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Relevant bibliographies by topics / Acanthocyte

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Journal articles

Academic literature on the topic 'Acanthocyte'

Author: Grafiati

Published: 11 March 2023

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Journal articles on the topic "Acanthocyte"

1

Maslak, P. "Acanthocyte." ASH Image Bank 2005, no. 0420 (2005): 101352. http://dx.doi.org/10.1182/ashimagebank-2005-101352.

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2

Darras, Alexis, Kevin Peikert, Antonia Rabe, et al. "Acanthocyte Sedimentation Rate as a Diagnostic Biomarker for Neuroacanthocytosis Syndromes: Experimental Evidence and Physical Justification." Cells 10, no. 4 (2021): 788. http://dx.doi.org/10.3390/cells10040788.

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(1) Background: Chorea-acanthocytosis and McLeod syndrome are the core diseases among the group of rare neurodegenerative disorders called neuroacanthocytosis syndromes (NASs). NAS patients have a variable number of irregularly spiky erythrocytes, so-called acanthocytes. Their detection is a crucial but error-prone parameter in the diagnosis of NASs, often leading to misdiagnoses. (2) Methods: We measured the standard Westergren erythrocyte sedimentation rate (ESR) of various blood samples from NAS patients and healthy controls. Furthermore, we manipulated the ESR by swapping the erythrocytes

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3

Doll, D. C., A. F. List, D. A. Dayhoff, T. S. Loy, Q. S. Ringenberg, and J. W. Yarbro. "Acanthocytosis associated with myelodysplasia." Journal of Clinical Oncology 7, no. 10 (1989): 1569–72. http://dx.doi.org/10.1200/jco.1989.7.10.1569.

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Dysplastic hematopoiesis associated with erythrocyte macrocytosis is a morphologic hallmark of myelodysplasia. We report the cases of six patients with myelodysplasia in which acanthocytosis was the predominant red blood cell (RBC) abnormality. In each case acanthocytes represented 5% to 10% of circulating RBC forms and was the primary reason for referral in two cases. None of the patients had comorbid conditions known to be associated with acanthocyte formation. Myelodysplasia should be considered in the differential diagnosis of acanthocytosis, particularly in the anemic, elderly individual.

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4

Slavova-Azmanova, Neli S., Nicole Kucera, Jiulia Satiaputra, et al. "Gain-of-function Lyn induces anemia: appropriate Lyn activity is essential for normal erythropoiesis and Epo receptor signaling." Blood 122, no. 2 (2013): 262–71. http://dx.doi.org/10.1182/blood-2012-10-463158.

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Key Points Gain-of-function Lyn mice develop hemolytic anemia with acanthocyte red blood cells and display compensatory extramedullary erythropoiesis. Hyperactive Lyn notably alters Epo receptor signaling, particularly an Akt-FoxO3 pathway, enhancing viability and delaying differentiation.

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5

Bayreuther, C., M. Borg, C. Ferrero-Vacher, A. Chaussenot, and C. Lebrun. "Choréo-acanthocytose sans acanthocytes." Revue Neurologique 166, no. 1 (2010): 100–103. http://dx.doi.org/10.1016/j.neurol.2009.03.005.

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6

Rabe, Antonia, Alexander Kihm, Alexis Darras, et al. "The Erythrocyte Sedimentation Rate and Its Relation to Cell Shape and Rigidity of Red Blood Cells from Chorea-Acanthocytosis Patients in an Off-Label Treatment with Dasatinib." Biomolecules 11, no. 5 (2021): 727. http://dx.doi.org/10.3390/biom11050727.

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Background: Chorea-acanthocytosis (ChAc) is a rare hereditary neurodegenerative disease with deformed red blood cells (RBCs), so-called acanthocytes, as a typical marker of the disease. Erythrocyte sedimentation rate (ESR) was recently proposed as a diagnostic biomarker. To date, there is no treatment option for affected patients, but promising therapy candidates, such as dasatinib, a Lyn-kinase inhibitor, have been identified. Methods: RBCs of two ChAc patients during and after dasatinib treatment were characterized by the ESR, clinical hematology parameters and the 3D shape classification in

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7

Shah, J. R., D. P. Patkar, and R. N. Kamat. "A Case of McLeod Phenotype of Neuroacanthocytosis Brain MR Features and Literature Review." Neuroradiology Journal 26, no. 1 (2013): 21–26. http://dx.doi.org/10.1177/197140091302600103.

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Huntington's disease and neuroacanthocytosis may present similar clinical and MRI features. It is important to differentiate these findings since treatment and prognosis vary vastly between them. The aim of this article is to familiarize radiologists with the differentiating features of Huntington's disease and various diseases comprising neuroacanthocytosis. A 40-year-old Indian man with extrapyramidal symptoms was referred for MRI. The clinical diagnosis was Huntington's disease, but there were a few atypical clinical features such as a history of biting the tongue, tics, marked hyporeflexia

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8

Tant, M. S., J. H. Lumsden, R. M. Jacobs, and B. N. Bonnett. "Evaluation of acanthocyte count as a diagnostic test for canine haemangiosarcoma." Comparative Clinical Pathology 12, no. 4 (2004): 174–81. http://dx.doi.org/10.1007/s00580-003-0493-z.

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9

Peikert, Kevin, Hannes Glaß, Enrica Federti, et al. "Targeting Lyn Kinase in Chorea-Acanthocytosis: A Translational Treatment Approach in a Rare Disease." Journal of Personalized Medicine 11, no. 5 (2021): 392. http://dx.doi.org/10.3390/jpm11050392.

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Chorea-acanthocytosis (ChAc) is a neurodegenerative disease caused by mutations in the VPS13A gene. It is characterized by several neurological symptoms and the appearance of acanthocytes. Elevated tyrosine kinase Lyn activity has been recently identified as one of the key pathophysiological mechanisms in this disease, and therefore represents a promising drug target. Methods: We evaluated an individual off-label treatment with the tyrosine kinase inhibitor dasatinib (100 mg/d, 25.8–50.4 weeks) of three ChAc patients. Alongside thorough safety monitoring, we assessed motor and non-motor scales

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10

Sae-Ung, N., P. Srisukkho, S. Archwichaiudom, K. Sairos, Y. Jungtrakul, and S. Anutrakulchai. "Advantage of acanthocyte flag message of UriSed 3 pro automated microscopy urine analyzer." Clinica Chimica Acta 493 (June 2019): S476. http://dx.doi.org/10.1016/j.cca.2019.03.1005.

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