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Relevant bibliographies by topics / Achilles tendinopathy (AT)

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Academic literature on the topic 'Achilles tendinopathy (AT)'

Author: Grafiati

Published: 10 December 2022

Last updated: 28 January 2023

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Journal articles on the topic "Achilles tendinopathy (AT)"

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Roche, A. J., and J. D. F. Calder. "Achilles tendinopathy." Bone & Joint Journal 95-B, no. 10 (October 2013): 1299–307. http://dx.doi.org/10.1302/0301-620x.95b10.31881.

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The two main categories of tendo Achillis tendon disorder are broadly classified by anatomical location to include non-insertional and insertional conditions. Non-insertional Achilles tendinopathy is often managed conservatively, and many rehabilitation protocols have been adapted and modified, with excellent clinical results. Emerging and popular alternative therapies, including a variety of injections and extracorporeal shockwave therapy, are often combined with rehabilitation protocols. Surgical approaches have developed, with minimally invasive procedures proving popular. The management of insertional Achilles tendinopathy is improved by recognising coexisting pathologies around the insertion. Conservative rehabilitation protocols as used in non-insertional disorders are thought to prove less successful, but such methods are being modified, with improving results. Treatment such as shockwave therapy is also proving successful. Surgical approaches specific to the diagnosis are constantly evolving, and good results have been achieved.

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Arya, Shruti, and Kornelia Kulig. "Tendinopathy alters mechanical and material properties of the Achilles tendon." Journal of Applied Physiology 108, no. 3 (March 2010): 670–75. http://dx.doi.org/10.1152/japplphysiol.00259.2009.

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The purpose of this study was to investigate the in vivo material and mechanical properties of the human Achilles tendon in the presence of tendinopathy. Real-time ultrasound imaging and dynamometry were used to assess Achilles tendon stiffness, Young's modulus, stress, strain, and cross-sectional area (CSA) in 12 individuals with Achilles tendinopathy and 12 age- and gender-matched controls. The results of this study suggest that tendinopathy weakens the mechanical and material properties of the tendon. Tendinopathic tendons had greater CSA, lower tendon stiffness, and lower Young's modulus. These alterations in mechanical characteristics may put the Achilles tendon at a higher risk to sustain further injury and prolong the time to recovery. Results from this study may be used to design treatment strategies that specifically target these deficits, leading to faster and permanent recovery from tendinopathy.

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Oh, Seo Yoon, Do Kyung Kim, Soo Hong Han, Hyun Hae Lee, Yunhui Jeong, Minjung Baek, Hyeongkyung Kim, Wooyeol Ahn, and Soonchul Lee. "Sustained Exposure of Substance P Causes Tendinopathy." International Journal of Molecular Sciences 21, no. 22 (November 16, 2020): 8633. http://dx.doi.org/10.3390/ijms21228633.

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Recently, neuromediators such as substance P (SP) have been found to be important factors in tendon homeostasis. Some studies have found SP to be the cause of inflammation and tendinopathy, whereas others have determined it to be a critical component of tendon healing. As demonstrated by these conflicting findings, the effects of SP on tendinopathy remain unclear. In this study, we hypothesized that the duration of SP exposure determines its effect on the tendons, with repetitive long-term exposure leading to the development of tendinopathy. First, we verified the changes in gene and protein expression using in vitro tenocytes with 10-day exposure to SP. SP and SP + Run groups were injected with SP in their Achilles tendon every other day for 14 days. Achilles tendons were then harvested for biomechanical testing and histological processing. Notably, tendinopathic changes with decreased tensile strength, as observed in the Positive Control, were observed in the Achilles in the SP group compared to the Negative Control. Subsequent histological analysis, including Alcian blue staining, also revealed alterations in the Achilles tendon, which were generally consistent with the findings of tendinopathy in SP and SP + Run groups. Immunohistochemical analysis revealed increased expression of SP in the SP group, similar to the Positive Control. In general, the SP + Run group showed worse tendinopathic changes. These results suggest that sustained exposure to SP may be involved in the development of tendinopathy. Future research on inhibiting SP is warranted to target SP in the treatment of tendinopathy and may be beneficial to patients with tendinopathy.

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Yasui, Youichi, Ichiro Tonogai, Andrew J. Rosenbaum, Yoshiharu Shimozono, Hirotaka Kawano, and John G. Kennedy. "The Risk of Achilles Tendon Rupture in the Patients with Achilles Tendinopathy: Healthcare Database Analysis in the United States." BioMed Research International 2017 (2017): 1–4. http://dx.doi.org/10.1155/2017/7021862.

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Introduction. Disorders of the Achilles tendon can be broadly classified into acute and chronic entities. Few studies have established chronic Achilles tendinopathy as a precursor to acute Achilles ruptures. In this study, we assessed the relationship between Achilles tendinopathy and rupture, clarifying the incidence of rupture in the setting of underlying tendinopathy.Methods. The United Healthcare Orthopedic Dataset from the PearlDiver Patient Record Database was used to identify patients with ICD-9 codes for Achilles rupture and/or Achilles tendinopathy. The number of patients with acute rupture, chronic tendinopathy, and rupture following a prior diagnosis of tendinopathy was assessed.Results.Four percent of patients with an underlying diagnosis of Achilles tendinopathy went on to sustain a rupture (7,232 patients). Older patients with tendinopathy were most vulnerable to subsequent rupture.Conclusions. The current study demonstrates that 4.0% of patients who were previously diagnosed with Achilles tendinopathy sustained an Achilles tendon rupture. Additionally, older patients with Achilles tendinopathy were most vulnerable. These findings are important as they can help clinicians more objectively council patients with Achilles tendinopathy.

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Dakin, Stephanie Georgina, Julia Newton, Fernando O. Martinez, Robert Hedley, Stephen Gwilym, Natasha Jones, Hamish A. B. Reid, et al. "Chronic inflammation is a feature of Achilles tendinopathy and rupture." British Journal of Sports Medicine 52, no. 6 (November 8, 2017): 359–67. http://dx.doi.org/10.1136/bjsports-2017-098161.

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BackgroundRecent investigation of human tissue and cells from positional tendons such as the rotator cuff has clarified the importance of inflammation in the development and progression of tendon disease. These mechanisms remain poorly understood in disease of energy-storing tendons such as the Achilles. Using tissue biopsies from patients, we investigated if inflammation is a feature of Achilles tendinopathy and rupture.MethodsWe studied Achilles tendon biopsies from symptomatic patients with either mid-portion tendinopathy or rupture for evidence of abnormal inflammatory signatures. Tendon-derived stromal cells from healthy hamstring and diseased Achilles were cultured to determine the effects of cytokine treatment on expression of inflammatory markers.ResultsTendinopathic and ruptured Achilles highly expressed CD14+ and CD68+ cells and showed a complex inflammation signature, involving NF-κB, interferon and STAT-6 activation pathways. Interferon markers IRF1 and IRF5 were highly expressed in tendinopathic samples. Achilles ruptures showed increased PTGS2 and interleukin-8 expression. Tendinopathic and ruptured Achilles tissues expressed stromal fibroblast activation markers podoplanin and CD106. Tendon cells isolated from diseased Achilles showed increased expression of pro-inflammatory and stromal fibroblast activation markers after cytokine stimulation compared with healthy hamstring tendon cells.ConclusionsTissue and cells derived from tendinopathic and ruptured Achilles tendons show evidence of chronic (non-resolving) inflammation. The energy-storing Achilles shares common cellular and molecular inflammatory mechanisms with functionally distinct rotator cuff positional tendons. Differences seen in the profile of ruptured Achilles are likely to be attributable to a superimposed phase of acute inflammation and neo-vascularisation. Strategies that target chronic inflammation are of potential therapeutic benefit for patients with Achilles tendon disease.

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Koike, Yoichi, Hans K. Uhthoff, Nanthan Ramachandran, Geoffrey P. Doherty, Martin Lecompte, David S. Backman, and Guy Trudel. "Achilles Tendinopathy." Critical Reviews in Physical and Rehabilitation Medicine 16, no. 2 (2004): 109–32. http://dx.doi.org/10.1615/critrevphysrehabilmed.v16.i2.30.

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Tabane, Ck. "Achilles tendinopathy." South African General Practitioner 3, no. 1 (March 2022): 22–25. http://dx.doi.org/10.36303/sagp.2022.3.1.0109.

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Kaplan, Kelby, Ovidio Olivencia, Melissa Dreger, William J. Hanney, and Morey J. Kolber. "Achilles Tendinopathy." Strength and Conditioning Journal 41, no. 5 (October 2019): 24–40. http://dx.doi.org/10.1519/ssc.0000000000000485.

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PAAVOLA, MIKA, PEKKA KANNUS, TERO A. H. JÄRVINEN, KARIM KHAN, LÁSZLO JÓZSA, and MARKKU JÄRVINEN. "ACHILLES TENDINOPATHY." Journal of Bone and Joint Surgery-American Volume 84, no. 11 (November 2002): 2062–76. http://dx.doi.org/10.2106/00004623-200211000-00024.

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Cook, J. L., K. M. Khan, and C. Purdam. "Achilles tendinopathy." Manual Therapy 7, no. 3 (August 2002): 121–30. http://dx.doi.org/10.1054/math.2002.0458.

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Dissertations / Theses on the topic "Achilles tendinopathy (AT)"

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Costa, Matthew. "Achilles tendinopathy and rupture." Thesis, University of East Anglia, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.426770.

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Grävare, Silbernagel Karin. "Achilles tendinopathy : evaluation and treatment /." Göteborg : Department of Orthopaedics, Institute of Clinical Sciences, The Sahlgrenska Academy at Göteborg University, 2006. http://hdl.handle.net/2077/504.

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Åström, Mats. "On the nature and etiology of chronic achilles tendinopathy." Lund : Dept. of Orthopaedics, Malmö University Hospital, Lund University, 1997. http://catalog.hathitrust.org/api/volumes/oclc/39705581.html.

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Shalabi, Adel. "Magnetic resonance imaging in chronic achilles tendinopathy /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7349-811-4/.

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O'Neill, Seth. "A biomechanical approach to Achilles tendinopathy management." Thesis, University of Leicester, 2017. http://hdl.handle.net/2381/40134.

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Achilles tendinopathy (AT) is a debilitating condition affecting active and inactive subjects. The aetiology of AT is contentious but most theoretical models are underpinned by the same core elements: increased tendon stress that exceeds the ability of the tendon to repair, eventually leading to tissue degradation. Proposed risk factors affect tissue load or the ability of the tendon to repair, unfortunately many proposed risk factors lack biological plausibility. This thesis reports a mixed methods approach to analyse risk factors for AT development. A Delphi study (using world tendon experts) was used to develop a consensus on risk factors for AT which were then used in an epidemiological study of UK runners – a high risk group for tendinopathy who have not previously been examined. Plantarflexor strength was identified as a potential contributor in AT development and this led to the development of a theory on why current treatment modalities work. This was further examined using a systematic review to determine whether changes occur in the muscle or tendon with the most common rehabilitation method (eccentric exercise). This review highlighted evidence for changes in plantaflexor power, amongst other factors. These findings were used to inform two studies examining how plantarflexor power was affected by AT and ultimately how rehabilitation altered these elements. The thesis reports multiple novel findings related to risk factors, the mechanism by which eccentric exercise may work, the effect of AT on plantarflexor function and how rehabilitation affects these deficits. The experimental studies contained in this thesis are some of the largest studies in the area, these studies identify significant torque and endurance deficits of the plantarflexors highlighting that Soleus appears to be most affected, whilst the later study reported how an eccentric rehabilitation regime improved these deficits but failed to fully resolve them. This offers an important clinical target but also highlights that rehabilitation needs to be for a longer duration with further modifications to resolve these identified plantarflexor deficits.

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Crouzier, Marion. "Muscle coordination and musculoskeletal disorders : investigation of Achilles tendinopathy." Thesis, Nantes, 2020. http://www.theses.fr/2020NANT2013.

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Le tendon d'Achille est composé de trois sous-tendons qui proviennent chacun d'un chef du triceps surae: gastrocnemius medialis, gastrocnemius lateralis, et soleus. Une distribution non optimale de la contrainte imposée au tendon d'Achille contribuerait au développement de la tendinopathie d'Achille. D’autre part, la distribution des contraintes sur le tendon d'Achille est en partie déterminée par la distribution de la force entre les chefs des triceps surae. L'objectif général de cette thèse était d’étudier le rôle de la coordination musculaire (définie comme la distribution de force entre les muscles) dans le développement d’une tendinopathie d'Achille. Les forces musculaires de chaque muscle ont été estimées à partir de l'activation, du volume et de l'architecture musculaires. Les résultats ont montré que (i) la distribution de l’activation musculaire dans le triceps surae est robuste dans le temps, et varie considérablement d'un individu à l'autre; (ii) il existe une corrélation positive significative entre la distribution de la surface de section transversale physiologique et la distribution de l'activation au sein des gastrocnemii; (iii) le gastrocnemius lateralis contribue significativement moins à la force totale produite par le triceps sural chez les personnes atteintes de tendinopathie d'Achille par rapport aux contrôles. L’altération de la coordination musculaire pourrait être une cause, comme elle pourrait être une conséquence de la tendinopathie d’Achille
The Achilles tendon is made of three subtendons that each arises from a different head of the triceps surae: gastrocnemius medialis, gastrocnemius lateralis and soleus. Non-optimal distribution of load within Achilles tendon would contribute to the development of Achilles tendinopathy. Moreover, there is evidence that the distribution of load or strain within the Achilles tendon is partly determined by the distribution of force among the heads of the triceps surae. The overall aim of this thesis was to provide a deeper understanding of the role of muscle coordination (i.e. the distribution of force among muscles) on the development of Achilles tendinopathy. Individual muscle forces were estimated from muscle activation, volume and architecture. Results showed that (i) the distribution of activation among triceps surae is robust between days, and varies greatly between individuals; (ii) there is a significant positive correlation between the distribution of physiological cross-sectional area and the distribution of activation among gastrocnemii; (iii) muscle coordination among the triceps surae differs in people with Achilles tendinopathy compared with controls, with the gastrocnemius lateralis contributing significantly less to total triceps surae force in people with Achilles tendinopathy. Whether this altered strategy is a cause or a consequence of Achilles tendinopathy should be further explored

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Saunders, Colleen Jayne. "Extracellular matrix gene sequence variant analyses and Achilles tendinopathy." Doctoral thesis, University of Cape Town, 2013. http://hdl.handle.net/11427/3199.

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Includes abstract.
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The primary aim of this thesis was to identify additional genetic elements predisposing individuals to risk of AT using a candidate gene, case-control genetic association approach, and to propose the biological mechanisms underlying this genetic risk. Candidate genes (COMP, THBS2, COL27A1, TNC, COL3A1, COL5A2 and COL5A3) were selected based on their chromosomal location and/or the biological function of their encoded proteins within the extracellular matrix (ECM) of the tendon.

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Hutchison, Anne-Marie. "The treatment and management of patients with a chronic mid body achilles tendinopathy." Thesis, Swansea University, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.678301.

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Azevedo, Liane. "Risk factors for Achilles tendinopathy in runners - an investigation of selected intrinsic, kinematic, kinetic and muscle activity factors that are associated with Achilles tendinopathy." Doctoral thesis, University of Cape Town, 2008. http://hdl.handle.net/11427/2772.

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The overarching purpose of this study was to investigate the multifactorial aetiology of Achilles tendinopathy. Variables such as training and injury history, lower limb alignment, running biomechanics and movement variability were investigated. This study also aims to understand how different sensory inputs, such as shoe or pain may affect biomechanics. Thirty four uninjured runners (UN) and twenty one runners with Achilles tendinopathy (AT) composed the population sample for this study. Questionnaire and lower limb measurements were used to investigate the multifactorial aetiology of the injury. Selected kinetic, kinematic and muscle activity parameters were employed to analyse the biomechanics aetiology of the injury. Runners performed 10 running trials at a self-selected speed in two running shoes with different hardness. Additionally, the UN runners ran for 10 min on the treadmill while the AT runners ran on the treadmill until they developed pain in the Achilles tendon. The results of the study showed that AT runners have a higher incidence of previous injury, run for more years, have reduced hamstring flexibility, and are heavier and taller than uninjured runners. The study also showed a reduced integrated electromyography activity (IEMG) of tibialis anterior and rectus femoris in the AT group during the running cycle. Stride to stride variability was similar between UN and AT runners but the biomechanics variability between participants were lower for the AT runners. No specific biomechanical adaptations were found between the two different shoe conditions (soft vs. hard). Similarly, biomechanics parameters were not altered at the onset of pain, but the reduced IEMG activity of tibialis anterior and rectus femoris were presented before and during the pain condition. It can, therefore, be concluded that runners with Achilles tendinopathy present reduced muscle activity during running which may be either a novel aetiological factor, or an adaptive response to the injury. The lower variability between runners with AT may indicate that these runners are less able to adjust their biomechanics according to their different functional behaviour or external input signals but this may require further investigation. Finally, it can also be concluded that the sensory inputs such as shoes and pain do not change this muscle activity pattern.

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Bjur, Dennis. "The human Achilles tendon : innervation and intratendinous production of nerve signal substances - of importance in understanding the processes of Achilles tendinosis." Doctoral thesis, Umeå universitet, Idrottsmedicin, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-30284.

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Tendinopathies are painful tendon conditions of presumably multifactorial genesis. In tendinosis, as in Achilles tendinosis, there is apart from pain also morphological changes which are described as degenerative with no signs of inflammation. The exact mechanisms behind these conditions are still, to a large extent, unknown. Pain, being the foremost impairing symptom, leads us to the hypothesis that nerves are deeply involved in the symptoms and processes of Achilles tendinosis. Locally produced nerve signal substances may also be involved in the processes. Knowledge of the innervation patterns within the tendon and knowledge on a possible local nerve signal substance production are therefore of utmost importance. There is a lack of information on these aspects. The specific aims of this thesis were 1) to investigate the innervation patterns regarding general, sensory, cholinergic and sympathetic innervations, and 2) to examine for the possible occurrence of a production of nerve signal substances and a presence of receptors related to these in the tendon cells, the tenocytes. Painfree normal and tendinosis Achilles tendons were examined. Immunohistochemistry, using antibodies against the general nerve marker PGP9.5, the synthesizing enzymes for acetylcholine (choline acetyltransferase; ChAT), and catecholamines (tyrosine hydroxylase; TH), the vesicular acetylcholine transporter (VAChT), neuropeptide Y (NPY), substance P and calcitonin gene-related peptide, was applied. Immunohistochemistry was also used for the delineation of muscarinic (M2R), adrenergic (α1-AR) and NPY-ergic (Y1 and Y2) receptors. To detect mRNA for TH and ChAT, in situ hybridization was used. In normal Achilles tendons, as well as in the tendinosis tendons, there was a very scanty innervation within the tendon tissue proper, the main general, sensory and sympathetic innervations being found in the paratendinous loose connective tissue. Interestingly, the tenocytes showed immunoreactions for ChAT, VAChT, TH, M2R, α1-AR and Y1R. The reactions were clearly more observable in tendons of tendinosis patients than in those of controls. The tenocytes of tendinosis patients also displayed mRNA reactions for ChAT and TH. Nevertheless, all tenocytes in the tendinosis specimens did not show these reactions. Immunoreactions for α1-AR, M2R and Y1R were also seen for blood vessel walls. The present thesis shows that there is a very limited innervation within tendon tissue proper, whilst there is a substantial innervation in the paratendinous loose connective tissue. It also gives evidence for an occurrence of production of catecholamines and acetylcholine in tenocytes, especially for tendinosis tendons. Furthermore, that ACh, catecholamines and NPY can have effects on these, as well as on blood vessels, via the receptors observed. The observations suggest that Achilles tendon tissue, whilst containing a very scarce innervation, exhibits autocrine/paracrine cholinergic/catecholaminergic/NPY-ergic effects that are upregulated in tendinosis. These findings are of great importance as the results of such effects may mimic processes that are known to occur in tendinosis. That includes effects related to proliferation and angiogenesis, and blood vessel and collagen regulating effects. In conclusion, within the Achilles tendon there is a very scarce innervation, whilst there appears to be a marked local production of nerve signal substances in Achilles tendinosis, namely in the tenocytes, the cells also harbouring receptors for these substances. The observations give a new insight into how the tendon tissue of the Achilles tendon is influenced by signal substances and may give options for new treatments of Achilles tendinosis.

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Books on the topic "Achilles tendinopathy (AT)"

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Achilles tendon. Rijeka: InTech, 2012.

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service), SpringerLink (Online, ed. The Achilles Tendon: Treatment and Rehabilitation. New York, NY: Springer-Verlag New York, 2009.

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Bennett, Paul. My Achilles' Heel: How to Overcome Achilles Tendinopathy. Lulu Press, Inc., 2014.

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Jessica, Terry. Healing Achilles Tendinopathy: The Beginners Guide to Achilles Tendinopathy Cure, Treatment, Management and Recovery for Your Complete Wellness. Independently Published, 2022.

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Lawrence, Daniel. Lower Limb Tendinopathy: Achilles, Patellar, Hamstring and Gluteal. Independently Published, 2018.

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Peers, Koen. Extracorporeal Shock Wave Therapy in Chronic Achilles & Patellar Tendinopathy. Leuven Univ Pr, 2003.

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Nunley, James A. The Achilles Tendon: Treatment and Rehabilitation. Springer, 2010.

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Book chapters on the topic "Achilles tendinopathy (AT)"

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Schaaf, Stephen, Ma Calus V. Hogan, and Adam S. Tenforde. "Achilles Tendon." In Tendinopathy, 251–63. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-65335-4_16.

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de Vos, Robert Jan, Pieter P. R. N. d’Hooghe, Peter de Leeuw, and Gino M. M. J. Kerkhoffs. "Achilles Tendinopathy." In Sports and Traumatology, 213–33. Paris: Springer Paris, 2014. http://dx.doi.org/10.1007/978-2-8178-0523-8_19.

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Maffulli, Nicola, Umile Giuseppe Longo, Stefano Campi, and Vincenzo Denaro. "Achilles Tendinopathy." In Evidence-Based Orthopedics, 872–78. Oxford, UK: Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444345100.ch102.

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Yang, Ajax. "Achilles Tendinopathy." In Musculoskeletal Sports and Spine Disorders, 291–93. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-50512-1_66.

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Hunt, Kenneth J. "Achilles Tendinopathy." In Muscle and Tendon Injuries, 345–54. Berlin, Heidelberg: Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-54184-5_33.

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Orlandi, Davide, and Enzo Silvestri. "Achilles Tendinopathy." In Ultrasound-guided Musculoskeletal Procedures, 87–90. Milano: Springer Milan, 2015. http://dx.doi.org/10.1007/978-88-470-5764-7_13.

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Maffulli, Nicola, and Francesco Oliva. "Achilles Tendinopathy." In European Instructional Lectures, 191–99. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-11832-6_17.

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Maffulli, Nicola, Alessio Giai Via, and Francesco Oliva. "Achilles Tendinopathy." In The Lower Limb Tendinopathies, 149–64. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-33234-5_10.

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Canata, Gian Luigi, and Valentina Casale. "Insertional Achilles Tendinopathy." In Sports Injuries of the Foot and Ankle, 349–57. Berlin, Heidelberg: Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-58704-1_31.

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Aicale, Rocco, and Nicola Maffulli. "Non-insertional Achilles Tendinopathy." In Foot and Ankle Disorders, 855–67. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-95738-4_38.

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Conference papers on the topic "Achilles tendinopathy (AT)"

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Elmallah, Reem, and Enas Elattar. "AB0912 EXTRACORPOREAL SHOCKWAVE VERSUS MUSCULOSKELETAL MESOTHERAPY FOR ACHILLES TENDINOPATHY IN ATHLETES." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.4818.

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Thornton, Gail M., Xinxin Shao, May Chung, Paul Sciore, Richard S. Boorman, David A. Hart, and Ian K. Y. Lo. "Mechanical Loading Uniquely Up-Regulates MMP-13 in Rat Supraspinatus Tendon But Not Achilles Tendon." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-173778.

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Tendon injuries and chronic pain, known as tendinopathy, are extremely common in supraspinatus and Achilles tendons. Despite their prevalence, the pathogenesis of these conditions is poorly understood. One of most common cited factors for injuries to the rotator cuff tendons is “impingement”, namely, mechanical compression of the supraspinatus tendon by the overlying coracoacromial arch [1]. To date, few studies have investigated the molecular processes underlying this condition.

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Tumilty, Steve, Joanne Munn, J. Haxby Abbott, Suzanne Mcdonough, Deirdre A. Hurley, Jeffrey R. Basford, G. David Baxter, and Leonardo Longo. "Laser Therapy in the Treatment of Achilles Tendinopathy: A Randomised Controlled Trial." In LASER FLORENCE 2009: A Gallery Through the Laser Medicine World. AIP, 2010. http://dx.doi.org/10.1063/1.3453776.

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Corrigan, Patrick, and Karin Grävare Silbernagel. "19 Do clinical measures relate to running asymmetries in patients with achilles tendinopathy?" In Scandinavian Sports Medicine Congress. BMJ Publishing Group Ltd and British Association of Sport and Exercise Medicine, 2019. http://dx.doi.org/10.1136/bjsports-2019-scandinavianabs.19.

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Connizzo, Brianne K., Kenneth W. Liechty, and Louis J. Soslowsky. "Altered Mechanical Properties and Fiber Re-Alignment in Diabetic Mouse Supraspinatus and Achilles Tendons." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80129.

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Tendons function to transfer load, maintain alignment and permit motion in joints. To perform these functions, tendons have complex mechanical behavior which is modulated by the tissue’s structure and composition, such as the collagen fibers and surrounding extracellular matrix. When these matrix proteins are altered, the mechanical properties are also altered, which could potentially lead to reduced loading and healing capacity. Diabetes is a metabolic disease which, among other co-morbidities, has been associated with Achilles tendon disorganization and tendinopathy, as well as increased overall joint stiffness in humans [1]. We have recently reported that the skin from the Db/Db diabetic mouse, a model of Type II Diabetes, as well as the skin from human diabetics, have impaired biomechanical properties compared to non-diabetic skin as the result of altered extracellular matrix composition. [2]. However, the mechanical properties of tendons from these animals have never been studied and could serve as a unique model of altered collagen structure as well as provide further understanding to the cause of tendinous injuries in the diabetic population. Therefore, the objective of this study is to measure the tensile mechanical properties and collagen fiber re-alignment in the db/db mouse model compared to non-diabetic controls. We hypothesize that tendon stiffness and modulus will be increased in the db/db group in the insertion site and midsubstance, and that db/db tendons will re-align earlier and faster during the testing protocol.

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Boesen, Anders Ploug, Henning Langberg, Rudi Hansen, Peter Malliaras, and Morten Ilum Boesen. "19 High-volume injection with and without corticosteroid in chronic midportion achilles tendinopathy – a randomised double blinded prospective study." In Abstracts from the Scandinavian Congress of Medicine and science in Sports, 2018. BMJ Publishing Group Ltd and British Association of Sport and Exercise Medicine, 2018. http://dx.doi.org/10.1136/bjsports-2018-099334.19.

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Färnqvist, Kenneth, Amit Chauhan Anuj, Dylan Morrissey, and Peter Malliaras. "12 Identification of prognostic factors for patient outcomes during exercise intervention for achilles tendinopathy: a systematic review and meta-analysis." In Abstracts from the Scandinavian Congress of Medicine and science in Sports, 2018. BMJ Publishing Group Ltd and British Association of Sport and Exercise Medicine, 2018. http://dx.doi.org/10.1136/bjsports-2018-099334.12.

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Iglesias, Javier González, Aitor Ruiz de Lara Osacar, Carlos Fernandez Gonzalez, Javier Teijeiro López, Manuel Mira Llopis, and Serena Iannone Lado. "C0088 Ultrasound-guided intratissue percutaneous electrolysis, dry-needling, diathermy and eccentric exercise in achilles tendinopathy in runners. a case series." In 2nd Rehabilitative Ultrasound Imaging Symposium in Physical Therapy, Madrid, Spain, 3–5 June 2016. BMJ Publishing Group Ltd and British Association of Sport and Exercise Medicine, 2018. http://dx.doi.org/10.1136/bjsports-2018-099763.35.

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Haleem, MSc Zubair, Stephen Kelly, Dev Pyne, Simon Lack, and Dylan Morrissey. "30 A new protocol for sonographic evaluation of mid-portion achilles tendinopathy: a test retest intra and inter-rater reliability study." In Scandinavian Sports Medicine Congress. BMJ Publishing Group Ltd and British Association of Sport and Exercise Medicine, 2019. http://dx.doi.org/10.1136/bjsports-2019-scandinavianabs.30.

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Marco, Sergio Romero. "C0059 A proposal cross-sectional analytic study with repeated measures: the role of hypertrophy plantaris muscle belly as a possible cause of achilles tendinopathy and plantar fasciosis in athletes." In 2nd Rehabilitative Ultrasound Imaging Symposium in Physical Therapy, Madrid, Spain, 3–5 June 2016. BMJ Publishing Group Ltd and British Association of Sport and Exercise Medicine, 2018. http://dx.doi.org/10.1136/bjsports-2018-099763.15.

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