blood group antigens by Western blotting analysis and sandwich enzyme-linked immunosorbent assay using blood group-specific monoclonal antibodies (MoAbs) and a lectin. Two major plasma proteins showing apparent molecular weight of about 180 Kd and 270 Kd by sodium dodecyl sulfate polyacrylamide gel electrophoresis reacted with blood group-specific MoAbs

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45

Matsui, T., Y. Fujimura, S. Nishida, and K. Titani. "Human plasma alpha 2-macroglobulin and von Willebrand factor possess covalently linked ABO(H) blood group antigens in subjects with corresponding ABO phenotype." Blood 82, no. 2 (1993): 663–68. http://dx.doi.org/10.1182/blood.v82.2.663.bloodjournal822663.

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We recently identified ABO(H) blood group structures in Asn-linked sugar chains of human von Willebrand factor (vWF) purified from factor VIII concentrates (J Biol Chem 267:8723, 1992). We surveyed plasma glycoproteins carrying ABO(H) blood group antigens by Western blotting analysis and sandwich enzyme-linked immunosorbent assay using blood group-specific monoclonal antibodies (MoAbs) and a lectin. Two major plasma proteins showing apparent molecular weight of about 180 Kd and 270 Kd by sodium dodecyl sulfate polyacrylamide gel electrophoresis reacted with blood group-specific MoAbs and Ulex

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Yamamoto, Fumiichiro, and Miyako Yamamoto. "Molecular genetic basis of porcine histo-blood group AO system." Blood 97, no. 10 (2001): 3308–10. http://dx.doi.org/10.1182/blood.v97.10.3308.

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Abstract Histo-blood group A and B antigens are oligosaccharide antigens important in transfusion and transplantation medicine. The final steps in the synthesis of these antigens are catalyzed by glycosyltransferases encoded by the functional alleles at the ABO locus. Humans have 3 major alleles (A, B, and O), whereas pigs are known to have only A and O alleles. This paper reports the molecular genetic basis of the porcine AO system. The porcine A gene is homologous to the ABO genes in humans and other species. It encodes an α1 → 3N-acetyl-D-galactosaminyltransferase that synthesizes A antigen

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Marano, Giuseppe, Stefania Vaglio, Liviana Catalano, Simonetta Pupella, Giancarlo Liumbruno, and Massimo Franchini. "The Role of ABO Blood Type in Thrombosis Scoring Systems." Seminars in Thrombosis and Hemostasis 43, no. 05 (2017): 525–29. http://dx.doi.org/10.1055/s-0037-1599143.

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AbstractIn addition to their major role in transfusion medicine, there is increasing evidence that ABO blood group antigens (complex carbohydrate molecules widely expressed on the surface of red blood cells and several other cell types) are implicated in the development of a wide array of pathologic conditions. In particular, intense research has been dedicated over the last 50 years to the study of the association between non-O blood type and the risk of developing cardiovascular disorders. Several pathways have been hypothesized to explain this relationship, the most reasonable implying the

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Yamamoto, Fumiichiro. "Review: ABO blood group system - ABH oligosaccharide antigens, anti-A and anti-B, A and B glycosyltransferases, and ABO genes." Immunohematology 20, no. 1 (2020): 3–22. http://dx.doi.org/10.21307/immunohematology-2019-418.

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Cohen, Miriam, Nancy Hurtado-Ziola, and Ajit Varki. "ABO blood group glycans modulate sialic acid recognition on erythrocytes." Blood 114, no. 17 (2009): 3668–76. http://dx.doi.org/10.1182/blood-2009-06-227041.

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Abstract ABH(O) blood group polymorphisms are based on well-known intraspecies variations in structures of neutral blood cell surface glycans in humans and other primates. Whereas natural antibodies against these glycans can act as barriers to blood transfusion and transplantation, the normal functions of this long-standing evolutionary polymorphism remain largely unknown. Although microbial interactions have been suggested as a selective force, direct binding of lethal pathogens to ABH antigens has not been reported. We show in this study that ABH antigens found on human erythrocytes modulate

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Vojvodic, Svetlana. "Distribution of ABO, Rh, MNSs, Kell and Duffy blood-group antigens in population of Vojvodina." Medical review 56, no. 3-4 (2003): 173–77. http://dx.doi.org/10.2298/mpns0304173v.

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Introduction Analysis of erythrocyte blood group antigen polymorphisms and genetic variability in population of Vojvodina was performed by investigating gene and genotype frequencies which determine antigens of ABO, Rh, MNSs, Kell and Duffy blood-group systems. Material and methods We investigated 350 unrelated persons from Vojvodina in regard to appurtenance of ABO, Rh, MNSs, Kell and Duffy blood-group systems. We calculated gene, genotype, phenotype frequencies and proportion significance test. Results and discussion Results of investigation revealed that gene and genotype frequencies of inv

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